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Dissertations / Theses on the topic 'Feline immunodeficiency virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 31 January 2023

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1

Caney, Sarah Madeline Amanda. "Mucosal immunopathogenesis of feline immunodeficiency virus infection." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341499.

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2

Camerini, Valentina. "Translational control of feline immunodeficiency virus (FIV)." Lyon, École normale supérieure (sciences), 2006. http://www.theses.fr/2006ENSL0387.

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3

Buck, Wayne R. "Neuropathogenic mechanisms of feline immunodeficiency virus infection." Columbus, Ohio : Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1078414064.

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Thesis (Ph. D.)--Ohio State University, 2004.<br>Title from first page of PDF file. Document formatted into pages; contains xiv, 144 p.; also includes graphics (some col.). Includes abstract and vita. Co-advisors: Lawrence E. Mathes and Maria H. Neff, Dept. of Veterinary Biosciences. Includes bibliographical references (p. 122-144).

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4

Leal, Rodolfo Assis Oliveira. "Recombinant feline interferon omega therapy in cats naturally infected with Feline Immunodeficiency Virus : clinical, viral and immunological relevance." Doctoral thesis, Universidade de Lisboa. Faculdade de Medicina Veterinária, 2014. http://hdl.handle.net/10400.5/7458.

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Tese de Doutoramento em Ciências Veterinárias Especialidade de Clínica<br>Type-I Interferons are well-known cytokines which among their main functions are key components of the host immune response against viral infections. Due to its immune modulation properties, they are commonly used in the therapeutic approach of various diseases such as retroviral infections. Recombinant feline interferon omega (rFeIFN-ω) is the first interferon licensed for use in veterinary medicine. Although it is commonly administered in retroviral infections, namely in Feline Immunodeficiency Virus (FIV) and Feline L

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5

Haining, Hayley. "Studies of the pathogenesis of feline immunodeficiency virus." Thesis, University of Glasgow, 2004. http://theses.gla.ac.uk/5512/.

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The project had three aims: namely: - (i) to investigate the in vitro cell tropism of a range of field isolates from cats at different stages of disease and compare their phenotype with the well-characterised prototype viruses FIV-PET and FIV-GL8. (ii) to study the pathogenicity of these viruses in vivo in order to examine any correlation between virulence in vivo and tropism in vitro. (iii) to look at the role of the env gene in the pathogenicity of FIV. In vitro studies of cell tropism revealed that isolates from cats in the terminal stage of disease had a greater ability to utilise CXCR4 th

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6

Gemeniano, Maria Lourdes Charmaine. "Characterization of Orf-A of feline immunodeficiency virus /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2002. http://uclibs.org/PID/11984.

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7

Bęczkowski, Paweł. "Virus evolution in the progression of natural feline immunodeficiency virus infection." Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4186/.

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Feline immunodeficiency virus (FIV) is an important pathogen of domestic cats which in some cases can lead to feline AIDS. It shares many similarities with its human counterpart and is studied to understand correlates of immune-protection and mechanisms of disease progression in cats, both to improve the welfare of infected cats and as an animal model for the pathogenesis of HIV infection in humans. FIV is believed to evolve during the course of infection as a result of the error prone nature of reverse transcriptase and recombination between viral variants, but relatively little is known abou

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8

Chan, Chi Ngai. "Cell signalling and feline immunodeficiency virus growth and latency." Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/3889/.

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The replication of CD4+ T cell-tropic retroviruses such as the human immunodeficiency virus-1 (HIV-1) and the feline immunodeficiency virus (FIV) is intricately linked to host cell signalling and activation status. This intimate relationship between the viruses and their respective hosts plays crucial roles in the pathogenesis of each virus. Focusing on FIV, this thesis examined several questions relating to virus replication and cell signalling. Firstly, the possible cell stimulatory effect when FIV Env binds to its primary receptor CD134 was investigated and results suggested that FIV does n

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9

Grant, Susan Elizabeth. "Studies on haemopoiesis in early feline immunodeficiency virus infection." Thesis, University of Glasgow, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296042.

