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Journal articles on the topic 'Fomepizol'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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1

. "Fomepizol als antidotum bij de behandeling van ethyleenglycolvergiftiging." Medisch-Farmaceutische Mededelingen 39, no. 3 (2001): 55–56. http://dx.doi.org/10.1007/bf03057688.

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2

Siekmeyer, Werner, Hatmut Neurath, Wieland Kiess, Herbert Desel, and Nicolaus Schwerk. "Diagnostik und Therapie von Glykolintoxikationen im Kindesalter." Kinder- und Jugendmedizin 06, no. 01 (2006): 33–37. http://dx.doi.org/10.1055/s-0037-1617887.

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ZusammenfassungGlykolintoxikationen stellen ein seltenes, akut lebensbedrohliches Krankheitsbild dar. Klinisch stehen neben den neurologischen Symptomen eine ausgeprägte metabolische Azidose mit großer Anionenlücke sowie eine große osmotische Lücke im Vordergrund. Beweisend ist der Nachweis von Glykol bzw. seiner Metabolite im Blut, jedoch verfügen nur wenige Zentren über die Möglichkeit, Glykol qualitativ bzw. quantitativ im Serum zu bestimmen. Die Diagnose muss deshalb zunächst klinisch gestellt werden. Allein der Verdacht erfordert die sofortige Einleitung einer adäquaten Therapie mit einem Antidot (Fomepizol oder Ethanol). Dieser Artikel soll dazu dienen, die typischen klinischen und paraklinischen Befunde sowie die Therapie von Glykolintoxikationen ausführlich darzustellen.

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3

Ringler, Sandra, Roman Gmuer, Katrin Faber, Jörg Bleisch, and Simon Andreas Müggler. "CME: Ethylenglykol-Intoxikation." Praxis 107, no. 20 (2018): 1097–106. http://dx.doi.org/10.1024/1661-8157/a003071.

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Zusammenfassung. Ethylenglykol ist ein süsslich schmeckender Alkohol (einfachster zweiwertiger Alkohol, Synonym «Glykol»), der in handelsüblichen Frostschutzmitteln und anderen industriellen Lösungen verwendet wird. Ohne adäquate Therapiemassnahmen kann eine Intoxikation mit Ethylenglykol zu einer schweren metabolischen Azidose, zum akuten Nierenversagen und zum Tod führen. Nach gastrointestinaler Resorption findet die Metabolisierung überwiegend hepatisch, unter anderem durch die Alkoholdehydrogenase, statt und resultiert in einer schweren metabolischen Azidose mit Anionenlücke und der Bildung von Kalziumoxalat-Kristallen, die sich in verschiedenen Geweben ablagern können. In der Niere führt dies zur akuten Tubulusnekrose mit potenziell reversiblem Nierenversagen. Therapeutisch entscheidend ist die schnellstmögliche Inhibition der Alkoholdehydrogenase durch Fomepizol oder Ethanol, um die Bildung toxischer Metaboliten zu verhindern. Die Hämodialyse bietet die effektivste Methode der sekundären Dekontamination. Bei frühem Therapiebeginn ist die Prognose günstig.

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4

&NA;. "Fomepizole." Dimensions of Critical Care Nursing 20, no. 5 (2001): 29. http://dx.doi.org/10.1097/00003465-200109000-00007.

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5

Hovda, KE, and D. Jacobsen. "Expert opinion: fomepizole may ameliorate the need for hemodialysis in methanol poisoning." Human & Experimental Toxicology 27, no. 7 (2008): 539–46. http://dx.doi.org/10.1177/0960327108095992.

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Fomepizole is now the antidote of choice in methanol poisoning. The use of fomepizole may also change the indications for hemodialysis in these patients. We have addressed this change in a review of articles on methanol poisonings. Review of the literature (through PubMed®) combined with our own experiences from two recent methanol outbreaks in Estonia and Norway. The efficiency of dialysis during fomepizole treatment was reported in only a few reports. One recent study challenged the old indications, suggesting a new approach with delayed or even no hemodialysis. Methanol-poisoned patients on fomepizole treatment may be separated into two categories: 1) The critically ill patient, with severe metabolic acidosis (base deficit >15 mM) and/or visual disturbances should be given buffer, fomepizole and immediate hemodialysis: dialysis removes the toxic anion formate, and assists in correcting the metabolic acidosis, thereby also reducing formate toxicity. The removal of methanol per se is not important in this setting because fomepizole prevents further production of formic acid. 2) The stable patient, with less metabolic acidosis and no visual disturbances, should be given buffer and fomepizole. This treatment allows for the possibility to delay, or even drop, dialysis in this setting, because patients will not develop more clinical features from methanol poisoning when fomepizole and bicarbonate is given in adequate doses. Indications and triage for hemodialysis in methanol poisonings should be modified. Delayed hemodialysis or even no hemodialysis may be an option in selected cases.

