Relevant bibliographies by topics / Foam inhibitors

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Foam inhibitors'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Foam inhibitors"

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Ang, Justin K., and Charles W. Bamforth. "Foam inhibitors from specialty malts." Journal of the Institute of Brewing 120, no. 3 (2014): 193–200. http://dx.doi.org/10.1002/jib.141.

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Zhang, Xun, Jing Yang, Pengfei Xie, Hao Liu, Qiang Deng, and Fengwei Dai. "Experimental study on controlled-release inhibitor foam for restraining spontaneous combustion of coal." Energy Exploration & Exploitation 38, no. 4 (2020): 1159–77. http://dx.doi.org/10.1177/0144598720910266.

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A new method is proposed based on temperature-controlled self-reaction to generate and release inhibitors in the form of foam at a specific temperature, which can overcome the disadvantages of short effective time and low efficiency in the inhibition of the spontaneous combustion of coal when inhibitors are released in advance, and greatly increase the action range of inhibitor through foam diffusion. The proposed temperature-controlled foaming system was prepared with hollow spheres as solution carriers, NaHCO3 and acetic acid as basic reactants, reaction-generated CO2 as foaming gas, sodium
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Tang, Chao-Ke, Guo-Hua Tang, Guang-Hui Yi, et al. "Effect of Apolipoprotein A-I on ATP Binding Cassette Transporter A1 Degradation and Cholesterol Efflux in THP-1 Macrophage-derived Foam Cells." Acta Biochimica et Biophysica Sinica 36, no. 3 (2004): 218–26. http://dx.doi.org/10.1093/abbs/36.3.218.

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Abstract Cholesterol-loaded macrophage foam cells are a central component of atherosclerotic lesions. ATP binding cassette transporter A1 (ABCA1), the defective molecule in Tangier disease, mediates the efflux of phospholipid and cholesterol from cells to apolipoprotein A-I (apoA-I), reversing foam cell formation. This study investigated the effect of apoA-I on ABCA1 degradation and cholesterol efflux in THP-1 macrophage-derived foam cells. After exposure of the cultured THP-1 macrophage-derived foam cells to apoA-I for different time, cholesterol efflux, ABCA1 mRNA and protein levels were det
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Ritchie, Helen, Alec Jamieson, and Nuala A. Booth. "Regulation, Location and Activity of Plasminogen Activator Inhibitor 2 (PAI-2) in Peripheral Blood Monocytes, Macrophages and Foam Cells." Thrombosis and Haemostasis 77, no. 06 (1997): 1168–73. http://dx.doi.org/10.1055/s-0038-1656132.

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SummaryMonocytes, macrophages and foam cells are central to atherogenesis. We have examined the potential ability of monocytes, macrophages and foam cells to affect the stability of deposited fibrin, characteristic of the atherosclerotic plaque, by their production of plasminogen activators and their inhibitors. Monocytes respond to thrombin and LPS by up-regulation of PAI-2 synthesis, and PAI-2 is their major product among the plasminogen activators/inhibitors. In contrast, macrophages and foam cells, while they did produce PAI-2, did not respond to thrombin and LPS by an increase in its synt
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Lantz, Sean, Jack Zakarian, Scott Deskin, and Ashlie Martini. "Filtration Effects on Foam Inhibitors and Optically Detected Oil Cleanliness." Tribology Transactions 60, no. 6 (2017): 1159–64. http://dx.doi.org/10.1080/10402004.2017.1285089.

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Hejna, Aleksander. "Clays as Inhibitors of Polyurethane Foams’ Flammability." Materials 14, no. 17 (2021): 4826. http://dx.doi.org/10.3390/ma14174826.

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Polyurethanes are a very important group of polymers with an extensive range of applications in different branches of industry. In the form of foams, they are mainly used in bedding, furniture, building, construction, and automotive sectors. Due to human safety reasons, these applications require an appropriate level of flame retardance, often required by various law regulations. Nevertheless, without the proper modifications, polyurethane foams are easily ignitable, highly flammable, and generate an enormous amount of smoke during combustion. Therefore, proper modifications or additives shoul
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Terasaki, Michishige, Hironori Yashima, Yusaku Mori, et al. "A Dipeptidyl Peptidase-4 Inhibitor Inhibits Foam Cell Formation of Macrophages in Type 1 Diabetes via Suppression of CD36 and ACAT-1 Expression." International Journal of Molecular Sciences 21, no. 13 (2020): 4811. http://dx.doi.org/10.3390/ijms21134811.

