Academic literature on the topic 'Fibromes'

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Journal articles on the topic "Fibromes"

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Fernandez, Hervé, Amélie Gervaise, and Renaud de Tayrac. "Fibromes utérins." EMC - Gynécologie 1, no. 1 (January 2006): 1–11. http://dx.doi.org/10.1016/s0246-1064(02)00063-4.

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Brabant, G., J. Y. Charvolin, and D. Houzé De L’Aulnoit. "Résection Hystéroscopique des Fibromes." Journal de Gynécologie Obstétrique et Biologie de la Reproduction 35, no. 1 (January 2006): 99–100. http://dx.doi.org/10.1016/s0368-2315(06)77425-7.

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Rhemimet, M., A. Kallali, S. Boujida, N. Zeraidi, A. Lakhdar, A. Baidada, and A. Kharbach. "POLYMYOMECTOMIE RAPPORTANT 20 FIBROMES : A PROPOS DUN CAS ET REVUE DE LA LITERATURE." International Journal of Advanced Research 9, no. 5 (May 31, 2021): 512–16. http://dx.doi.org/10.21474/ijar01/12866.

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Les fibromes uterins sont des tumeurs des cellules musculaires lisses il sagit des tumeurs gynecologiques benignes les plus courantes chez les femmes en age de procreer. Leur presence est souvent constatee dans le cadre de lexploration qui est menee chez un couple aux prises avec linfertilite par ailleurs, ces fibromes sont dorigine monoclonale. Il est rare de constater des fibromes avant lapparition des premières règles de plus, les fibromes connaissent habituellement une regression après la menopause. Ils reagissent aux hormones et les oestrogènes semblent en promouvoir la croissance. Il a ete demontre que les concentrations locales en oestrogènes sont plus elevees dans les myomes que dans le myomètre environnant, ce qui sexplique peut-etre par une concentration accrue en aromatase. La reactivite hormonale semble etre accrue dans le cas des myomes sous-muqueux, par comparaison avec celle que presentent les myomes sous-sereux.Lechographie constitue un moyen adequat, rapide, sur et rentable de determiner la taille, le nombre et lemplacement des fibromes .La prise en charge de ces myomes peut etre une abstention ou un traitement medical ou un traitement chirurgical conservateur polymyomectomie ou radicale une hysterectomie.
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Le Dref, O., J. P. Pelage, and D. Jacob. "Fibromes utérins. Embolisation : pratiques actuelles." EMC - Gynécologie 1, no. 1 (January 2006): 1–6. http://dx.doi.org/10.1016/s0246-1064(05)43160-6.

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Lefèbvre, Guylaine G., George Vilos, and Murray Asch. "Embolisation des fibromes utérins (EFU)." Journal of Obstetrics and Gynaecology Canada 26, no. 10 (October 2004): 913–28. http://dx.doi.org/10.1016/s1701-2163(16)30142-6.

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Le Dref, O., J. P. Pelage, and D. Jacob. "Fibromes utérins. Embolisation : pratiques actuelles." EMC - Gynécologie-Obstétrique 2, no. 3 (August 2005): 261–68. http://dx.doi.org/10.1016/j.emcgo.2005.04.001.

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Kovacsik, H. "Traitement endovasculaire des fibromes utérins." JMV-Journal de Médecine Vasculaire 46, no. 5 (October 2021): S13—S14. http://dx.doi.org/10.1016/j.jdmv.2021.08.183.

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Bordessoules, A., and H. Trillaud. "Traitement des fibromes uterins par embolisation." Journal de Radiologie 89, no. 10 (October 2008): 1249. http://dx.doi.org/10.1016/s0221-0363(08)75713-5.

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Le Dref, O., J. P. Pelage, and D. Jacob. "Les fibromes utérins. Embolisation : pratiques actuelles." Gynécologie Obstétrique & Fertilité 32, no. 12 (December 2004): 1057–63. http://dx.doi.org/10.1016/j.gyobfe.2004.08.021.

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Ravina, J. H. "Les fibromes utérins. Embolisation : pratiques actuelles." Gynécologie Obstétrique & Fertilité 33, no. 5 (May 2005): 363–64. http://dx.doi.org/10.1016/j.gyobfe.2005.04.004.

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Dissertations / Theses on the topic "Fibromes"

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MOLLARD, JOELLE. "Endometriose, fibromes et traitement substitutif de la menopause." Aix-Marseille 2, 1994. http://www.theses.fr/1994AIX20703.

