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Dissertations / Theses on the topic 'Función barrera intestinal'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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1

Pigrau, Pastor Marc. "Molecular mechanisms and effect of acute and psychosocial stress on the intestinal barrier function. Implications on the irritable bowel syndrome." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/665970.

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El estrés psicosocial es uno de los grandes problemas de salud en las sociedades modernas. En condiciones fisiológicas el estrés representa una amenaza para la homeostasis. En respuesta al estrés se genera una respuesta coordinada que implica a los sistemas autonómico, endocrino e inmunitario para mantener la estabilidad del medio. Una sobreexposición al estrés en individuos susceptibles, altera esta respuesta adaptativa pudiendo dar lugar al desarrollo de enfermedades. Uno de los órganos más vulnerables al estrés es el tracto gastrointestinal (GI), ya que las enfermedades más comunes del trac
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Estévez, Medina Javier. "Toll-like receptors as modulators of intestinal barrier function." Doctoral thesis, Universitat Autònoma de Barcelona, 2016. http://hdl.handle.net/10803/400146.

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Las patologías funcionales (síndrome del intestino irritable, IBS) e inflamatorias gastrointestinales (enfermedad inflamatoria intestinal, IBD) se caracterizan por alteraciones de la función barrera epitelial, con un aumento de la permeabilidad, y cambios en la microbiota intestinal. Los receptores de tipo Toll (TLRs) participan en el reconocimiento bacteriano en el intestino y en el control neuroinmune local, estando, por tanto, implicados en la regulación de la función barrera del epitelio intestinal. El objetivo de este trabajo ha sido caracterizar la implicación de los receptores TLR5 y
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Fernández, Blanco Joan Antoni. "Función Barrera Epitelial en un Modelo de Disfunción Intestinal Inducido por Parasitosis con Trichinella spiralis en la Rata." Doctoral thesis, Universitat Autònoma de Barcelona, 2012. http://hdl.handle.net/10803/107862.

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La función barrera intestinal constituye la primera línea de defensa del tracto digestivo. Ésta se ve condicionada por el transporte hidroelectrolítico, la permeabilidad y la motilidad intestinales. Alteraciones de estas funciones favorecen la estimulación continua, por antígenos y microorganismos luminales, del sistema inmune local llevando a estados inflamatorios más o menos crónicos. Estos cambios parecen caracterizar diversas patologías inflamatorias y funcionales gastrointestinales, incluyendo la enfermedad inflamatoria intestinal y el síndrome de intestino irritable. Los mastocito
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Guilarte, Clavero Mar. "El mastocito: célula clave en la microinflamación intestinal y en la regulación de la función barrera mediada por el estrés en el síndrome del intestino irritable con predominio de diarrea." Doctoral thesis, Universitat Autònoma de Barcelona, 2014. http://hdl.handle.net/10803/283645.

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El Síndrome del Intestino Irritable (SII), clásicamente considerada una enfermedad del colon, es un trastorno altamente prevalente, con gran impacto en la calidad de vida y pocas opciones terapéuticas eficaces. Se desconoce cuál es su etiología, pero el estrés se ha asociado al desarrollo y mantenimiento del SII, en base a que los pacientes afectos padecen más estrés crónico que la población sana. Los mecanismos fisiopatológicos subyacentes no se conocen con exactitud, pero destaca, como hallazgo común relacionado con la sintomatología, la alteración de la función barrera intestinal, junto co
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Brufau, Bonet Maria Teresa. "Efecte dels β-galactomannans sobre la funció intestinal de barrera en les infeccions per Salmonella". Doctoral thesis, Universitat de Barcelona, 2016. http://hdl.handle.net/10803/400155.

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L’administració d’antibiòtics a dosis subterapèutiques va ser una estratègia extensament utilitzada a partir dels anys 50 en la producció animal per a la prevenció d’infeccions i com a promotors del creixement. Tanmateix, la creixent aparició de bacteris resistents als antibiòtics va comportar que l’any 2006, la Unió Europea (UE) prohibís l’ús dels antibiòtics com a promotors del creixement (AGP) en la nutrició animal (CE n. 1831/2003). Aquesta prohibició, però, va fer repuntar el nombre de casos d’infeccions en animals i toxiinfeccions alimentàries en humans que fins aleshores havien estat co
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6

Vieira, Milena Morais. "Retinol, proteÃnas transportadoras, carotenÃides, proteÃnas de fase aguda e barreira funcional intestinal em crianÃas de uma comunidade urbana de Fortaleza." Universidade Federal do CearÃ, 2007. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1596.

