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Journal articles on the topic 'Gastrointestinal motiliy'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Poli, E., R. Raddino, G. Pela', M. Gargano, and P. Ghirardi. "Effects of phosphorated carbohydrates on gastrointestinal motiliy “In vitro”." Pharmacological Research Communications 20 (September 1988): 312. http://dx.doi.org/10.1016/s0031-6989(88)80442-9.

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2

Sakahara, Satoshi, Zuoyun Xie, Kanako Koike, et al. "Physiological characteristics of gastric contractions and circadian gastric motility in the free-moving conscious house musk shrew (Suncus murinus)." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, no. 4 (2010): R1106—R1113. http://dx.doi.org/10.1152/ajpregu.00278.2010.

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Although many studies have demonstrated the physiological action of motilin on the migrating motor complex, the precise mechanisms remain obscure. To obtain new insights into the mechanisms, we focused on the house musk shrew ( Suncus murinus, suncus used as a laboratory name) as a small model animal for in vivo motilin study, and we studied the physiological characteristics of suncus gastrointestinal motility. Strain gauge transducers were implanted on the serosa of the gastric body and duodenum, and we recorded gastrointestinal contractions in the free-moving conscious suncus and also examin

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3

Boivin, M., M. Riberdy, M. C. Raymond, L. Trudel, and P. Poitras. "Motilin and the postprandial motility of the antrum." Canadian Journal of Physiology and Pharmacology 70, no. 11 (1992): 1491–95. http://dx.doi.org/10.1139/y92-211.

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This study was designed to establish whether the rise in plasma motilin observed after a meal in humans can influence the postprandial motor activity of the antrum. Antroduodenal postprandial motility profiles and indices obtained from 5 controls and 5 subjects infused with exogenous synthetic motilin (0.1 μg∙kg−1) or with the motilin receptor agonist erythromycin lactobionate (200 mg) were compared. Motilin infusion increased plasma motilin concentrations about 5 times above the physiological range but failed to modify the normal postprandial contractile response. On the other hand, in 4 of t

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4

Gordon, Dan. "Erythromycin-responsive motilin receptor regulates upper gastrointestinal motility." Gastroenterology 117, no. 3 (1999): 524. http://dx.doi.org/10.1016/s0016-5085(99)70440-3.

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5

Malfertheiner, P., M. G. Sarr, M. P. Spencer, and E. P. DiMagno. "Effect of duodenectomy on interdigestive pancreatic secretion, gastrointestinal motility, and hormones in dogs." American Journal of Physiology-Gastrointestinal and Liver Physiology 257, no. 3 (1989): G415—G422. http://dx.doi.org/10.1152/ajpgi.1989.257.3.g415.

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We tested the hypothesis that the duodenum is necessary to coordinate interdigestive pancreatic trypsin secretion with gastrointestinal motility and determined whether duodenectomy altered interdigestive cycles of plasma motilin and pancreatic polypeptide and their relationship to trypsin secretion and motility. Consequently, in normal and duodenectomized dogs, we measured trypsin secretion, gastrointestinal motility, and plasma concentrations of motilin and pancreatic polypeptide during the interdigestive period. After duodenectomy, peaks of trypsin secretion continued to cycle at normal inte

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6

Ohno, Tetsuro, Erito Mochiki, and Hiroyuki Kuwano. "The Roles of Motilin and Ghrelin in Gastrointestinal Motility." International Journal of Peptides 2010 (February 3, 2010): 1–6. http://dx.doi.org/10.1155/2010/820794.

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In structure, ghrelin resembles motilin. The two peptides are considered to be members of the motilin-ghrelin peptide family. Motilin is considered to be an endocrine regulator of the interdigestive migrating contractions, the fasted motor pattern in the gastrointestinal (GI) tract. It has been reported that ghrelin stimulates GI motility. The gastrokinetic capacity of ghrelin has been well documented in the rodent. However, there have been few positive reports of the gastrokinetic capacity of ghrelin in dogs. Some reports with human subjects have shown that an i.v. ghrelin injection accelerat

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7

Kamerling, I. M. C., A. D. Van Haarst, J. Burggraaf, et al. "Dose-related effects of motilin on proximal gastrointestinal motility." Alimentary Pharmacology & Therapeutics 16, no. 1 (2002): 129–35. http://dx.doi.org/10.1046/j.1365-2036.2002.01142.x.

