Relevant bibliographies by topics / Fibroblast growth factors

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Fibroblast growth factors'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2024

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Journal articles on the topic "Fibroblast growth factors"

1

Thomas, Kenneth A. "Fibroblast growth factors." FASEB Journal 1, no. 6 (1987): 434–40. http://dx.doi.org/10.1096/fasebj.1.6.3315806.

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Baird, Andrew, and Patricia A. Walicke. "Fibroblast growth factors." British Medical Bulletin 45, no. 2 (1989): 438–52. http://dx.doi.org/10.1093/oxfordjournals.bmb.a072333.

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Mason, Ivor. "Fibroblast growth factors." Current Biology 13, no. 9 (2003): R346. http://dx.doi.org/10.1016/s0960-9822(03)00270-7.

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Cheng, Maye F., Faizah S. Abdullah та Matthew B. Buechler. "Essential growth factor receptors for fibroblast homeostasis and activation: Fibroblast Growth Factor Receptor (FGFR), Platelet Derived Growth Factor Receptor (PDGFR), and Transforming Growth Factor β Receptor (TGFβR)". F1000Research 13 (21 травня 2024): 120. http://dx.doi.org/10.12688/f1000research.143514.2.

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Fibroblasts are cells of mesenchymal origin that are found throughout the body. While these cells have several functions, their integral roles include maintaining tissue architecture through the production of key extracellular matrix components, and participation in wound healing after injury. Fibroblasts are also key mediators in disease progression during fibrosis, cancer, and other inflammatory diseases. Under these perturbed states, fibroblasts can activate into inflammatory fibroblasts or contractile myofibroblasts. Fibroblasts require various growth factors and mitogenic molecules for su
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Cheng, Maye F., Faizah S. Abdullah, and Matthew B. Buechler. "Essential growth factor receptors for fibroblast homeostasis and activation." F1000Research 13 (February 19, 2024): 120. http://dx.doi.org/10.12688/f1000research.143514.1.

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Fibroblasts are cells of mesenchymal origin that are found throughout the body. While these cells have several functions, their integral roles include maintaining tissue architecture through the production of key extracellular matrix components, and participation in wound healing after injury. Fibroblasts are also key mediators in disease progression during fibrosis, cancer, and other inflammatory diseases. Under these perturbed states, fibroblasts can activate into inflammatory fibroblasts or contractile myofibroblasts. Fibroblasts require various growth factors and mitogenic molecules for su
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Chen, Gregory, and Reza Forough. "Fibroblast Growth Factors, Fibroblast Growth Factor Receptors, Diseases, and Drugs." Recent Patents on Cardiovascular Drug Discovery 1, no. 2 (2006): 211–24. http://dx.doi.org/10.2174/157489006777442478.

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Onoue, H., Y. Ebi, H. Nakayama, XM Ru, Y. Kitamura, and J. Fujita. "Suppressive effect of Sl/Sld mouse embryo-derived fibroblast cell lines on diffusible factor-dependent proliferation of mast cells." Blood 74, no. 5 (1989): 1557–62. http://dx.doi.org/10.1182/blood.v74.5.1557.1557.

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Abstract Two modes of mast cell growth are present, one dependent on diffusible growth factors (interleukins [IL] 3 and 4) and another dependent on contact with fibroblasts. The 3T3 fibroblast cell lines derived from WCB6F1-+/+ mouse embryos supported the proliferation of cultured mast cells (CMC), whereas the 3T3 fibroblast cell lines from WCB6F1-Sl/Sld mouse embryos did not. To investigate the relationship between growth factor-dependent and fibroblast-dependent growths of mast cells, we cocultured CMC and 3T3 fibroblasts in the presence of diffusible growth factors. WCB6F1-+/+ mouse embryo-
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Onoue, H., Y. Ebi, H. Nakayama, XM Ru, Y. Kitamura, and J. Fujita. "Suppressive effect of Sl/Sld mouse embryo-derived fibroblast cell lines on diffusible factor-dependent proliferation of mast cells." Blood 74, no. 5 (1989): 1557–62. http://dx.doi.org/10.1182/blood.v74.5.1557.bloodjournal7451557.

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Two modes of mast cell growth are present, one dependent on diffusible growth factors (interleukins [IL] 3 and 4) and another dependent on contact with fibroblasts. The 3T3 fibroblast cell lines derived from WCB6F1-+/+ mouse embryos supported the proliferation of cultured mast cells (CMC), whereas the 3T3 fibroblast cell lines from WCB6F1-Sl/Sld mouse embryos did not. To investigate the relationship between growth factor-dependent and fibroblast-dependent growths of mast cells, we cocultured CMC and 3T3 fibroblasts in the presence of diffusible growth factors. WCB6F1-+/+ mouse embryo-derived 3
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Metzler, Veronika Maria, Christian Pritz, Anna Riml, et al. "Separation of cell survival, growth, migration, and mesenchymal transdifferentiation effects of fibroblast secretome on tumor cells of head and neck squamous cell carcinoma." Tumor Biology 39, no. 11 (2017): 101042831770550. http://dx.doi.org/10.1177/1010428317705507.

