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1

Boukerrouche, Mohammed oussama. "Gastric Conduit Necrosis in Esophageal Surgery." International Journal of Cell Biology and Physiology 05, no. 01 (2022): 01–05. http://dx.doi.org/10.55640/ijcbp-511.

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This study aims to investigate the occurrence, risk factors, clinical presentation, and management of gastric conduit necrosis following esophageal reconstruction, providing insights into its impact on patient outcomes and strategies for prevention and treatment. A retrospective analysis was conducted on patients who underwent esophageal reconstruction with gastric conduit creation between January 2010 and December 2023. Clinical records, including surgical notes, postoperative outcomes, and follow-up data, were reviewed to identify cases of gastric conduit necrosis. Risk factors such as surgi
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2

Gandhi, Suraj R., Neha S. Shenoy, Vini Joseph, et al. "Extensive gastric necrosis in an infant." International Surgery Journal 8, no. 9 (2021): 2816. http://dx.doi.org/10.18203/2349-2902.isj20213625.

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Gastric necrosis is a rare condition, more so in children and can be potentially fatal. The sloughing of the necrosed wall results in perforations or large defects in the gastric wall. We report a rare case of extensive gastric necrosis in a 2 months old female infant, managed successfully. Child presented with non-bilious vomiting and excessive cry. There was history of recurrent upper abdominal distension. X-ray of abdomen showed massive pneumoperitoneum. Intra operatively, posterior gastric wall was sloughed off and lower end of feeding tube was lying in the peritoneal cavity. Necrotic wall
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3

Kant, Shashi. "Gastric tip necrosis." European Journal of Cardio-Thoracic Surgery 28, no. 2 (2005): 359. http://dx.doi.org/10.1016/j.ejcts.2005.04.027.

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4

Yeo, R. "Avascular gastric necrosis." British Journal of Surgery 77, no. 4 (1990): 475. http://dx.doi.org/10.1002/bjs.1800770438.

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5

Cullinane, Daniel C., Amanda R. Hein, and Henry I. Grant. "Gastric Necrosis Due to Gastric Distension." American Surgeon 85, no. 3 (2019): 137–38. http://dx.doi.org/10.1177/000313481908500308.

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6

Albuquerque, Andreia, Pedro Bastos, and Guilherme Macedo. "Gastric necrosis caused by gastric banding." Gastrointestinal Endoscopy 75, no. 5 (2012): 1098–99. http://dx.doi.org/10.1016/j.gie.2011.12.024.

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7

de Papp, Erika, Kenneth J. Drobatz, and Dez Hughes. "Plasma lactate concentration as a predictor of gastric necrosis and survival among dogs with gastric dilatation-volvulus: 102 cases (1995-1998)." Journal of the American Veterinary Medical Association 215, no. 1 (1999): 49–52. http://dx.doi.org/10.2460/javma.1999.215.01.49.

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Objective To determine relationships between plasma lactate concentration and gastric necrosis and between plasma lactate concentration and outcome for dogs with gastric dilatation-volvulus. Design Retrospective study. Animals 102 dogs. Procedure Information on signalment, history, plasma lactate concentration, medical and surgical treatment, cost of hospitalization, and outcome was retrieved from medical records. Results 69 of 70 (99%) dogs with plasma lactate concentration < 6.0 mmol/L survived, compared with 18 of 31 (58%) dogs with plasma lactate concentration > 6.0 mmol/L (1 dog eut
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8

López, Rocío González, M. Muinelo Lorenzo, J. Pérez Grobas, et al. "Idiopathic gastric partial necrosis." Open Journal of Gastroenterology 03, no. 07 (2013): 314–16. http://dx.doi.org/10.4236/ojgas.2013.37055.

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9

FRUCHTER, G. "Gastric necrosis: a complication of gastric banding." American Journal of Gastroenterology 98, no. 9 (2003): S216. http://dx.doi.org/10.1016/s0002-9270(03)01424-2.

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10

Fruchter, Gerald, Vlado Simko, Hatem Shoukeir, Hueldine Webb, and Ayse Aytaman. "GASTRIC NECROSIS: A COMPLICATION OF GASTRIC BANDING." American Journal of Gastroenterology 98 (September 2003): S216. http://dx.doi.org/10.1111/j.1572-0241.2003.08391.x.

