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Relevant bibliographies by topics / FHA

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Academic literature on the topic 'FHA'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

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Journal articles on the topic "FHA"

1

Golshani, Maryam, Waheed Ur Rahman, Adriana Osickova, Jana Holubova, Jinery Lora, Nataliya Balashova, Peter Sebo, and Radim Osicka. "Filamentous Hemagglutinin of Bordetella pertussis Does Not Interact with the β2 Integrin CD11b/CD18." International Journal of Molecular Sciences 23, no. 20 (October 20, 2022): 12598. http://dx.doi.org/10.3390/ijms232012598.

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The pertussis agent Bordetella pertussis produces a number of virulence factors, of which the filamentous hemagglutinin (FhaB) plays a role in B. pertussis adhesion to epithelial and phagocytic cells. Moreover, FhaB was recently found to play a crucial role in nasal cavity infection and B. pertussis transmission to new hosts. The 367 kDa FhaB protein translocates through an FhaC pore to the outer bacterial surface and is eventually processed to a ~220 kDa N-terminal FHA fragment by the SphB1 protease. A fraction of the mature FHA then remains associated with bacterial cell surface, while most of FHA is shed into the bacterial environment. Previously reported indirect evidence suggested that FHA, or its precursor FhaB, may bind the β2 integrin CD11b/CD18 of human macrophages. Therefore, we assessed FHA binding to various cells producing or lacking the integrin and show that purified mature FHA does not bind CD11b/CD18. Further results then revealed that the adhesion of B. pertussis to cells does not involve an interaction between the bacterial surface-associated FhaB and/or mature FHA and the β2 integrin CD11b/CD18. In contrast, FHA binding was strongly inhibited at micromolar concentrations of heparin, corroborating that the cell binding of FHA is ruled by the interaction of its heparin-binding domain with sulfated glycosaminoglycans on the cell surface.

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2

Julio, Steven M., and Peggy A. Cotter. "Characterization of the Filamentous Hemagglutinin-Like Protein FhaS in Bordetella bronchiseptica." Infection and Immunity 73, no. 8 (August 2005): 4960–71. http://dx.doi.org/10.1128/iai.73.8.4960-4971.2005.

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ABSTRACT Filamentous hemagglutinin (FHA) is a large (>200 kDa), rod-shaped protein expressed by bordetellae that is both surface-associated and secreted. FHA mediates bacterial adherence to epithelial cells and macrophages in vitro and is absolutely required for tracheal colonization in vivo. The recently sequenced Bordetella bronchiseptica genome revealed the presence of a gene, fhaS, that is nearly identical to fhaB, the FHA structural gene. We show that although fhaS expression requires the BvgAS virulence control system, it is maximal only under a subset of conditions in which BvgAS is active, suggesting an additional level of regulation. We also show that, like FHA, FhaS undergoes a C-terminal proteolytic processing event and is both surface-associated and secreted and that export across the outer membrane requires the channel-forming protein FhaC. Unlike FHA, however, FhaS was unable to mediate adherence of B. bronchiseptica to epithelial cell lines in vitro and was not required for respiratory tract colonization in vivo. In a coinfection experiment, a ΔfhaS strain was out-competed by wild-type B. bronchiseptica, indicating that fhaS is expressed in vivo and that FhaS contributes to bacterial fitness in a manner revealed when the mutant must compete with wild-type bacteria. These data suggest that FHA and FhaS perform distinct functions during the Bordetella infectious cycle. A survey of various Bordetella strains revealed two distinct fhaS alleles that segregate according to pathogen host range and that B. parapertussis hu most likely acquired its fhaS allele from B. pertussis horizontally, suggesting fhaS may contribute to host-species specificity.

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Tsai, Ming-Daw. "FHA." Structure 10, no. 7 (July 2002): 887–88. http://dx.doi.org/10.1016/s0969-2126(02)00795-5.

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4

Wilson, Dan R., Annette Siebers, and B. Brett Finlay. "Antigenic Analysis of Bordetella pertussis Filamentous Hemagglutinin with Phage Display Libraries and Rabbit Anti-Filamentous Hemagglutinin Polyclonal Antibodies." Infection and Immunity 66, no. 10 (October 1, 1998): 4884–94. http://dx.doi.org/10.1128/iai.66.10.4884-4894.1998.

