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Relevant bibliographies by topics / Fibrosis and cirrhosis

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Academic literature on the topic 'Fibrosis and cirrhosis'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Fibrosis and cirrhosis"

1

Sebay, Ahmed M. El, Pavel N. Abramov та Seidfatima M. Borunova. "Мolecular diagnosis of fibrotic and cirrhotic changes in the liver of dogs". Veterinariya, Zootekhniya i Biotekhnologiya 1, № 98 (2022): 24–33. http://dx.doi.org/10.36871/vet.zoo.bio.202201004.

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Liver fibrosis or cirrhosis are major outcomes of long-standing chronic liver injury in which most dogs remain asymptomatic until fatal liver insufficiency is developed. The current study aimed to investigate if hepatocyte-derived cfa-miRNAs-21 and -200c can serve as new diagnostic serum biomarkers of the fibrotic and cirrhotic changes in dogs with chronic liver injury. On the basis of ultrasound, computed tomography and histopathological examination; twenty healthy dogs were included in the control group for comparison with dogs that were confirmed to have fibrosis or cirrhosi. Cfa-miRNA -20

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Che, Ying, Youjung Chien, Yuli Zhu, et al. "GSDMD-Dependent Neutrophil Extracellular Traps Mediate Portal Vein Thrombosis and Associated Fibrosis in Cirrhosis." International Journal of Molecular Sciences 25, no. 16 (2024): 9099. http://dx.doi.org/10.3390/ijms25169099.

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Portal vein thrombosis (PVT) is a challenging and controversial complication of cirrhosis. Experimental models that reproduce cirrhotic PVT and effective pharmacological therapies are limited. We aimed to investigate the nature course and mechanisms of PVT in cirrhosis. A novel PVT model was developed via two-step total portal vein ligation in healthy and thioacetamide (TAA)-cirrhotic rats. Circulating and liver-infiltrating neutrophils were isolated from individuals with cirrhosis to examine neutrophil extracellular traps (NETs) and explore their unique characteristics and implications in PVT

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Chirapongsathorn, Sakkarin. "Stage and natural history of cirrhosis." Thai Journal of Hepatology 1, no. 2 (2018): 14–18. http://dx.doi.org/10.30856/th.jhep2018vol1iss2_04.

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Cirrhosisis the final pathway of any chronic liver disease and can lead to a number of complications,including hepatocellular carcinoma. Liver fibrosis is the key role of disease progression in cirrhosis. Advanced liver fibrosis and early stage of cirrhosis are not usually clinically detectable or symptomatic.As patients develop more extensive hepatic fibrosis potentially resulting in development of cirrhoticcomplications such as variceal hemorrhage. Prognosis and survival is markedly better in patients with early stage of cirrhosis. Now there are many stages classification of cirrhosis define

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Moreau, Johanna, Pascaline Bouzy, Julien Guillard, et al. "Analysis of Hepatic Fibrosis Characteristics in Cirrhotic Patients with and without Hepatocellular Carcinoma by FTIR Spectral Imaging." Molecules 25, no. 18 (2020): 4092. http://dx.doi.org/10.3390/molecules25184092.

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The evolution of cirrhosis is marked by quantitative and qualitative modifications of the fibrosis tissue and an increasing risk of complications such as hepatocellular carcinoma (HCC). Our purpose was to identify by FTIR imaging the spectral characteristics of hepatic fibrosis in cirrhotic patients with and without HCC. FTIR images were collected at projected pixel sizes of 25 and 2.7 μm from paraffinized hepatic tissues of five patients with uncomplicated cirrhosis and five cirrhotic patients with HCC and analyzed by k-means clustering. When compared to the adjacent histological section, the

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Chaopathomkul, Bundit, Ornalin Boonsirisak, and Krit Pongpirul. "Correlation of the Stiffness of Hepatocellular Carcinoma and Surrounding Liver Parenchyma by Point Shear Wave Elastography." Journal of Diagnostic Medical Sonography 35, no. 1 (2018): 10–15. http://dx.doi.org/10.1177/8756479318801587.

