Relevant bibliographies by topics / Fusion musculaire

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Fusion musculaire'

Author: Grafiati
Published: 25 January 2025

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Journal articles on the topic "Fusion musculaire"

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Girardi, Francesco, Anissa Taleb, Lorenzo Giordani, Bruno Cadot та Fabien Le Grand. "La signalisation TGFβ contrôle la fusion cellulaire et la régénération musculaire". Les Cahiers de Myologie, № 19 (червень 2019): 33–34. http://dx.doi.org/10.1051/myolog/201919013.

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Mounier, Rémi, та Bénédicte Chazaud. "L’axe PPARγ-GDF3 dans les macrophages contrôle la fusion myogénique au cours de la régénération musculaire". médecine/sciences 33, № 5 (2017): 466–69. http://dx.doi.org/10.1051/medsci/20173305003.

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Kumari, Pushpa. "Histopathological changes and elaboration of Mucosubstances induced by metacid in the large intestine of fingerlings of Channa punctatus (Bloch)." PROCEEDINGS OF THE ZOOLOGICAL SOCIETY OF INDIA 23, no. 02 (2024): 275. https://doi.org/10.59467/pzsi.2024.23.275.

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The Communication reports the metacid (an organophosphate pesticide) induced histopathological and histochemical alterations in the large intestine of fingerlings of a fresh water air breathing snake headed murrel fish, Channa punctatus (Bloch). The fishes were exposed to sublethal concentration (8.4ppm) of metacid for 96 hrs. Histologically large intestine contained usual four layers mucosa, submucosa, muscularis and serosa. Metacid intoxication caused histopathologically changes in the large intestine were intestinal villi was totally destroyed, the mucosal layer was entirely separated from
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Hoh, Daniel J., Michael Y. Wang, and Stephen L. Ritland. "Anatomic Features of the Paramedian Muscle-Splitting Approaches to the Lumbar Spine." Operative Neurosurgery 66, suppl_1 (2010): ons—13—ons—25. http://dx.doi.org/10.1227/01.neu.0000350866.25760.33.

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Abstract Background: Intermuscular approaches can expose the lumbar spine and minimize muscular trauma and injury. The segmental anatomy of the posterior lumbar musculature allows surgical access through separation of muscle groups and fascicles and provides one to develop intermuscular working channels while preserving the integrity of the muscles and their function. In addition, preservation of the accompanying neurovascular bundles minimizes blood loss, tissue atrophy, and pain. With these approaches, a variety of procedures for decompression, discectomy, interbody fusion, or pedicle screw
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Chargé, Sophie B. P. "Interleukine-4 et fusion des cellules musculaires." médecine/sciences 19, no. 12 (2003): 1185–87. http://dx.doi.org/10.1051/medsci/200319121185.

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Verma, Abhisht, Isha Garg, and Anamendra Sharma. "Klippel–Feil Syndrome with Limited Rotation Range of Motion: Our Observation on Radiographic Evaluation of Cervical Spine Case Series." Journal of Bone and Joint Diseases 39, no. 2 (2024): 90–94. http://dx.doi.org/10.4103/jbjd.jbjd_13_24.

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Abstract Background: The aim is to identify radiographic changes in the cervical spine or occipitocervical junction in patients with restricted lateral bending and lateral rotation due to Klippel–Feil syndrome. It is important to consider this altered mobility or fusion when planning surgery for hypermobile segments. Observation: In Klippel–Feil syndrome, restricted rotation is typically associated with occipitocervical involvement, but it can also be caused by long fusion of the cervical vertebrae alone. If left untreated in early childhood, this fusion and the relatively mobile lower segment
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Diab, Mohammad. "Scapulothoracic Fusion for Facioscapulohumeral Muscular Dystrophy." Journal of Bone and Joint Surgery (American) 87, no. 10 (2005): 2267. http://dx.doi.org/10.2106/jbjs.d.02952.

