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Books on the topic 'Fetal size'

Author: Grafiati

Published: 4 June 2021

Last updated: 21 February 2023

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1

Kautz, J. Edward. Effects of harvest on feral rock dove survival, nest success and population size. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1990.

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2

Briggs, Gerald G. Drugs in pregnancy and lactation: A reference guide to fetal and neonatal risk. 3rd ed. Baltimore: Williams & Wilkins, 1990.

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3

1935-, Freeman Roger K., and Yaffe Sumner J. 1923-, eds. Drugs in pregnancy and lactation: A reference guide to fetal and neonatal risk. 6th ed. Philadelphia, Pa: Lippincott Williams & Wilkins, 2002.

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4

Briggs, Gerald G. Drugs in pregnancy and lactation: A reference guide to fetal and neonatal risk on CD-ROM. [Philadelphia, Pa.]: Lippincott, Williams & Wilkins, 1999.

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5

Ried, Sibylle. Epilepsy, pregnancy, and the child. Oxford: Blackwell Science, 1996.

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6

1939-, Scanlon John W., ed. Perinatal anesthesia. Boston: Blackwell Scientific Publications, 1985.

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7

1935-, Freeman Roger K., and Yaffe Sumner J. 1923-, eds. Drugs in Pregnancy and Lactation. 7th ed. Baltimore: Lippincott Williams & Wilkins, 2005.

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8

P, Barker D. J., ed. Fetal and placental size and risk of hypertension in adult life. 1990.

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9

Anne, Dorita. The effect of colony size on fetal resorption and secondary sex ratio in domestic Chinchilla laniger. 1985.

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10

Archer, Nick, and Nicky Manning. Nuchal translucency and the heart. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199230709.003.0019.

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Introduction 264The nuchal scan 266Management 268Nuchal translucency describes sonolucent tissue in the posterior aspect of the fetal neck; the size can be measured with accuracy during the 1st trimester of pregnancy and an increase in the measurement is associated with an increased risk of chromosomal abnormality. If fetal karyotype is normal: ...

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11

Schreuder, Michiel F. Renal tubular dysgenesis. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0350.

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Renal tubular dysgenesis involves the absence or incomplete differentiation of proximal tubular nephron segments. Due to the lack of a patent nephron, it is characterized by (fetal) anuria and subsequent oligohydramnios, pulmonary hypoplasia, premature birth with severe and refractory arterial hypotension, and fetal or neonatal death. The main cause for renal tubular dysgenesis is a genetic mutation in the renin–angiotensin system, which has shown an autosomal recessive trait. Maternal use of angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers during pregnancy can have

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12

Wilton, Niall, Brian J. Anderson, and Bruno Marciniak. Anatomy, physiology, and pharmacology in paediatric anaesthesia. Edited by Jonathan G. Hardman and Neil S. Morton. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0069.

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Anaesthesia for children is tempered by changes that occur during both growth and development. Drug dose is affected by size and clearance maturation processes as well as the changing body composition that occurs with age. All organ systems undergo these maturation changes and most are complete within the first few years of life. Normal physiological variables in infancy and childhood are quite different from adults. The central nervous, cardiovascular, and respiratory systems are particularly important. Cerebral immaturity and plasticity impacts sensitivity to drugs, pain responses, and behav

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13

Davidson, Andrew, Adrian Bosenberg, and Stephen Stayer. Neonatal anaesthesia. Edited by Jonathan G. Hardman and Neil S. Morton. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0070.

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Neonatal anaesthesia requires an understanding of how neonates differ from adults and older children in anatomy, physiology, and pharmacology. There are also pathological and surgical conditions in neonates that are associated with unique anaesthesia challenges. Organ systems are generally immature, reducing the clearance of many drugs, while different water and fat content results in altered volumes of distribution. Pharmacological management is further complicated by a lack of basic pharmacokinetic data for the use of most anaesthetic drugs in neonates. At birth, there is a transition from a

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14

A, Malecki Richard, and U.S. Fish and Wildlife Service., eds. Effects of harvest on feral rock dove survival, nest success and population size. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1991.

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15

Drugs in Pregnancy and Lactation: A Reference Guide to Fetal and Neonatal Risk. Williams & Wilkins, 1994.

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16

Alchi, Bassam, and David Jayne. The patient with antiphospholipid syndrome with or without lupus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0164.