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ABSTRACT Although substantial advancements have been made in the development of efficacious acellular vaccines against Bordetella pertussis, continued progress requires better understanding of the antigenic makeup of B. pertussis virulence factors, including filamentous hemagglutinin (FHA). To identify antigenic regions of FHA, phage display libraries constructed by using random fragments of the 10-kbp EcoRI fragment ofB. pertussis fhaB were affinity selected with rabbit anti-FHA polyclonal antibodies. Characterization of antibody-reactive clones displaying FHA-derived peptides identified 14 antigenic regions, each containing one or more epitopes. A number of clones mapped within regions containing known or putative FHA adhesin domains and may be relevant for the generation of protective antibodies. The immunogenic potential of the phage-displayed peptides was assessed indirectly by comparing their recognition by antibodies elicited by sodium dodecyl sulfate (SDS)-denatured and native FHA and by measuring the inhibition of this recognition by purified FHA. FHA residues 1929 to 2019 may contain the most dominant linear epitope of FHA. Clones mapping to this region accounted for ca. 20% of clones recovered from the initial library selection and screening procedures. They are strongly recognized by sera against both SDS-denatured and native FHA, and this recognition is readily inhibited by purified FHA. Given also that this region includes a factor X homolog (J. Sandros and E. Tuomanen, Trends Microbiol. 1:192–196, 1993) and that the single FHA epitope (residues 2001 to 2015) was unequivocally defined in a comparable study by E. Leininger et al. (J. Infect. Dis. 175:1423–1431, 1997), peptides derived from residues of 1929 to 2019 of FHA are strong candidates for future protection studies.

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Geisthövel. "Functional hyperandrogenism – classification, etiology, diagnostic and therapy." Therapeutische Umschau 59, no. 4 (April 1, 2002): 163–73. http://dx.doi.org/10.1024/0040-5930.59.4.163.

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Die dargestellte Klassifizierung des Funktionellen Hyperandrogenismus (FHA) basiert auf lange und seit jüngerer Zeit bekannten klinischen Erkenntnissen, die durch neuere molekularbiologische Vorstellungen untermauert werden. Man kann annehmen, dass sich der FHA aus verschiedenen, organ- oder systemspezifischen Entitäten zusammensetzt, die letztlich unterschiedliche Diagnostik- und Therapiestrategien zur Folge haben. Die Bezeichnung PCOS», welche die verschiedenen Entitäten ungenau und schieflastig umfasst, sollte durch die entsprechenden, adäquaten Begriffsbestimmungen ersetzt werden. Obwohl schon umfangreiche Denkansätze und Kenntnisse zu molekularbiologischen Abläufen beim FHA vorliegen, sind mit Ausnahme der hier nicht besprochenen FHA-Gruppe III (FAHA) die ätiologischen Folgerungen noch nicht gefestigt und die diagnostischen wie therapeutischen Konsequenzen weiterhin limitiert. Allerdings sind in der nahen Zukunft auf der Basis der jüngsten Erkenntnisse des menschlichen Genoms und den neuen genetischen screenings-Möglichkeiten (Microarrays) Weiterentwicklungen zu erwarten.

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Durocher, Daniel, and Stephen P. Jackson. "The FHA domain." FEBS Letters 513, no. 1 (December 20, 2001): 58–66. http://dx.doi.org/10.1016/s0014-5793(01)03294-x.

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7

Alonso, Sylvie, Kévin Pethe, Nathalie Mielcarek, Dominique Raze, and Camille Locht. "Role of ADP-Ribosyltransferase Activity of Pertussis Toxin in Toxin-Adhesin Redundancy with Filamentous Hemagglutinin duringBordetella pertussis Infection." Infection and Immunity 69, no. 10 (October 1, 2001): 6038–43. http://dx.doi.org/10.1128/iai.69.10.6038-6043.2001.

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ABSTRACT Pertussis toxin (PT) and filamentous hemagglutinin (FHA) are two major virulence factors of Bordetella pertussis. FHA is the main adhesin, whereas PT is a toxin with an A-B structure, in which the A protomer expresses ADP-ribosyltransferase activity and the B moiety is responsible for binding to the target cells. Here, we show redundancy of FHA and PT during infection. Whereas PT-deficient and FHA-deficient mutants colonized the mouse respiratory tract nearly as efficiently as did the isogenic parent strain, a mutant deficient for both factors colonized substantially less well. This was not due to redundant functions of PT and FHA as adhesins, since in vitro studies of epithelial cells and macrophages indicated that FHA, but not PT, acts as an adhesin. An FHA-deficient B. pertussis strain producing enzymatically inactive PT colonized as poorly as did the FHA-deficient, PT-deficient strain, indicating that the ADP-ribosyltransferase activity of PT is required for redundancy with FHA. Only strains producing active PT induced a local transient release of tumor necrosis factor alpha (TNF-α), suggesting that the pharmacological effects of PT are the basis of the redundancy with FHA, through the release of TNF-α. This may lead to damage of the pulmonary epithelium, allowing the bacteria to colonize even in the absence of FHA.

