Relevant bibliographies by topics / Gastrointestinal gas

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Gastrointestinal gas'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 March 2023

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Journal articles on the topic "Gastrointestinal gas"

1

Sferra, Thomas J., and Leo A. Heitlinger. "GASTROINTESTINAL GAS FORMATION AND INFANTILE COLIC." Pediatric Clinics of North America 43, no. 2 (April 1996): 489–510. http://dx.doi.org/10.1016/s0031-3955(05)70417-x.

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Lauritano, E. C., A. Gasbarrini, and G. R. Corazza. "Introduction: Bacteria, gas and functional gastrointestinal disorders." Digestive and Liver Disease Supplements 3, no. 2 (July 2009): 25–26. http://dx.doi.org/10.1016/s1594-5804(09)00011-4.

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Malagelada, J.-R. "Sensation and gas dynamics in functional gastrointestinal disorders." Gut 51, Supplement 1 (July 1, 2002): i72—i75. http://dx.doi.org/10.1136/gut.51.suppl_1.i72.

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ter Laan, Mark, Erik Totte, Rob A. van Hulst, Klaas van der Linde, Wim van der Kamp, and Jean-Pierre E. Pierie. "Cerebral gas embolism due to upper gastrointestinal endoscopy." European Journal of Gastroenterology & Hepatology 21, no. 7 (July 2009): 833–35. http://dx.doi.org/10.1097/meg.0b013e328310aefc.

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Ochiai, T., K. Igri, Y. Kumagai, M. Iida, and S. Yamazaki. "Gastrointestinal: Massive portal venous gas and pneumatosis intestinalis." Journal of Gastroenterology and Hepatology 25, no. 6 (May 26, 2010): 1178. http://dx.doi.org/10.1111/j.1440-1746.2010.06361.x.

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KANEKO, Syoji, Yuki TSUSHIMA, Yutaka YOSHIDA, and Masanori KAYANO. "Gas chromatographic determination of dimethylpolysiloxane in gastrointestinal drugs." Bunseki kagaku 35, no. 3 (1986): 189–92. http://dx.doi.org/10.2116/bunsekikagaku.35.3_189.

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Rösch, T., B. Kapfer, U. Will, W. Baronius, M. Strobel, R. Lorenz, and K. Ulm. "New Techniques Accuracy of Endoscopic Ultrasonography in Upper Gastrointestinal Submucosal Lesions: a Prospective Multicenter Study." Scandinavian Journal of Gastroenterology 37, no. 7 (January 2002): 856–62. http://dx.doi.org/10.1080/gas.37.7.856.862.

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Groot, Evelien F. de. "Flatography: Detection of gastrointestinal diseases by faecal gas analysis." World Journal of Gastrointestinal Pharmacology and Therapeutics 6, no. 4 (2015): 111. http://dx.doi.org/10.4292/wjgpt.v6.i4.111.

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Kuo, S., W. Chang, C. Yu, and C. Hsieh. "Silent hepatic portal venous gas following upper gastrointestinal endoscopy." Endoscopy 41, S 02 (June 2009): E121—E122. http://dx.doi.org/10.1055/s-0029-1214657.

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Mehl, ML, B. Seguin, RW Norrdin, S. Geddes, and SJ Withrow. "Idiopathic pneumoperitoneum in a dog." Journal of the American Animal Hospital Association 37, no. 6 (November 1, 2001): 549–51. http://dx.doi.org/10.5326/15473317-37-6-549.

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A 13-year-old, neutered male standard poodle with tachypnea and abdominal distension was diagnosed with pneumoperitoneum. Pneumoperitoneum can be due to a perforated gastrointestinal tract, penetrating abdominal wounds, gas-producing bacterial peritonitis, or it can be iatrogenically introduced during surgery. Idiopathic pneumoperitoneum is a condition diagnosed in humans after exclusion of perforated gastrointestinal tract and other known causes of free intra-abdominal gas. This report suggests that dogs may suffer from a similar syndrome.
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Dissertations / Theses on the topic "Gastrointestinal gas"

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Dias, Bianca Paludeto. "Efeitos cardiorrespiratórios e motilidade gastrointestinal da infusão contínua de butorfanol em equinos anestesiados pelo isofluorano /." Araçatuba : [s.n.], 2010. http://hdl.handle.net/11449/88188.