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6

Battistella, Marisa. "Fomepizole as an Antidote for Ethylene Glycol Poisoning." Annals of Pharmacotherapy 36, no. 6 (2002): 1085–89. http://dx.doi.org/10.1345/aph.1a397.

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OBJECTIVE: To assess the use of fomepizole in the treatment of ethylene glycol poisoning in the absence of hemodialysis. DATA SOURCES: A MEDLINE search (1966–May 2001) of English-language literature pertaining to the use of fomepizole in ethylene glycol poisoning was performed. Key search terms included fomepizole, ethylene glycol poisoning, and hemodialysis. References cited in those articles were also evaluated. DATA SYNTHESIS: Results from a number of case reports and a prospective trial suggest that fomepizole is a safe and effective antidote in the treatment of ethylene glycol poisoning. CONCLUSIONS: Case reports and 1 prospective trial have shown that, in the absence of both renal dysfunction and significant metabolic acidosis, the use of fomepizole should obviate the need for hemodialysis. However, the decision to add hemodialysis in the treatment of ethylene glycol poisoning based on plasma ethylene glycol concentrations is still debatable. A randomized, controlled trial is needed to determine the exact criteria for adding hemodialysis.

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7

&NA;. "Alcohol/fomepizole." Reactions Weekly &NA;, no. 1251 (2009): 4–5. http://dx.doi.org/10.2165/00128415-200912510-00009.

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8

Chen, Yng-Tay, Jiunn-Wang Liao, and Dong-Zong Hung. "Protective effects of fomepizole on 2-chloroethanol toxicity." Human & Experimental Toxicology 29, no. 6 (2010): 507–12. http://dx.doi.org/10.1177/0960327109358612.

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2-Chloroethanol (2-CE) is a widely used industrial solvent. In Taiwan, Taiwanese farmers apply 2-CE on grape-vines to accelerate grape growth, a practice that in some cases have caused poisoning in humans. Thus, there is strong interest in identifying antidotes to 2-CE. This study examines the protective role in 2-CE intoxicated rats. Alcohol dehydrogenase and glutathione were hypothesized to be important in the metabolism of 2-CE. This study used fomepizole, an alcohol dehydrogenase inhibitor, and chemicals that affected glutathione metabolism to study 2-CE toxicity. Notably, fomepizole 5 mg/kg significantly increased median lethal dose (LD50) of 2-CE from 65.1 to 180 mg/kg and reduced the production of a potential toxic metabolite chloroacetaldehyde (CAA) in animal plasma. In contrast, disulfiram (DSF), an aldehyde dehydrogenase inhibitor, increased the toxicity of 2-CE on the lethality in rats. Additional or pretreatment with N-acetylcysteine (NAC) and fomepizole significantly reduced plasma CAA concentrations. Fomepizole also significantly reduced 2-CEinhibited glutathione activity. Otherwise, pretreatment with NAC for 4 days followed by co-treatment with fomepizole significantly decreased formation of the metabolic CAA. These results indicated that its catalytic enzyme might play a vital role during 2-CE intoxication, and the combination of fomepizole and NAC could be a protective role in cases of acute 2-CE intoxication.

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9

Hall, Tammy L. "Fomepizole in the treatment of ethylene glycol poisoning." CJEM 4, no. 03 (2002): 199–204. http://dx.doi.org/10.1017/s1481803500006382.