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Conselho Nacional de Desenvolvimento CientÃfico e TecnolÃgico<br>A deficiÃncia de vitamina A (DVA) à considerada um importante problema de saÃde pÃblica em todo o mundo e o papel da DVA na integridade epitelial intestinal ainda nÃo està bem compreendido. O objetivo do trabalho foi investigar a associaÃÃo do retinol (vitamina A) e carotenÃides com a barreira funcional intestinal em crianÃas de uma comunidade urbana em Fortaleza, no Nordeste do Brasil. Participaram do estudo 102 crianÃas de 2 meses a 9 anos de idade. O estado nutricional das crianÃas foi avaliado atravÃs do escore âzâ de peso po
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7

Costa, Aline Vasques da. "Efeitos do treinamento aeróbio sobre a morfologia e função barreira da mucosa intestinal em ratos wistar." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/39/39135/tde-01062017-151656/.

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O treinamento aeróbio de forma crônica está correlacionado com menor grau de inflamação e pode ser considerado um dos principais responsáveis por criar condições favoráveis para o equilíbrio intestinal. A mucosa do intestino consiste em uma monocamada de células epiteliais com função principal de absorção de nutrientes e formação de barreira contra entrada de potenciais antígenos que podem prejudicar sua função. A permeabilidade da barreira intestinal pode ser controlada por diferentes processos. Um aumento de antígenos luminais como lipopolissacarídeo (LPS) pode gerar um aumento na permeabili
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8

Bueti, Deanna. "Immunomodulatory cytokines regulate intestinal epithelial barrier function /." Title page and abstract only, 2003. http://web4.library.adelaide.edu.au/theses/09SB/09sbb9289.pdf.

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9

LOZANO, Marisol Garzon. "Cohort study of associations between intestinal protozoa infection and intestinal barrier funcion, nutritional status, and neurodevelopment in infants from Republic of São Tomé." Doctoral thesis, Instituto de Higiene e Medicina Tropical, 2017. http://hdl.handle.net/10362/45433.

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Enquadramento Em lactentes de países de baixo e médio rendimento, Giardia lamblia, Cryptosporidium e Entamoeba hystolitica são agentes prevalentes em infeções intestinais. As interações hospedeiro-parasita podem levar a uma resposta inflamatória da mucosa e aumento da permeabilidade intestinal. Clinicamente, isto pode refletir-se por impacto negativo no crescimento e neurodesenvolvimento. Os efeitos destas infeções intestinais subclínicas na saúde infantil têm sido pouco estudado. Objetivo Analisar, em crianças assintomáticas de São Tomé, as associações entre infeções por parasitas intestin
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10

Beltinger, Johannes Hermann. "Studies on colonic epithelial ion transport and barrier function." Thesis, University of Nottingham, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311747.

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11

Terciolo, Chloé. "Effets de Saccharomyces boulardii CNCM I-745 sur le complexe d'adhérence E-cadhérine/caténines dans les maladies inflammatoires chroniques de l'intestin : impact sur la barrière épithéliale intestinale." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5045/document.

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Dans de nombreuses pathologies digestives dont les maladies inflammatoires chroniques intestinales (MICI), l'intégrité de la barrière épithéliale est rompue. Cette perte d'intégrité est notamment due à la réduction ou la perte d'expression des jonctions adhérentes composées du complexe E-cadhérine/caténines. Il est donc important d'identifier de nouvelles molécules capables de réguler ce complexe dans les MICI. C'est dans ce contexte que nous nous sommes intéréssés à une levure non pathogène, Saccharomyces boulardii (Sb) utilisée dans la prévention et le traitement de désordres gastro-intestin
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12

Moeser, Adam James. "Mechanisms of Prostaglandin-Stimulated Recovery of Mucosal Barrier Function in the Ischemia-Injured Porcine Intestine: Role of Intestinal Ion Transport." NCSU, 2006. http://www.lib.ncsu.edu/theses/available/etd-04252006-123724/.