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The purpose of this study was to assess the correlation between hepatocellular carcinoma (HCC) and surrounding liver parenchyma stiffness using point shear wave elastography (pSWE). HCC was diagnosed using the criteria of the American Association for the Study of Liver Diseases. Liver fibrosis was classified into three groups (nonsignificant fibrosis, significant fibrosis, and cirrhosis). pSWE was performed on the HCC and the adjacent hepatic parenchyma and was expressed as kilopascal (kPa). A total of 59 HCC patients with 64 tumors were included in the study. The mean stiffnesses of HCC and l

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Han, Man-Hoon, Jee Hyun Lee, Gyeonghwa Kim, et al. "Expression of the Long Noncoding RNA GAS5 Correlates with Liver Fibrosis in Patients with Nonalcoholic Fatty Liver Disease." Genes 11, no. 5 (2020): 545. http://dx.doi.org/10.3390/genes11050545.

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Background: Advanced liver fibrosis is the most important prognostic factor in nonalcoholic fatty liver disease (NAFLD). The long noncoding RNA (lncRNA), growth arrest-specific transcript 5 (GAS5), is associated with the inhibition of liver fibrogenesis, and its levels are decreased in cirrhotic liver. Methods: We analyzed 51 patients with NAFLD, the diagnosis of which was confirmed by liver biopsy. Expression of GAS5 in both the liver and plasma of the patients was analyzed using a quantitative real-time polymerase chain reaction according to the fibrosis stage. Results: Plasma GAS5 expressio

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Bravo, E., E. D'Amore, F. Ciaffoni, and C. L. Mammola. "Evaluation of the spontaneous reversibility of carbon tetrachloride-induced liver cirrhosis in rabbits." Laboratory Animals 46, no. 2 (2012): 122–28. http://dx.doi.org/10.1258/la.2012.011035.

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There is a general consensus that liver fibrosis in humans is potentially reversible, while scepticism prevails on the concept that cirrhosis can be truly reversed. The availability of suitable experimental models is fundamental for disease research. The experimental murine model of liver cirrhosis induced by carbon tetrachloride (CCl4) reproduces both the histological picture of the postnecrotic cirrhosis and its biochemical and clinical parameters. Normal hepatic structure is modified by formation of regeneration nodules. Fibrosis represents a morphological element of disease and an effect o

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Lafoz, Erica, Genís Campreciós, Héctor García-Calderó, et al. "Impact of lifestyle interventions targeting physical exercise and caloric intake on cirrhosis regression in rats." American Journal of Physiology-Gastrointestinal and Liver Physiology 321, no. 6 (2021): G603—G616. http://dx.doi.org/10.1152/ajpgi.00191.2021.

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We have developed two advanced cirrhosis regression experimental models with persistent relevant fibrosis and portal hypertension and an associated deteriorated metabolism that mimic what happens in patients. LI, despite improving metabolism, did not enhance the regression process in our cirrhotic models. CR did not further reduce PP, hepatic fibrosis, or HSC activation. MEE exhibited a profibrogenic effect in the liver blunting cirrhosis regression. One of the potential explanations of this worsening could be ammonia accumulation.

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Delgado, María Gabriela, Jordi Gracia-Sancho, Giusi Marrone, et al. "Leptin receptor blockade reduces intrahepatic vascular resistance and portal pressure in an experimental model of rat liver cirrhosis." American Journal of Physiology-Gastrointestinal and Liver Physiology 305, no. 7 (2013): G496—G502. http://dx.doi.org/10.1152/ajpgi.00336.2012.

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Increased hepatic vascular resistance mainly due to elevated vascular tone and to fibrosis is the primary factor in the development of portal hypertension in cirrhosis. Leptin, a hormone associated with reduction in nitric oxide bioavailability, vascular dysfunction, and liver fibrosis, is increased in patients with cirrhosis. We aimed at evaluating whether leptin influences the increased hepatic resistance in portal hypertension. CCl4-cirrhotic rats received the leptin receptor-blocker ObR antibody, or its vehicle, every other day for 1 wk. Hepatic and systemic hemodynamics were measured in b

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Jian, Jianbo, Xinyan Zhao, Lili Qin, et al. "Three-dimensional visualization of fibrous tissues in cirrhotic rats via X-ray phase-contrast computed tomography with iodine staining." Journal of Synchrotron Radiation 26, no. 4 (2019): 1354–60. http://dx.doi.org/10.1107/s1600577519006064.

