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Books on the topic 'Fetal heart development'

Author: Grafiati
Published: 9 March 2023

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1

Jeffrey, Lipshitz, ed. Perinatal development of the heart and lung: Proceedings of the First International Christie Conference, Banff, Alberta, Canada. Ithaca, N.Y: Perinatology Press, 1987.

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2

J, Moss Arthur, and Allen Hugh D, eds. Moss and Adams' heart disease in infants, children, and adolescents: Including the fetus and young adult. 7th ed. Philadelphia: Lippincott Williams & Wilkins, 2008.

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3

Karen, Bellenir, ed. Congenital disorders sourcebook: Basic information about disorders acquired during gestation, including spina bifida, hydrocephalus, cerebral palsy, heart defects, craniofacial abnormalities, fetal alcohol syndrome, and more, along with current treatment options and statistical data. Detroit, MI: Omnigraphics, 1997.

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4

J, Judd Sandra, ed. Congenital disorders sourcebook: Basic consumer health information about nonhereditary birth defects and disorders related to prematurity, gestational injuries, congenital infections, and birth complications, including heart defects, hydrocephalus, spina bifida, cleft lip and palate, cerebral palsy, and more; along with facts about the prevention of birth defects, fetal surgery and other treatment options, research initiatives, a glossary of related terms, and resources for additional information and support. 2nd ed. Detroit, MI: Omnigraphics, 2007.

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5

(Editor), Simcha Yagel, Norman H. Silverman (Editor), and Ulrich Gembruch (Editor), eds. Fetal Cardiography. Taylor & Francis, 2002.

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6

Monk, Catherine, and Amie Ashley Hane. Fetal and Infant Neurobehavioral Development. Edited by Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.20.

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This chapter reviews the literature examining fetal and infant neurobehavioral development. Basic fetal neurological development and neurobehavioral functioning are reviewed. Major fetal neurobehavioral milestones and their assessment are addressed and include fetal behavioral states, heart rate, movement, and responsivity to stimuli. The processes of neurological growth from birth to age 2 are reviewed. Infant neurobehavioral development is addressed and includes state regulation and sleep, physical growth and motor development, and the basic processes underlying social-emotional development. For fetus and infant, research examining the associations between neurobehavioral development and maternal distress and poverty is reviewed. The implications for future directions in fetal-infant neurobehavioral research are discussed.
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7

Moss and Adams' Heart Disease in Infants, Children, and Adolescents : Including the Fetus and Young Adult (2 Volume Set). 6th ed. Lippincott Williams & Wilkins, 2001.

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8

J, Moss Arthur, Adams Forrest H, and Emmanouilides George C. 1926-, eds. Moss and Adams heart disease in infants, children, and adolescents: Including the fetus and young adult. 5th ed. Baltimore: Williams & Wilkins, 1995.

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9

Emmanouilides, George C., Arthur J. Moss, and Forrest H. Adams. Moss and Adams Heart Disease in Infants, Children, and Adolescents: Including the Fetus and Young Adult (2 Volume Set). 5th ed. Williams & Wilkins, 1995.

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10

1933-, Moller James H., and Neal William A. 1940-, eds. Fetal, neonatal, and infant cardiac disease. Norwalk, Conn: Appleton & Lange, 1990.

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11

Allen, Hugh D., David J. Driscoll, Robert E. Shaddy, and Timothy F. Feltes. Moss and Adams' Heart Disease in Infants, Children, and Adolescents: Including the Fetus and Young Adult. 7th ed. Lippincott Williams & Wilkins, 2007.

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12

Toomey, Kay Ann. The predictive validity of fetal heart rate for infant development at four months of age. 1986.

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13

Dempsey, Adam Abraham. Computational exploration of the human cardiovascular genome and its application to understanding human fetal heart development. 2003.

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14

Wilton, Niall, Brian J. Anderson, and Bruno Marciniak. Anatomy, physiology, and pharmacology in paediatric anaesthesia. Edited by Jonathan G. Hardman and Neil S. Morton. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0069.

