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Dissertations / Theses on the topic 'GABA'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Kragler, Andrea. "GABA-Transporter." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-9774.

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2

Picard, Raymonde. "Charakterisierung funktioneller Domänen für GABA und Furosemid auf GABAA-Rezeptoren." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=971995141.

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3

Ong, Jennifer. "GABA and GABA-receptors in the enteric nervous system /." Title page, contents and summary only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09pho582.pdf.

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4

Davies, Martin. "The GABA transporter and the regulation of the GABA¦A receptor." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1993. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq23915.pdf.

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5

Aanesen, Arthur. "Gaba and human spermatozoa : characterization and regulation of gaba transport proteins /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980925aane.

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6

Mendu, Suresh Kumar. "Role of GABA and GABAA Channels in T lymphocytes and Stem cells." Doctoral thesis, Uppsala universitet, Institutionen för neurovetenskap, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-172541.

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GABA (gamma-aminobutyric acid) is best known for its physiological function in the central nervous system. In the brain GABA is the main inhibitory neurotransmitter where it decreases excitability of neurons and neuronal networks. The balance between excitation evoked by glutamate and inhibition evoked by GABA is the base from where the brain works. It is fair to say that glutamate is like the gas-pedal and GABA the brake that keeps the brain running at a normal speed. But, it is not only in the brain that GABA is taking a part in a physiological process vital to life. GABA is present in bl

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7

Gardner-Fortier, Catherine. "Développement d'un fromage fonctionnel renfermant un composé bioactif, l'acide gama-aminobutyrique (GABA)." Thesis, Université Laval, 2011. http://www.theses.ulaval.ca/2011/28143/28143.pdf.

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8

CHAMBON, JEAN-PIERRE. "Caracterisation de derives pyridazinyl-gaba comme ligands antagonistes du recepteur gaba-a." Université Louis Pasteur (Strasbourg) (1971-2008), 1987. http://www.theses.fr/1987STR13002.

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9

Chambon, Jean-Pierre. "Caractérisation de dérivés pyridazinyl-gaba comme ligands antagonistiques du récepteur gaba-A." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37603758b.

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10

Namwindwa, Ernest Sinvula. "GABA and glutamate mimetics." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.376436.

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11

Babateen, Omar M. "GABA signaling regulation by GLP-1 receptor agonists and GABA-A receptors modulator." Doctoral thesis, Uppsala universitet, Fysiologi, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-282431.

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GABA (γ-aminobutyric acid) is the main neuroinhibitory transmitter in mammalian brains. It binds to GABA-A and GABA-B receptors. The GABA-A receptors are ligand-gated chloride channels. A variety of GABA-A receptor agonists and antagonists have been developed to study the GABA-mediated inhibition and to explore new medications. In this thesis I have examined the role of GABA in brain tumors and the effects of the metabolic hormone GLP-1 on GABAergic signaling in neurons. I studied if GABA-A receptors subunits were expressed and formed functional ion channels in the glioblastoma cell line U304

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12

Ferguson, Shane C. D. J. "GABA and retino-tectal development." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0019/MQ55206.pdf.

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13

Tabor, Alethea Bernice. "Synthesis of GABA-T inhibitors." Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305797.

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14

Hosie, Alastair Marshall. "GABA receptors of Drosophila melanogaster." Thesis, University of Cambridge, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324973.

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15

Ali, Saima. "Regulation of the cell surface expression and the function of GABA¦BR1a by GABA¦BR2." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0027/MQ50433.pdf.

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16

Chen, Jianping. "The Effects of Chronic Ethanol Intake on the Allosteric Interaction Between GABA and Benzodiazepine at the GABAA Receptor." Thesis, University of North Texas, 1992. https://digital.library.unt.edu/ark:/67531/metadc501231/.

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This study examined the effects of chronic ethanol intake on the density, affinity, and allosteric modulation of rat brain GABAA receptor subtypes. In the presence of GABA, the apparent affinity for the benzodiazepine agonist flunitrazepam was increased and for the inverse agonist R015-4513 was decreased. No alteration in the capacity of GABA to modulate flunitrazepam and R015-4513 binding was observed in membranes prepared from cortex, hippocampus or cerebellum following chronic ethanol intake or withdrawal. The results also demonstrate two different binding sites for [3H]RO 15-4513 in rat ce

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17

Rahman, Mozibur. "Effects of neuroactive steroids on the recombinant GABAA receptor in Xenopus oocyte." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1112.

