Relevant bibliographies by topics / Function of genes

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Function of genes'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Function of genes"

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Hedlund, Petter. "Genes and erectile function." Current Opinion in Urology 13, no. 5 (September 2003): 397–403. http://dx.doi.org/10.1097/00042307-200309000-00007.

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Olfson, Emily, and David A. Ross. "Genes Orchestrating Brain Function." Biological Psychiatry 82, no. 3 (August 2017): e17-e19. http://dx.doi.org/10.1016/j.biopsych.2017.06.003.

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Sharon, Amir, Alin Finkelstein, Neta Shlezinger, and Ido Hatam. "Fungal apoptosis: function, genes and gene function." FEMS Microbiology Reviews 33, no. 5 (September 2009): 833–54. http://dx.doi.org/10.1111/j.1574-6976.2009.00180.x.

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Ishiura, S., S. Suo, and N. Sasagawa. "Genes involved in cognitive function." Seibutsu Butsuri 41, supplement (2001): S10. http://dx.doi.org/10.2142/biophys.41.s10_3.

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Ivanova, Elena, and Gavin Kelsey. "Imprinted genes and hypothalamic function." Journal of Molecular Endocrinology 47, no. 2 (July 28, 2011): R67—R74. http://dx.doi.org/10.1530/jme-11-0065.

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Genomic imprinting is an important and enigmatic form of gene regulation in mammals in which one copy of a gene is silenced in a manner determined by its parental history. Imprinted genes range from those with constitutive monoallelic silencing to those, typically more remote from imprinting control regions, that display developmentally regulated, tissue-specific or partial monoallelic expression. This diversity may make these genes, and the processes they control, more or less sensitive to factors that modify or disrupt epigenetic marks. Imprinted genes have important functions in development and physiology, including major endocrine/neuroendocrine axes. Owing to is central role in coordinating growth, metabolism and reproduction, as well as evidence from genetic and knockout studies, the hypothalamus may be a focus for imprinted gene action. Are there unifying principles that explain why a gene should be imprinted? Conflict between parental genomes over limiting maternal resources, but also co-adaptation between mothers and offspring, have been invoked to explain the evolution of imprinting. Recent reports suggest there may be many more genes imprinted in the hypothalamus than hitherto expected, and it will be important for these new candidates to be validated and to determine whether they conform to current notions of how imprinting is regulated. In fully evaluating the role of imprinted genes in the hypothalamus, much work needs to be done to identify the specific neuronal populations in which particular genes are expressed, establish whether there are pathways in common and whether imprinted genes are involved in long-term programming of hypothalamic functions.
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Davies, William, Phoebe M. Y. Lynn, Dinko Relkovic, and Lawrence S. Wilkinson. "Imprinted genes and neuroendocrine function." Frontiers in Neuroendocrinology 29, no. 3 (June 2008): 413–27. http://dx.doi.org/10.1016/j.yfrne.2007.12.001.

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Juszczak, Grzegorz R., and Adrian M. Stankiewicz. "Glucocorticoids, genes and brain function." Progress in Neuro-Psychopharmacology and Biological Psychiatry 82 (March 2018): 136–68. http://dx.doi.org/10.1016/j.pnpbp.2017.11.020.

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Thiem, Suzanne M. "Baculovirus genes affecting host function." In Vitro Cellular & Developmental Biology - Animal 45, no. 3-4 (February 27, 2009): 111–26. http://dx.doi.org/10.1007/s11626-008-9170-5.

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Fonseca, Ghislaine V., José Humberto M. Tambor, Marina P. Nobrega, Rafael Santos, and Francisco G. Nobrega. "Sugarcane genes related to mitochondrial function." Genetics and Molecular Biology 24, no. 1-4 (December 2001): 175–81. http://dx.doi.org/10.1590/s1415-47572001000100024.