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10

Samman, Ayman. "The role of virus neutralisation in immunity to feline immunodeficiency virus infection." Thesis, University of Glasgow, 2010. http://theses.gla.ac.uk/1554/.

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Feline immunodeficiency virus (FIV) is an important veterinary pathogen with comparative significance because of its similarities to its human counterpart HIV. Since FIV is the only non-primate lentivirus which induces AIDS-like symptoms in its natural host, it serves as a valuable animal model for both prophylactic and therapeutic studies of HIV. It is accepted that the induction of neutralising antibodies (NAbs) is a key element in the control of lentiviral infection, since T-cell based vaccines alone failed to prevent infection in most experimental animal model systems. In this project a ro

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11

Frey, Susan Carol Stankewitz. "The role of CXCR4 in feline immunodeficiency virus cell entry /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/11487.

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12

Meers, Joanne. "The effects of antiviral agents on feline immunodeficiency virus infection." Thesis, Meers, Joanne (1994) The effects of antiviral agents on feline immunodeficiency virus infection. PhD thesis, Murdoch University, 1994. https://researchrepository.murdoch.edu.au/id/eprint/53284/.

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Feline immunodeficiency virus (FIV) is a recently discovered lentivirus with many structural and replicative similarities to human immunodeficiency virus (HIV), and has been suggested as a useful animal model for the evaluation of anti-HIV chemotherapy. The successful use of the model is dependent on the development of techniques to accurately measure the response to antiviral treatment in FIV-infected cats. FIV was isolated by the co-cultivation of peripheral blood mononuclear cells (PBMC) from seropositive cats with PBMC of seronegative donor cats, or with MYA-1 cells (a feline T-lymphobl

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13

Hopper, Cherida Dawm. "Studies on the epizootiology of feline immunodeficiency virus infection in cats." Thesis, University of Bristol, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335478.

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14

Garg, Himanshu. "Feline Immunodeficiency Virus (FIV) Envelope Glycoprotein-Mediated Cell Fusion and Apoptosis." NCSU, 2003. http://www.lib.ncsu.edu/theses/available/etd-11042003-141554/.

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Feline Immunodeficiency Virus (FIV) and Human Immunodeficiency Virus (HIV) are lentiviruses that are remarkable similar in their genomic organization, receptor usage and pathogenesis. Based on this FIV has evolved into a well-established small animal model for studying AIDS. FIV and HIV cause a progressive depletion of T cells via a still unknown mechanism though numerous studies support a role of membrane expressed HIV env glycoprotein in apoptotic killing of CD4+ T cells. HIV env glycoprotein is a heterodimer of surface expressed gp120 that binds to CD4 and a chemokine receptor and transmemb

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15

McEwan, William. "Factors affecting replication and cross-species transmission of feline immunodeficiency virus." Thesis, University of Glasgow, 2010. http://theses.gla.ac.uk/1388/.

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In order to successfully invade a new species, lentiviruses must overcome restriction factors, dominant blocks to replication, and be able to make use of available host factors such as entry receptors for replication. Human immunodeficiency virus (HIV-1) infection is blocked at a post-entry stage by rhesus macaque TRIM5α, an effect that is enhanced by host factor cyclophilin A (CypA), but largely evades restriction by the human TRIM5α variant. HIV-1 replication is also inhibited by simian APOBEC3G but is able to evade restriction by human APOBEC3G by inducing its degradation through expression

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16

Cheng, Heather H. "Envelope and receptor determinants for infection by an immunodeficiency-associated feline leukemia virus /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/5010.

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17

Gu, Qinyong [Verfasser]. "The molecular interaction of feline immunodeficiency virus Vif with feline APOBEC3 and Cullin 5 / Qinyong Gu." Gießen : Universitätsbibliothek, 2018. http://d-nb.info/1162840072/34.

