Relevant bibliographies by topics / Ganglions autonomes

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Ganglions autonomes'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Ganglions autonomes.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Ganglions autonomes"

1

Sienkiewicz, W., A. Dudek, A. Chroszcz, M. Janeczek, and J. Kaleczyc. "Distribution and immunohistochemical properties of autonomic neurons supplying the ovine hip joint capsule." Veterinární Medicína 63, No. 6 (2018): 261–70. http://dx.doi.org/10.17221/61/2017-vetmed.

Full text
Abstract:
Combined retrograde tracing and double labelling immunohistochemistry were applied to study the distribution and chemical coding of autonomic neurons projecting to the ovine hip joint capsule. As revealed by retrograde tracing, fast blue-positive autonomic neurons supplying the lateral side of the hip joint capsule and the medial side of the hip joint capsule were located within the lumbar and sacral of the ipsilateral sympathetic chain ganglia and within the caudal mesenteric ganglion. Immunohistochemistry revealed that nearly all (sympathetic chain ganglia: 96% and caudal mesenteric ganglion: 98.8%) the neurons were adrenergic in nature (positive for dopamine β-hydroxylase). Many retrogradely labelled neurons also displayed immunoreactivity to neuropeptide Y (approximately 34% of fast blue-positive neurons within caudal mesenteric ganglion and sympathetic chain ganglia). Populations of Met-Enk<sup>+</sup> (20%) and Leu-Enk<sup>+</sup> (6%) neurons were present only in the sympathetic chain ganglia while within caudal mesenteric ganglion no enkephalinergic-labelled neurons were noted. Only a small population (2.2%) of hip joint capsule-projecting neurons were Gal-IR and they were observed only within the caudal mesenteric ganglion. No cholinergic neurons involved in the innervation of the hip joint capsule were found. However, fast blue-positive nerve cell bodies were surrounded by numerous cholinergic nerve fibres often forming basket-like formations. Single Gal<sup>+</sup> nerve fibres were found in the intraganglionic connective tissue. Substance P-positive or calcitonin gene-related peptide-positive intraganglionic nerve terminals were very numerous and formed “baskets” surrounding fast blue-positive perikarya within sympathetic chain ganglias and caudal mesenteric ganglion.
APA, Harvard, Vancouver, ISO, and other styles
2

King, B. F., and J. H. Szurszewski. "Peripheral reflex pathways involving abdominal viscera: transmission of impulses through prevertebral ganglia." American Journal of Physiology-Gastrointestinal and Liver Physiology 256, no. 3 (1989): G581—G588. http://dx.doi.org/10.1152/ajpgi.1989.256.3.g581.

Full text
Abstract:
In neurophysiological terms, divergence describes the transmission of impulse traffic from a single afferent line, through an integrating nervous system, and out into multiple efferent lines. This feature has been ascribed to the vertebrate central nervous system and invertebrate ganglionic systems but has not yet been associated with the autonomic nervous system in mammals. Therefore, this study investigated the degree of divergence of afferent impulse traffic through a mammalian autonomic ganglion, the inferior mesenteric ganglion (IMG) in guinea pig. Multiunit discharges were recorded extracellularly from the peripheral nerves, which emerge from the IMG, to determine the lines of efferent outflow (i.e., divergence) of impulse traffic generated by stimulating central efferent and peripheral afferent nerves. Pathways interrupted by a cholinergic ganglion synapse were identified by using hexamethonium. Pathways running directly through the IMG were identified by studying the effects of dividing nerves surgically. A complex arrangement of ascending and descending pathways was revealed, showing a neural network that interconnects the upper gastrointestinal tract, distal colon, and pelvic viscera via prevertebral ganglia.
APA, Harvard, Vancouver, ISO, and other styles
3

Khan, Sabrina, Jean Schoenen, and Messoud Ashina. "Sphenopalatine ganglion neuromodulation in migraine: What is the rationale?" Cephalalgia 34, no. 5 (2013): 382–91. http://dx.doi.org/10.1177/0333102413512032.