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Poulain-Godefroy, Odile, Nathalie Mielcarek, Nathalie Ivanoff, Franck Remoué, Anne-Marie Schacht, Nigel Phillips, Camille Locht, André Capron, and Gilles Riveau. "Bordetella pertussis Filamentous Hemagglutinin Enhances the Immunogenicity of Liposome-Delivered Antigen Administered Intranasally." Infection and Immunity 66, no. 4 (April 1, 1998): 1764–67. http://dx.doi.org/10.1128/iai.66.4.1764-1767.1998.

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ABSTRACT In an attempt to increase the immunogenicity of mucosally delivered antigens, we incorporated the Bordetella pertussisfilamentous hemagglutinin (FHA) adhesin into liposomes containing the glutathione S-transferase of Schistosoma mansoni (Sm28GST) as a model antigen. Outbred mice immunized twice intranasally with liposomes containing a constant suboptimal dose of Sm28GST and increasing doses of FHA produced anti-Sm28GST antibodies in a FHA dose-dependent manner. The addition of 3 μg of FHA to the liposomes induced more than 10-fold-higher anti-Sm28GST antibody titers, compared to those induced by liposomes without FHA. The presence of FHA did not alter the nature of the humoral immune response, and the sera contained anti-Sm28GST immunoglobulin G1 (IgG1), IgG2a, and IgG2b. However, anti-Sm28GST IgA was only detected when at least 3 μg of FHA was added to the preparation. These results show a promising potential for FHA to enhance the immunogenicity of mucosally administered antigens incorporated into liposomes.

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9

Luo, Shukun, Xiaoran Xin, Li-Lin Du, Keqiong Ye, and Yi Wei. "Dimerization Mediated by a Divergent Forkhead-associated Domain Is Essential for the DNA Damage and Spindle Functions of Fission Yeast Mdb1." Journal of Biological Chemistry 290, no. 34 (July 9, 2015): 21054–66. http://dx.doi.org/10.1074/jbc.m115.642538.

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MDC1 is a key factor of DNA damage response in mammalian cells. It possesses two phospho-binding domains. In its C terminus, a tandem BRCA1 C-terminal domain binds phosphorylated histone H2AX, and in its N terminus, a forkhead-associated (FHA) domain mediates a phosphorylation-enhanced homodimerization. The FHA domain of the Drosophila homolog of MDC1, MU2, also forms a homodimer but utilizes a different dimer interface. The functional importance of the dimerization of MDC1 family proteins is uncertain. In the fission yeast Schizosaccharomyces pombe, a protein sharing homology with MDC1 in the tandem BRCA1 C-terminal domain, Mdb1, regulates DNA damage response and mitotic spindle functions. Here, we report the crystal structure of the N-terminal 91 amino acids of Mdb1. Despite a lack of obvious sequence conservation to the FHA domain of MDC1, this region of Mdb1 adopts an FHA-like fold and is therefore termed Mdb1-FHA. Unlike canonical FHA domains, Mdb1-FHA lacks all the conserved phospho-binding residues. It forms a stable homodimer through an interface distinct from those of MDC1 and MU2. Mdb1-FHA is important for the localization of Mdb1 to DNA damage sites and the spindle midzone, contributes to the roles of Mdb1 in cellular responses to genotoxins and an antimicrotubule drug, and promotes in vitro binding of Mdb1 to a phospho-H2A peptide. The defects caused by the loss of Mdb1-FHA can be rescued by fusion with either of two heterologous dimerization domains, suggesting that the main function of Mdb1-FHA is mediating dimerization. Our data support that FHA-mediated dimerization is conserved for MDC1 family proteins.

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Li, J., G. I. Lee, S. R. Van Doren, and J. C. Walker. "The FHA domain mediates phosphoprotein interactions." Journal of Cell Science 113, no. 23 (December 1, 2000): 4143–49. http://dx.doi.org/10.1242/jcs.113.23.4143.