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18

Kraase, Martin [Verfasser]. "The virus-receptor interaction in the pathogenesis of feline immunodeficiency virus infection / Martin Kraase." Berlin : Freie Universität Berlin, 2014. http://d-nb.info/1049687663/34.

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19

Leal, Magda Liliana Garcia. "Avaliação da freqüência da infecção por micoplasmas hemotrópicos em gatos com linfoma." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-17042009-141814/.

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Com o objetivo de avaliar a freqüência de infecção por micoplasmas hemotrópicos em gatos com linfoma e seu impacto na ocorrência de anemias nesses animais, foram analisadas amostras sangüíneas de 14 animais com diagnóstico de linfoma, sem qualquer tratamento prévio e 14 amostras de sangue de gatos hígidos, por meio da técnica de PCR-Nested. Utilizaram-se primers que amplificam fragmentos do gene 16S rRNA dos micoplasmas. Eritrograma e bioquímica sérica foram realizados, assim como testes sorológicos imunoenzimáticos (ELISA) para ambos os retrovírus. Anemia foi observada em 28,6% (4/14) dos gat

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20

Shen, Xiaoying. "Infection of newborn kittens with a feline immunodeficiency virus vif-deletion mutant /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2004. http://uclibs.org/PID/11984.

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21

Rogers, Melinda Cadd. "Differential thrombospondin expression on T lymphocytes in a Feline Immunodeficiency Virus model." NCSU, 2007. http://www.lib.ncsu.edu/theses/available/etd-06152007-103921/.

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CD4+CD25+ T regulatory cells represent an important subset of lymphocytes whose function is to suppress autoimmune disease and control normal immune responses. There is much research indicating a direct role for TGF-beta expressed on the surface of Tregs in the suppressor function of these cells. TGF-beta, whether bound to the cell surface or secreted, is produced as part of a complex that renders the cytokine inactive. Thrombospondin is the primary activator of biologically latent TGF-beta. This research demonstrates that thrombospondin is expressed on the surface of T lymphocytes isolated fr

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22

del, Fierro Gloria M. "Clinical, immunological and pathological aspects of experimental feline immunodeficiency virus (FIV) infection." Thesis, del Fierro, Gloria M. (1995) Clinical, immunological and pathological aspects of experimental feline immunodeficiency virus (FIV) infection. PhD thesis, Murdoch University, 1995. https://researchrepository.murdoch.edu.au/id/eprint/53477/.

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Feline immunodeficiency virus is a recently discovered feline lentivirus which clinically resembles human HIV-1 virus inducing immune dysfunction and secondary infections in cats. FIV presence has been confirmed throughout the world in domestic, feral and zoo cat populations. Older, free-roaming, diseased male cats are at the greatest risk of acquiring the infection. There are no specific clinical signs restricted to FIV infection. However, weight loss, recurrent fever of undetermined origin, unthriftiness, inappetence, pyrexia, lymphadenopathy, anaemia, leucopenia and myeloproliferative d

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23

Gonsales, Fernanda Fidelis. "Ocorrência de Chlamydophila felis e do plasmídeo críptico em gatis nas cidades de São Paulo e Osasco." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/10/10134/tde-09092014-153345/.

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A infecção de trato respiratório superior em gatos é uma afecção muito frequente em indivíduos que vivem em abrigos, com elevada morbidade e em alguns casos, fatal. O herpesvírus felino tipo1 (FHV-1) e a Chlamydophila felis estão entre os principais causadores. O FHV-1 ocasiona quadros de espirros, secreção nasal e alterações oculares como conjuntivite. A C. felis é responsável pelos piores casos de conjuntivite e apresenta um plasmídeo críptico como possível fator de virulência. A presença dos retrovírus da leucemia felina (FeLV) e/ou imunodeficiência dos felinos (FIV) debilita a função do si

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24

Tran, Khanh Van Nhu. "The Effect Of Methamphetamine On Astrocytes With Implications For Feline Immunodeficiency Virus And Cxcr4." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1211857305.