Full text
Abstract:
Objective The objective of this article is to review the prospect of treating migraine with sphenopalatine ganglion (SPG) neurostimulation. Background Fuelled by preliminary studies showing a beneficial effect in cluster headache patients, the potential of treating migraine with neurostimulation has gained increasing interest within recent years, as current treatment strategies often fail to provide adequate relief from this debilitating headache. Common migraine symptoms include lacrimation, nasal congestion, and conjunctival injection, all parasympathetic manifestations. In addition, studies have suggested that parasympathetic activity may also contribute to the pain of migraineurs. The SPG is the largest extracranial parasympathetic ganglion of the head, innervating the meninges, lacrimal gland, nasal mucosa, and conjunctiva, all structures involved in migraine with cephalic autonomic symptoms. Conclusion We propose two possible mechanisms of action: 1) interrupting the post-ganglionic parasympathetic outflow to inhibit the pain and cephalic autonomic symptoms, and 2) modulating the sensory processing in the trigeminal nucleus caudalis. To further explore SPG stimulation in migraineurs as regards therapeutic potential and mode of action, randomized clinical trials are warranted.
APA, Harvard, Vancouver, ISO, and other styles
4

Martin, D. S., and J. R. McNeill. "Whole body vascular capacitance response to vasopressin is mediated by autonomic function." American Journal of Physiology-Heart and Circulatory Physiology 261, no. 2 (1991): H493—H499. http://dx.doi.org/10.1152/ajpheart.1991.261.2.h493.

Full text
Abstract:
Effects of intravenous infusions of arginine vasopressin (AVP) on whole body vascular capacitance were determined in anesthetized cats when autonomic nervous system function was intact and, in other cats, when reflexes were blocked by the ganglionic blocking agent pentolinium. With the use of the constant cardiac output-reservoir technique, changes in reservoir volume were assumed to reflect reciprocal changes in whole body vascular capacitance. Relationships between the dose of AVP and the plasma concentration of the peptide achieved during infusions were not significantly different in the two groups of animals. Blood pressure responses to AVP were greater, whereas heart rate responses to the peptide were abolished in ganglion-blocked cats. In cats with intact autonomic function, reservoir volume decreased by 1.6, 4.2, and 7.8 ml/kg at AVP doses of 1, 10, and 100 ng.kg-1.min-1, respectively. In contrast, in ganglion-blocked cats, reservoir volume did not change significantly at 1.0 and 10 ng.kg-1.min-1 of AVP, and the highest dose caused a much smaller change in volume (3 ml/kg) than that observed in cats with intact autonomic function (7.8 ml/kg). Systemic compliance was unchanged by AVP in both groups of animals, suggesting that the increases in whole body vascular capacitance were likely due to changes in unstressed volume. The results suggest that reflexively mediated changes in autonomic function increase whole body vascular capacitance during elevations in the circulating levels of AVP to plasma concentrations that are biologically relevant. These findings may explain how AVP decreases cardiac output in animals with an intact autonomic nervous system.
APA, Harvard, Vancouver, ISO, and other styles
5

Wood, Adam, Salvatore Docimo, and David E. Elkowitz. "Cardiovascular Disease and its Association with Histological Changes of the Left stellate Ganglion." Clinical Medicine Insights: Pathology 3 (January 2010): CPath.S4285. http://dx.doi.org/10.4137/cpath.s4285.

Full text
Abstract:
Mounting evidence has demonstrated that the autonomic system plays a role in the morbidity and mortality of certain cardiovascular disease states. Ventricular arrhythmias have been associated with the level of sympathetic activation. We attempted to determine if the presence of fibrosis, a marker for previous ischemic events, correlates with an increase in the number of left stellate ganglion nerve cell bodies which is indicative of hypersympathetic stimulation to the myocardial tissue. Left stellate ganglia were removed, sectioned and prepared using hematoxylin and eosin and Masson's trichrome stain. The interventricular septum of the heart corresponding to the stellate ganglion samples were removed, serially sectioned, and stained with hematoxylin and eosin and Masson's trichrome stain. The samples were described using a grading scale to quantify the percentage of fibrosis. Ganglion nerve cell bodies were then individually counted in three separate high-powered fields. A student's T-test was used to statistically evaluate the data. Stellate ganglions were sampled from 32 cadavers. Fibrosis was present within 72% (23/32) of the interventricular septums that were sampled. Nine interventricular septums were found to be free of fibrosis. For those interventricular septums that were positive for the presence of fibrosis, the mean left stellate ganglion nerve cell bodies was 39.8 (Range: 26-51). For those interventricular septums that were negative for the presence of fibrosis, the mean left stellate ganglion nerve cell bodies was 34.3 (Range: 27-46). The difference between the mean nerve cell bodies for interventricular septums with fibrosis and without fibrosis was found to be statistically significant ( P = 0.048). Histological changes in terms of the number of left stellate ganglion nerve cell bodies seem to be dependent upon the presence of fibrosis within the interventricular septum. Considering fibrosis of the interventricular septum is a marker for previous ischemic events, an increase in the number of nerve cell bodies of the left stellate ganglion in the presence of fibrosis suggests an association does exist between hypersympathetic stimulation to the myocardial tissue and myocardial infarction. Further research into this association is warranted in order to determine if left stellate ganglion blockade is a viable treatment option for arrhythmias following myocardial infarctions.
APA, Harvard, Vancouver, ISO, and other styles
6