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ABSTRACT The management of ethylene glycol poisoning is reviewed, with a focus on the use of the new antidote fomepizole. Ethylene glycol is a widely used industrial agent that is also easily obtained commercially, usually as radiator antifreeze. Ingestion of as little as 30 to 60 mL can result in death or serious permanent disability. Traditional management of poisoning includes the use of ethanol, with or without hemodialysis. Activated charcoal is not indicated, and gastric lavage may be beneficial only in the first hour after ingestion. Cofactors such as pyridoxine and thiamine may be beneficial in patients deficient in these vitamins. A new antidote, fomepizole, has recently been approved for use in Canada. Like ethanol, it is a competitive inhibitor of alcohol dehydrogenase. Potential benefits of fomepizole include its ease of administration and lack of serious adverse effects. Fomepizole may be recommended over ethanol in situations in which avoidance of ethanol-induced side effects is imperative or when ethanol is not readily available. Further studies are required to verify its comparative efficacy and cost-effectiveness compared to ethanol.

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10

&NA;. "Focus on…fomepizole." Nursing 30, no. 4 (2000): 32cc8. http://dx.doi.org/10.1097/00152193-200030040-00021.

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11

Beatty, Lorri, Robert Green, Kirk Magee, and Peter Zed. "A Systematic Review of Ethanol and Fomepizole Use in Toxic Alcohol Ingestions." Emergency Medicine International 2013 (2013): 1–14. http://dx.doi.org/10.1155/2013/638057.

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Objectives. The optimal antidote for the treatment of ethylene glycol or methanol intoxication is not known. The objective of this systematic review is to describe all available data on the use of ethanol and fomepizole for methanol and ethylene glycol intoxication.Data Source. A systematic search of MEDLINE and EMBASE was conducted.Study Selection. Published studies involving the use of ethanol or fomepizole, or both, in adults who presented within 72 hours of toxic alcohol ingestion were included. Our search yielded a total of 145 studies for our analysis. There were no randomized controlled trials, and no head-to-head trials.Data Extraction. Variables were evaluated for all publications by one independent author using a standardized data collection form.Data Synthesis. 897 patients with toxic alcohol ingestion were identified. 720 (80.3%) were treated with ethanol (505 Me, 215 EG), 146 (16.3%) with fomepizole (81 Me, 65 EG), and 33 (3.7%) with both antidotes (18 Me, 15 EG). Mortality in patients treated with ethanol was 21.8% for Me and 18.1% for EG. In those administered fomepizole, mortality was 17.1% for Me and 4.1% for EG. Adverse events were uncommon.Conclusion. The data supporting the use of one antidote is inconclusive. Further investigation is warranted.

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12

Wilson, Nicola. "SP2 Management of ethylene glycol poisoning in an adolescent: a clinical pearl." Archives of Disease in Childhood 105, no. 9 (2020): e1.2-e2. http://dx.doi.org/10.1136/archdischild-2020-nppg.2.

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Situation16 year old girl admitted with suspected ingestion of ethylene glycol. She was treated with fomepizole and continuous renal replacement therapy (CRRT).How the Pharmacy Team ContributedEthanol was prescribed until fomepizole arrived. The volume of ethanol to be administered was calculated wrongly by the consultant due to confusion about the available strength. The Paediatric Intensive Care Unit (PICU) pharmacist intervened and the correct dosage information was given.PICU pharmacist used Toxbase to determine the correct treatment of ethylene glycol poisoning and advised on dosing regime, including adjustment due to CRRT. The pharmacist facilitated prescribing on the electronic system (added drug to system, set up administration instructions and assisted with prescribing – pharmacist was not an Independent Prescriber). This allowed medical staff to concentrate on resuscitation, monitoring cardiac function, inserting intravenous lines and obtaining access for CRRT.The PICU pharmacist and pharmacy technician co-ordinated initial supply of fomepizole. Fomepizole is usually ordered directly from the manufacturer during office hours. The patient presented in the early evening so further supply had to be obtained from a hospital hundreds of miles away after referring to the Rarely Used Medicines list. Pharmacist contacted appropriate on-call pharmacist and arranged for transfer of medicines via courier. Pharmacy technician arranged for further supply form the manufacturer the following day.Pharmacy technician arranged supply of additional dialysis fluids for CRRT due to the higher than usual administration rate.Without contribution from the pharmacy team the patient is likely to have been given the wrong dose of ethanol and fomepizole, and there would have been delay in initiation of treatment followed by an interruption, as it was wrongly assumed that it was kept as stock in the adjoining ‘adult hospital’ and subsequent supply from a local hospital would not have been sought by ward staff until original supply ran out.OutcomeEthylene glycol poisoning was confirmed on laboratory testing. Levels of ethylene glycol fell steadily over 36 hours, allowing CRRT and fomepizole to stop. Patient was discharged from PICU after 48 hours with no apparent long-lasting effects, but was referred to various specialities including renal, gastroenterology and psychology.Patient and family denied knowledge of intentional or accidental ingestion. Police investigation was inconclusive.Lessons to be LearnedLarger supplies of fomepizole are now kept in stock within Health Board. Supplies were missing from emergency cupboards when stock was needed, despite being on stock lists, necessitating courier fees to transfer stock from elsewhere. Procedures reviewed to ensure that stock is available in emergency cupboards at all times.This patient demonstrated that current (target) stock levels of fomepizole were inadequate for providing treatment during CRRT as the required doses are substantially higher (administered every four hours rather than every twelve hours) and would have lasted less than 12 hours for this average sized teenager. National Rarely Used Medicines list was updated to reflect actual stock levels and other hospitals increased their stockholding due to the realisation that existing stock was inadequate and that further supplies were hard to obtain out of working hours.