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The forkhead-associated (FHA) domain is a phosphopeptide-binding domain first identified in a group of forkhead transcription factors but is present in a wide variety of proteins from both prokaryotes and eukaryotes. In yeast and human, many proteins containing an FHA domain are found in the nucleus and involved in DNA repair, cell cycle arrest, or pre-mRNA processing. In plants, the FHA domain is part of a protein that is localized to the plasma membrane and participates in the regulation of receptor-like protein kinase signaling pathways. Recent studies show that a functional FHA domain consists of 120–140 amino acid residues, which is significantly larger than the sequence motif first described. Although FHA domains do not exhibit extensive sequence similarity, they share similar secondary and tertiary structures, featuring a sandwich of two anti-parallel (beta)-sheets. One intriguing finding is that FHA domains may bind phosphothreonine, phosphoserine and sometimes phosphotyrosine, distinguishing them from other well-studied phosphoprotein-binding domains. The diversity of proteins containing FHA domains and potential differences in binding specificities suggest the FHA domain is involved in coordinating diverse cellular processes.

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Dissertations / Theses on the topic "FHA"

1

Morris, Erin Rebecca. "FHA domain genes of Arabidopsis /." free to MU campus, to others for purchase, 2004. http://wwwlib.umi.com/cr/mo/fullcit?p3144443.

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2

Hartman, Matěj. "Funkční analýza rizik (FHA) malého letounu." Master's thesis, Vysoké učení technické v Brně. Fakulta strojního inženýrství, 2013. http://www.nusl.cz/ntk/nusl-230779.

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The object of this diploma thesis is to perform Functional Hazard Assessment of small four-seat aircraft according to Federal Aviation Regulations Part 23, which would be similar to present aircrafts on market. Input data were acquired by research of systems aircrafts use on current market. On this basis the Functional Hazard Assessment was performed ad aircraft level. Total loss of power supply was qualified as Catastrophic therefore is used in following assessment. A preliminary failure rate assessment of typical parts used in electrical system was performed at the end of diploma thesis. For the most crucial parts a simple model was created and failure rate computed.

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Qin, Dongyan. "Specificity and structural modeling of FHA domain of CHK2 and a general characterization of FHA domain of caenorhabditis elegans CHK2." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1061304007.

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Li, Hongyuan. "Structure of KI67 FHA domain and its binding to HNIFK." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1060882032.

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Lee, Hyun. "Structural and functional characterization of the yeast DUN1 FHA domain." The Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=osu1155669890.

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Jakl, Jan. "Funkční analýza rizik (FHA) 4-místného letounu pro osobní dopravu." Master's thesis, Vysoké učení technické v Brně. Fakulta strojního inženýrství, 2010. http://www.nusl.cz/ntk/nusl-229297.

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At the beginning this master's thesis includes of a comprehensive review of aircraft accidents in this category, 2-6-digit aircraft for passenger transport. Since this work focused on autopilot, so naturally there is a basic overview of most common autopilots, which can be found in these aircraft now, but in the future. Functional hazard analysis (FHA) for the 4-seater plane for passenger services primarily investigates cases of catastrophic malfunction, which in most cases accompanied by the likelihood taken from different databases. The airplane, which is created for this analysis will preferably equipped with instruments for IFR flights. There is also a brief overview of the regulations necessary for the installation of these systems in the airplane. At the end of this work is to design the dashboard, a design layout of equipment for future aircraft, with an emphasis on maximum transparency.

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7

Baiyee, Martha N. "Attitudes of secondary school students toward home economics according to FHA membership." Virtual Press, 1991. http://liblink.bsu.edu/uhtbin/catkey/774765.

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Yongkiettrakul, Suganya. "Molecular structure and specific interaction of fha domains of saccharomyces cerevisiae rad53." The Ohio State University, 2004. http://rave.ohiolink.edu/etdc/view?acc_num=osu1083529702.

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9

Iles, Natasha J. "The role of a divergent FHA domain in DNA single-strand break repair." Thesis, University of Sussex, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487930.