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HUYS, CHRISTIAN. "Imagerie des fibromes pleuraux : a propos de 4 cas." Angers, 1994. http://www.theses.fr/1994ANGE1075.

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Nassif, Elie. "Les fibromes sous-pleuraux : revue générale à propos d'un cas." Montpellier 1, 1994. http://www.theses.fr/1994MON11171.

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LICATA, JACQUES. "Tactiques operatoires des fibromes uterins : a propos d'une serie personnelle." Aix-Marseille 2, 1990. http://www.theses.fr/1990AIX20318.

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Sabourdy, Frédérique. "Poxivirus et tumeurs : influence des facteurs de croissance viraux sur la formation des fibromes de Shope." Toulouse 3, 2004. http://www.theses.fr/2004TOU30277.

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La famille des Poxviridae compte trois virus tumorigènes. Les mécanismes empruntés par ces virus pour induire des tumeurs sont inconnus et semblent originaux. Certains poxvirus codent des homologues de l'EGF qui représentent de bons candidats pour participer à la tumorigénèse. Après une présentation générale de la biologie des Poxviridae, l'auteur s'interesse plus particulièrement au virus du fibrome de Shope (VFS), responsable de fibromes cutanés chez le lapin, dont il présente la symptomatologie et la pathogénie. Elle rappelle les caractéristiques des facteurs de croissance EGF-like cellulaires et viraux et leurs liens avec la tumorigénèse. L'auteur montre que le facteur de croissance du VFS, SFGF, active la prolifération des fibroblastes dans les tumeurs, et pourrait participer aux modifications morphologiques dont ils sont les cibles. Le facteur de croissance du virus myxomateux, MGF, ne peut le remplacer
Only three poxviruses are tumorigenic. The mechanisms they use to induce tumours remain unknown. Some of them encode EGF-like homologs, which represent good candidates for triggering tumorigenesis. The author first gives a general overview of the poxvirus biology. Then, she presents poxvirus tumorigenesis characteristics, using Shope fibroma virus (SFV) as an example. She describes cellular and viral EGF-like growth factors, showing how these peptides could be linked with tumour formation. According to her experimental results, SFV growth factor, SFGF, activates fibroblasts proliferation in fibromas, and could also be involved in their morphological abnormalities. Myxoma virus growth factor, MGF, cannot substitute for SFGF
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Payafar, Alireza. "L'évaluation par l'I. R. M. Des fibromes utérins traités par la triptolérine." Montpellier 1, 1993. http://www.theses.fr/1993MON11144.

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AVISSE, LAMARRE ANNABELLE. "Donnees cliniques actuelles sur les fibromes pleuraux : a propos de cinq observations." Amiens, 1992. http://www.theses.fr/1992AMIEM109.

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DELVINCOURT, BARTHES MONIQUE, and NICOLAS VIRNOT. "Les fibromes cardiaques : 1 cas chez un nourrisson ; revue de la litterature depuis 1971." Lille 2, 1988. http://www.theses.fr/1988LIL2M028.

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Jourdain, Olivier. "Une nouvelle méthode de traitement des fibromes : la thermothérapie interstitielle au laser Nd:Yag : étude préliminaire." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23009.

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Banegas, Danielle. "Contribution à l'étude de la dégénérescence sarcomateuse des fibromes de l'utérus : à propos d'une observation." Montpellier 1, 1989. http://www.theses.fr/1989MON11215.

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Books on the topic "Fibromes"

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Dubuisson, Jean-Bernard. Les fibromes utérins. Paris: Arnette, 1994.

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Niels, Høiby, and Pedersen Svend Stenvang, eds. Cystic fibrosis, basic and clinical research: Proceedings of the 17th Annual Meeting of the European Working Group for Cystic Fibrosis, Copenhagen, 17-21 June 1991. Amsterdam: Excerpta Medica, 1992.

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Symposium, Ciba Foundation. Fibrosis. London: Pitman, 1985.

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Segars, James. Fibroids. Chichester, West Sussex: John Wiley & Sons, 2013.

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Segars, James H., ed. Fibroids. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.

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Rittié, Laure, ed. Fibrosis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7113-8.

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Dubuisson, Jean-Bernard. Uterine fibroids. Paris: Arnette, 1997.

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Cystic fibrosis. Oxford: Oxford University Press, 2010.