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To accurately characterize cirrhosis, knowledge of the 3D fibrous structures is essential. Histology is the gold standard in cirrhosis screening, but it mainly provides structural information in 2D planes and destroys the 3D samples in the process. The aim of this study was to evaluate the potential of X-ray phase-contrast computed tomography (PCCT) with iodine staining for the 3D nondestructive visualization of internal structural details in entire cirrhotic livers with histopathologic correlation. In this study, cirrhotic livers induced by carbon tetrachloride (CCl4) in rats were imaged via

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Dissertations / Theses on the topic "Fibrosis and cirrhosis"

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Jobara, Kanta. "Whey-hydrolyzed peptide-enriched immunomodulating diet prevents progression of liver cirrhosis in rats." Kyoto University, 2014. http://hdl.handle.net/2433/189665.

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Yasuda, Katsutaro. "A nonhuman primate model of liver fibrosis towards cell therapy for liver cirrhosis." Kyoto University, 2020. http://hdl.handle.net/2433/258975.

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Wang, Xin Maggie. "Fibroblast activation protein in cell biology and liver fibrosis." Thesis, The University of Sydney, 2007. https://hdl.handle.net/2123/28106.

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Fibroblast activation protein (FAP) is a type 11 cell surface protein of the dipeptidyl peptidase IV (DPIV) gene family. FAP has two peptidase activities, DP activity and a recently reported narrow prolyl endopeptidase activity, which is restricted to Gly-Pro derived substrates. The only known natural substrate of FAP is a collagen type I (CN-I) specific gelatinase activity. In contrast, as the closest relative of PAP, DPIV has only DP activity. Nine chemokines, including CXCL12, are DPIV substrates and DPIV binds to fibronectin (FN). The natural ligands of FAP are unknown. FAP is not de

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Chang, Ping. "Quantitative evaluation of hepatic morphological alterations and pharmacokinetic changes of cationic drugs in fibrosis-inducing hepatic diseases /." St. Lucia, Qld, 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16767.pdf.

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Rodríguez, Navarro Sarai. "New and combined treatments for cirrhosis and portal hypertension: effects on hemodynamics and hepatic fibrosis in experimental animal models." Doctoral thesis, Universitat Autònoma de Barcelona, 2016. http://hdl.handle.net/10803/390964.

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La cirrosi, la causa més freqüent d’hipertensió portal (HTP) en els països occidentals, és considerada una malaltia amb múltiples fases, que evoluciona des d'etapes inicials asimptomàtiques a un estat de cirrosi descompensada amb diverses manifestacions clíniques, les quals representen la principal causa de mort i de trasplantament hepàtic a tot el món. Per tant, caldria adaptar les diferents teràpies per a la cirrosi a cadascuna de les fases de la malaltia. Malgrat els avenços realitzats en les últimes dècades per entendre millor la fisiopatologia de la HTP i poder desenvolupar noves estratèg

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Gangadharan, Bevin. "Proteomics in viral disease." Thesis, University of Oxford, 2006. http://ora.ox.ac.uk/objects/uuid:c66c53ed-a824-4f99-8f2b-d2bc65a984c7.

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The separation, identification, and characterisation of the proteins present in a tissue or biological sample is called ‘proteomics’. This technique can be used for example to identify biomarkers and investigate signalling pathways. Increasingly, proteomics is being applied to the analysis of virus related samples; here two such examples are described. Presently there is no reliable non-invasive way of assessing liver fibrosis. Here a novel 2D-PAGE based proteomics study was used to identify potential fibrosis biomarkers. Serum from patients with varying degrees of hepatic scarring induced by

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Mooney, Paul Joseph. "The use of alpha-1-acid glycoprotein as a putative non-invasive marker of fibrosis and prognostic indicator of cirrhosis." Thesis, University of Strathclyde, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426354.