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Orientador: Paulo Sérgio Patto dos Santos
Banca: Newton Nunes
Banca: Valéria Nobre Leal de Souza Oliva
Resumo: Objetivou-se avaliar os efeitos hemogasométricos, cardiorrespiratórios, qualidade da recuperação, alterações na motilidade gastrointestinal e cortisol plasmático de equinos submetidos à orquiectomia e anestesiados com isofluorano associado à infusão continua de butorfanol. Foram utilizados doze equinos adultos, distribuídos em dois grupos dois grupos, butorfanol (GB) e controle (GC). Os animais foram pré-medicados com xilazina, induzidos com quetamina associada ao midazolam e mantidos com isofluorano. Ao GB foi administrado butorfanol (0, 025 mg/kg IV) em bolus seguido de por infusão continua do fármaco (13µg/kg/h). As observações das variáveis foram feitas no momento basal e a cada 15 minutos por 75 minutos. Foi constatada redução na FC no M45 e M75 no GC e no M45 e M60 no GB comparados ao M0. A PAS, no GC, aumentou no M30 e M45 comparado ao M0. Já a PAD e PAM aumentaram em ambos os grupos quando comparadas ao valor basal. Houve redução da motilidade gastrointestinal por 60 minutos no GB. Foi observada elevação do cortisol plasmático em M60 e T30 em relação ao M0 em ambos os grupos. Os resultados obtidos permitiram concluir que a infusão contínua de butorfanol em equinos anestesiados pelo isofluorano promove discreta redução na motilidade gastrointestinal
Abstract: The aim of this study was to evaluate the blood gases, cardiorespiratory effects, quality of recovery, changes in intestinal motility and plasma cotisol in isoflurane-anesthetized horses associated to continuous rate infusion of butorphanol and submitted to orchiectomy. Twelve adult horses distributed into two groups (BG) and control group (CG). The animals were premedicated with xylazine, induced with ketamine combined with midazolam and maintained with isoflurane. In the BG, butorphanol (0.025 mg-1 kg-1 IV) was administered followed by continuous rate infusion (13µg-1kg-1hour) of the opiod. Observations of the variables were carried out at baseline and every 15 minutes during 75 minutes. Reduction in HR was observed in M45 and M75 in the CG and M45 and M60 in BG compared to M0. The SAP increased in the CG at M30 and M45 compared to M0 whereas DAP and MAP increased in both groups compared to baseline. PaO2 and RT decreased over time in both groups. There was a reduction of gastrointestinal motility by 60 minutes in GB. And plasma cortisol was higher in M60 and T30 than M0 in both groups. The results allow conclude that the continuous rate infusion of butorphanol in horses anesthetized with isoflurane a modest intestinal motility reduction
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Dias, Bianca Paludeto [UNESP]. "Efeitos cardiorrespiratórios e motilidade gastrointestinal da infusão contínua de butorfanol em equinos anestesiados pelo isofluorano." Universidade Estadual Paulista (UNESP), 2010. http://hdl.handle.net/11449/88188.