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8

Luiking, Y. C., A. C. van der Reijden, T. L. Peeters, G. P. van Berge Henegouwen, and L. M. A. Akkermans. "Motilin bolus infusion cannot repeatedly induce interdigestive gastrointestinal motility." European Journal of Gastroenterology & Hepatology 10, no. 12 (1998): A84. http://dx.doi.org/10.1097/00042737-199812000-00265.

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9

Kawamura, Tomoe, Bunzo Matsuura, Teruki Miyake, Masanori Abe, Yoshiou Ikeda, and Yoichi Hiasa. "Effects of Motilin Receptor Agonists and Ghrelin in Human motilin receptor Transgenic Mice." International Journal of Molecular Sciences 20, no. 7 (2019): 1521. http://dx.doi.org/10.3390/ijms20071521.

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Gastrointestinal motility is regulated by neural factors and humoral factors. Both motilin and ghrelin improve gastrointestinal motility, but many issues remain unclear. We prepared human motilin receptor transgenic (Tg) mice and performed experiments evaluating the effects of motilin, erythromycin (EM), and ghrelin. EM and ghrelin promoted gastric emptying (GE) when administered either peripherally or centrally to Tg mice. Atropine (a muscarinic receptor antagonist) counteracted GE induced by centrally administered EM, but not that induced by peripherally administered EM. The administration o

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10

Gold-Smith, Fuchsia D., Ruma G. Singh, and Maxim S. Petrov. "Elevated Circulating Levels of Motilin are Associated with Diabetes in Individuals after Acute Pancreatitis." Experimental and Clinical Endocrinology & Diabetes 128, no. 01 (2019): 43–51. http://dx.doi.org/10.1055/a-0859-7168.

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Abstract Aim The study aimed to investigate the associations between glycaemic control after acute pancreatitis and gastrointestinal motility, using plasma motilin concentration and gastroparesis cardinal symptom index score as proxies. Methods This cross-sectional study recruited a total of 93 individuals after acute pancreatitis. Gastroparesis cardinal index scores, demographic and anthropometric factors, as well as pancreatitis-related factors were analysed. Fasting venous blood was collected to measure motilin, glycated haemoglobin, and fasting blood glucose. Linear regression analyses wer

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11

Chung, S. A., G. R. Greenberg, and N. E. Diamant. "Relationship of postprandial motilin, gastrin, and pancreatic polypeptide release to intestinal motility during vagal interruption." Canadian Journal of Physiology and Pharmacology 70, no. 8 (1992): 1148–53. http://dx.doi.org/10.1139/y92-159.

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Experiments were performed to determine how postprandial motilin, gastrin, and pancreatic polypeptide plasma concentrations measured during vagal blockade relate to coincident small intestinal motility patterns. Feeding produced a postprandial pattern of intestinal motility coincident with a sustained increase in gastrin and pancreatic polypeptide and a decline in motilin plasma concentrations. Vagal blockade replaced the fed pattern with one similar to migrating motor complex (MMC) activity. Highest motilin plasma concentrations were observed during phase III of this MMC-like activity, as occ

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12

Zhang, Sanyin, Peng Yang, Xixiong Li, et al. "Comparative Researches of Semen Arecae and Charred Semen Arecae on Gastrointestinal Motility, Motilin, Substance P, and CCK in Chronically Stressed Rats." Evidence-Based Complementary and Alternative Medicine 2017 (2017): 1–8. http://dx.doi.org/10.1155/2017/1273561.

