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Journal articles on the topic 'Food toxicosis'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Edmondson, A. J., B. B. Norman, and D. Suther. "Survey of state veterinarians and state veterinary diagnostic laboratories for selenium deficiency and toxicosis in animals." Journal of the American Veterinary Medical Association 202, no. 6 (1993): 865–72. http://dx.doi.org/10.2460/javma.1993.202.06.865.

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Summary: Surveys were performed in which the state veterinarian and the major state veterinary diagnostic laboratories in every state were asked about selenium (Se) deficiency and toxicosis in livestock and wildlife in their state. Selenium-deficiency diseases were diagnosed in 46 states and were reported to be an important livestock problem in regions of 37 states; deficiencies were diagnosed in wildlife in 10 states. Natural Se toxicosis was a rare problem, with only 7 states reporting naturally developing Se toxicosis. Oversupplementation with Se was reported as a cause of toxicosis in 15 s

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2

Huang, Yan, Jing Ge, Yongjie Wang, Sarah Shelby, Palika Morse та Ken Coffey. "283 Quercetin Antagonizes Tall Fescue Toxicosis-Induced Cardiovascular Toxicity in Lamb (Ovis Aries) via Modulating PGC-1α Mediated Mitochondrial Biogenesis". Journal of Animal Science 100, Supplement_3 (2022): 131–35. http://dx.doi.org/10.1093/jas/skac247.250.

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Abstract Quercetin is a natural flavonoid with a higher antioxidant capacity and anti-inflammatory. Tall fescue [Schedonorus arundinaceus (Schreb.) Dumort; E+] is perennial cool-season grass for grazing livestock in North America. Fescue infected with the endophyte (Neotyphodium coenophialum) produces chemicals toxic to livestock. However, the impact of quercetin on alleviating fescue toxicosis has received only limited attention. The objective of this experiment was to investigate the mitigation effect of quercetin on fescue toxicosis-induced cardiotoxicity of lambs. A total number of 24 Dorp

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3

Banholzer, E., and K. Heinritzi. "Selenium toxicosis in fattening pigs." Journal of Animal Physiology and Animal Nutrition 80, no. 1-5 (1998): 158–62. http://dx.doi.org/10.1111/j.1439-0396.1998.tb00519.x.

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4

Classen, Dyneah, Kent Schwartz, Darin Madson, and Steve Ensley. "Microcystin toxicosis in nursery pigs." Journal of Swine Health and Production 25, no. 4 (2017): 198–205. http://dx.doi.org/10.54846/jshap/999.

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This case report documents a clinical case of blue-green algae toxicosis, caused by microcystin toxins, in 5-week-old pigs. Mortality during the investigation was elevated by approximately 7.5% in three affected groups, with a final mortality of 11.4%, and 50% of the population demonstrating clinical signs of various degrees. Affected pigs grew slowly and had distended abdomens. Histological examination of tissue samples revealed hepatic centrilobular necrosis with chronic-active periacinar individual hepatocyte necrosis and regeneration or centrilobular hepatocyte necrosis with hemorrhage. Ad

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5

Hall, Jeffery O. "Ionophore Use and Toxicosis in Cattle." Veterinary Clinics of North America: Food Animal Practice 16, no. 3 (2000): 497–509. http://dx.doi.org/10.1016/s0749-0720(15)30083-9.

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6

Ensley, Steve. "Ionophore Use and Toxicosis in Cattle." Veterinary Clinics of North America: Food Animal Practice 36, no. 3 (2020): 641–52. http://dx.doi.org/10.1016/j.cvfa.2020.07.001.

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7

Reed, K. F. M., Z. N. Nie, L. V. Walker, W. J. Mace, and S. G. Clark. "Weather and pasture characteristics associated with outbreaks of perennial ryegrass toxicosis in southern Australia." Animal Production Science 51, no. 8 (2011): 738. http://dx.doi.org/10.1071/an11016.