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Made available in DSpace on 2014-06-11T19:23:09Z (GMT). No. of bitstreams: 0 Previous issue date: 2010-08-30Bitstream added on 2014-06-13T20:10:41Z : No. of bitstreams: 1 dias_bp_me_araca.pdf: 394360 bytes, checksum: 6ce31366bd34a531d11d750f0d95e3ed (MD5)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Objetivou-se avaliar os efeitos hemogasométricos, cardiorrespiratórios, qualidade da recuperação, alterações na motilidade gastrointestinal e cortisol plasmático de equinos submetidos à orquiectomia e anestesiados com isofluorano associado à infusão continua de butorfanol. Foram utilizados doze equinos adultos, distribuídos em dois grupos dois grupos, butorfanol (GB) e controle (GC). Os animais foram pré-medicados com xilazina, induzidos com quetamina associada ao midazolam e mantidos com isofluorano. Ao GB foi administrado butorfanol (0, 025 mg/kg IV) em bolus seguido de por infusão continua do fármaco (13µg/kg/h). As observações das variáveis foram feitas no momento basal e a cada 15 minutos por 75 minutos. Foi constatada redução na FC no M45 e M75 no GC e no M45 e M60 no GB comparados ao M0. A PAS, no GC, aumentou no M30 e M45 comparado ao M0. Já a PAD e PAM aumentaram em ambos os grupos quando comparadas ao valor basal. Houve redução da motilidade gastrointestinal por 60 minutos no GB. Foi observada elevação do cortisol plasmático em M60 e T30 em relação ao M0 em ambos os grupos. Os resultados obtidos permitiram concluir que a infusão contínua de butorfanol em equinos anestesiados pelo isofluorano promove discreta redução na motilidade gastrointestinal
The aim of this study was to evaluate the blood gases, cardiorespiratory effects, quality of recovery, changes in intestinal motility and plasma cotisol in isoflurane-anesthetized horses associated to continuous rate infusion of butorphanol and submitted to orchiectomy. Twelve adult horses distributed into two groups (BG) and control group (CG). The animals were premedicated with xylazine, induced with ketamine combined with midazolam and maintained with isoflurane. In the BG, butorphanol (0.025 mg-1 kg-1 IV) was administered followed by continuous rate infusion (13µg-1kg-1hour) of the opiod. Observations of the variables were carried out at baseline and every 15 minutes during 75 minutes. Reduction in HR was observed in M45 and M75 in the CG and M45 and M60 in BG compared to M0. The SAP increased in the CG at M30 and M45 compared to M0 whereas DAP and MAP increased in both groups compared to baseline. PaO2 and RT decreased over time in both groups. There was a reduction of gastrointestinal motility by 60 minutes in GB. And plasma cortisol was higher in M60 and T30 than M0 in both groups. The results allow conclude that the continuous rate infusion of butorphanol in horses anesthetized with isoflurane a modest intestinal motility reduction
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Rodriguez, Frausto Heriberto. "Development and analytical validation of a gas chromatography-mass spectrometry method for the assessment of gastrointestinal permeability and intestinal absorptive capacity in dogs." [College Station, Tex. : Texas A&M University, 2008. http://hdl.handle.net/1969.1/ETD-TAMU-3257.

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Cedernaes, Jonathan. "Intestinal Gene Expression Profiling and Fatty Acid Responses to a High-fat Diet." Doctoral thesis, Uppsala universitet, Funktionell farmakologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-196207.

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The gastrointestinal tract (GIT) regulates nutrient uptake, secretes hormones and has a crucial gut flora and enteric nervous system. Of relevance for these functions are the G protein-coupled receptors (GPCRs) and the solute carriers (SLCs). The Adhesion GPCR subfamily is known to mediate neural development and immune system functioning, whereas SLCs transport e.g. amino acids, fatty acids (FAs) and drugs over membranes. We aimed to comprehensively characterize Adhesion GPCR and SLC gene expression along the rat GIT. Using qPCR we measured expression of 78 SLCs as well as all 30 Adhesion GPCRs in a twelve-segment GIT model. 21 of the Adhesion GPCRs had a widespread (≥5 segments) or ubiquitous (≥11 segments) expression. Restricted expression patterns were characteristic for most group VII members. Of the SLCs, we found the majority (56 %) of these transcripts to be expressed in all GIT segments. SLCs were predominantly found in the absorption-responsible gut regions. Both Adhesion GPCRs and SLCs were widely expressed in the rat GIT, suggesting important roles. The distribution of Adhesion GPCRs defines them as a potential pharmacological target. FAs constitute an important energy source and have been implicated in the worldwide obesity increase. FAs and their ratios – indices for activities of e.g. the desaturase enzymes SCD-1 (SCD-16, 16:1n-7/16:0), D6D (18:3n-6/18:2n-6) and D5D (20:4n-6/20:3n-6) – have been associated with e.g. overall mortality and BMI. We examined whether differences in FAs and their indices in five lipid fractions contributed to obesity susceptibility in rats fed a high fat diet (HFD), and the associations of desaturase indices between lipid fractions in animals on different diets. We found that on a HFD, obesity-prone (OP) rats had a higher SCD-16 index and a lower linoleic acid (LA) proportions in subcutaneous adipose tissue (SAT) than obesity-resistant rats. Desaturase indices were significantly correlated between many of the lipid fractions. The higher SCD-16 may indicate higher SCD-1 activity in SAT in OP rats, and combined with lower LA proportions may provide novel insights into HFD-induced obesity. The associations between desaturase indices show that plasma measurements can serve as proxies for some lipid fractions, but the correlations seem to be affected by diet and weight gain.
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Goh, Hong Eng. "A new structural summary of the MMPI-2 for evaluating personal injury claimants." University of Southern Queensland, Faculty of Sciences, 2006. http://eprints.usq.edu.au/archive/00001434/.