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25

Wooding, Anita. "Antiviral efficacy of nine nucleoside reverse transcriptase inhibitors against feline immunodeficiency virus in feline peripheral blood mononuclear cells." Diss., Ludwig-Maximilians-Universität München, 2015. http://nbn-resolving.de/urn:nbn:de:bvb:19-182515.

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The purpose of the study reported here was to compare the antiviral efficacy against feline immunodeficiency virus (FIV) and cytotoxicity in feline peripheral blood mononuclear (PBM) cells of 9 nucleoside reverse transcriptase inhibitors (NRTIs), three of which had not been evaluated against FIV in feline cells before. PBM cells were isolated from the blood of three specific pathogen-free (SPF) cats. The cytotoxic effects of the test compounds were determined by colorimetric quantification of a formazan product resulting from bioreduction of a tetrazolium reagent by viable PBM cells. Each co

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26

Cravo, Joana Acciaioli Pena. "Efeito do tratamento por interferão ómega de origem felina (rFeIFN-Ω) na evolução clínica de gatos naturalmente infectados com os vírus da imunodeficiência (FIV) e leucemia felinas (FeLV) e na excreção de vírus respiratórios concomitantes". Master's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2011. http://hdl.handle.net/10400.5/3793.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>O vírus da imunodeficiência felina (FIV) e o vírus da leucemia felina (FeLV) são retrovírus responsáveis por duas das doenças infecciosas mais comuns e que põem em risco o bem-estar e a vida dos gatos em todo o mundo. Os imunoestimuladores, tais como os interferões rHuIFN-α e rFeIFN-ω, são amplamente utilizados em Medicina Veterinária no tratamento de gatos com FIV e FeLV. No entanto, a administração de rHuIFN-α a felinos tem limitações devido à produção de anticorpos contra o fármaco. O rFeIFN-ω, recentemente desenvolvido, mostrou u

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27

Sirage, Carla Sofia Ramos Alves. "Avaliação da expressão de mediadores imunitários em gatos infectados com o vírus da Leucemia Felina (FeLV) e tratados com interferão ómega recombinante felino (rFeIFN-ω)". Master's thesis, Universidade de Lisboa. Faculdade de Medicina Veterinária, 2014. http://hdl.handle.net/10400.5/7195.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>O interferão ómega felino (rFeIFN-ω) é atualmente o único interferão licenciado para uso médico-veterinário tendo-se mostrado eficaz no tratamento de gatos infectados pelo FeLV: melhora o seu estado clínico, prolonga a sua longevidade e reduz a excreção de vírus concomitantes. Contudo, o efeito do rFeIFN-ω como antiviral tem sido questionado, acreditando-se que este fármaco atue apenas ao nível da imunidade inata. Resultados publicados pelo nosso grupo reforçam esta teoria, reportando um aumento dos níveis séricos de proteínas de fas

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28

Liu, Pinghuang. "Virus infection and evolution in the central nervous system following intracerebroventricular inoculation with Feline Immunodeficiency Virus." NCSU, 2005. http://www.lib.ncsu.edu/theses/available/etd-10302005-212859/.

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HIV-1 infection of the central nervous system (CNS) results in neurological impairments in subpopulation of HIV-infected individuals which range from from mild cognitive/motor disorder (MCMD) to HIV-associated dementia (HAD). HIV-1 associated neurological diseases are still a big problem even with the introduction of combined antiretroviral therapy. However the mechanisms of CNS infection and pathogenesis that lead to HAD are still not completely clear. HIV-1 CNS infection occurs soon after peripheral infection. Subsequent to infection, the CNS may act as a protected anatomical reservoir for l

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29

Sturgess, Christopher Paul. "Studies on mucosal effector mechanisms in feline immunodeficiency virus (FIV) infection in cats." Thesis, University of Bristol, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388027.

