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Books on the topic 'Fetal origins of adult disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

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1

Huang, He-Feng, and Jian-Zhong Sheng, eds. Gamete and Embryo-fetal Origins of Adult Diseases. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-007-7772-9.

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2

Langley-Evans, S. C., ed. Fetal nutrition and adult disease: programming of chronic disease through fetal exposure to undernutrition. Wallingford: CABI, 2004. http://dx.doi.org/10.1079/9780851998213.0000.

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3

Marelyn, Wintour E., and Owens Julie A, eds. Early life origins of health and disease. New York, N.Y: Springer Science+Business Media, 2006.

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4

Gregory, Bock, Whelan Julie, and Symposium on the Childhood Environment and Adult Disease (1990 : Ciba Foundation), eds. The Childhood environment and adult disease. Chichester: Wiley, 1991.

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5

C, Azmitia Efrain, Björklund Anders 1945-, and New York Academy of Sciences., eds. Cell and tissue transplantation into the adult brain. New York, N.Y: New York Academy of Sciences, 1987.

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6

A perinatal strategy for preventing adult disease: The role of long-chain polyunsaturated fatty acids. Boston, Mass: Kluwer Academic Publishers, 2002.

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7

Fetal & Infant Origins of Adult Disease. Bmj Publishing Group, 1992.

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8

Fetal and Infant Origins of Adult Disease. Amer College of Physicians, 1992.

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9

Huang, He-Feng, and Jian-Zhong Sheng. Gamete and Embryo-fetal Origins of Adult Diseases. Springer, 2016.

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10

Huang, He-Feng, and Jian-Zhong Sheng. Gamete and Embryo-fetal Origins of Adult Diseases. Springer, 2013.

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11

Barker, David J. Fetal Origins of Cardiovascular and Lung Disease. Taylor & Francis Group, 2000.

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12

C, Langley-Evans S., ed. Fetal nutrition and adult disease: Programming of chronic disease through fetal exposure to undernutrition. Cambridge, MA: CABI Pub. in association with The Nutrition Society, 2004.

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13

Fetal Origins of Cardiovascular and Lung Disease (Lung Biology in Health and Disease). Informa Healthcare, 2001.

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14

Langley-Evans, S. C. Fetal Nutrition and Adult Disease: Programming of Chronic Disease through Fetal Exposure to Undernutrition (Frontiers in Nutritional Science). CABI, 2004.

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15

(Editor), Deborah Hodgson, and Christopher Coe (Editor), eds. Perinatal Programming: Early Life Determinants of Adult Health & Disease. Informa Healthcare, 2005.

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16

M, Hodgson Deborah, and Coe Christopher L, eds. Perinatal programming: Early life determinants of adult health & disease. London: Taylor & Francis, 2006.

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17

Archer, Nick, and Nicky Manning. Aetiology of fetal heart disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198766520.003.0002.

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This chapter discusses the aetiology of fetal heart disease, including discussion on maternal factors, genetic factors, genetic causes, risk of associated non-cardiac anomalies, prevention of congenital heart disease, and the fetal origins of health and disease.

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18

J, Moss Arthur, Adams Forrest H, and Emmanouilides George C. 1926-, eds. Moss and Adams heart disease in infants, children, and adolescents: Including the fetus and young adult. 5th ed. Baltimore: Williams & Wilkins, 1995.

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19

Moss and Adams' Heart Disease in Infants, Children, and Adolescents : Including the Fetus and Young Adult (2 Volume Set). 6th ed. Lippincott Williams & Wilkins, 2001.

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20

P, Newnham John, and Ross Michael G, eds. Early life origins of human health and disease. Basel: Karger, 2009.

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21

Emmanouilides, George C., Arthur J. Moss, and Forrest H. Adams. Moss and Adams Heart Disease in Infants, Children, and Adolescents: Including the Fetus and Young Adult (2 Volume Set). 5th ed. Williams & Wilkins, 1995.

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22

FAHA, Hugh D. Allen MD FACC FAAP. Moss & Adams’ Heart Disease in Infants, Children, and Adolescents, Including the Fetus and Young Adult. LWW, 2016.

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23

Allen, Hugh D., David J. Driscoll, Robert E. Shaddy, and Timothy F. Feltes. Moss and Adams' Heart Disease in Infants, Children, and Adolescents: Including the Fetus and Young Adult. 7th ed. Lippincott Williams & Wilkins, 2007.

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24

J, Moss Arthur, and Allen Hugh D, eds. Moss and Adams' heart disease in infants, children, and adolescents: Including the fetus and young adult. 7th ed. Philadelphia: Lippincott Williams & Wilkins, 2008.

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25

Page, Kathleen C., Susan Ozanne, and Endla Katalin Anday, eds. Impact of Fetal & Early Postnatal Nutrition on the Developing Brain: Implication for Adult Disease. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-724-8.

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26

Das, Undurti N. Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer, 2012.