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The Minnesota Multiphasic Personality Inventory-2 (MMPI-2) is a popular measure of psychosocial functioning and psychopathology in the assessment of individuals in a variety of settings. However, the method of construction employed with the MMPI more than 60 years ago with psychiatric patients challenges the applicability of the scales for determining the psychosocial functioning of individuals from different settings. The restandardisation conducted in 1987 made no effort to eradicate the item overlap that was a result of the criterion keying method with contrasted groups. Although restandardized and updated with more contemporary language and content, the original psychiatric constructs were retained in order to maintain continuity with its predecessor. The aims of this investigation were to develop a new structure for the MMPI-2 constructed at the item-level, empirically derived and which specifically represents the dimensions that are relevant and appropriate in evaluating the psychosocial functioning of personal injury claimants. This task included comparisons with a comparable scale-level analysis and developing optimal scoring strategies where items in components and facets are allocated weightings based upon their strength of association. Study 1 was conducted using a sample of 2989 personal injury claimants assessed in Australia and the United States of America. The final sample of 3230, included 241 normal individuals, was utilized to develop a scale-level structure from 79 standard MMPI-2 scales and subscales. A nine-component solution consisting of General Maladjustment /Emotional Distress, Asocial Beliefs, Social Vulnerability, Somatic Complaints, Psychological Disturbance, Impulsive Expression, Antisocial Practices, Stereotypic Fears and Family Difficulties was derived using principal component analysis. However, intercorrelation between components in the structure signaled the need to develop a structure that would eradicate problems that were perpetuated by item overlap. The second study was conducted with a set of best practice procedures with the same clinical sample of 2989 personal injury claimants as Study 1. Forty-one components were derived through principal component analysis. Through the application of a set of criteria, a 35-component solution was retained. The pattern coefficients from the allocation of items to components determined the weightings to be applied to each item. Further analysis of the 35 components derived a substructure of 37 facets. The 35 components included only 442 of the 567 items, with the reliability coefficients of the first 25 components that ranged between .5 and .97, and the remaining 10 components that ranged from .29 to .49. The latter unreliable components were not included in the final Structural Summary, leaving 25 components (400 items) and their 33 facets for interpretation. Hence, in demonstrating the utility of the newly-derived structure, only 25 components and their 33 facets were interpreted. The 25 components were grouped conceptually into six domains. In the emotional domain were Psychological Distress (PsyDist), Anger, Fears, Psychotic Symptoms (PsyS), Paranoia (Par), Irritability (Irrit), Elation (Elat), Fear of the Dark (FD), and Financial Worry (FinWo). Somatic Complaints (SomC), Sexual Concerns (SexCon), and Gastrointestinal Problems (GasP) made up the measures in the physiological domain. In the behavioural domain were Cognitive Difficulties (CogDiff), Stimulus-Seeking (StimuS), Discipline (Dis), and Delinquency (Del) whilst the interpersonal domain was formed by Social Withdrawal (SoW), Negative Interpersonal Attitude (NIA), Timidity (Tim), Lie, Dissatisfaction with Self (DWS) and Family Relationship Difficulties (FReD). Alcoholism (Alco) was the only measure in the substance abuse domain, and the gender domain was comprised of Masculinity (Mas) and Femininity (Fem). The third study established preliminary normative means and standard deviations using a small opportunistic Australian university student sample (N = 219). No substantial gender differences were found but gender norms were maintained to facilitate comparisons with the traditional MMPI-2 approach. Comparisons of frequency of 'true' item response between the Australian university student sample and the U.S. restandardisation sample found relatively little differences and permitted evaluation of between sample differences on components and facets. The utility of the structure was demonstrated with the illustration of two clinical case examples, and a comparison was made with the standard MMPI-2 scales and subscales. The Structural Summary for the MMPI-2 demonstrated discriminative measures of psychosocial functioning that were a result of no item overlap, and the ability to attend to the different levels of intensity of self-report items because of differential weightings.
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Books on the topic "Gastrointestinal gas"

