Relevant bibliographies by topics / Fibroblast activation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Fibroblast activation'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Fibroblast activation"

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Caporarello, Nunzia, Jeffrey A. Meridew, Dakota L. Jones та ін. "PGC1α repression in IPF fibroblasts drives a pathologic metabolic, secretory and fibrogenic state". Thorax 74, № 8 (2019): 749–60. http://dx.doi.org/10.1136/thoraxjnl-2019-213064.

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Idiopathic pulmonary fibrosis (IPF) is a fatal ageing-related disease linked to mitochondrial dysfunction. The present study aimed to determine whether peroxisome proliferator activated receptor gamma co-activator 1-alpha (PPARGC1A, encoding PGC1α), a master regulator of mitochondrial biogenesis, is diminished in IPF and controls pathologic fibroblast activation. Primary human IPF, control lung fibroblasts and fibroblasts sorted from bleomycin-injured mice were used to evaluate the expression and function of PGC1α. In vitro PGC1α manipulation was performed by small interfering RNA knockdown or
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Cai, Zhou, Hua Guo, Jing Qian та ін. "Effects of bone morphogenetic protein 4 on TGF-β1-induced cell proliferation, apoptosis, activation and differentiation in mouse lung fibroblasts via ERK/p38 MAPK signaling pathway". PeerJ 10 (27 липня 2022): e13775. http://dx.doi.org/10.7717/peerj.13775.

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Fibroblasts, in particular myofibroblasts, are the critical effector cells in idiopathic pulmonary fibrosis (IPF), a deadly lung disease characterized by abnormal lung remodeling and the formation of “fibroblastic foci”. Aberrant activation of TGF-β1 is frequently encountered and promotes fibroblast proliferation, activation, and differentiation in pulmonary fibrosis. Hence, the inhibition of TGF-β1-induced lung fibroblast activation holds promise as a therapeutic strategy for IPF. The present study aimed to investigate the potential effect and underlying mechanisms of bone morphogenetic prote
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Xin, Yanguo, Wenchao Wu, Jing Qu, et al. "Inhibition of Mitofusin-2 Promotes Cardiac Fibroblast Activation via the PERK/ATF4 Pathway and Reactive Oxygen Species." Oxidative Medicine and Cellular Longevity 2019 (April 16, 2019): 1–16. http://dx.doi.org/10.1155/2019/3649808.

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Mitofusin-2 (Mfn2) is a key outer mitochondrial membrane protein, which maintains normal mitochondrial dynamics and function. However, its role in cardiac fibroblast activation remains poorly understood. In the present study, a rat model of transverse aortic constriction (TAC) was established to observe the cardiac fibroblast activation in vivo. TGF-β1 treatment for 24 hours was used to induce cardiac fibroblast activation in vitro. As a result, the expression of Mfn2 decreased in the hypertrophic heart tissues and cardiac fibroblasts treated with TGF-β1. siMfn2 and adenovirus were applied to
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Liu, Fei, David Lagares, Kyoung Moo Choi, et al. "Mechanosignaling through YAP and TAZ drives fibroblast activation and fibrosis." American Journal of Physiology-Lung Cellular and Molecular Physiology 308, no. 4 (2015): L344—L357. http://dx.doi.org/10.1152/ajplung.00300.2014.

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Pathological fibrosis is driven by a feedback loop in which the fibrotic extracellular matrix is both a cause and consequence of fibroblast activation. However, the molecular mechanisms underlying this process remain poorly understood. Here we identify yes-associated protein (YAP) (homolog of drosophila Yki) and transcriptional coactivator with PDZ-binding motif (TAZ) (also known as Wwtr1), transcriptional effectors of the Hippo pathway, as key matrix stiffness-regulated coordinators of fibroblast activation and matrix synthesis. YAP and TAZ are prominently expressed in fibrotic but not health
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Heng, Madalene. "Phosphorylase Kinase Inhibition Therapy in Burns and Scalds." BioDiscovery 20 (February 24, 2017): e11207. https://doi.org/10.3897/biodiscovery.20.e11207.

