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Books on the topic 'Gas Poisoning'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Hepola, Jouko. Sulfur transformations in catalytic hot-gas cleaning of gasification gas. Espoo [Finland]: Technical Research Centre of Finland, 2000.

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2

Fromm, Eckehard. Kinetics of metal-gas interactions at low temperatures: Hydriding, oxidation, poisoning. Berlin: Springer Verlag, 1998.

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3

(Firm), Metra Martech, and Great Britain. Consumer Affairs Directorate., eds. Deaths from poisoning by carbon monoxide from non-gas fuelled domestic heating appliances in rented accommodation. London: Department of Trade and Industry, 1998.

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4

1950-, Paxman Jeremy, ed. A higher form of killing: The secret history of gas and germ warfare. London: Arrow, 2002.

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5

A, Barron Ruth, Rumack Barry H. 1942-, and Physicians for Human Rights (U.S.), eds. Bloody Sunday: Trauma in Tbilisi : the events of April 9, 1989 and their aftermath : report of a medical mission to Soviet Georgia. Somerville, MA (58 Day St., Suite 202, Somerville 02144): Physicians for Human Rights, 1990.

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6

L, Dungworth D., and Mohr U, eds. Inhalation toxicology: The design and interpretation of inhalation studies and their use in risk assessment. Berlin: Springer-Verlag, 1988.

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7

O, McClellan R., and Henderson R. F, eds. Concepts in inhalation toxicology. New York: Hemisphere Pub. Corp., 1989.

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8

Conway, Jeffery. Blood poisoning: Poems. New York City: Cold Calm Press, 1995.

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9

J, Miller Fred, Boorman Gary A, and Chemical Industry Institute of Toxicology., eds. Nasal toxicity and dosimetry of inhaled xenobiotics: Implications for human health. Washington, DC: Taylor & Francis, 1994.

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10

G, Penney David, ed. Carbon monoxide toxicity. Boca Raton: CRC Press, 2000.

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11

Y, Ong Kwok, ed. Detection technologies for chemical warfare agents and toxic vapors. Boca Raton, Fla: CRC Press, 2005.

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12

New Jersey. Legislature. General Assembly. Regulatory Oversight Committee. Committee meeting of Assembly Regulatory Oversight Committee: The committee will discuss the implementation of the Sudan Divesture Act, P.L. 2005, c. 162; discuss the justice gap in New Jersey, which refers to the need for increasing legal representation resources for low-income state residents; revisit the status and implementation of Danielle's Law, P.L. 2003, c. 191; and revisit the status of removing adjudicated juveniles with mental illness from juvenile correction facilities to provide them with mental health treatment : [December 8, 2005, Trenton, New Jersey]. Trenton, NJ: The Unit, 2005.

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13

Great Britain National Health Insurance. Atlas of Gas Poisoning. Creative Media Partners, LLC, 2022.

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14

Great Britain National Health Insurance. Atlas of Gas Poisoning. Creative Media Partners, LLC, 2022.

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15

Medical Aspects of Mustard Gas Poisoning. Creative Media Partners, LLC, 2022.

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16

Medical Aspects of Mustard Gas Poisoning. Creative Media Partners, LLC, 2022.

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17

Weller, Carl Vernon, and Aldred Scott Warthin. Medical Aspects of Mustard Gas Poisoning. Creative Media Partners, LLC, 2018.

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18

Weller, Carl Vernon, and Aldred Scott Warthin. The Medical Aspects Of Mustard Gas Poisoning. Kessinger Publishing, LLC, 2007.

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19

The Medical Aspects of Mustard gas Poisoning. Franklin Classics, 2018.

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20

Kilburn, M.D., Kaye H. Brain Robber: The Poisoning of America by Rotten Egg Gas. ABC-CLIO, LLC, 2009.

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21

Fromm, Eckehard. Kinetics of Metal-Gas Interactions at Low Temperatures: Hydriding, Oxidation, Poisoning. Springer, 2012.

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22

Annane, Djillali, and B. Jérôme Aboab. Management of carbon monoxide poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0328.

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CO poisoning is the commonest cause of toxic death. Carbon monoxide is colourless, odourless, and tasteless, and is produced under various conditions. When people inhale CO, the gas diffuses rapidly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Subsequently, CO causes oxygen deprivation of all body tissues. CO also induces oxidative stress and systemic inflammation. After CO poisoning a broad variety of symptoms may occur. Survivors of CO poisoning often present with persistent neurological sequels or develop delayed neurological symptoms. There is poor correlation between carboxyhaemoglobin levels and clinical symptoms. The presence of coma, underlying co-morbid conditions and need for mechanical ventilation are the main prognostic factors. Management includes prompt extraction from the toxic environment and breathing 100% oxygen, although the role and practicalities of hyperbaric oxygen therapy remain controversial.

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23

Freemantle, Michael. Gas! Gas! Quick, Boys!: How Chemistry Changed the First World War. History Press Limited, The, 2011.

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24

Fromm, Eckehard. Kinetics of Metal-Gas Interactions at Low Temperatures: Hydriding, Oxidation, Poisoning (Springer Series in Surface Sciences). Springer, 2001.

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25

Veterans At Risk: The Health Effects of Mustard Gas and Lewsite. National Academies Press, 1993.

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26

Hovda, Knut Erik, and Dag Jacobsen. Management of alcohol poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0327.