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XRCC1 plays a major role in the repair of these lesions in mammalian cells by binding and/or activating many components of the single-strand break repair (SSBR) pathway. One such component is polynucleotide kinase (PNK) which possesses a divergent forkhead associated (FHA) domain that binds CK2-phosphorylated XRCC1. Aprataxin has a similar divergent FHA domain that also interacts with XRCC1 and which has been implicated in SSBR. In this thesis, yeast two-hybrid analysis indicated that PNK interacted with the pro-apoptotic protein Hippi in a manner dependent on the PNK FHA domain. In addition, a novel protein containing a similar FHA domain to PNK and aprataxin was identified and denoted APLF (Aprataxin and PNK-Like Factor). APLF was also shown to bind XRCC1 in a manner dependent on its FHA domain. Furthermore, this interaction was greatly stimulated by CK2-phosphorylation of XRCC1. APLF interacted with the double-strand break repair (DSBR) factor XRCC4. APLF was modified following DNA damage, presumably by phosphorylation. Nuclear localisation of YFP-APLF was promoted by the presence of XRCC1. Moreover, YFP-APLF colocalised with RFP-XRCC1 in DNA damage-induced nuclear foci following H₂O₂ treatment. Novel interaction partners of APLF identified by employing a yeast two-hybrid library screen included Ku86/XRCC5 and KEAP1. These data suggest a role for APLF in the cellular response to DNA strand breaks.

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10

Song, Haiyan. "Structural and Functional Study of the Interaction between Ki67 FHA Domain and NIFK." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1204523912.

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Books on the topic "FHA"

1

Austin, Teatro Chicano de. FHA. Alexandria, VA: Alexander Street Press, 2005.

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RCED, United States General Accounting Office. FHA internal controls. Washington, D.C: The Office, 1993.

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United States. Dept. of Housing and Urban Development, ed. FHA multifamily development. [Washington, D.C.?: U.S. Dept. of Housing and Urban Development, 1995.

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Office, General Accounting. FHA internal controls. Washington, D.C: The Office, 1992.

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Lewis, Kathy. Understanding FHA & VA loans. Marietta, Ga: Capstone Institute of Mortgage Finance, 1999.

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Diehl, Cheryl J. HUD/FHA quality control manual. 2nd ed. Chicago (111 E. Wacker Dr., Chicago 60601): Institute of Financial Education, 1994.

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Lewis, Kathy. FHA underwriter's: Direct endorsement survival handbook. Marietta, Ga: Capstone Institute of Mortgage Finance, 1999.

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8

Santi, Albert. Questions & answers for FHA, VA & conventional loans. 3rd ed. Memphis, TN: Mortgage Techniques, 1987.

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Santi, Albert. Questions & answers for FHA, VA & conventional loans. 2nd ed. Germantown, TN: Mortgage Techniques, 1985.

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Santi, Albert. Questions & answers for FHA, VA & conventional loans. 2nd ed. Memphis, TN: Mortgage Techniques, 1986.

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Book chapters on the topic "FHA"

1

Durocher, Daniel. "The FHA Domain." In Modular Protein Domains, 143–62. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527603611.ch7.

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Feldstein, Sylvan G. "How to Analyze FHA-Insured Mortgage Hospital Bonds." In The Handbook of Municipal Bonds, 1049–54. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781119198093.ch65.

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Courchane, Marsha, Rajeev Darolia, and Peter Zorn. "Fha Loans and Policy Responses to Credit Availability." In Lessons from the Financial Crisis, 155–61. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118266588.ch21.

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Zhan, Haoxi, and Xiaobing Pei. "FHA: Fast Heuristic Attack Against Graph Convolutional Networks." In Discovery Science, 151–65. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-88942-5_12.

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Cheng, Kui. "Effect of Mn on Protein Adsorption on Mn-TCP/FHA Coatings." In Key Engineering Materials, 881–84. Stafa: Trans Tech Publications Ltd., 2007. http://dx.doi.org/10.4028/0-87849-422-7.881.

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Cai, Xiao Xiao, Ping Gong, Yi Man, Zhi Qing Chen, and Gang He. "The Construction and Characterization of Nano-FHA Bioceramic Coating on Titanium Surface." In Key Engineering Materials, 333–36. Stafa: Trans Tech Publications Ltd., 2007. http://dx.doi.org/10.4028/0-87849-422-7.333.

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Cesarelli, Mario, M. Ruffo, M. Romano, P. Bifulco, F. Kovacs, and S. Iaccarino. "An algorithm for FHR extraction from FHS signals." In IFMBE Proceedings, 1374–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-89208-3_326.