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Gold, Susan Dudley. Cystic fibrosis. New York: Crestwood House, 2000.

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Bjorklund, Ruth. Cystic fibrosis. New York: Marshall Cavendish Benchmark, 2009.

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Book chapters on the topic "Fibromes"

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Boyer, L., E. Dumousset, N. Mazet, A. Ravel, A. Alfidja Lankoande, and P. Chabrot. "Embolisation utérine pour fibromes." In Collection de la Société française d’imagerie cardiaque et vasculaire, 315–31. Paris: Springer Paris, 2012. http://dx.doi.org/10.1007/978-2-287-99170-7_19.

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Mazabraud, André. "Cortico-metaphyseal fibrous defect, non-ossifying fibroma, multiple fibromas, benign fibrous histiocytoma." In Pathology of bone tumours, 153–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-95839-7_12.

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Bährle-Rapp, Marina. "Fibrose, auch: Fibrosis." In Springer Lexikon Kosmetik und Körperpflege, 206. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_3985.

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Bährle-Rapp, Marina. "Fibrom, auch: Fibroma." In Springer Lexikon Kosmetik und Körperpflege, 206. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_3981.

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Bratani, L., Ole Welling, and H. E. Schaller. "Fibrome." In Plastische Chirurgie, 299–300. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-48849-2_19.

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Leppert, Phyllis, Mazen Fouany, and James H. Segars. "Understanding Uterine Fibroids." In Fibroids, 1–10. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.ch1.

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Heitmann, Ryan J., Cindy M. P. Duke, William H. Catherino, and Alicia Y. Armstrong. "Surgical Treatments and Outcomes." In Fibroids, 109–19. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.ch10.

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Cookingham, Lisa Marii, Alicia Y. Armstrong, Aradhana Venkatesan, and James H. Segars. "Rare Fibroid Syndromes." In Fibroids, 120–33. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.ch11.

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Christman, Gregory M., Courtney A. Marsh, and Elizabeth J. Campbell. "Counseling the Patient with Uterine Fibroids." In Fibroids, 134–44. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.ch12.

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Younger, Joshua, K. Maravet Baig-Ward, James H. Segars, and Ayman Al-Hendy. "The Clinical Spectrum of Fibroid Disease." In Fibroids, 11–23. Oxford: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118456996.ch2.

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Conference papers on the topic "Fibromes"

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Lafont, J., J. H. Catherine, M. Lejeune, U. Ordioni, R. Lan, and F. Campana. "Manifestations buccales de la sclérose tubéreuse de Bourneville." In 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603014.

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L’objectif de ce travail est de faire le point sur les manifestations buccales de la sclérose tubéreuse de Bourneville (STB) à travers le cas d’un jeune patient. Un jeune homme de 15 ans était adressée pour la mise en place de minivis orthodontique afin de fermer des espaces d’agénésies de 35 et 45. L’interrogatoire retrouvait une STB dont les manifestations épileptiques étaient traitées par de la lamotrigine 75mg/j et de la carbamazépine LP 200mg/j. L’examen clinique exo-buccal retrouvait des macules hypochromiques sur le membre inférieur droit, des angiofibromes faciaux et une malformation vasculaire jugale gauche. L’examen endo-buccal retrouvait de multiples lésions buccales sur les papilles interdentaires pouvant évoquer des fibromes ou des hamartomes. Une biopsie était réalisée et retrouvait un revêtement malpighien, discrètement hyperplasique et sans atypie cellulaire. Les faisceaux collagènes du conjonctif étaient mêlés à de nombreux fibroblastes aux noyaux réguliers, sans mitose visible. Les cellules inflammatoires, essentiellement mononuclées, étaient dispersées mais tendaient à se regrouper autour de vaisseaux nombreux et hyperplasiques. L’examen concluait à un fibrome. Aucun traitement buccal n’était proposé devant l’absence de symptôme et de demande esthétique. La STB est une maladie génétique autosomique dominante avec une incidence de 1/10 000. Elle est liée à une mutation du gène TSC1 sur le chromosome 9 ou du gène TSC2 sur le chromosome 16 qui perturbe la sécrétion d’une protéine régulant la voie mTOR. C’est une maladie multisystème avec une expression clinique variable. Les principaux symptômes sont l’épilepsie, le retard mental et la présence d’adénomes sébacés, mais la maladie est associée à un polymorphisme clinique rendant le diagnostic difficile. La conférence de consensus de 2012 a ainsi défini des critères diagnostiques majeurs (lésions cutanées, oculaires, cérébrales, cardiaques, pulmonaires, rénales,..) et mineurs dont deux sont bucco-dentaires. Le diagnostic est retenu devant deux critères majeurs ou un critères majeur et deux critères mineurs. Les signes oraux sont la présence de trois ou plus puits d’émail et deux ou plus fibromes gingivaux. Les fibromes gingivaux atteindraient 50 à 70% des patients. La région antérieure maxillaire semble la plus touchée. L’exérèse est indiquée en cas de gêne esthétique ou de saignements associés. Actuellement, les inhibiteurs de mTOR représentent une option thérapeutique proposée dans la prise en charge des patients atteints de STB. La STB est une pathologie rare. La présence de lésions buccales fait partie des critères diagnostiques.
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Perez, R., B. Salmon, N. Moreau, and AL Ejeil. "Les fibromes ossifiants périphériques : Cas clinique et Revue de la littérature." In 65ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2017. http://dx.doi.org/10.1051/sfco/20176502001.

