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Journal articles on the topic 'Fat metabolism'

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1

Pei, Liming, and Ronald M. Evans. "Retrofitting Fat Metabolism." Cell Metabolism 9, no. 6 (2009): 483–84. http://dx.doi.org/10.1016/j.cmet.2009.05.006.

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2

Leslie, Eric, Christine Mermier, and Len Kravitz. "Exercise and Fat Metabolism." ACSM'S Health & Fitness Journal 26, no. 3 (2022): 34–39. http://dx.doi.org/10.1249/fit.0000000000000768.

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3

Landecker, Hannah. "Postindustrial Metabolism: Fat Knowledge." Public Culture 25, no. 3 (2013): 495–522. http://dx.doi.org/10.1215/08992363-2144625.

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4

Gleeson, Michael. "Basic metabolism I: fat." Surgery (Oxford) 23, no. 3 (2005): 83–88. http://dx.doi.org/10.1383/surg.23.3.83.63111.

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5

Samra, J. S., L. K. M. Summers, and K. N. Frayn. "Sepsis and fat metabolism." British Journal of Surgery 83, no. 9 (1996): 1186–96. http://dx.doi.org/10.1046/j.1365-2168.1996.02445.x.

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6

McAndrew, Philomena F. "Fat Metabolism and Cancer." Surgical Clinics of North America 66, no. 5 (1986): 1003–12. http://dx.doi.org/10.1016/s0039-6109(16)44037-5.

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7

Vogan, Kyle. "TM6SF2 and fat metabolism." Nature Genetics 46, no. 7 (2014): 665. http://dx.doi.org/10.1038/ng.3023.

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8

Samra, J. S., L. K. M. Summers, and K. N. Frayn. "Sepsis and fat metabolism." British Journal of Surgery 83, no. 9 (1996): 1186–96. http://dx.doi.org/10.1002/bjs.1800830906.

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9

Jeukendrup, A. E., and R. Randell. "Fat burners: nutrition supplements that increase fat metabolism." Obesity Reviews 12, no. 10 (2011): 841–51. http://dx.doi.org/10.1111/j.1467-789x.2011.00908.x.

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10

Gimble, Jeffrey M., and Z. Elizabeth Floyd. "Fat circadian biology." Journal of Applied Physiology 107, no. 5 (2009): 1629–37. http://dx.doi.org/10.1152/japplphysiol.00090.2009.

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While adipose tissue has long been recognized for its major role in metabolism, it is now appreciated as an endocrine organ. A growing body of literature has emerged that identifies circadian mechanisms as a critical regulator of adipose tissue differentiation, metabolism, and adipokine secretory function in both health and disease. This concise review focuses on recent data from murine and human models that highlights the interplay between the core circadian regulatory proteins and adipose tissue in the context of energy, fat, and glucose metabolism. It will be important to integrate circadia
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11

Egashira, Yukari, and Hiroo Sanada. "Dietary Fat and Tryptophan Metabolism." Nippon Eiyo Shokuryo Gakkaishi 55, no. 6 (2002): 357–60. http://dx.doi.org/10.4327/jsnfs.55.357.

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12

Moro, Cedric. "Natriuretic peptides and fat metabolism." Current Opinion in Clinical Nutrition and Metabolic Care 16, no. 6 (2013): 645–49. http://dx.doi.org/10.1097/mco.0b013e32836510ed.

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13

Campbell, P. J., M. G. Carlson, and N. Nurjhan. "Fat metabolism in human obesity." American Journal of Physiology-Endocrinology and Metabolism 266, no. 4 (1994): E600—E605. http://dx.doi.org/10.1152/ajpendo.1994.266.4.e600.

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Excessive fat turnover and oxidation might cause the insulin resistance of carbohydrate metabolism in obese humans. We studied the response of free fatty acid (FFA) metabolism in lean and obese volunteers to sequential insulin infusions of 4, 8, 25, and 400 mU.m-2.min-1. The insulin dose-response curves for suppression of FFA concentration, FFA turnover ([1-14C]palmitate), and lipolysis ([2H5]glycerol) were shifted to the right in the obese subjects (insulin concentrations that produced a half-maximal response, lean vs. obese: 103 +/- 21 vs. 273 +/- 41, 96 +/- 11 vs. 264 +/- 44, and 101 +/- 23
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14

BJÖRNTORP, PER. "Fat Cell Distribution and Metabolism." Annals of the New York Academy of Sciences 499, no. 1 (2006): 66–72. http://dx.doi.org/10.1111/j.1749-6632.1987.tb36198.x.

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15

&NA;. "Free Communication/Poster - Fat Metabolism." Medicine & Science in Sports & Exercise 40, Supplement (2008): 47. http://dx.doi.org/10.1249/01.mss.0000321008.13304.48.

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16

Blaak, Ellen. "Gender differences in fat metabolism." Current Opinion in Clinical Nutrition and Metabolic Care 4, no. 6 (2001): 499–502. http://dx.doi.org/10.1097/00075197-200111000-00006.

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17

Yip, Rupert G. C., and M. Michael Wolfe. "GIF biology and fat metabolism." Life Sciences 66, no. 2 (1999): 91–103. http://dx.doi.org/10.1016/s0024-3205(99)00314-8.

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18

Cray, S. "Fat metabolism during propofol infusion." British Journal of Anaesthesia 82, no. 3 (1999): 473. http://dx.doi.org/10.1093/bja/82.3.473.

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19

Machann, Jürgen, Andreas Fritsche, and Fritz Schick. "New Insights into Fat Metabolism." German Research 27, no. 3 (2005): 21–23. http://dx.doi.org/10.1002/germ.200590028.

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20

Ranneries, Claudia, Jens Bülow, Benjamin Buemann, Niels Juel Christensen, Joop Madsen, and Arne Astrup. "Fat metabolism in formerly obese women." American Journal of Physiology-Endocrinology and Metabolism 274, no. 1 (1998): E155—E161. http://dx.doi.org/10.1152/ajpendo.1998.274.1.e155.

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An impaired fat oxidation has been implicated to play a role in the etiology of obesity, but it is unclear to what extent impaired fat mobilization from adipose tissue or oxidation of fat is responsible. The present study aimed to examine fat mobilization from adipose tissue and whole body fat oxidation stimulated by exercise in seven formerly obese women (FO) and eight matched controls (C). Lipolysis in the periumbilical subcutaneous adipose tissue, whole body energy expenditure (EE), and substrate oxidation rates were measured before, during, and after a 60-min bicycle exercise bout of moder
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21

Ko, Seong-Hee, and YunJae Jung. "Energy Metabolism Changes and Dysregulated Lipid Metabolism in Postmenopausal Women." Nutrients 13, no. 12 (2021): 4556. http://dx.doi.org/10.3390/nu13124556.

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Aging women experience hormonal changes, such as decreased estrogen and increased circulating androgen, due to natural or surgical menopause. These hormonal changes make postmenopausal women vulnerable to body composition changes, muscle loss, and abdominal obesity; with a sedentary lifestyle, these changes affect overall energy expenditure and basal metabolic rate. In addition, fat redistribution due to hormonal changes leads to changes in body shape. In particular, increased bone marrow-derived adipocytes due to estrogen loss contribute to increased visceral fat in postmenopausal women. Enha
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22

Williams, Christine M. "Lipid metabolism in women." Proceedings of the Nutrition Society 63, no. 1 (2004): 153–60. http://dx.doi.org/10.1079/pns2003314.

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Differences in whole-body lipid metabolism between men and women are indicated by lower-body fat accumulation in women but more marked accumulation of fat in the intra-abdominal visceral fat depots of men. Circulating blood lipid concentrations also show gender-related differences. These differences are most marked in premenopausal women, in whom total cholesterol, LDL-cholesterol and triacylglycerol concentrations are lower and HDL-cholesterol concentration is higher than in men. Tendency to accumulate body fat in intra-abdominal fat stores is linked to increased risk of CVD, metabolic syndro
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23

Corvera, Silvia. "Perinatal fat progenitors shape adult metabolism." Nature Metabolism 4, no. 8 (2022): 963–64. http://dx.doi.org/10.1038/s42255-022-00626-5.

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24

Wang, Thomas J. "The Natriuretic Peptides and Fat Metabolism." New England Journal of Medicine 367, no. 4 (2012): 377–78. http://dx.doi.org/10.1056/nejmcibr1204796.

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25

Carlson, Grant W. "The Breast, Fat, and Steroid Metabolism." Plastic and Reconstructive Surgery 123, no. 3 (2009): 117e—118e. http://dx.doi.org/10.1097/prs.0b013e31819a35db.

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26

"Internal Body Clock Controls Fat Metabolism." Asian Journal of Cell Biology 6, no. 1 (2010): 24. http://dx.doi.org/10.3923/ajcb.2011.24.24.

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27

&NA;. "Free Communication/Slide - Fat Metabolism 1." Medicine & Science in Sports & Exercise 40, Supplement (2008): 36. http://dx.doi.org/10.1249/01.mss.0000320804.73699.a1.

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28

&NA;. "Free Communication/Slide - Fat Metabolism 2." Medicine & Science in Sports & Exercise 40, Supplement (2008): 51. http://dx.doi.org/10.1249/01.mss.0000321073.88564.ba.

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29

Wiener, Michael, Michael M. Rothkopf, Gail Rothkopf, and Jeffrey Askanazi. "Fat Metabolism in Injury and Stress." Critical Care Clinics 3, no. 1 (1987): 25–56. http://dx.doi.org/10.1016/s0749-0704(18)30560-8.

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30

Kim, Eun Ju, Yeon Kyung Kim, Ji Eun Kim, et al. "UV Modulation of Subcutaneous Fat Metabolism." Journal of Investigative Dermatology 131, no. 8 (2011): 1720–26. http://dx.doi.org/10.1038/jid.2011.106.

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31

Piché, Marie-Eve, and Paul Poirier. "Obesity, ectopic fat and cardiac metabolism." Expert Review of Endocrinology & Metabolism 13, no. 4 (2018): 213–21. http://dx.doi.org/10.1080/17446651.2018.1500894.

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32

Hwang, S., LK Sarna, YL Siow, and K O. "High Fat Diet Disrupts Homocysteine Metabolism." Canadian Journal of Cardiology 29, no. 10 (2013): S168—S169. http://dx.doi.org/10.1016/j.cjca.2013.07.256.

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33

Loftus, Thomas M. "Introduction: Fat metabolism and adipose homeostasis." Seminars in Cell & Developmental Biology 10, no. 1 (1999): 1–2. http://dx.doi.org/10.1006/scdb.1998.0270.

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34

Deitel, M. "It's a Fat, Fat, Fat, Fat World!" Obesity Surgery 14, no. 7 (2004): 869–70. http://dx.doi.org/10.1381/0960892041719536.

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35

Lecka-Czernik, Beata. "Marrow fat metabolism is linked to the systemic energy metabolism." Bone 50, no. 2 (2012): 534–39. http://dx.doi.org/10.1016/j.bone.2011.06.032.

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36

FARNWORTH, E. R., and J. K. G. KRAMER. "FAT METABOLISM IN GROWING SWINE: A REVIEW." Canadian Journal of Animal Science 67, no. 2 (1987): 301–18. http://dx.doi.org/10.4141/cjas87-029.

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At birth, piglets have little body fat that can be mobilized. The influx of high-fat milk causes a rapid increase in body fat stores and a depression of lipogenic enzyme activity. Conversely, lipolytic enzyme activity increases after birth. Changing the fat intake of sucking piglets affects the amount of fat deposition. The length of the sucking period also influences body composition at weaning. Weaning produces a pronounced but temporary decrease in total body lipid, despite an increase in fat synthesis. The effect of weaning on lipolysis is not clear due to a lack of experimental data. Duri
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37

Liu, Li, Qingbo Kong, Zhuoya Xiang, et al. "Integrated Analysis of Transcriptome and Metabolome Provides Insight into Camellia oleifera Oil Alleviating Fat Accumulation in High-Fat Caenorhabditis elegans." International Journal of Molecular Sciences 24, no. 14 (2023): 11615. http://dx.doi.org/10.3390/ijms241411615.

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Camellia oil (CO) is a high medicinal and nutritional value edible oil. However, its ability to alleviate fat accumulation in high-fat Caenorhabditis elegans has not been well elucidated. Therefore, this study aimed to investigate the effect of CO on fat accumulation in high-fat C. elegans via transcriptome and metabolome analysis. The results showed that CO significantly reduced fat accumulation in high-fat C. elegans by 10.34% (Oil Red O method) and 11.54% (TG content method), respectively. Furthermore, CO primarily altered the transcription levels of genes involved in longevity regulating p
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38

Cardoso, Filipa. "The brain-fat connection." Science 378, no. 6619 (2022): 485. http://dx.doi.org/10.1126/science.ade2132.

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39

Van Horn, Linda. "Metabolism: Assessment and Applications Vary by Fat and Fat-Free Mass." Journal of the American Dietetic Association 111, no. 11 (2011): 1641. http://dx.doi.org/10.1016/j.jada.2011.09.019.

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40

Han, Gi Ppeum, Geun Hyeon Park, Jong Hyuk Kim, Hyeon Seok Choi, Hwan Ku Kang, and Dong Yong Kil. "PSVII-19 Transcriptomic analysis of the liver of laying hens fed diets containing supplemental fat at early laying stages." Journal of Animal Science 97, Supplement_3 (2019): 353. http://dx.doi.org/10.1093/jas/skz258.703.

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Abstract Feeding diets containing supplemental fat to laying hens is reported to ameliorate pathogenesis of fatty liver hemorrhagic syndrome (FLHS). However, molecular mechanisms for this positive effect have not been investigated. Thus, we conducted a transcriptomic analysis of the liver of laying hens fed diets containing supplemental fat at early laying stages. Two dietary treatments included basal diets with no supplemental fat and basal diets supplemented with 3.0% tallow. A total of 256 18-week-old Hy-line Brown laying hens were allotted to 1 of 2 treatments. Diets were fed to hens for 1
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41

Schwartz, Michael W. "The neuroscience of metabolism." Open Access Government 46, no. 1 (2025): 100–101. https://doi.org/10.56367/oag-046-11943.

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The neuroscience of metabolism Given that the brain can detect and respond to changing levels of body fat and blood sugar, Michael W. Schwartz, MD from the University of Washington Medicine Diabetes Institute, explains how the brain can be targeted to treat obesity and diabetes. Until recently, obesity was perceived even in medical circles as a personal failing characterized by a lack of willpower. However, as more is learned about the biological system known as ‘energy homeostasis,’ this perception is changing. Controlled primarily by the brain, energy homeostasis works by matching energy int
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42

Li, M., D. Gu, N. Xu, et al. "Gut carbohydrate metabolism instead of fat metabolism regulated by gut microbes mediates high-fat diet-induced obesity." Beneficial Microbes 5, no. 3 (2014): 335–44. http://dx.doi.org/10.3920/bm2013.0071.

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The aim of this study was to investigate the mechanisms underlying the involvement of gut microbes in body weight gain of high-fat diet-fed obesity-prone (obese) and obesity-resistant (lean) mice. C57BL/6 mice were grouped into an obese group, a lean group and a normal control group. Both obese and lean mice were fed a high-fat diet while normal control mice were fed a normal diet; they were observed for six weeks. The results showed that lean mice had lower serum lipid levels, body fat and weight gain than obese mice. The ATPase, succinate dehydrogenase and malate dehydrogenase activities in
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43

Lack, Edward B. "Fat Metabolism and its Relevance to Liposuction." American Journal of Cosmetic Surgery 14, no. 3 (1997): 263–67. http://dx.doi.org/10.1177/074880689701400306.

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Advances in liposuction, primarily involving the tumescent technique of local anesthesia, have fostered the advent of large-volume liposuction. Such major alterations in the volume of an organ system must have physiologic implications. This paper attempts to describe fat metabolism as it relates to fat storage and expenditure and the possible effects of large-volume liposuction on fat metabolism.
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44

Qureshi, Asjid, and Peter G. Kopelman. "Leptin - fat messenger or fat controller?" Clinical Endocrinology 47, no. 2 (1997): 169–71. http://dx.doi.org/10.1046/j.1365-2265.1997.2931092.x.

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45

Roche, Helen M. "Dietary carbohydrates and triacylglycerol metabolism." Proceedings of the Nutrition Society 58, no. 1 (1999): 201–7. http://dx.doi.org/10.1079/pns19990026.

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There is a growing body of scientific evidence which demonstrates that plasma triacylglycerol (TAG) concentration, especially in the postprandial state, is an important risk factor in relation to the development of CHD. Postprandial hypertriacylglycerolaemia is associated with a number of adverse metabolic risk factors, including the preponderance of small dense LDL, low HDL-cholesterol concentrations and elevated factor VII activity. Traditionally, a low-fat high-carbohydrate diet was used to prevent CHD because it effectively reduces plasma cholesterol concentrations, but this dietary regime
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46

Romanauska, Anete. "Seeing fat inside the nucleus." Science 374, no. 6571 (2021): 1066. http://dx.doi.org/10.1126/science.abm8139.

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47

Skrzypski, Marek, and Paweł A. Kołodziejski. "Special Issue: Lipid Metabolism, Adipogenesis and Fat Tissue Metabolism: Gene Regulation." Genes 14, no. 5 (2023): 1121. http://dx.doi.org/10.3390/genes14051121.

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48

HONG, QIN, CHEN XIA, HU XIANGYING, and YUAN QUAN. "Capsinoids suppress fat accumulation via lipid metabolism." Molecular Medicine Reports 11, no. 3 (2014): 1669–74. http://dx.doi.org/10.3892/mmr.2014.2996.

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49

Bloor, W. R. "FAT METABOLISM IN THE EARLY 1900'S." Nutrition Reviews 10, no. 7 (2009): 193–95. http://dx.doi.org/10.1111/j.1753-4887.1952.tb01145.x.

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50

Romijn, J. A., E. F. Coyla, L. Sidossis, J. F. HoroWit, and R. R. Wolfe. "EFFECTS OF EXERCISE INTENSITY ON FAT METABOLISM." Medicine & Science in Sports & Exercise 24, Supplement (1992): S72. http://dx.doi.org/10.1249/00005768-199205001-00432.

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