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Mitochondria function as metabolic powerhouses by generating energy through oxidative phosphorylation and have become the focus of renewed interest due to progress in understanding the subtleties of their biogenesis and the discovery of the important roles which these organelles play in senescence, cell death and the assembly of iron-sulfur (Fe/S) centers. Using proteins from the yeast Saccharomyces cerevisiae, Homo sapiens and Arabidopsis thaliana we searched the sugarcane expressed sequence tag (SUCEST) database for the presence of expressed sequence tags (ESTs) with similarity to nuclear genes related to mitochondrial functions. Starting with 869 protein sequences, we searched for sugarcane EST counterparts to these proteins using the basic local alignment search tool TBLASTN similarity searching program run against 260,781 sugarcane ESTs contained in 81,223 clusters. We were able to recover 367 clusters likely to represent sugarcane orthologues of the corresponding genes from S. cerevisiae, H. sapiens and A. thaliana with E-value <= 10-10. Gene products belonging to all functional categories related to mitochondrial functions were found and this allowed us to produce an overview of the nuclear genes required for sugarcane mitochondrial biogenesis and function as well as providing a starting point for detailed analysis of sugarcane gene structure and physiology.
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Vieira, Elaine, Thomas P. Burris, and Ivan Quesada. "Clock genes, pancreatic function, and diabetes." Trends in Molecular Medicine 20, no. 12 (December 2014): 685–93. http://dx.doi.org/10.1016/j.molmed.2014.10.007.

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Dissertations / Theses on the topic "Function of genes"

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Brown, Raquel Monique. "RHOX GENES FUNCTION DURING FOLLICULOGENESIS." OpenSIUC, 2011. https://opensiuc.lib.siu.edu/theses/725.

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Mammalian ovulation is a complex, hormone-dependent developmental program in which several events must take place in an ordered progression to ensure that the oocyte is competent for fertilization. This process requires the coordinated expression of many genes which must be turned on and off in the right place at the right time for proper development of the follicle. While the hormone signals from the brain that initiate ovulation are known, the master control genes which regulate this process are not well known. Homeobox proteins are potential candidates to perform as master regulators. Homeobox proteins are DNA-binding proteins that regulate the transcription of downstream genes and thereby control biological events. We recently identified a new homeobox gene cluster on the mouse X chromosome that are only expressed in reproductive tissues. These reproductive homeobox (Rhox) homeobox genes are expressed in the ovary, placenta, testis, and epididymis, and thus are good candidates to regulate both male and female reproductive tissue development and physiology. Rhox gene expression fell into three categories: Class I exhibited peak expression prior to ovulation (0-8 hours after hCG), Class II were predominantly expressed during ovulation (8-16 hours after hCG), and Class III peaked after ovulation. The slightly overlapping windows of peak Rhox gene expression suggest that these genes may regulate specific events during the ovulatory cycle. The founding member of the cluster, Rhox5, is highly expressed in granulosa cells of pre-ovulatory follicles. We previously reported that Rhox5-null female mice are viable and fertile, suggesting that RHOX5 is either not essential for ovulation, or that one of the other RHOX factors may compensate functionally in granulosa cells. In order to identify potentially redundant RHOX factors, we examined the expression patterns of all 32 Rhox genes using an eCG primed, hCG induced superovulation model, in wild-type, Rhox5-null, and heterozygous littermate mice. Expression levels of Rhox1, exhibited peak expression prior to being hormonal primed, was reduced in the Rhox5-null animal. However, Rhox8 mRNA and protein were reduced at 2h and 4h post hCG, but recovered once the follicles passed the antral stage of development. Conversely, in progesterone receptor knockout mice (PRKO), Rhox8 exhibited normal stimulation by eCG, but failed to reach its peak mRNA level at 8h post-hCG found in WT mice. This suggests a model in which Rhox8 transcription is dependent upon RHOX5 during early folliculogenesis and progesterone during the periovulatory window when RHOX5 normally wanes. Subsequent promoter analysis in granulosa cells revealed essential homeobox binding and progesterone response elements within Rhox8's 5'-flanking region. Transfection of RHOX5 and PGR expression plasmids stimulated, whereas dominant negative and mutant constructs inhibited, Rhox8 promoter activity. At present, the specific impact of misregulation of Rhox5 and Rhox8 during early folliculogenesis is not known. However, follicle counts from serially sectioned ovaries, extirpated from normal cycling animals, indicated that Rhox5-null mice possess ~50% fewer follicles than heterozygous littermates. Loss of RHOX5 in Sertoli cells results in male subfertility characterized by poor germ cell survival due in part to the misregulation of metabolism promoting genes. One of these genes, Ins2, is also stimulated by RHOX5 and RHOX8 in granulosa cells, suggesting impaired insulin signaling may contribute reduced follicle development in Rhox5-null ovaries.
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Fitter, David W. "Function of GOLDEN2-like genes in Arabidopsis." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393774.