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11

Li, Wei-Feng, Ruo-Yu Gao, Jing-Wen Xu, and Xi-Qiu Yu. "Endoscopic treatment of bleeding gastric ulcer causing gastric wall necrosis: A case report." World Journal of Gastrointestinal Surgery 16, no. 4 (2024): 1203–7. http://dx.doi.org/10.4240/wjgs.v16.i4.1203.

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BACKGROUND Gastric wall necrosis is a rare complication of endoscopic treatment for bleeding gastric ulcer, which may exacerbate the patient’s condition once it occurs and may even require surgical intervention for treatment. CASE SUMMARY A 59-year-old man was admitted to our department with melena. Endoscopy revealed a giant ulcer in the gastric antrum with a visible vessel in its center, which was treated with sclerosants and tissue glue injection and resulted in necrosis of the gastric wall. CONCLUSION Injection of sclerosants and tissue glue may lead to gastric wall necrosis, which is a se
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12

Aydin, Ibrahim, Ahmet Pergel, Ahmet Fikret Yucel, Dursun Ali Sahin, and Ender Ozer. "Gastric Necrosis due to Acute Massive Gastric Dilatation." Case Reports in Medicine 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/847238.

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Gastric necrosis due to acute massive gastric dilatation is relatively rare. Vascular reasons, herniation, volvulus, acute gastric dilatation, anorexia, and bulimia nervosa play a role in the etiology of the disease. Early diagnosis and treatment are highly important as the associated morbidity and mortality rates are high. In this case report, we present a case of gastric necrosis due to acute gastric dilatation accompanied with the relevant literature.
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13

Shah, Malay B., James G. Bittner, and Michael A. Edwards. "Rett Syndrome and Gastric Perforation." American Surgeon 74, no. 4 (2008): 315–17. http://dx.doi.org/10.1177/000313480807400407.

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Rett Syndrome is associated with decreased peristaltic esophageal waves and gastric dysmotility, resulting in swallowing difficulties and gastric dilation. Rarely, gastric necrosis and perforation occur. Our case represents the third reported case of gastric necrosis and perforation associated with Rett Syndrome. A 31-year-old female after 11 hours of intermittent emesis and constant, sharp abdominal pain presented with evidence of multiorgan system failure including hypovolemic shock, metabolic acidosis, coagulopathy, and hepatorenal failure. A chest radiograph revealed intra-abdominal free a
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14

Shukla, RamMohan, KartikChandra Mandal, Sujay Maitra, et al. "Gastric volvulus with partial and complete gastric necrosis." Journal of Indian Association of Pediatric Surgeons 19, no. 1 (2014): 49. http://dx.doi.org/10.4103/0971-9261.125968.

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15

Murata, Toshihiro, Hirokazu Uetsuka, Masashi Uda, Osamu Kawamata, Hajime Nakai, and Tamotsu Ohta. "A Case of Gastric Necrosis with Gastric Cancer." Japanese Journal of Gastroenterological Surgery 41, no. 5 (2008): 505–9. http://dx.doi.org/10.5833/jjgs.41.505.

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16

Kirchmayr, Werner, Karlheinz Ammann, Franz Aigner, Helmut G. Weiss, and Hermann Nehoda. "Pouch Dilatation after Gastric Banding Causing Gastric Necrosis." Obesity Surgery 11, no. 6 (2001): 770–72. http://dx.doi.org/10.1381/09608920160558768.

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17

Iannelli, Antonio, Enrico Facchiano, Eric Sejor, Patrick Baqué, Thierry Piche, and Jean Gugenheim. "Gastric Necrosis: a Rare Complication of Gastric Banding." Obesity Surgery 15, no. 8 (2005): 1211–14. http://dx.doi.org/10.1381/0960892055002194.

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18

Ramage, L., J. Deguara, A. Davies, et al. "Gastric tube necrosis following minimally invasive oesophagectomy is a learning curve issue." Annals of The Royal College of Surgeons of England 95, no. 5 (2013): 329–34. http://dx.doi.org/10.1308/003588413x13629960045751.

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Introduction Gastric tube necrosis following oesophagectomy is thought to have an increased association with a minimally invasive technique. Some suggest gastric ischaemic preconditioning may reduce ischaemic complications. We discuss our series of 155 consecutive minimally invasive oesophagectomies (MIOs), including a number of cases of gastric tube ischaemia, of which 4 (2.6%) developed conduit necrosis. Methods Data were collected prospectively of MIOs carried out by a single surgeon between 2005 and 2011. Cases of gastric tube necrosis were identified. Results Overall, 155 patients were id
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19

Vargas, Frédéric, Gilles Hilbert, Ruddy Valentino, et al. "An idiopathic total gastric necrosis." Intensive Care Medicine 27, no. 1 (2000): 331–32. http://dx.doi.org/10.1007/s001340000764.