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Aims. To compare the effects of Semen Arecae (SA) and Charred Semen Arecae (CSA) on gastrointestinal motility, motilin, substance P (SP), and cholecystokinin (CCK) in chronically stressed rats. Methods. Rats were randomly divided into control group and stress group. Rats in stress group were randomly exposed to a variety of unpredictable stimulations for 21 days. Then, the rats were treated orally with distilled water, SA, CSA, and mosapride for 7 days. Gastric residue rate and intestinal propulsion rate were evaluated. Serum levels of motilin and SP were measured by enzyme-linked immunosorben

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13

Apu, Auvijit Saha, Anupom Mondal, Takio Kitazawa, Shota Takemi, Takafumi Sakai, and Ichiro Sakata. "Molecular cloning of motilin and mechanism of motilin-induced gastrointestinal motility in Japanese quail." General and Comparative Endocrinology 233 (July 2016): 53–62. http://dx.doi.org/10.1016/j.ygcen.2016.05.017.

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14

Muller, E. L., P. A. Grace, R. L. Conter, J. J. Roslyn, and H. A. Pitt. "Influence of motilin and cholecystokinin on sphincter of Oddi and duodenal mobility." American Journal of Physiology-Gastrointestinal and Liver Physiology 253, no. 5 (1987): G679—G683. http://dx.doi.org/10.1152/ajpgi.1987.253.5.g679.

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The sphincter of Oddi and the duodenum exhibit cyclical activity in phase with the migrating myoelectric complex. Both motilin and cholecystokinin have been shown to modulate gastrointestinal and sphincter of Oddi motility. However, previous studies have not monitored the effects of these hormones on simultaneously recorded sphincter of Oddi and duodenum pressures. The present investigation was undertaken, therefore, to determine the influence of both motilin and cholecystokinin on simultaneously recorded sphincter of Oddi and duodenal motility. In seven anesthetized prairie dogs, a triple-lum

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15

Nakamura, Hiroyuki, Tadashi Asano, Koichi Haruta, and Keisuke Takeda. "Gastrointestinal motor inhibition by exogenous human, salmon, and eel calcitonin in conscious dogs." Canadian Journal of Physiology and Pharmacology 73, no. 1 (1995): 43–49. http://dx.doi.org/10.1139/y95-006.

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Effects of synthetic eel (E-), salmon (S-), and human (H-) calcitonin (CT) on gastrointestinal motility were studied in conscious beagle dogs, which had been implanted with strain gauge force transducers. Intramuscular administration of E-, S-, or H-CT interrupted gastric migrating motor complexes, digestive pattern, and gastric emptying. The order of potency was E-CT = S-CT > H-CT. Motor inhibition induced by CT occurred independently of plasma immunoreactive motilin levels or hypocalcemia. In addition, E-CT and S-CT induced vomiting without a retrograde giant contraction (RGC) during the

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16

Ogawa, Atsushi, Erito Mochiki, Mitsuhiro Yanai, et al. "Interdigestive migrating contractions are coregulated by ghrelin and motilin in conscious dogs." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 302, no. 2 (2012): R233—R241. http://dx.doi.org/10.1152/ajpregu.00078.2011.

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During fasting, gastrointestinal (GI) motility is characterized by cyclical motor contractions. These contractions have been referred to as interdigestive migrating contractions (IMCs). In dogs and humans, IMCs are known to be regulated by motilin. However, in rats and mice, IMCs are regulated by ghrelin. It is not clear how these peptides influence each other in vivo. The aim of the present study was to investigate the relationship between ghrelin and motilin in conscious dogs. Twenty healthy beagles were used in this study. Force transducers were implanted in the stomach, duodenum, and jejun

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17

Koželuhová, Jana, Karel Balihar, and Lucie Zdrhová. "Diagnosis of gastrointestinal motility disorders." Vnitřní lékařství 64, no. 6 (2018): 611–20. http://dx.doi.org/10.36290/vnl.2018.085.

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18

Oono, Tetsuro. "Gastrointestinal Motility." Kitakanto Medical Journal 63, no. 1 (2013): 93–94. http://dx.doi.org/10.2974/kmj.63.93.

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19

Bharucha, Adil E. "Gastrointestinal motility." Gastroenterology 120, no. 4 (2001): 1056. http://dx.doi.org/10.1016/s0016-5085(01)83920-2.

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20

Weisbrodt, Norman W. "Gastrointestinal motility." Gastroenterology 89, no. 6 (1985): 1445. http://dx.doi.org/10.1016/0016-5085(85)90680-8.

