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Journal articles on the topic 'Functional renal reserve'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Amiel, C., F. Blanchet, G. Friedlander, and A. Nitenberg. "Renal Functional Reserve." Nephrology Dialysis Transplantation 5, no. 9 (1990): 763–70. http://dx.doi.org/10.1093/ndt/5.9.763.

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2

Ratanasrimetha, Praveen, Miguel Quirich, and Sorot Phisitkul. "Renal functional reserve." Southwest Respiratory and Critical Care Chronicles 6, no. 25 (2018): 26–30. http://dx.doi.org/10.12746/swrccc.v6i25.481.

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Serum creatinine and glomerular filtration rate (GFR) are the current standard tests tomeasure kidney function. The baseline GFR does not reflect full function of the kidney sincehuman kidneys do not always work at full capacity. Similarly, serum creatinine is not a sensitivemeasure for kidney function or injury. In healthy individuals the GFR physiologically increasesin response to certain stresses or stimuli, such as protein loading.Renal functional reserve (RFR) is defined as the difference between the maximalglomerular filtration rate (generally determined after oral or intravenous protein

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3

Friedlander, G., F. Blanchet, and C. Amiel. "Renal functional reserve." Toxicology Letters 46, no. 1-3 (1989): 227–35. http://dx.doi.org/10.1016/0378-4274(89)90131-8.

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4

Palsson, Ragnar, and Sushrut S. Waikar. "Renal Functional Reserve Revisited." Advances in Chronic Kidney Disease 25, no. 3 (2018): e1-e8. http://dx.doi.org/10.1053/j.ackd.2018.03.001.

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5

Ronco, Claudio, Rinaldo Bellomo, and John Kellum. "Understanding renal functional reserve." Intensive Care Medicine 43, no. 6 (2017): 917–20. http://dx.doi.org/10.1007/s00134-017-4691-6.

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6

G. Dj, Ishkabulova, Kholmuradova Z.E., and Rahmonkulov Sh. I. "CHANGE OF FUNCTIONAL KIDNEY RESERVE IN CHILDREN IN DYSMETABOLIC NEPHROPATHIES." International Journal of Medical Sciences And Clinical Research 3, no. 10 (2023): 47–54. http://dx.doi.org/10.37547/ijmscr/volume03issue10-09.

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The urgency of the problem. The development of renal failure depends on secondary hemodynamic metabolic factors than on the activity of the primary pathological process. Both non-modifiable and potentially modifiable risk factors for the development of renal failure have been identified (2, 7). Among the potentially reversible risk factors for the development of renal failure, glomerular hyperfiltration and interglomerular hypertension under the influence of angiotensin II (ANG II) areof high importance.

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7

Ueno, Akira, and Kiichiro Tago. "GLOMERULAR FUNCTION AND RENAL FUNCTIONAL RESERVE." Japanese Journal of Urology 81, no. 12 (1990): 1787–99. http://dx.doi.org/10.5980/jpnjurol1989.81.1787.

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8

Tago, K., Y. Takihana, and A. Ueno. "Renal Functional Reserve in Kidney Transplants." Aktuelle Urologie 21, S 1 (1990): 37–41. http://dx.doi.org/10.1055/s-2008-1060670.

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9

Dhaene, Michel, Jean-Pierre Sabot, Yvan Philippart, Jean-Marc Doutrelepont, Jean-Louis Vanherweghem, and Charles Toussaint. "Renal Functional Reserve of Transplanted Kidneys." Nephron 44, no. 2 (1986): 157–58. http://dx.doi.org/10.1159/000184224.

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10

Koratala, Abhilash, and Amir Kazory. "Renal functional reserve and pregnancy outcomes." Kidney International 92, no. 3 (2017): 768. http://dx.doi.org/10.1016/j.kint.2017.04.038.

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11

PECLY, I. M. D., V. GENELHU, and E. A. FRANCISCHETTI. "Renal functional reserve in obesity hypertension." International Journal of Clinical Practice 60, no. 10 (2006): 1198–203. http://dx.doi.org/10.1111/j.1742-1241.2006.01037.x.

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12

Sackmann, H., T. Tran-Van, I. Tack, H. Hanaire-Broutin, J. P. Tauber, and J. L. Ader. "Renal functional reserve in IDDM patients." Diabetologia 41, no. 1 (1998): 86–93. http://dx.doi.org/10.1007/s001250050871.

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13

Fliser, D., M. Zeier, R. Nowack, and E. Ritz. "Renal functional reserve in healthy elderly subjects." Journal of the American Society of Nephrology 3, no. 7 (1993): 1371–77. http://dx.doi.org/10.1681/asn.v371371.

