Relevant bibliographies by topics / Fetal germ cell

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Fetal germ cell'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Fetal germ cell"

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Frazier, A. Lindsay, Christopher Weldon, and James Amatruda. "Fetal and neonatal germ cell tumors." Seminars in Fetal and Neonatal Medicine 17, no. 4 (2012): 222–30. http://dx.doi.org/10.1016/j.siny.2012.05.004.

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Western, P., J. Van Den Bergen, D. Miles, R. Ralli, and A. Sinclair. "002. REGULATION OF PLURIPOTENCY AND CELL CYCLE IN FETAL GERM CELLS." Reproduction, Fertility and Development 21, no. 9 (2009): 2. http://dx.doi.org/10.1071/srb09abs002.

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The germ cell lineage is unique in that it must ensure that the genome retains the complete developmental potential (totipotency) that supports development in the following generation. This is achieved through a number of mechanisms that prevent the early germ cell lineage from somatic differentiation and promote the capactity for functional totipotency. Part of this process involves the retained germ line expression of key genes that regulate pluripotency in embryonic stem cells, embryonic germ cells and some embryonal carcinoma cells, the stem cells of testicular tumours. Despite this, germ
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Angenard, Gaëlle, Vincent Muczynski, Hervé Coffigny, et al. "Cadmium Increases Human Fetal Germ Cell Apoptosis." Environmental Health Perspectives 118, no. 3 (2010): 331–37. http://dx.doi.org/10.1289/ehp.0900975.

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Isaacs, Hart. "Perinatal (fetal and neonatal) germ cell tumors." Journal of Pediatric Surgery 39, no. 7 (2004): 1003–13. http://dx.doi.org/10.1016/j.jpedsurg.2004.03.045.

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Lawaetz, Andreas C., and Kristian Almstrup. "Involvement of epigenetic modifiers in the pathogenesis of testicular dysgenesis and germ cell cancer." Biomolecular Concepts 6, no. 3 (2015): 219–27. http://dx.doi.org/10.1515/bmc-2015-0006.

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AbstractTesticular germ cell cancer manifests mainly in young adults as a seminoma or non-seminoma. The solid tumors are preceded by the presence of a non-invasive precursor cell, the carcinoma in situ cell (CIS), which shows great similarity to fetal germ cells. It is therefore hypothesized that the CIS cell is a fetal germ cell that has been arrested during development due to testicular dysgenesis. CIS cells retain a fetal and open chromatin structure, and recently several epigenetic modifiers have been suggested to be involved in testicular dysgenesis in mice. We here review the possible in
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van Vorstenbosch, C. J., E. Spek, B. Colenbrander, and C. J. Wensing. "The ultrastructure of normal fetal and neonatal pig testis germ cells and the influence of fetal decapitation on the germ cell development." Development 99, no. 4 (1987): 553–63. http://dx.doi.org/10.1242/dev.99.4.553.

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The development of germ cells in the male pig was investigated ultrastructurally in normal and decapitated fetuses. The age ranged respectively from 30 days p.c. till one month after birth and from 52 days p.c. until birth. The ultrastructural organization of the germ cells changes dramatically between 30 days p.c. and 52 days p.c. which coincides with the formation of ‘true’ sex cords. From 52 days p.c. onwards the morphology is rather stable: cells show a ‘hydrated’ appearance and typical cell bridges. There is no obvious difference in the ultrastructure of germ cells in decapitated animals,
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Childs, Andrew J., Philippa Saunders, and Richard A. Anderson. "Modeling Human Fetal Germ Cell Development In Vitro." Biology of Reproduction 78, Suppl_1 (2008): 300. http://dx.doi.org/10.1093/biolreprod/78.s1.300.

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Chappell, Vesna A., Brett D. Keiper, and Christopher B. Geyer. "Translational Control During Fetal Male Germ Cell Development." Biology of Reproduction 87, Suppl_1 (2012): 137. http://dx.doi.org/10.1093/biolreprod/87.s1.137.

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De Felici, Massimo, Francesca Klinger, Federica Campolo, Carmela Balistreri, Marco Barchi, and Susanna Dolci. "To Be or Not to Be a Germ Cell: The Extragonadal Germ Cell Tumor Paradigm." International Journal of Molecular Sciences 22, no. 11 (2021): 5982. http://dx.doi.org/10.3390/ijms22115982.