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Fibroblasts play a central role in tumor invasion, recurrence, and metastasis in head and neck squamous cell carcinoma. The aim of this study was to investigate the influence of tumor cell self-produced factors and paracrine fibroblast–secreted factors in comparison to indirect co-culture on cancer cell survival, growth, migration, and epithelial–mesenchymal transition using the cell lines SCC-25 and human gingival fibroblasts. Thereby, we particularly focused on the participation of the fibroblast-secreted transforming growth factor beta-1.Tumor cell self-produced factors were sufficient to e
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COUTTS, JACQUELINE C., and JOHN T. GALLAGHER. "Receptors for fibroblast growth factors." Immunology and Cell Biology 73, no. 6 (1995): 584–89. http://dx.doi.org/10.1038/icb.1995.92.

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Dissertations / Theses on the topic "Fibroblast growth factors"

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Patel, Ambreen. "Fibroblast growth factors and retinal cell genesis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0025/MQ48033.pdf.

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Detvisitsakun, Chanitchote. "Functional characterization of a Baculovirus fibroblast growth factor." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/239.

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Harmer, N. J. "Structural studies of fibroblast growth factors and their receptors." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603723.

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Recent crystal structures have suggested two models for the complex between FGFs, FGF receptors (FGFRs) and the proteoglycan heparin that mediates signalling, and have provided insight into how FGFs show differing affinities for the range of FGFRs. I have examined complexes of FGF, FGFR and heparin by size-exclusion chromatography, analytical ultracentrifugation and mass spectrometry. This analysis suggests that both of the crystal structures faithfully represent the state of the molecules in solution. From this, I conclude that the origin of the difference in the two models lies in the prepar
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Ding, Su Sin. "Fibroblast growth factors in gastrointestinal development, homeostasis and injury." Thesis, Imperial College London, 2009. http://hdl.handle.net/10044/1/5497.

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Fibroblast growth factors (FGFs) and their receptors (FGFRs) are essential for controlling cell growth and proliferation, angiogenesis, wound healing and tumourigenesis. In mammals, there are twenty-three FGFs and five FGFRs, with each FGFR having different ligand binding specificities. FGFs are thought to act in a paracrine manner, in which they are secreted by one cell and activate FGFRs on another cell type, and such interaction helps to establish the fundamental crosstalk between epithelium and mesenchyme during development, homeostasis and tumourigenesis. This thesis aims to investigate t
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Moenner, Michel. "Étude du mécanisme d'action des facteurs de croissance "Fibroblast Growth Factors" (FGF)." Paris 12, 1988. http://www.theses.fr/1988PA120010.

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Pour les deux types cellulaires etudies, il apparait que le signal mitogene induit par la formation du complexe fgf-recepteurs est independant de l'activation du cycle des polyphosphoinositides et de l'activation de la proteine kinase c
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Moenner, Michel. "Etude du mécanisme d'action des facteurs de croissance "fibroblast growth factors", FGF." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb376165817.

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Scarpa, Richard C. "Neurotensin potentiates the proliferative effects of growth factors in human embryonic lung fibroblasts /." Thesis, Connect to Dissertations & Theses @ Tufts University, 2004.

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Thesis (Ph.D.)--Tufts University, 2004.<br>Adviser: David E. Cochrane. Submitted to the Dept. of Biology. Includes bibliographical references (leaves 137-165). Access restricted to members of the Tufts University community. Also available via the World Wide Web;
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Abud, Helen E. "Examination of methods for the study of FGFs during mouse development." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260765.

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Vidyasagar, Rishma. "Characterisation of a suitable surface for the study of FGF : oligosaccharide interactions." Thesis, University of Liverpool, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288274.

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Yeoh, Joyce Siew Gaik. "Regulatory role of fibroblast growth factors on hematopoietic stem cells." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/299000842.

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Books on the topic "Fibroblast growth factors"

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Baird, Andrew, 1954 July 27-, Klagsbrun Michael, and New York Academy of Sciences., eds. The Fibroblast growth factor family. New York Academy of Sciences, 1991.

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Pedro, Cuevas, ed. Fibroblast growth factor in the cardiovascular system. I. Holzapfel, 2002.

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Mullen, Elizabeth Anne. An inhibitor of angiogenesis isolated from bovine cornea. s.n.], 1992.