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Koyama, Yukinori. "Effects of oral intake of hydrogen water on liver fibrogenesis in mice." Kyoto University, 2014. http://hdl.handle.net/2433/185194.

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Rui, Leandro Almeida. "Avaliação da terapia celular utilizando células-tronco mesenquimais para o tratamento de cirrose hepática em ratos Wistar." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-09012015-085311/.

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A cirrose hepática é uma doença crônica e irreversível com modificações histológicas caracterizadas por nódulos de regeneração e desarranjo de hepatócitos, com aumento difuso de tecido conjuntivo e perda de função. As células-tronco têm sido amplamente estudadas para o tratamento de injúrias hepáticas. Fibrose e cirrose foram induzidas em ratos Wistar, através da aplicação de tioacetamida (TAA) intraperitoneal a 200 mg/kg por 8 ou 14 semanas, seguido de terapia celular com aplicação intravenosa única de 1x106 células-tronco de membrana amniótica de ratas. Amostras de sangue foram coletadas par

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Silva, Elizangela dos Anjos. "Avaliação morfológica e molecular pós-tratamento com os fatores hepatotróficos, da cirrose hepática induzida em ratas pela tioacetamida: análise a curto e longo prazo." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-03042012-163500/.

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A cirrose hepática é um processo irreversível caracterizado pelo desequilíbrio entre a deposição e a degradação de componentes da matriz extracelular (MEC), acarretando disfunção hepatocelular e aumento da resistência ao fluxo sanguíneo, resultando em insuficiência hepática e na hipertensão portal. A cirrose em cães, muitas vezes é identificada nos estágios finais, já que o estágio inicial é assintomático. Em humanos, no estágio final da cirrose, o transplante é o único tratamento. Desta forma, a busca por tratamentos alternativos torna-se imprescindível. A administração de fatores hepatotrófi

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Books on the topic "Fibrosis and cirrhosis"

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C, Surrenti, ed. Fat-storing cells and liver fibrosis: Proceedings of the 71st Falk Symposium held in Florence, Italy, July 1-3, 1993. Kluwer, 1994.

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Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Cystic fibrosis-associated liver disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0022.

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Pathophysiology 162Clinical features 162Diagnosis 163Management 164Cystic fibrosis (CF) is an autosomal recessive disease resulting from mutations in the gene coding for the cystic fibrosis transmembrane conductance regulator (CFTR) (see Chapter 21). CFTR functions as a transmembrane chloride channel in the apical membrane of most secretory epithelia and the disease thus affects lungs, pancreas, exocrine glands, gut, and liver. In CF-associated liver disease the biliary tract is most commonly involved in a spectrum from asymptomatic to biliary cirrhosis. The liver disease runs from mild and su

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Burmer, Karl. CBD Oil for Cirrhosis: Complete Guide to the Treatment of Liver Fibrosis. Independently Published, 2019.

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Shimizu, Hideharu, Tomasz G. Rogula, and Philip R. Schauer. Safety and Efficacy of Bariatric Surgery in Patients with Cirrhosis. Edited by Tomasz Rogula, Philip Schauer, and Tammy Fouse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190608347.003.0021.

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Perioperative risks for morbidly obese patients with cirrhosis are significant, and surgeons should consider these risks carefully in deciding on the type of bariatric procedure to be performed. The benefits of bariatric surgery for cirrhotic patients include substantial weight loss, improvements in metabolic diseases, and potential regression of fibrosis, which can also increase their eligibility and candidacy for liver transplantation. There is currently a lack of strong evidence, but the restrictive bariatric procedures are the safest options for carefully selected patients with cirrhosis.

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Keshav, Satish, and Palak Trivedi. Chronic liver failure. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0210.