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A series of experiments were conducted to determine physiologic mechanisms of mucosal repair in the ischemia-injured intestine. The first experiment (Chapter III) investigated the contributory role of individual Cl- channels in the recovery of barrier function in ischemia-injured porcine ileum. Ischemia-injured porcine ileal mucosa was mounted in Ussing chambers. Short circuit current (Isc) and transepithelial resistance (TER) were measured in response to PGE2 and pharmacologic inhibitors of epithelial Cl- channels. Overall, results from these studies demonstrate that ClC-2-mediated intestinal
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13

Willemsen, Linette Eustachia Maria. "Intestinal barrier function: regulation of epithelial permeability and mucin expression." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/74526.

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14

Groschwitz, Katherine R. "Mast cell-mediated intestinal barrier function in homeostasis and disease." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1291389792.

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15

Gil, Cardoso Katherine. "Grape seed proanthocyanidins as modulators of the inflammatory response and barrier function in the intestine." Doctoral thesis, Universitat Rovira i Virgili, 2018. http://hdl.handle.net/10803/663376.

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La disfunció intestinal es basa en un estat proinflamatori en l'intestí i en una funció de barrera defectuosa, característiques comunes de diverses malalties cròniques intestinals, però també associades amb patologies metabòliques relacionades amb la dieta, com és l’obesitat. En aquest sentit, les proantocianidines són compostos bioactius naturals de la família dels flavonoides que han mostrat posseir propietats antiinflamatòries i que podrien tenir efectes significatius en l’entorn intestinal, influint en la fisiologia i la bioquímica de l'intestí. En aquest marc, la present tesi va ser disse
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Shifflett, Donnie Edward. "REGULATION OF COX-2 AND PGE2-DEPENDENT RECOVERY OF INTESTINAL BARRIER FUNCTION." NCSU, 2004. http://www.lib.ncsu.edu/theses/available/etd-06302004-144347/.

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The regulation of intestinal barrier function is of importance in various clinical situations. For example, in cases of ischemia/reperfusion injury, there is a disruption in the gastrointestinal barrier. This may lead to release of pro-inflammatory cytokines and neutrophil (PMN) infiltration. It has been demonstrated that COX-2, which is upregulated during inflammation, and its downstream product PGE2 are important for recovery of barrier function, yet their regulation has not been fully characterized. Study 1: To study the effects of PMNs on acutely injured mucosa, we applied PMNs isolated fr
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HU, Li-Li. "STAT3 in intestinal epithelial cells regulates barrier function and anti-bacterial response." Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p1465075.

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Thesis (M.S.)--University of California, San Diego, 2009.<br>Title from first page of PDF file (viewed July 22, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references (p. 57-63).
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Le, Nga Thi Thanh. "Regulation of Intestinal Epithelial Barrier and Immune Function by Activated T Cells." Case Western Reserve University School of Graduate Studies / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1599833768774075.

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19

Münch, Andreas. "Collagenous colitis : The influence of inflammation and bile acids on intestinal barrier function." Doctoral thesis, Linköpings universitet, Gastroenterologi och hepatologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-56291.

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Background and aims: Collagenous colitis (CC) is a diarrheal disorder with an incidence rate of 5-6/100000 inhabitants, affecting mainly middle-aged women. The diagnosis is made by histology of the colonic mucosa. Classical findings are a thickened subepithelial collagenous layer and chronic inflammation in the lamina propria. In inflammatory bowel disease (IBD) the mucosal barrier function is important in pathogenesis. The main purpose of the thesis was therefore to describe the barrier function in CC. The cause of CC is uncertain but the condition seems to be associated with bile acid malabs
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Carlsson, Anders. "Role of mast cells and probiotics in the regulation of intestinal barrier function." Doctoral thesis, Linköpings universitet, Avdelningen för kliniska vetenskaper, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-100770.