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Verster, Joris C., Thomas M. Tzschentke, Kieran O’Malley, Francis C. Colpaert, Bart Ellenbroek, Bart Ellenbroek, R. Hamish McAllister-Williams, et al. "FAA." In Encyclopedia of Psychopharmacology, 531. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_597.

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Clemson, Lindy, J. Rick Turner, J. Rick Turner, Farrah Jacquez, Whitney Raglin, Gabriela Reed, Gabriela Reed, et al. "FHS." In Encyclopedia of Behavioral Medicine, 799. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100649.

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Ward, Tracey, Raphael Bernier, Cora Mukerji, Danielle Perszyk, James C. McPartland, Ellen Johnson, Susan Faja, et al. "FBA." In Encyclopedia of Autism Spectrum Disorders, 1253. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1698-3_100589.

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Conference papers on the topic "FHA"

1

Zhang, Meng, Chunsheng Cui, Shulin Liu, and Xiaojian Yi. "Reliability technology using FTA, FMECA, FHA and FRACAS: A review." In 2021 IEEE International Conference on Sensing, Diagnostics, Prognostics, and Control (SDPC). IEEE, 2021. http://dx.doi.org/10.1109/sdpc52933.2021.9563512.

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Allen, Christopher K., Andrew J. Goupee, Jeffrey Lindner, and Robert Berry. "Simulation of a Floating Offshore Wind Turbine With an Integrated Response Mitigation Technology." In ASME 2018 1st International Offshore Wind Technical Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/iowtc2018-1087.

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This work investigates the implementation of a novel, NASA-developed Fluid Harmonic Absorber (FHA) technology to mitigate platform motions and structural loads that can lead to lighter platforms, increased turbine performance, and ultimately, a lower LCOE. The novel damping strategy takes advantage of existing water ballast in the VolturnUS semi-submersible platform to achieve significant performance gains with minimal additional equipment and complexity. NREL’s FOWT software FAST is modified to include the primary features of the FHA technology. A study of the University of Maine-developed VolturnUS semi-submersible FOWT augmented with FHA technology is undertaken to quantify global performance of the system. When compared to the baseline technology, numerical simulations of a redesigned platform utilizing the FHA dampers indicate a reduction of 15.8% in hull structural material. Finally, the improvements in LCOE resulting from this mass reduction are assessed to demonstrate the advantages of NASA’s FHA technology for FOWT applications.

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Oeder, Christian, and Thomas Duerbaum. "ZVS investigation of llc converters based on FHA assumptions." In 2013 IEEE Applied Power Electronics Conference and Exposition - APEC 2013. IEEE, 2013. http://dx.doi.org/10.1109/apec.2013.6520669.

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Paviet-Hartmann, Patricia, Amber Wright, Edward Mausolf, Keri Campbell, and Frederic Poineau. "Application of Formohydroxamic Acid in Nuclear Processing: Synthesis and Complexation With Technetium-99." In 18th International Conference on Nuclear Engineering. ASMEDC, 2010. http://dx.doi.org/10.1115/icone18-29028.

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Acetohydroxamic acid (AHA) is an organic ligand planned for use in the Uranium Extraction (UREX) process. It reduces neptunium and plutonium, and the resultant hydrophilic complexes are separated from uranium by extraction with tributyl phosphate (TBP) in a hydrocarbon diluent. AHA undergoes hydrolysis to acetic acid which will impede the recycling of nitric acid. During recent discussions of the UREX process, it has been proposed to replace AHA by formohydroxamic acid (FHA). FHA will undergo hydrolysis to formic acid which is volatile, thus allowing the recycling of nitric acid. The reported reduction potentials of AHA and pertechnetate (TcO4−) indicated that it may be possible for AHA to reduce technetium, altering its fate in the fuel cycle. At UNLV, it has been demonstrated that TcO4− undergoes reductive nitrosylation by AHA under a variety of conditions. The resulting divalent technetium is complexed by AHA to form the pseudo-octahedral trans-aquonitrosyl (diacetohydroxamic)-technetium(II) complex ([TcII(NO)(AHA)2H2O]+). In this paper, we are reporting the synthesis of FHA and its complex formation with technetium along with the characterization of FHA crystals achieved by NMR and IR spectroscopy. Two experiments were conducted to investigate the complexation of FHA with Tc and the results were compared with previous data on AHA. The first experiment involved the elution of Tc from a Reillex HP anion exchange resin, and the second one monitored the complexation of technetium with FHA by UV-visible spectrophotometry.