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18

Ebrik-Al, Akoum Sahar. "Nouveaux ligands du récepteur gaba b, nouvelles pyrrolidin-2-ones : études chimique et pharmacologique." Lille 2, 1995. http://www.theses.fr/1995LIL2P261.

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19

ANSAR, M'HAMMED. "Agonistes et antagonistes de l'acide gamma-aminobutyrique au niveau du recepteur gaba-b : etudes chimique et pharmacologique." Lille 2, 1995. http://www.theses.fr/1995LIL2P251.

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20

Mortensen, Martin. "GABAa receptor pharmacology : electrophysiological studies of agonist activity on reconstituted human GABAa receptors /." [Cph.] : Department of Pharmacology, The Royal Danish School of Pharmacy, 2002. http://www.dfh.dk/phd/defences/martinmortensen.htm.

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21

BOUCHET, MARIE-JEANNE. "Marquage irreversible du recepteur gaba#a." Université Louis Pasteur (Strasbourg) (1971-2008), 1996. http://www.theses.fr/1996STR13281.

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L'objectif de notre recherche est le marquage irreversible du site de liaison de l'acide -aminobutyrique (gaba#a) en vue de sa caracterisation moleculaire. Le recepteur ionotrope gaba#a est un hetero-oligomere regroupant autour d'un canal chlorure plusieurs sites de liaison qui interagissent entre eux: gaba, benzodiazepine, canal, barbiturates, steroides, ainsi que d'autres moins bien definis. Le recepteur gaba#a aurait une structure pentamerique, composee d'au moins trois des seize sous-unites ( 1-6, 1-4, 1-3, , p1-2) que la biologie moleculaire a permis mettre en evidence a ce jour, sans qu'

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22

Langlois, Anaïs. "Rôle du BDNF dans le développement des synapses GABAergiques de l'hippocampe de rat." Thesis, Aix-Marseille, 2012. http://www.theses.fr/2012AIXM4089/document.

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Le cerveau immature est le siège de processus développementaux qui permettent de passer d'une structure primitive à un réseau mature et fonctionnel. L'activité synaptique spontanée générée dans le système nerveux en développement joue un rôle fondamental dans ces processus. Un des principaux moyens par lesquels cette activité peut être traduite en changement phénotypique au niveau neuronal est la sécrétion de neurotrophines. Les neurotrophines sont sécrétées par les neurones et contrôlent toutes les étapes du développement neuronal. Dans l'hippocampe en développement, la neurotrophine principa

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23

Kramer, Vasko [Verfasser]. "Synthese 18 F-markierter Liganden zur Visualisierung der GABA-Bindungsstelle des GABA A-Rezeptors mittels PET / Vasko Kramer." Mainz : Universitätsbibliothek Mainz, 2012. http://d-nb.info/1025406451/34.

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24

Marandi, Nima. "Entwicklungsspezifische Wirkmechanismen der Neurotransmitter GABA und Glyzin." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-10831.

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25

McGonigle, Ian Vincent. "Molecular pharmacology of an insect GABA receptor." Thesis, University of Cambridge, 2010. https://www.repository.cam.ac.uk/handle/1810/226857.

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Cys-loop receptors are ligand-gated ion channels that are involved in fast synaptic neurotransmission in the central and peripheral nervous system. The Cys-loop receptor RDL ('resistant to dieldrin') is a GABA-gated chloride channel from Drosophila melanogaster and is a major target site for insecticides. The aim of this dissertation was to characterise RDL receptors with particular focus on the agonist binding site. To assess the potency of a range of GABA analogues on RDL receptors, I expressed receptors in Xenopus oocytes and used voltage-clamp electrophysiology to detect receptor responses

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26

Jax, Sabine. "Cyclische GABA-Derivate und Nicotinanaloga als Maleinimiden /." München : Hieronymus, 1999. http://www.gbv.de/dms/bs/toc/300875053.pdf.

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27

Alyami, A. M. "Pharmacology of benzodiazepines and GABA in intestine." Thesis, University of Bradford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.384251.

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28

Taylor, Alison R. "Electrophysiological studies of GABA receptors in insects." Thesis, Open University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278439.

