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Relevant bibliographies by topics / Gastric necrosis

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Academic literature on the topic 'Gastric necrosis'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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Journal articles on the topic "Gastric necrosis"

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Boukerrouche, Mohammed oussama. "Gastric Conduit Necrosis in Esophageal Surgery." International Journal of Cell Biology and Physiology 05, no. 01 (2022): 01–05. http://dx.doi.org/10.55640/ijcbp-511.

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This study aims to investigate the occurrence, risk factors, clinical presentation, and management of gastric conduit necrosis following esophageal reconstruction, providing insights into its impact on patient outcomes and strategies for prevention and treatment. A retrospective analysis was conducted on patients who underwent esophageal reconstruction with gastric conduit creation between January 2010 and December 2023. Clinical records, including surgical notes, postoperative outcomes, and follow-up data, were reviewed to identify cases of gastric conduit necrosis. Risk factors such as surgi

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Gandhi, Suraj R., Neha S. Shenoy, Vini Joseph, et al. "Extensive gastric necrosis in an infant." International Surgery Journal 8, no. 9 (2021): 2816. http://dx.doi.org/10.18203/2349-2902.isj20213625.

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Gastric necrosis is a rare condition, more so in children and can be potentially fatal. The sloughing of the necrosed wall results in perforations or large defects in the gastric wall. We report a rare case of extensive gastric necrosis in a 2 months old female infant, managed successfully. Child presented with non-bilious vomiting and excessive cry. There was history of recurrent upper abdominal distension. X-ray of abdomen showed massive pneumoperitoneum. Intra operatively, posterior gastric wall was sloughed off and lower end of feeding tube was lying in the peritoneal cavity. Necrotic wall

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Kant, Shashi. "Gastric tip necrosis." European Journal of Cardio-Thoracic Surgery 28, no. 2 (2005): 359. http://dx.doi.org/10.1016/j.ejcts.2005.04.027.

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Yeo, R. "Avascular gastric necrosis." British Journal of Surgery 77, no. 4 (1990): 475. http://dx.doi.org/10.1002/bjs.1800770438.

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Cullinane, Daniel C., Amanda R. Hein, and Henry I. Grant. "Gastric Necrosis Due to Gastric Distension." American Surgeon 85, no. 3 (2019): 137–38. http://dx.doi.org/10.1177/000313481908500308.

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Albuquerque, Andreia, Pedro Bastos, and Guilherme Macedo. "Gastric necrosis caused by gastric banding." Gastrointestinal Endoscopy 75, no. 5 (2012): 1098–99. http://dx.doi.org/10.1016/j.gie.2011.12.024.

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de Papp, Erika, Kenneth J. Drobatz, and Dez Hughes. "Plasma lactate concentration as a predictor of gastric necrosis and survival among dogs with gastric dilatation-volvulus: 102 cases (1995-1998)." Journal of the American Veterinary Medical Association 215, no. 1 (1999): 49–52. http://dx.doi.org/10.2460/javma.1999.215.01.49.

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Objective To determine relationships between plasma lactate concentration and gastric necrosis and between plasma lactate concentration and outcome for dogs with gastric dilatation-volvulus. Design Retrospective study. Animals 102 dogs. Procedure Information on signalment, history, plasma lactate concentration, medical and surgical treatment, cost of hospitalization, and outcome was retrieved from medical records. Results 69 of 70 (99%) dogs with plasma lactate concentration < 6.0 mmol/L survived, compared with 18 of 31 (58%) dogs with plasma lactate concentration > 6.0 mmol/L (1 dog eut

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López, Rocío González, M. Muinelo Lorenzo, J. Pérez Grobas, et al. "Idiopathic gastric partial necrosis." Open Journal of Gastroenterology 03, no. 07 (2013): 314–16. http://dx.doi.org/10.4236/ojgas.2013.37055.

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FRUCHTER, G. "Gastric necrosis: a complication of gastric banding." American Journal of Gastroenterology 98, no. 9 (2003): S216. http://dx.doi.org/10.1016/s0002-9270(03)01424-2.

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Fruchter, Gerald, Vlado Simko, Hatem Shoukeir, Hueldine Webb, and Ayse Aytaman. "GASTRIC NECROSIS: A COMPLICATION OF GASTRIC BANDING." American Journal of Gastroenterology 98 (September 2003): S216. http://dx.doi.org/10.1111/j.1572-0241.2003.08391.x.

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Dissertations / Theses on the topic "Gastric necrosis"

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Goto, Hidemi. "Helicobacter pylori and gastric diseases." Nagoya University School of Medicine, 2003. http://hdl.handle.net/2237/5386.