Orozco, Adriana Vega, Zulema Sosa, Verónica Fillipa, Fabian Mohamed, and Ana María Rastrilla. "The cholinergic influence on the mesenteric ganglion affects the liberation of ovarian steroids and nitric oxide in oestrus day rats: characterization of an ex vivo system." Journal of Endocrinology 191, no. 3 (2006): 587–98. http://dx.doi.org/10.1677/joe.1.06859.

Full text
Abstract:
The axons that constitute the ovarian nervous plexus originate mostly in the principal neurons of the superior mesenteric ganglion (SMG) that is part of the sympathetic ganglionic chain and exhibits cholinergic receptors. In order to observe the effect of acetylcholine, the main neurotransmitter in the ganglionic transmission, the purpose of the present work was: first, to standardize an integrated ex vivo superior mesenteric ganglion-ovarian nervous plexus-ovary (SMG-ONP-O) system in oestrus day rats; secondly, to determine if the ganglionic cholinergic stimulus modifies the release of nitric oxide and steroids in the ovary compartment in the absence of humoral factors; and thirdly, to investigate if there are differences in the responses between the left and right ovaries caused by the neural stimulus. The ex vivo experimental left and right systems were developed and standardized. The systems were incubated in Krebs–Ringer phosphate buffer in a Dubnoff metabolic shaker. The progesterone release was determined to standardize the incubation times, obtaining different responses between the left and right systems, which shows that both systems have their own autonomic tone. Non-specific stimulation with KCl in the ganglion compartment provoked different responses in terms of release of progesterone and oestradiol. Progesterone decreased in the left and right systems. However, oestradiol diminished at short times and increased at 60 and 120 min in the left ovary, whereas it increases at 30 and 60 min in the right ovary. These different responses show the sensitivity and viability of both systems. When acetylcholine was used in the ganglion compartment, the release of nitric oxide, progesterone, androstenedione and oestradiol was evaluated. The liberation of nitrite increased at 15, 30 and 60 min in the left system and decreased in the right system at 120 min. Progesterone showed a decrease in its release at 15, 30 and 120 min and androstenedione at 15 min in the left ovary compartment. In the right ovary, only progesterone decreased in relation to the control at 120 min while androstenedione did not show significant changes. Oestradiol showed an increase in the left ovary compartment at all the studied times, while in the right ovary it did not show any changes. These results indicate that the neural stimulus from the superior mesenteric ganglion through the ovarian nervous plexus is one of the factors modulating the secretory activity of the ovarian steroids and nitric oxide. The system is viable and also shows a different sensitivity of the left ovary in relation to the right one at least in this cycle stage, characterized by marked irrigation and profound structural changes in the ovary.
APA, Harvard, Vancouver, ISO, and other styles
7

Vernino, Steven, Phillip A. Low, and Vanda A. Lennon. "Experimental Autoimmune Autonomic Neuropathy." Journal of Neurophysiology 90, no. 3 (2003): 2053–59. http://dx.doi.org/10.1152/jn.00408.2003.