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Sengul, Meryem Senay. "Two Odorant-Binding Protein Genes in Mosquitoes: Comparative Genomics, Expression, and Function." Diss., Virginia Tech, 2008. http://hdl.handle.net/10919/26578.

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Insect Odorant-Binding Proteins (OBPs) are small, water-soluble molecules that solubilize hydrophobic odorant molecules in the sensillum lymph and transport them to their cognate receptors in the olfactory receptor neurons. With the availability of the genome sequence of the African malaria mosquito, Anopheles gambiae, there has been a profound interest in the characterization and functional analyses of Obp genes in order to understand the molecular basis of mosquito host-seeking behavior. However, no direct evidence has been found for specific functions of any mosquito OBPs. In this study, I describe the comparative genomics and expression analyses on two mosquito Obp genes (Obp1 and Obp7) as well as efforts to determine their functions. Both of these Obp genes were identified in Anopheles stephensi and only Obp7 gene was identified in Anopheles quadriannulatus by screening bacterial artificial chromosome (BAC) libraries of these species. Comparative analyses revealed several interesting features including segments of conserved non-coding sequences (CNSs) that contain potential regulatory elements relevant to olfactory tissue development and blood-feeding. The expression profiles of these genes were examined in detail in the Asian malaria mosquito An. stephensi. Obp1 and Obp7 transcripts were significantly higher in females than male mosquitoes and they were predominantly found in the antenna, which is the primary olfactory organ of mosquitoes. Twenty-four hours after a blood meal, mRNA levels of these two genes were significantly reduced in the maxillary palp and proboscis, referred to as secondary olfactory organs of mosquitoes. These findings collectively indicate that Obp1 and Obp7 genes in An. stephensi likely function in female olfactory response and may be involved in behaviors related to blood-feeding. To investigate the function of these Obp genes more directly, a Sindbis virus based expression system is established to knockdown the two Obp gene orthologs in Aedes aegypti. The effective knockdown of Obp1 and Obp7 genes (8 and 100-fold, respectively) is accomplished in female mosquito olfactory tissues. The potential for a systematic analysis of the molecular players involved in mosquito olfaction using this newly developed technique is discussed. Such analysis will provide the foundation for interfering with mosquito host-seeking behavior for the prevention of disease transmission.
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Auburn, Richard Peter. "Screening for genes involved in Drosophila neuromuscular function." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621081.

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Andrews, Tallulah. "Clustering genes by function to understand disease phenotypes." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:06bfce1f-4ae0-4715-9ee3-290c43ae9b18.

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Developmental disorders including: autism, intellectual disability, and congenital abnormalities are present in 3-8% of live births and display a huge amount of phenotypic and genetic heterogeneity. Traditionally, geneticists have identified individual monogenic diseases among these patients but a majority of patients fail to receive a clinical diagnosis. However, the genomes of these patients frequently harbour large copynumber variants (CNVs) but their interpretation remains challenging. Using pathway analysis I found significant functional associations for 329 individual phenotypes and show that 39% of these could explain the patients’ multiple co-morbid phenotypes; and multiple associated genes clustered within individual CNVs. I showed there was significantly more such clustering than expected by chance. In addition, the presence of a multiple functionally-related genes is a significant predictor of CNV pathogenicity beyond the presence of known disease genes and size of the CNV. This clustering of functionally-related genes was part of a broader pattern of functional clusters across the human genome. These genome-wide functional clusters showed tissuespecific expression and some evidence of chromatin-domain level regulation. Furthermore, many genome-wide functional clusters were enriched in segmental duplications making them prone to CNV-causing mutations and were frequently seen disrupted in healthy individuals. However, the majority of the time a pathogenic CNV affected the entire functional cluster, where as benign CNVs tended to affect only one or two genes. I also showed that patients with CNVs affecting the same functional cluster are significantly more phenotypically similar to each other than expected even if their CNVs do not affect any of the same genes. Lastly, I considered one of the major limitations in pathway analysis, namely ascertainment biases in functional information due to the prioritization of genes linked to human disease, and show how the modular nature of gene-networks can be used to identify and prioritize understudied genes.
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Lopes, Miguel. "The function of Msx genes in vascular development." Paris 6, 2011. http://www.theses.fr/2011PA066343.