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13

Goldfarb, David S. "Fomepizole for ethyleneglycol poisoning." Lancet 354, no. 9190 (1999): 1646. http://dx.doi.org/10.1016/s0140-6736(05)77133-6.

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14

&NA;. "Approvals for fomepizole and busulfan." Inpharma Weekly &NA;, no. 1319 (2002): 22. http://dx.doi.org/10.2165/00128413-200213190-00047.

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15

Najafi, Chris C., Leonard J. Hertko, Jerrold B. Leikin, and Stephen M. Korbet. "Fomepizole in Ethylene Glycol Intoxication." Annals of Emergency Medicine 37, no. 3 (2001): 358–59. http://dx.doi.org/10.1067/mem.2001.113734.

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16

Chiu, Michael H., Natalia Jaworska, Nicholas L. Li, and Mark Yarema. "Massive Acetaminophen Overdose Treated Successfully with N-Acetylcysteine, Fomepizole, and Hemodialysis." Case Reports in Critical Care 2021 (July 11, 2021): 1–5. http://dx.doi.org/10.1155/2021/6695967.

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Acetaminophen overdose is one of the most common causes of acute hepatic failure in the developed world. There is strong evidence for N-acetylcysteine (NAC) as a safe and effective antidote for acetaminophen toxicity. However, there is less clarity in the management of massive overdoses (acute, single ingestions > 500 mg/kg with 4-hour equivalent concentrations ~6000 μmol/L) which are often associated with metabolic acidosis and multiorgan dysfunction. In such ingestions, the role of adjuvant treatments such as fomepizole and extracorporeal removal is unclear. We present a case of a 20-year-old female presenting with an acute ingestion of over 120 grams (1764.7 mg/kg) and an acetaminophen concentration of 5880 μmol/L who developed refractory shock, decreased level of consciousness, and metabolic acidosis requiring mechanical ventilation and vasopressor support. She was treated with gastric decontamination with activated charcoal, IV NAC, fomepizole, and hemodialysis. The patient had complete clearance of acetaminophen by 32 hours after presentation and normalization of her acid base and hemodynamic status without any organ failure. This case highlights the potential benefit of a triple strategy of NAC, fomepizole, and early hemodialysis in massive acetaminophen overdose, potentially sparing complications of prolonged intubation and ICU hospitalization.

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17

Yaworski, H., W. Palatnick, C. Oleschuk, S. Ringland, and M. Tenebein. "P163: Methanol poisoning by inhalation: a case series." CJEM 20, S1 (2018): S115. http://dx.doi.org/10.1017/cem.2018.361.

