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Books on the topic 'Fetal autopsy'

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1

Becker, M. J. Pathology of late fetal stillbirth. Churchill Livingstone, 1989.

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2

E, Becker Anton, ed. Pathology of late fetal stillbirth. Churchill Livingstone, 1989.

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3

Gilbert-Barness, Enid. Handbook of pediatric autopsy pathology. Humana Press, 2005.

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4

Perinatal pathology. 2nd ed. Saunders, 1996.

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5

Debich-Spicer, Diane E., and Enid Gilbert-Barness. Handbook of Pediatric Autopsy Pathology. Humana, 2010.

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6

Debich-Spicer, Diane E., and Enid Gilbert-Barness. Handbook of Pediatric Autopsy Pathology. Humana Press Inc.,U.S., 2004.

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7

Gilbert-Barness, Enid, Diane E. Spicer, and Thora S. Steffensen. Handbook of Pediatric Autopsy Pathology. Springer, 2013.

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8

Gilbert-Barness, Enid, Diane E. Spicer, and Thora S. Steffensen. Handbook of Pediatric Autopsy Pathology. Springer, 2013.

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9

Gilbert-Barness, Enid, and Diane E. Debich-Spicer. Handbook of Pediatric Autopsy Pathology. Humana Press, 2004.

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10

Cohen, Marta C., and Irene Scheimberg. Pediatric and Perinatal Autopsy Manual. University of Cambridge ESOL Examinations, 2014.

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11

The Pediatric and Perinatal Autopsy Manual with DVD-ROM. Cambridge University Press, 2014.

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12

Fetal and Neonatal Pathology. 3rd ed. Springer, 2001.

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13

Keeling, Jean W. Fetal and Neonatal Pathology. Springer, 2013.

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14

Keeling, Jean W. Fetal and Neonatal Pathology. Springer, 2014.

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15

Dr. Larissa V. Furtado M.D., Dr. Jessica M. Comstock M.D., and Dr. Staci Bryson M.D. Diagnostic Pathology : Fetal Histology: Published by Amirsys. Lippincott Williams & Wilkins, 2013.

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16

(Editor), Jean W. Keeling, and T. Yee Khong (Editor), eds. Fetal and Neonatal Pathology. 4th ed. Springer, 2007.

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17

Sheppard, Mary N. Myocardial non-compaction. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0026.

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Abstract:
Isolated left ventricular non-compaction is a controversial entity which has only been reported in the past 30 years. It is becoming more frequently diagnosed due to the use of echocardiography and MRI. It can present in fetal life, infancy, childhood, and adult life. Clinically, the patient can present with cardiac arrhythmias, cardiac failure, systemic emboli due to thrombosis within the ventricles, and sudden death. It can be a genetic entity associated with mutations in many genes associated with hypertrophic cardiomyopathy, dilated cardiomyopathy, and arrhythmogenic cardiomyopathy. It is a rare entity found at autopsy and is more common in children than adults. In the past the prognosis has been considered worse in children then in adults. Treatment is usually empirical, dealing with the cardiac failure, arrhythmias, and thromboemboli.
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