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30

Hora, Aline Santana da. "Micoplasmas hemotrópicos como potenciais agentes causadores de anemia em felinos domésticos." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-05092008-104907/.

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Com o objetivo de avaliar a magnitude da infecção por micoplasmas hemotrópicos nos felinos anêmicos, amostras sangüíneas de 270 felinos anêmicos (Ht≤29%) e 53 felinos saudáveis foram submetidas à exames hematológicos, bioquímicos séricos (proteína total, albumina, fosfatase alcalina, alanina aminotransferase, aspartato aminotransferase, gamaglutamil transferase, bilirrubinas, uréia e creatinina), avaliação citológica do esfregaço sangüíneo e testes moleculares para a detecção de material genético de Mycoplasma spp. Foram encontradas 25 amostras positivas no grupo dos felinos anêmicos, pela

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31

Brown, Abigail Louise. "Investigation into the use of feline CD40 ligand as an adjuvant in a DNA vaccine against feline immunodeficiency virus." Thesis, University of Glasgow, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412964.

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32

Veiga, Rafael Guerreiro. "Clínica e cirurgia de pequenos animais." Master's thesis, Universidade de Évora, 2016. http://hdl.handle.net/10174/19245.

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Este relatório foi elaborado na sequencia do estágio curricular realizado pelo autor, ente 28 de setembro de 2015 e 28 de março de 2016, no Hospital Clínico Veterinario de la Universidad CEU Cardenal Herrera (HCV CEU-UCH), em Alfara del Patriarca, Valência, Espanha. A infeciologia foi a área da clínica médica mais representativa (28%), sendo o vírus da imunodeficiência felina (FIV) o agente infecioso registado mais frequentemente (19%). É importante reconhecer e diagnosticar esta infeção de forma a aplicar um maneio adequado aos pacientes infetados, melhorando a sua qualidade de vida e preveni

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33

Silva, Flávio Roberto Chaves da. "Prevalência das infecções pelo vírus da leucemia viral felina e da imunodeficiência viral felina na cidade de Porto Alegre." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2007. http://hdl.handle.net/10183/12699.

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O vírus da imunodeficiência felina (FIV) é um membro da subfamília Lentivirinae da família Retroviridae. A infecção é caracterizada por imunodepressão com um declíneo progressivo dos linfócitos T CD4+, propiciando, desta maneira, o surgimento de infecções oportunistas. Já o vírus da leucemia viral felina (FeLV) pertence a subfamília Oncovirinae da família Retroviridae. Este vírus é um importante patógeno dos gatos domésticos que causa uma variedade de desordens neoplásicas e degenerativas e também apresenta distribuição mundial. O presente estudo compreendeu um levantamento da prevalência das

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34

Zanutto, Marcelo de Souza. "Dinâmica da infecção toxoplásmica em felinos infectados pelo Vírus da Imunodeficiência Felina." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-13082007-094957/.

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Para avaliar se a dinâmica da infecção toxoplásmica em gatos infectados pelo VIF é diferente daquela que ocorre em gatos não infectados por esse retrovírus, gatos adultos infectados pelo Vírus da Imunodeficiência Felina (VIF) clade B assintomáticos (n=7) (Grupo I: VIF+TOXO+), e gatos sem a infecção viral (n=7) (Grupo III: VIF-TOXO+) foram inoculados pela via oral com cistos de Toxoplasma gondii cepa P. Os animais foram avaliados por meio do exame clínico, mensuração de anticorpos IgM e IgG anti-T. gondii pela Reação de Imunofluorescência Indireta, eliminação e quantificação de oocistos pela té

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35

Techakriengkrai, Navapon. "Investigating the role of target cell availability in the pathogenesis of feline immunodeficiency virus infection." Thesis, University of Glasgow, 2016. http://theses.gla.ac.uk/7429/.