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Severe outbreaks of toxicosis caused by the natural endophyte Neotyphodium lolii in perennial ryegrass (Lolium perenne L.) have occasionally resulted in heavy loss of livestock in Victoria and Tasmania. Meteorological records were examined from locations where acute toxicosis was recorded. High rainfall (>350 mm) in spring–summer was an obvious common feature; typically, it prolonged the period of high growth rate of pasture, increasing predicted spring–summer (September–March) growth (by ~36%) and increasing the predicted digestibility of organic matter of summer pasture (1 February) by 2.

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8

Shimshoni, Jakob Avi, Malka Britzi, Paolo S. Pozzi, et al. "Acute maduramicin toxicosis in pregnant gilts." Food and Chemical Toxicology 68 (June 2014): 283–89. http://dx.doi.org/10.1016/j.fct.2014.03.034.

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9

SATO, Reeko, Hiroyuki YAMAGISHI, Yoshihisa NAITO, et al. "Feline Vitamin D Toxicosis Caused by Commercially Available Cat Food." Journal of the Japan Veterinary Medical Association 46, no. 7 (1993): 577–81. http://dx.doi.org/10.12935/jvma1951.46.577.

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10

KHAN, M. A., and G. C. KOZUB. "RESPONSE OF ANGUS, CHAROLAIS, AND HEREFORD BULLS TO BLACK FLIES (Simulium spp), WITH AND WITHOUT PHOSMET TREATMENT." Canadian Journal of Animal Science 65, no. 1 (1985): 269–72. http://dx.doi.org/10.4141/cjas85-033.

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Clinical toxicosis, used as an indicator of black-fly tolerance, was not as pronounced in Charolais bulls as in Angus or Hereford bulls, whether or not they were treated dermally with 25 mg phosmet/kg body weight. During the first 20 days after exposure to black flies, the combined weight loss of the treated and untreated Charolais bulls was lower than that of their Angus or Hereford counterparts. Up to 33 days PT, the combined gains of the treated bulls of the three breeds were intermittently higher than those of the controls. Key words: Black flies, beef bulls, phosmet, response, toxicosis,

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11

Wages, D. P., M. D. Ficken, M. E. Cook, and J. Mitchell. "Salt Toxicosis in Commercial Turkeys." Avian Diseases 39, no. 1 (1995): 158. http://dx.doi.org/10.2307/1591997.

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12

Poudel, Sanjok, Wayne E. Zeller, John Fike, and Gabriel Pent. "Condensed Tannins Attributes: Potential Solution to Fescue Toxicosis?" Agriculture 13, no. 3 (2023): 672. http://dx.doi.org/10.3390/agriculture13030672.

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Tall fescue (Schedonorus arundinaceous (Schreb.) Dumort. nom. cons. Lolium arundinaceum (Schreb.) Darbysh.) toxicosis results from the consumption of alkaloids released by wild-type endophytes (Epichloe coenophiala) that live in symbiosis with the plant. Alkaloid consumption causes significant production and reproductive losses which cost the U.S. beef industry approximately $2 billion every year. Incorporating species that contain condensed tannins (CTs) into forage systems may be an effective strategy to reduce the effects of fescue toxicosis in livestock. It has been hypothesized that stabl

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13

Murphy, Mike, and Emily Cornwell. "223 Survey of veterinarians who use pentobarbital for euthanasia suggests knowledge gaps regarding animal disposal." Journal of Animal Science 102, Supplement_2 (2024): 239–40. http://dx.doi.org/10.1093/jas/skae102.273.