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The increase in GFR after an amino acid (AA) load, the so-called renal functional reserve, is impaired in the aged rat. Whether the renal functional reserve predicts the progression of renal disease in humans is controversial, but it is possible that age-related alterations of renal hemodynamics are relevant for the evolution of renal disease in the elderly. We compared renal hemodynamics before and after an AA infusion in 15 healthy normotensive subjects of young age (seven women, eight men; median age, 26 yr; range, 23 to 32) and in 10 subjects of old age (six women, four men; median age, 70

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14

Gaipov, Abduzhappar, Yalcin Solak, Nurlan Zhampeissov, et al. "Renal functional reserve and renal hemodynamics in hypertensive patients." Renal Failure 38, no. 9 (2016): 1391–97. http://dx.doi.org/10.1080/0886022x.2016.1214052.

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15

Nakamura, H., Y. Daidoh, T. Asano, and T. Mochizuki. "Higher renal functional reserve in well-functioning renal grafts." Transplantation Proceedings 29, no. 1-2 (1997): 231–32. http://dx.doi.org/10.1016/s0041-1345(96)00074-7.

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16

McDougal, W. Scott. "Measurement of Renal Functional Reserve in Children." Journal of Urology 175, no. 1 (2006): 313. http://dx.doi.org/10.1097/00005392-200601000-00111.

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17

Mansy, H., D. Patel, S. Tapster, A. Torrance, and R. Wilkinson. "Four Ways to Recruit Renal Functional Reserve." Clinical Science 69, s12 (1985): 12P. http://dx.doi.org/10.1042/cs069012p.

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18

Hellerstein, S., M. Berenbom, P. Erwin, N. Wilson, and S. DiMaggio. "Measurement of Renal Functional Reserve in Children." Journal of Urology 175, no. 1 (2006): 313. http://dx.doi.org/10.1016/s0022-5347(05)00095-9.

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19

Gozhenko, Anatoliy I., Anna S. Kuznetsova, Yelena S. Kuznetsova, and Tatiana N. Byts. "FUNCTIONAL RENAL RESERVE IN DIABETIC NEPHROPATHY PATIENTS." Pathophysiology 25, no. 3 (2018): 220–21. http://dx.doi.org/10.1016/j.pathophys.2018.07.133.

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20

Cleper, Roxana, Miriam Davidovitz, Rafael Halevi, and Bella Eisenstein. "Renal functional reserve after acute poststreptococcal glomerulonephritis." Pediatric Nephrology 11, no. 4 (1997): 473–76. http://dx.doi.org/10.1007/s004670050319.

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21

Sharma, Aashish, Marìa Jimena Mucino, and Claudio Ronco. "Renal Functional Reserve and Renal Recovery after Acute Kidney Injury." Nephron Clinical Practice 127, no. 1-4 (2014): 94–100. http://dx.doi.org/10.1159/000363721.

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22

DeSanto, Natale Gaspare, Giovambattista Capasso, Pietro Anastasio, et al. "Renal Functional Reserve in Children with and without Renal Disease." Nephron 59, no. 1 (1991): 1–6. http://dx.doi.org/10.1159/000186508.

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23

Ivanov, D. D., A. I. Gozhenko, and L. M. Savytska. "INDIVIDUALIZATION OF RENOPROTECTION IN DEPENDENCE FROM ESTIMATED GLOMERULAR FILTRATION RATE AND RENAL FUNCTIONAL RESERVE." Nephrology (Saint-Petersburg) 23, no. 1 (2019): 9–14. http://dx.doi.org/10.24884/1561-6274-2019-23-1-9-14.

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Renoprotection – a system of measures primarily pharmacological control, contributing to the continued preservation of renal function. Any medical activity or medicines that lead to a slowdown, suspension or reversion of eGFR could be considered as renoprotective. Currently, renoprotective drugs with evidence-based efficacy include angiotensin-converting enzyme inhibitors (ACEIs), and recently SGLT2 inhibitors. It is known that the therapeutic efficacy of renoprotective drugs depends on the initial eGFR. Hence, choosing renoprotection medicines, it is necessary to consider not only the estimat

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24

Ader, J. L., I. Tack, D. Durand, T. Tran-Van, L. Rostaing, and J. M. Suc. "Renal functional reserve in kidney and heart transplant recipients." Journal of the American Society of Nephrology 7, no. 8 (1996): 1145–52. http://dx.doi.org/10.1681/asn.v781145.