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DIAB, MOHAMMAD, BASIL T. DARRAS, and FREDERIC SHAPIRO. "SCAPULOTHORACIC FUSION FOR FACIOSCAPULOHUMERAL MUSCULAR DYSTROPHY." Journal of Bone and Joint Surgery-American Volume 87, no. 10 (2005): 2267–75. http://dx.doi.org/10.2106/00004623-200510000-00017.

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Brook, P. D., J. D. Kennedy, L. M. Stern, A. D. Sutherland, and B. K. Foster. "Spinal Fusion in Duchenne's Muscular Dystrophy." Journal of Pediatric Orthopaedics 16, no. 3 (1996): 324–31. http://dx.doi.org/10.1097/01241398-199605000-00006.

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Miller, Freeman, Colin F. Moseley, and Jan Koreska. "SPINAL FUSION IN DUCHENNE MUSCULAR DYSTROPHY." Developmental Medicine & Child Neurology 34, no. 9 (2008): 775–86. http://dx.doi.org/10.1111/j.1469-8749.1992.tb11516.x.

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Dissertations / Theses on the topic "Fusion musculaire"

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BLENEAU, SEVERINE. "Etude par rmn bi-dimensionnelle de la fusion des myoblastes et de la regeneration musculaire." Paris 6, 1999. http://www.theses.fr/1999PA066063.

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La regeneration musculaire met en jeu des cellules myoblastes qui se repliquent puis fusionnent pour reconstituer la fibre musculaire. Les etats de replication et de fusion de myoblastes en culture ont ete etudies par rmn bi-dimensionnelle du proton. Les spectres des cellules en fusion presentent des signaux lipidiques intenses, contrairement a ceux des cellules en replication. D'autres cellules presentent egalement des signaux lipidiques intenses en fonction de leur etat. Dans l'objectif d'etablir une comparaison, des cellules transformees, des lymphocytes actives, des cellules vegetales en f
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Lý, Hà My. "Characterization of a novel molecular pathway linking metabolic regulation and muscle stem cell fate." Electronic Thesis or Diss., Lyon 1, 2024. http://www.theses.fr/2024LYO10193.

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Le muscle strié squelettique possède une capacité de régénération grâce à une population de cellules souches musculaires (CSM ou cellules satellites) qui gardent une capacité à exprimer un programme myogénique lors d'une lésion musculaire. Une perte de la capacité myogénique des CSM est associée à la perte musculaire avec l'âge, mais aussi à des maladies invalidantes telles que les myopathies. Une des caractéristiques de la fonction des CSMs est leur plasticité métabolique qui supporte le programme myogénique. Sur la base de travaux antérieurs du laboratoire mettant en évidence le rôle central
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Girardi, Francesco. "TGFbeta signalling pathway in muscle regeneration : an important regulator of muscle cell fusion." Electronic Thesis or Diss., Sorbonne université, 2019. http://www.theses.fr/2019SORUS114.

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La régénération musculaire s’appuie sur une réserve de cellules souches résidant dans le muscle appelées cellules satellites (MuSCs). Les MuSCs sont quiescentes et peuvent s’activer à la suite d’une blessure du muscle afin de former des progéniteurs amplificateurs (myoblastes) qui se différencieront et fusionneront pour former de nouvelles myofibres. Durant ce processus, un réseau complexe de voies de signalisation est impliqué, parmi lequel la signalisation du facteur de croissance transformant bêta (TGFβ) joue un rôle fondamental. Précédents rapports ont proposé de nombreuses fonctions pour
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De, la celle Marie. "Etude de deux aspects de la myogenèse chez l'embryon de poulet : la transition epitheliomesenchymateuse et la fusion des myoblastes." Aix-Marseille 2, 2009. http://theses.univ-amu.fr.lama.univ-amu.fr/2009AIX22077.pdf.