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What I need to know about gas. Bethesda, MD: Department of Health & Human Services, NIH, National Institute of Diabetes and Digestive and Kidney Diseases, National Digestive Diseases Information Clearinghouse, 2011.

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Lo que usted debe saber sobre los gases: What you need to know about gases. Bethesda, MD: U.S. Department of Health & Human Services, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases, National Digestive Diseases Information Clearinghouse, 2011.

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Parker, James N., and Philip M. Parker. The 2002 official patient's sourcebook on gas. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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Stewart, Melissa. Blasts of gas: The secrets of breathing, burping, and farting. New York: Marshall Cavendish Benchmark, 2009.

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Blasts of gas: The secrets of breathing, burping, and farting. New York: Marshall Cavendish Benchmark, 2009.

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ill, Chen Xi, ed. Mei ge ren dou hui "pu". Xinbei Shi: You fu wen hua shi ye gu fen you xian gong si, 2016.

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Peikin, Steven R. Gastrointestinal health: A self-help nutritional program to prevent, cure, or alleviate irritable bowel syndrome, ulcers, heartburn, gas, constipation & many other digestive disorders. New York: Quill, 2001.

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Gastrointestinal health: A self-help nutritional program to prevent, cure, or alleviate irritable bowel syndrome, ulcers, heartburn, gas, constipation, and many other digestive disorders. New York, NY: HarperCollins, 1991.

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Berkson, Lindsey. Healthy digestion the natural way: Preventing and healing heartburn, constipation, gas, diarrhea, inflammatory bowel and gallbladder diseases, ulcers, irritable bowel syndrome, food allergies and more. New York: Wiley, 2000.

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Zen yang sheng huo bu sheng qi. Taibei Shi: Zhong yang ri bao she, 1990.

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Book chapters on the topic "Gastrointestinal gas"

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Snape, William J. "Bloating and Gas." In Gastrointestinal Motility, 249–55. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4803-4_24.

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Urita, Yoshihisa, Toshiyasu Watanabe, Ikutaka Takemoto, Yosuke Sasaki, Tadashi Maeda, Manabu Matsumoto, Yoshiko Honda, Nagato Shimada, Hitoshi Nakajima, and Motonobu Sugimoto. "Intraluminal Gas and Gastrointestinal Diseases." In Gas Biology Research in Clinical Practice, 6–14. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000321936.

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Macfarlane, George T., and Glenn R. Gibson. "Carbohydrate Fermentation, Energy Transduction and Gas Metabolism in the Human Large Intestine." In Gastrointestinal Microbiology, 269–318. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-4111-0_9.

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Almansa, Cristina, Kenneth DeVault, and Lesley A. Houghton. "Gas and Bloating." In Functional and Motility Disorders of the Gastrointestinal Tract, 113–23. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1498-2_10.

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Naito, Yuji, Kazuhiko Uchiyama, Tomohisa Takagi, and Toshikazu Yoshikawa. "CO and Its Application to Gastrointestinal Disease." In Gas Biology Research in Clinical Practice, 35–42. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000321939.

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Ock, Chan Young, So Jung Han, Ki-Seok Choi, Eun-Hee Kim, Ju Hyun Kim, and Ki-Baik Hahm. "Hydrogen Sulfide in the Gastrointestinal Tract: Friend or Foe?" In Gas Biology Research in Clinical Practice, 65–72. Basel: KARGER, 2011. http://dx.doi.org/10.1159/000321942.