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21

Layer, P., A. T. Chan, V. L. Go, and E. P. DiMagno. "Human pancreatic secretion during phase II antral motility of the interdigestive cycle." American Journal of Physiology-Gastrointestinal and Liver Physiology 254, no. 2 (1988): G249—G253. http://dx.doi.org/10.1152/ajpgi.1988.254.2.g249.

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We determined if changes in the irregular motor activity of phase II, the dominant motility phase in awake fasting humans, are associated with fluctuations in pancreatic secretion by intubating the upper gastrointestinal tract of 15 healthy humans and recording antral and duodenal motility and obtaining duodenal samples for one or two interdigestive motility cycles. Antral phase II activity was graded as having low, intermediate, or high frequency of contractions and related to duodenal trypsin output and plasma concentrations of motilin and human pancreatic polypeptide (HPP), a marker of vaga

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22

Tack, J., B. Coulie, A. Wilmer, T. Peeters, and J. Janssens. "Actions of the 5-hydroxytryptamine 1 receptor agonist sumatriptan on interdigestive gastrointestinal motility in man." Gut 42, no. 1 (1998): 36–41. http://dx.doi.org/10.1136/gut.42.1.36.

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Background—Pharmacological studies of the enteric nervous system have shown the presence of several subtypes of 5-hydroxytryptamine (5HT) receptor, which might be involved in control of the migrating motor complex.Aims—To study the effect of sumatriptan, an agonist of enteric neuronal 5HT1P receptors, on interdigestive motility in man.Subjects and methods—In 12 healthy subjects, interdigestive motility was recorded manometrically in the upper gastrointestinal tract. In seven subjects blood samples were drawn every 15 minutes for radioimmunoassay of motilin and somatostatin. After two phase 3s

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23

Rodriguez-Sinovas, A., M. Jimenez, P. De Clercq, T. L. Peeters, and P. Vergara. "Rhythmic oscillating complexes in gastrointestinal tract of chickens: a role for motilin." American Journal of Physiology-Gastrointestinal and Liver Physiology 272, no. 4 (1997): G916—G922. http://dx.doi.org/10.1152/ajpgi.1997.272.4.g916.

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Rhythmic oscillating complex (ROC) is a highly organized gastrointestinal motility pattern recently described in fasted avian species. ROCs show several high-speed aborad-propagated contractions that progressively change into others of orad direction. In addition, chickens show migrating motor complexes (MMC) in both fed and fasting states. Recently, motilin was isolated and characterized from chicken small intestine. Accordingly, the aim of this study was to learn whether chicken motilin might be involved in either ROC or MMC induction. Electromyographic recordings were obtained from differen

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24

Zhang, Huijuan, Jing Wang, Yingli Liu, and Baoguo Sun. "Glutinous rice amylopectin can adjust the plasma gut-regulated peptide levels in rhubarb-induced spleen deficiency rats." Food & Function 7, no. 2 (2016): 938–42. http://dx.doi.org/10.1039/c5fo00859j.

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The pharmacological effects of glutinous rice (GR) and GR amylopectin (GRA) on the gastrointestine were investigated in rhubarb-induced spleen deficiency rats by determining the levels of gastrointestinal hormones such as the peptides serum gastrin, amylase motilin, and somatostatin.

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25

Zhang, Weiwei, Xuelian Zhang, Yanyan Zhang, et al. "Analysis of Changes of Intestinal Flora in Elderly Patients with Alzheimer’s Disease and Liver Cancer and Its Correlation with Abnormal Gastrointestinal Motility." Journal of Oncology 2021 (August 13, 2021): 1–5. http://dx.doi.org/10.1155/2021/7517379.