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Chronic liver failure is the functional syndrome resulting from cirrhosis. Clinical features of chronic hepatic decompensation include encephalopathy, coagulopathy, and hepatocellular jaundice. Cirrhosis is the final common pathway for a variety of chronic liver diseases and is characterized by fibrosis and the conversion of normal liver architecture into structurally abnormal nodules. There often exists a poor correlation between biopsy findings and the clinical presentation. Some individuals with cirrhosis are asymptomatic and have a reasonably good life expectancy, while others have severe

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Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Fatty liver disease in children. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0055.

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Demographics 406Pathophysiology 406Differential diagnoses 407Presenting features 407Investigation 408Management 409Fatty liver disease is now increasingly recognized in children, particularly in the setting of obesity.The term non-alcoholic steatohepatitis (NASH) was first coined in 1980 by Ludwig to describe a pattern of liver injury in adults in which the liver histology was consistent with alcoholic hepatitis, but in whom significant alcohol consumption was denied. NASH can be considered as part of a broader spectrum of non-alcoholic fatty liver disease that extends from simple steatosis th

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Voilliot, Damien, Jaroslaw D. Kasprzak, and Eduardo Bossone. Diseases with a main influence on right ventricular function. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0060.

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As an important and independent predictive factor of morbidity and mortality, right ventricular (RV) function should be carefully assessed in patients with chronic obstructive lung disease, lung fibrosis, liver cirrhosis, or obesity. RV assessment requires a complete study of the ‘RV-pulmonary circulation unit’ with estimation of RV preload, RV intrinsic contractility, and RV afterload. Therefore, estimation of pulmonary arterial pressure, pulmonary vascular resistance, and left ventricular systolic and diastolic function should be included in this evaluation, in addition to conventional RV sy

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Plotnik, Adam N., and Stephen Kee. Advancing the TIPS Sheath Through a Difficult Cirrhotic Liver: Pay It Forward Off the Balloon. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0076.

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The “pay it forward off the balloon” technique for advancing the transjugular intrahepatic portosystemic shunt (TIPS) sheath may be employed during a difficult TIPS case when the interventionalist has already accessed the portal vein but cannot get the standard 10 Fr TIPS sheath through the fibrotic tract into the portal vein to thereby allow placement of a standard TIPS covered stent. In some patients, the fibrotic recoil of the parenchymal hepatic tract can be so severe that the basic balloon dilation maneuver fails. The pay it forward off the balloon technique employs the use of a 6 mm × 4-

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Book chapters on the topic "Fibrosis and cirrhosis"

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Gonzalez, Raul S., and Kay Washington. "Fibrosis and Cirrhosis." In Non-Neoplastic Liver Pathology. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-31424-2_8.

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Arthur, Michael J. P. "Mechanisms of Progression and Regression of Liver Fibrosis." In Liver Cirrhosis. Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-68343-8_1.

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Rockey, Don C. "Hepatic Fibrosis and Cirrhosis." In Yamada' s Textbook of Gastroenterology. John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118512074.ch105.

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Wells, Rebecca G. "Hepatic Fibrosis and Cirrhosis." In Molecular Pathology Library. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7107-4_30.

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Saffioti, Francesca, and Massimo Pinzani. "Pathogenesis and Evolution of Liver Fibrosis: Cirrhosis or Cirrhoses?" In Complications of Cirrhosis. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13614-1_1.

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Izumi, Namiki, Nobuharu Tamaki, Yasuhiro Asahina, and Masayuki Kurosaki. "Fibrosis and fibrogenesis." In Cirrhosis: A practical guide to management. John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118412640.ch05.

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Ueno, Takato, Toru Nakamura, Hikaru Ueno, Kyuichi Tanikawa, and Michio Sata. "Present Status and Future of Gene Therapy for Hepatic Fibrosis." In Liver Cirrhosis. Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-68343-8_5.

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Kayano, Kozo, Isao Sakaida, and Kiwamu Okita. "Inhibitory Effects of the Herbal Medicine Sho-saiko-to on Liver Fibrosis." In Liver Cirrhosis. Springer Japan, 2001. http://dx.doi.org/10.1007/978-4-431-68343-8_6.

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Rau, Nikhiel B., and Nezam H. Afdhal. "Noninvasive Markers of Fibrosis in the Assessment of Cirrhosis." In Complications of Cirrhosis. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13614-1_5.

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Mehrabi, Sara, Enza Genco, Mariacristina Maturi, and Mirko D’Onofrio. "Chronic Hepatitis and Liver Fibrosis/Cirrhosis." In Imaging of the Liver and Intra-hepatic Biliary Tract. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-38983-3_15.

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Conference papers on the topic "Fibrosis and cirrhosis"

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Sewell-Loftin, M. K., and W. David Merryman. "The Role of SRC in Strain- and Ligand- Dependent Phenotypic Modulation of Mouse Embryonic Fibroblasts." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53604.

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Connective tissue fibrosis represents a significant portion of mortality and morbidity in our society. These diseases include many illnesses such as heart valve disease, atherosclerosis, macular degeneration, and cirrhosis, meaning that millions of lives are affected by these conditions each year. Fibrotic tissues form when quiescent fibroblasts activate becoming myofibroblasts, the phenotype of active tissue construction and fibrosis. During this process, the cells produce smooth muscle α-actin (αSMA), a contractile element considered to be the hallmark of cellular activation [1]. Following t

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Islam, Sibgha, Attique Ur Rehman, Sabeen Javaid, Tahir Muhammad Ali, and Ali Nawaz. "An Integrated Machine Learning Framework for Classification of Cirrhosis, Fibrosis, and Hepatitis." In 2022 Third International Conference on Latest trends in Electrical Engineering and Computing Technologies (INTELLECT). IEEE, 2022. http://dx.doi.org/10.1109/intellect55495.2022.9969404.

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"Hepatoprotective Effect of Typhonium flagelliforme against Thioacetamide Induced Liver Cirrhosis in Rats." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.643.

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Typhonium flagelliforme (T. flagelliforme) was utilized in outdated medication for handling numerous syndromes. This study aimed to investigate hepatoprotection effects of T. flagelliforme against thioacetamide (TAA) hepatotoxicity in rodents.Thirty rodents arbitrarily separated five clusters. Collection 1 was intraperitoneally injected distilled water thrice /week and fed (p.o) daily with 10% Tween 20 to eight weeks. Collection 2-5 i.p. injected with 200 mg/kg TAA three times thrice per week for 8 weeks and fed 10% Tween 20, 50 mg/kg silymarin, 250 and 500 mg/kg of T. flagelliforme extract da

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A. M, Asbelshiny, and Nabeela M. "Attention-Enhanced Residual Networks for Advanced Liver Fibrosis Classification in Deep Learning Models." In International Conference on Modern Trends in Engineering and Management (ICMTEM-25). International Journal of Advanced Trends in Engineering and Management, 2025. https://doi.org/10.59544/fzwf8587/icmtem25p16.

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Liver fibrosis, a precursor to cirrhosis and liver cancer, requires early and accurate classification to improve patient outcomes. Traditional image analysis techniques and conventional deep learning models like U Net face challenges in capturing fine grained features essential for fibrosis staging. To address these limitations, this study introduces an advanced deep learning architecture Attention Enhanced Residual Network (Attention ResNet) designed specifically for liver fibrosis classification. The proposed model integrates improved residual modules within both encoder and decoder paths to

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Roan, Esra, and Kumar Vemaganti. "A Strain Rate Dependent Constitutive Model for Liver Tissue: Experiments and Modeling." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192921.

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The mechanical characterization of liver tissue can be highly beneficial in the management of liver diseases and injuries (1). Liver is the most frequently injured abdominal organ due to injuries that are generally caused by motor vehicle accidents and falls from heights, both of which result in rapid decelerations (2). Moreover, in the US alone, more than 20,300 people die each year as a result of complications arising from chronic liver diseases and the associated fibrosis and cirrhosis.

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Fanous, J., D. Mcnierney, J. Chen, et al. "Multi-Organ Complexities in Telomere Biology Disorders: Liver Cirrhosis, Pulmonary Fibrosis, and Transplantation Challenges." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a5945.