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20

Grover, Kunal, Bingru Xie, and Weizheng Wang. "Alcohol Induced Ischemic Gastric Necrosis." American Journal of Gastroenterology 103 (September 2008): S306. http://dx.doi.org/10.14309/00000434-200809001-00776.

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21

Putnam, Tyler, Erica Turse, and Kambiz Kadkhodayan. "2698 Spontaneous Vasogenic Gastric Necrosis." American Journal of Gastroenterology 114, no. 1 (2019): S1490. http://dx.doi.org/10.14309/01.ajg.0000600324.23701.74.

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22

Yorke, Joseph, Frank Enoch Gyamfi, Ronald Awoonor-Williams, et al. "Acute Gastric Necrosis in a Teenager." Case Reports in Surgery 2020 (September 25, 2020): 1–4. http://dx.doi.org/10.1155/2020/8882179.

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Gastric infarction is a rare condition often associated with high mortality due to a delay in diagnosis. The stomach which has a rich supply of blood is a rare site for such a condition. Gastric infarction has a long list of etiological factors. We report a case of a patient who was managed successfully following gastric infarction from gastric dilatation. An 18-year-old female student presented with a three-day history of abdominal pain associated with abdominal distension of two days. The abdomen was distended with generalized tenderness, rebound tenderness, and guarding. Bowel sounds were a
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23

Rakotomena, Davidà S., Aurélia Rakotondrainibe, Gabrielle E. P. Andrianah, Tovohery A. Rajaonera, and Hery N. Rakoto-Ratsimba. "Necrose Etendue De L’estomac Sur Dilatation Gastrique Chez Un Patient Suivi Pour Tumeur Cerebelleuse." European Scientific Journal, ESJ 14, no. 27 (2018): 21. http://dx.doi.org/10.19044/esj.2018.v14n27p21.

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Acute necrosis with a gastric dilatation is very rare. It usually occurs on psychiatric and neurological field. The preoperative diagnosis is difficult, and it is often revealed by radiological exams. With or without surgery, mortality remains high. We report a case of gastric necrosis in a patient with a severe neurological history. The clinical signs were vomiting, without intestinal transit disorders, and poor general condition. The radiological exams revealed gastric gas gangrene in the stomach wall. The surgical management determined the limit of gastric necrosis and gastrectomy was perfo
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24

Ishiguro, Toru, Youichi Kumagai, Tomojiro Ono, et al. "Usefulness of Indocyanine Green Angiography for Evaluation of Blood Supply in a Reconstructed Gastric Tube During Esophagectomy." International Surgery 97, no. 4 (2013): 340–44. http://dx.doi.org/10.9738/cc159.1.

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Abstract We report a case of necrosis of a reconstructed gastric tube in a 77-year-old male patient who had undergone esophagectomy. At the time of admission, the patient had active gastric ulcers, but these were resolved by treatment with a proton pump inhibitor. Subtotal esophagectomy with gastric tube reconstruction was performed. Visually, the reconstructed gastric tube appeared to be well perfused with blood. Using indocyanine green (ICG) fluorescence imaging the gastroepiploic vessels were well enhanced and no enhancement was visable 3 to 4 cm from the tip of the gastric tube. Four days
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25

Gundes, Ebubekir, Tevfik Kucukkartallar, Ahmet Tekin, and Murat Cakir. "Gastric necrosis and perforation caused by acute gastric dilatation." Turkish Journal of Surgery 30, no. 3 (2014): 180–82. http://dx.doi.org/10.5152/ucd.2013.34.

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26

REEVE, THOMAS, BLAIR JACKSON, CAROL SCOTT-CONNER, and CHARLES SLEDGE. "Near-Total Gastric Necrosis Caused by Acute Gastric Dilatation." Southern Medical Journal 81, no. 4 (1988): 515–17. http://dx.doi.org/10.1097/00007611-198804000-00027.

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27

Ghanayem, B. I., P. J. Boor, and A. E. Ahmed. "Acrylonitrile-induced gastric mucosal necrosis: role of gastric glutathione." Journal of Pharmacology and Experimental Therapeutics 232, no. 2 (1985): 570–77. https://doi.org/10.1016/s0022-3565(25)20621-8.