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Severe burns and scalds almost always result in unsightly hypertrophic scarring. Among the important processes involved in scarring are fibroblast formation and transformation of fibroblasts into myofibroblasts. Myofibroblasts contain α-smooth muscle actin which has contractile properties and can lead to wound contraction and hypertrophic scarring. Phosphorylase kinase (PhK), expressed within 5 mins of injury, is among the earliest enzymes released after tissue damage. It is responsible for activation of NF-kB, which in turn activates over 200 different genes related to inflammation, fibroblas
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Aravamudhan, Aja, Andrew J. Haak, Kyoung Moo Choi, et al. "TBK1 regulates YAP/TAZ and fibrogenic fibroblast activation." American Journal of Physiology-Lung Cellular and Molecular Physiology 318, no. 5 (2020): L852—L863. http://dx.doi.org/10.1152/ajplung.00324.2019.

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Idiopathic pulmonary fibrosis (IPF) results in scarring of the lungs by excessive extracellular matrix (ECM) production. Resident fibroblasts are the major cell type involved in ECM deposition. The biochemical pathways that facilitate pathological fibroblast activation leading to aberrant ECM deposition are not fully understood. Tank binding protein kinase-1 (TBK1) is a kinase that regulates multiple signaling pathways and was recently identified as a candidate regulator of fibroblast activation in a large-scale small-interfering RNA (siRNA) screen. To determine the effect of TBK1 on fibroblas
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Tang, Rui, Yung-Chun Wang, Xiaohan Mei, et al. "LncRNA GAS5 attenuates fibroblast activation through inhibiting Smad3 signaling." American Journal of Physiology-Cell Physiology 319, no. 1 (2020): C105—C115. http://dx.doi.org/10.1152/ajpcell.00059.2020.

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Transforming growth factor-β (TGF-β)-induced fibroblast activation is a key pathological event during tissue fibrosis. Long noncoding RNA (lncRNA) is a class of versatile gene regulators participating in various cellular and molecular processes. However, the function of lncRNA in fibroblast activation is still poorly understood. In this study, we identified growth arrest-specific transcript 5 (GAS5) as a novel regulator for TGF-β-induced fibroblast activation. GAS5 expression was downregulated in cultured fibroblasts by TGF-β and in resident fibroblasts from bleomycin-treated skin tissues. Ove
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Stauffer, Winston T., Erik A. Blackwood, Khalid Azizi, Randal J. Kaufman, and Christopher C. Glembotski. "The ER Unfolded Protein Response Effector, ATF6, Reduces Cardiac Fibrosis and Decreases Activation of Cardiac Fibroblasts." International Journal of Molecular Sciences 21, no. 4 (2020): 1373. http://dx.doi.org/10.3390/ijms21041373.

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Activating transcription factor-6 α (ATF6) is one of the three main sensors and effectors of the endoplasmic reticulum (ER) stress response and, as such, it is critical for protecting the heart and other tissues from a variety of environmental insults and disease states. In the heart, ATF6 has been shown to protect cardiac myocytes. However, its roles in other cell types in the heart are unknown. Here we show that ATF6 decreases the activation of cardiac fibroblasts in response to the cytokine, transforming growth factor β (TGFβ), which can induce fibroblast trans-differentiation into a myofib
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Margulis, Alexander, Karl H. Nocka, Nancy L. Wood, Stanley F. Wolf, Samuel J. Goldman, and Marion T. Kasaian. "MMP dependence of fibroblast contraction and collagen production induced by human mast cell activation in a three-dimensional collagen lattice." American Journal of Physiology-Lung Cellular and Molecular Physiology 296, no. 2 (2009): L236—L247. http://dx.doi.org/10.1152/ajplung.90462.2008.