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Une des étapes les plus importantes de la formation de circulatoire système est la mise en place d’un réseau vasculaire. Les vaisseaux sont composés d’une couche endothéliale intérieure et d’une couche murale extérieure (cellules musculaires lisses [VSMCs] pour les artères et les veines ; péricytes pour les capillaires et les veinules). Dans l’embryon de souris, Msx1lacZ et Msx2lacZ sont exprimés dans des VSMCs (vaisseaux intersomitiques et carotide respectivement) et dans quelques cellules endothéliales de l’aorte (Goupille et al. , 2008). Chez l’adulte, Msx1lacZ et Msx2lacZ sont surtout exprimés dans les cellules musculaires lisses des artères périphériques. De plus, Msx1lacZ est aussi exprimé dans les péricytes des capillaires du muscle squelettique. Nous avons inactivé les deux gènes Msx spécifiquement dans des cellules murales en combinant les allèles Msx1lacz, Msx2lox et α-Sm22Cre (Lopes et al. In press). Des analyses de projection tomographique optique ont démontré un excès de bifurcations dans les vaisseaux céphaliques des embryons mutants à E11. 5. Les artères vertébrale et carotide présentent un calibre augmenté, directement associé à une réduction de couverture par les cellules musculaires lisses. Tirant profit d’un nouvel allèle Msx1CreERT2, nous avons démontré, par traçage cellulaire, que l’anomalie initiale concernait une population de précurseurs des VSMCs. Le phénotype anormal est dû à une réduction du signal BMP dans ces précurseurs, qui entraîne la sous-expression des gènes codant pour les métalloprotéase-2 et métalloprotéase-9. Ces deux gènes sont impliqués dans la migration des précurseurs et leur intégration dans la couche murale
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Lind, Johanna. "Memory, genes, and brain imaging : relating the APOE gene to brain function and structure /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-110-4/.

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Elstob, Philip Ronald. "Hox gene function and cell identity in Drosphila." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.272353.

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Heinrich, Stefanie May. "Human SP-A- genes, structure, function- and lung diseases." Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-138347.

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Cwajna, Mark. "The sequential function of Hox genes in limb development." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=96918.

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Loss-of-function experiments have demonstrated that the establishment of the limb architecture relies upon the function of HoxA and HoxD genes, which are required from early limb bud stages onwards. While there is a tight link between Hox genes and skeletal development, Hox function during osteochondrogenesis remains unclear.Without HoxA/D genes there is an early development arrest, resulting in drastic truncation of the limb skeleton. The severity of this mutation made its analysis uninformative regarding the specific function of HoxA/D genes at later stages of limb development. In order to investigate Hox function during later stages of osteochondrogenesis, we generated conditional Hox inactivations in the osteochondrogenic lineage to circumvent the early affect of HoxA/D loss of function.Comparing the ubiquitous Hox deficiency and specific inactivation in limb skeleton progenitors suggests that HoxA/D genes are needed early on for normal bone patterning and also at later stages to support normal bone growth.
Des expériences de perte de fonction ont démontré que la mise en place de l'architecture du squelette des membres requière la fonction des gènes HoxA et HoxD. Cependant, le rôle de ces gènes au cours de l'osteochondrogenèse est inconnu. Le but de mon projet de recherche était de définir ce rôle.En absence des gènes HoxA/D, le développement des membres est compromis dès les stades précoces, bien avant le début de l'osteochondrogenèse. Par conséquent, j'ai généré et analysé des inactivations conditionnelles des gènes HoxA/D dans le lignage osteochondrogenic afin d'établir le rôle de ces gènes au cours du développement osseux. Mes résultats montrent que la croissance des os en développement requiert la fonction des gènes HoxA/D dans le lignage osteochodrogénique. De plus, la comparaison entre l'inactivation ubiquitaire et conditionnelle permet de mieux comprendre la contribution précoce et tardif des gènes HoxA/D dans la formation du squelette des membres.
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Books on the topic "Function of genes"

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Genes and cardiovascular function. New York: Springer, 2011.

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Ostadal, Bohuslav, Makoto Nagano, and Naranjan S. Dhalla, eds. Genes and Cardiovascular Function. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1.

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NATO Advanced Research Workshop on Major Histocompatibility Complex--Genes/Molecules/Function (1987 Bar Harbor, Me.). H-2 antigens: Genes, molecules, function. New York: Plenum Press, 1987.