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Renal functional impairment paradoxically often seems less severe in kidney than in heart-transplant recipients (KTR and HTR, respectively) when both are submitted to cyclosporine therapy. Renal functional reserve (RFR), elicited by a 3-h intravenous amino acid infusion, was examined in 12 KTR and 13 HTR at 7 to 8 months, appropriately compared with either eight one-kidney or 12 two-kidney healthy control subjects (1K.C and 2K.C, respectively). Baseline GFR was 54 +/- 4 mL/min in KTR and 71 +/- 4 mL/min in HTR (P < 0.05). During amino acid infusion, the maximum increase in GFR (which repres

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25

Cassidy, Michael J. D., and Rosemarie M. Beck. "Renal Functional Reserve in Live Related Kidney Donors." American Journal of Kidney Diseases 11, no. 6 (1988): 468–72. http://dx.doi.org/10.1016/s0272-6386(88)80081-7.

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26

Semiz, Serap, Iffet Bircan, GÜLsÜN GÜLay Yilmaz, Binnur KarayalÇIn, and Ayfer GÜR GÜVen. "Renal functional reserve in insulin dependent diabetic children." Pediatrics International 40, no. 4 (1998): 341–44. http://dx.doi.org/10.1111/j.1442-200x.1998.tb01944.x.

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27

Bertoni, E., A. Rosati, M. Zanazzi, et al. "Functional reserve and hyperfiltration after cadaveric renal transplantation." Transplantation Proceedings 33, no. 7-8 (2001): 3363–64. http://dx.doi.org/10.1016/s0041-1345(01)02448-4.

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28

Bach, Dieter, Heidi Mrowka, Stefan Schauseil, and Bernd Grabensee. "Renal Functional Reserve in Patients with IgA Glomerulopathy." Renal Failure 16, no. 5 (1994): 617–27. http://dx.doi.org/10.3109/08860229409044889.

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29

Guleria, Sandeep, Monish Aron, SN Mehta, SC Tiwari, MG Karmarkar, and Rekha Sharma. "Renal functional reserve in live related kidney donors." Indian Journal of Urology 19, no. 1 (2002): 63. http://dx.doi.org/10.4103/0970-1591.21083.

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30

Hozhenko, A. I., A. V. Kravchuk, V. M. Sirman, O. P. Nikitenko, and L. V. Romaniv. "Functional Renal Reserve: Physiological Value of Renal Reserve and Substantiation of the Method of Its Determination." KIDNEYS, no. 4.14 (December 9, 2015): 7–11. http://dx.doi.org/10.22141/2307-1257.0.4.14.2015.74890.

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31

Jufar, Alemayehu H., Yugeesh R. Lankadeva, Clive N. May, Andrew D. Cochrane, Rinaldo Bellomo, and Roger G. Evans. "Renal functional reserve: from physiological phenomenon to clinical biomarker and beyond." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 319, no. 6 (2020): R690—R702. http://dx.doi.org/10.1152/ajpregu.00237.2020.

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Glomerular filtration rate (GFR) is acutely increased following a high-protein meal or systemic infusion of amino acids. The mechanisms underlying this renal functional response remain to be fully elucidated. Nevertheless, they appear to culminate in preglomerular vasodilation. Inhibition of the tubuloglomerular feedback signal appears critical. However, nitric oxide, vasodilator prostaglandins, and glucagon also appear important. The increase in GFR during amino acid infusion reveals a “renal reserve,” which can be utilized when the physiological demand for single nephron GFR increases. This

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32

Husain-Syed, Faeq, Fiorenza Ferrari, Horst-Walter Birk, et al. "Pre-transplant renal functional reserve and renal function after lung transplantation." Journal of Heart and Lung Transplantation 39, no. 9 (2020): 970–74. http://dx.doi.org/10.1016/j.healun.2020.05.011.

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33

Galimova, Saida Sh, Nuriya I. Abdrakhmanova, Konstantin S. Mochalov, et al. "Functional reserve of oxygen-dependent metabolism of phagocytes in blood of patients with severe hemorrhagic fever with renal syndrome complicated by acute renal failure." I.P. Pavlov Russian Medical Biological Herald 33, no. 1 (2025): 61–70. https://doi.org/10.17816/pavlovj487160.