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Mast cell-fibroblast interactions may contribute to fibrosis in asthma and other disease states. Fibroblast contraction is known to be stimulated by coculture with the human mast cell line, HMC-1, or by mast cell-derived agents. Matrix metalloproteinases (MMPs) can also mediate contraction, but the MMP-dependence of mast cell-induced fibroblast contractility is not established, and the consequences of mast cell activation within the coculture system have not been fully explored. We demonstrate that activation of primary human mast cells (pHMC) with IgE receptor cross-linking, or activation of
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Cheng, Maye F., Faizah S. Abdullah, and Matthew B. Buechler. "Essential growth factor receptors for fibroblast homeostasis and activation." F1000Research 13 (February 19, 2024): 120. http://dx.doi.org/10.12688/f1000research.143514.1.

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Fibroblasts are cells of mesenchymal origin that are found throughout the body. While these cells have several functions, their integral roles include maintaining tissue architecture through the production of key extracellular matrix components, and participation in wound healing after injury. Fibroblasts are also key mediators in disease progression during fibrosis, cancer, and other inflammatory diseases. Under these perturbed states, fibroblasts can activate into inflammatory fibroblasts or contractile myofibroblasts. Fibroblasts require various growth factors and mitogenic molecules for su
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Dissertations / Theses on the topic "Fibroblast activation"

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Wang, Xin Maggie. "Fibroblast activation protein in cell biology and liver fibrosis." Thesis, The University of Sydney, 2007. https://hdl.handle.net/2123/28106.

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Fibroblast activation protein (FAP) is a type 11 cell surface protein of the dipeptidyl peptidase IV (DPIV) gene family. FAP has two peptidase activities, DP activity and a recently reported narrow prolyl endopeptidase activity, which is restricted to Gly-Pro derived substrates. The only known natural substrate of FAP is a collagen type I (CN-I) specific gelatinase activity. In contrast, as the closest relative of PAP, DPIV has only DP activity. Nine chemokines, including CXCL12, are DPIV substrates and DPIV binds to fibronectin (FN). The natural ligands of FAP are unknown. FAP is not de
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Oers, Johanna Maria Margareta van. "Activation of the fibroblast growth factor receptor 3 in bladder cancer." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2007. http://hdl.handle.net/1765/10475.

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Geitgey, Delaney Kate. "Evaluating the role of fibroblast activation protein and fibroblast growth factor 21 in growth hormone-induced adipose tissue fibrosis." Ohio University Honors Tutorial College / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ouhonors1587596581428154.

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Dolivo, David. "Fibroblast activation and pro-fibrotic phenotypes: modulation by FGF2 and MAPK signaling." Digital WPI, 2018. https://digitalcommons.wpi.edu/etd-dissertations/477.

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Fibrotic diseases are a leading cause of morbidity and mortality in the developed world. Despite this, the lack of therapies for fibrotic pathological disease states is severe. A large part of the reason for this lack of viable therapies is due to an incomplete understanding of the early processes driving tissue fibrosis, as well as the dismal results of pharmacologic monotherapies at the clinical trial stage in humans thus far. Therefore, better understanding of the upstream mechanisms driving tissue fibrosis is imperative. One of the common mechanisms underlying all fibroses is the presence
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Kraman, Matthew. "Suppression of intitumor immunity by stromal cells expressing fibroblast activation protein-alpha." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609547.

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Aguilera, Rojas Matias Ignacio [Verfasser]. "Cellular crosstalk between canine fibroblasts and a mast cell tumour cell line and its significance in fibroblast activation / Matias Ignacio Aguilera Rojas." Berlin : Freie Universität Berlin, 2020. http://d-nb.info/1218530553/34.

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Shamhart, Patricia E. "The impact of the extracellular matrix and type 1 diabetes on cardiac fibroblast activation." Kent State University / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=kent1283881511.

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Hamson, Elizabeth. "Functional Characterisation and Outcomes of Post Translational Modifications Driven by Fibroblast Activation Protein Enzyme Activity." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/14891.