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Drucker, Beverly Joyce. Functional and developmental studies on members of the fibroblast growth factor family. [Columbia University], 1993.

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Dalton, Stephen Lewis. Cell attachment controls fibronectin and α51 integrin levels in fibroblasts: Implications for anchorage-dependent and -independent growth. [Columbia University], 1993.

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Westman, Jacob. Synthesis of oligosaccharides related to heparin and heparan sulphate and their binding to fibroblast growth factors. Dept. of Organic Chemistry, Arrhenius Laboratory, Stockholm University, 1995.

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1945-, Lippman Marc E., Ortho Pharmaceutical Corporation, and University of California, Los Angeles., eds. Growth regulation of cancer: Proceedings of an Ortho-UCLA Symposium on Growth Regulation of Cancer, held at Park City, Utah, January 17-23, 1987. Liss, 1988.

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Kjell, Fuxe, and Wenner-Grenska samfundet, eds. Trophic regulation of the basal ganglia: Focus on dopamine neurons. Pergamon, 1994.

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1945-, Lippman Marc E., Dickson Robert B. 1952-, and University of California, Los Angeles., eds. Growth regulation of cancer II: Proceedings of a UCLA symposium, held at Keystone, Colorado, January 21-27, 1989. Wiley-Liss, 1990.

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Walters, Jean Elizabeth. Immunochemical studies on fibroblast growth factor-1 and fibroblast growth factor receptor 1. Oxford Brookes University, 1998.

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Book chapters on the topic "Fibroblast growth factors"

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Baird, A., and P. Böhlen. "Fibroblast Growth Factors." In Peptide Growth Factors and Their Receptors I. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-49295-2_7.

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Cronauer, Marcus V., and Wolfgang A. Schulz. "Fibroblast Growth Factors." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_2175-2.

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Baird, A., and P. Böhlen. "Fibroblast Growth Factors." In Peptide Growth Factors and Their Receptors I. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4612-3210-0_7.

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Cronauer, Marcus V., and Wolfgang A. Schulz. "Fibroblast Growth Factors." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_2175.

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Cronauer, Marcus V., and Wolfgang A. Schulz. "Fibroblast Growth Factors." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2175.

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Chiodelli, Paola, and Marco Presta. "Fibroblast Growth Factors." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_5710-1.

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Chiodelli, Paola, and Marco Presta. "Fibroblast Growth Factors." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57401-7_5710.

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Moscatelli, David. "Fibroblast Growth Factors." In Cytokines of the Lung. CRC Press, 2022. http://dx.doi.org/10.1201/9781003066927-3.

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Ornitz, David M., and Gabriel Waksman. "Fibroblast Growth Factor Receptors." In Growth Factors and Wound Healing. Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-1876-0_9.

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Burgess, Wilson H. "Structure-Function Analysis of Fibroblast Growth Factor-1 (Acidic Fibroblast Growth Factor)." In Growth Factors, Peptides and Receptors. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2846-3_28.

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Conference papers on the topic "Fibroblast growth factors"

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Hurley, Jennifer R., and Daria A. Narmoneva. "Fibroblasts Induce Mechanical Changes in the Extracellular Environment and Enhance Capillary-Like Network Formation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193093.

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Cardiac tissue engineering studies have demonstrated the importance of revascularization in engineered grafts for successful implantation and regeneration [1]. Understanding the myocardium’s complex cellular organization and the interactions between the major cardiac cell types (cardiomyocytes, endothelial cells, and cardiac fibroblasts) is critical for revascularization. Our previous studies have shown the importance of cardiomyocyte-endothelial interactions [2]. However, there is limited information available on endothelial-fibroblast interactions. We and others have previously observed that
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Kwon, Youngjoo, and Geun Yeong Park. "Abstract 5088: Fibroblast-derived factors enhance growth of ovarian cancer cells." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5088.

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Seitz, T., K. Freese, WE Thasler, and C. Hellerbrand. "Expression of paracrine fibroblast growth factors in hepatic stellate cells and hepatic fibrosis." In 37. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0040-1721957.

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Woo, K. V., C. Weinheimer, J. Nigro, and D. M. Ornitz. "Protective Role of Fibroblast Growth Factors in Group 3 Hypoxia Induced Pulmonary Hypertension." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4033.

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Singh, Ankur, Shalu Suri, Ted T. Lee, et al. "Adhesive Signature-Based, Label-Free Isolation of Human Pluripotent Stem Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80044.