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Onay, Murat. "A New and Fast Optimization Algorithm: Fox Hunting Algorithm (FHA)." In 2016 International Conference on Applied Mathematics, Simulation and Modelling. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/amsm-16.2016.35.

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Motte, Christian, Martin Niehuis, Sophie-Cathrin Fischer, Karel Stastny, and Peter Jeschke. "Functional Hazard Assessment of the Propulsion Control System Designed for a Small Hybrid Electric Aircraft." In GPPS Chania22. GPPS, 2022. http://dx.doi.org/10.33737/gpps22-tc-15.

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This paper presents the Functional Hazard Assessment (FHA) of a Propulsion Control System (PCS) designed for a hybrid electric aircraft considering the Failure Conditions (FCs) that are novel due to the electrification of the propulsion system. It will be worked out whether these FCs are covered by current certification regulations. After defining the functionality, the FCs for a hybrid electric propulsion system are established, based on current standards and also the Working Group 113 (WG-113) recommendations. The FHA and Preliminary System Safety Assessment (PSSA) approaches are used to evaluate and improve the architecture of the control system. The system functions on propulsion level are defined and allocated to different subsystems. Their FCs and effects are then established. Based on the applicable standards and recommendations of WG-113, these effects are classified, and the Safety Requirements (SRs) are then defined. The FHA results include 23 FCs related to the hybrid system, some of which are not addressed by the current regulatory framework. In summary, this paper outlines how electrified powertrain systems will affect new propulsion architectures from a safety perspective and how the current regulatory framework and the work of the WG-113 could be improved by addressing new FCs resulting from the FHA. By suggesting SRs derived from the FHA, this paper also discusses what system/safety activities need to be undertaken in order to design a propulsion architecture that is compliant with the applicable certification documents.

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Jain, Akshat, and Ivan Clemente Massimiani. "LCC Resonant Converter Design and Transfer Function Computation Using FHA Analysis." In 2021 4th Biennial International Conference on Nascent Technologies in Engineering (ICNTE). IEEE, 2021. http://dx.doi.org/10.1109/icnte51185.2021.9487672.

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Baimel, D., M. Mellincovsky, M. Sitbon, Y. Darhovsky, and A. Kuperman. "Modified FHA-based Diode Rectifier Representation for SN-Compensated Inductive WPT Links." In 2021 IEEE 19th International Power Electronics and Motion Control Conference (PEMC). IEEE, 2021. http://dx.doi.org/10.1109/pemc48073.2021.9432501.

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Huang, Hong. "FHA-based voltage gain function with harmonic compensation for LLC resonant converter." In 2010 IEEE Applied Power Electronics Conference and Exposition - APEC 2010. IEEE, 2010. http://dx.doi.org/10.1109/apec.2010.5433473.

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Zuo, Yu, Haowei Niu, Ruihong Zhang, and Xuewei Pan. "The Modified FHA and Simplified Time-doamin Analysis methodologies for LLC Resonant Converter." In 2021 IEEE 12th Energy Conversion Congress & Exposition - Asia (ECCE-Asia). IEEE, 2021. http://dx.doi.org/10.1109/ecce-asia49820.2021.9479161.

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Reports on the topic "FHA"

1

Aragon, Diego, Andrew Caplin, Sumit Chopra, John Leahy, Yann LeCun, Marco Scoffier, and Joseph Tracy. Reassessing FHA Risk. Cambridge, MA: National Bureau of Economic Research, March 2010. http://dx.doi.org/10.3386/w15802.

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Caplin, Andrew, Anna Cororaton, and Joseph Tracy. Is the FHA Creating Sustainable Homeownership? Cambridge, MA: National Bureau of Economic Research, June 2012. http://dx.doi.org/10.3386/w18190.

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3

Gyourko, Joseph, and Joseph Tracy. Unemloyment and Unobserved Credit Risk in the FHA Single Family Mortgage Insurance Fund. Cambridge, MA: National Bureau of Economic Research, March 2013. http://dx.doi.org/10.3386/w18880.

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4

Hendershott, Patric, and William Schultz. Equity and Nonequity Determinants of FHA Single-Family Mortgage Foreclosures in the 1980s. Cambridge, MA: National Bureau of Economic Research, August 1993. http://dx.doi.org/10.3386/w4440.