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Introduction: Methanol intoxication is a well-recognized toxicological emergency. While most cases of significant methanol poisoning occur via ingestion, there are reports in the literature of poisoning resulting from the inhalational route. We report a series of methanol intoxications secondary to inhalational abuse of a methanol containing lacquer thinner presenting to an inner city Emergency Department. Methods: A laboratory database was searched for methanol levels > 5 mmol/L. (16mg/dL). from January 1, 2010 to December 31, 2015. A chart review was completed to determine mode of poisoning, clinical presentation, treatment, and disposition. Results: We found 35 patients who made a total of 83 emergency department (ED) visits with a methanol level > 5mmol/L. (16mg/dL). The methanol levels ranged from 5.3-39.6 mmol/L. (16.96 -126.72 mg/dL) . 73% of poisonings were secondary to inhalation of a methanol-containing lacquer thinner. The median age of these patients was 43 years, and 49% were male. The majority of patients (96%) resided in the core area. The most frequent chief complaints were substance abuse/intoxication, gastrointestinal complaints, and chest pain. 18% of patients described visual symptoms. Treatments were fomepizole only (59%), fomepizole plus hemodialysis (26%), and hemodialysis alone (2%). 49% of patients were discharged from the ED, while 28% and 23% were admitted to an intensive care unit (ICU) and an internal medicine ward respectively. There were no cases of blindness. We describe a cohort of patients who developed methanol poisoning from inhalation of a methanol containing lacquer thinner that required treatment with fomepizole and hemodialysis. While almost 1/3 of these patients were admitted to ICU, 49% were discharged from the emergency department after a course of fomepizole. The etiology of this outbreak was found to be a change in the formulation of the lacquer thinner, substituting a higher concentration of methanol for toluene. The manufacturer and a number of local retail outlets were contacted. This resulted in the product being taken off the shelves by the retail outlets, and eventually, a change in the product formulation by the manufacturer, with a resultant decrease in the methanol content. After these actions, we have not seen any additional presentations of inhalational methanol intoxication. Conclusion: We report the largest case series to date of patients who presented with methanol intoxication, requiring fomepizole and/or hemodialysis, secondary to inhalation of a methanol containing lacquer thinner. Physician advocacy regarding the etiology of this outbreak resulted in collaboration with retail outlets and subsequent action by the manufacturer. This ended the outbreak.

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18

Gracia, Rebeca, Brian Latimer, and Kenneth E. McMartin. "Kinetics of fomepizole in pregnant rats." Clinical Toxicology 50, no. 8 (2012): 743–48. http://dx.doi.org/10.3109/15563650.2012.716518.

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19

Casavant, M. J. "Fomepizole in the Treatment of Poisoning." PEDIATRICS 107, no. 1 (2001): 170–71. http://dx.doi.org/10.1542/peds.107.1.170.

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20

&NA;. "Fomepizole effective in ethylene glycol poisoning." Inpharma Weekly &NA;, no. 1180 (1999): 19. http://dx.doi.org/10.2165/00128413-199911800-00034.

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21

Mycyk, Mark B., and Jerrold B. Leikin. "Antidote Review: Fomepizole for Methanol Poisoning." American Journal of Therapeutics 10, no. 1 (2003): 68–70. http://dx.doi.org/10.1097/00045391-200301000-00015.

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22

Osterhoudt, Kevin C. "Fomepizole Therapy for Pediatric Butoxyethanol Intoxication." Journal of Toxicology: Clinical Toxicology 40, no. 7 (2002): 929–30. http://dx.doi.org/10.1081/clt-120016967.

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23

Sande, Margaret, David Thompson, and Andrew A. Monte. "Fomepizole for severe disulfiram-ethanol reactions." American Journal of Emergency Medicine 30, no. 1 (2012): 262.e3–262.e5. http://dx.doi.org/10.1016/j.ajem.2010.11.014.

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24

Sivilotti, Marco L. A. "Ethanol: Tastes Great! Fomepizole: Less Filling!" Annals of Emergency Medicine 53, no. 4 (2009): 451–53. http://dx.doi.org/10.1016/j.annemergmed.2008.07.038.

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25

Przystanowicz, Jędrzej, Barbara Zielińska-Psuja, Joanna Kowalówka-Zawieja, and Karina Sommerfeld. "Acid-base balance in acute ethylene glycol poisoning in rats treated with fomepizole." Journal of Medical Science 83, no. 1 (2014): 29–36. http://dx.doi.org/10.20883/medical.e40.