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Abstract To assess 1) knowledge and practices of veterinarians regarding disposal of euthanized animals, 2) the extent to which veterinarians communicate with their clients about potential risks of rendering pentobarbital-euthanized animals, and 3) the extent to which veterinarians communicate potential relay toxicosis and environmental risks of pentobarbital-euthanized animals to clients. Over a 3-wk period in early 2021, 16,831 of the 99,500 members of the AVMA were surveyed, with 2,093 responses (a 12% response rate). Respondents were assigned to 1 of 3 categories on the basis of their answ

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14

Provenza, Frederick D., John V. Nolan, and Justin J. Lynch. "Temporal contiguity between food ingestion and toxicosis affects the acquisition of food aversions in sheep." Applied Animal Behaviour Science 38, no. 3-4 (1993): 269–81. http://dx.doi.org/10.1016/0168-1591(93)90025-k.

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15

Coenen, M., E. Landes, and G. Assmann. "Selenium toxicosis in the horse - case report." Journal of Animal Physiology and Animal Nutrition 80, no. 1-5 (1998): 153–57. http://dx.doi.org/10.1111/j.1439-0396.1998.tb00518.x.

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16

McLain-Romero, J., R. Creamer, H. Zepeda, J. Strickland, and G. Bell. "The toxicosis of Embellisia fungi from locoweed (Oxytropis lambertii) is similar to locoweed toxicosis in rats1." Journal of Animal Science 82, no. 7 (2004): 2169–74. http://dx.doi.org/10.2527/2004.8272169x.

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17

Mote, Ryan S., Nicholas S. Hill, Karan Uppal, ViLinh T. Tran, Dean P. Jones, and Nikolay M. Filipov. "Metabolomics of fescue toxicosis in grazing beef steers." Food and Chemical Toxicology 105 (July 2017): 285–99. http://dx.doi.org/10.1016/j.fct.2017.04.020.

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18

Render, J. A., W. W. Carlton, E. J. Hinsman, and J. J. Turek. "Pathology of 6-aminonicotinamide toxicosis in the rabbit." Food and Chemical Toxicology 23, no. 9 (1985): 795–803. http://dx.doi.org/10.1016/0278-6915(85)90279-0.

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19

Andreev, Vladimir P., A. I. Andriyanov, and Z. V. Plakhotskaya. "RISKS OF METAL TOXYCOSES FROM THE RECEPTION OF FOOD OF NATURAL ORIGIN IN THE COASTAL LINE OF THE NORTHERN SEAS." Hygiene and sanitation 97, no. 3 (2018): 254–58. http://dx.doi.org/10.18821/0016-9900-2018-97-3-254-258.

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Due to the multifold gain of industrial emissions of heavy metals in the world the danger of the use wild plants and animals in food has increased. Seaweed and shellfish have the high ability to accumulate toxins in their bodies. There is considered the intake of aquatic organisms of the Northern seas with food, taken directly from their habitats. The analysis of available literature data showed that in the conditions of the White and Barents seas, the probability of metal toxicosis seem to be negligible.

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20

Nikolyan, Sona, Anaida Tsakanyan, Nune Andreasyan, and Samvel Martirosyan. "THE PREVALENCE OF BACTERIA OF THE GENUS PROTEUS SPP. IN DIFFERENT TYPES OF RESEARCH FOOD PRODUCTS." Criminalistics and Forensics, no. 67 (August 9, 2022): 372–78. http://dx.doi.org/10.33994/kndise.2022.67.38.

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This article discusses the issue of contamination of food products by microorganisms of the genus Proteus, which actively multiply on a protein nutrient medium, causing the rotting of meat, fish, and other protein-rich foods. It is noted that recently in catering facilities there are often cases of toxic infections caused by the presence of these particular bacteria. The article draws attention to the fact that acute enteroidea, occurring as gastroenteritis, gastritis, or colienteritis, among young children, especially those with reduced immunity, is more severe, accompanied by symptoms of tox

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21

Igbokwe, Ikechukwu Onyebuchi, Ephraim Igwenagu, and Nanacha Afifi Igbokwe. "Aluminium toxicosis: a review of toxic actions and effects." Interdisciplinary Toxicology 12, no. 2 (2019): 45–70. http://dx.doi.org/10.2478/intox-2019-0007.

