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Relevant bibliographies by topics / FAT10

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Academic literature on the topic 'FAT10'

Author: Grafiati

Published: 15 June 2024

Last updated: 31 July 2025

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Journal articles on the topic "FAT10"

1

Cao, Jinjing, Annette Aichem, Michael Basler, Gerardo Omar Alvarez Salinas, and Gunter Schmidtke. "Phosphorylated FAT10 Is More Efficiently Conjugated to Substrates, Does Not Bind to NUB1L, and Does Not Alter Degradation by the Proteasome." Biomedicines 12, no. 12 (2024): 2795. https://doi.org/10.3390/biomedicines12122795.

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Background: FAT10 is a member of the ubiquitin-like modifier family. Similar to ubiquitin, FAT10 has a distinct enzyme cascade consisting of E1-activating, E2-conjugating, and possibly several E3-ligating enzymes, which will covalently link FAT10 to substrate proteins in order to target them directly for proteasomal degradation. FAT10 was reported to be phosphorylated by IKKβ during infection with influenza A virus. Methods: To assess the difference between the FAT10-dependent degradation of phosphorylated FAT10 and the non-phosphorylated FAT10 wild type (FAT10 WT), a mutated FAT10 that mimick

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Hipp, Mark Steffen, Birte Kalveram, Shahri Raasi, Marcus Groettrup, and Gunter Schmidtke. "FAT10, a Ubiquitin-Independent Signal for Proteasomal Degradation." Molecular and Cellular Biology 25, no. 9 (2005): 3483–91. http://dx.doi.org/10.1128/mcb.25.9.3483-3491.2005.

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ABSTRACT FAT10 is a small ubiquitin-like modifier that is encoded in the major histocompatibility complex and is synergistically inducible by tumor necrosis factor alpha and gamma interferon. It is composed of two ubiquitin-like domains and possesses a free C-terminal diglycine motif that is required for the formation of FAT10 conjugates. Here we show that unconjugated FAT10 and a FAT10 conjugate were rapidly degraded by the proteasome at a similar rate. Fusion of FAT10 to the N terminus of very long-lived proteins enhanced their degradation rate as potently as fusion with ubiquitin did. FAT10

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Schnell, Leonie, Alina Zubrod, Nicola Catone, Johanna Bialas, and Annette Aichem. "Tumor necrosis factor mediates USE1-independent FAT10ylation under inflammatory conditions." Life Science Alliance 6, no. 11 (2023): e202301985. http://dx.doi.org/10.26508/lsa.202301985.

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The ubiquitin-like modifier FAT10 is up-regulated in many different cell types by IFNγ and TNFα (TNF) and directly targets proteins for proteasomal degradation. FAT10 gets covalently conjugated to its conjugation substrates by the E1 activating enzyme UBA6, the E2 conjugating enzyme USE1, and E3 ligases including Parkin. To date, USE1 was supposed to be the only E2 enzyme for FAT10ylation, and we show here that a knockout of USE1 strongly diminished FAT10 conjugation. Remarkably, under inflammatory conditions in the presence of TNF, FAT10 conjugation appears to be independent of USE1. We repor

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Jia, Yue, Ping Ji, and Samuel W. French. "The Role of FAT10 in Alcoholic Hepatitis Pathogenesis." Biomedicines 8, no. 7 (2020): 189. http://dx.doi.org/10.3390/biomedicines8070189.

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FAT10 expression is highly up-regulated by pro-inflammatory cytokines IFNγ and TNFα in all cell types and tissues. Increased FAT10 expression may induce increasing mitotic non-disjunction and chromosome instability, leading to tumorigenesis. In this review, we summarized others’ and our work on FAT10 expression in liver biopsy samples from patients with alcoholic hepatitis (AH). FAT10 is essential to maintain the function of liver cell protein quality control and Mallory–Denk body (MDB) formation. FAT10 overexpression in AH leads to balloon degeneration and MDB aggregation formation, all of wh

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Mah, Mei Min, Nicola Roverato, and Marcus Groettrup. "Regulation of Interferon Induction by the Ubiquitin-Like Modifier FAT10." Biomolecules 10, no. 6 (2020): 951. http://dx.doi.org/10.3390/biom10060951.

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The revelation that the human major histocompatibility complex (MHC) class I locus encodes a ubiquitin-like protein designated HLA-F adjacent transcript 10 (FAT10) or ubiquitin D (UBD) has attracted increasing attention to the function of this protein. Interestingly, the pro-inflammatory cytokines interferon (IFN)-γ and tumor necrosis factor (TNF) α synergize to strongly induce FAT10 expression, thereby suggesting a role of FAT10 in the immune response. Recent reports that FAT10 downregulates type I interferon production while it upregulates IFN-γ pose mechanistic questions on how FAT10 differ

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Arshad, Maria, Nazefah Abdul Hamid, Mun Chiang Chan, et al. "NUB1 and FAT10 Proteins as Potential Novel Biomarkers in Cancer: A Translational Perspective." Cells 10, no. 9 (2021): 2176. http://dx.doi.org/10.3390/cells10092176.

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Cancer increases the global disease burden substantially, but it remains a challenge to manage it. The search for novel biomarkers is essential for risk assessment, diagnosis, prognosis, prediction of treatment response, and cancer monitoring. This paper examined NEDD8 ultimate buster-1 (NUB1) and F-adjacent transcript 10 (FAT10) proteins as novel biomarkers in cancer. This literature review is based on the search of the electronic database, PubMed. NUB1 is an interferon-inducible protein that mediates apoptotic and anti-proliferative actions in cancer, while FAT10 is a ubiquitin-like modifier

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Canaan, Allon, Xiaofeng Yu, Carmen J. Booth, et al. "FAT10/Diubiquitin-Like Protein-Deficient Mice Exhibit Minimal Phenotypic Differences." Molecular and Cellular Biology 26, no. 13 (2006): 5180–89. http://dx.doi.org/10.1128/mcb.00966-05.

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ABSTRACT The FAT10 gene encodes a diubiquitin-like protein containing two tandem head-to-tail ubiquitin-like domains. There is a high degree of similarity between murine and human FAT10 sequences at both the mRNA and protein levels. In various cell lines, FAT10 expression was shown to be induced by gamma interferon or by tumor necrosis factor alpha. In addition, FAT10 expression was found to be up-regulated in some Epstein-Barr virus-infected B-cell lines, in activated dendritic cells, and in several epithelial tumors. However, forced expression of FAT10 in cultured cells was also found to pro

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Schregle, Richard, Stefanie Mueller, Daniel F. Legler, Jérémie Rossy, Wolfgang A. Krueger, and Marcus Groettrup. "FAT10 localises in dendritic cell aggresome-like induced structures and contributes to their disassembly." Journal of Cell Science 133, no. 14 (2020): jcs240085. http://dx.doi.org/10.1242/jcs.240085.

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ABSTRACTDendritic cell (DC) aggresome-like induced structures (DALIS) are protein aggregates of polyubiquitylated proteins that form transiently during DC maturation. DALIS scatter randomly throughout the cytosol and serve as antigen storage sites synchronising DC maturation and antigen presentation. Maturation of DCs is accompanied by the induction of the ubiquitin-like modifier FAT10 (also known as UBD), which localises to aggresomes, structures that are similar to DALIS. FAT10 is conjugated to substrate proteins and serves as a signal for their rapid and irreversible degradation by the 26S

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Boehm, Annika N., Johanna Bialas, Nicola Catone, et al. "The ubiquitin-like modifier FAT10 inhibits retinal PDE6 activity and mediates its proteasomal degradation." Journal of Biological Chemistry 295, no. 42 (2020): 14402–18. http://dx.doi.org/10.1074/jbc.ra120.013873.

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The retina-specific chaperone aryl hydrocarbon interacting protein-like 1 (AIPL1) is essential for the correct assembly of phosphodiesterase 6 (PDE6), which is a pivotal effector enzyme for phototransduction and vision because it hydrolyzes cGMP. AIPL1 interacts with the cytokine-inducible ubiquitin-like modifier FAT10, which gets covalently conjugated to hundreds of proteins and targets its conjugation substrates for proteasomal degradation, but whether FAT10 affects PDE6 function or turnover is unknown. Here, we show that FAT10 mRNA is expressed in human retina and identify rod PDE6 as a ret

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Saxena, Kritika, Nicola Domenico Roverato, Melody Reithmann та ін. "FAT10 is phosphorylated by IKKβ to inhibit the antiviral type-I interferon response". Life Science Alliance 7, № 1 (2023): e202101282. http://dx.doi.org/10.26508/lsa.202101282.

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IFN-I secretion provides a rapid host defense against infection with RNA viruses. Within the host cell, viral RNA triggers the activation of the RIG-I signaling pathway, leading to the production of IFN-I. Because an exaggerated IFN-I response causes severe tissue damage, RIG-I signaling is tightly regulated. One of the factors that control the IFN-I response is the ubiquitin-like modifier FAT10, which is induced by TNF and IFNγ and targets covalently FAT10-linked proteins for proteasomal degradation. However, the mechanism of how FAT10 modulates IFN-I secretion remains to be fully elucidated.

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Dissertations / Theses on the topic "FAT10"

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Bialas, Johanna [Verfasser]. "The influence of FAT10 on the ubiquitin pathway and The search for FAT10-specific E3 ligases / Johanna Bialas." Konstanz : KOPS Universität Konstanz, 2018. http://d-nb.info/1215032919/34.