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Emch, Gregory Simon. "EFFECTS OF TUMOR NECROSIS FACTOR-ALPHA ON DORSAL VAGAL COMPLEX NEURONS THAT EXERT REFLEX CONTROL OF THE GASTROINTESTINAL TRACT." The Ohio State University, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=osu1018475175.

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Peterson, Richard A. II. "Investigations into the role of proinflammatory cytokines in the pathogenesis of gastric epithelial proliferation in chronic helicobacter pylori gastritis." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1055881993.

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Prévost, Frédéric. "Gastrites ulcéro-nécrotiques aiguës cryptogéniques : à propos de 13 observations." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25288.

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Ouziel, Romy. "Contribution des carences en vitamines A et D dans le processus inflammatoire des maladies alcooliques du foie." Doctoral thesis, Universite Libre de Bruxelles, 2015. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/217907.

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Les maladies alcooliques du foie (MAF) sont responsables de 3,8% de la mortalité mondiale et restent une cause majeure de mortalité par maladies hépatiques, plus importante que l’hépatite C. Malgré le fardeau qu’elles représentent, tant sur le plan de la morbi-mortalité que sur le plan économique, il n’existe que très peu d’options thérapeutiques hormis l’abstinence et la transplantation hépatique. Une meilleure compréhension de la pathophysiologie et des facteurs impliqués dans les MAF permettrait d’identifier de nouvelles cibles thérapeutiques afin d’améliorer la prise en charge et le pronos

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Books on the topic "Gastric necrosis"

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Ware, Lorraine B. Pathophysiology of acute respiratory distress syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0108.

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The acute respiratory distress syndrome (ARDS) is a syndrome of acute respiratory failure characterized by the acute onset of non-cardiogenic pulmonary oedema due to increased lung endothelial and alveolar epithelial permeability. Common predisposing clinical conditions include sepsis, pneumonia, severe traumatic injury, and aspiration of gastric contents. Environmental factors, such as alcohol abuse and cigarette smoke exposure may increase the risk of developing ARDS in those at risk. Pathologically, ARDS is characterized by diffuse alveolar damage with neutrophilic alveolitis, haemorrhage,

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Rajendram, Rajkumar. Management of acute pancreatitis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0191.

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The major causes of morbidity and mortality in acute pancreatitis are organ dysfunction and infection of necrotic tissue. Management should aim to prevent, or to diagnose and treat, the complications of pancreatic inflammation, and any predisposing factors to avoid recurrence. Medical management is essentially supportive with oxygen, intravenous fluids, analgesia, enteral or parenteral nutrition, and correction of metabolic abnormalities. Patients with severe acute pancreatitis are unlikely to resume prompt oral intake so nutritional support is also required. Post-pyloric feeding is not requir

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Book chapters on the topic "Gastric necrosis"

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Khan, Mansoor. "Gastric Necrosis from Short Gastric Embolization." In Encyclopedia of Trauma Care. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_131.

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Tejedor-Tejada, Javier, Ameya Deshmukh, Ahmed Mohammed Elmeligui, and Jose Nieto. "EUS Gastric Access for Therapeutic Endoscopy for Management of a Walled Off Necrosis with a LAM Stent in Gastric Bypass Anatomy." In Atlas of Interventional EUS. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-9340-3_32.

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Rezaeian, F., R. Wettstein, P. Mörsdorf, M. D. Menger, and Y. Harder. "Pharmacological induction of the “delay phenomenon”: Ghrelin, a gastric peptide to prevent musculocutaneous necrosis from ischemia?" In Deutsche Gesellschaft für Chirurgie. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-00625-8_103.

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"Post Embolization Gastric Necrosis." In Encyclopedia of Trauma Care. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_101185.

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Primrose, Sandy B. "Helicobacter pylori and Gastric Ulcers." In Microbiology of Infectious Disease. Oxford University Press, 2022. http://dx.doi.org/10.1093/oso/9780192863843.003.0010.

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Gastritis and peptic ulcers are caused by Helicobacter pylori and can be treated with antibiotics. Helicobacter pylori is the only bacterium that can cause cancer. To grow at the pH of the stomach, the bacterium produces urease that cleaves urea to ammonia and bicarbonate. The ammonia neutralizes the stomach acid and damages the gastric mucosa. The urease requires nickel for action and Helicobacter produces nickel-scavenging proteins. The increased pH caused by ammonia liquefies the mucus layer and allows the bacteria to attach to the gastric epithelium where it uses a type VI secretion system

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Rao Thammineedi, Subramanyeshwar, Srijan Shukla, Nusrath Syed, Ajesh Raj Saksena, Sujit Chyau Patnaik, and Pratap Reddy Ramalingam. "Scope of Real Time Fluorescence Imaging in Esophagectomy." In Esophageal Surgery - Current Principles and Advances [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.107267.