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27

Das, Undurti N. Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer London, Limited, 2011.

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28

Das, Undurti N. A Perinatal Strategy for Preventing Adult Disease: The Role of Long-Chain Polyunsaturated Fatty Acids. Springer, 2002.

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29

Macnab, Andrew J., Abdallah Daar, and Christoff Pauw, eds. Health in Transition: Translating developmental origins of health and disease science to improve future health in Africa. African Sun Media, 2020. http://dx.doi.org/10.18820/9781928357759.

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At STIAS, the ‘Health in Transition’ theme includes a programme to address the epidemic rise in the incidence of non-communicable diseases (NCDs) such as Type 2 diabetes, hypertension, obesity, coronary heart disease and stroke in Africa. The aim is to advance awareness, research capacity and knowledge translation of science related to the Developmental Origins of Health and Disease (DOHaD) as a means of preventing NCDs in future generations. Application of DOHaD science is a promising avenue for prevention, as this field is identifying how health and nutrition from conception through the first 1 000 days of life can dramatically impact a developing individual’s future life course, and specifically predicate whether or not they are programmed in infancy to develop NCDs in later life. Prevention of NCDs is an essential strategy as, if unchecked, the burden of caring for a growing and ageing population with these diseases threatens to consume entire health budgets, as well as negatively impact the quality of life of millions. Africa in particular needs specific, focussed endeavors to realize the maximal preventive potential of DOHaD science, and a means of generating governmental and public awareness about the links between health in infancy and disease in adult life. This volume summarizes the expertise and experience of a leading group of international scientists led by Abdallah Daar brought together at STIAS as part of the ‘Health in Transition’ programme.

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30

Ruiz-Villalba, Adrián, Nikolaos Frangogiannis, and José Maria Pérez-Pomares. Origin and diversity of cardiac fibroblasts: developmental substrates of adult cardiac fibrosis. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0012.

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Cardiac connective tissues are primarily formed by cardiac fibroblasts (CF) of diverse embryonic origins. Whereas CF specific roles in cardiac morphogenesis remain under-researched, their involvement in adult cardiac fibrosis is clinically relevant. Cardiac fibrosis is a common element of several chronic cardiac conditions characterized by the loss of ventricular wall mechanical function, ultimately driving to heart failure. In the ischaemic heart early reparative fibrosis evidences the very restricted regenerative potential of the myocardium. In non-ischaemic diseases fibrosis is activated by unknown signals. We summarize current knowledge on the origin of CFs and their developmental roles, and discuss the differential disease-dependent response of different CF subpopulations to various pathological stimuli. We also describe the characteristic cell-cell and cell-matrix interactions that determine the fibrotic remodelling of the myocardium. We analyse experimental models for the study of cardiac fibrosis, and suggest future directions in the search for new markers and therapeutic targets.

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31

Ahmed, Ahmed I., Sarah Aldhaheri, and Allison Bannick. Inherited Metabolic Diseases (IMDs) and Pregnancy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0030.

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Inherited metabolic diseases (IMDs) are rare genetic disorders: clinically heterogeneous, and they can present at any age. With the expanded newborn screening panels, many of the IMDs have been successfully screened. Early diagnosis and treatment of these conditions have led to improved neurological outcomes and overall survival of these individuals, and now many of them are reaching childbearing age. Despite treatment, the potential presence of preexisting organ involvement may not only impact their fertility potentials but also may impose a higher risk of adverse maternal and fetal outcomes. Pregnancy leads to an extra strain on maternal metabolism; this may result in the manifestation of symptoms of a previously unknown disease or a progression of a known disease. This chapter will address the possible complications of some inherited disorders of metabolism that are associated with maternal or fetal neurological manifestations such as disorders of energy metabolism (eg, mitochondrial disorders, adult onset urea cycle disorders, ornithine transcarbamylase (OTC) deficiency, amino acidopathies, phenylketonuria (PKU), and impaired fatty acid oxidation disorders). We will provide special emphasis on the available potential treatments and plan of care during pregnancy and postpartum periods.

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32

Pérez-Pomares, José M., and Robert Kelly, eds. The ESC Textbook of Cardiovascular Development. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.001.0001.

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A rapid inspection of the table of contents shows that we have grouped relevant cardiovascular developmental topics in five different sections, which move progressively from basic research to clinical relevance, concluding with a glance at the near future of this fast-moving field. All of these sections deal with concepts that are critical to understanding from where and how cardiac chambers (atria and ventricles), valves (atrioventricular and arterial), great vessels (aortic and pulmonary trunks), cardiac conduction system (nodes, bundles, and Purkinje fibres), and coronary blood vessels form. Throughout the book there is continuous reference to experimental animal models for developmental processes, including the mouse, chick, and zebrafish, often involving the application of state of the art technological innovations. This has allowed us to illustrate the more likely origins of specific forms of congenital heart disease, and to elaborate on the developmental substrate of certain forms of adult cardiovascular disease.

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