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Barilo, N. F. WHC-SD-W252-FHA-001, Rev. 0: Preliminary fire hazard analysis for Phase II Liquid Effluent Treatment and Disposal Facility, Project W-252. Office of Scientific and Technical Information (OSTI), May 1995. http://dx.doi.org/10.2172/83798.

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Turner, Paul, and John O'Brien. Review of the FSA’s research programme on food hypersensitivity. Food Standards Agency, June 2021. http://dx.doi.org/10.46756/sci.fsa.bka542.

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The overarching mission of the Food Standards Agency (FSA) is tothe ensure that food is safe, food is what it says it is and that consumers can make informed choices about what to eat. These are of central importance to consumers with food hypersensitivity(FHS).Food hypersensitivity (FHS) encompasses both immune-mediated food hypersensitivity (food allergy and coeliac disease) and non-immune food intolerances. FHS is a complex, multifactorial disease of concern to multiple stakeholders including consumers with FHS, their families, clinicians, regulatory agencies and policy makers, scientists, food manufacturers and food business operators. It affects around 5-8% of children and 2-3% of adults in the UK, and although rare, can be fatal. Public concern over FHS has grown in recent years. In the UK and elsewhere, food recalls due to the presence of undeclared allergens feature predominantly in food alerts; legislation over food labelling has become clearer, and consumers and producers are more aware of FHS. The FSA has been a major funder of research into FHS for over 2 decades, and the outputs of the research programme has had significant impacts at a national and global scale, most notably in the area of the prevention of FHS in children and the presence of declared and undeclared allergens in food products. Strengthening protections for consumers with FHS is a top priority for the FSA. The FSA has established a Food Hypersensitivity Programme Board to oversee and coordinate its work in this area. The working group was tasked with reviewing the research into FHS supported by the Food Standards Agency to date, and prioritising those priority areas where the current scientific evidence is limited and therefore should be a focus for future research investment. The aim –to make the UK the best country in the world for consumers with food hypersensitivity.

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Knibb, Rebecca, Lily Hawkins, and Dan Rigby. Food Sensitive Study: Wave Two Survey. Food Standards Agency, September 2022. http://dx.doi.org/10.46756/sci.fsa.nyx192.

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Food hypersensitivities (FH) include food allergy, food intolerance and coeliac disease. Food allergy and coeliac disease involve an immune mediated reaction to certain foods; food intolerance is caused by a non-immune mediated reaction (such as an enzymatic or pharmacological effect). Each of these FHs result in unpleasant symptoms if the food is eaten in sufficient quantity, with food allergic reactions sometimes resulting in life-threatening symptoms. Management of FH by an individual or members of their family therefore involves constant vigilance and risk assessment to determine if a food is safe to eat. Research over the last twenty years has demonstrated that this burden, along with the unpredictable nature of FH reactions, has an impact on quality of life (QoL). QoL encompasses our emotions, physical health, the environment we live in, our social networks and day-to-day activities. FH has been shown to have an impact on many of these areas, however there are still research gaps. In particular, many studies focus on children, adolescents or parents rather than the adult population and little is known about those with food intolerances. In order to make a comprehensive characterisation and evaluation of the burden caused by living with FH, the day-to-day management of FH and associated inconveniences, the FSA has commissioned this project, led by Aston University. The project is called the FoodSensitive study and this report relates to findings for workstream one, a survey to assess the impact of FH on QoL. This survey was carried out in two waves, one year apart. This report covers the second wave and a comparison of wave one and two for those participants who completed both waves.

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King, Lucy. FSA Consumer segmentation. Food Standards Agency, September 2021. http://dx.doi.org/10.46756/sci.fsa.bmo506.

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For our audiences, it is important to find out how their attitudes and behaviours relating to food safety differ, in order to understand who is more likely to take food safety risks and in what context. This is essential for effective communications and helps us to shape food safety policy. The audiences in these documents have been created using attitudinal and behavioural segmentation that categorises people based on their attitudes to food and their reported hygiene and food safety behaviours.

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Flanagan, George F., David Eugene Holcomb, and Sacit M. Cetiner. FHR Generic Design Criteria. Office of Scientific and Technical Information (OSTI), June 2012. http://dx.doi.org/10.2172/1045234.

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Flanagan, G. F., D. E. Holcomb, and S. M. Cetiner. FHR Generic Design Criteria. Office of Scientific and Technical Information (OSTI), June 2012. http://dx.doi.org/10.2172/1054143.

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