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Holmes, Hal, Eli Vlaisavljevich, Ee Lim Tan, Keat G. Ong, and Rupak M. Rajachar. "Magnetoelastic Materials as Novel Bioactive Coatings to Improve Integration of Percutaneous Implants." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53308.

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Fibroblastic activity is an innate function of the host response. In the presence of many percutaneous biomedical implants, this activity becomes uncontrollable, resulting in significant fibrous overgrowth at the soft tissue-implant interface [1]. The aberrant cell growth associated with pathological fibrosis can lead to extensive remodeling and excessive synthesis of extracellular matrix (ECM) components, preventing proper integration [2]. Furthermore, these areas of irregular fibrotic activity can also serve as sites for opportunistic infection [3]. In brief, interfacial fibrosis is often responsible for the ultimate failure and increased risk of infection of percutaneous biomedical implants.
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Baker, Brendon M., Grace D. O’Connell, Sounok Sen, Ashwin S. Nathan, Dawn M. Elliott, and Robert L. Mauck. "Multi-Lamellar and Multi-Axial Maturation of Cell-Seeded Fiber-Reinforced Tissue Engineered Constructs." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176434.

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The architecture of load-bearing fibrous tissues is optimized to enable a specific set of mechanical functions. This organization arises from a complex process of cell patterning, matrix deposition, and functional maturation [1]. In their mature state, these tissues span multiple length scales, encompassing nanoscale interactions of cells with extracellular matrix to the centimeter length scales of the anatomic tissue volume and shape. Two structures that typify dense fibrous tissues are the meniscus of the knee and the annulus fibrosus (AF) of the intervertebral disc (IVD). The mechanical function of the wedge-shaped knee meniscus is based on its stiff prevailing circumferential collagen architecture that resists tensile deformation [2,3]. Adding to its complexity, radial tie fibers and sheets are interwoven amongst these fibers, increasing stiffness in the transverse direction and binding the tissue together [4]. In the annulus fibrosus, multiple anisotropic lamellae are stacked in concentric rings with their prevailing fiber directions alternating above and below the horizontal axis in adjacent layers [5]. The high circumferential tensile properties of this laminate structure allow it to resist bulging of the nucleus pulposus with compressive loading of the spine. Given their structural properties, unique form, and demanding mechanical environments, the knee meniscus and the AF region of the IVD represent two of the most challenging tissues to consider for functional tissue engineering.
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SILVA, MATHEUS ROBERTO DA, ANA LUIZA GOMES DO NASCIMENTO BATISTA, CARLA FERNANDA DE FREITAS TEIXEIRA, GILMARA MORAIS DE ARAUJO, JOICE RAQUEL URBANO DO NASCIMENTO, VICTOR CESAR URQUIZA CANDEIA, and MILENA NUNES ALVES DE SOUSA. "RELAÇÃO DOS PRINCIPAIS ASPECTOS DA FIBROSE CÍSTICA: UMA BREVE REVISÃO DE LITERATURA." In Brazilian Congress. brazco, 2020. http://dx.doi.org/10.51162/brc.health2020-00015.