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Introduction. Ethylene glycol (EG) is relatively nontoxic but undergoes a multi-step oxidation to toxic metabolites, aldehydes and acids. The accumulation of organic acids, mainly glycolates, leads to the development of profound, life-threatening metabolic acidosis. A key therapy is an antidotal treatment with fomepizole (4-MP), the inhibitor of the first step of EG biotransformation enzyme, alcohol dehydrogenase.Aim. The aim of the study was to demonstrate the efficacy of fomepizole in the prevention of acid-base balance disorders in acute ethylene glycol poisonings in rats.Material and methods. Adult male Wistar rats were given EG (p.o.) with single (i.p.) or multiple (p.o.) doses of 4-MP (EG 3830 and 5745 mg/kg, respectively, 4-MP in single dose of 10 mg/kg or 15 mg/kg followed by 10 mg/kg every 12 hours). Blood gas analysis was performed and blood pH, bicarbonate concentration and base excess were evaluated.Results and conclusions. The single dose of 4-MP was effective in preventing a decrease in blood pH, bicarbonate concentration and base excess during the entire experimental period (pH 7.35 vs 7.21 at hour 12, bicarbonate concentration 27.2 vs 18.3 mmol/dm3 at hour 8, base excess 1.8 vs -8.2 mmol/dm3 at hour 18). The multiple administration of 4-MP started 2 hours after EG poisoning resulted in rapid restoration of proper values of acid- -base balance parameters. Fomepizole is highly efficacious in restraining the acid-base balance disorders which are concomitant with acute ethylene glycol poisonings.

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26

Brent, Jeffrey, Kenneth McMartin, Scott Phillips, Cynthia Aaron, and Ken Kulig. "Fomepizole for the Treatment of Methanol Poisoning." New England Journal of Medicine 344, no. 6 (2001): 424–29. http://dx.doi.org/10.1056/nejm200102083440605.

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27

Miller, Neil R. "FOMEPIZOLE FOR THE TREATMENT OF METHONOL POISONING." Evidence-Based Eye Care 3, no. 2 (2002): 114–15. http://dx.doi.org/10.1097/00132578-200204000-00024.

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28

Weinman, Steven A. "A new antidote in review: Fomepizole (Antizol)." Journal of Emergency Nursing 24, no. 4 (1998): 333–34. http://dx.doi.org/10.1016/s0099-1767(98)90108-8.

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29

Brent, Jeffrey. "Fomepizole for Ethylene Glycol and Methanol Poisoning." New England Journal of Medicine 360, no. 21 (2009): 2216–23. http://dx.doi.org/10.1056/nejmct0806112.

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30

Velez, Larissa I., Greene Shepherd, Yong Chan Lee, and Daniel C. Keyes. "Ethylene glycol ingestion treated only with fomepizole." Journal of Medical Toxicology 3, no. 3 (2007): 125–28. http://dx.doi.org/10.1007/bf03160922.

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31

Mégarbane, Bruno, Stephen W. Borron, Hervé Trout, et al. "Treatment of acute methanol poisoning with fomepizole." Intensive Care Medicine 27, no. 8 (2001): 1370–78. http://dx.doi.org/10.1007/s001340101011.

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32

Wong, S., R. King, S. Molai, et al. "361: Medication Errors Occurring in Fomepizole Administration." Annals of Emergency Medicine 54, no. 3 (2009): S114. http://dx.doi.org/10.1016/j.annemergmed.2009.06.393.

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33

McMartin, Kenneth E., C. Simon Sebastian, David Dies, and Dag Jacobsen. "Kinetics and metabolism of fomepizole in healthy humans." Clinical Toxicology 50, no. 5 (2012): 375–83. http://dx.doi.org/10.3109/15563650.2012.683197.

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34

Brown, M. J., M. W. Shannon, A. Woolf, and E. W. Boyer. "Childhood Methanol Ingestion Treated With Fomepizole and Hemodialysis." PEDIATRICS 108, no. 4 (2001): e77-e77. http://dx.doi.org/10.1542/peds.108.4.e77.

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35

Borron, S. "Fomepizole in treatment of uncomplicated ethylene glycol poisoning." Lancet 354, no. 9185 (1999): 831. http://dx.doi.org/10.1016/s0140-6736(99)02711-7.

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36

Borron, Stephen W., Bruno Mégarbane, and Frédéric J. Baud. "Fomepizole in treatment of uncomplicated ethylene glycol poisoning." Lancet 354, no. 9181 (1999): 831. http://dx.doi.org/10.1016/s0140-6736(99)80015-4.