Full text
Abstract:
Antibodies specific for the neuronal ganglionic nicotinic acetylcholine receptor (nAChR) are found in high titer in serum of patients with subacute autonomic failure. This clinical disorder is known as autoimmune autonomic neuropathy (AAN). Rabbits immunized with a neuronal nAChR α3 subunit fusion protein produce ganglionic nAChR antibodies and develop autonomic failure (experimental AAN, or EAAN). We used quantitative measures of autonomic function to demonstrate that this animal model of neuronal nAChR autoimmunity recapitulates the cardinal autonomic features of AAN in humans. The severity of dysautonomia in the rabbit ranges from isolated cardiovagal impairment to severe panautonomic failure with fixed mydriasis, gastroparesis, dry eyes, impaired heart rate variability, hypotension, and low plasma catecholamines. The severity of autonomic failure correlates with serum antibody levels. Immunohistochemical staining of superior cervical ganglia and myenteric plexus neurons demonstrates intact presynaptic nerve terminals and intact postsynaptic neurons containing cytoplasmic nAChR, but lacking surface nAChR. These findings define the autonomic physiology and histopathology of this novel animal model and support the concept that AAN in humans is a disorder of ganglionic cholinergic synaptic transmission caused by ganglionic nAChR antibodies.
APA, Harvard, Vancouver, ISO, and other styles
8

María Dolores Ramos Jiménez, Lucía Santos Martín, Nicolás Cordero Tous, and Carmen de la Linde Valverde. "Cirugía de implantación de microestimulador del ganglio esfenopalatino. Manejo anestésico y revisión de la literatura." Revista Electrónica AnestesiaR 11, no. 3 (2019): 2. http://dx.doi.org/10.30445/rear.v11i3.715.

Full text
Abstract:
La cefalea en racimos es un tipo de cefalea trigémino-autonómica con síntomas marcadamente disfuncionales debidos al intenso dolor y a la aparición de signos disautonómicos hemicraneales ipsilaterales recurrentes. El tratamiento médico no siempre resulta eficaz o puede no tolerarse debido a sus efectos adversos. El desarrollo de técnicas quirúrgicas que inciden en el tratamiento a través de la estimulación de estructuras directamente relacionadas con el dolor, supone una nueva y esperanzadora herramienta terapéutica para este tipo de pacientes. Además de una cirugía correcta, el tipo de anestesia para la intervención debe ser cuidadosa y adecuarse a la necesidad de trabajar sobre un campo quirúrgico oro-facial amplio. Presentamos dos casos de pacientes con cefalea en racimos de evolución crónica y refractaria a tratamiento, sometidos a la implantación quirúrgica de un microestimulador en el ganglio esfenopalatino bajo anestesia general. ABSTRACT Sphenopalatine ganglion microstimulator surgical insertion. Anesthetic management and literature review Cluster headache is a trigeminal-autonomic headache with severe symptoms due to recurrent intense pain and ipsilateral autonomic manifestations. Medical treatment is not always succesful or may be poorly tolerated for side effects. The development of surgical technics who involve the stimulation of the pain centers, is a new and hopeful tool for these patients. Althought a correct surgery, anesthesia may be careful and must fit into a board surgical orofacial field. We expose a case for chronic and refractory cluster headache, treated by sphenopalatine ganglion microstimulator surgical insertion under general anesthesia.
APA, Harvard, Vancouver, ISO, and other styles
9

Smith, Peter A., Hsinyo Chen, Dmitry E. Kurenny, Alexander A. Selyanko, and Jeffrey A. Zidichouski. "Regulation of the M current: transduction mechanism and role in ganglionic transmission." Canadian Journal of Physiology and Pharmacology 70, S1 (1992): S12—S18. http://dx.doi.org/10.1139/y92-238.