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Dipeptidyl peptidase-4 (DPP-4) inhibitors have been reported to play a protective role against atherosclerosis in both animal models and patients with type 2 diabetes (T2D). However, since T2D is associated with dyslipidemia, hypertension and insulin resistance, part of which are ameliorated by DPP-4 inhibitors, it remains unclear whether DPP-4 inhibitors could have anti-atherosclerotic properties directly by attenuating the harmful effects of hyperglycemia. Therefore, we examined whether a DPP-4 inhibitor, teneligliptin, could suppress oxidized low-density lipoprotein (ox-LDL) uptake, foam ce
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Keewan, Mohammad, Fawzi Banat, Priyabrata Pal, Jerina Zain, and Emad Alhseinat. "Foaming of industrial lean methyldiethanolamine solution in the presence of hydrocarbon and fatty acid based corrosion inhibitors." Oil & Gas Science and Technology – Revue d’IFP Energies nouvelles 73 (2018): 76. http://dx.doi.org/10.2516/ogst/2018073.

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In natural gas sweetening alkanolamine processes one of the regularly used chemical is the corrosion inhibitor. For better operation of the plant it is essential to understand the effect of their presence on foaming of industrial lean Methyldiethanolamine (MDEA) used as solvents at different temperatures. This study aimed at investigating the effect of HydroCarbon Based (HCB) and fatty acid based corrosion inhibitor having chemical name Bis(2-Hydroxyethyl)Cocoalkylamine (BHCL) on the foaming tendency of industrial real lean MDEA solutions. Experiments were conducted with different operating pa
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Plotkin, Jesse D., Michael G. Elias, Anthony L. Dellinger та Christopher L. Kepley. "NF-κB inhibitors that prevent foam cell formation and atherosclerotic plaque accumulation". Nanomedicine: Nanotechnology, Biology and Medicine 13, № 6 (2017): 2037–48. http://dx.doi.org/10.1016/j.nano.2017.04.013.

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Terasaki, Michishige, Munenori Hiromura, Yusaku Mori, et al. "A Dipeptidyl Peptidase-4 Inhibitor Suppresses Macrophage Foam Cell Formation in Diabetic db/db Mice and Type 2 Diabetes Patients." International Journal of Endocrinology 2018 (December 9, 2018): 1–9. http://dx.doi.org/10.1155/2018/8458304.

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Dipeptidyl peptidase-4 (DPP-4) inhibitors could have antiatherosclerotic action, in addition to antihyperglycemic roles. Because macrophage foam cells are key components of atherosclerosis, we investigated the effect of the DPP-4 inhibitor teneligliptin on foam cell formation and its related gene expression levels in macrophages extracted from diabetic db/db (C57BLKS/J Iar -+Leprdb/+Leprdb) mice and type 2 diabetes (T2D) patients ex vivo. We incubated mouse peritoneal macrophages and human monocyte-derived macrophages differentiated by 7-day culture with oxidized low-density lipoprotein in the
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Dissertations / Theses on the topic "Foam inhibitors"

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DeLozier, Greg. "Part 1: Employing conventional defoamer emulsions to enhance the flotation removal of flexographic news inks." Diss., Available online, Georgia Institute of Technology, 2005, 2003. http://etd.gatech.edu/theses/available/ipstetd-1016/.

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Pritchard-Jones, Rowan. "Expression of a novel inhibitory form of VEGF in skin and skin cancer." Thesis, University of Bristol, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.435867.

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Wang, Min. "Identification of molecular cloning of trypsin inhibitor form the skins of oriental ranid frogs." Thesis, Queen's University Belfast, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534666.

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Nagel, Jutta Maria. "Modulation des metastatischen Potentials einer humanen Fibrosarkomzelllinie durch Ko-Expression von TIMP-1 (tissue inhibitor of metalloproteinases-1) und einer löslichen Form des Urokinase-Rezeptors." [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=974167282.