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37

Bestic, Michelle, Martha Blackford, and Michael Reed. "Fomepizole: A Critical Assessment of Current Dosing Recommendations." Journal of Clinical Pharmacology 49, no. 2 (2009): 130–37. http://dx.doi.org/10.1177/0091270008327142.

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38

Brent, Jeffrey, Kenneth McMartin, Scott Phillips, et al. "Fomepizole for the Treatment of Ethylene Glycol Poisoning." New England Journal of Medicine 340, no. 11 (1999): 832–38. http://dx.doi.org/10.1056/nejm199903183401102.

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39

Clark, Jason D. "Intravenous fomepizole: Treating ethylene glycol and methanol toxicity." AirMed 21, no. 3 (2002): 22–24. http://dx.doi.org/10.1067/mmj.2002.124222.

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40

Clark, Jason D. "Intravenous fomepizole: Treating ethylene glycol and methanol toxicity." Air Medical Journal 21, no. 3 (2002): 22–24. http://dx.doi.org/10.1016/s1067-991x(02)70033-3.

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41

Maskell, Kevin F., Sara Beckett, and Kirk L. Cumpston. "Methanol Kinetics in Chronic Kidney Disease After Fomepizole." American Journal of Therapeutics 23, no. 6 (2016): e1949-e1951. http://dx.doi.org/10.1097/mjt.0000000000000394.

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42

Mégarbane, Bruno, Dominique Fompeydie, Robert Garnier, and Frédéric J. Baud. "Treatment of a 1,4-Butanediol Poisoning with Fomepizole." Journal of Toxicology: Clinical Toxicology 40, no. 1 (2002): 77–80. http://dx.doi.org/10.1081/clt-120002888.

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43

Hovda, Knut Erik, Kirsti Svendsen Andersson, Petter Urdal, and Dag Jacobsen. "Methanol and Formate Kinetics During Treatment with Fomepizole." Clinical Toxicology 43, no. 4 (2005): 221–27. http://dx.doi.org/10.1081/clt-200058936.

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Hovda, Knut Erik, Kirsti Svendsen Andersson, Petter Urdal, and Dag Jacobsen. "Methanol and Formate Kinetics During Treatment with Fomepizole." Clinical Toxicology 43, no. 4 (2005): 221–27. http://dx.doi.org/10.1081/clt-58936.

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Preiss, D. "Fomepizole and ethanol for treating ethylene glycol poisoning." BMJ 348, jan06 17 (2014): g70. http://dx.doi.org/10.1136/bmj.g70.

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Detaille, Thierry, Pierre Wallemacq, Stephan Clément de Cléty, Roger Vanbinst, Guy Dembour, and Philippe Hantson. "Fomepizole alone for severe infant ethylene glycol poisoning." Pediatric Critical Care Medicine 5, no. 5 (2004): 490–91. http://dx.doi.org/10.1097/01.pcc.0000128600.17670.ff.

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Baum, C. R., C. B. Langman, E. E. Oker, C. A. Goldstein, S. R. Aviles, and J. K. Makar. "Fomepizole Treatment of Ethylene Glycol Poisoning in an Infant." PEDIATRICS 106, no. 6 (2000): 1489–91. http://dx.doi.org/10.1542/peds.106.6.1489.

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Lepik, Katherine J., Jeffrey R. Brubacher, Christopher R. DeWitt, et al. "Bradycardia and hypotension associated with fomepizole infusion during hemodialysis." Clinical Toxicology 46, no. 6 (2008): 570–73. http://dx.doi.org/10.1080/15563650701725128.

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Carnero, Guillermo, Manish Nepal, and James Hartle. "51 Severe Methanol Poisoning Requiring Recurrent Hemodialysis and Fomepizole." American Journal of Kidney Diseases 57, no. 4 (2011): B29. http://dx.doi.org/10.1053/j.ajkd.2011.02.054.

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Schicchi, Azzurra, Hélène Besson, Riana Rasamison, Marie-Pierre Berleur, and Bruno Mégarbane. "Fomepizole to treat disulfiram-ethanol reaction: a case series." Clinical Toxicology 58, no. 9 (2019): 922–25. http://dx.doi.org/10.1080/15563650.2019.1708091.

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