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Abstract Aluminium (Al) is frequently accessible to animal and human populations to the extent that intoxications may occur. Intake of Al is by inhalation of aerosols or particles, ingestion of food, water and medicaments, skin contact, vaccination, dialysis and infusions. Toxic actions of Al induce oxidative stress, immunologic alterations, genotoxicity, pro-inflammatory effect, peptide denaturation or transformation, enzymatic dysfunction, metabolic derangement, amyloidogenesis, membrane perturbation, iron dyshomeostasis, apoptosis, necrosis and dysplasia. The pathological conditions associa

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22

Subapriya, S., S. Vairamuthu, B. Murali Man, and C. Balachandr. "Clinicopathological Investigation on Thiram Toxicosis in Broiler Chicken." International Journal of Poultry Science 6, no. 4 (2007): 242–44. http://dx.doi.org/10.3923/ijps.2007.242.244.

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23

Subapriya, S., S. Vairamuthu, B. Murali Man, and C. Balachandr. "Pathomorphological Changes in Thiram Toxicosis in Broiler Chicken." International Journal of Poultry Science 6, no. 4 (2007): 251–54. http://dx.doi.org/10.3923/ijps.2007.251.254.

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24

Stonecipher, Clinton A., Benedict T. Green, Kevin D. Welch, Dale R. Gardner, and Daniel Cook. "Larkspur toxicosis alters basal diet intake by cattle." Applied Animal Science 39, no. 5 (2023): 257–63. http://dx.doi.org/10.15232/aas.2023-02405.

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25

Rademacher, Christopher, Scott Radke, and Laura Greiner. "Management of sodium ion toxicosis – water deprivation syndrome." Journal of Swine Health and Production 31, no. 6 (2023): 295–97. http://dx.doi.org/10.54846/jshap/1353.

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If a pig is exposed to prolonged periods without water or excessive salt intake, the sodium content of the brain increases. The most severe problems occur when a pig is rehydrated with unfettered water access. The high concentration of sodium in the brain draws water in and the brain swells inside the rigid calvarium causing neurologic clinical signs and ultimately, death. Rehydration must occur as a slow process over a period of many hours to prevent brain swelling. Water introduction must occur over a period of hours with slow water introduction to allow for the sodium ion exchange.

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26

Saidova, Nilufar Akhrorovna. "CHANGES IN THE MOUTH CAVITY AND METABOLIC DISORDERS DURING TOXICOSIS IN THE 1st HALF OF PREGNANCY." Results of National Scientific Research International Journal 3, no. 12 (2024): 188–93. https://doi.org/10.5281/zenodo.14564385.

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The problem of early toxicosis today is quite relevant. There are frequently occurring (vomiting of pregnant women, salivation) and rare forms of early toxicosis (dermatoses of pregnant women, tetany, osteomalacia, acute yellow liver atrophy, bronchial asthma of pregnant women). In this work, the most frequent clinical manifestation of early toxicosis, namely vomiting of pregnant women, will be considered in more detail. Vomiting of pregnant women is a complex clinical syndrome, which is characterized by digestive disorders, leading to significant disorders: dehydration, changes in metabolism

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27

Corbee, Ronald Jan, and Louis C. Penning. "COMMD1 Exemplifies the Power of Inbred Dogs to Dissect Genetic Causes of Rare Copper-Related Disorders." Animals 11, no. 3 (2021): 601. http://dx.doi.org/10.3390/ani11030601.

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Wilson’s Disease is a rare autosomal recessive disorder in humans, often presenting with hepatic copper overload. Finding the genetic cause of a rare disease, especially if it is related to food constituents like the trace element copper, is a Herculean task. This review describes examples of how the unique population structure of in-bred dog strains led to the discovery of a novel gene and two modifier genes involved in inherited copper toxicosis. COMMD1, after the discovery in 2002, was shown to be a highly promiscuous protein involved in copper transport, protein trafficking/degradation, re

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28

Gilbert, Robinette W., Jean E. Sander, and Thomas P. Brown. "Copper Sulfate Toxicosis in Commercial Laying Hens." Avian Diseases 40, no. 1 (1996): 236. http://dx.doi.org/10.2307/1592396.

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29

Williams, Katherine, Edwin Benjamin Rajo, Andrew R. Weaver, Daniel H. Poole, and Carrie L. Pickworth. "103 Impact of Fescue Variety on Fecal Egg Counts in Mature Beef Cattle." Journal of Animal Science 101, Supplement_1 (2023): 78–79. http://dx.doi.org/10.1093/jas/skad068.093.

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Abstract Fescue toxicosis decreases beef cattle performance in a multitude of ways, including decreased health and immunity. Cattle challenged by fescue toxicosis are often grazing in an environment favorable for development of gastrointestinal nematodes (GIN). Interactions of fescue toxicosis and parasite challenge may result in further performance and economic losses. Many beef producers treat animals regularly for GIN parasites with little insight on necessity or effectiveness of anthelmintic products. Overuse of anthelmintics can result in development of anthelmintic resistance in GIN popu

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30

Kulach, P. V., I. M. Nityaga, and M. I. Shopinskaya. "VETERINARY AND SANITARY ASSESSMENT OF THE QUALITY OF FARM-MADE SAUSAGE PRODUCTS." Problems of Veterinary Sanitation, Hygiene and Ecology 1, no. 2 (2020): 147–51. http://dx.doi.org/10.36871/vet.san.hyg.ecol.202002004.

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The article presents the results of laboratory studies of farm-made sausage products. The results of microbiological studies of sausages indicate that the product does not contain pathogens of food-borne disease and toxicosis, as well as another pathogenic microflora. This indicates high parameters of sausage production, sanitary indicators of raw materials, good sanitary condition of technical equipment and premises of the sausage shop in compliance with technical conditions of heat treatment of products.

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31

Haliburton, John C., and Sandra E. Morgan. "Nonprotein Nitrogen-Induced Ammonia Toxicosis and Ammoniated Feed Toxicity Syndrome." Veterinary Clinics of North America: Food Animal Practice 5, no. 2 (1989): 237–49. http://dx.doi.org/10.1016/s0749-0720(15)30974-9.

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32

Subapriya, S., S. Vairamuthu, B. Murali Man, and C. Balachandr. "Growth Performance Studies in Thiram Toxicosis in Broiler Chicken." International Journal of Poultry Science 6, no. 4 (2007): 248–50. http://dx.doi.org/10.3923/ijps.2007.248.250.

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33

Daft, B. M., A. A. Bickford, and M. A. Hammarlund. "Experimental and Field Sulfaquinoxaline Toxicosis in Leghorn Chickens." Avian Diseases 33, no. 1 (1989): 30. http://dx.doi.org/10.2307/1591064.

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34

Malinak, Chad M., Charles C. Hofacre, Stephen R. Collett, et al. "Tribasic Copper Chloride Toxicosis in Commercial Broiler Chicks." Avian Diseases 58, no. 4 (2014): 642–49. http://dx.doi.org/10.1637/10864-051514-case.1.

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35

Reed, K. F. M., J. L. Vaughan, L. J. Cummins, and D. D. Moore. "Impact of mycotoxins and of a mycotoxin deactivator on alpacas grazing perennial ryegrass infected with wild endophyte (Neotyphodium spp.)." Animal Production Science 50, no. 9 (2010): 902. http://dx.doi.org/10.1071/an10068.

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Liveweight gain, animal health and the effectiveness of a mycotoxin deactivator were studied on an old pasture that contained 61% perennial ryegrass. Sixty-seven percent of the ryegrass population was infected with endophyte (Neotyphodium spp.). The pasture was fenced into two halves and two groups of 28 alpaca male weaners were rotated between the two plots. Nine to 10 Suris and 18–19 Huacayas were allocated to each group. One group was fed a concentrate supplement (100 g/head per day) and the other was fed the same supplement to which was added the toxin deactivator, Mycofix® Plus (5 g/100 g

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36

Poudel, Sanjok, Gabriel J. Pent, John H. Fike, Brittany Harlow, and Wayne Zeller. "161 Effects of Sericea Lespedeza Supplementation on Steers Grazing Wild-Type Endophyte-Infected Tall Fescue." Journal of Animal Science 102, Supplement_1 (2024): 50–51. http://dx.doi.org/10.1093/jas/skae019.060.

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Abstract Fescue toxicosis poses a significant challenge to the United States beef industry, resulting in considerable economic losses. This syndrome arises from the consumption of tall fescue (Schedonorus arundinaceous) infected with the wild-type endophyte (WE; Epichloe coenophiala), which produces toxic alkaloids. The most effective solution for mitigating fescue toxicosis is renovating WE tall fescue pastures to novel endophyte-infected (NE) tall fescue. However, pasture renovation is costly and may not be feasible under certain conditions, with the economic profitability taking up to 3 yr.

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37

Alfaro, Gastón F., and Sonia J. Moisá. "Fescue toxicosis: a detrimental condition that requires a multiapproach solution." Animal Frontiers 12, no. 5 (2022): 23–28. http://dx.doi.org/10.1093/af/vfac063.

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38

Yusupova, K. V., N. N. Mishina, E. I. Semyonov, G. S. Kashevarov, and Z. H. Sagdeeva. "Screening of adaptogens potentially effective in mycotoxicosis." International Journal of Veterinary Medicine, no. 2 (June 28, 2024): 115–23. http://dx.doi.org/10.52419/issn2072-2419.2024.2.115.

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Microscopic mold fungi are pathogens invisible to the naked eye that secrete dangerous metabolic products – mycotoxins. When exposed to various factors, mycotoxins can accumulate in plant and animal products. Getting into food and, as a result, into a living organism, they can lead to the appearance of mycoses and mycotoxicoses. The most common T-2 toxin of the Russian Federation territory. Due to the impossibility of completely eliminating microscopic fungi, the search for affordable preventive measures with detoxifying, immunostimulating properties remains relevant and promising. The aim of

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39

Popova, O. S., and V. A. Baryshev. "Use of sorption premixes in fish farming as a prevention of nutritional toxicosis." Legal regulation in veterinary medicine, no. 4 (January 8, 2023): 109–11. http://dx.doi.org/10.52419/issn2782-6252.2022.4.109.

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Climate change is a fact with serious implications that extend to pollution, seafood safety and global food security.The studies were carried out in 2022, on the basis of the FGBOU HE SPbGUVM. Guppy aquarium fish were taken as objects of exploratory research. In the amount of 45 pieces. All objects were placed in the same habitat conditions: 3 aquariums of 10 liters each, males were selected, 10 pcs each. Three groups were created: the first group was given acetate as a lead toxicant, at the rate of 1g/10l, which is 10 times higher than the MPC, which is 0.1g/l according to existing regulation

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40

Seay, Taylor B., Brooke A. Clemmons, Emily A. Melchior, et al. "PSII-27 Effect of endophyte-infected tall fescue seed and red clover isoflavones on the serum metabolites in beef steers." Journal of Animal Science 98, Supplement_4 (2020): 374–75. http://dx.doi.org/10.1093/jas/skaa278.658.