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INTRODUCTION: Hemorrhagic fever with renal syndrome (HFRS) is one of the most common zoonotic infections, and acute renal failure (ARF) is a serious complication of HFRS. A mechanism inalienable from the pathogenesis of HFRS is activation of oxygen-dependent metabolism of phagocytes and the generation of reactive oxygen species (ROS) by them. Overactivation of oxygen-dependent processes induces oxidative damage to molecular structures and underlies the development of ARF leading to marked alterations of reserve capacities of phagocytes. AIM: Evaluation of the functional reserve of oxygen-depen

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34

EARLE, KENNETH ANTHONY, SEEMA MEHROTRA, RAYMOND NEIL DALTON, ELIZABETH DENVER, and RAMASAMYIYER SWAMINATHAN. "Defective Nitric Oxide Production and Functional Renal Reserve in Patients with Type 2 Diabetes Who Have Microalbuminuria of African and Asian Compared with White Origin." Journal of the American Society of Nephrology 12, no. 10 (2001): 2125–30. http://dx.doi.org/10.1681/asn.v12102125.

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Abstract. Diabetic nephropathy is a leading cause of end-stage renal failure. Its incidence is higher and is increasing in persons of Indo-Asian and African-Caribbean (African-Asian) compared with those of white origin. Nitric oxide deficiency is associated with progressive renal disease. It was hypothesized that differences in the capacity to increase glomerular filtration (functional renal reserve) would exist between these racial groups in relation to nitric oxide availability. Patients with type 2 diabetes of African-Asian (n = 9) and white (n = 9) origin with microalbuminuria were studied

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35

Khimion, Liudmyla, Liliia Tymoshchuk та Mariia Rybytska. "The Significance of Renal Functional Reserve in Еssential Hypertension". Family Medicine, № 4 (30 грудня 2016): 81–84. http://dx.doi.org/10.30841/2307-5112.4.2016.248534.

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In the structure of causes of the end1stage chronic kidney disease is essential hypertension (EH) which takes the leading role. One of the pathogenic mechanisms of hypertensive nephropathy is a violation of intrarenal blood flow, which clinically manifesting with hyperfiltration. The objective: of the frequency of subclinical renal damage of patients with stage I EAH by water and salt loading and comparing these data with the standard criteria of essential nephropathy with hypertension. Patients and methods. The study involved 30 patients aged 27 to 59 years with diagnosed grade 1

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36

NUNLEY, JULIA R., ANNE KING, TOM COMSTOCK, MARC POSNER, CAROL MARSHALL та DOMENIC A. SICA. "CYCLOSPORINEʼS EFFECT ON FUNCTIONAL RESERVE IN THE RENAL ALLOGRAFT". Transplantation 51, № 1 (1991): 190–92. http://dx.doi.org/10.1097/00007890-199101000-00030.

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37

Gaipov, Abduzhappar, Yalcin Solak, Nurlan Zhampeissov, et al. "SP111RENAL FUNCTIONAL RESERVE AND RENAL HEMODYNAMICS IN HYPERTENSIVE PATIENTS." Nephrology Dialysis Transplantation 31, suppl_1 (2016): i122. http://dx.doi.org/10.1093/ndt/gfw159.20.

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38

De Nicola, Luca, Roland C. Blantz, and Francis B. Gabbai. "Renal functional reserve in treated and untreated hypertensive rats." Kidney International 40, no. 3 (1991): 406–12. http://dx.doi.org/10.1038/ki.1991.226.

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39

Ladefoged, Soren D., Erling Pedersen, Martin Skielboe, Hans K. Lauritsen, and Niels Fogh-Andersen. "Renal Functional Reserve After an Acute Intravenous Lipid Load." Journal of Renal Nutrition 3, no. 4 (1993): 186–90. http://dx.doi.org/10.1016/s1051-2276(12)80093-8.

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40

Woitas, R. P., J. Heller, D. Iven, U. Spengler, and T. Sauerbruch. "Nitric oxide and renal functional reserve in compensated cirrhosis." Gastroenterology 108, no. 4 (1995): A1199. http://dx.doi.org/10.1016/0016-5085(95)29097-4.

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41

Losito, A., F. Fortunati, I. Zampi, and A. Del Favero. "Impaired renal functional reserve and albuminuria in essential hypertension." BMJ 296, no. 6636 (1988): 1562–64. http://dx.doi.org/10.1136/bmj.296.6636.1562.

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42

Khimion, Liudmyla, Liliia Tymoshchuk та Mariia Rybytska. "The Significance of Renal Functional Reserve in Еssential Hypertension". Family Medicine, № 4 (5 жовтня 2016): 81–84. https://doi.org/10.30841/2307-5112.4.2016.248534.