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Saiki, Masaaki. "Adenovirus-mediated gene transfer of a truncated form of fibroblast growth factor receptor inhibits growth of glioma cells both in vitro and in vivo." Kyoto University, 2000. http://hdl.handle.net/2433/151415.

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Rondeau, Eric. "Les activateurs du plasminogene du rein humain : identification, localisation, facteurs de secretion." Paris 6, 1987. http://www.theses.fr/1987PA066607.

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Les glomerules isoles du cortex renal chez l'homme, contiennent des activateurs du plasminogene. L'activateur tissulaire du plasminogene (t-pa) represente 80% de l'activite profibrinolytique et l'eurokinase 20% de cette activite. Les cellules epitheliales glomerulaires en culture contiennent l'urokinase. L'activite profibrinolytique des surnageant glomerulaires augmente lorsque les glomerules sont incubes en presence du calcium et a ph alcalin. Les acides gras polyinsatures (acide arachidonique, acide eicosopentaenoique ou acide eicosatrienoique) augmentent cette activite alors que les acides
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Tsakanikas, Loukia. "Normal form analysis of a mean-field inhibitory neuron model." Thesis, 2012. http://spectrum.library.concordia.ca/974722/1/Tsakanikas_MSC_F2012.pdf.

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In neuroscience one of the open problems is the creation of the alpha rhythm detected by the electroencephalogram (EEG). One hypothesis is that the alpha rhythm is created by the inhibitory neurons only. The mesoscopic approach to understand the brain is the most appropriate to mathematically modelize the EEG records of the human scalp. In this thesis we use a local, mean-field potential model restricted to the inhibitory neuron population only to reproduce the alpha rhythm. We perform extensive bifurcation analysis of the system using AUTO.We use Kuznetsov’s method that combines the ce
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Xie, Zheng-Ming, and 謝正明. "Inhibitory effects of Laminaria japonica fermented product on low density lipoprotein oxidation and foam cell formation." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/8t53vr.

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碩士<br>國立臺灣海洋大學<br>生命科學暨生物科技學系<br>107<br>In this experiment, we investigated the difference between Laminaria japonica water extract (JWE) and Laminaria japonica fermented product (JFP) on functional groups, inhibition of low density lipoprotein (LDL) oxidation and inhibition of foam cell formation using the model of oxidized low density lipoprotein (oxLDL)-induced macrophage. Results exhibited that JFP had better inhibition of copper ion-induced LDL oxidation than JWE. In cell viability experiment, it was showed that JFP increased cell viabilities in the concentration range of 10 to 150 μg/ml,
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Li, Yu-Hsuan, and 李佑軒. "Antioxidant and anti-inflammatory activities of several edible and medicinal mushrooms and their inhibitory ability on foam cell formation." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/83441685437776319690.

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碩士<br>國立中興大學<br>食品暨應用生物科技學系所<br>103<br>Appropriate inflammatory responses might protect human body from diseases, but excessive inflammation may be harmful to cells and organs leading to many chronic diseases. ROS plays an important role in oxidative stress and modulates the secretion of inflammatory mediators. The formation of foam cells plays an important role in the development of atherosclerosis process. Edible and medicinal mushrooms contain various kinds of bioactive compounds, presenting multiple bioactivities. Thus, mushrooms are good sources to be used for health foods. In this stud
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Novotná, Eliška. "Vliv foam rollingu na mechano-nociceptivní a vibrotaktilní čití." Master's thesis, 2019. http://www.nusl.cz/ntk/nusl-435650.

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The thesis deals with the evaluation of the influence of foam rolling on selected parameters of somatosensory perception (pressure-algic threshold, vibrotactile sensation). The theoretical part discusses fascial tissue, summarizes the current knowledge of foam rolling and focuses on the mechanisms of pain and vibrotactile sensation. The research part consists of a randomized blind study on a group of 15 healthy probands (11 women, 4 men), which evaluates the effect of foam rolling. The effect is objectivized by pressure algometry and vibrametry on the m. rectus femoris and m. biceps femoris of
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Books on the topic "Foam inhibitors"

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Afford, Simon C. The form and function of ling alpha proteinase inhibitor in bronchoalveolar lavage fluid from healthy individuals. University of Birmingham, 1988.

