Relevant bibliographies by topics / GABAergic transmissioni

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'GABAergic transmissioni'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "GABAergic transmissioni"

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Liu, Tao, Tsugumi Fujita, and Eiichi Kumamoto. "Acetylcholine and norepinephrine mediate GABAergic but not glycinergic transmission enhancement by melittin in adult rat substantia gelatinosa neurons." Journal of Neurophysiology 106, no. 1 (2011): 233–46. http://dx.doi.org/10.1152/jn.00838.2010.

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GABAergic and glycinergic inhibitory synaptic transmissions in substantia gelatinosa (SG; lamina II of Rexed) neurons of the spinal dorsal horn play an important role in regulating nociceptive transmission from the periphery. It has not yet been well known whether each of the inhibitory transmissions plays a distinct role in the regulation. We report an involvement of neurotransmitters in GABAergic but not glycinergic transmission enhancement produced by the PLA2 activator melittin, where the whole-cell patch-clamp technique is applied to the SG neurons of adult rat spinal cord slices. Glycine
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Zhu, Ping Jun, and Vincent A. Chiappinelli. "Nicotine Modulates Evoked GABAergic Transmission in the Brain." Journal of Neurophysiology 82, no. 6 (1999): 3041–45. http://dx.doi.org/10.1152/jn.1999.82.6.3041.

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The effects of nicotine on evoked GABAergic synaptic transmission were examined using whole cell recordings from neurons of the lateral spiriform nucleus in embryonic chick brain slices. All synaptic activities were abolished by the GABAA receptor antagonist, bicuculline (20 μM). Under voltage-clamp with KCl-filled pipettes (holding potential −70 mV), nicotine (0.1–1.0 μM) increased the frequency of spontaneous GABAergic currents in a dose-dependent manner. Nicotine enhanced electrically evoked GABAergic transmission only at relatively low concentrations of 50–100 nM (but not 25 nM), which app
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Donato, Roberta, and Andrea Nistri. "Relative Contribution by GABA or Glycine to Cl−-Mediated Synaptic Transmission on Rat Hypoglossal Motoneurons In Vitro." Journal of Neurophysiology 84, no. 6 (2000): 2715–24. http://dx.doi.org/10.1152/jn.2000.84.6.2715.

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The relative contribution by GABA and glycine to synaptic transmission of motoneurons was investigated using an hypoglossus nucleus slice preparation from neonatal rats. Spontaneous, miniature, or electrically evoked postsynaptic currents (sPSCs, mPSCs, ePSCs, respectively) mediated by glycine or GABA were recorded under whole cell voltage clamp after blocking excitatory glutamatergic transmission with kynurenic acid. The overall majority of Cl−-mediated sPSCs was glycinergic, while only one-third was GABAergic; 70 ± 10% of mPSCs were glycinergic while 22 ± 8% were GABAergic. Tetrodotoxin (TTX
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Rao, G. Prasad. "Anxiety Disorders and Gabaergic Transmission." Indian Journal of Psychological Medicine 28, no. 1 (2006): 5–6. http://dx.doi.org/10.1177/0975156420060101.

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Sergeeva, Olga A. "GABAergic transmission in hepatic encephalopathy." Archives of Biochemistry and Biophysics 536, no. 2 (2013): 122–30. http://dx.doi.org/10.1016/j.abb.2013.04.005.

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Gafurov, Boris, and Suzanne B. Bausch. "GABAergic transmission facilitates ictogenesis and synchrony between CA3, hilus, and dentate gyrus in slices from epileptic rats." Journal of Neurophysiology 110, no. 2 (2013): 441–55. http://dx.doi.org/10.1152/jn.00679.2012.

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The impact of regional hippocampal interactions and GABAergic transmission on ictogenesis remain unclear. Cortico-hippocampal slices from pilocarpine-treated epileptic rats were compared with controls to investigate associations between seizurelike events (SLE), GABAergic transmission, and neuronal synchrony within and between cortico-hippocampal regions. Multielectrode array recordings revealed more prevalent hippocampal SLE in epileptic tissue when excitatory transmission was enhanced and GABAergic transmission was intact [removal of Mg2+ (0Mg)] than when GABAergic transmission was blocked [
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Schubert, Timm, Daniel Kerschensteiner, Erika D. Eggers, et al. "Development of Presynaptic Inhibition Onto Retinal Bipolar Cell Axon Terminals Is Subclass-Specific." Journal of Neurophysiology 100, no. 1 (2008): 304–16. http://dx.doi.org/10.1152/jn.90202.2008.