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Ryu, Stella [Verfasser]. "Investigation of the FAT10 conjugation pathway / Stella Ryu." Konstanz : Bibliothek der Universität Konstanz, 2012. http://d-nb.info/105034880X/34.

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Ahmad, Faiz [Verfasser]. "The Search for Deconjugating Enzymes of FAT10 / Faiz Ahmad." Konstanz : Bibliothek der Universität Konstanz, 2016. http://d-nb.info/1159513368/34.

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Bürger, Stefanie [Verfasser]. "The Ubiquitin-like modifier FAT10 in tolerance induction / Stefanie Bürger." Konstanz : Bibliothek der Universität Konstanz, 2013. http://d-nb.info/1110770529/34.

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Schwab, Ricarda [Verfasser]. "Investigation of the interaction of FAT10 and VCP (p97) / Ricarda Schwab." Konstanz : Bibliothek der Universität Konstanz, 2015. http://d-nb.info/1144178703/34.

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Mah, Mei Min [Verfasser]. "The Role of FAT10 in Regulating the Interferon Response / Mei Min Mah." Konstanz : KOPS Universität Konstanz, 2019. http://d-nb.info/1202012833/34.

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Spinnenhirn, Valentina [Verfasser]. "Functional analysis of the ubiquitin-like modifier FAT10 in autophagy / Valentina Spinnenhirn." Konstanz : Bibliothek der Universität Konstanz, 2015. http://d-nb.info/1112604391/34.

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Kluge, Kathrin Christiane [Verfasser]. "Characterisation of the Interaction between FAT10 and its Substrate Protein p62 / Kathrin Christiane Kluge." Konstanz : Bibliothek der Universität Konstanz, 2014. http://d-nb.info/1112745238/34.

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Schregle, Richard [Verfasser]. "The Ubiquitin-like Modifier FAT10 in Dendritic Cell Aggresome-like Induced Structures / Richard Schregle." Konstanz : KOPS Universität Konstanz, 2018. http://d-nb.info/121985266X/34.

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10

Bernard, Lucie. "Rôle de FAT10 dans la sénescence des hépatocytes et le développement de la NASH." Electronic Thesis or Diss., Université de Lille (2022-....), 2023. https://pepite-depot.univ-lille.fr/ToutIDP/EDBSL/2023/2023ULILS039.pdf.

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L'accumulation d'hépatocytes sénescents a été identifiée comme un facteur clé dans la progression des maladies du foie gras non alcooliques (NAFLDs), qui correspondent à un spectre de pathologies hépatiques chroniques, allant de la simple stéatose jusqu'au développement d'une stéato-hépatite non alcoolique (NASH), d'une cirrhose voire d'un carcinome hépatocellulaire (HCC). Cependant, les mécanismes et acteurs participant à la régulation de la sénescence au cours de la NASH sont encore peu décrits. L'objectif de cette thèse a donc été d'étudier les mécanismes contrôlant la sénescence des hépato

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Books on the topic "FAT10"

1

Raasi, Shahri. Functional analysis of FAT10, a cytokine-inducible ubiquitin-like protein. 2001.

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Nayak, R. V., N. A. Mousa, and International Joint Power Generation Conference (1990 Boston, Mass.). Combustion Modeling and Burner Replacement Strategies/Fact10/No G00523: Presented at the 1990 International Joint Power Generation Conference, Boston, ... 21-25, 1990 (Fact (Series), Vol. 10,). Amer Society of Mechanical, 1992.

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Book chapters on the topic "FAT10"

1

Pelzer, Christiane, and Marcus Groettrup. "FAT10." In Subcellular Biochemistry. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6676-6_19.

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Aichem, Annette, and Marcus Groettrup. "Detection and Analysis of FAT10 Modification." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-474-2_7.

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3

Lukasiak, Sebastian, Kai Breuhahn, Claudia Schiller, Gunter Schmidtke, and Marcus Groettrup. "Quantitative Analysis of Gene Expression Relative to 18S rRNA in Carcinoma Samples Using the LightCycler® Instrument and a SYBR GreenI-based Assay: Determining FAT10 mRNA Levels in Hepatocellular Carcinoma." In Methods in Molecular Biology. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-040-3_5.

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Bu, Tiao, Lingling Wang, Xiaolong Wu, et al. "Interacting Fat1 and Dchs Planar Cell Polarity Proteins Supported by Fjx1 Serve as Heterodimeric Intercellular Bridges Crucial to Support Spermatogenesis." In Advances in Experimental Medicine and Biology. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-82990-1_15.