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Objective. To investigate the changes of intestinal flora in elderly patients with Alzheimer’s disease and liver cancer and its correlation with abnormal gastrointestinal motility. Methods. From January 2018 to December 2020, 102 elderly patients with Alzheimer’s disease and liver cancer were selected as the observation group. Eighty-nine healthy patients during the same period were selected as the control group. The two groups of intestinal flora (intestinal microbial diversity) were detected by real-time fluorescent quantitative PCR (RT-qPCR) and high-throughput sequencing. The two groups of

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26

Cai, Guang-Xian. "Simotang enhances gastrointestinal motility, motilin and cholecystokinin expression in chronically stressed mice." World Journal of Gastroenterology 17, no. 12 (2011): 1594. http://dx.doi.org/10.3748/wjg.v17.i12.1594.

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27

Zhang, Teng-Yue, Peng Wang, Qing Wang, et al. "Saussurea Lappa Modulates Gastrointestinal Motility, Motilin and Cholecystokinin Expression in Ulcer Rats." Journal of Gastroenterology and Hepatology Research 5, no. 2 (2016): 2021–27. http://dx.doi.org/10.17554/j.issn.2224-3992.2016.05.625.

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28

Hirata, Tadatoshi, Toshihiw Nagai, Masaki Fujimura, Carl J. Pfeiffer, Haruaki Yajima, and Takayoshi Tobe. "Interactions of Motilin and Neurotensin on Feline Gastrointestinal Motility and Lymph Flow." Digestive Surgery 2, no. 4 (1985): 225–30. http://dx.doi.org/10.1159/000171710.

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29

Nakamura, Hiroyuki, Takeshi Iwai, Tadashi Asano, et al. "Effects of motilin receptor agonists on gastrointestinal motility and hemodynamics in dogs." Japanese Journal of Pharmacology 71 (1996): 109. http://dx.doi.org/10.1016/s0021-5198(19)36676-4.

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30

Song, C. W., C. D. Kim, K. Y. Lee, T. M. Chang, and W. Y. Chey. "Cisapride and renzapride stimulate plasma motilin release and gastrointestinal motility in dogs." Gastroenterology 103, no. 4 (1992): 1400. http://dx.doi.org/10.1016/0016-5085(92)91652-k.

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31

Shi, Yanan, Yingsong Zheng, Jingming Xu, et al. "Gastrointestinal Motility and Gut Hormone Secretion in response to Shenhuang Plaster in a Postoperative Ileus Rat Model." Evidence-Based Complementary and Alternative Medicine 2021 (June 1, 2021): 1–10. http://dx.doi.org/10.1155/2021/8859579.

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Postoperative ileus (POI), a gastrointestinal function disorder, is a complication that arises from surgery. Shenhuang plaster (SHP) application to the Shenque acupoint (CV8) to promote the recovery of gastrointestinal function has achieved definite curative effects in clinical settings; however, the underlying pharmacological mechanism remains unknown. In this study, we evaluated the effects of SHP using a Sprague Dawley rat POI model. Then, gastrointestinal transit in different rat groups was evaluated by the movement of fluorescein-labelled dextran. Ghrelin, obestatin, motilin (MTL), and va

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32

&NA;. "Lower gastrointestinal motility." Nuclear Medicine Communications 15, no. 1 (1994): 1–3. http://dx.doi.org/10.1097/00006231-199401000-00001.

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33

Haghiashtiani, Ghazaleh, and Michael C. McAlpine. "Sensing gastrointestinal motility." Nature Biomedical Engineering 1, no. 10 (2017): 775–76. http://dx.doi.org/10.1038/s41551-017-0146-1.

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34

Vantrappen, G., J. Janssens, G. Coremans, and R. Jian. "Gastrointestinal motility disorders." Digestive Diseases and Sciences 31, S9 (1986): 5–25. http://dx.doi.org/10.1007/bf01295987.

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35

Smout, André J. P. M., and Marco W. Mundt. "Gastrointestinal motility testing." Best Practice & Research Clinical Gastroenterology 23, no. 3 (2009): 287–98. http://dx.doi.org/10.1016/j.bpg.2009.04.006.

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36

Inatomi, Nobuhiro, Hiroshi Satoh, Yoshitaka Haki, Naoto Hashimoto, Zen Itoh, and Satoshi Omura. "EM-523, an erythromycin derivative, induces motilin-like gastrointestinal motility in conscious dogs." Japanese Journal of Pharmacology 46 (1988): 70. http://dx.doi.org/10.1016/s0021-5198(19)57055-x.