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Anaesthesia for children is tempered by changes that occur during both growth and development. Drug dose is affected by size and clearance maturation processes as well as the changing body composition that occurs with age. All organ systems undergo these maturation changes and most are complete within the first few years of life. Normal physiological variables in infancy and childhood are quite different from adults. The central nervous, cardiovascular, and respiratory systems are particularly important. Cerebral immaturity and plasticity impacts sensitivity to drugs, pain responses, and behaviour and increases potential harm from apoptosis with anaesthesia. The heart undergoes a transition from fetal to adult circulation during the first few weeks of life. Undiagnosed congenital defects are not uncommon. The neonate is very susceptible to conditions that trigger an increase in pulmonary vascular resistance, with reversion to fetal circulatory patterns. Respiratory anatomy and mechanics affect the propensity to apnoea, airway maintenance, artificial ventilation modalities, uptake of inhalational agents, and tracheal tube sizes. Metabolic rate and oxygen requirements increase with decreasing age. This physiology influences diverse aspects that include the rate of desaturation during apnoea, hypoglycaemia during starvation, cardiac output, drug metabolism, fluid requirements, and heat production or loss.
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15

Khamashta, Munther A., Graham R. V. Hughes, and Guillermo Ruiz-Irastorza. Anti-phospholipid antibody syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0120.

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The anti-phospholipid syndrome (APS) described almost 30 years ago, is now recognized as a major cause of deep vein thrombosis, stroke, and heart attacks in young people (<45 years of age). It is also the commonest treatable cause of recurrent miscarriages and a major cause of late fetal death. Other clinical manifestations are cardiac valvular disease, livedo reticularis, renal thrombotic microangiopathy, thrombocytopenia, haemolytic anaemia, epilepsy, and cognitive impairment. The presence of anti-phospholipid antibodies (aPL) has been closely related to the development of thrombosis and complications in pregnancy. However, not all patients with aPL will develop the clinical features. Lupus anticoagulant is generally thought to be more strongly associated with the risk of clinical manifestations of APS than anticardiolipin and anti ?2-glycoprotein I antibodies. The exact pathogenic mechanisms leading to thrombosis and/or pregnancy morbidity are poorly understood. Therapy of thrombosis is based on long-term oral anti-coagulation and patients with arterial events should be treated aggressively. Obstetric care is based on combined medical-obstetric high-risk management and treatment with aspirin and heparin.
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16

Khamashta, Munther A., Graham R. V. Hughes, and Guillermo Ruiz-Irastorza. Anti-phospholipid antibody syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0120_update_001.

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Abstract:
The anti-phospholipid syndrome (APS) described almost 30 years ago, is now recognized as a major cause of deep vein thrombosis, stroke, and heart attacks in young people (<45 years of age). It is also the commonest treatable cause of recurrent miscarriages and a major cause of late fetal death. Other clinical manifestations are cardiac valvular disease, livedo reticularis, renal thrombotic microangiopathy, thrombocytopenia, haemolytic anaemia, epilepsy, and cognitive impairment. The presence of anti-phospholipid antibodies (aPL) has been closely related to the development of thrombosis and complications in pregnancy. However, not all patients with aPL will develop the clinical features. Lupus anticoagulant is generally thought to be more strongly associated with the risk of clinical manifestations of APS than anticardiolipin and anti ?2-glycoprotein I antibodies. The exact pathogenic mechanisms leading to thrombosis and/or pregnancy morbidity are poorly understood. Therapy of thrombosis is based on long-term oral anti-coagulation and patients with arterial events should be treated aggressively. Obstetric care is based on combined medical-obstetric high-risk management and treatment with aspirin and heparin.
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17

Kalter, Harold. Mortality and Maldevelopment. Springer, 2008.

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18

Mortality and Maldevelopment: Part I: Congenital cardiovascular malformations. Springer, 2007.

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