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Fibroblast Activation Protein (FAP) is a cellXsurface anchored dimeric protease, closely related to Dipeptidyl Peptidase (DPP) 4. This atypical serine protease has both dipeptidyl peptidase and endopeptidase activities, cleaving substrates at a postXproline bond. FAP expression is difficult to detect in nonXdiseased adult organs, but is greatly up regulated in sites of tissue remodelling, which includes liver fibrosis, lung fibrosis, atherosclerosis, arthritis, tumours and embryonic tissues. Due to its restricted expression pattern and dual enzymatic activities, FAP is emerging as a unique the
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Millul, Jacopo. "Drug Conjugates Specific to Fibroblast Activation Protein and Carbonic Anhydrase IX for Applications in Oncology." Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1193822.

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Despite the different therapeutic options available, more than 10 million deaths every year are caused by cancer, thus representing one of the majors’ causes of death worldwide. Lung cancer (1.8 million), breast cancer (700’000), colorectal cancer (950’000), and liver cancer (830’000) represent half of the global cancer incidence. Common approaches for the treatment of cancer include surgery, radiotherapy (external beam radiation and Radioligand Therapy, RLT), chemotherapy and immunotherapy. While radiotherapy indistinctively targets all cells within the tumor environment, chemotherapeutic
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胡, 興柏. "ACTIVATION OF FIBROBLAST-DERIVED MATRIX METALLOPROTEINASE-2 BY COLON CANCER CELLS IN NON-CONTACT CO-CULTURES." Doctoral thesis, Kyoto University, 2000. http://hdl.handle.net/2433/151419.

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京都大学<br>0048<br>新制・課程博士<br>博士(医学)<br>甲第8550号<br>医博第2272号<br>新制||医||747(附属図書館)<br>UT51-2000-M14<br>京都大学大学院医学研究科内科系専攻<br>(主査)教授 鍋島 陽一, 教授 月田 承一郎, 教授 千葉 勉<br>学位規則第4条第1項該当<br>Doctor of Medical Science<br>Kyoto University<br>DAM
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Books on the topic "Fibroblast activation"

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Guo, Chun. Divergent regulation of MMP-2 secretion and activation in adult rat cardiac fibroblasts. De Montfort University, 2002.

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Pulkki, Kari. Activation of synovial fibroblasts by macrophage-derived factors: Characterization of arthritis-associated and cytokine-induced biochemical and morphological changes in cultured synovial fibroblasts. K. Pulkki, 1988.

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Giesel, Frederik L., and Rodney Hicks. Fibroblast Activation Protein Imaging, an Issue of PET Clinics. Elsevier, 2023.

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Grimbacher, Bodo. Activation of early growth response transcripts and cytokines in rheumatoid synovial fibroblasts. 1994.

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Pitzalis, Costantino, Frances Humby, and Michael P. Seed. Synovial pathology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or
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Denton, Christopher P., and Pia Moinzadeh. Systemic sclerosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0121.

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The term 'scleroderma' describes a group of conditions in which the development of thickened, fibrotic skin is a cardinal feature. This includes localized forms of scleroderma (e.g. morphoea) and also systemic forms of the disease that are more correctly termed systemic sclerosis. Systemic sclerosis (SSc) is a multiorgan, autoimmune disease that has a high clinical burden and mortality, due to affecting the skin as well as internal organs. As with other related diseases there is a female predominance and marked clinical diversity. The pathogenesis of SSc is not fully elucidated; it includes en
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Vantieghem, Katleen. Photoproduction of Vitamin D3 & Activation into 1a, 25-dihydroxyvitamin D3 in Human Epidermal Keratinocytes, Dermal Fibroblasts & Other Cells. Leuven Univ Pr, 2006.

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Book chapters on the topic "Fibroblast activation"

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Saxena, Amit, and Nikolaos G. Frangogiannis. "Fibroblast Activation in the Infarcted Myocardium." In Cardiac Fibrosis and Heart Failure: Cause or Effect? Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17437-2_2.