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Abstract Fescue toxicosis reduces animal performance, costing approximately $2 billion dollars to the beef industry annually. Emerging research has demonstrated effects of fescue toxicosis can be reduced by consumption of red clover isoflavones. The objective of the current study was to evaluate the effect of isoflavone supplementation with endophyte infected seed consumption on serum metabolites in beef steers. Angus steers (n = 36) were randomly allocated to treatments in a 2×2 factorial arrangement of endophyte-infected (E+) or endophyte-free (E-) tall fescue seed, with (P+) or without (P-)

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41

Jovanović, M., D. Trailović, V. Kukolj, et al. "An outbreak of fumonisin toxicosis in horses in Serbia." World Mycotoxin Journal 8, no. 4 (2015): 387–91. http://dx.doi.org/10.3920/wmj2014.1812.

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This paper reports on an outbreak of fumonisin toxicosis in a stable with 100 horses. Twenty-one horses were affected and fifteen died within a month. The animals were not febrile and exhibited poor appetite, somnolence, head pressing, blindness, ataxia, lethargy, and seizures, ending with death. The post mortem examination of the head of one 18-month-old colt revealed large, bilateral, slightly asymmetrical large areas of necrosis of the cerebral white matter. The lesions involved the frontal, parietal and occipital lobes only. Histological findings revealed leukomalacia with necrosis, infilt

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42

Grote, Ally J., Allison Morgan, Ken Coffey, Christine Nieman, Dirk Philipp, and Beth B. Kegley. "PSXVI-27 Using tannin to mitigate tall fescue toxicosis." Journal of Animal Science 99, Supplement_3 (2021): 480–81. http://dx.doi.org/10.1093/jas/skab235.847.

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Abstract Fescue toxicosis has a negative impact on livestock production. Tannins could bind to tall fescue toxins rendering them ineffective. The objective was to improve health and well-being of sheep offered tall fescue infected with the endophytic fungus (Neotyphodium coenophialum) by binding toxins in the rumen with condensed tannins. Non-pregnant, non-lactating ewes (n=20; 57±1.3 kg initial BW) were housed in individual 1×1.5-m pens with grate flooring and were offered ad libitum access to either non-toxic novel-endophyte infected tall fescue haylage (NE), or toxic endophyte infected tall

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43

VANDERKOP, PERRY A., and JAMES D. MacNEIL. "PROTECTION PROVIDED BY SODIUM SELENITE AGAINST AN ORAL TOXIC DOSE OF MONENSIN IN BROILER CHICKS." Canadian Journal of Animal Science 69, no. 2 (1989): 477–82. http://dx.doi.org/10.4141/cjas89-053.

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Monensin is an ionophoric antibiotic that is widely used in cattle, swine and poultry production. In poultry it is used for its anticoccidial properties. Over the past decade several cases of flock poisoning due to monensin toxicity have been reported. Studies with cattle and swine demonstrate that Se-vitamin E pretreatment can provide a protective effect from an induced monensin toxicosis. In this study, 192 male broiler chicks were given one of eight diets, varying in the concentration of supplemented Se, as sodium selenite, from 0.5 to 15.0 mg kg−1, for a period of 4 wk. An oral dose of mon

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44

Leong, Veronica Yin-Ming, and T. P. Brown. "Toxicosis in Broiler Chicks Due to Excess Dietary Ethoxyquin." Avian Diseases 36, no. 4 (1992): 1102. http://dx.doi.org/10.2307/1591583.

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45

Reznichenko, Aleksei, Svetlana Vodianitskaia, Iana Masalykina, and Andrei Manokhin. "Hepatoprotective effect of hypoxenum against exogenous toxicosis of white rats." BIO Web of Conferences 27 (2020): 00079. http://dx.doi.org/10.1051/bioconf/20202700079.

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Any disturbances in the organism caused by infection, drug administration, vaccination, etc., are accompanied with disturbance of liver functions. Various xenobiotics entering animal organisms with food or water have the highest hepatotoxicity. Thus, an important trend in modern research is a search for substances increasing liver capacity against pathological activities, strengthening its detergent actions. The goal of this research was to study hepatoprotector properties of hypoxenum using the experimental acute toxic hepatitis model in white rats. The acute toxic hepatitis was caused by inj

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Subapriya, S., S. Vairamuthu, B. Murali Man, and C. Balachandr. "A Study on Immunopathologic Effect of Thiram Toxicosis in Broiler Chicken." International Journal of Poultry Science 6, no. 4 (2007): 245–47. http://dx.doi.org/10.3923/ijps.2007.245.247.