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Chez les vertébrés, les somites, structures transitoires du mésoderme, sont à l'origine de tous les muscles squelettiques des membres et du corps. Les muscles embryonnaires se forment en deux phases séquentielles à partir du dermomyotome épithélial (partie dorsale du somite). 1ère phase : les cellules des quatre lèvres du dermomyotome délaminent pour former le myotome primaire, composé de fibres musculaires mononucléées et post‐mitotiques. 2nde phase : les cellules de la partie centrale du dermomyotome subissent une transition épithélio‐mésenchymateuse (EMT) qui permet l'entrée massive dans le
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Wu, Melissa P. "Enhancing Myoblast Fusion for Therapy of Muscular Dystrophies." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:10740.

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Skeletal muscle is a major organ comprising 30-40% of the human body mass. The coordination of processes resulting in mature muscle requires many genes, and their loss can result in debilitating muscle disorders. Of the strategies being developed to cure muscle diseases, enhancement of the natural process of muscle cell fusion in existing or introduced myogenic cells has great therapeutic potential. In this work, we determined whether a drug that stimulates proliferation and fusion of myoblasts could alleviate murine Duchenne muscular dystrophy. We also studied the necessity of a gene that is
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Lafuste, Peggy Authier François-Jérôme. "Mécanismes moléculaires impliqués dans la fusion des cellules précurseurs myogéniques humains." Créteil : Université de Paris-Val-de-Marne, 2004. http://doxa.scd.univ-paris12.fr:80/theses/th0214091.pdf.

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Fongy, Anais. "Implication potentielle des protéines de fusion mitochondriale dans l'ontogenèse des processus bioénergétiques musculaires chez l'oiseau." Thesis, Lyon 1, 2013. http://www.theses.fr/2013LYO10276/document.

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Les jeunes oiseaux exposés au froid assurent leur homéothermie en stimulant les oxydations mitochondriales dans les muscles squelettiques. L’exposition prolongée au froid accroit les capacités de thermogenèse musculaire grâce à une plasticité bioénergétique mitochondriale dont le contrôle reste hypothétique. Chez les mammifères, des protéines de fusion (les mitofusines (Mfns) et OPA1(OPtic Atrophy 1)) participent au remaniement des réseaux dynamiques mitochondriaux dans de multiples types cellulaires. Le but de ce travail de thèse était de caractériser l’expression d’homologues aviaires des pr
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Félix, de Melo Juliana. "Molécules de fusion et facteurs de transcription dans les macrophages et cellules musculaires squelettiques de rats : l'effet de la dénutrition néonatale." Compiègne, 2012. http://www.theses.fr/2012COMP2000.

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Cette thèse en co-tutelle a été effectuée à l’Université de Technologie de Compiègne (UTC - France) et à l’Université Fédérale de Pernambuco (UFPE - Brésil). En France, les activités ont été réalisées dans l’unité de Biomécanique-Bioingénierie (CNRS UMR 6600) de l’UTC, et au Brésil, dans le Laboratoire de Physiologie de la Nutrition Naíde Teodósio (LAFINNT) du Département de Nutrition de l’UFPE, dans le Laboratoire d’Immunopathologie Keizo Asami (LIKA-UFPE) et au sein du Centre de Recherche Aggeu Magalhães (Fiocruz PE). Une déficience nutritionnelle pendant la période critique du développement
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Petrany, Michael J. "Consequences of Cell Fusion and Multinucleation for Skeletal Muscle Development and Disease." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1595847866440328.

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Jacquemin, Virginie. "Mécanismes cellulaires et moléculaires impliqués dans l'hypertrophie des myotubes humains induite par l'IGF-1." Paris 7, 2006. http://www.theses.fr/2006PA077195.

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L'IGF-1 (Insulin-like Growth factor 1) est un facteur de croissance sécrété par le foie, mais aussi exprimé localement au niveau du muscle où il joue un rôle majeur dans la régulation de la masse. Ainsi, surexprimé au niveau du muscle de souris, l'IGF-1 est capable d'induire une hypertrophie des fibres musculaires et de limiter la perte de masse musculaire liée à l'âge. Dans cette étude, réalisée sur modèle de myoblastes primaires humains, la capacité de l'IGF-1 à induire une hypertrophie des myotubes a pu être retrouvée. En traitant les cultures par l'IGF-1 après 3 jours de différenciation, n
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Books on the topic "Fusion musculaire"

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Alami, Ilias, and Adam D. Dixon. The Spectre of State Capitalism. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/9780198925224.001.0001.