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28

Low, Vincent H. S., and Richard I. Thompson. "Gastric emphysema due to necrosis from massive gastric distention." Clinical Imaging 19, no. 1 (1995): 34–36. http://dx.doi.org/10.1016/0899-7071(94)00011-z.

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29

Barker, J. A., H. Burnett, and G. L. Carlson. "Gastric necrosis complicating acute gastric dilatation after Nissen fundoplication." Case Reports 2011, jun16 1 (2011): bcr0220113811. http://dx.doi.org/10.1136/bcr.02.2011.3811.

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30

Todd, S. Rob, Gary T. Marshall, and Alan H. Tyroch. "Acute Gastric Dilatation Revisited." American Surgeon 66, no. 8 (2000): 709–10. http://dx.doi.org/10.1177/000313480006600801.

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First described by S.E. Duplay in 1833, acute gastric dilatation has since been well documented in the literature. Several theories of the pathogenesis of acute gastric dilatation have been postulated. In 1842, Karl Freiherr von Rokitansky described the superior mesenteric artery syndrome, followed by W. Brinton in 1859 with the atonic theory. C.R. Morris et al. introduced debilitation and anesthesia as predisposing factors. Although rare, gastric necrosis is the most severe consequence of acute gastric dilatation. Vascular insufficiency secondary to increased intragastric pressure is the crit
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31

Ahmadinejad, Mjtaba, Elham Valimohammdi, Ghazale Molaverdi, Seydehamideh Hashemi, Mohammad Hadi Bahri, and Javad Zebarjadi Bagherpour. "Can Heroin Poisoning Lead to Stomach Necrosis? A Case Report." International Journal of Medical Toxicology and Forensic Medicine 12, no. 4 (2022): 38932. http://dx.doi.org/10.32598/ijmtfm.v12i4.38932.

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Background: According to the last report of the United Nations Office on Drugs and Crime (UNOCD), opiate use (including heroin) is 1.2% of the world population. In Iran, about 2 million people are drug addicts. Heroin reduces gastric motility and prolongs gastric emptying time and causes gastric dilation which can be a reason for gastric ischemia. Gastric ischemia is an uncommon condition due to the rich gastric blood perfusion and collateral arteries. As some studies show, gastric dilation can be the cause of gastric necrosis. Methods: A 22-year-old woman presented to our hospital with severe
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32

De Vega, Marta, Carlos Miliani, Juan Antonio Martinez-Piñeiro, Debora Acin, and Fernando Pereira. "PS01.077: GASTRIC NECROSIS WITH PERFORATION AS LATE COMPLICATION OF NISSEN FUNDUPLICATION." Diseases of the Esophagus 31, Supplement_1 (2018): 71. http://dx.doi.org/10.1093/dote/doy089.ps01.077.

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Abstract Background Gastric necrosis and perforation after Nissen funduplication is a rare and life-treatening complication. Prompt diagnosis of acute gastric dilatation is mandatory to avoid gastric necrosis. A tight funduplication can cause a gastric dilatation because of the inability to vomit. Other causes of gastric dlatation include trauma, volvulus, anorexia and bulimia diabetes, polyphagia, acute infections and others. Methods A 56-year-old man was admited to emergency room with acute-onset epigastric pain and persistent nausea for the past 4 hours. Nine years ago he underwent a laparo
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33

Vallieres, Eric, and Paul F. Waters. "Incarcerated Parahiatal Hernia with Gastric Necrosis." Annals of Thoracic Surgery 44, no. 1 (1987): 82–83. http://dx.doi.org/10.1016/s0003-4975(10)62365-x.

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34

Pueyo Périz, Eva María, Serafín Andrés Pérez, Aitana García Tejero, and Carlota García Fernández. "Gastric Necrosis Secondary to Inguinal Incarceration." Cirugía Española (English Edition) 94, no. 3 (2016): 180. http://dx.doi.org/10.1016/j.cireng.2016.02.009.

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35

Papanikolaou, I., P. Foukas, A. Sioulas, et al. "A case of gastric ischemic necrosis." Endoscopy 43, S 02 (2011): E342. http://dx.doi.org/10.1055/s-0030-1256795.

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36

Ajit, Nisha, Kovid Trivedi, and Robert Walter. "Wilkie’s Syndrome Causing Acute Gastric Necrosis." Chest 150, no. 4 (2016): 407A. http://dx.doi.org/10.1016/j.chest.2016.08.420.