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The intestinal mucosa is the largest contact area and one of the most important barriers to the outside environment. It is highly specialized in aiding us digest and absorb nutrients. It is daily exposed to several potentially dangerous substances and microorganisms, which if they were allowed to pass into the body, could give rise to diseases. Throughout the small intestine certain sites specialized in antigen sampling are found. These sites are named Peyer’s patches and are lymphoid follicles. The epithelium covering the Peyer’s patches is called follicle-associated epithelium and is special
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Luo, Su. "Individual and combined toxicity of a mycotoxin, the deoxynivalenol and a trace metal, the cadmium on the intestine." Thesis, Toulouse, INPT, 2019. http://www.theses.fr/2019INPT0072.

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Le déoxynivalénol (DON) est une mycotoxine de type trichothécène de type B, principalement produite par le genre Fusarium. C'est l'une des mycotoxines les plus répandues, elle est largement trouvée dans les céréales et les produits dérivés des céréales. Le cadmium est un composant de la croûte terrestre et un polluant environnemental courant. C'est un métal trace non essentiel et toxique pour la santé des humains et des animaux. Bien que la toxicité individuelle du DON et du Cd ait été bien étudiée, leur effet combiné est peu étudié. L'intestin étant le premier organe ciblé par les contaminant
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Edrees, W. K. "The effect of lower limb ischaemia-reperfusion injury on intestinal permeability and the systemic inflammatory response." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391116.

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White, J. S. "Models of intestinal barrier function and their application in the study of biliary obstruction." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368529.

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Keita, Åsa. "Barrier function of the follicle-associated epithelium in stress and Crohn's disease /." Linköping : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-9271.

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Delbue, da Silva Deborah [Verfasser]. "Defective epithelial barrier function in chronic inflammation of the intestinal mucosa / Deborah Delbue da Silva." Berlin : Freie Universität Berlin, 2021. http://d-nb.info/1238074669/34.

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Lima, NoÃlia Leal. "SuplementaÃÃo de alanil-glutamina em crianÃas de uma comunidade carente de Fortaleza-CE: impacto sobre a barreira intestinal e o estado nutricional infantil." Universidade Federal do CearÃ, 2006. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=618.

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FundaÃÃo de Amparo à Pesquisa do Estado do CearÃ<br>IntroduÃÃo: Apesar do reconhecimento das alteraÃÃes intestinais associadas à desnutriÃÃo e dos efeitos trÃficos da alanil-glutamina (AG) na funÃÃo de barreira intestinal, medida pela taxa de excreÃÃo urinÃria de Lactulose: Manitol, ainda sÃo escassos os estudos para determinar o efeito de suplementaÃÃo oral da AG em crianÃas desnutridas. Objetivos: Examinar o efeito de suplementaÃÃo oral de AG ou placebo glicina (G) na funÃÃo de barreira intestinal e crescimento em crianÃas sob risco nutricional residentes na comunidade do Parque UniversitÃri
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Rodríguez, Lagunas M. José. "Papel de la cascada del ácido araquidónico en la función epitelial de barrera en un modelo de células intestinales Caco-2." Doctoral thesis, Universitat de Barcelona, 2013. http://hdl.handle.net/10803/117258.

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El epitelio intestinal forma una barrera que permite el paso de nutrientes pero restringe el de substancias potencialmente nocivas. La alteración de dicha función se relación a con enfermedades gastrointestinales como la enfermedad inflamatoria intestinal (IBD) en la que, además, la producción de eicosanoides en la mucosa intestinal se encuentra incrementada. Por ese motivo, el objetivo del presente trabajo ha sido investigar el papel de los eicosanoides producidos en la cascada del ácido araquidónico en la regulación de la función epitelial de barrera en un modelo intestinal de células Caco-2
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Keita, Åsa. "Barrier function of the Follicle-Associated Epithelium in Stress and Crohn's disease." Doctoral thesis, Linköpings universitet, Kirurgi, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-9271.

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Crohns sjukdom är en kronisk inflammatorisk tarmsjukdom av okänd orsak. Det tidigaste tecknet på Crohns sjukdom är mikroskopiska sår i det s.k. follikelassocierade epitelet (FAE) som täcker ansamlingar av immunceller i tarmen. FAE är specialiserat för att fånga innehåll från tarmen och transportera det till underliggande immunvävnad. Denna funktion är viktig för att inducera skyddande immunsvar, men den utgör också en ingångsväg för sjukdomsalstrande bakterier. Crohns sjukdom är associerat med ett kraftigt ökat immunsvar mot bakterier, och sjukdomsförloppet kan ändras av stress. Det övergripan
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Yanagihashi, Yuichi. "A novel smooth septate junction-associated membrane protein, Snakeskin, is required for intestinal barrier function in Drosophila." Kyoto University, 2012. http://hdl.handle.net/2433/157465.

