Relevant bibliographies by topics / Gastrin-releasing peptide

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Academic literature on the topic 'Gastrin-releasing peptide'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 March 2023

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Dissertations / Theses on the topic "Gastrin-releasing peptide"

1

Kallingal, George J. "The Role of Gastrin-releasing Peptide in Photic Entrainment." Kent State University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=kent1208972062.

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2

Schmidt, Anna Laura. "Alvos moleculares em meduloblastoma : um estudo in vitro." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2010. http://hdl.handle.net/10183/21418.

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Meduloblastoma é o tumor intracranial mais comum em crianças, provavelmente derivado de células precursoras da camada granular externa do cerebelo durante seu desenvolvimento. O tratamento padrão consiste em cirurgia, radioterapia e quimioterapia, que produzem graves sequelas nos pacientes e garantem uma sobrevida baixa, o que demonstra a necessidade de novas alternativas terapêuticas para a doença. Evidências demonstram que o receptor do peptídeo liberador de gastrina (GRPR) está superexpresso em diversos tumores humanos, assim como seu agonista (GRP) pode atuar como um fator de crescimento a
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3

Marrone, Bianca Fontana. "Perfil de expressão do receptor do peptídeo liberador de gastrina em materiais de biópsia de pacientes portadores de melanoma maligno." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/61720.

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Introdução: A incidência de melanoma maligno (MM) está aumentando mundialmente e o manejo de pacientes com doença avançada representa um difícil problema. Durante décadas, a quimioterapia foi o tratamento padrão no tratamento de pacientes com MM metastático. Entretanto, essa modalidade tem produzido resultados desapontadores. Recentemente, com os avanços no conhecimento sobre os eventos moleculares relacionados ao desenvolvimento do MM, novas drogas dirigidas a alvos moleculares de relevância na doença têm sido identificadas. O peptídeo liberador de gastrina (GRP) é um peptídeo neuroendócrino,
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4

Retzloff, Lauren Brooke Smith Charles J. "Design, synthesis, and evaluation of radiolabeled bombesin conjugates for the diagnosis of breast cancer." Diss., Columbia, Mo. : University of Missouri-Columbia, 2009. http://hdl.handle.net/10355/6740.

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The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on March 25, 2010). Vita. Thesis advisor: Charles J. Smith. "December 2009" Includes bibliographical references.
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5

Schweinsberg, Christian. "Novel 99mTc-labeled bombesin analogues with improved pharmacokinetics for targeting of gastrin-releasing-peptide receptor-positive tumors /." Zürich : ETH/PSI, 2008. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=17952.

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6

Farias, Caroline Brunetto de. "Interação funcional entre o receptor do peptídeo liberador de gastrina e a via de sinalização do AMP cíclico/proteína quinase A : um estudo in vitro e in vivo." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/17832.

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Muitas evidências demonstram que o peptídeo liberador de gastrina (GRP) é um fator de crescimento que afeta funções neuroendócrinas, incluindo proliferação e diferenciação celular, comportamento alimentar, formação de memória, respostas a estresses, desenvolvimento de neoplasias, desordens neurológicas e psiquiátricas. Porém, os eventos moleculares pelos quais isso ocorre ainda não são totalmente compreendidos. No presente estudo, nós avaliamos as interações entre o receptor do peptídeo liberador de gastrina (GRPR) e a via de sinalização celular da PKA, tanto na proliferação celular de gliobla
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7

Varasteh, Zohreh. "Bombesin Antagonists for Targeting Gastrin-Releasing Peptide Receptor-Positive Tumors : Design, Synthesis, Preclinical Evaluation and Optimization of Imaging Agents." Doctoral thesis, Uppsala universitet, Plattformen för preklinisk PET, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-232123.

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This thesis is focused on the development, preclinical evaluation, and optimization of radiotracers for the detection of gastrin-releasing peptide receptor (GRPR)-expressing tumors. The work is divided into three distinct parts: (1) the development of bombesin (BN) antagonist (RM26)-based imaging radiotracers for the detection of GRPR-expressing tumors using different positron emission tomography (PET) and single photon emission computed tomography (SPECT) radionuclides (68Ga, 18F and 111In), (2) the establishment of a method to monitor the ligand-G protein-coupled receptor (GPCR) interaction
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8

Clark, David Albert. "The effect of protease inhibitors and gastrin releasing peptide receptor antagonists on human small cell lung cancer growth." Diss., The University of Arizona, 1993. http://hdl.handle.net/10150/186391.

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The effect of the protease inhibitors BBI and aprotinin on the in vitro clonal growth of two human small cell lung cancer (SCLC) cell lines was investigated. Also, the effect of BBI on processing of proGRP and on mRNA levels for prohormone convertase 1 and 2 (PC1 and PC2) in SCLC cells was examined. Finally, the in vivo distribution of a BN/GRP receptor antagonist in mice was studied. In vitro growth of NCI-H345 and NCI-N592 SCLC cells was inhibited by the protease inhibitors BBI and aprotinin. When NCI-H345 cells were incubated with BBI and BN in the same plates, the addition of BN appeared t
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9

Murkar, Anthony. "Exploring Novel Treatment Approaches for Post-Traumatic Stress Disorder." Thesis, Université d'Ottawa / University of Ottawa, 2020. http://hdl.handle.net/10393/40040.

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Post-traumatic stress disorder is a disorder characterized by an inability to extinguish traumatic memories and heightened reactivity to emotional stimuli. Due to the heightened resistance of traumatic memories to extinction, treatment for PTSD has been challenging and is limited to behavioral therapies targeted at reducing responsivity to threatening stimuli. Currently there are no standard pharmacological interventions that are specific to PTSD; rather, drugs used appear to target symptoms of some of the co-morbid conditions, such as anxiety (e.g. benzodiazepines) or depression (antidepressa
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10

Giacchetti, Sylvie. "Facteurs de croissance et cancers du sein : étude du récepteur à la bombesine-GRP (Gastrin Releasing Peptide) in vitro." Paris 6, 1990. http://www.theses.fr/1990PA062036.

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