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Eisen, Tim. The patient with renal cell cancer. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0172.

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Renal cancer is the commonest malignancy of the kidney and worldwide, accounts for between 2% and 3% of the total cancer burden. The mainstay of curative treatment remains surgery. There have been significant advances in surgical technique, the most important ones being nephron-sparing surgery and laparoscopic nephrectomy. The medical treatment of advanced renal cell cancer has only improved markedly in the last decade with the development of antiangiogenic tyrosine-kinase inhibitors, inhibitors of mammalian target of rapamycin, and a diminished role for immunotherapy.Tyrosine-kinase inhibitor
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Naked Drunk And Writing Shed Your Inhibitions And Craft A Compelling Memoir Or Personal Essay. Ten Speed Press, 2010.

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Bhole, Malini. Complement deficiencies. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0299.

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The complement system comprises a group of heat-labile proteins which form part of the innate immune system. The main physiological functions of the complement system include defence against pyogenic bacterial infections, clearance of immune complexes and products of inflammatory damage, and acting as a bridge between the innate and adaptive immune system. The complement system is regulated by various complement inhibitors (regulatory proteins) that are present in both the classical pathway and the alternate pathway and which regulate and prevent spontaneous activation of the complement system
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Volberda, Henk, Frans van den Bosch, and Kevin Heij. Introduction. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198792048.003.0001.

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Chapter 1 starts with the case of Kodak and the development of the electronic still camera to illustrate the pivotal importance of business model innovation. This opening chapter looks at why business model innovation is needed, sets out the research model and key research questions, and outlines the main elements to be discussed in subsequent chapters. These include: the changing competitive environment; business model innovation strategies; levers of business model innovation; catalysts and inhibitors in business model innovation; and competitive advantages of new business models. These elem
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Krueger, Darcy A., and Jamie Capal. Familial CNS Tumor Syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0136.

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Tuberous sclerosis complex is an autosomal dominant multi-system disease that involves the skin, brain, heart, lungs, and kidneys and is associated with seizures including infantile spasms, intellectual disability, autism and pulmonary and heart disease. Skin lesions can be particularly disfiguring and infantile spasms can be associated with marked cognitive decline. The outlook for patients has improved markedly with the recognition that TSC is caused by upregulation of the mammalian target of rapamycin (mTOR) enzyme, which connects energy needs and supply with cellular and neuronal growth. m
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Leung, Doris G. Other Proven and Putative Autoimmune Disorders of the Peripheral Nervous System. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0098.

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Myasthenia gravis is in most cases an autoimmune disorder of the neuromuscular junction in which antibodies are directed at nicotinic acetylcholine receptors or other synaptic proteins, such as the MusK protein that is involved in the formation of the formation and maturation of the motor endplate. Less commonly, myasthenia gravis can result from antibodies directed to presynaptic calcium channels as a side effect of paraneoplastic antibodies (Lambert-Eaton syndrome) or from a developmental paucity of acetylcholine receptors in the neonatal form of the disease. Treatment is usually a combinati
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Phillips, Katharine A. Insight and Delusional Beliefs in Body Dysmorphic Disorder. Edited by Katharine A. Phillips. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190254131.003.0009.

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This chapter discusses insight (“delusionality”) in body dysmorphic disorder (BDD). BDD beliefs span a broad range of insight, from good to absent insight (i.e., delusional beliefs). About 70% of patients have poor or absent insight. Early emerging clues suggest possible neurobiologic bases of poorer insight in BDD. BDD’s delusional form (characterized by the absence of insight) appears to be the same disorder as its nondelusional form rather than a separate psychotic disorder. Consistent with this, serotonin-reuptake inhibitor (SRI) monotherapy is efficacious for delusional BDD as well as non
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Deramecourt, Vincent, Florence Lebert, and Florence Pasquier. Frontotemporal dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0036.