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Feline immunodeficiency virus (FIV) is a naturally occurring lentivirus of domestic cats, which shares many similarities with its human counterpart, human immunodeficiency virus (HIV). FIV infects its main target cell, the CD4+ T lymphocyte, via interactions with its primary receptor CD134 (an activation marker expressed on activated CD4+ T lymphocytes), and, the chemokine receptor CXCR4. According to the different ways in which FIV isolates interact with CD134, FIV may be categorised into two groups. The first group contains strains that tend to dominate during the earlier phase of infection,

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36

Hayes, Kathleen A. "The feline immunodeficiency virus infected cat as a model of AIDS pathogenesis and antiviral therapy /." The Ohio State University, 1993. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487843314697158.

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37

Azevedo, Inês Ribeiro Pereira Siborro. "Terapêutica com interferão-w em gatos infectados pelo vírus da imunodeficiência felina e/ou pelo vírus da leucemia felina : avaliação clínica, análises laboratoriais e excreção de vírus concomitantes do tracto digestivo." Master's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2012. http://hdl.handle.net/10400.5/5083.

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Os interferões são as proteínas da inflamação mais bem estudadas, encontram-se no mercado dois tipos de interferão: o interferão recombinante humano alfa (reHuIFN-α) e o interferão recombinante felino ómega (reFeIFN-ω). Ambos podem ser usados no tratamento dos sintomas das duas retroviroses felinas, o vírus da imunodeficiência felina (FIV) e o vírus da leucemia felina (FeLV), duas doenças infecto-contagiosas de importante expressão na prática clínica, que afectam fortemente o estado hígido dos animais infectados e que, até ao momento, não tem cura. O reHuIFN-α encontrasse no mercado a um preço

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38

Phadke, Anagha. "CD8+ T cell antiviral activity: mechanism of induction and the suppression of emerging feline immunodeficiency virus strains." Diss., Texas A&M University, 2003. http://hdl.handle.net/1969.1/5990.

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In the present studies, the essential role of inducer cells for the induction of soluble anti-viral activity against feline immunodeficiency virus (FIV) was investigated. Induction of suppression of FIV replication was found to not strictly require autologous cells and was probably not FIV specific. Suppression was maximum when the inducer cells and the effector CD8+ T cells were in contact with each other, suggesting a potential role for membrane antigen interactions and/or cytokines in the induction process. Additionally, flow cytometry analysis demonstrated a significant increase in the per

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39

Wooding, Anita [Verfasser], and Katrin [Akademischer Betreuer] Hartmann. "Antiviral efficacy of nine nucleoside reverse transcriptase inhibitors against feline immunodeficiency virus in feline peripheral blood mononuclear cells / Anita Wooding. Betreuer: Katrin Hartmann." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2015. http://d-nb.info/1072038404/34.

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40

Haipek, Katia. "Avaliação das subpopulações de linfócitos T CD4+, linfócitos T CD8+ e da razão CD4+/CD8+ em gatos com gengivite crônica e infectados naturalmente pelo vírus da imunodeficiência dos felinos (FIV)." Universidade de São Paulo, 2006. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-25052007-143025/.

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A gengivite crônica e intratável observada em gatos infectados pelo vírus da imunodeficiência felina (FIV) é um problema bastante freqüente na clínica de pequenos animais. O papel do FIV na etiologia da estomatite persistente ainda está por ser determinado. As manifestações orais são freqüentemente os primeiros sintomas observados em pacientes humanos infectados pelo HIV e podem ser usadas como indicadores da progressão da doença. O objetivo do presente estudo foi quantificar os linfócitos T CD4+, T CD8+ e a razão CD4+/CD8+ em uma colônia de gatos com gengivite crônica e naturalmente infectado

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41

Gupta, Soumi. "Construction and characterization of an attenuated feline immunodeficiency virus proviral DNA vaccine that co-expresses interferon gamma /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2003. http://uclibs.org/PID/11984.