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Abstract The state is back, and it means business. Since the turn of the twenty-first century, state-owned enterprises, sovereign funds, and policy banks have vastly expanded their control over assets and markets. Concurrently, governments have experimented with increasingly assertive modalities of statism, from techno-industrial policies and spatial development strategies to economic nationalism and trade and investment restrictions. This book argues that we are currently witnessing a historic arc in the trajectories of state intervention, characterized by a drastic reconfiguration of the sta
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Book chapters on the topic "Fusion musculaire"

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Garcia, Luis, Patrick Dreyfus, Martine Pinçon-Raymond, et al. "Phenotypic and Functional Reversion of Muscular Dysgenesis by Heterotypic Fibroblast-Myotube Fusion In Vitro." In Myoblast Transfer Therapy. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5865-7_16.

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Christensen, Nedra, and Howard Saal. "Cleft Lip and/or Cleft Palate and Other Craniofacial Anomalies." In Pediatric Nutrition In Chronic Diseases And Developmental Disorders. Oxford University PressNew York, NY, 2005. http://dx.doi.org/10.1093/oso/9780195165647.003.0025.

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Abstract Cleft lip with or without cleft palate (CL/P) and cleft palate (CP) result from incomplete merging and fusion of embryonic processes during formation of the face. Microforms of clefting are also known to occur, the best example being submucous CP. In this condition there is incomplete fusion of the muscular layers of the velum (soft palate) with fusion of the overlying mucosa. In children and adults with submucous CP, the clinical features are highly variable, ranging from an isolated, inconspicuous clinical finding of bifid uvula to severe speech (hypernasality) and feeding difficulties, often with attendant chronic otitis media. Cleft palate and CL/P have multiple etiologies as well. They are often associated with underlying syndromes. Additionally, orofacial clefts are classified by anatomical involvement. This includes unilateral cleft lip, bilateral cleft lip, cleft lip with cleft palate (generally indicating involvement of the primary and secondary palates) cleft palate, and cleft palate secondary to Pierre Robin sequence4 (Figs. 25–1, 25–2, and 25–3). Hundreds of disorders have been associated with orofacial clefts, including both genetic and teratogenic conditions. Among the most common causes of cleft palate is the Pierre Robin sequence. This specifically refers to disruption of normal elevation of the fusion of the palatal shelves secondary to micrognathia and a posterally placed tongue (Fig. 25–2). Like all orofacial clefts, Pierre Robin sequence can be caused by multiple underlying disorders.
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Sousa, Hugo, José Roberto Pimenta Godoy, Rogério Wagner da Silva, et al. "VENTRE ADICIONAL NO MÚSCULO QUADRÍCEPS FEMORAL: IMPLICAÇÕES BIOMECÂNICAS E CLÍNICAS." In Variações Anatômicas: o avanço da ciência no Brasil. Editora Científica Digital, 2023. http://dx.doi.org/10.37885/230412726.

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O músculo quadríceps femoral é um dos componentes do compartimento anterolateral da coxa, cobrindo quase toda a face anterior e a lateral do fêmur. É formado pelos músculos vasto lateral (MVL), vasto medial, vasto intermédio e reto femoral. Há relatos de variações na morfologia macroscópica e microscópica do quadríceps femoral na literatura. Os tópicos relacionados às variações, podem ser sumarizados como: acréscimo de uma cabeça, deixando de ser quadríceps e passando a ser designado como quinticeps femoris; fusão entre vasto lateral e vasto intermediário; tipo de fibra muscular ao longo do ventre e tamanho da fibra muscular. Por tudo isso, este trabalho tem como objetivo descrever os aspectos morfológicos e morfométricos do achado de um ventre muscular adicional no vasto lateral. Durante dissecação de rotina, do membro inferior esquerdo de um cadáver do sexo masculino, adulto, com idade indeterminada, foi reconhecida uma variação no MVL, localizada no terço distal do músculo em questão. constatou-se que subjacente ao trato iliotibial, havia um ventre muscular bem delimitado, com epimísio próprio, distinto ao MVL, cujas origens encontravam-se na linha áspera do fêmur e no septo intermuscular, e cujas inserções no retináculo lateral do joelho e na borda lateral da patela. Assim, ao analisar estas características, convencionou-se designar esta variação encontrada de músculo vasto lateral oblíquo (MVLO). Diante do exposto, ressalta-se que presente variação, MVLO, independente da frequência, deve ser de conhecimento dos profissionais de saúde que trabalham ou pretendem trabalhar com diagnósticos e/ou intervenções cirúrgicas relacionadas ao complexo articular do joelho.
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Conference papers on the topic "Fusion musculaire"