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Tanaree, D. D., J. M. Duringer, D. W. Bohnert, and A. M. Craig. "Microarray evaluation of bovine hepatic gene response to fescue toxicosis." World Mycotoxin Journal 6, no. 4 (2013): 419–26. http://dx.doi.org/10.3920/wmj2012.1482.

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‘Fescue toxicosis’ is a disease in livestock caused by ingestion of ergot alkaloids produced by the fungal endophyte Neotyphodium coenophialum in tall fescue; it is estimated to cost 1 billion USD in damages per year to the beef industry alone. Clinical signs include decreased reproductive fitness, necrosis of extremities, and reduced average daily gain and milk production. Little is known about the cellular mechanisms that mediate these toxic sequelae. We evaluated the effects of ergovaline-based fescue toxicosis on gene expression via oligonucleotide microarray. Liver biopsies were obtained

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Sagdeeva, Z. Kh, A. V. Sofronova, D. R. Sagdeev, L. E. Matrosova, S. A. Tanaseva, and E. I. Idrisova. "Organoleptic, physico-chemical and microbiological parameters of broiler chicken meat in combined toxicosis." International Journal of Veterinary Medicine, no. 1 (April 2, 2024): 184–91. http://dx.doi.org/10.52419/issn2072-2419.2024.1.184.

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Increasing the productivity of animals and birds, resistance to non-infectious and infectious diseases remains a priority in agricultural production. Currently, the problem of obtaining environmentally friendly animal feed and food of animal origin is acute. The main natural and anthropogenic pollutants of feed and food raw materials are mycotoxins, toxic elements and pesticides. It is important to highlight the role of the influence of combinations of natural and anthropogenic pollutants on a living organism. Mycotoxins, toxic elements and pesticides, in addition to their polytoxic effect on

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Fisher, P., and C. O'Connor. "Oral toxicity of p-aminopropiophenone to ferrets." Wildlife Research 34, no. 1 (2007): 19. http://dx.doi.org/10.1071/wr06125.

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Ferrets (Mustela furo) are pests in New Zealand and new methods are being sought for their control. The hydrochloride form of p-aminopropiophenone (PAPP) was highly toxic to ferrets when delivered by gavage, with LD50 and LD99 values of 15.52 and 20.80 mg kg–1, respectively. Signs of toxicosis progressed through pallor and cyanosis, impaired coordination, prostration with reduced responses to stimuli, with death in ~2 h. In a preliminary assessment of effective doses of PAPP in bait, 19 of 20 ferrets consumed chick carcasses containing ~46 mg PAPP within 40 min. Ferrets that ate chick baits al

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Thomas, Ivan R., Ken Coffey, Christine C. Nieman, Ally J. Grote, and Sarah W. Alvey. "PSVI-5 Using Red Clover Extract to Mitigate Tall Fescue Toxicosis." Journal of Animal Science 101, Supplement_2 (2023): 344–45. http://dx.doi.org/10.1093/jas/skad341.391.

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Abstract Tall fescue toxicosis is caused by ergot alkaloids produced by the fungus Neotyphodium coenophialum that resides within the majority of the tall fescue plants in the Southeastern and lower Midwestern US. Tall fescue (Festuca arundinacea) toxicosis is estimated to cost livestock producers over $1 billion dollars annually through reduced growth and reproductive performance. Improvements in performance by cattle grazing toxic tall fescue can have substantial economic advantages for livestock producers throughout the tall fescue region. Numerous methods to mitigate tall fescue toxicosis h

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