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Books on the topic 'Foramen oval patente'

Author: Grafiati

Published: 10 September 2021

Last updated: 6 February 2022

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1

Amin, Zahid, Jonathan M. Tobis, Horst Sievert, and John D. Carroll, eds. Patent Foramen Ovale. Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-4987-3.

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2

Abbott, Maude E. Two cases of widely patent foramen ovale. s.n., 1996.

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3

PFO and the diver: Patency of cardiac foramen ovale : a risk factor for dysbaric disorders? Best Pub., 2007.

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4

Carroll, John D., Horst Sievert, Zahid Amin, and Jonathan M. Tobis. Patent Foramen Ovale. Springer, 2017.

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5

Carroll, John D., Horst Sievert, Zahid Amin, and Jonathan M. Tobis. Patent Foramen Ovale. Springer, 2014.

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6

Kokkinidis, Damianos G., Harsimran Sachdeva Singh, George Giannakoulas, Aristeidis H. Katsanos, Guillaume Turc, and Vincent Thijs, eds. Patent Foramen Ovale (PFO) Closure for Prevention of Stroke. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-254-0.

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7

Turc, Guillaume, David Calvet, and Jean-Louis Mas. Cardiac aetiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0005.

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Cardiac aetiology accounts for approximately 20% of strokes in young adults. Although atrial fibrillation is a leading cause of stroke in the general population, it is uncommon in young adults. In such patients, more diverse causes of ischaemic stroke are observed, including valvular heart diseases, infective endocarditis, Libman–Sacks endocarditis, dilated cardiomyopathies, congenital heart diseases, myocardial infarction, and intracardiac tumours. Patent foramen ovale is commonly observed in young adults with ischaemic stroke, but this association may be incidental in a sizeable proportion of patients. Young adults who are the most likely to have a stroke-related patent foramen ovale are also those with the lowest recurrence risk.

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8

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Atrial septal defects. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0076_update_004.

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Secundum atrial septal defects, such as ostium secundum defect, sinus venosus defect, and patent foramen ovale, are discussed. Indications of closure and the recommendations of ACC/AHA and ESC are presented.

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9

Chong, Ji Y., and Michael P. Lerario. Hole in My Heart. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0015.

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The presence of a patent foramen ovale (PFO) is associated with cryptogenic stroke, especially in young patients. Medical therapy for patients with stroke and PFO includes antiplatelet therapy or anticoagulation. PFO closure is not routinely recommended for stroke prevention.

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10

Patent Foramen Ovale Closure for Stroke, Myocardial Infarction, Peripheral Embolism, Migraine, and Hypoxemia. Elsevier, 2020. http://dx.doi.org/10.1016/c2018-0-02016-2.

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11

Thorne, Sara, and Sarah Bowater. Septal defects. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759959.003.0009.

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This chapter explores atrial septal defects (ASDs), including ostium secundum ASD, ostium primum ASD, sinus venosus ASD, coronary sinus defect, and patent foramen ovale. Ventricular septal defects (VSDs) are also discussed, including definition and incidence, cardiac associations, presentation, physical signs, investigation, and management. A further section explores atrioventricular septal defects (AVSDs), including associations, incidence and recurrence, clinical presentation, investigation, surgical management, and late complications after repair AVSD repair.

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12

Archer, Nick, and Nicky Manning. Septal anomalies. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199230709.003.0009.

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Atrial septal defects 122Ventricular septal defects 124Atrioventricular septal defects 134• The presence of a patent foramen ovale (PFO) is essential for right-to-left flow of oxygenated blood returning from the placenta to reach vital organs.• Distinguishing between PFO and an ASD is difficult and the diagnosis can only be made with certainty if the atrial septum is virtually absent. It is suggested that if the size of the gap in the atrial septum is greater than the diameter of the AA then the possibility of a significant ASD postnatally should be considered....

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13

Crawford, Michael H., Edward A. Gill, and John D. Carroll. Percutaneous Closure of Patent Foramen Ovale, An Issue of Cardiology Clinics (The Clinics: Internal Medicine). Saunders, 2005.

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14

Krel, Regina, and Paul G. Mathew. Seeing Spots. Edited by Angela O’Neal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190609917.003.0004.

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Migraine and its association with stroke is a topic that has received much attention due to the high prevalence of migraine and the often devastating outcomes of stroke. There is a nearly two-fold increased risk of stroke in patients with migraine. In addition, this risk is higher in younger adults, particularly women under 45 years old, and in those with increased frequency of migraine with aura attacks. This chapter seeks to explore migraine-induced stroke, migrainous infarction, as well as the risk associated with ischemic stroke in patients with migraines. Furthermore, proposed mechanisms for stroke related to migraine, such as cortical spreading depression, arterial dissection, and patent foramen ovale, will be discussed.

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15

Kelley, Roger E. Cardiac Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0188.

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Heart disease is a major contributor to stroke and other neurologic disorders in adults. Cardioembolic stroke accounts for roughly 15% of all stroke, and the most common mechanism is cardiac arrhythmia, with atrial fibrillation the leading contributor. Anticoagulation such as using aspirin or warfarin is chosen based on the presence of associated risk factors including congestive heart failure, hypertension, age, and diabetes mellitus. Heart failure ranks second in the incidence of stroke from cardioembolism, with other risk factors being endocarditis, severe cardiomyopathy, acute myocardial infarction, and patent foramen ovale. Recent clinical trials indicate that induction of total body hyopthermia after cardiac arrest to a target temperature of 32°C to 34°C, for 24 hours, had a more favorable neurological outcome compared with a normothermia group.

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16

Transcatheter Closure Of Asds And Pfos A Comprehensive Assessment. Not Avail, 2010.

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