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37

Kitazawa, Takio, Shunji Ichikawa, Toshihide Yokoyama, Akio Ishii, and Katsuichi Shuto. "Stimulating action of KW-5139 (Leu13-motilin) on gastrointestinal motility in the rabbit." British Journal of Pharmacology 111, no. 1 (1994): 288–94. http://dx.doi.org/10.1111/j.1476-5381.1994.tb14058.x.

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38

Iwai, Takeshi, Hiroyuki Nakamura, Hisanori Takanashi, et al. "Hypotensive mechanism of [Leu13]motilin in dogs in vivo and in vitro." Canadian Journal of Physiology and Pharmacology 76, no. 12 (1998): 1103–9. http://dx.doi.org/10.1139/y98-138.

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The effects of [Leu13]motilin were examined in vivo after its intravenous administration into anesthetized dogs and in vitro with isolated preparations of canine mesenteric artery. [Leu13]Motilin (0.1-10 nmol·kg-1, i.v.) induced both strong and clustered phasic contractions in the gastric antrum and duodenum. At doses of over 1 nmol·kg-1, [Leu13]motilin also produced transient decreases in arterial blood pressure, left ventricular pressure, maximum rate of rise of left ventricular pressure, and total peripheral resistance, and an increase in aortic blood flow and heart rate. A selective motili

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39

Kusano, Motoyasu, Hiroko Hosaka, Akiyo Kawada, et al. "Gastrointestinal Motility and Functional Gastrointestinal Diseases." Current Pharmaceutical Design 20, no. 16 (2014): 2775–82. http://dx.doi.org/10.2174/13816128113199990572.

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40

Boivin, M., L. R. Pinelo, S. St-Pierre, and P. Poitras. "Neural mediation of the motilin motor effect on the human antrum." American Journal of Physiology-Gastrointestinal and Liver Physiology 272, no. 1 (1997): G71—G76. http://dx.doi.org/10.1152/ajpgi.1997.272.1.g71.

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To elucidate the mode of action of motilin on the stimulation of human gastrointestinal motility, we studied the effect of exogenous motilin during muscarinic or serotoninergic pharmacological blockade. Manometric recording of the interdigestive antroduodenal motility was carried out in 27 healthy volunteers until the appearance of a spontaneous antral phase III. The tested blocker was then administered intravenously and was followed 30 min later by a 10-min infusion of synthetic human motilin (50 ng/kg). Motilin administered on a background of saline induced a premature phase III migrating fr

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Mu, Dao-zhou, Mei Xue, Jian-jun Xu, et al. "Antidepression and Prokinetic Effects of Paeoniflorin on Rats in the Forced Swimming Test via Polypharmacology." Evidence-Based Complementary and Alternative Medicine 2020 (July 11, 2020): 1–10. http://dx.doi.org/10.1155/2020/2153571.

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Paeoniflorin, an organic compound extracted from the roots of the white peony (Paeonia lactiflora) plant, has previously been shown to exert antidepression and prokinetic effects. The traditional Chinese prescription Si-Ni-San, of which paeoniflorin is a constituent, is often used in treating depression and functional gastrointestinal disorders. The effectiveness of Si-Ni-San has been shown to be less effective in a paeoniflorin-deleted form. The present study further investigates whether paeoniflorin alone is as effective as herbal prescriptions in which the compound is a constituent, specifi

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42

O'Neil, Heather S., and Hélène Marquis. "Listeria monocytogenes Flagella Are Used for Motility, Not as Adhesins, To Increase Host Cell Invasion." Infection and Immunity 74, no. 12 (2006): 6675–81. http://dx.doi.org/10.1128/iai.00886-06.