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In the human embryo, the genetic program that orchestrates germ cell specification involves the activation of epigenetic and transcriptional mechanisms that make the germline a unique cell population continuously poised between germness and pluripotency. Germ cell tumors, neoplasias originating from fetal or neonatal germ cells, maintain such dichotomy and can adopt either pluripotent features (embryonal carcinomas) or germness features (seminomas) with a wide range of phenotypes in between these histotypes. Here, we review the basic concepts of cell specification, migration and gonadal coloni
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Spiller, Cassy M., Josephine Bowles, and Peter Koopman. "Nodal/Cripto signaling in fetal male germ cell development: implications for testicular germ cell tumors." International Journal of Developmental Biology 57, no. 2-3-4 (2013): 211–19. http://dx.doi.org/10.1387/ijdb.130028pk.

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Dissertations / Theses on the topic "Fetal germ cell"

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Cowan, Gillian. "Fetal germ cell differentiation and the impact of the somatic cells." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/4164.

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Specification of a germ cell lineage and appropriate maturation are essential for the transfer of genetic information from one generation to the next. Germ cells form from pluripotent precursor cells that migrate into the gonadal ridge and undergo commitment to either the female or male lineage. In the fetal ovary, germ cells enter meiotic prophase I, then arrest at the diplotene stage; in the testis germ cells do not begin meiosis until puberty. Abnormal differentiation of germ cells can result in malignant transformation. Somatic cells play a key role in modulating the developmental fate of
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Mitchell, Roderick T. "Germ cell development in the human and marmoset fetal testis and the origins of testicular germ cell tumours." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4818.

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Normal germ cell development in the human testis is crucial for subsequent fertility and reproductive health. Disruption of testis development in fetal life can result in deleterious health consequences such as testicular dysgenesis syndrome (TDS), which includes disorders, such as cryptorchidism, hypospadias, infertility and testicular germ cell tumours (TGCT). A rat model of TDS in which rats are exposed to phthalates in utero has been validated, but does result in the development of TGCT. In humans, TGCTs result from transformation of pre-neoplastic carcinoma in-situ (CIS) cells and these C
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Okumura, Leah M. "Germ cell nuclear factor is not required for the down-regulation of pluripotency markers in fetal ovarian germ cells." Thesis, Massachusetts Institute of Technology, 2012. http://hdl.handle.net/1721.1/77781.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Biology, 2012.<br>Cataloged from PDF version of thesis.<br>Includes bibliographical references.<br>In mouse, germ cells retain expression of the pluripotency markers Oct4 and Nanog longer than any other cells in the body. While somatic cells repress these markers during gastrulation, female germ cells continue to express them until around the time of meiotic initiation. It is not yet clear why pluripotency markers are downregulated with this particular timing, nor is it understood what factors are involved in their repression. I
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Kasai, Shinya. "Haploinsufficiency of Bcl-x leads to male specific defects in fetal germ cells : differential regulation of germ cell apoptosis between the sexes." Kyoto University, 2004. http://hdl.handle.net/2433/148262.

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Ene, Adriana. "Meiotic prophase progression and germ cell elimination in fetal and neonatal mouse ovaries." Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=92367.

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In most mammalian species, all oogonia cease mitotic proliferation and enter meiosis in fetal ovaries. Furthermore, more than half of the maximum number of germ cells is eliminated from ovaries by neonatal life, thus limiting the oocyte reserve for reproduction. The cause or mechanism of this female germ cell loss remains largely unknown. A major loss occurs in the oocytes which reach the pachytene stage of meiotic prophase, suggesting that oocytes with meiotic or recombination errors may be eliminated by a checkpoint mechanism. It remains to be determined whether oocytes are eliminated by apo
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Jobling, Matthew S. "Fetal germ cell development in the rat testis and the impact of di (n-Butyl) phthalate exposure." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4803.

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During gonad development and fetal life, the germ cells (GC) undergo a range of different developmental processes necessary for correct postnatal gametogenesis and the production of the next generation. If these fetal events are disrupted by genetic or environmental factors, there could be severe consequences that may not present until adulthood. This is of particular importance in relation to human testicular GC tumours (TGCT), the most common cancer of young men, as TGCT is thought to arise from fetal GCs that have failed to differentiate normally during development and thus persist into adu
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Barkley, Nicole Marie Garverick Henry Allen. "Characterization of apoptosis in the developing bovine fetal ovary association with germ cell loss /." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/6105.

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The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract, appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on October 9, 2009) Thesis advisor: Dr. H. Allen Garverick. Vita. Includes bibliographical references.
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He, Jing. "Investigating the expression and function of DAZL and BOLL during human oogenesis." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25481.

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Fetal germ cell development is a key stage of female reproductive life. The DAZ family proteins (DAZ, DAZL and BOLL) are RNA-binding proteins with critical roles in murine germ cell development but their expression and potential targets in the human are largely unknown. The studies in this Thesis investigated the expression and function of DAZL and BOLL in human fetal ovary. Both DAZL and BOLL mRNA are increased dramatically at the time of entry into meiosis. Immunohistochemical analysis with specific meiotic markers suggested that DAZL and BOLL have distinct spatial-temporal expression patter
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Muczynski, Vincent. "Polluants environnementaux et développement du testicule foetal humain : effets et mécanismes des phtalates." Phd thesis, Université Paris Sud - Paris XI, 2011. http://tel.archives-ouvertes.fr/tel-00631554.

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Au cours des dernières décennies, nous avons progressivement vu augmenter un certain nombre d'anomalies de la fonction de reproduction masculine dans les pays industrialisés. Ces constatations ont fait émerger l'hypothèse selon laquelle certains polluants de notre environnement pourraient altérer le développement du testicule fœtal et ainsi être responsables de ces anomalies. Parmi les composants incriminés se trouvent les phtalates, largement répandus dans l'environnement. Ces composés ont été décrits comme reprotoxiques, ils altèrent le développement de la lignée germinale dans différentes e
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Hurtado, Gonzalez Pablo Ignacio. "The consequences of fetal exposure to analgesics for germ cells." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/29631.

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Despite the general advice of avoiding medication during pregnancy, the majority of pregnant woman use one or more ‘over the counter’ analgesics. During the last few years there has been growing evidence that analgesic exposure, such as paracetamol, ibuprofen or indomethacin, during pregnancy can have detrimental effects on rodent and human fetal gonads. The majority of previous studies have focused in alterations in testosterone production and male reproductive disorders. However, few studies have analysed the effect of these analgesics on fetal germ cells and possible consequences on fertili
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Books on the topic "Fetal germ cell"

1

Brian, Dale, ed. In vitro fertilization. 2nd ed. Cambridge University Press, 2000.

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1946-, Elder Kay, ed. In vitro fertilization. Cambridge University Press, 1997.

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Alan, Trounson, and Wood Carl, eds. Atlas of fine structure of human sperm penetration, eggs, and embryos cultured in vitro. Praeger, 1985.

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Book chapters on the topic "Fetal germ cell"

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Spiller, Cassy M., Guillaume Burnet, and Josephine Bowles. "Mouse Fetal Germ Cell Isolation and Culture Techniques." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-4017-2_13.

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Leendert, Robert A. Olie, H. J. Looijenga, et al. "Growth of Human Seminoma Cells on STO Feeder Depends on Phenotype, Presence of Fetal Calf Serum and Added Growth Factors." In Germ Cell Tumours III. Elsevier, 1994. http://dx.doi.org/10.1016/b978-0-08-042198-8.50028-5.

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Felici, Massimo De, and Susanna Dolci. "Chapter 7 Cellular Interactions of Mouse Fetal Germ Cells In In Vitro Systems." In Current Topics in Developmental Biology. Elsevier, 1987. http://dx.doi.org/10.1016/s0070-2153(08)60623-7.

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Conference papers on the topic "Fetal germ cell"

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Looijenga, Leendert H. J., Hendrik Wermann, Ad Gillis, et al. "Abstract LB-67: Global DNA methylation in fetal human germ cells and germ cell tumors: correlation with differentiation and cisplatin resistance." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-67.

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Soker, Shay, Dawn Delo, Samira Neshat, and Anthony Atala. "Amniotic Fluid Derived Stem Cells for Cardiac Muscle Therapies." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192492.

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Many forms of pediatric and adult heart disease are accompanied by high morbidity and mortality, as the heart muscle has limited regenerative potential. Cell therapy has been proposed as a means to promote the regeneration of injured heart muscle. We have established lines of broad spectrum multipotent stem cells derived from primitive fetal cells present in human amniotic fluid (hAFS) cells (1). AFS cells offer several advantages: They are easy to isolate and grow (no feeder layers needed), are highly expansive including clonal growth and they can differentiate into all germ layers. In the cu
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