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Esophagectomy is a challenging surgery that is known to be associated with high rates of morbidity. Anastomotic leaks, pneumonia, conduit necrosis and chyle leaks are the commonly reported complications. Perfusion assessment and tissue injection based fluorescence guided surgery (FGS) are the newer clinical applications of fluorescent dyes. With the advent and integration of real time fluorescence imaging with the existing minimal access platforms, the esophageal surgeon can employ these techniques to potentially improve outcomes. During thoracic dissection, thoracic duct lymphography, fluores

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"Construction, analysis and correlation of dose response logistic curves for repurposed drugs anticancer effects on fibrosarcoma in hamsters." In Book of Abstracts - RAD 2025 Conference. RAD Centre, Niš, Serbia, 2025. https://doi.org/10.21175/rad.abstr.book.2025.7.1.

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Logarithmic dose response curves are typically „S“-shaped, monotonic and can be fit to a classical Hill equation. The Hill equation is logistic curve – an extension of the sigmoid functions.Although in vitro (cell lines) anticancer effects of some nononcological pleiotropic drugs, such as metformin and itraconazole are reported, in vivo (experimental animals) and clinical investigations, especially of their combinations, are not yet conducted. The study was conducted on Syrian golden hamsters (10-12 weeks old; weight, ~80 g). The animals were randomized into groups (6 hamsters per group). The

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"Rescue control experiments to regain the knockdown anticancer effects of repurposed drugs on fibrosarcoma in hamsters." In Book of Abstracts - RAD 2025 Conference. RAD Centre, Niš, Serbia, 2025. https://doi.org/10.21175/rad.abstr.book.2025.7.3.

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Literature analysis shows that repurposed non-oncological drugs, such as metformin and disulfiram, act through a lot of same anticancer mechanisms, on the same targets, in the same direction. Also, for majority of these mechanisms, non-oncological drug deoxycholic acid acts in the opposite pro- cancer direction. If the rescuing target/protein is expressed at sufficient level that it can reciprocally modulate the pathway of interest, the rescue experiment provide evidence for underlying mechanism of investigated process. To test whether NF-kB and HIF-1a inhibition underlies the anticancer mecha

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Rosa, Hanna Bárbara Lopes, and Eduarda Cristina Borges Dorneles. "Intussuscepção." In Emergências em gastroenterologia abordagem prática para estudantes. Even3 Publicações, 2025. https://doi.org/10.29327/5495368.1-13.

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A intussuscepção é a invaginação de um segmento do trato gastrointestinal no segmento adjacente, causando obstrução e isquemia. Predomina em crianças, sendo rara em adultos, onde cerca de 90% dos casos estão associados a patologias preexistentes, como neoplasias. Fatores de risco incluem alterações anatômicas, pólipos, aderências pós-cirúrgicas, tumores benignos, divertículos, inflamações intestinais e uso de tubo de gastrostomia. A fisiopatologia envolve a invaginação de um segmento intestinal (intussusceptum) no segmento adjacente (intussuscipiens), resultando em obstrução e isquemia, podend

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Conference papers on the topic "Gastric necrosis"

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Ouazzani, S., D. Lorenzo, M. Fernandez, et al. "Regeneration of Epithelialized Digestive Continuity after Endoscopic Stenting for Circumferential Oesogastric Junction Necrosis Following Roux-EN-Y Gastric Bypass." In ESGE Days 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1724277.

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De Angelis, V., M. Zambon, L. Toffolo, C. Donada, G. L. Molaro, and R. Zuin. "ACTIVATION OF FACTOR VII IS RELATED TO BLEEDING TENDENCY IN LIVER CIRRHOSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644800.

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Coagulation abnormalities are among the number of potential risk factors toinitiate the bleeding episodes from gastrcr-esophageal varices in liver cirrhosis. The impairment of liver clearance of activated coagulation factors, the release of thromboplastin-like activity from the necrotic liver cells and the hemodynamic changes due to expanded bollaterals may all contribute to activate the coagulation cascade.However, little is known about the mechanisms leading to this activation. Activated Factor VII (FVIIa) is known totrigger both intrinsec and extrinsec coagulation pathway. Therefore, we mea

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Reports on the topic "Gastric necrosis"

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Lin, Chin-Lang, Xiu-Wei Li, Kevin Sheu, and Chien-Ho Wang. Comparison of different trans-gastric stents for drainage of necrotic collection in patients with necrotizing pancreatitis: a systematic review and network meta-analysis of randomized control trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.12.0086.

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