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Silva, Tarcisio Rubens da, Rayana Elias Maia, and Taísa de Abreu Marques Nogueira. "Progressive thoracolumbar scoliosis culminating in the diagnosis of young pompe disease: case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.044.

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Introduction: Pompe’s disease is a neuromuscular condition caused by a metabolic disorder of autosomal recessive inheritance. The deficit of acid alpha-glucosidase causes accumulation of glycogen in the lysosomes of the striated and cardiac muscle. It presents in childhood: hypotonia and cardiorespiratory impairment; but at late-onset: axial and waist muscle weakness. Case report: Patient, female, 20 years old, non-consanguineous parents, with good intra-uterus fetal mobility, was born by cesarean delivery weighing 3.7 kilograms and 51 centimeters. She first walked without support and spoke he
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Belluco, Paolo, Monica Bordegoni, and Umberto Cugini. "A Techinque Based on Muscular Activation for Interacting With Virtual Environments." In ASME 2011 World Conference on Innovative Virtual Reality. ASMEDC, 2011. http://dx.doi.org/10.1115/winvr2011-5539.

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Interacting with computers by using the bodily motion is one of the challenging topics in the Virtual Reality field, especially as regards the interaction with large scale virtual environments. This paper presents a device for interacting with a Virtual Reality environment that is based on the detection of the muscular activity and movements of the user by the fusion of two different signals. The idea is that through muscular activities a user is capable of moving a cursor in the virtual space, and making some actions through gestures. The device is based on an accelerometer and on electromyog
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LoMauro, Antonella, Marianna Romei, Sandra Gandossini, Nereo Bresolin, Maria Grazia D'Angelo, and Andrea Aliverti. "Scoliosis and spinal fusion (SF) correlate with spirometry but not with breathing pattern at rest in Duchenne muscular dystrophy (DMD)." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4141.

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Mieites, Verónica, Arturo Pardo, José A. Gutiérrez-Gutiérrez, et al. "Colorimetric fusion of attenuation and birefringence in OCT signatures: a screening tool for evaluating muscular degradation in alpha-sarcoglican deficit murine models." In Optical Coherence Imaging Techniques and Imaging in Scattering Media, edited by Maciej Wojtkowski, Yoshiaki Yasuno, and Benjamin J. Vakoc. SPIE, 2023. http://dx.doi.org/10.1117/12.2670555.

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Lustosa, Alysson Bastos, João Paulo Holanda Soares, Iago Mateus Rocha Leite, Rilciane Maria dos Reis Ribeiro, and Olívio Feitosa Costa Neto. "SECRETORY CARCINOMA BREAST IN A YOUNG MAN." In XXIV Congresso Brasileiro de Mastologia. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s1075.

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Introduction: Secretory carcinoma of the breast is a rare disease, accounting for approximately 0.15% of breast cancer cases. This entity was first described in a child in 1966. However, one of the largest case series with SEER data, encompassing a total of 190 patients, showed that the median age at diagnosis was 56 years, and it can affect both sexes, being much more common in women. In this same series, 58% and 40% of patients were positive for estrogen and progesterone hormone receptors, respectively. Most cases (86.86%) were well to moderately differentiated tumors without lymph node invo
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