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37

MAGALHÃES, Maria Angélica B., Alfredo J. A. BARBOSA, Juliano A. FIGUEIREDO, Luiz R. ALBERTI, and Andy PETROIANU. "EFFECTS OF DIFFERENT PERIODS OF GASTRIC ISCHAEMIA IN THE VIABILITY OF THE TISSUE OF BODY, FUNDUS AND ANTRUM REGION OF RABBIT STOMACH." ABCD. Arquivos Brasileiros de Cirurgia Digestiva (São Paulo) 28, no. 3 (2015): 167–70. http://dx.doi.org/10.1590/s0102-67202015nahead00001.

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BACKGROUND: Despite the rich vascular arcade of the stomach, gastric ischemia represents an important medical challenge and can be the consequence of obstructive or non-obstructive vascular processes of pathological or iatrogenic origin. AIM: To assess the effects of acute gastric ischaemia on the different regions of the stomach. METHOD: Fifteen New Zeland rabbits were divided into three groups: group 1, animals were observed during 3 h; group 2, during 6 h; group 3, during 12 h. Rabbit stomachs were subjected to devascularization of the greater and lesser curvatures. After predetermined time
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38

Parys, S., and S. Werapitiya. "Acutely slipped gastric band with gastric necrosis and massive haemorrhage." Journal of Surgical Case Reports 2013, no. 8 (2013): rjt053. http://dx.doi.org/10.1093/jscr/rjt053.

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39

Laverick, Sophie, Udoka Ogbuneke, Ioannis Koumoutsos, and Zahed Ahmed. "Acute oesophageal necrosis: an important differential in the elderly population with haematemesis." BMJ Case Reports 14, no. 7 (2021): e243055. http://dx.doi.org/10.1136/bcr-2021-243055.

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Acute oesophageal necrosis is a rare cause of haematemesis associated with high mortality and morbidity in elderly patients with multiple comorbidities. Acute oesophageal necrosis is thought to be caused by a combination of hypoperfusion, vascular disease and duodenal disease causing transient gastric outlet obstruction and therefore reflux of gastric contents. The subsequent necrosis is associated with significant morbidity and mortality. We present a case of an 83-year-old man presenting with sepsis secondary to gallstone cholangitis, who developed haematemesis 2 days post admission. Oesopho
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40

Gün, Feryal, Latif Abbasoğlu, and Alaaddin Çelik. "Acute Gastric Perforation after Acid Ingestion." Journal of Pediatric Gastroenterology and Nutrition 35, no. 3 (2002): 360–62. http://dx.doi.org/10.1002/j.1536-4801.2002.tb07835.x.

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ABSTRACTGastric outlet obstruction is a common late result after acid ingestion; early complications, such as gastric necrosis or perforations are unusual. This is a report of a patient with the history of strong acid ingestion who underwent total gastrectomy due to perforation and extensive necrosis of the stomach.
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41

Liu, Chi-Feng, Chun-Ching Lin, Mei-Hsiu Lin, Yi-Shiu Lin, and Song-Chow Lin. "Cytoprotection by Propolis Ethanol Extract of Acute Absolute Ethanol-Induced Gastric Mucosal Lesions." American Journal of Chinese Medicine 30, no. 02n03 (2002): 245–54. http://dx.doi.org/10.1142/s0192415x02000387.

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Acute p.o. administration of absolute ethanol (1.0 ml/kg) to fasted rats produced extensive necrosis of gastric mucosa. Pretreatment with p.o. administration of propolis ethanol extract (PEE) could effectively and dose-dependently prevent such necrosis. This protective effect is called "cytoprotection." The maximal cytoprotective effect against absolute ethanol (AE)-induced gastric mucosal lesion was observed 1 hour after PEE administration. A gross examination of the gastric mucosa showed a marked improvement in groups receiving PEE. In order to further investigate the gastric protective mech
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42

Vostroknutov, I. V., A. V. Kholmansky, A. F. Egorov, et al. "Successful treatment of a patient with idiopathic gastric necrosis." Grekov's Bulletin of Surgery 181, no. 1 (2022): 104–6. http://dx.doi.org/10.24884/0042-4625-2022-181-1-104-106.

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Necrosis of the stomach as an independent disease is quite rare, even less often - total idiopathic necrosis. We present a case of successful treatment of a patient with idiopathic stomach necrosis with a good outcome.
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43

Askarpour, Shahnam, Hazhir Javaherizadeh, Mahboobeh Rashidi, Mahmood Khoshkhabar, and Afshin Rezazadeh. "Gastric Necrosis in a Previously Healthy Child: A Case Report." Middle East Journal of Digestive Diseases 16, no. 3 (2024): 200–202. http://dx.doi.org/10.34172/mejdd.2024.392.

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Gastric necrosis is a very rare surgical emergency in a previously healthy child. A 13-year-old boy with abdominal pain and coffee-ground vomiting was admitted to the emergency department. Physical examination revealed signs of peritonitis and septic shock. The patient underwent a laparotomy. Gastric necrosis and discoloration of the lower esophagus and duodenum due to ischemia were present. Distention of gastric and duodenum was also seen. Total gastrectomy and Roux-en-Y esophagojejunostomy were done. The patient underwent a chest computed tomography (CT), and patchy ground-glass opacity was
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44

Nishikawa, Katsunori, Yujiro Tanaka, Yuichiro Tanishima, et al. "PS01.168: IS IT POSSIBLE TO PREVENT GASTRIC TUBE NECROSIS FOLLOWING ESOPHAGECTOMY?" Diseases of the Esophagus 31, Supplement_1 (2018): 97. http://dx.doi.org/10.1093/dote/doy089.ps01.168.

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Abstract Background Gastric tube necrosis (GN) following esophagectomy is a rare, but critical and life threatening complication. Unlike anastomotic leakage due to local ischemia, GN involves extensive full thickness ischemia resulting from vascular insufficiency. Most cases of GN need total or partial replacement of gastric tube. Although quantitative assessment of tissue perfusion during esophageal surgery contributed to reduce the incidence of postoperative anastomotic complications, GN remains a serious complication to be solved. Methods Data were collected retrospectively from 271 patient
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45

Thalmaier, Ulrike, Norbert Lehn, Klaus Pfeffer, Manfred Stolte, Michael Vieth, and Wulf Schneider-Brachert. "Role of Tumor Necrosis Factor Alpha in Helicobacter pylori Gastritis in Tumor Necrosis Factor Receptor 1-Deficient Mice." Infection and Immunity 70, no. 6 (2002): 3149–55. http://dx.doi.org/10.1128/iai.70.6.3149-3155.2002.

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ABSTRACT Increased gastric production of interleukin 8 and tumor necrosis factor alpha (TNF-α) has been implicated in the pathogenesis of Helicobacter pylori-associated gastroduodenal disease. In the present study we used a mouse model to demonstrate whether loss of the tumor necrosis factor receptor 1 (TNF-R1) function leads to differences in gastric inflammation or the systemic immune response in H. pylori infection. Six different clinical isolates of H. pylori (three cytotoxin-positive and three cytotoxin-negative strains) were adapted to C57BL/6 mice. TNF-R1-deficient (TNF-R1−/−) mice (n =
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46

Powell, John L., Joseph Payne, Clinton L. Meyer, and Paul R. Moncla. "Gastric necrosis associated with acute gastric dilatation and small bowel obstruction." Gynecologic Oncology 90, no. 1 (2003): 200–203. http://dx.doi.org/10.1016/s0090-8258(03)00204-x.

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47

Murakami, Chikako, Wataru Irie, Chizuko Sasaki, et al. "Extensive gastric necrosis secondary to acute gastric dilatation: A case report." Legal Medicine 36 (February 2019): 85–88. http://dx.doi.org/10.1016/j.legalmed.2018.11.007.

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48

Lunca, Sorinel, Michel Vix, Andrew Rikkers, Francesco Rubino, and Jacques Marescaux. "Late Gastric Prolapse with Pouch Necrosis after Laparoscopic Adjustable Gastric Banding." Obesity Surgery 15, no. 4 (2005): 571–75. http://dx.doi.org/10.1381/0960892053723420.

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49

MILLIS, DARRYL L., JOE G. HAUPTMAN, and ROBERT B. FULTON. "Abnormal Hemostatic Profiles and Gastric Necrosis in Canine Gastric Dilatation‐Volvulus." Veterinary Surgery 22, no. 2 (1993): 93–97. http://dx.doi.org/10.1111/j.1532-950x.1993.tb01680.x.

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50

Lee, Dosang, Kiyoung Sung, and Jun Hyun Lee. "Acute Gastric Necrosis Due to Gastric Outlet Obstruction Accompanied with Gastric Cancer and Trichophytobezoar." Journal of Gastric Cancer 11, no. 3 (2011): 185. http://dx.doi.org/10.5230/jgc.2011.11.3.185.

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