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Monteiro, Sandra Maria Nunes. "Efficacy of glutamine, peptides and vitamins A and E supplementation on diarrheal disease induced by methotrexate and cholera toxin: improvement of intestinal barrier function." Universidade Federal do CearÃ, 2004. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=596.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>A desnutriÃÃo, desidrataÃÃo e agentes antineoplÃsicos, tais como o metotrexato (MTX), sÃo imediatos causadores das doenÃas diarrÃicas. Objetivando investigar a mucosite intestinal (MI) induzida por MTX, a eficÃcia das soluÃÃes de reidrataÃÃo oral (SRO) acrescidas com glutamina (Gln), alanilglutamina (Ala-Gln), Hyprol 4107 (HYP) e hyfoama (HYFO) utilizou-se a perfusÃo intestinal. E para a suplementaÃÃo com vitaminas A (VITA) e E (VITE), Gln e peptÃdeos, utilizou-se as avaliaÃÃes morfomÃtricas, metabÃlicas e a de permeabilidade intes
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Malone, Frances R. "Luminal nutrition during hepatopoietic cell transplantation and its effects on indices of intestinal barrier function in children and adults /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/7344.

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Schmitt, Romain. "S-nitrosoglutathion et perméabilité intestinale : de la mise en place d’un modèle de chambre de Ussing à l’étude de l’impact de ce donneur de monoxyde d’azote sur la barrière intestinale Involved factors in the maintenance of intestinal barrier homeostasis: a review How to guarantee results from ex vivo studies based on Ussing chamber system for studying intestinal barrier integrity and function Comparison between two derivatization methods of nitrite ion labeled with 15N applied to liquid chromatography-tandem mass spectrometry Influence of S-nitrosoglutathione metabolism on its intestinal permeability Luminal S-nitrosoglutathione effects on intestinal barrier in an ex vivo model of Ussing chamber." Thesis, Université de Lorraine, 2019. http://www.theses.fr/2019LORR0342.

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Le monoxyde d’azote (NO) est connu pour ses nombreuses actions afin de maintenir l’homéostasie de la barrière intestinale, régulant la sécrétion de mucus, le stress oxydant, le système immunitaire… Le S-nitrosoglutathion (GSNO), donneur de NO naturellement produit par l’organisme, a montré une action bénéfique sur le renforcement de la barrière intestinale. L’enjeu est aujourd’hui de comprendre les mécanismes inhérents à cette observation. Pour ce faire une étude en Chambre de Ussing a été menée. Une fois le modèle d’étude validé par le suivi de marqueurs de perméabilité, les études histologiq
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Malinowski, Maciej Borys [Verfasser]. "The direct and systemic influence of immunosuppressive drugs on intestinal glucose absorption, barrier function and chloride secretion in rat models / Maciej Borys Malinowski." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2011. http://d-nb.info/1026344786/34.

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L'Huillier, Clement. "Etude de l'impact de supplémentations en acides aminés dans un modèle murin d'anorexie associé à une activité physique. Glutamine, but not branched-chain amino acids, restores intestinal barrier function during activity-based anorexia Influence of glutamine and branched-chain amino acids supplementation during refeeding in activity-based anorectic mice." Thesis, Normandie, 2019. http://www.theses.fr/2019NORMR123.

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L’anorexie mentale est un trouble majeur du comportement alimentaire décrit dans le DSM-V affectant en majorité les femmes et se caractérisant principalement par une réduction sévère de l’apport calorique. Elle est fréquemment associée à des troubles anxiodépressifs, une hyperactivité physique ainsi qu’à des troubles fonctionnels digestifs (TFI). Bien que sa prévalence soit faible, il s’agit de la pathologie mentale avec le plus fort taux de mortalité et un risque de rechute très élevé. Le but de cette thèse était de vérifier l’efficacité d’une supplémentation orale en acides aminés tels que l
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Aguanno, Doriane. "Modulation de la fonction de barrière intestinale et des jonctions serrées par les N-acyl homosérine lactones, molécules du quorum sensing bactérien Increased jejunal permeability in human obesity is revealed by a lipid challenge and is linked to inflammation and type 2 diabetes Intestinal plasticity in response to nutrition and gastrointestinal surgery." Thesis, Sorbonne université, 2020. http://www.theses.fr/2020SORUS009.