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Read, N. W. "Gastrointestinales Gas." In Gastroenterologische Pathophysiologie, 21–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71876-2_3.

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Vieira, Adriana, Ana Gramacho, Dora Rolo, Nádia Vital, Maria João Silva, and Henriqueta Louro. "Cellular and Molecular Mechanisms of Toxicity of Ingested Titanium Dioxide Nanomaterials." In Advances in Experimental Medicine and Biology, 225–57. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-88071-2_10.

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AbstractAn exponential increase in products containing titanium dioxide nanomaterials (TiO2), in agriculture, food and feed industry, lead to increased oral exposure to these nanomaterials (NMs). Thus, the gastrointestinal tract (GIT) emerges as a possible route of exposure that may drive systemic exposure, if the intestinal barrier is surpassed. NMs have been suggested to produce adverse outcomes, such as genotoxic effects, that are associated with increased risk of cancer, leading to a concern for public health. However, to date, the differences in the physicochemical characteristics of the NMs studied and other variables in the test systems have generated contradictory results in the literature. Processes like human digestion may change the NMs characteristics, inducing unexpected toxic effects in the intestine. Using TiO2 as case-study, this chapter provides a review of the works addressing the interactions of NMs with biological systems in the context of intestinal tract and digestion processes, at cellular and molecular level. The knowledge gaps identified suggest that the incorporation of a simulated digestion process for in vitro studies has the potential to improve the model for elucidating key events elicited by these NMs, advancing the nanosafety studies towards the development of an adverse outcome pathway for intestinal effects.
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Cave, Nick. "Gastrointestinal Gas." In Canine and Feline Gastroenterology, 124–28. Elsevier, 2013. http://dx.doi.org/10.1016/b978-1-4160-3661-6.00015-8.

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Ammoury, Rana, and Sandeep Gupta. "Colic and Gastrointestinal Gas." In Pediatric Gastrointestinal and Liver Disease, 119–26. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4377-0774-8.10011-9.

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Conference papers on the topic "Gastrointestinal gas"

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Huang, Hen-Wei, Claas Ehmke, Christoph Steiger, Ian Ballinger, Miguel Jimenez, Nhi Phan, Haoying Sun, et al. "In Situ Detection of Gastrointestinal Inflammatory Biomarkers Using Electrochemical Gas Sensors." In 2022 44th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2022. http://dx.doi.org/10.1109/embc48229.2022.9871468.

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Huang, Hen-Wei, David de Gruijl, Philip Fritz, Abhijay Kemkar, Ian Ballinger, George Selsing, Peter Ray Chai, and Giovanni Traverso. "Encapsulation of Gas Sensors to Operate in the Gastrointestinal Tract for Continuous Monitoring." In 2022 IEEE Sensors. IEEE, 2022. http://dx.doi.org/10.1109/sensors52175.2022.9967279.

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Rotbart, A., K. Kalantar-zadeh, JZ Ou, CK Yao, JG Muir, and PR Gibson. "PWE-005 New in vitro human faecal fermentation system for diagnosis and treatment of gastrointestinal disorders using continuous intestinal gas profiling." In British Society of Gastroenterology, Annual General Meeting, 19–22 June 2017, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2017. http://dx.doi.org/10.1136/gutjnl-2017-314472.250.

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Adjei, Prince E., Zenebe M. Lonseko, and Nini Rao. "GAN-Based Synthetic Gastrointestinal Image Generation." In 2020 17th International Computer Conference on Wavelet Active Media Technology and Information Processing (ICCWAMTIP). IEEE, 2020. http://dx.doi.org/10.1109/iccwamtip51612.2020.9317341.

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Sams, Ataher, and Homaira Huda Shomee. "GAN-Based Realistic Gastrointestinal Polyp Image Synthesis." In 2022 IEEE 19th International Symposium on Biomedical Imaging (ISBI). IEEE, 2022. http://dx.doi.org/10.1109/isbi52829.2022.9761447.

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Sams, Ataher, and Homaira Huda Shomee. "GAN-Based Realistic Gastrointestinal Polyp Image Synthesis." In 2022 IEEE 19th International Symposium on Biomedical Imaging (ISBI). IEEE, 2022. http://dx.doi.org/10.1109/isbi52829.2022.9761447.

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Thilakasiri, Pathum, Tracy Nero, Michael W. Parker, Matthias Ernst, and Ashwini L. Chand. "Abstract 4962: Repurposingbazedoxifeneto suppress gastrointestinal cancer growth." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4962.

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Thilakasiri, Pathum, Tracy Nero, Michael W. Parker, Matthias Ernst, and Ashwini L. Chand. "Abstract 4962: Repurposingbazedoxifeneto suppress gastrointestinal cancer growth." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4962.

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Mehra, Neha, Sandhya Rani, Promila Sharma, and Pratibha Joshi. "Ergonomic Assessment of Activities of Front Office Worker in Selected Hospitality Units and Record Related Health." In Applied Human Factors and Ergonomics Conference. AHFE International, 2019. http://dx.doi.org/10.54941/ahfe100150.

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Abstract:
Often when we imagine the kind of workers who get workplace injuries, we think of those who need to exert a lot of physical energy on the job. It is true that employees in these work environments may be at greater risk for injury, but office workers are also at risk. Front Office work is rapidly changing, as new developments in computer technology come along which can make our jobs easier, but which also can present new problems for both management and employees. There is enough information contained here to allow a single employee to set up their own workstation to suit the way they work. This paper provides with the information and tools necessary to analyze office jobs related health problems. A descriptive research enumerates the existing conditions in which the researcher has no control over the variables and can report only actual conditions. The interview schedule was found to be an appropriate tool, which would adequately gather information pertaining to research work. Considering the objective of the study, research was carried out at Pancham Hotel of Bareilly. A sample size is a definite plan for obtaining a sample from a given population. Total sample sizes of 30 were selected. Many office injuries are caused by the repetitive tasks that put strain on our muscles and joints. There are several health related problems faced by office workers which affect their daily working schedule such as chronic back pain, gastric bronchial, asthma, diabetes, depression, arthritis, anaemia and headache. Sixty percent respondents quite seldom suffered from digestion difficulties whereas 12 percent respondents quite often and only 16 percent respondent almost always suffered from digestion difficulties. Several research studies reveals that gastrointestinal complaints of gastric upset, gas, constipation, poor eating etc. are strongly correlated with front office work.
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Schmidt, M., and O. Hülsmann. "Carmelite spirit for the treatment of stress-related gastrointestinal symptoms." In GA – 69th Annual Meeting 2021, Virtual conference. Georg Thieme Verlag, 2021. http://dx.doi.org/10.1055/s-0041-1736920.

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Reports on the topic "Gastrointestinal gas"

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Newman-Toker, David E., Susan M. Peterson, Shervin Badihian, Ahmed Hassoon, Najlla Nassery, Donna Parizadeh, Lisa M. Wilson, et al. Diagnostic Errors in the Emergency Department: A Systematic Review. Agency for Healthcare Research and Quality (AHRQ), December 2022. http://dx.doi.org/10.23970/ahrqepccer258.

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Objectives. Diagnostic errors are a known patient safety concern across all clinical settings, including the emergency department (ED). We conducted a systematic review to determine the most frequent diseases and clinical presentations associated with diagnostic errors (and resulting harms) in the ED, measure error and harm frequency, as well as assess causal factors. Methods. We searched PubMed®, Cumulative Index to Nursing and Allied Health Literature (CINAHL®), and Embase® from January 2000 through September 2021. We included research studies and targeted grey literature reporting diagnostic errors or misdiagnosis-related harms in EDs in the United States or other developed countries with ED care deemed comparable by a technical expert panel. We applied standard definitions for diagnostic errors, misdiagnosis-related harms (adverse events), and serious harms (permanent disability or death). Preventability was determined by original study authors or differences in harms across groups. Two reviewers independently screened search results for eligibility; serially extracted data regarding common diseases, error/harm rates, and causes/risk factors; and independently assessed risk of bias of included studies. We synthesized results for each question and extrapolated U.S. estimates. We present 95 percent confidence intervals (CIs) or plausible range (PR) bounds, as appropriate. Results. We identified 19,127 citations and included 279 studies. The top 15 clinical conditions associated with serious misdiagnosis-related harms (accounting for 68% [95% CI 66 to 71] of serious harms) were (1) stroke, (2) myocardial infarction, (3) aortic aneurysm and dissection, (4) spinal cord compression and injury, (5) venous thromboembolism, (6/7 – tie) meningitis and encephalitis, (6/7 – tie) sepsis, (8) lung cancer, (9) traumatic brain injury and traumatic intracranial hemorrhage, (10) arterial thromboembolism, (11) spinal and intracranial abscess, (12) cardiac arrhythmia, (13) pneumonia, (14) gastrointestinal perforation and rupture, and (15) intestinal obstruction. Average disease-specific error rates ranged from 1.5 percent (myocardial infarction) to 56 percent (spinal abscess), with additional variation by clinical presentation (e.g., missed stroke average 17%, but 4% for weakness and 40% for dizziness/vertigo). There was also wide, superimposed variation by hospital (e.g., missed myocardial infarction 0% to 29% across hospitals within a single study). An estimated 5.7 percent (95% CI 4.4 to 7.1) of all ED visits had at least one diagnostic error. Estimated preventable adverse event rates were as follows: any harm severity (2.0%, 95% CI 1.0 to 3.6), any serious harms (0.3%, PR 0.1 to 0.7), and deaths (0.2%, PR 0.1 to 0.4). While most disease-specific error rates derived from mainly U.S.-based studies, overall error and harm rates were derived from three prospective studies conducted outside the United States (in Canada, Spain, and Switzerland, with combined n=1,758). If overall rates are generalizable to all U.S. ED visits (130 million, 95% CI 116 to 144), this would translate to 7.4 million (PR 5.1 to 10.2) ED diagnostic errors annually; 2.6 million (PR 1.1 to 5.2) diagnostic adverse events with preventable harms; and 371,000 (PR 142,000 to 909,000) serious misdiagnosis-related harms, including more than 100,000 permanent, high-severity disabilities and 250,000 deaths. Although errors were often multifactorial, 89 percent (95% CI 88 to 90) of diagnostic error malpractice claims involved failures of clinical decision-making or judgment, regardless of the underlying disease present. Key process failures were errors in diagnostic assessment, test ordering, and test interpretation. Most often these were attributed to inadequate knowledge, skills, or reasoning, particularly in “atypical” or otherwise subtle case presentations. Limitations included use of malpractice claims and incident reports for distribution of diseases leading to serious harms, reliance on a small number of non-U.S. studies for overall (disease-agnostic) diagnostic error and harm rates, and methodologic variability across studies in measuring disease-specific rates, determining preventability, and assessing causal factors. Conclusions. Although estimated ED error rates are low (and comparable to those found in other clinical settings), the number of patients potentially impacted is large. Not all diagnostic errors or harms are preventable, but wide variability in diagnostic error rates across diseases, symptoms, and hospitals suggests improvement is possible. With 130 million U.S. ED visits, estimated rates for diagnostic error (5.7%), misdiagnosis-related harms (2.0%), and serious misdiagnosis-related harms (0.3%) could translate to more than 7 million errors, 2.5 million harms, and 350,000 patients suffering potentially preventable permanent disability or death. Over two-thirds of serious harms are attributable to just 15 diseases and linked to cognitive errors, particularly in cases with “atypical” manifestations. Scalable solutions to enhance bedside diagnostic processes are needed, and these should target the most commonly misdiagnosed clinical presentations of key diseases causing serious harms. New studies should confirm overall rates are representative of current U.S.-based ED practice and focus on identified evidence gaps (errors among common diseases with lower-severity harms, pediatric ED errors and harms, dynamic systems factors such as overcrowding, and false positives). Policy changes to consider based on this review include: (1) standardizing measurement and research results reporting to maximize comparability of measures of diagnostic error and misdiagnosis-related harms; (2) creating a National Diagnostic Performance Dashboard to track performance; and (3) using multiple policy levers (e.g., research funding, public accountability, payment reforms) to facilitate the rapid development and deployment of solutions to address this critically important patient safety concern.
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