Relevant bibliographies by topics / Fibroblast-pneumocyte factor

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  1. Journal articles
  2. Dissertations / Theses

Academic literature on the topic 'Fibroblast-pneumocyte factor'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Fibroblast-pneumocyte factor"

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Buch, S., R. N. Han, J. Liu, A. Moore, J. D. Edelson, B. A. Freeman, M. Post, and A. K. Tanswell. "Basic fibroblast growth factor and growth factor receptor gene expression in 85% O2-exposed rat lung." American Journal of Physiology-Lung Cellular and Molecular Physiology 268, no. 3 (March 1, 1995): L455—L464. http://dx.doi.org/10.1152/ajplung.1995.268.3.l455.

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Lungs exposed to elevated O2 concentrations suffer an initial loss of type I pneumocytes, followed by a reparative type II pneumocyte hyperplasia. We hypothesized that type II pneumocyte hyperplasia after exposure of young adult rats to 85% O2 in vivo would be temporally related to 1) an increased concentration of intrapulmonary basic fibroblast growth factor (bFGF), a potent stimulator of type II pneumocyte DNA synthesis in vitro, and 2) an upregulation of pneumocyte receptors for bFGF (FGF-R). Increased rat lung bFGF mRNA, relative to air-exposed control animals, was observed at 4 days of ex
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King, George, Megan E. Smith, Max H. Cake, and Heber C. Nielsen. "What is the identity of fibroblast-pneumocyte factor?" Pediatric Research 80, no. 6 (August 8, 2016): 768–76. http://dx.doi.org/10.1038/pr.2016.161.

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Torday, John Steven. "Commentary on “What is the identity of fibroblast pneumocyte factor (FPF)?”." Pediatric Research 81, no. 2 (October 24, 2016): 391. http://dx.doi.org/10.1038/pr.2016.209.

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Ballard, Philip L. "Commentary on the identity of fibroblast pneumocyte factor: rat vs. human." Pediatric Research 82, no. 1 (May 31, 2017): 4–5. http://dx.doi.org/10.1038/pr.2017.85.

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Nielsen, Heber C., George King, and Max H. Cake. "Response to “Commentary on identity of fibroblast pneumocyte factor: rat vs. human”." Pediatric Research 82, no. 1 (May 31, 2017): 6–7. http://dx.doi.org/10.1038/pr.2017.86.

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Sakurai, Maromi K., Sang Lee, Danielle A. Arsenault, Vania Nose, Jay M. Wilson, John V. Heymach, and Mark Puder. "Vascular endothelial growth factor accelerates compensatory lung growth after unilateral pneumonectomy." American Journal of Physiology-Lung Cellular and Molecular Physiology 292, no. 3 (March 2007): L742—L747. http://dx.doi.org/10.1152/ajplung.00064.2006.

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We hypothesize that compensatory lung growth after unilateral pneumonectomy in a murine model is, in part, angiogenesis dependent and can be altered using angiogenic agents, possibly through regulation of endothelial cell proliferation and apoptosis. Left pneumonectomy was performed in mice. Mice were then treated with proangiogenic factors [vascular endothelial growth factor (VEGF); basic fibroblast growth factor (bFGF)], VEGF receptor antibodies (MF-1, DC101), and VEGF receptor small molecule chemical inhibitors. Lung volume and mass were measured. The lungs were analyzed using immunohistoch
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King, George, Jolanta E. Damas, Max H. Cake, David Berryman та Garth L. Maker. "Influence of glucocorticoids, neuregulin-1β, and sex on surfactant phospholipid secretion from type II cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 306, № 3 (1 лютого 2014): L292—L298. http://dx.doi.org/10.1152/ajplung.00297.2013.

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Glucocorticoids induce lung fibroblasts to produce fibroblast-pneumocyte factor, a peptide that stimulates type II cells to synthesize pulmonary surfactant. This effect is known to be more apparent in cells derived from female fetuses, a characteristic that has been attributed to sex-linked differences in the fibroblasts. In the current study, it has been shown that dexamethasone enhances both β-adrenergic receptor (β-AR) activity (1.3- to 1.6-fold increase) and (−)-isoproterenol-induced secretion of surfactant (1.8- to 1.9-fold increase) in type II cells. However, fibroblast-conditioned media
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Colvin, Jennifer S., Andrew C. White, Stephen J. Pratt, and David M. Ornitz. "Lung hypoplasia and neonatal death inFgf9-null mice identify this gene as an essential regulator of lung mesenchyme." Development 128, no. 11 (June 1, 2001): 2095–106. http://dx.doi.org/10.1242/dev.128.11.2095.

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Mammalian lung develops as an evagination of ventral gut endoderm into the underlying mesenchyme. Iterative epithelial branching, regulated by the surrounding mesenchyme, generates an elaborate network of airways from the initial lung bud. Fibroblast growth factors (FGFs) often mediate epithelial-mesenchymal interactions and mesenchymal Fgf10 is essential for epithelial branching in the developing lung. However, no FGF has been shown to regulate lung mesenchyme. In embryonic lung, Fgf9 is detected in airway epithelium and visceral pleura at E10.5, but is restricted to the pleura by E12.5. We r
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Tanswell, A. K., R. N. N. Han, S. J. Buch, and L. J. Fraher. "Circulating Factors That Modify Lung Cell DNA Synthesis Following Exposure to Inhaled Oxidants. III. Effect of Plasma on Lung Pneumocyte and Fibroblast DNA Synthesis Following Exposure of Adult Rats to 85% Oxygen." Experimental Lung Research 17, no. 5 (January 1991): 869–86. http://dx.doi.org/10.3109/01902149109064323.

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Srisuma, Sorachai, Soumyaroop Bhattacharya, Feng Tu, Barry Starcher, and Thomas J. Mariani. "Fibroblast Growth Factor Receptor Deficiency Results in Abnormal Type II Pneumocyte Gene Expression and Dysregulation of Matrix Production." FASEB Journal 21, no. 5 (April 2007). http://dx.doi.org/10.1096/fasebj.21.5.a10.

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Dissertations / Theses on the topic "Fibroblast-pneumocyte factor"

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Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and characterization of fibroblast-pneumocyte factor /." Access via Murdoch University Digital Theses Project, 2007. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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au, G. Maker@murdoch edu, and Garth Lucas Maker. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Murdoch University, 2008. http://wwwlib.murdoch.edu.au/adt/browse/view/adt-MU20080430.141113.

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The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou
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Maker, Garth Lucas. "Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor." Maker, Garth Lucas (2008) Regulation of surfactant production by fetal type II pneumocytes and the characterization of fibroblast-pneumocyte factor. PhD thesis, Murdoch University, 2008. http://researchrepository.murdoch.edu.au/486/.

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Abstract:
The fetal lung undergoes extensive physiological and biochemical maturation prior to birth in preparation for its postnatal function as an organ for gas exchange. Pulmonary surfactant, a substance that reduces surface tension and prevents alveolar collapse, is produced by type II pneumocytes within the lung. Reduced ability to produce surfactant leads to neonatal respiratory distress syndrome. Synthesis of the phospholipid component of surfactant, phosphatidylcholine (PC), is stimulated by fibroblast-pneumocyte factor (FPF), a protein expressed by fibroblast cells within the fetal lung. Althou
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