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ABSTRACT Flagellar structures contribute to the virulence of multiple gastrointestinal pathogens either as the effectors of motility, as adhesins, or as a secretion apparatus for virulence factors. Listeria monocytogenes is a food-borne, gram-positive pathogen that uses flagella to increase the efficiency of epithelial cell invasion (A. Bigot, H. Pagniez, E. Botton, C. Frehel, I. Dubail, C. Jacquet, A. Charbit, and C. Raynaud, Infect. Immun. 73:5530-5539, 2005; L. Dons, E. Eriksson, Y. Jin, M. E. Rottenberg, K. Kristensson, C. N. Larsen, J. Bresciani, and J. E. Olsen, Infect. Immun. 72:3237-32

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Zabielski, R., P. Kiela, T. Onaga, H. Mineo, S. Kato, and P. C. Gregory. "Effect of neural blockades, gastrointestinal regulatory peptides, and diversion of gastroduodenal contents on periodic pancreatic secretion in the preruminant calf." Canadian Journal of Physiology and Pharmacology 73, no. 11 (1995): 1616–24. http://dx.doi.org/10.1139/y95-723.

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The role of nerves, gastrointestinal peptides, and gastroduodenal contents in the regulation of pancreatic periodic function were studied in preruminant calves. Nine male, Friesian calves were surgically fitted with pancreatic and duodenal catheters, abomasal and duodenal cannulae, and duodenal electrodes. Pancreatic secretion oscillated in phase with the duodenal migrating myoelectric complex. Pancreatic secretion and duodenal motility were abolished by intravenous atropine (5 μg∙kg−1∙min−1). The frequency of pancreatic and duodenal cycles was similarly increased by motilin and decreased by p

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44

Leming, S., J. Broad, S. J. Cozens, et al. "GSK962040: a small molecule motilin receptor agonist which increases gastrointestinal motility in conscious dogs." Neurogastroenterology & Motility 23, no. 10 (2011): 958—e410. http://dx.doi.org/10.1111/j.1365-2982.2011.01770.x.

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45

Zhou, Qiu-Juan, Zhan-Guo Nie, Zhong-Ming Dai, Xin Zhang, and Xiang Dong. "Gastrointestinal motility and changes of serum motilin and gastrin in conscious heat stroke rats." World Chinese Journal of Digestology 22, no. 21 (2014): 3003. http://dx.doi.org/10.11569/wcjd.v22.i21.3003.

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46

Washabau, Robert J. "Gastrointestinal motility disorders and gastrointestinal prokinetic therapy." Veterinary Clinics of North America: Small Animal Practice 33, no. 5 (2003): 1007–28. http://dx.doi.org/10.1016/s0195-5616(03)00076-7.

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47

Farmer, Adam D., S. Mark Scott, and Anthony R. Hobson. "Gastrointestinal motility revisited: The wireless motility capsule." United European Gastroenterology Journal 1, no. 6 (2013): 413–21. http://dx.doi.org/10.1177/2050640613510161.

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Guo, Xuan, Yin Xu, Hua-liang Tan, Xiao-juan Wang, and Lin Xiao. "The Key Ingredient Acacetin in Weishu Decoction Alleviates Gastrointestinal Motility Disorder Based on Network Pharmacology Analysis." Mediators of Inflammation 2021 (September 21, 2021): 1–12. http://dx.doi.org/10.1155/2021/5265444.

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Abstract:

Background. Gastrointestinal motility disorder is a common gastrointestinal disease, which seriously affects life quality. Traditional Chinese medicine (TCM) has been widely used as an alternative therapy for gastrointestinal motility disorder. Acacetin is a natural flavonoid compound that has antioxidant and anti-inflammatory, antidepressant, and anticancer properties. However, the efficacy of Acacetin in the treatment of gastrointestinal motility disorders has not been studied. Our aim was to investigate the mechanism of Acacetin-alleviated gastrointestinal motility disorder and its efficacy

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Törnblom, Hans, Magnus Simrén, and Hasse Abrahamsson. "Gastrointestinal motility and neurogastroenterology." Scandinavian Journal of Gastroenterology 50, no. 6 (2015): 685–97. http://dx.doi.org/10.3109/00365521.2015.1027265.

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Lee, Oh Young. "Gastrointestinal Motility Modulating Drugs." Journal of the Korean Medical Association 52, no. 9 (2009): 920. http://dx.doi.org/10.5124/jkma.2009.52.9.920.

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