Full text
Abstract:
Slow excitatory postsynaptic potentials in sympathetic ganglia often involve suppression of a voltage-dependent potassium current termed the M current. This current is suppressed by the muscarinic action of acetylcholine, by peptides such as luteinizing hormone releasing hormone, and sometimes by α-adrenoceptor agonists. Activation of β-adrenoceptors sometimes produces weak potentiation. The voltage dependence of the M current is such that its suppression increases the excitability of ganglionic neurones. Since this sometimes leads to spontaneous discharge, activation of the slow excitatory postsynaptic potential mechanism (or modulation of M current) within a sympathetic ganglion produces effects that manifest in the autonomic outflow to the target organ. In frogs, M currents are present in the neurones of both paravertebral sympathetic ganglia and cardiac parasympathetic ganglia. Since the M current is suppressed by adrenaline in the parasympathetic ganglia and these ganglia often receive adrenergic fibres from sympathetic ganglia, this might reflect an important means of interaction between the two branches of the autonomic system. At the cellular level, M-current suppression is little affected by drugs that interfere with membrane phosphorylation–dephosphorylation processes. This observation is discussed in relationship to the current understanding of the transduction mechanism for agonist-induced M-current suppression.Key words: autonomic nerve, K+ channel, G protein, muscarinic mechanism, adrenergic mechanism.
APA, Harvard, Vancouver, ISO, and other styles
10

Guo, Song, Katrine Falkenberg, Henrik Winther Schytz, Anthony Caparso, Rigmor Højland Jensen, and Messoud Ashina. "Low frequency activation of the sphenopalatine ganglion does not induce migraine-like attacks in migraine patients." Cephalalgia 40, no. 9 (2020): 966–77. http://dx.doi.org/10.1177/0333102420921156.

Full text
Abstract:
Introduction Cephalic autonomic symptoms occur in 27‒73% of migraine patients during attacks. The role of parasympathetic activation in migraine attack initiation remains elusive. Low frequency stimulation of the sphenopalatine ganglion increases parasympathetic outflow. In this study, we hypothesized that low frequency stimulation of the sphenopalatine ganglion would provoke migraine-like attacks in migraine patients. Methods In a double-blind randomized sham-controlled crossover study, 12 migraine patients with a sphenopalatine ganglion neurostimulator received low frequency or sham stimulation for 30 min on two separate days. We recorded headache characteristics, cephalic autonomic symptoms, ipsilateral mechanical perception and pain thresholds, mean blood flow velocity in the middle cerebral artery (VMCA) and diameter of the superficial temporal artery during and after stimulation. Results Five patients (42%) reported a migraine-like attack after low frequency stimulation compared to six patients (50%) after sham ( p = 1.000). We found a significant increase in mechanical detection thresholds during low frequency stimulation compared to baseline ( p = 0.007). Occurrence of cephalic autonomic symptoms and changes in mechanical perception thresholds, VMCA and diameter of the superficial temporal artery showed no difference between low frequency stimulation compared to sham ( p = 0.533). Conclusion Low frequency stimulation of the sphenopalatine ganglion did not induce migraine-like attacks or autonomic symptoms in migraine patients. These data suggest that increased parasympathetic outflow by the sphenopalatine ganglion neurostimulator does not initiate migraine-like attacks. Study protocol: ClinicalTrials.gov registration number NCT02510742
APA, Harvard, Vancouver, ISO, and other styles

Dissertations / Theses on the topic "Ganglions autonomes"

1

Hamel, Olivier Robert Roger. "Ganglions végétatifs céphaliques anatomie descriptive, topographique et fonctionnelle; intérêts cliniques. /." [S.l.] : [s.n.], 2004. http://theses.univ-nantes.fr/thesemed/SPEhamel.pdf.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

SCHUMANN, MUHAMMAD AHMAD. "ELECTROPHYSIOLOGICAL ANALYSIS OF CHOLECYSTOKININ ACTIONS IN MAMMALIAN INFERIOR MESENTERIC GANGLION (AUTONOMIC REFLEX)." Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/183872.