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Evely, R. S., F. E. Preston, D. R. Triger, C. R. M. Hay, M. C. Greves, and J. C. E. Underwood. "TYPE III PRO-COLLAGEN PEPTIDE IN LIVER DISEASE IN HAEMOPHILIA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644022.

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During the past 10 years we have carried out liver biopsies on haemophiliacs with biochemical evidence of chronic liver disease (CLD). To date 44 biopsies have been obtained from 35 patients. Histological diagnoses are Chronic Persistent Hepatitis (CPH) 24, Chronic Aggressive Hepatitis (CAH) 11 and Cirrhosis 9. Serial biopsies indicate that progressive liver disease is now a serious problem in haemophilia. Liver biopsy is not without risk and therefore it is important to identify factors which may be of value in predicting the nature of the liver disease or its progression. Since intra-hepatic

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Nagel, M., A. Arslanow, M. Nguyen-Tat, et al. "SEAL program – Early detection of liver fibrosis and cirrhosis by screening of the general population." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402107.

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Nagel, M., A. Arslanow, M. Nguyen-Tat, et al. "SEAL program - Early detection of liver fibrosis and cirrhosis by screening of the general population." In 37. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0040-1721995.

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Allam, Dalia, Hala Abdalla, and Dalia Allam. "P61 Assessment of clinically significant portal hypertension among periportal fibrosis versus liver cirrhosis patients using fibroscan." In Abstracts of the BSG Annual Meeting, 20–23 June 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-bsg.119.

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Reports on the topic "Fibrosis and cirrhosis"

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Liu, Xiaopei, Dan Liu, and Cong’e Tan. Gut microbiome-based machine learning for diagnostic prediction of liver fibrosis and cirrhosis: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0133.

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Review question / Objective: The invasive liver biopsy is the gold standard for the diagnosis of liver cirrhosis. Other non-invasive diagnostic approaches, have been used as alternatives to liver biopsy, however, these methods cannot identify the pathological grade of the lesion. Recently, studies have shown that gut microbiome-based machine learning can be used as a non-invasive diagnostic approach for liver cirrhosis or fibrosis, while it lacks evidence-based support. Therefore, we performed this systematic review and meta-analysis to evaluate its predictive diagnostic value in liver cirrhos

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Jin, Dachuan, Gao Peng, Shunqin Jin, Tao Zhou, Baoqiang Guo, and Guangming Li. Comparison of therapeutic effects of anti-diabetic drugs on non-alcoholic fatty liver disease patients without diabetes: A network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0014.

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Review question / Objective: To evaluate the efficacy of different anti-diabetic drugs in the treatment of non-diabetic non-alcoholic disease by network meta-analysis, and find the best intervention. Condition being studied: Non-alcoholic fatty liver disease (NAFLD) refers to the disease in which the liver fat content exceeds 5%, and excludes the secondary causes of alcohol, infection, drugs or other specific metabolic diseases. As a spectrum of disorders, it includes hepatocyte steatosis and steatohepatitis at the initial stage, liver fibrosis at the later stage, cirrhosis at the final stage,

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Sowjanya, Dr Kaniti, Dr Bongu Srinivas, and Dr Metta Lakshmana Rao. A STUDY ON FIBROSCAN COMPARED TO AST TO PLATELET RATIO INDEX(APRI) FOR ASSESSMENT OF LIVER FIBROSIS WITH NONALCOHOLIC FATTY LIVER DISEASE(NAFLD). World Wide Journals, 2023. http://dx.doi.org/10.36106/ijar/1606016.

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Background : Nonalcoholic fatty liver disease (NAFLD) is increasingly recognized as a cause of chronic liver disease, and often results in the devastating outcomes of decompensated liver cirrhosis and hepatocellular carcinoma and is an important feature of metabolic syndromes and insulin resistance. The assessment of liver brosis is essential for predicting the prognosis and outcome of all forms of chronic liver disease. A liver biopsy is the gold standard for the assessment of liver brosis, but it has its limitations, which include life-threatening complications. Alternative methods of non-

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