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Ethyl alcohol poisoning is by far the most common alcohol poisoning, and it can usually be handled by simple symptomatic treatment. Methanol and ethylene glycol poisonings share many characteristics, often presenting with a metabolic acidosis of unknown origin. Obtaining the diagnosis could be difficult, and assays for the toxic alcohol are seldom available at hand. The diagnosis should thus be supported by the use of the anion- and osmolal gaps, and/or a simple and specific formate enzymatic method measuring the toxic metabolite itself. A delayed diagnosis of poisonings will often have fatal consequences, in spite of effective treatment available. Treatment mainly consists of buffer, antidote, haemodialysis, and folinic acid (if methanol is suspected) in addition to supportive care. The long-term prognosis of cerebral (unless hypoxic damage)—and kidney function in ethylene glycol poisonings - is good given adequate treatment. Diethylene glycol is also highly toxic, but the knowledge on toxicity is limited. We recommend treatment similar to ethylene glycol poisonings. Other alcohol poisonings are less severe, and will usually present without pronounced metabolic acidosis.

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27

Farmer, Penny. Lead Pollution from Motor Vehicles 1974-86: A Select Bibliography. Taylor & Francis Group, 1987.

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28

Winternitz, Milton Charles, and United States Army Dept of Pathology. Collected Studies on the Pathology of War Gas Poisoning: From the Department of Pathology and Bacteriology Medical Science Section, Chemical Warfare Service. Creative Media Partners, LLC, 2023.

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29

Richter, Donald. Chemical Soldiers: British Gas Warfare in World War One. Pen & Sword Books Limited, 1993.

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30

Richter, Donald. Chemical Soldiers: British Gas Warfare in World War One. Pen & Sword Books Limited, 1993.

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31

Richter, Donald. Chemical Soldiers: British Gas Warfare in World War One. Pen & Sword Books Limited, 1993.

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32

M. C. (Milton C. ) Winternitz and Yale University Anthony N Brady Memo. Collected Studies on the Pathology of War Gas Poisoning from the Department of Pathology and Bacteriology, Medical Science Section, Chemical Warfare Service, under the Direction of M. C. Winternitz . . Creative Media Partners, LLC, 2021.

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33

Richter, Donald. Chemical Soldiers: British Gas Warfare in World War One (Modern War Studies). Trans-Atlantic Publications, 1994.

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34

Dean, Amy E., and Keith Tower. Fire Protection GD on H -OS. National Fire Protection Association (NFPA), 1991.

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35

Fire protection guide on hazardous materials. 9th ed. Quincy, MA: National Fire Protection Association, 1986.

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36

Neligan, Patrick J., and Clifford S. Deutschman. Management of metabolic acidosis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0256.

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Metabolic acidosis (MA) commonly complicates critical illness, usually manifesting as a fall in arterial pH (<7.4) accompanied by a concomitant fall in serum bicarbonate concentration. Acidosis caused by unmeasured anions (UMA), can be distinguished from Hyperchloraemic acidosis by demonstrating a widening of the anion gap (AG). AG should be corrected for albumin and lactate. The base deficit (BD) calculates degree of metabolic acidosis and represents the amount of strong cation required to restore the pH to 7.4. Neither the AG nor the BD specify the cause of acidosis, and are unhelpful in the setting of mixed disorders. The base deficit gap (BDG) is used to calculate the effect of free water, sodium, chloride and albumin on the BD. It is the difference between BDcalc and BDmeasured (on a blood gas) and represents UMA. The strong ion gap more robustly calculates the amount of UMA than AG or BDG, and may be more accurate at predicting outcomes in the emergency room. Lactic acidosis is due to hypovolaemia until otherwise proven. In the majority of cases aggressive fluid resuscitation is warranted. In the presence of normal tissue blood flow regional hypoperfusion, poisoning or exogenous catecholamines should be considered. Ketoacidosis is due to intracellular glucose deficiency, caused by hypoinsulinaemia or starvation. The former is treated with isotonic crystalloid and insulin. Renal acidosis is treated with renal replacement therapy or recovery of renal function.

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37

(Editor), Gary A. Boorman, ed. Nasal Toxicity And Dosimetry Of Inhaled Xenobiotics. CRC, 1995.

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38

Penney, David G. Carbon Monoxide Toxicity. Taylor & Francis Group, 2019.

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39

Penney, David G. Carbon Monoxide Toxicity. Taylor & Francis Group, 2010.

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40

Sun, Yin. Detection Technologies for Chemical Warfare Agents and Toxic Vapors. Taylor & Francis Group, 2004.

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41

Sun, Yin, and Kwok Y. Ong. Detection Technologies for Chemical Warfare Agents and Toxic Vapors. Taylor & Francis Group, 2004.

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42

Sun, Yin, and Kwok Y. Ong. Detection Technologies for Chemical Warfare Agents and Toxic Vapors. Taylor & Francis Group, 2004.

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43

Sun, Yin, and Kwok Y. Ong. Detection Technologies for Chemical Warfare Agents and Toxic Vapors. Taylor & Francis Group, 2004.

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44

Chemical Soldiers. Pen and Sword, 2014.

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45

Christie, Agatha. Und dann gabs keines mehr. Scherz, 2003.

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46

Und dann gabs keines mehr. Hachette Colletions, 2008.

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