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Neamtu, M., C. E. Cotrutz, A. Filioreanu, T. Petreus, and A. Neamtu. "Molecular Dynamics Simulation of Fibroblast Growth Factors Receptors Activation." In IFMBE Proceedings. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-04292-8_69.

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Davis, Terence. "Progeroid Syndromes: Role of Accelerated Fibroblast Senescence and p38 Activation." In Tumor Dormancy, Quiescence, and Senescence, Vol. 3. Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-9325-4_3.

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Mousavi, Mohammad Javad, Jafar Karami, Masoumeh Alimohammadi, Farid Solaymani-Mohammadi, and Nima Rezaei. "Fibroblast Activation Protein (FAP): A Key Modulator of the Cancer Microenvironment." In Handbook of Cancer and Immunology. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-030-80962-1_357-1.

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Chen, Hai-Lan, Philip S. Rudland, John A. Smith, and David G. Fernig. "Activation of Basic Fibroblast Growth Factor (bFGF) by Heparan Sulphate (HS)." In Intercellular Signalling in the Mammary Gland. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1973-7_10.

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Pellegrini, L., D. F. Burke, and T. L. Blundell. "Activation Mechanism of Fibroblast Growth Factor Receptor Tyrosine Kinase Revealed by Crystal Structure of Fibroblast Growth Factor Receptor Ectodomain Bound to Fibroblast Growth Factor and Heparin." In Insulin & Related Proteins - Structure to Function and Pharmacology. Springer Netherlands, 2002. http://dx.doi.org/10.1007/0-306-47582-0_15.

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Jaye, Michael, Gregg Crumley, and Joseph Schlessinger. "Analysis and Transfection of Human Acidic Fibroblast Growth Factor (aFGF) cDNA Clones and Structural Analysis of the Human aFGF Gene reveals Features Shared within a Family of Homologous Proteins." In Activation of Hormone and Growth Factor Receptors. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1936-5_5.

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Kawano, Daniel F., Carlos H. T. de Paula da Silva, and Carlton A. Taft. "Design of Inhibitors of the Human Fibroblast Activation Protein α as a Strategy to Hinder Metastasis and Angiogenesis." In Emerging Research in Science and Engineering Based on Advanced Experimental and Computational Strategies. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-31403-3_11.

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Keane, F. M., S. Chowdhury, T. W. Yao, et al. "Chapter 5. Targeting Dipeptidyl Peptidase-4 (DPP-4) and Fibroblast Activation Protein (FAP) for Diabetes and Cancer Therapy." In Drug Discovery. Royal Society of Chemistry, 2011. http://dx.doi.org/10.1039/9781849733151-00118.

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Riemann, Dagmar, Jana Röntsch, Bettina Hause, Jürgen Langner, and Astrid Kehlen. "Cell-Cell Contact Between Lymphocytes and Fibroblast-Like Synovioctyes Induces Lymphocytic Expression of Aminopeptidase n/cd13 and Results in Lymphocytic Activation." In Cellular Peptidases in Immune Functions and Diseases 2. Springer US, 2002. http://dx.doi.org/10.1007/0-306-46826-3_6.

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Conference papers on the topic "Fibroblast activation"

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Abe, Shuichi, Hiroyasu Okazaki, Masami Kishi, et al. "Fibrocyte Regulates Lung Fibroblast Activation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4460.

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Aravamudhan, A., A. Haak, G. Ligresti, et al. "Targeting TBK1 to Reduce Fibroblast Activation." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5351.

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Perl, A. K. T., J. Green, and K. Wagner. "Diabetes Mellitus Impairs Regenerative Fibroblast Activation." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6649.

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Schwarz, Johannes, and Oliver Eickelberg. "Mitochondrial Fusion Processes Modulate Lung Fibroblast Activation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1926.

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Van Deventer, James A., and Karl D. Wittrup. "Abstract 2138: Targeting cancer-associated fibroblasts using antibodies recognizing fibroblast activation protein-alpha." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2138.

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Jin, Jiefu, James D. Barnett, Balaji Krishnamachary, Yelena Mironchik, Hisataka Kobayashi та Zaver Bhujwalla. "Abstract 3360: Phototheranostics of cancer associated fibroblasts by targeting fibroblast activation protein-α". У Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3360.

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Zhou, J., and Y. Y. Sanders. "Targeting Repressive Histone H3K27me3 Medicated Lung Fibroblast Activation." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a2200.

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Hurley, Jennifer R., and Daria A. Narmoneva. "Endothelial-Fibroblast Interactions in Angiogenesis and Matrix Remodeling." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206534.

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Revascularization is critical for successful regeneration of ischemic cardiac tissue after injury. To achieve revascularization in engineered cardiac grafts, it is necessary to understand the interactions between major cardiac cell types. The importance of cardiomyocyte-endothelial interactions in angiogenesis is well documented [1]; however, less is known about interactions between endothelial and stromal cells, fibroblasts in particular. Studies indicate that during capillary assembly, fibroblasts (FBs) provide chemical signaling via growth factor expression and endothelial activation and pr
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Liu, Fei, Thomas H. Sisson, Jeffrey C. Horowitz, and Daniel J. Tschumperlin. "Matrix Stiffness Regulates Fibroblast Activation Through PAI-1 Expression." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1033.

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Milara, Javier, Adela Serrano, Teresa Peiró, et al. "Aclidinium Partially Prevents Human Lung Fibroblast Activation In Vitro." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3587.

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Reports on the topic "Fibroblast activation"

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Techakumphu, Mongkol, Pranee Numchaisrika, Ratajuk Rungsiwiwat, Somchai Suwajanakorn, Kamtorn Pruksananonda, and Pramuan Virutamasen. Development of somatic cell nuclear transfer techique for biomedical and agricultural research : report. Chulalongkorn University, 2004. https://doi.org/10.58837/chula.res.2004.85.

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To develop somatic cell nuclear transfer by using the rabbit as a mode in biomedical and agricultural research. The experiments are described in nine chapters: Chapter 1: Introduction, Chapter 2: Superovulation and oocyte recipient production in rabbits, Chapter 3: A technique for adult and foetal fibroblast cell culture, for donor cell preparation in somatic nuclear transfer, Chapter 4: Preliminary studies on somatic cell nuclear transfer in rabbits, Chapter 5: In vitro development of rabbit cloned embryos using adult fibroblast cells, after post-activation treatment with cyelohexamide and 6-
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Kelly, Jr, Simms Thomas J., Huang Avis E., Mazur Yan, and Anna. Fibroblast Activation Protein-Alpha, a Serine Protease that Facilitates Metastasis by Modification of Diverse Microenvironments. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada588423.

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Brennen, Nathaniel. Evaluation of Fibroblast Activation Protein-Alpha (FAP) as a Diagnostic Marker and Therapeutic Target in Prostate Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada481625.

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P. Hoven, Voravee, Kohn, Joachim, and Adisorn Poopattanapong. Immobilization of RGD Peptides on surface of Tyrosine-Derived Polycarbonate to enhance cell adhesion and proliferation. Chulalongkorn University, 2006. https://doi.org/10.58837/chula.res.2006.35.

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This research has focused on chemical immobilization of RGD-containing peptides (RGD, RGDS, GRGDS) on the surface of tyrosine-derived polycarbonates having carboxyl pendant groups, poly(DTE-co-20%DT carbonate) through a two-step reaction. The first step involved an activation of carboxyl groups by N-hydroxysuccinimide (NHS) in the presence of 1-(3-dimethylaminopropyl)-3-ethylcarbodiimide hydrochloride (EDCI). The second step was a covalent attachment of RGD-containing peptides. The success of peptides. The success of peptide immobilization was determined by the ninhydrin method and x-ray photo
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