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Synaptic integration is modulated by inhibition onto the dendrites of postsynaptic cells. However, presynaptic inhibition at axonal terminals also plays a critical role in the regulation of neurotransmission. In contrast to the development of inhibitory synapses onto dendrites, GABAergic/glycinergic synaptogenesis onto axon terminals has not been widely studied. Because retinal bipolar cells receive subclass-specific patterns of GABAergic and glycinergic presynaptic inhibition, they are a good model for studying the development of inhibition at axon terminals. Here, using whole cell recording
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Vázquez-Gómez, Elizabeth, Andy Hernández-Abrego, Jassiel Mejía-Piedras, and Jesús García-Colunga. "Regulation of Hippocampal GABAergic Transmission by Fluoxetine and Its Metabolite Norfluoxetine." Receptors 3, no. 1 (2024): 1–12. http://dx.doi.org/10.3390/receptors3010001.

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Major depression is related to dysfunction of the GABAergic pathway. Interestingly, the antidepressant fluoxetine modifies GABAergic neurotransmission in human and animal models of depression. However, the effects of norfluoxetine (the main metabolite of fluoxetine) on GABAergic neurotransmission have not yet been studied. Therefore, we explored whether fluoxetine and/or norfluoxetine may regulate GABAergic transmission and whether these substances interact with GABAA receptors in hippocampal CA1 stratum radiatum interneurons. For these purposes, we recorded the firing profile, GABAergic spont
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Rubi, Lena, and Jean-Marc Fritschy. "Increased GABAergic transmission in neuropeptide Y-expressing neurons in the dopamine-depleted murine striatum." Journal of Neurophysiology 123, no. 4 (2020): 1496–503. http://dx.doi.org/10.1152/jn.00059.2020.

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As the main input nucleus of the basal ganglia, the striatum plays a central role in planning, control, and execution of movement and motor skill learning. More than 90% of striatal neurons, so-called medium spiny neurons (MSN), are GABAergic projection neurons, innervating primarily the substantia nigra pars reticulata or the globus pallidus internus. The remaining neurons are GABAergic and cholinergic interneurons, synchronizing and controlling striatal output by reciprocal connections with MSN. Besides prominent local cholinergic influence, striatal function is globally regulated by dopamin
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Petukhova, Elena, Daria Ponomareva, Karin Rustler, Burkhard Koenig, and Piotr Bregestovski. "Action of the Photochrome Glyght on GABAergic Synaptic Transmission in Mouse Brain Slices." International Journal of Molecular Sciences 23, no. 18 (2022): 10553. http://dx.doi.org/10.3390/ijms231810553.

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Glyght is a new photochromic compound described as an effective modulator of glycine receptors at heterologous expression, in brain slices and in zebrafish larvae. Glyght also caused weak inhibition of GABAA-mediated currents in a cell line expressing α1/β2/γ2 GABAA receptors. However, the effects of Glyght on GABAergic transmission in the brain have not been analysed, which does not allow a sufficiently comprehensive assessment of the effects of the compound on the nervous system. Therefore, in this study using whole-cell patch-clamp recording, we analysed the Glyght (100 µM) action on evoked
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Dissertations / Theses on the topic "GABAergic transmissioni"

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MAURI, MARIO. "Cellule staminali mesenchimali: potenziali modulatori del sistema nervoso centrale." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/39835.

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Bone marrow-derived mesenchymal stem cells (MSCs) account for a small population of cells of the non-hematopoietic component of bone marrow. MSCs are multipotent stem cells endowed with neurotrophic potential combined to immunological properties, making them a promising therapeutic tool for neurodegenerative disorders. Although the mechanisms by which they act are still largely unknown, trans-differentiation, paracrine and autocrine actions have been hypothesized. Here we focus on the study of the effects exerted by rat MSCs on CNS neurons and oligodendrocytes by using a simplified in vitro c
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Garden, Derek Leonard Frank. "GABAergic transmission in the perirhinal cortex in vitro." Thesis, University of Bristol, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.274770.

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Mellor, Jack Robert. "Electrophysiological investigation of the mechanisms underlying GABAergic synaptic transmission." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624122.

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Fan, Kai Yoon. "GABAergic synaptic transmission, plasticity and integration in the subthalamic nucleus." Thesis, University of Sheffield, 2012. http://etheses.whiterose.ac.uk/3167/.

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Diana, Marco Alberto. "Charakterisierung von einer retrograden Modulation inhibitorischer synaptischer Transmission im Kleinhirn der Ratte." Doctoral thesis, [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=971021988.

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Asseri, Khalid. "Effects of AMBD and isovaline on GABAergic transmission in thalamic neurons." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/35078.

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In central neurons, the endogenous amino acid γ-aminobutyric acid (GABA) exerts synaptic inhibition mediated through ionotropic GABAA-, or metabotropic GABAB-receptors. These receptors exist on both pre- and postsynaptic membranes. The synthetic structural analogues of GABA, 6-aminomethyl-3-methyl-4H-1,2,4-benzothiadiazine-1,1-dioxide (AMBD) and R-isovaline have received little study on synaptic inhibition in the mammalian thalamus. AMBD was originally proposed as a taurine antagonist whereas R-isovaline is a non-biogenic amino acid that increases postsynaptic K⁺ conductance of thalamocortica
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Cao, Zhiwen, and 曹志文. "GABAergic transmission in developmental establishment of a gravity-related spatial reference." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47151304.

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In rats, the subnuclei of the inferior olive (IO) and thalamus exist topographic spatial representation to sinusoidal horizontal linear translations along either the antero-posterior or interaural direction. To examine the effect of GABAergic neurotransmission within the vestibular nucleus on the establishment of gravity-related topographic spatial representation in relay station of the central vestibular pathway, GABAA receptor antagonist bicuculline was used to chronically perturb GABA transmission within the vestibular nucleus of postnatal rats. Implantation of bicuculline-loaded or salin
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Ma, Ying [Verfasser]. "Effects of enhancing GABAergic transmission on sleep-associated memory consolidation / Ying Ma." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2012. http://d-nb.info/1021331007/34.

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Antonelli, Roberta. "The role of prolyl-isomerase PIN1 in GABAergic and glutamatergic synaptic transmission." Doctoral thesis, SISSA, 2015. http://hdl.handle.net/20.500.11767/4893.

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The correct functioning of the central nervous system relies on the rapid and efficient communication between neurons. This occurs at highly specialized functional points of contact called synapses. Synapses are extremely plastic in structure and function, strongly influenced by their own histories of impulse traffic and by signals from nearby cells. Synaptic contacts are fundamental to the development, homeostasis and remodeling of neural circuits. All these events are achieved through different mechanisms operating at both pre- and postsynaptic sites. At the level of the post synaptic densit
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Yuan, Ning. "DISTINCT MODULATORY EFFECTS OF DOPAMINE ON EXCITATORY CHOLINERGIC AND INHIBITORY GABAERGIC SYNAPTIC TRANSMISSION IN DROSOPHILA." Ohio University / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1149001533.

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Books on the topic "GABAergic transmissioni"

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Giovanni, Biggio, Costa Erminio, and Capo Boi Conference on Neuroscience (4th : 1985 : Villasimius, Italy), eds. GABAergic transmission and anxiety. Raven Press, 1986.

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2

Giovanni, Biggio, Concas Alessandra, and Costa Erminio, eds. GABAergic synaptic transmission: Molecular, pharmacological, and clinical aspects. Raven Press, 1992.

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3

L, Alkon Daniel, and National Institute of Neurological and Communicative Disorders and Stroke, eds. Long-term transformation of an inhibitory into an excitatory GABAergic synaptic response. National Institute of Neurological and Communicative Disorders and Stroke, 1993.

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Biggio, Giovanni, and Alessandra Concas. Gabaergic Synaptic Transmission: Molecular, Pharmacological, and Clinical Aspects (Advances in Biochemical Psychopharmacology). Raven Pr, 1992.

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Giovanni, Giuseppe Di, Adam C. Errington, and Vincenzo Crunelli. Extrasynaptic GABAA Receptors. Springer, 2014.

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Giovanni, Giuseppe Di, Adam C. Errington, and Vincenzo Crunelli. Extrasynaptic GABAA Receptors. Springer, 2014.

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Giovanni, Giuseppe Di, Adam C. Errington, and Vincenzo Crunelli. Extrasynaptic GABAA Receptors. Springer, 2016.

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Book chapters on the topic "GABAergic transmissioni"

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Manji, Husseini K., Jorge Quiroz, R. Andrew Chambers, et al. "GABAergic Transmission." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1398.

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Gaiarsa, Jean-Luc, and Yezekiel Ben-Ari. "Ontogenesis of Gabaergic and Glutamatergic Synaptic Transmission." In Advances in Behavioral Biology. Springer US, 2001. http://dx.doi.org/10.1007/0-306-47612-6_5.

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Bartos, Marlene, Jonas-Frederic Sauer, Imre Vida, and Ákos Kulik. "Fast and Slow GABAergic Transmission in Hippocampal Circuits." In Springer Series in Computational Neuroscience. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-99103-0_5.

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Bartos, Marlene, Jonas-Frederic Sauer, Imre Vida, and Ákos Kulik. "Fast and Slow GABAergic Transmission in Hippocampal Circuits." In Hippocampal Microcircuits. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-0996-1_5.

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Sipilä, Sampsa T., and Kai K. Kaila. "GABAergic Transmission and Neuronal Network Events During Hippocampal Development." In Developmental Plasticity of Inhibitory Circuitry. Springer US, 2009. http://dx.doi.org/10.1007/978-1-4419-1243-5_7.

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Dieudonné, Stéphane, and Marco Alberto Diana. "Postsynaptic Determinants of Inhibitory Transmission at Mixed GABAergic/Glycinergic Synapses." In Co-Existence and Co-Release of Classical Neurotransmitters. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09622-3_7.

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Costa, E., C. Ferrarese, A. Guidotti, M. Miyata, and I. Mocchetti. "Multiple Signal in GABAergic Transmission: A New Family of Modulatory Peptides." In Receptor-Receptor Interactions. Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-08949-9_29.

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Costa, E., C. Ferrarese, A. Guidotti, M. Miyata, and I. Mocchetti. "Multiple Signal in GABAergic Transmission: A New Family of Modulatory Peptides." In Receptor-Receptor Interactions. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5415-4_29.

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Maneuf, Y. P., and J. M. Brotchie. "Cannabinoid Receptor-Mediated Modulation of Gabaergic Transmission in the Basal Ganglia." In Advances in Behavioral Biology. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0194-1_21.

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Woodin, Melanie A., and Mu-ming Poo. "Activity-Dependent Modification of Cation-Chloride Cotransporters Underlying Plasticity of Gabaergic Synaptic Transmission." In Excitatory-Inhibitory Balance. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0039-1_6.

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Conference papers on the topic "GABAergic transmissioni"

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Cao, Yixuan. "The mechanisms of action of antiepileptic drugs affecting GABAergic and Glutamatergic synaptic transmission." In International Conference on Modern Medicine and Global Health (ICMMGH 2023), edited by Sheiladevi Sukumaran. SPIE, 2023. http://dx.doi.org/10.1117/12.2692185.

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Li, Guoshi, Stacy Cheng, Frank Ko, Scott L. Raunch, Gregory Quirk, and Satish S. Nair. "Computational Modeling of Lateral Amygdala Neurons During Acquisition and Extinction of Conditioned Fear, Using Hebbian Learning." In ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-15078.

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The amygdaloid complex located within the medial temporal lobe plays an important role in the acquisition and expression of learned fear associations (Quirk et al. 2003) and contains three main components: the lateral nucleus (LA), the basal nucleus (BLA), and the central nucleus (CE) (Faber and Sah, 2002). The lateral nucleus of the amygdala (LA) is widely accepted to be a key site of plastic synaptic events that contributes to fear learning (Pare, Quirk, LeDoux, 2004). There are two main types of neurons within the LA and the BLA: principal pyramidal-like cells which form projection neurons
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