Full text
Abstract:
Cholecystokinin (CCK)-like immunoreactive materials have been localized in neurons with cell bodies in the colon and axons in the IMG of the guinea pig. The physiological significance of neuronal CCK in sympathetic prevertebral ganglia is unknown. The goal of the present studies is to test the hypothesis the CCK is a neurotransmitter in the IMG of guinea pig and rabbit. In vitro IMG preparations with or without a segment of the colon attached were utilized to conduct intracellular recordings of potentials elicited in the neurons by pressure-ejected CCK₈. The peptide triggered a depolarization with rapid onset (1-5 s) and a rate of rise (1.6 ± 0.4 mV/s) in 95% of the neurons tested. Values of the ED₅₀ for effecting depolarization average 1.1 ± 0.5 pmoles. In 59% of the cells, the depolarization was associated with a decrease in R(in) and in 20% with an increase. The remaining cells showed no change in R(in). G(Na) and G(K) were increased and decreased, respectively; potential-dependence characteristics revealed a null potential of 36 ± 9 mV in those cells exhibiting a decrease in R(in). Gastrin, caerulein, and CCK₂₇₋₃₃ effected similar depolarization. CCK₈-evoked depolarization imitated the depolarization produced either by colon distension or by nerve stimulation. Upon repeated administration of CCK₈, the response of the cells to the peptide underwent tachyphylaxis. In addition, CCK₈ desensitized the depolarization evoked by stimulation in 50% of the cells. Furthermore, in an equal percentage of neurons, CCK₈ depressed responses of the colon distension-induced depolarization. The CCK₈ has both pre- and postsynaptic sites of action is supported by lowering Ca²⁺ and administering TTX (3 μM), which caused no effect and depressed 30% of CCK-induced depolarization respectively. Spantide (SP antagonist) blocked the response to SP, but not to CCK₈, in 5 out of 6 neurons, indicating separate receptor sites for SP and CCK₈. Moveover, completely desensitizing the cell response to SP or VIP did not cross desensitize its response to CCK₈ as observed in 6 neurons. In the rabbit IMG, the physiological significance of CCK₈ excitation is unknown, since colon distension did not elicit any depolarization. These results support the hypothesis that CCK₈ or a related peptide is a neurotransmitter mediating reflex activity between the colon and the IMG in guinea pig.
APA, Harvard, Vancouver, ISO, and other styles
3

Young, Tim. "Cardiovascular autonomic responses in pre- and post-ganglionic models of chronic autonomic failure." Thesis, Imperial College London, 2008. http://hdl.handle.net/10044/1/4718.

Full text
Abstract:
Multiple System Atrophy (MSA) and Pure Autonomic Failure (PAF) are contrasting models of Chronic Autonomic failure. PAF primarily involves the post-ganglionic autonomic nervous system, whilst in MSA the pre-ganglionic structures are impaired. My central hypothesis is that this underlying neuropathological difference between MSA and PAF will lead to differing cardiovascular responses. I will assess the cardiovascular effects of known pressor and vasomotor stimuli (mental arithmetic, cold pressor test, isometric exercise, water ingestion, inhaled CO₂ and inspiratory gasp) in MSA and PAF. Neurohormonal aspects will be explored by comparing the cardiovascular effects of the α2-adrenoceptor agonist clonidine with serum noradrenaline levels in these groups, as well as comparing supine antidiuretic hormone (ADH) levels after head up tilt and correlating these with supine blood pressure (BP). As well as contrasting the cardiovascular responses, I will use the water ingestion studies to examine effects on orthostatic hypotension, a common complication of both MSA and PAF. To measure cardiovascular responses during these studies I have used the Portapres II device to obtain continuous, non-invasive, beat-to-beat measurements of BP and heart rate (HR). Subsequent Model flow analysis using Beatscope software has then been used to calculate further cardiovascular indices, including cardiac output (CO), stroke volume (SV) and total peripheral resistance (TPR). In addition, intermittent BP and HR measurements have been obtained with an automated sphygmomanometer (Dinamap). Finally, peripheral vasomotor responses have been recorded by means of the Laser Doppler perfusion meter.
APA, Harvard, Vancouver, ISO, and other styles
4

Xue, Zhi-Gang. "Recherches sur la différenciation du système nerveux periphérique chez les oiseaux : mise en évidence et propriétés des précurseurs de type autonome présents dans les ganglions sensoriels." Paris 13, 1987. http://www.theses.fr/1987PA132008.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Jordan, Jens. "Pharmakologische Dissektion des Baroreflexes beim Menschen." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2002. http://dx.doi.org/10.18452/13806.