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29

Casalotti, S. O. "Molecular characterization of the GABA [A, ] receptor." Thesis, Imperial College London, 1988. http://hdl.handle.net/10044/1/46989.

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30

Bilbe, Graeme. "Molecular studies on the gaba-benzodiazepine receptor." Thesis, Imperial College London, 1985. http://hdl.handle.net/10044/1/37639.

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31

Sudjadi. "Analysis of cloned genes for GABA catabolism." Thesis, University of Leicester, 1991. http://hdl.handle.net/2381/35163.

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A putative hpa+ clone has been isolated allowing an Hpa-Hpc+ mutant CD03 to growth on 4-HPA. Detection of a 4-HPA monooxygenase from extracts was unsuccessful. When it became apparent that gab genes had been cloned the emphasis switched to detailed analysis of those genes. The gab genes were isolated using suppression of a mutant CT101 defective in the sad gene. It seems that the cloned gabDT genes encoding NADP-dependent SSA dehydrogenase and GABA transaminase are responsible for these, not the gabD gene only. Possible explanation of these phenomena are described. E.coli C and K-12 cannot uti

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32

Smith, K. R. "Molecular determinates of GABA-A receptor trafficking." Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1212232/.

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Gamma amino-butyric acid type A receptors (GABAA receptors) are the main mediators of inhibitory neurotransmission in the CNS. These chloride permeable ligand-gated ion channels are intrinsic to maintaining the excitatory/inhibitory balance of neuronal circuits, a balance that is essential to ensure proper workings of the nervous system. The strength of the inhibitory synapse can be rapidly altered by GABAA receptor trafficking mechanisms, which are tightly regulated by many interacting proteins and post-translational modifications. The identification of GABAA receptor associated proteins is a

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33

Twelvetrees, A. E. "Molecular mechanisms of GABA-A receptor trafficking." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/815656/.

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Gamma-aminobutyric acid type A receptors (GABA-A receptors) are the main sites of fast synaptic inhibition in the brain. Regulating the numbers of GABA-A receptors at post-synaptic sites is a key mechanism for regulating the strength of inhibitory synaptic transmission. How GABA-A receptors are rapidly transported to synapses is unknown although the trafficking protein huntingtin associated protein 1 (HAP1) is known to regulate surface GABA-A receptor numbers by an uncharacterised mechanism. This study focuses on how HAP1 regulates GABA-A receptor trafficking. This research demonstrates that G

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34

Masiulis, Simonas. "Structural studies of human GABA-A receptors." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:159d7e7f-3654-45cd-a261-4283100b906d.

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Type-A Î3-amino-butyric acid receptors (GABA<sub>A</sub>Rs) are pentameric ligand-gated ion channels (pLGICs), which mediate the majority of fast inhibitory neurotransmission in the animal central nervous system. Their dysfunction is related to numerous conditions including epilepsy, insomnia, anxiety, panic disorders, depression and schizophrenia. GABA<sub>A</sub>Rs are therefore major targets of clinically important drugs, including benzodiazepines and the intravenous general anaesthetics etomidate and propofol, as well as endogenous modulators, for example neurosteroids. Despite recent pro

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35

Richter, Grant. "Gaba Drugs For Motor Recovery After Stroke." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/25088.

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Clinical reports describe a rare but striking recovery of brain function post stroke after taking the non-benzodiazepine ‘Z-drug’ zolpidem. The underlying mechanism for this is still unknown. This body of work investigates the relationship of Z-drugs and stroke and serves three purposes. Firstly, it furthers our understanding of the particular GABAA receptor subtypes Z-drugs modulate. Benzodiazepines bind between the α-γ2 subunit interfaces (where α is α1, α2, α3, or α5). An analogous binding site is present when γ2 is substituted for γ1 or γ3 but has limited and conflicting pharmacological i

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36

McDonald, Emily F. "Expression of GABA receptors in human sclera." Thesis, Queensland University of Technology, 2014. https://eprints.qut.edu.au/68604/2/Emily_McDonald_Thesis.pdf.

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37

SITAMZE, JEAN-MARIE. "Synthese d'antagonistes du gaba a proprietes ascaricides." Strasbourg 1, 1993. http://www.theses.fr/1993STR15071.

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38

Riffault, Baptiste. "Plasticité GABAergique et épilepsie : focus sur le proBDNF." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM4005/document.