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L'intestin abrite le principal microbiote du corps humain et exerce donc un rôle de barrière. Les jonctions serrées (JS) contrôlent la perméabilité paracellulaire des cellules épithéliales et sont des acteurs majeurs de la barrière intestinale, dérégulée dans les Maladies Inflammatoires Chroniques de l'Intestin (MICI). Les N-acyl homosérine lactones (AHL), molécules du quorum sensing, un système bactérien de communication, sont également capables d’interagir avec l'hôte. Nous avons récemment identifié l'AHL 3-oxo-C12:2, diminuée dans les MICI. Notre objectif est d’étudier l’impact de cette nou
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Juric, Marina [Verfasser]. "Segment-specific impact of TNF-[alpha]-induced inflammation on HCO3-homeostasis and epithelial barrier function in the murine intestine / Marina Juric." Hannover : Technische Informationsbibliothek und Universitätsbibliothek Hannover (TIB), 2013. http://d-nb.info/103280369X/34.

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Moeser, Adam. "Mechanisms of prostaglandin-stimulated recovery of mucosal barrier function in the ischemia-injured porcine intestine role of intestinal ion transport /." 2006. http://www.lib.ncsu.edu/theses/available/etd-04252006-123724/unrestricted/etd.pdf.

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Snelling, Anna M., Louissa Macfarlane-Smith, Kalliopi Bitzopoulou, B. S. Reddya, J. MacFiea, and M. Gatta. "Commensal bacteria do translocate across the intestinal barrier in surgical patients." 2007. http://hdl.handle.net/10454/2429.

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Shifflett, Donnie Edward. "Regulation of COX-2 and PGE₂-dependent recovery of intestinal barrier function." 2004. http://www.lib.ncsu.edu/theses/available/etd-06302004-144347/unrestricted/etd.pdf.

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Dong, Charlotte. "Mechanisms of Glucagon-like Peptide-2-mediated Effects on Intestinal Barrier Function in Health and Irinotecan-induced Enteritis." Thesis, 2013. http://hdl.handle.net/1807/42819.

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Abstract:
Glucagon-like peptide-2 (GLP-2) is an intestinal hormone that promotes gut growth through an insulin-like growth factor (IGF)-1 and intestinal epithelial (IE)-IGF-1 receptor (R)-dependent pathway. GLP-2 also promotes epithelial barrier function by as yet unknown mechanisms. I hypothesized that GLP-2-mediated effects on barrier function requires the IE-IGF-1R. Chronic GLP-2 treatment enhanced barrier function by decreasing gastrointestinal permeability in vivo and increasing jejunal resistance ex vivo. These responses were abolished in inducible IE-IGF-1R knockout (KO) animals. Additionally, ep
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Su, Hsing-Min, and 蘇倖民. "Effect of garlic oil and its organosulfur compounds－diallyl sulfide on the intestinal barrier function and T-lymphocyte function of lipopolysaccharide injected rats." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/74506793666316455717.

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Wu, Shi-Wei, and 伍世瑋. "Effects of N-Acetyl-glucosamine on the repair of intestinal mucosa barrier and immune function in DSS-induced C57BL/6J mice." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/02742640737037660835.

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Abstract:
碩士<br>中山醫學大學<br>營養學系碩士班<br>103<br>Objectives：The aim of study was to determine effects of N-acetylglucosamine (NAG) on the mucosa repair, immune function, oxidative stress and inflammatory-related pathway in a dextran sulfate sodium (DSS)-induced inflammatory bowel disease mouse model. Materials and Methods：C57BL/6J mice (age 5.5wk) were randomly divided into vehicle ( RO water) and DSS-induced groups (3% w/v DSS water) for 5 days (induction period) during which mice were fed fiber-free AIN-93 diets. In the following diet period, DSS-induced mice were further assigned to the three experimental
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