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42

SIDONI, Marli. "Biotecnologia IgY aplicada ao imunodiagn?stico da infec??o pelo v?rus da imunodefici?ncia felina." Universidade Federal Rural do Rio de Janeiro, 2016. https://tede.ufrrj.br/jspui/handle/jspui/2384.

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Submitted by Jorge Silva (jorgelmsilva@ufrrj.br) on 2018-08-24T19:19:55Z No. of bitstreams: 1 2016 - Marli Sidoni.pdf: 1735106 bytes, checksum: 03907fc3437a44bf96b94d6040184407 (MD5)<br>Made available in DSpace on 2018-08-24T19:19:55Z (GMT). No. of bitstreams: 1 2016 - Marli Sidoni.pdf: 1735106 bytes, checksum: 03907fc3437a44bf96b94d6040184407 (MD5) Previous issue date: 2016-12-01<br>This work focused on deploying IgY technology into developing ELISA immunoenzymatic test to FIV diagnose, contributing to the establishment of a production model for a kit in the field of veterinary medicine. Th

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Hellard, Éléonore. "Des concepts et méthodes associés à la co-circulation des virus dans les populations naturelles d’hôtes à la nécessité d’interdisciplinarité : l’exemple du chat et de ses virus." Thesis, Lyon 1, 2012. http://www.theses.fr/2012LYO10049.

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De nombreux parasites circulent dans les populations naturelles. Au sein d’un hôte, souvent pluri-infecté, ils peuvent interagir, augmentant ou réduisant le risque d’infection et les symptômes d’autres pathogènes. L’étude de ces interactions commence seulement dans les populations naturelles. Les enjeux sont cruciaux : détecter les interactions d’intérêt, estimer la probabilité de coinfection, comprendre la cocirculation des parasites. La détection des interactions sur le terrain est compliquée par la nature des données (e.g., présence-absence) et les facteurs confondants créant des associatio

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Kuhnt, Leah Ann Johnson Calvin M. "Magnetic Resonance Imaging of radiation-induced thymic atrophy as a model for pathologic changes in acute feline immunodeficiency virus infection." Auburn, Ala, 2008. http://hdl.handle.net/10415/1536.

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Mexas, Angela Marie. "CD4+CD25+ REGULATORY T CELLS ARE INFECTED AND ACTIVATED PHENOTYPICALLY AND FUNCTIONALLY DURING ACUTE INFECTION WITH FELINE IMMUNODEFICIENCY VIRUS." NCSU, 2007. http://www.lib.ncsu.edu/theses/available/etd-11022007-103144/.

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HIV-induced AIDS may be mediated by the activation of immunosuppressive CD4+CD25+ T regulatory cells (Tregs). Tregs have been shown to regulate CD4+ and CD8+ immune responses to HIV and FIV antigens in vitro. We tested the hypothesis that Tregs become infected and activated during the acute stage of FIV infection leading to the suppression of CD4+ T helper cell responses. Cats were experimentally infected with FIV-NCSU-1 and blood and lymph node biopsies were collected at 1 week intervals following inoculation. Real-Time PCR was used to determine plasma viremia and relative number of FIV copie

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Figueiredo, Andreza Soriano. "Quantificação e seqüênciamento do gene da transcriptase reversa em gatos naturalmente infectados com vírus da imunodeficiência felina tratado com AZT /." Botucatu : [s.n.], 2007. http://hdl.handle.net/11449/98345.