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Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by recurrent arterial or venous thrombosis and/or pregnancy loss, accompanied by laboratory evidence of antiphospholipid antibodies (aPL), namely anticardiolipin antibodies (aCL), lupus anticoagulant (LA), and antibodies directed against beta-2 glycoprotein 1 (β‎‎‎2GP1). APS may occur as a ‘primary’ form, ‘antiphospholipid syndrome,’ without any known systemic disease or may occur in the context of systemic lupus erythematosus (SLE), ‘SLE-related APS’. APS may affect any organ system and displays a broad spectrum of thromb

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17

Drugs in Pregnancy and Lactation: A Reference Guide to Fetal and Neonatal Risk. 7th ed. Lippincott Williams & Wilkins, 2005.

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18

King, Carolyn, and David Forsyth, eds. Handbook of New Zealand Mammals. CSIRO Publishing, 2021. http://dx.doi.org/10.1071/9781486306299.

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The Handbook of New Zealand Mammals is the only definitive reference on all the land-breeding mammals recorded in the New Zealand region (including the New Zealand sector of Antarctica). It lists 65 species, including native and exotic, wild and feral, living and extinct, residents, vagrants and failed introductions. It describes their history, biology and ecology, and brings together comprehensive and detailed information gathered from widely scattered or previously unpublished sources. The description of each species is arranged under standardised headings for easy reference. Because t

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19

Drug-Induced Pathology. Springer, 2011.

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20

Barrach, H. J., H. V. Gärtner, M. Habs, E. Grundmann, and H. W. Altmann. Drug-Induced Pathology. Springer London, Limited, 2012.

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21

Goffin, Eric, Laura Labriola, and Michel Jadoul. Bacterial and fungal infections in patients on peritoneal dialysis. Edited by Jonathan Himmelfarb. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0270.

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Infections specifically related to peritoneal dialysis include peritonitis on the one hand, and exit-site and tunnel infections on the other hand.The diagnosis of peritonitis rests on the classical triad of cloudy dialysate, abdominal pain, and presence of < 100 white-cells (including < 50 % polymorphonuclear cells) within the dialysate effluent. Because peritonitis is associated with high mortality and morbidity rates, empiric antibiotics should be initiated without delay, covering both Gram-positive and Gram-negative organisms. Most regimens include vancomycin or a first-generation cep

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22

S, Zagon Ian, and Slotkin Theodore A, eds. Maternal substance abuse and the developing nervous system. San Diego: Academic Press, 1992.

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23

Zagon, Ian S., and Theodore A. Slotkin. Maternal Substance Abuse and the Developing Nervous System. Elsevier Science & Technology Books, 2012.

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24

J, Akhurst R., Kavlock Robert J, and Daston George P, eds. Drug toxicity in embryonic development: Advances in understanding mechanisms of birth defects. Berlin: Springer, 1997.

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25

Drug Toxicity in Embryonic Dev Ii: Adv Understanding Mechanisms of Birth Defects; Mechanistic, Etc. Springer-Verlag Telos, 1997.

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26

Drug toxicity in embryonic development I/II: Advances in understanding mechanisms of birth defects. Berlin: Springer, 1996.

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27

(Editor), R. J. Akhurst, George P. Daston (Editor), and Robert J. Kavlock (Editor), eds. Drug Toxicity in Embryonic Dev I: Adv Understanding Mechanisms of Birth Defects: Morphogenesis Etc. Springer-Verlag Telos, 1997.

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28

J, Kavlock Robert, and Daston George P, eds. Drug toxicity in embryonic development I: Advances in understanding mechanisms of birth defects : morphogenesis and processes at risk. Berlin: Springer, 1996.

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29

J, Kavlock Robert, and Daston George P, eds. Drug toxicity in embryonic development II: Advances in understanding mechanisms of birth defects : mechanistic understanding of human developmental toxicants. Berlin: Springer, 1996.

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30

Ried, Sibylle, Sibylle Ri, and Gertrud Beck-Managetta. Epilepsy, Pregnancy and the Child. Blackwell Science, 1997.

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31

Medications & Mothers' Milk: 2019. Springer Publishing Company, Incorporated, 2017.

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32

Drugs in Pregnancy and Lactation. Lippincott Williams & Wilkins, 2011.

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