Full text
Abstract:
Komplette pharmakologische Unterbrechung des Baroreflexes mittels eines Ganglienblockers führt zu einer starken Zunahme der Wirkung vasoaktiver Substanzen auf den Blutdruck. Eine ähnliche Überempfindlichkeit gegenüber vasoaktiven Substanzen ist auch bei Erkrankungen zu beobachten, die mit Schädigungen des afferenten oder des efferenten Schenkels des Baroreflexes einhergehen. Variabilität der Baroreflexfunktion innerhalb der Population trägt somit in erheblichem Umfang zur Variabilität des Ansprechens auf vasoaktive Substanzen bei. Durch Vergleich der Wirkung kreislaufwirksamer Substanzen oder physiologischer Interventionen vor und während Ganglienblockade können zentrale und periphere Effekte voneinander unterschieden werden. Mit dieser Methode können Änderungen der vaskulären Sensitivität und der Pufferfunktion des Baroreflexes bei Krankheitszuständen charakterisiert werden.<br>Complete pharmacological interruption of the baroreflex using ganglionic blockade is associated with a profound increase in the blood pressure response to vasoactive substances. Similar hypersensitivity to vasoactive substances can be observed in disorders that involve the afferent arc or the efferent arc of the baroreflex. Therefore, interindividual variability in baroreflex function contributes substantially to the variability in the responsiveness to vasoactive substances. Comparison of the response to cardiovascular medications before and during ganglionic blockade can be used to dissect central and peripheral effects. This approach is also useful to characterize changes in vascular sensitivity and in baroreflex buffering function in diseases.
APA, Harvard, Vancouver, ISO, and other styles
6

Merighi, Adalberto. "Light and electron microscopical studies on the distribution of peptides and 'classical' neurotransmitters in dorsal root ganglion cells and in the dorsal horn of the spinal cord." Thesis, Imperial College London, 1990. http://hdl.handle.net/10044/1/46446.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Xue, Zhi-Gang. "Recherches sur la différenciation du système nerveux périphérique chez les oiseaux mise en évidence et propriétés des précurseurs de type autonome présents dans les ganglions sensoriels /." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37610856d.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Zhou, Xiangdong. "Brain-derived Neurotrophic Factor in Autonomic Nervous System: Nicotinic Acetylcholine Receptor Regulation and Potential Trophic Effects." Connect to full-text via OhioLINK ETD Center, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1130160629.

Full text
Abstract:
Thesis (Ph.D.)--Medical University of Ohio, 2005.<br>"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Medical Sciences." Major advisor: Joseph F. Margiotta. Includes abstract. Document formatted into pages: iii, 226 p. Title from title page of PDF document. Bibliography: pages 80-92,130-139,149-225.
APA, Harvard, Vancouver, ISO, and other styles
9

Dimitriadou, Violetta. "Donnees anatomophysiologiques en faveur d'un role non vasomoteur de l'innervation autonome des vaisseaux cerebraux : role trophique au cours du developpement et de l'age adulte, possibilite d'une intervention indirecte parl'intermediaire de cellules." Paris 6, 1988. http://www.theses.fr/1988PA066200.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Maillet, Brigitte. "Analyse de l’activité neuronale dans le ganglion stellaire en relation avec la fonction cardiaque." Thèse, 2010. http://hdl.handle.net/1866/4492.

Full text
Abstract:
Quatre microélectrodes ont été insérées dans le ganglion stellaire gauche (GS) de préparations canines in vivo pour évaluer la décharge des potentiels d’action dans les neurones situés dans ce ganglion périphérique durant un état cardiovasculaire stable et suivant des injections systémiques et locales de nicotine. Durant les périodes de contrôle, des changements mineurs ont été observés dans la pression artérielle systolique, dans le rythme cardiaque et dans le temps de conduction atrio-ventriculaire. L’activité générée par les neurones du GS est demeurée relativement constante à l’intérieure de chaque chien, mais variait entre les préparations. L’administration de nicotine systémique a altéré les variables physiologiques et augmenté l’activité neuronale. Même si différents changements au niveau des variables physiologiques ont été observés entre les animaux, ces changements demeuraient relativement constants pour un même animal. La dynamique de la réponse neuronale était similaire, mais l’amplitude et la durée variaient entre et au sein des chiens. L’injection de nicotine dans une artère à proximité du GS a provoqué une augmentation marquée des potentiels d’action sans faire changer les variables physiologiques. La technique d’enregistrement permet donc de suivre le comportement de multiples populations de neurones intrathoraciques situés dans le GS. La relation entre l’activation neuronale du GS et les changements physiologiques sont stables pour chaque chien, mais varient entre les animaux. Cela suggère que le poids relatif des boucles de rétroaction impliquées dans la régulation cardiovasculaire peut être une caractéristique propre à chaque animal.<br>Four micro-electrodes were inserted in the left stellate ganglion (SG) of in vivo canine preparations to evaluate the firing of neuronal somata located in this peripheral ganglion during stable cardiovascular state and following local and systemic injection of nicotine. During control periods, minor changes were observed in systolic arterial pressure, the heart rhythm and the atrioventricular conduction time. The activity generated by SG neurons remained relatively constant within each dog, but the firing rate was variable among the preparations. Systemic nicotine administration altered the physiological variables and increased the neuronal activity. Although different patterns of physiological changes were observed among the preparations, it remained invariant upon successive injections in each animal. The behaviour of the neuronal response was similar but varies in amplitude and duration both within and between the dogs. Local injections of nicotine in an artery close to the SG induced a brief and huge burst of neuronal firing, but did not influence the physiological response. The recording technique thus permit to follow the behaviour of multiple intrathoracic neuronal populations located in the SG. The relation between the SG firing and the physiological changes is stable in each dog, but differed between the animals. It suggests that the weight of the different feedback loops involved in the cardiovascular regulation might be a characteristic feature of each animal and/or the position of the electrodes in the SG is critical, since different neuronal populations are present and could react differently.
APA, Harvard, Vancouver, ISO, and other styles