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Generation of human induced pluripotent stem cells (hiPSCs) from fibroblasts and other somatic cells represents a highly promising strategy to produce auto- and allo-genic cell sources for therapeutic approaches as well as novel models of human development and disease1. Reprogramming protocols involve transduction of the Yamanaka factors Oct3/4, Sox2, Klf4, and c-Myc into the parental somatic cells, followed by culturing the transduced cells on mouse embryonic fibroblast (MEF) or human fibroblast feeder layers, and subsequent mechanical dissociation of pluripotent cell-like colonies for propag
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Rieke, Damian T., Zhixiang Zuo, Katharina Endhardt, et al. "Abstract 2832: Fibroblast growth factors in head and neck cancer: Genetic alterations and therapeutic targeting with ponatinib." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2832.

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Epstein Shochet, Gali, Becky Bardenstein-Wald, Elizabetha Brook, and David Shitrit. "Transforming growth factor beta (TGF-ß) pathway activation by IPF fibroblast-derived soluble factors is mediated by IL-6 trans-signaling." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3352.

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Guzy, R., B. Ansbro, E. Reed, and N. O. Dulin. "Fibroblast Growth Factor 2 Accelerates Myofibroblast Dedifferentiation in Primary Human Lung Fibroblasts." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4042.

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Shreiber, David I., Paul A. J. Enever, and Robert T. Tranquillo. "Stress-Dependent Effects of Platelet-Derived Growth Factor-BB on Fibroblast Migration and Traction Differ in Collagen and Fibrin." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2575.

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Abstract We used our novel assays of cell behavior in tissue equivalents to study the dose-response effects of PDGF-BB on RDF migration and traction in mechanically stressed and stress-free type I collagen and fibrin gels. PDGF-BB increased fibroblast migration significantly in all assays, but the effects on traction depended on the presence of stress and the nature of the ECM. PDGF-BB decreased fibroblast traction in stressed collagen gels, but increased traction in stress-free gels. No statistical conclusion could be inferred for stressed fibrin gels, and increasing PDGF-BB decreased tractio
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Gould, Russell, Karen Chin, Puifai Santisakultam та ін. "Anisotropic Strain Fields Enhance Matrix Remodeling Through Elevated TGF-β Signaling". У ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53805.

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In this work, we demonstrate the unique effect of controlled anisotropic strain on fibroblast behavior in 3D engineered tissue environments. Anisotropy of biaxial strain resulted in increased cellular orientation and collagen fiber alignment. Transforming growth factor beta-1 (TGFβ1) gene expression and pSmad2 nuclear translocation increased with biaxial directionality. Myofibroblastic alpha-smooth muscle actin (α-SMA) decreased with applied strain similar to mechanically unloaded hydrogels. Collectively, these results demonstrate a novel mechanobiological mechanism by which fibroblasts develo
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Reports on the topic "Fibroblast growth factors"

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Funkenstein, Bruria, and Shaojun (Jim) Du. Interactions Between the GH-IGF axis and Myostatin in Regulating Muscle Growth in Sparus aurata. United States Department of Agriculture, 2009. http://dx.doi.org/10.32747/2009.7696530.bard.

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Growth rate of cultured fish from hatching to commercial size is a major factor in the success of aquaculture. The normal stimulus for muscle growth in growing fish is not well understood and understanding the regulation of muscle growth in fish is of particular importance for aquaculture. Fish meat constitutes mostly of skeletal muscles and provides high value proteins in most people's diet. Unlike mammals, fish continue to grow throughout their lives, although the size fish attain, as adults, is species specific. Evidence indicates that muscle growth is regulated positively and negatively by
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Welling, Bradley. Fibroblast Growth Factor Regeneration of Tympanic Membrane Perforations. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada591173.

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Ittmann, Michael M. Fibroblast Growth Factor Receptor-4 and Prostate Cancer Progression. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada446482.

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Ittmann, Michael M. Fibroblast Growth Factor Receptor-4 and Prostate Cancer Progression. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada462818.

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Ittmann, Michael M. Fibroblast Growth Factor Receptor-4 and Prostate Cancer Progression. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada476956.

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Kagan, Benjamin L. Growth Factor Regulation of an Angiogenic Factor, the Fibroblast Growth Factor-Binding Protein (FGF-BP), in Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398106.

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Kagan, Benjamin L. Growth Factor Regulation of an Angiogenic Factor, the Fibroblast Growth Factor-Binding Protein (FGF-BP), in Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada410065.

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Wieder, Robert. The Role of Basic Fibroblast Growth Factor in Human Breast Cancer. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada302241.

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Tondravi, Mehrdad. Optimization of Fibroblast Growth Factor-1 as an Anabolic Agent for Osteoporosis. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada394773.

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Burgess, Wilson H. Optimization of Fibroblast Growth Factor-1 as an Anabolic Agent for Osteoporosis. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/adb233740.

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