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A fibrose cistica (FC) e uma doenca genetica, cronica, progressiva e de cunho letal. Este trabalho teve como objetivo realizar uma revisao de literatura sobre as caracteristicas gerais da mucoviscidose. Para tanto, utilizou-se as plataformas eletronicas SciELO, Scopus, PubMed/MEDLINE e periodicos CAPES, sendo a pesquisa realizada utilizando os termos: fibrose cistica ou cystic fibrosis. Selecionaram-se os artigos desde a decada de 1950 ate o ano de 2018. Ao longo dos ultimos anos varias descobertas estimuladas principalmente pela alta prevalencia e letalidade da doenca, tais como, o sequenciamento da principal mutacao do gene, culminando na formacao de um aminoacido mutado, envolvido na patogenese da FC e a constatacao do envolvimento multissistemico, caracterizado por uma patologia pulmonar progressiva, doenca que acomete o sistema hepatico e ocasiona disfuncao pancreatica exocrina, problemas na capacidade intestinal, infertilidade masculina e concentracao elevada de eletrolitos no suor. Todo esse processo evolutivo resulta em diagnostico cada vez mais precoce e tratamento mais eficaz com enfoque na qualidade de vida e sobrevida dos pacientes. Apesar disso, varias lacunas no ambito genetico, a titulo de exemplificacao, ainda existem para consolidacao de um tratamento mais eficiente e ate a chegada da cura. ,
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Reza, Anna T., and Steven B. Nicoll. "Dynamic Hydrostatic Pressurization Differentially Regulates Extracellular Matrix Elaboration by Bovine Inner and Outer Annulus Fibrosus Cells Seeded on 3-D Polymer Scaffolds." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176539.

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Current surgical treatments for intervertebral disc (IVD) degeneration result in decreased mobility of the spine [1]. A tissue engineering approach may provide an alternative that restores both IVD structure and function. The IVD is comprised of three distinct regions: the outer annulus fibrosus (OA), inner annulus fibrosus (IA), and the nucleus pulposus (NP). Each of the cell populations within these regions possess unique phenotypic properties that are greatly influenced by environmental factors, such as the surrounding 3-D extracellular matrix (ECM) and mechanical loading (i.e., hydrostatic pressurization) [2]. As such, both the 3-D scaffold and in vitro culture conditions may have marked effects on the development of tissue-engineered IVD constructs. Although the influence of mechanical loading on IVD cells and explants has been investigated, no prior studies have examined the impact of hydrostatic pressurization on OA and IA cells in 3-D culture. Therefore, the objective of this study was to determine the effects of dynamic hydrostatic pressurization on OA and IA cells seeded on 3-D fibrous poly(glycolic acid)-poly(L-lactic acid) (PGA-PLLA) scaffolds. We hypothesized that the application of hydrostatic pressure would promote increased production of type II collagen and chondroitin sulfate proteoglycan in both OA- and IA-seeded constructs.
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Labus, K. M., A. H. Hsieh, and C. M. Puttlitz. "Lamellar and Interlamellar Shear Compared Across Regions of the Annulus Fibrosus." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14309.

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Computational models of the intervertebral disc commonly use continuum descriptions that treat the annulus fibrosus as a single material rather than discretely modeling the lamellae and interlamellar interactions [1,2]. However, modeling the mechanics of individual lamellae and the interlamellar region can aid in the understanding of degenerative disc disease and its treatment. Previous work has demonstrated that fibrous connections between lamellae as well as bridges spanning across layers exist, but the mechanical contributions of these structures have largely remained uncharacterized [3]. Studying interlamellar shear mechanics may provide insights into the structure-function relationships of the annulus. The purpose of this study was to compare the mechanical shear in the interlamellar and lamellar regions, model the stress-stretch relationships of these areas utilizing a hyperelastic strain energy function, and compare the shear properties across multiple locations of the intervertebral disc.
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McCaffrey, Katherine, Karen Rose, and John P. Abraham. "Numerical Simulation of Cryosurgery as a Potential Treatment for Uterine Fibroids." In 2010 14th International Heat Transfer Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/ihtc14-22106.