Books on the topic "Ganglions autonomes"

1

Parker, Philip M., and James N. Parker. Ganglions: A medical dictionary, bibliography, and annotated research guide to Internet references. ICON Health Publications, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

Clinical handbook of mindfulness. Springer, 2009.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

McLachlan, A. J. Autonomic Ganglia (Autonomic Nervous System). Informa Healthcare, 1995.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

1918-, Karczmar A. G., Kōketsu Kyōzō 1922-, and Nishi Syogoro, eds. Autonomic and enteric ganglia: Transmission and its pharmacology. Plenum Press, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

1918-, Karczmar A. G., Kōketsu Kyōzō 1922-, and Nishi Syogoro, eds. Autonomic and enteric ganglia: Transmission and its pharmacology. Plenum Press, 1986.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

Geoffrey, Burnstock, and Hoyle, Charles H. V., 1955-, eds. Autonomic neuroeffector mechanisms. Harwood Academic Publishers, 1992.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Clinical Handbook of Mindfulness. Springer, 2010.

Find full text
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Ganglions autonomes"

1

Karczmar, Alexander G. "Historical Development of Concepts of Ganglionic Transmission." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Koketsu, K., and Alexander G. Karczmar. "General Concepts of Ganglionic Transmission and Modulation." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Christ, Daryl D., and Nae J. Dun. "Presynaptic Modulation: Endogenous Substances with Ganglionic Depressant Actions." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Karczmar, Alexander G., and Nae J. Dun. "Pharmacology of Synaptic Ganglionic Transmission and Second Messengers." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_13.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Karczmar, Alexander G. "Autonomic Disease and Clinical Applications of Ganglionic Agents." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_20.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Karczmar, Alexander G. "Ganglionic Transmission as a Model for CNS Function." In Autonomic and Enteric Ganglia. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4615-9436-9_21.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Tronvik, Erling, and Rigmor Jensen. "The Role of the Sphenopalatine Ganglion in Headache Conditions: New Insights." In Cluster Headache and other Trigeminal Autonomic Cephalgias. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-12438-0_10.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Purves, Dale, David A. Johnson, and Richard I. Hume. "Regulation of Synaptic Connections in the Rabbit Ciliary Ganglion." In Ciba Foundation Symposium 83 - Development of the Autonomic Nervous System. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720653.ch12.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Kiraly, M., E. Tribollet, M. Dolivo, and J. J. Dreifuss. "A Presynaptic Action of Vasopressin in the Superior Cervical Ganglion of the Rat." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_58.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Mascorro, J. A., and R. D. Yates. "Anatomy and Morphology of Chromaffin Paraganglia Associated with the Inferior Mesenteric Ganglion in Cats." In Histochemistry and Cell Biology of Autonomic Neurons and Paraganglia. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72749-8_32.

Full text
APA, Harvard, Vancouver, ISO, and other styles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography