Academic literature on the topic 'Gadolinium chlorure'

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Journal articles on the topic "Gadolinium chlorure"

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&NA;. "Gadolinium chloride." Reactions Weekly &NA;, no. 1317 (September 2010): 25. http://dx.doi.org/10.2165/00128415-201013170-00081.

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DEAN, PETER B., PEKKA NIEMI, LEENA KIVISAARI, and MARTTI KORMANO. "Comparative Pharmacokinetics of Gadolinium DTPA and Gadolinium Chloride." Investigative Radiology 23, no. 1 (September 1988): S261. http://dx.doi.org/10.1097/00004424-198809000-00057.

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DEAN, PETER B., PEKKA NIEMI, LEENA KIVISAARI, and MARTTI KORMANO. "Comparative Pharmacokinetics of Gadolinium DTPA and Gadolinium Chloride." Investigative Radiology 23 (September 1988): S258—S260. http://dx.doi.org/10.1097/00004424-198809001-00055.

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Spencer, Andrew, Susan Wilson, and Ernest Harpur. "Gadolinium chloride toxicity in the mouse." Human & Experimental Toxicology 17, no. 11 (November 1998): 633–37. http://dx.doi.org/10.1177/096032719801701108.

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1 Groups of five male and five female CD-1 mice received a single intravenous injection of gadolinium chloride at dosages of 0 (saline control), 0.05, 0.1 and 0.2 mmol/ kg. All mice were necropsied 48 h post dose. 2 Plasma analysis showed increases in concentrations of lactate dehydrogenase (both sexes), aspartate amino-transferase and alanine aminotransferase (females only) in the 0.2 mmol/kg group. Cholesterol was elevated at all dosages in both sexes whilst globulin was raised in both sexes at 0.1 and 0.2 mmol/kg. 3 Histological lesions were present at all dosages and increased in severity in a dose-related fashion. The most common lesions were: mineral emboli in capillaries, accumulation of mineral in the mono-nuclear phagocytic system, hepatocellular necrosis, and lymphoid depletion, necrosis and mineralisation in the spleen. 4 Such observations are similar to those in rats given gadolinium chloride and should be assessed when evaluating the toxicological profile of gadolinium containing compounds being developed for nuclear magnetic resonance imaging.
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Klein, Andrés, Juan Oyarzún, Cristian Cortez, and Silvana Zanlungo. "Gadolinium Chloride Rescues Niemann–Pick Type C Liver Damage." International Journal of Molecular Sciences 19, no. 11 (November 14, 2018): 3599. http://dx.doi.org/10.3390/ijms19113599.

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Niemann–Pick type C (NPC) disease is a rare neurovisceral cholesterol storage disorder that arises from loss of function mutations in the NPC1 or NPC2 genes. Soon after birth, some patients present with an aggressive hepatosplenomegaly and cholestatic signs. Histopathologically, the liver presents with large numbers of foam cells; however, their role in disease pathogenesis has not been explored in depth. Here, we studied the consequences of gadolinium chloride (GdCl3) treatment, a well-known Kupffer/foam cell inhibitor, at late stages of NPC liver disease and compared it with NPC1 genetic rescue in hepatocytes in vivo. GdCl3 treatment successfully blocked the endocytic capacity of hepatic Kupffer/foam measured by India ink endocytosis, decreased the levels CD68—A marker of Kupffer cells in the liver—and normalized the transaminase levels in serum of NPC mice to a similar extent to those obtained by genetic Npc1 rescue of liver cells. Gadolinium salts are widely used as magnetic resonance imaging (MRI) contrasts. This study opens the possibility of targeting foam cells with gadolinium or by other means for improving NPC liver disease. Synopsis: Gadolinium chloride can effectively rescue some parameters of liver dysfunction in NPC mice and its potential use in patients should be carefully evaluated.
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Spencer, Andrew J., Susan A. Wilson, John Batchelor, Alexandra Reid, Jeremy Pees, and Ernest Harpur. "Gadolinium Chloride Toxicity in the Rat." Toxicologic Pathology 25, no. 3 (May 1997): 245–55. http://dx.doi.org/10.1177/019262339702500301.

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Spencer, A., S. Wilson, and E. Harpur. "Gadolinium chloride toxicity in the mouse." Human & Experimental Toxicology 17, no. 11 (November 1, 1998): 633–37. http://dx.doi.org/10.1191/096032798678908035.

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Simon, A., and T. Koehler. "A new gadolinium nitride chloride—Gd3Cl6N." Journal of the Less Common Metals 116, no. 1 (February 1986): 279–92. http://dx.doi.org/10.1016/0022-5088(86)90236-5.

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Khushkhov, KH B., M. K. Vindizheva, A. S. Uzdenova, and Z. A. Zhanikaeva. "Joint electroreduction of lanthanum, gadolinium and boron in halide melts." Journal of Mining and Metallurgy, Section B: Metallurgy 39, no. 1-2 (2003): 275–80. http://dx.doi.org/10.2298/jmmb0302275k.

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The joint electroreduction of La, Gd and B from chloride-fluoride melts has been studied by cyclic voltametry. Based on the analysis of voltamograms the possibility of electrosynthesis of lanthanum-gadolinium borides from chloride-fluoride melts has been shown.
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Lim, Jong Min, and Sang Woo Kim. "Synthesis of Nickel Coated Gadolinia Doped Ceria Nanopowder by Microwave Radiation." Materials Science Forum 569 (January 2008): 77–80. http://dx.doi.org/10.4028/www.scientific.net/msf.569.77.

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Nickel coated gadolinium doped ceria (GDC) powder was synthesized by microwave radiation and combustion. For the synthesis, the precipitates of gadolinium cerium oxycarbonate hydrate (GdxCe2-xO(CO3)2·H2O) were formed by a microwave radiated reaction between cerium nitrate (Ce(NO3)3.6H2O) and gadolinium nitrate (Gd(NO3)3.6H2O) and urea (CO(NH2)2), then nickel coatings on the gadolinium cerium oxycarbonate hydrate were performed by further microwave reaction between nickel chloride and urea. The shape and size of the gadolinium cerium oxycarbonate hydrate particles were critically dependent on aging time during microwave radiation. The irregular particles were transformed to rod shape particles with well-crystallized with increasing aging time to 40 min at 70 - 80°C because of the gradual decomposition of urea during microwave radiation. Small nickel precursor particles were homogeneously coated on the gadolinium cerium oxycarbonate hydrate particles with rod shape with aid of microwave radiation at 80 °C for 40 min. As a result, the nickel coated GDC nanopowders were sucessfully produced by the microwave radiation synthesis and further microwave combusted at 450°C for 20 min.
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Dissertations / Theses on the topic "Gadolinium chlorure"

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Mahiou, Rachid. "Transferts d'excitation optique et effets non lineaires dans des materiaux concentres en gadolinium." Clermont-Ferrand 2, 1987. http://www.theses.fr/1987CLF2E379.

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Fluorescence de nagdf::(4), k::(2)gdf::(5), gdcl::(3). Excitation selective dans l'ultraviolet (excitation a 1 photon) ou dans le rouge (excitation a 2 photons) d'une des composantes stark du multiplet **(6)p::(7/2) de gd**(3+). Le declin de la fluorescence entre 4,4k et la temperature ambiante permet de determiner le mode de diffusion de l'energie d'excitation. Effet de la densite d'excitation. Mise en evidence d'une fluorescence anti-stokes issue des niveaux **(6)g::(7/2), **(6)d::(3/2) et **(6)i::(7/2). Effet d'un champ magnetique
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Pernot, Paul. "Élaboration et caractérisation de composés binaires et ternaires du graphite avec les chlorures métalliques : Corrélations entre structures et propriétés physiques." Nancy 1, 1989. http://www.theses.fr/1989NAN10236.

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Préparation de composés d'insertion du graphique avec COCL#2, CDCL#2, CUCL#2, GDCL#3 et ALCL#3. Caractérisation structurale par diffraction RX et corrélation des données cristallochimiques avec les mesures de conductivité thermique et électrique
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Stenbäck, Anders. "Studies of Experimental Bacterial Translocation." Doctoral thesis, Uppsala University, Department of Surgical Sciences, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-5933.

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One of the main obstacles to maintaining patients with short bowel syndrome on parenteral nutrition, or successfully transplanting these patients with a small bowel graft, is the many severe infections that occur. Evidence is accumulating that translocating bacteria from the patient’s bowel causes a significant part of these infections. In this thesis bacterial translocation is studied in a Thiry-Vella loop of defunctionalised small bowel in the rat.

Bacterial translocation to the mesenteric lymph nodes (MLNs) occurs in almost 100% of the rats after three days. No systemic spread of bacteria is observed unless there is additional immunosupression with depletion of Kupffer cells in the liver. However, blocking the function of α/β T cells does not increase the translocation. Removal of MLNs does not either aggravate bacterial translocation in the Thiry-Vella loop model. Conversely, after small bowel transplantation translocating bacteria spread systemically if the MLNs are removed.

The Thiry-Vella loop should also be a suitable model for the testing of potentially translocation-inhibiting substances. Reinforcement of the intestinal barrier with glutamine or phosphatidylcholine proved insufficient in decreasing bacterial translocation. Even selective bowel decontamination with tobramycin failed to abolish bacterial translocation. Thus, it seems that the driving force for translocation in this model is strong regardless of the relatively small trauma of intestinal defunctionalisation.

Flow cytometric studies of the immune cells in the spleen MLNs showed a decrease in MHC class II positive T cells in the MLNs of the Thiry-Vella loop. Concurrently the number of macrophages increased with time as observed by immunohistochemistry. The fraction of MHC class II negative macrophages increased in the spleens of rats treated with glutamine.

In conclusion, the Thiry-Vella loop model offers possibilities of immunological as well as mechanistic studies on bacterial translocation from small intestine.

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Book chapters on the topic "Gadolinium chlorure"

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Korolenko, T., I. Svechnikova, K. Urazgaliyev, G. Vakulin, and S. Djanaeva. "Liver Cysteine Proteinases in Macrophage Depression Induced by Gadolinium Chloride." In Advances in Experimental Medicine and Biology, 315–21. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9613-1_41.

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Pardasani, R. T., and P. Pardasani. "Magnetic properties of gadolinium(III) chloride complex with 18-membered hexaazatetraimine macrocyclic ligand." In Magnetic Properties of Paramagnetic Compounds, 1313–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-53974-3_678.

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