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Aichem, Annette, Annika N. Boehm, Nicola Catone, Gunter Schmidtke, and Marcus Groettrup. "Analysis of modification and proteolytic targeting by the ubiquitin-like modifier FAT10." In Methods in Enzymology. Elsevier, 2019. http://dx.doi.org/10.1016/bs.mie.2018.12.040.

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Mittal, Vishnu, Abhinav Singhal, and Shushank Mahajan. "FAT16 File System Disk." In Data Recovery Techniques for Computer Forensics. BENTHAM SCIENCE PUBLISHERS, 2025. https://doi.org/10.2174/9789815274677125010009.

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Background: The historical development of the FAT16 file system highlights its inception, evolution, and key milestones. It explores the technological landscape that necessitated the creation of FAT16, shedding light on the challenges and requirements that shaped its design. Objective: The primary objective is to conduct a detailed analysis of the FAT16 file system considering its architecture, functionality, and historical significance. By dissecting the internal workings of FAT16, we aim to provide readers with a deeper comprehension of its strengths, weaknesses, and enduring relevance. Meth

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Adams, Susan. ""And the Sun Refused to Shine"." In Final Acts: The End of Life: Hospice and Palliative Care. Baywood Publishing Company, Inc., 2013. http://dx.doi.org/10.2190/fatc10.

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B. Pathak, Anand, and Satyam Satyarthi. "Head Neck Squamous Cell Cancer Genomics: Oncogenes, Tumor Suppressor Genes and Clinical Implications." In Molecular Mechanisms in Cancer. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.101044.

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Head Neck Squamous Cell Cancer is genomically heterogenous. Common somatic mutations involve TP53, CDKN2A, FAT1, NOTCH1, PIK3CA, KMT2D and NSD1, less frequently others. Epigenetic changes also contribute to HNSCC biology. Alterations in tumor suppressor genes is a major oncogenic event in HNSCC. Genomic heterogeneity exists between different subsites within head neck region and also between the primary and metastatic disease. Intratumor heterogeneity has also been recognized. Based on key genomic alterations, four major molecular subtypes have been identified. Multi-omics analysis has provided

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Conference papers on the topic "FAT10"

1

Noymai, Anukool, Urachada Ketprom, and Chaichana Mitrpant. "Increasing memory in FAT16 removable media of RFID handheld reader." In 2008 5th International Conference on Electrical Engineering/Electronics, Computer, Telecommunications and Information Technology (ECTI-CON). IEEE, 2008. http://dx.doi.org/10.1109/ecticon.2008.4600538.

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Irshad, Khushboo, Chitrangda Srivastava, Nargis Malik, et al. "Abstract 3175: FAT1 and the immunosuppressive milieu in glioblastoma tumors." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-3175.

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Dikshit, Bhawana, Parthaprasad Chattopadhyay, Subrata Sinha, and Kunzang Chosdol. "Abstract 4102: FAT1: A novel regulator of cancer and inflammation." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4102.

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Srivastava, Chitrangda, Khushboo Irshad, Parthaprasad Chattopadhyay, et al. "Abstract 3534: FAT1: A potential target of NFkB (RelA) in GBM." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3534.

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Gupta, Y., SS Shivajirao, K. Irshad, et al. "PO-125 FAT1 on salvador-warts-hippo (SWH) pathway in human glioblastoma." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.166.

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Halimah, Nova Nur. "KARAKTERISASI SENSOR HY-SRF05 DAN LOAD CELL SINGLE-POINT SEBAGAI PARAMETER PENGUKURAN ANTROPOMETRI PADA SISTEM PEMANTAUAN STATUS GIZI BAYI." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.fa10.

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Arafahnti, Bestari Laksmi, Umiatin Umiatin, and Heru Prasetio. "PENGARUH ENERGI LINAC TERHADAP RESPON FILM DOSIMETRI GAFCHROMIC." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2023. http://dx.doi.org/10.21009/03.1101.fa10.

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Connor, Ashton A., Jordan Lerner-Ellis, Mohammad R. Akbari, et al. "Abstract A23: Rare variants in the FAT1 gene may predispose to familial colorectal cancer." In Abstracts: AACR Special Conference: Colorectal Cancer: From Initiation to Outcomes; September 17-20, 2016; Tampa, FL. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.crc16-a23.

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Krasteva, V. M., G. H. Sigel, S. L. Semjonov, M. M. Bubnov, and M. I. Belovolov. "Pr3+ -doped Ge-S-I glasses and fibers for PDFA applications." In Optical Amplifiers and Their Applications. OSA, 1997. http://dx.doi.org/10.1364/oaa.1997.faw10.

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Liu, Chung Ji, Shu-Chun Lin, and Li-Han Lin. "Abstract 1234: Somatic mutations of FAT1 in oral cancer are associated with tumor progression and survival." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1234.

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