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Le facteur neurotrophique dérivé du cerveau (BDNF) synthétisé sous la forme d'un précurseur (proBDNF) qui peut être clivé pour donner sa forme mature (mBDNF). Le mBDNF et le proBDNF produisent des réponses physiologiques opposées par l'activation de deux classes distinctes de récepteurs transmembranaires : respectivement, le récepteur TrkB et p75NTR. Le ratio mature/pro-BDNF est un élément important impliqué dans la plasticité synaptique, la formation des circuits neuronaux et in fine les fonctions cognitives. Les altérations dans ce clivage peuvent ainsi expliquer l’émergence de conditions pa

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39

Engström, Thomas. "GABA-agonister som antipsykotika : En litteraturstudie kring GABA-agonisters potentiella roll som terapeutisk behandling av symptom förekommande vid schizofreni." Thesis, Umeå universitet, Farmakologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-137189.

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40

Buckley, Stella Tracey. "GabaA receptor-mediated neurotransmission in human alcoholic brain /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17286.pdf.

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41

VANDEVOORDE, VALERIE. "Antagonistes des recepteurs gaba-a : etude structurale et pharmacologique comparative." Strasbourg 1, 1989. http://www.theses.fr/1989STR15070.

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42

Ko, Francoise Dulcinea. "Effect of GABARAP (GABA§A-receptor-associated protein) on the interaction between the dopamine D¦5 and GABA§A receptors." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ58857.pdf.

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43

Glykys, Joseph Charalambos. "GABA[A] receptor subunits mediating tonic inhibition in the hippocampus and the main source of GABA responsible for their activation." Diss., Restricted to subscribing institutions, 2007. http://proquest.umi.com/pqdweb?did=1464110651&sid=1&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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44

Raster, Peter [Verfasser], and Burkhard [Akademischer Betreuer] König. "Synthesis of diarylethenes by cycloaddition & GABA-amides and photoswitchable GABAA-receptor ligands / Peter Raster. Betreuer: Burkhard König." Regensburg : Universitätsbibliothek Regensburg, 2014. http://d-nb.info/1068055804/34.

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45

Höffken, Oliver. "GABA-abhängige Modulation trainingsinduzierter Plastizität im menschlichen Kortex." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=967260124.

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46

Mohamed, Diana. "Kan GABA-transporthämmare fungera som läkemedel mot epilepsi?" Thesis, Linnaeus University, School of Natural Sciences, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-5694.

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<p>Epilepsi är ingen speciell sjukdom utan ett symtom på en hjärnskada eller störd nervcellsfunktion i hjärnan. Epileptiska anfall beror på abnorm urladdning i hjärnans nervceller. Idag lever omkring 60 000 d.v.s. 0,5-1 % av Sveriges befolkning med epilepsi. Risken att drabbas är störst under det första levnadsåret och efter 65-årsålder då risken att drabbas av stroke är som störst. Behandling av epilepsi används i syfte att hindra uppkomst av anfall och göra det möjligt för den drabbade att leva ett relativt normalt liv. Antiepileptika dämpar aktiviteten i hjärnan och reducerar därmed risken

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47

Olstad, Elisabeth. "Glutamate and GABA: Major Players in Neuronal Metabolism." Doctoral thesis, Norwegian University of Science and Technology, Department of Neuroscience, 2007. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-1511.

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<p>Disturbance of neuronal metabolism has implications for a number of neurological and psychiatric conditions, and enhanced knowledge of this is important in developing new methods for treating such disorders. The present research was undertaken to aid understanding of diseases related to disturbance in glutamate and γ-amino butyric acid (GABA) metabolism.</p><p>Two different types of neuronal cell cultures were used in these studies; one containing GABAergic neurons of cerebral neocortical origin and one containing cerebellar neurons. The latter consists primarily of glutamatergic granule ne

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48

Jackson, Michael F. "Frequency-dependent actions of GABA enhancing anticonvulsant drugs." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0022/NQ50191.pdf.

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49

Cao, Juxiang Locy Robert D. "Functional genomics of GABA metabolism in yeast thermotolerance." Auburn, Ala, 2008. http://repo.lib.auburn.edu/2007%20Fall%20Dissertations/Cao_Juxiang_41.pdf.

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50

Aydar, Ebru. "Pharmacology of GABA and glutamate receptors in insects." Thesis, Oxford Brookes University, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363779.

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