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Cortical deficits in schizophrenia: From genes to function. New York: Springer, 2008.

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Tsang, Michael Wai Kok. Characterisation of structure and function of mouse dishevelled genes. Dublin: University College Dublin, 1996.

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name, No. Tumor suppressor genes ; v. 2: Regulation, function, and medicinal applications. Totowa, NJ: Humana Press, 2003.

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Shearman, Claire Amy. Structure, function and regulation of modulation genes of Rhizobium leguminosarum. Norwich: University ofEast Anglia, 1986.

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Day, Trevor. Genetics: Investigating the function of genes and the science of heredity. New York: Rosen Central, 2012.

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International, Seminar/Workshop on the Molecular Structure Function and Assembly of ATP Synthases (1987 Honolulu Hawaii). Molecular structure, function, and assembly of the ATP synthases. New York: Plenum Press, 1989.

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International Meeting on Cholinesterases (3rd 1990 La Grande Motte, France). Cholinesterases: Structure, function, mechanism, genetics, and cell biology. Washington, DC: American Chemical Society, 1991.

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Book chapters on the topic "Function of genes"

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Dang, Chi V., and Linda A. Lee. "Retroviruses, Cancer Genes, and Tumor Suppressor Genes." In c-Myc Function in Neoplasia, 37–64. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22681-0_2.

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Hubacek, Jaroslav A., and Rudolf Poledne. "Genes and Plasma Lipids in Czech Slavic Population." In Genes and Cardiovascular Function, 149–57. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_15.

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Braveny, Pavel. "Johann Gregor Mendel: “Father of Modern Genetics”." In Genes and Cardiovascular Function, 3–5. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_1.

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Schaffer, Stephen W., and Chian Ju Jong. "MELAS Syndrome: Mediated by Impaired Taurinomethyluridine Synthesis." In Genes and Cardiovascular Function, 93–100. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_10.

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Poledne, Rudolf, and Jaroslav A. Hubacek. "Genetics of Myocardial Infarction." In Genes and Cardiovascular Function, 103–11. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_11.

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Ozaki, Kouichi, and Toshihiro Tanaka. "Genetic Background of Myocardial Infarction." In Genes and Cardiovascular Function, 113–20. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_12.

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Vasku, Anna, Jiri Blahak, Daniel Baumgartner, and Julie Bienertova-Vasku. "Angiotensin Converting Enzyme I/D Polymorphism and Cardiovascular Risk: Disclosed Story." In Genes and Cardiovascular Function, 121–34. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_13.

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Ravingerova, Tana, Adriana Adameova, Slavka Carnicka, Tara Kelly, Martina Nemcekova, Jana Matejikova, and Antigone Lazou. "PPARs and Myocardial Response to Ischemia in Normal and Diseased Heart." In Genes and Cardiovascular Function, 135–48. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_14.

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Leenen, Frans H. H., Shahrier Amin, Alexandre F. R. Stewart, and Frederique Tesson. "Genetic Basis of Salt-Sensitive Hypertension in Humans." In Genes and Cardiovascular Function, 161–75. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_16.

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Kunes, Jaroslav, Michaela Kadlecova, and Josef Zicha. "Gene–Environment Interactions: Their Role in Hypertension Development." In Genes and Cardiovascular Function, 177–84. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_17.

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Conference papers on the topic "Function of genes"

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Ong, Bruce A., Julian L. Allen, Jason Caboot, Oscar H. Mayer, Patrick Sleiman, and Hakon Hakonarson. "Gene Network Analysis Identifies Novel Lung Function Genes." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6024.

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Ma, Wencai, Xinwu Guo, Chenguang Wang, Yunyan Gu, Yuannv Zhang, Jiguang Xia, and Zheng Guo. "Extracting multi-function features for cancer genes." In 2010 Seventh International Conference on Fuzzy Systems and Knowledge Discovery (FSKD). IEEE, 2010. http://dx.doi.org/10.1109/fskd.2010.5569547.

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Ong, Bruce A., Jason Caboot, Julian Allen, Joseph McDonough, Patrick Sleiman, Jin Li, and Hakon Hakonarson. "Gene Network Analysis In A Pediatric Cohort Identifies Novel Lung Function Genes." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3488.

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Sun, Jingchun, Leng Han, and Zhongming Zhao. "Schizophrenia Genes: Characteristics of Function and Protein Interaction Networks." In 2008 International Conference on Biomedical Engineering And Informatics (BMEI). IEEE, 2008. http://dx.doi.org/10.1109/bmei.2008.227.

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Ray, Oliver, and Christopher H. Bryant. "Inferring the Function of Genes from Synthetic Lethal Mutations." In 2008 International Conference on Complex, Intelligent and Software Intensive Systems. IEEE, 2008. http://dx.doi.org/10.1109/cisis.2008.124.

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Lange, Nancy E., Sheryl Rifas-Shiman, Diane R. Gold, Matthew Gillman, and Augusto A. Litonjua. "Vitamin D Related Genes And Childhood Asthma And Lung Function." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5605.

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Becker, E. J., A. Faiz, M. Van Den Berge, W. Timens, P. S. Hiemstra, G. Liu, X. Zhang, et al. "A Bronchial Airway Gene Expression Signature of Future Lung Function Decline Is Enriched in XBP1-Regulated Genes." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7234.

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Chen, Yu Long, Su Gai Yin, and Yao Song Wu. "The Function and Interaction of Genes Related to Esophageal Squamous Cell Cancer." In 2011 5th International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2011. http://dx.doi.org/10.1109/icbbe.2011.5780083.

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Lussier, Yves A., Joanne Berghout, Francesca Vitali, Kenneth S. Ramos, Maricel Kann, and Jason H. Moore. "Reading Between the Genes: Computational Models to Discover Function from Noncoding DNA." In Pacific Symposium on Biocomputing 2018. WORLD SCIENTIFIC, 2017. http://dx.doi.org/10.1142/9789813235533_0046.

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Kerkhof, Marjan, H. M. Boezen, Alet H. Wijga, Bert Brunekreef, Henriette A. Smit, Johan C. de Jongste, Carel Thijs, et al. "Replicated COPD Genes, Transient Early Wheeze And Lung Function In Early Childhood." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3803.

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Reports on the topic "Function of genes"

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V, DE CRECY-LAGARD, and HANSON A. D. PHYLOGENOMICS - GUIDED VALIDATION OF FUNCTION FOR CONSERVED UNKNOWN GENES. Office of Scientific and Technical Information (OSTI), January 2012. http://dx.doi.org/10.2172/1032489.

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Kathuria, Hasmeena. Identification and Function of Ets Target Genes Involved in Lung Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada594333.

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Kathuria, Hasmeena. Identification and Function of Ets Target Genes Involved in Lung Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada573588.

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Kathuria, Hasmeena. Identification and Function of Ets Target Genes Involved in Lung Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada555912.

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Verma, Inder, and Quan Zhu. A Genetic Screen for Genes Involved in BRCA 1 Tumor Suppressor Function. Fort Belvoir, VA: Defense Technical Information Center, March 2007. http://dx.doi.org/10.21236/ada469482.

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Epstein, Jonathan A. Developing a Zebrafish Model of NF1 for Structure-Function Analysis and Identification of Modifier Genes. Fort Belvoir, VA: Defense Technical Information Center, April 2008. http://dx.doi.org/10.21236/ada485806.

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Epstein, Jonathan A. Developing a Zebrafish Model of NF1 for Structure-Function Analysis and Identification of Modifier Genes. Fort Belvoir, VA: Defense Technical Information Center, April 2009. http://dx.doi.org/10.21236/ada502775.

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Epstein, Jonathan A., and A. T. Look. Developing a Zebrafish Model of NF1 for Structure-Function Analysis and Identification of Modifier Genes. Fort Belvoir, VA: Defense Technical Information Center, April 2012. http://dx.doi.org/10.21236/ada570844.

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Borodovsky, M. New Markov Model Approaches to Deciphering Microbial Genome Function and Evolution: Comparative Genomics of Laterally Transferred Genes. Office of Scientific and Technical Information (OSTI), April 2013. http://dx.doi.org/10.2172/1073499.

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Selleck, Scott B. Understanding the Function of Tuberous Sclerosis Complex Genes in Neural Development: Roles in Synapse Assembly and Axon Guidance. Fort Belvoir, VA: Defense Technical Information Center, February 2012. http://dx.doi.org/10.21236/ada603854.

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