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In the structure of causes of the end-stage chronic kidney disease is essential hypertension (EH) which takes the leading role. One of the pathogenic mechanisms of hypertensive nephropathy is a violation of intrarenal blood flow, which clinically manifesting with hyperfiltration. <strong>The objective: </strong>of the frequency of subclinical renal damage of patients with stage I EAH by water and salt loading and comparing these data with the standard criteria of essential nephropathy with hypertension. <strong>Patients and methods. </strong>The study involved 30 patients aged 27 to

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43

Reverte, Virginia, Antonio Tapia, Analia Loria, Francisco Salazar, M. Teresa Llinas, and F. Javier Salazar. "COX2 inhibition during nephrogenic period induces ANG II hypertension and sex-dependent changes in renal function during aging." American Journal of Physiology-Renal Physiology 306, no. 5 (2014): F534—F541. http://dx.doi.org/10.1152/ajprenal.00535.2013.

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This study was performed to test the hypothesis that ANG II contributes to the hypertension and renal functional alterations induced by a decrease of COX2 activity during the nephrogenic period. It was also examined whether renal functional reserve and renal response to volume overload and high sodium intake are reduced in 3–4- and 9–11-mo-old male and female rats treated with vehicle or a COX2 inhibitor during nephrogenic period (COX2np). Our data show that this COX2 inhibition induces an ANG II-dependent hypertension that is similar in male and female rats. Renal functional reserve is reduce

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44

Molina, Enrique, José Herrera, and Bernardo Rodríguez-Iturbe. "The renal functional reserve in health and renal disease in school age children." Kidney International 34, no. 6 (1988): 809–16. http://dx.doi.org/10.1038/ki.1988.254.

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45

Dedov, I. I., N. A. Mukhin, M. V. Shestakova, et al. "Renal Functional Reserve in Diabetic Patients without Clinical Nephropathy: Comparisons with Renal Morphology." Diabetic Medicine 8, S2 (1991): S43—S47. http://dx.doi.org/10.1111/j.1464-5491.1991.tb02155.x.

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46

Khakimova, Dilyara Makhmutrievna, Adelya Nailevna Maksudova, and Il'dar Gazimdzhanovich Salikhov. "Renal functional reserve and tubular function in patents with type 2 diabetes mellitus." Diabetes mellitus 14, no. 2 (2011): 82–85. http://dx.doi.org/10.14341/2072-0351-5641.

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Aim. To study renal functional reserve and partial functions in patents with type 2 diabetes mellitus in the absence of renal lesionsMaterials and methods. We examined 42 patients (17 men and 24 women) aged 38-69 (mean 49.8?8.3) years with DM2 4.6?2.6 yr in duration.Control group comprised 32 practically healthy subjects. Intrarenal hemodynamics was estimated from RFR values. Ethanolamine, uric acid, Ca,and P levels were measured in sera and 24-hr urine; daily excretion of ammonia and aminonitrogen in the urine was determined. Results. The patients were divided into 2 groups based on the resul

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47

van Londen, Marco, Nicolien Kasper, Niek R. Hessels, et al. "Renal functional reserve capacity before and after living kidney donation." American Journal of Physiology-Renal Physiology 315, no. 6 (2018): F1550—F1554. http://dx.doi.org/10.1152/ajprenal.00064.2018.

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Compensatory gomerular filtration rate (GFR) increase after kidney donation results in a GFR above 50% of the predonation value. The renal functional reserve (RFR) assessed by the renal response to dopamine infusion (RFRdopa) is considered to reflect functional reserve capacity and is thought to be a tool for living donor screening. However, it is unknown if the RFRdopa predicts long-term kidney function. Between 1984 and 2017, we prospectively measured GFR (125I-iothalamate) and RFR by dopamine infusion in 937 living kidney donors. We performed linear regression analysis of predonation RFRdop

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48

Ivanov, D. D., A. I. Gozhenko, and L. N. Savitskaya. "Renoprotection and its association with eGFR and renal functional reserve." KIDNEYS 7, no. 4 (2018): 238–44. http://dx.doi.org/10.22141/2307-1257.7.4.2018.148512.

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49

Freemas, Jessica A., Morgan L. Worley, Mikaela C. Gabler, et al. "Renal Functional Reserve Is Attenuated During Mild Passive Heat Stress." Medicine & Science in Sports & Exercise 53, no. 8S (2021): 95. http://dx.doi.org/10.1249/01.mss.0000760224.44351.64.

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50

Nicola, L. D., R. C. Blantz, and F. B. Gabbai. "Renal Functional Reserve in the Early Stage of Experimental Diabetes." Diabetes 41, no. 3 (1992): 267–73. http://dx.doi.org/10.2337/diab.41.3.267.

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