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Orientador: João Pessoa Araújo Júnior<br>Banca: Lenice do Rosário de Souza<br>Banca: Alexandre Secorum Borges<br>Resumo: O Vírus da Imunodeficiência Felina (FIV) é um lentivírus que causa uma síndrome de imunodeficiência em gatos domésticos. O FIV tem sido particularmente utilizado em estudos de resistência viral aos análogos de nucleosídeos devido a Transcriptase Reversa (TR) apresentar propriedades físicas, catalíticas e sensibilidade às drogas semelhantes à TR do HIV. Os objetivos desse trabalho foram tratar com AZT gatos naturalmente infectados com o FIV, fazer o monitoramento da carga vir

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Thomas, Jan. "Feline immunodeficiency virus infection in domestic cats in Western Australia: Prevalence of natural infection and association with clinical and morphological disease." Thesis, Thomas, Jan (1997) Feline immunodeficiency virus infection in domestic cats in Western Australia: Prevalence of natural infection and association with clinical and morphological disease. PhD thesis, Murdoch University, 1997. https://researchrepository.murdoch.edu.au/id/eprint/53185/.

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A series of studies was undertaken to survey the prevalence of Feline Immunodeficiency Virus (FIV) infection in domestic cats in Western Australia and to establish the relationship between infection and states of clinical and morphological disease. The studies established that there was a high prevalence of FIV infection in sick cats in Western Australia. The historical data, clinical and clinicopathological changes affecting these animals were compared with those from sick cats unaffected by FIV. This revealed the typical disease picture seen with FIV infection in cats from Western Austra

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Figueiredo, Andreza Soriano [UNESP]. "Quantificação e seqüênciamento do gene da transcriptase reversa em gatos naturalmente infectados com vírus da imunodeficiência felina tratado com AZT." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/98345.

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Made available in DSpace on 2014-06-11T19:29:32Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-06-22Bitstream added on 2014-06-13T18:39:11Z : No. of bitstreams: 1 figueiredo_as_me_botfmvz.pdf: 290159 bytes, checksum: 38ae46f390705b8d387a7ef9c78d6040 (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>O Vírus da Imunodeficiência Felina (FIV) é um lentivírus que causa uma síndrome de imunodeficiência em gatos domésticos. O FIV tem sido particularmente utilizado em estudos de resistência viral aos análogos de nucleosídeos devido a Transcriptase Reversa (TR) apresen

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Botelho, Sílvia Maria Almeida. "Estudo epidemiológico do vírus da imunodeficiência felina e do vírus da leucemia felina em gatos errantes e assilvestrados da ilha de São Miguel, Açores." Master's thesis, Universidade de Lisboa. Faculdade de Medicina Veterinária, 2014. http://hdl.handle.net/10400.5/6751.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>O vírus da Leucemia Felina (FeLV) e o vírus da Imunodeficiência Felina (FIV) pertencem à família Retroviridae. São responsáveis por duas viroses que ameaçam a vida e o bem-estar do gato doméstico, e a conservação de felinos silvestres como o lince da Península Ibérica. O principal objetivo deste estudo epidemiológico foi detetar a presença do FIV e do FeLV em gatos residentes na ilha de São Miguel, Açores. A amostra foi constituída por 90 gatos selecionados em grupos de risco elevado ou com sinais clínicos compatíveis com estas vir

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Fernandes, Marta Freixo. "Avaliação retrospetiva da resposta ao tratamento cirúrgico da gengivo-estomatite crónica em gatos infetados com o vírus da imunodeficiência felina." Master's thesis, Universidade de Lisboa, Faculdade de Medicina Veterinária, 2019. http://hdl.handle.net/10400.5/18934.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>A gengivo-estomatite crónica felina é uma síndrome multifactorial que pode ocorrer em gatos infetados com o vírus da imunodeficiência felina como uma manifestação desta doença. O seu tratamento inclui diversas opções terapêuticas médicas, embora as extrações dentárias sejam a medida considerada mais eficaz até ao momento. O objetivo do presente trabalho consistiu em estudar a resposta ao tratamento cirúrgico da gengivo-estomatite crónica felina, em gatos infetados pelo vírus da imunodeficiência felina. A amostra em estudo foi const

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