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Nearly 80% of all women may suffer from menorrhagia caused by uterine fibroids (leiomyomas) which are benign tumors made up of muscle and fibrous tissue that grow from the muscular wall of the uterus. The vast majority of women whose symptoms are strong enough to require treatment obtain a hysterectomy. Other treatment options which are less invasive than hysterectomy include thermal therapies such as thermal ablation or cryosurgical removal of tissue. This project numerically evaluates the efficacy of a liquid-nitrogen-based cryotherapy for the treatment of uterine fibroids. A bioheat transfer model was utilized which includes both the effects of blood perfusion and the impacts of liquid-to-solid phase change. Changes in all physical properties including thermal conductivity, heat capacity, and perfusion rate were taken into account as the tissue passed through a range of temperatures where it would be transitioning from unfrozen to fully frozen. The numerical model was based on a one-dimensional unsteady bioheat equation. The results show that even for the direct-contact cooling, it is unlikely that intracellular ice would form during the procedure. On the other hand, based on data obtained from previous cell-survival studies, it was found that necrosis would occur when the cooling rates exceeded 30°/min. According to the present numerical results, necrosis would occur within the tissue up to a depth of approximately 5.8 mm, thereby ensuring that sufficient tissue would be cryosurgically destroyed to result in effective treatment.
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Samsonova, M. V., E. V. Kusraeva, A. L. Cherniaev, and N. V. Trushenko. "Histological Signs of Differential Diagnosis of Interstitial Pulmonary Fibrosis (IPF) and Fibrous Hypersensitive Pneumonitis." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3144.

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Higuita-Castro, Natalia, Cosmin Mihai, Derek J. Hansford, and Samir N. Ghadiali. "In-Vitro Model of the Microscale Alveolar Environment." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53648.

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The present work describes the development of a novel micro-nanoscale system that more closely resembles the alveolar-capillary barrier in the lung by recapitulating different parameters of the cellular microenvironment, including fibrous geometry, fiber stiffness, chemistry, and cell-cell interactions. The system consists of a three-dimensional multilayered structure. Two microchannel chambers that resemble alveolar/airway space and the capillary lumen, interfaced with a porous mesh of polymeric nanofibers that act as the basal substrate for seeding lung epithelial and endothelial cell. The top and bottom chambers of the device were fabricated using soft lithography techniques, while the nanofiber mesh was obtained via electrospinning. Human alveolar epithelial cells (A549) and human umbilical vein endothelial cells (HUVEC) were successfully co-cultured using this system. Various cellular and molecular biology techniques are being employed to investigate injury patterns and overall cell responses under different circumstances which mimic various lung disorders such acute lung injury, pulmonary fibrosis and emphysema.
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Reports on the topic "Fibromes"

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Dvorak, George J. Plasticity of Fibrous Composites. Fort Belvoir, VA: Defense Technical Information Center, May 1987. http://dx.doi.org/10.21236/ada184637.

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Mameti, Lilian. Open Hysterectomy for a Patient with Fibroids. Touch Surgery Publications, August 2019. http://dx.doi.org/10.18556/touchsurgery/2016.s0167.

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Mameti, Lilian. Open Hysterectomy for a Patient with Fibroids. Touch Surgery Simulations, August 2019. http://dx.doi.org/10.18556/touchsurgery/2019.s0167.

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Agarwal, Sandeep K. Cadherin-11 Regulation of Fibrosis through Modulation of Epithelial-to-Mesenchymal Transition: Implications for Pulmonary Fibrosis in Scleroderma. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada591380.

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Agarwal, Sandeep K. Cadherin-11 Regulation of Fibrosis through Modulation of Epithelial-to-Mesenchymal Transition: Implications for Pulmonary Fibrosis in Scleroderma. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada618226.

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Smith, David R. Thermal conductivity of fibrous glass board :. Gaithersburg, MD: National Bureau of Standards, 1997. http://dx.doi.org/10.6028/nist.tn.1391.

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Muszynski, L. C., and M. A. Rochefort. Fibrous Shotcrete for Expedient Repair of Structures. Fort Belvoir, VA: Defense Technical Information Center, September 1994. http://dx.doi.org/10.21236/ada311454.

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Artlett, Carol M. The Modulation of Fibrosis in Scleroderma by 3-Deoxyglucosone. Fort Belvoir, VA: Defense Technical Information Center, June 2008. http://dx.doi.org/10.21236/ada485008.

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Artlett, Carol M. The Modulation of Fibrosis in Scleroderma by 3-Deoxyglucosone. Fort Belvoir, VA: Defense Technical Information Center, June 2010. http://dx.doi.org/10.21236/ada541210.

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Velentgas, Priscilla, Donna Messner, and Evan Myers. Comparing Patient-Centered Outcomes after Treatment for Uterine Fibroids. Patient-Centered Outcomes Research Institute (PCORI), May 2018. http://dx.doi.org/10.25302/5.2018.ce.12114430.

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