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Frontotemporal dementia (FTD) is the second most common form of dementia in persons younger than 65 years after Alzheimer’s disease. The FTD spectrum is characterized by clinical, molecular and genetic heterogeneity. Core features of FTD are behavioural and language manifestations and the clinical spectrum of FTD currently includes a behavioural variant, progressive nonfluent aphasia and semantic dementia. The most common behavioural features are disinhibition, apathy, loss of empathy, hyperorality and perseveration. Neuroimaging usually demonstrates focal atrophy and hypometabolism in the ant
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Roze, Emmanuel, and Nenad Blau. Biogenic Monoamine Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0031.

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Biogenic monoamine disorders are a group of inherited diseases characterized by a defect in the synthesis, transport, or degradation of catecholamines and serotonin. The phenotype mostly reflects the pattern and severity of the monoamine deficiency. Movement disorders due to cerebral dopamine deficiency are almost always prominent, mostly in the form of dystonia and/or parkinsonism. These disorders are potentially devastating yet treatable. Early diagnosis and treatment are crucial to prevent ongoing brain dysfunction. Detection of hyperphenylalaninemia in a neonate could be a good clue to the
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Book chapters on the topic "Foam inhibitors"

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Darby, Ian A., and Alexis Desmoulière. "Scar Formation: Cellular Mechanisms." In Textbook on Scar Management. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_3.

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AbstractFibroblasts are key players in the maintenance of skin homeostasis and in orchestrating physiological tissue repair. Fibroblasts secrete and are embedded in a sophisticated extracellular matrix, and a complex and interactive dialogue exists between fibroblasts and their microenvironment. In addition to the secretion of the extracellular matrix, fibroblasts and myofibroblasts secrete extracellular matrix remodeling enzymes, matrix metalloproteinases and their inhibitors, and tissue inhibitors of metalloproteinases and are thus able to remodel the extracellular matrix. Myofibroblasts and their microenvironment form a network that evolves during tissue repair. This network has reciprocal actions affecting cell differentiation, cell proliferation, cell quiescence, or apoptosis and has actions on growth factor bioavailability by binding, sequestration, and activation. Mechanical forces also play a role in regulating the myofibroblast phenotype as cells are subjected to mechanical stress and mechanical signaling is activated. Innervation is also involved in both skin repair processes and differentiation of myofibroblasts. In pathological situations, for example, in excessive scarring, the dialogue between myofibroblasts and their microenvironment can be altered or disrupted, leading to defects in tissue repair or to pathological scarring, such as that seen in hypertrophic scars. Better understanding of the intimate dialogue between myofibroblasts and their local microenvironment is needed and will be important in aiding the identification of new therapeutic targets and discovery of new drugs to treat or prevent aberrant tissue repair and scarring.
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Staudt, H., D. Kling, and M. Nordlander. "Inhibitory effects of the Ca2+-antagonist Felodipine on the accumulation of monocytes and their transformation into foam cells in early atherosclerotic lesions." In Arteriosklerotische Gefäßerkrankungen. Vieweg+Teubner Verlag, 1992. http://dx.doi.org/10.1007/978-3-663-19646-4_44.

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Gerbixing, K. P., M. Steup, and E. Latzko. "Chelates of Fructose 1,6-Bisphosphate-Ions Function as Substrates and Free Fructose 1,6-Bisphosphate-Ions as Inhibitors of Fructose 1,6-Bisphosphatase Form B from Synechococcus Leopoliensis." In Progress in Photosynthesis Research. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-017-0516-5_100.

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Urano, Tetsumei, Kiyohito Serizawa, Kenji Sakakibara, et al. "The Cleavage of a Serine Protease Inhibitor at the Reactive Center by Its Target Protease: Analysis of the Substrate-Like Form of a Serpin." In Current Aspects of Blood Coagulation, Fibrinolysis, and Platelets. Springer Japan, 1993. http://dx.doi.org/10.1007/978-4-431-68323-0_5.

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Botkin, James H. "Scorch Inhibitors for Polyurethane Slabstock Foams." In Advances in Urethane Science and Technology. CRC Press, 2020. http://dx.doi.org/10.1201/9780367811679-3.

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Dewangan, Y. "Polysaccharide as Green Corrosion Inhibitor." In Sustainable Corrosion Inhibitors. Materials Research Forum LLC, 2021. http://dx.doi.org/10.21741/9781644901496-4.

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The carbohydrates associated with polysaccharide glycosidic bonds are tightly chained, usually linear and highly branched complex molecules. Their structure mainly consists of hydroxyl groups in the form of functional groups, in which an oxygen heterogeneous atom is present. Some polysaccharides have hetero atoms. Nitrogen and Sulfur in addition to oxygen, which have unshared electron pairs. Hetero atoms easily share their electron pair to the vacant d orbitals of the metal ion and prevent the metal from corrosion. Polysaccharides are biodegradable, renewable, inexpensive and environment friendly due to which they are easily used as corrosion inhibitors. The present study mentions some major research work in which polysaccharides are used as corrosion inhibitors. Their mixed type nature has been reported in most research papers, and in the case of steel metal, they mainly follow the Langmuir adsorption isotherm. Chemical (gravimetric analysis) and electrochemical (EIS &amp; PDP) studies are frequently used for the corrosion inhibition study. Some of the current research papers have also used computational or theoretical studies such as quantum chemical study and MD simulation. At the end of this book chapter, a discussion is also given regarding further research and direction related to the topic.
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Bourdon, Allen K., Greg Villareal, George Perry, and Clyde F. Phelix. "Alzheimer's and Parkinson's Disease Novel Therapeutic Target." In Research Anthology on Diagnosing and Treating Neurocognitive Disorders. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-3441-0.ch021.

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Thiazolidinedione (TZD) drugs (Takeda Pharmaceuticals and Metabolic Solutions Development Company) targeting inhibition of the mitochondrial pyruvate carrier (MPC) are currently being tested in clinical trials to prevent progression into mild cognitive impairment of Alzheimer's disease (AD) or in the pipeline to prevent neurodegeneration in Parkinson's disease (PD). These have Ki values in the µM range. This study was focused on identifying candidate drug precursors of the natural cinnamic acid products that might have good bioavailability in the nM ranges forming covalent thiol bonds with targets. In silico protein homology modeling and ligand docking has demonstrated that binding cysteine residues within the transport channel is a key part of the inhibitory mechanism. These are covalent thiohemiacetal bonds with the alpha-carbon, carboxylate group, off a phenol ring. Like the classic MPC inhibitors, these natural derivatives of hydroxycinnamic acid have a conjugated pi-system used to form thiol bonds with the cysteine residue via Michael addition.
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Ramesh, M. "Case-Studies on Green Corrosion Inhibitors." In Sustainable Corrosion Inhibitors. Materials Research Forum LLC, 2021. http://dx.doi.org/10.21741/9781644901496-9.

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Corrosion in metals and its alloys is an inevitable phenomenon but can be controlled by suitable classical methods like process control, cathode protection, surface treating methods, impurity reduction in metals and addition of metals to form alloys. Nevertheless, the employment of corrosion inhibitors is still a noteworthy and simplest of all the above processes in protecting the metals and alloys especially in acidic media. Protection of metals against corrosion not only prevents corrosion but also is beneficial in terms of money loss as far as industrial equipment, surfaces and vessels are concerned. Since the use of organic and inorganic inhibitors are highly discouraged due to their high cost and toxicity, necessity has adequately aroused the development of corrosion inhibitors which are natural and green. Trends, nowadays, focussed in controlling corrosion in various metals and alloys through green corrosion inhibitors consisting of natural elements alone. In contrast to the inorganic inhibitors, green corrosion inhibitors are characterized by biodegradability, low cost and meagre toxicity. Several researchers are now turning themselves towards the research of green inhibitors which are of no threat to humans and the ecosystem. The current discussion is focussed on the fundamentals of corrosion, corrosion inhibition, materials used for it and case studies of green inhibitors used for corrosion control in various conventional and monolithic metals.
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Roberts, Harold R., and Gilbert C. White. "The biology of haemostasis and thrombosis." In Oxford Textbook of Medicine. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.220601.

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Haemostasis—a component of the wound defence mechanism—is a process by which vessel wall components and platelets act in concert with procoagulant and anticoagulant proteins to form a plug of cells and cross-linked fibrin. The plug is later remodelled and replaced by new tissue as part of wound healing. These processes are very complex and involve highly controlled pathways of interaction between cells, glycans, and membrane-bound and soluble proteins of coagulation and fibrinolysis, as well as their cognate inhibitors....
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M. Gordon, Erlinda, Nicole L. Angel, Ted T. Kim, Don A. Brigham, Sant P. Chawla, and Frederick L. Hall. "Immune and Cell Cycle Checkpoint Inhibitors for Cancer Immunotherapy." In Advances in Precision Medicine Oncology. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96664.

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The rational design of immunotherapeutic agents has advanced with a fundamental understanding that both innate and adaptive immunity play important roles in cancer surveillance and tumor destruction; given that oncogenesis occurs and cancer progresses through the growth of tumor cells with low immunogenicity in an increasingly immunosuppressive tumor microenvironment. Checkpoint inhibitors in the form of monoclonal antibodies that block cancer’s ability to deactivate and evade the immune system have been widely indicated for a variety of tumor types. Through targeting the biological mechanisms and pathways that cancer cells use to interact with and suppress the immune system, immunotherapeutic agents have achieved success in inhibiting tumor growth while eliciting lesser toxicities, compared to treatments with standard chemotherapy. Development of “precise” bio-active tumor-targeted gene vectors, biotechnologies, and reagents has also advanced. This chapter presents ongoing clinical research involving immune checkpoint inhibitors, while addressing the clinical potential for tumor-targeted gene blockade in combination with tumor-targeted cytokine delivery, in patients with advanced metastatic disease, providing strategic clinical approaches to precision cancer immunotherapy.
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Conference papers on the topic "Foam inhibitors"

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Watson, Duncan K., Musaffar A. Khan, and M. Anas Qureshi. "Foam Sticks Application to Increase Production and Encapsulated Inhibitors to Mitigate Corrosion and Scale Formation." In SPE/PAPG Annual Technical Conference. Society of Petroleum Engineers, 2011. http://dx.doi.org/10.2118/156213-ms.

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Cassani, F., P. Ortega, A. Davila, W. Rodriguez, and J. Seranno. "Evaluation of Foam Inhibitors at the Jusepin Oil/Gas Separation Plant, El Furrial Field, Eastern Venezuela." In SPE Latin America Petroleum Engineering Conference. Society of Petroleum Engineers, 1992. http://dx.doi.org/10.2118/23681-ms.

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Carrell, R. W., P. D. Christey, and D. R. Boswell. "SERPINS: ANTITHROMBIN AND OTHER INHIBITORS OF COAGULATION AND FIBRINOLYSIS. EVIDENCE FROM AMINO ACID SEQUENCES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642896.

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A number of the key inhibitors of coagulation and fibrinolysis have recently been shown to be members of the same superfamily of serine protease inhibitors, the serpins. The archetypes of the group are alpha-l-antitrypsin and antithrombin and it includes antiplasmin, C1-inhibitor, heparin cofactor II and the newly recognised inhibitors of plasminogen activators and activated Protein C. Alignment of their structures shows that they have the same skeletal three-dimensional conformation and, by inference, the same general function mechanisms.The serpins have a reactive centre, primarily dependent
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Baker, J. B., M. P. McGrogan, C. Simonsen, R. L. Gronke, and B. W. Festoff. "STRUCTURE AND PROPERTIES OF PROTEASE NEXIN I." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644765.

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Human foreskin fibroblasts secrete several different serine protease inhibitors which differ in size and protease specificities. These proteins, called protease nexins (PNs) all form SDS-resistant complexes with their protease targets. Fibroblast surface receptors recognize the protease-PN complexes and mediate their delivery to lysosomes. PNI is a 45 kilodalton glycoprotein that rapidly inhibits several arg or lys-specific proteases including trypsin, thrombin, and urokinase (k assoc.∼ 4×l06,∼ 6×105 and ∼ 2×105, m−1s−1 respectively). Like antithrombin III, PNI binds heparin and inhibits throm
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Suzuki, Koji, Yoshihiro Deyashiki, Junji Nishioka, Kazunori Toma, and Shuji Yamamoto. "THE INHIBITOR OF ACTIVATED PROTEIN C: STRUCTURE AND FUNCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642963.

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In the final step of protein C pathway, activated protein C (APC) is neutralized with a plasma inhibitor, termed protein C inhibitor (PCI). PCI was first described by Marlar and Griffin (1980) and then isolated from human plasma as a homogeneous form and characterized by the authors (1983). PCI is a single chain glycoprotein with M 57,000 and a plasma concentration of 5 ug/ml. Analysis of a cDNA nucleotide sequence has clarified that a precursor of human PCI consists of a mature protein of 387 amino acid residues (M 43,759) and a signal peptide of 19 amino acid residues. Only one cysteine resi
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de Agostini, A., F. Barja, S. Carrel, P. C. Harpel, and M. Schapira. "C1 -INHIBITOR: STRUCTURE-ACTIVITY RELATIONSHIPS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642903.

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Cl-inhibitor [C1 -In] and other protease inhibitors of the serpin superfamily inactivate serine proteases by forming bimolecular enzyme-inhibitor complexes, a reaction that is associated with changes in the inhibitor conformation. To determine the significance of these changes, we have examined the influence of various treatments on the binding to C1-In of monoclonal antibody 4C3. This antibody was previously shown to bind to an epitope created during the reaction of C1-In with the Arg-specific protease plasma kallikrein [K]: the site for 4C3 was expressed on the K-C1-In complex, on C1-In clea
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Hadsbjerg, Casper, and Kristian Krejbjerg. "Challenges in Hydrate Plug Prevention in Pipelines Seen Over the Lifetime of a Field." In ASME 2009 28th International Conference on Ocean, Offshore and Arctic Engineering. ASMEDC, 2009. http://dx.doi.org/10.1115/omae2009-79446.

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When the oil and gas industry explores subsea resources in remote areas and at high water depths, it is important to have advanced simulation tools available in order to assess the risks associated with these expensive projects. A major issue is whether hydrates will form when the hydrocarbons are transported to shore in subsea pipelines, since the formation of a hydrate plug might shut down a pipeline for an extended period of time, leading to severe losses. The industry practices a conservative approach to hydrate plug prevention, which is the addition of inhibitors to ensure that hydrates c
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Yue, Zhiwei, Qiang Fu, Nancy Lang, and Chunfang Fan. "Liquid Scale Inhibitors for Metallic-Crosslinked Gel Fracturing Systems." In SPE International Oilfield Scale Conference and Exhibition. SPE, 2014. http://dx.doi.org/10.2118/spe-169806-ms.

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Abstract Scale inhibitors are important additives in fracturing fluids to help prevent mineral scale depositions during hydraulic fracturing, shut-in, and flowback stages. The inhibition mechanisms rely heavily on the interactions of certain functional groups from the inhibitor molecules and the lattice metals on the scale crystal surface. In stimulation using crosslinked gel fluids based on metallic crosslinkers, such as zirconium (Zr), titanium (Ti), or aluminum (Al), these metals form strong covalent bonds with guar and guar derivatives and therefore significantly increase overall gel stabi
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Loskutoff, D. J., J. Mimuro, and C. Hekman. "PLASMINOGEN ACTIVATOR INHIBITOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644763.

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Plasminogen activation provides an important source of localized proteolytic activity not only during fibrinolysis, but also during ovulation, cell migration, epithelial cell differentiation, tumor invasion and a variety of other physiological processes. Precise regulation of plasminogen activator (PA) activity thus constitutes a critical feature of many biological processes. This control is achieved in large part through the action of specific PA inhibitors (PAIs). Although 4 distinct PAIs have been detected,1the endothelial cellTderived inhibitor (PAI-1) is the only one that efficiently inhi
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Ny, T., L. Hansson, and B. Åstedt. "ISOLATION OF cDNA FOR TYPE-2 PLASMINOGEN ACTIVATOR INHIBITOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642855.

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The placental type plasminogen activator inhibitor (PAI-2) has been purified from extracts of human placenta and from a histiocytic lymphoma cell line. It is mainly an uPA inhibitor but it also inhibits the two-chain form of tPA.In order to determine the factors regulating PAI-2 gene expression and thereby clarify the physiological role of PAI-2 we have undertaken the molecular cloning of PAI-2 cDNA. A λgt11 expression library prepared from placental mRNA, was screened, immunologically using a monoclonal antibody probe developed against PAI-2 purified from human placenta. When 1.7×105 recombin
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