Relevant bibliographies by topics / Gastrointestinal Neoplasms

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Gastrointestinal Neoplasms'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 June 2025

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Journal articles on the topic "Gastrointestinal Neoplasms"

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Sharma, Supriya, and Vinay K. Kapoor. "Gastrointestinal Neuroendocrine Neoplasms." Indian Journal of Endocrine Surgery and Research 16, no. 2 (2022): 51–58. http://dx.doi.org/10.5005/jp-journals-10088-11177.

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Savari, Omid, Hope Hastings, Rania Rayes, and Joseph F. Tomashefski. "Neuroendocrine Neoplasia in Extranodal Marginal Zone Lymphoma of Mucosa-Associated Lymphoid Tissue (MALT Lymphoma) of the Lung: A Case Report and Immunohistochemistry Analysis of Eight Pulmonary MALT Lymphomas." International Journal of Surgical Pathology 26, no. 7 (2018): 660–63. http://dx.doi.org/10.1177/1066896918769745.

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Carcinoid tumorlets are peribronchiolar proliferations of neuroendocrine cells often associated with lung scars. Extranodal marginal zone lymphoma of mucosa-associated lymphoid tissue (MALT lymphoma) is a non-Hodgkin’s lymphoma that frequently involves the gastrointestinal tract but less commonly is described in the lung. Simultaneous occurrence of neuroendocrine neoplasms and MALT lymphoma is extraordinarily rare and has predominately been reported in the gastrointestinal tract. In this article, we describe the case of a 73-year-old female with coexisting pulmonary MALT lymphoma and carcinoid tumorlets of the right middle lobe. Retrospective series of 8 pulmonary MALT lymphomas are evaluated for neuroendocrine neoplasia by immunohistochemistry. No correlation between MALT lymphoma and neuroendocrine neoplasia was identified in this case series. While the concurrence of these distinctive neoplasms is most likely coincidental, the presence of a common risk factor, or one neoplasm as a risk factor for the other, deserves study of a larger group of pulmonary MALT lymphomas.
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Lee, Eun-Joo, Myung-Jin Chung, and Kyu-Shik Jeong. "Canine Intestinal Lymphangiectasia Concomitant with Renal Cell Carcinoma." Acta Veterinaria 71, no. 3 (2021): 351–60. http://dx.doi.org/10.2478/acve-2021-0030.

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Abstract The etiology of dilation of lymphatic vessels, termed as intestinal lymphangiectasia, remains unknown. In most cases, it occurs secondary to other pathologic conditions such as gastrointestinal neoplasms. However, only a few cases of canine intestinal lymphangiectasia concurrent with non-gastrointestinal neoplasms have been reported so far. Moreover, the correlation between intestinal lymphangiectasia and non-gastrointestinal neoplasms has not been discussed in any other literature. In this study, we report a rare case of intestinal lymphangiectasia concomitant with renal cell carcinoma in an 11 year old female mixed Maltese, suggesting that non-gastrointestinal neoplasms could be associated with the development of intestinal lymphangiectasia. On gross observation, the small intestine was irregularly swollen presenting an accordion like shape. Microscopic examination revealed prominent dilatation of the lymphatic vessels, especially, within the submucosa and muscularis layer. The lacteals within the villi were dilated and presented “club-shaped” tips. The carcinoma might trigger intestinal lymphangiectasia by compressing the main lymphatic vessels or the cisterna chyli, subsequently increasing the pressure of the lymphatic vessels in the gastrointestinal tract. Moreover, metastasis of the carcinoma to the gastrointestinal tract could induce intestinal lymphangiectasia. Thus, the occurrence of intestinal lymphangiectasia must be considered when an abdominal neoplasm is located around major lymphatic vessels.
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Del Piero, F., B. A. Summers, J. F. Cummings, G. Mandelli, and E. A. Blomme. "Gastrointestinal Stromal Tumors in Equids." Veterinary Pathology 38, no. 6 (2001): 689–97. http://dx.doi.org/10.1354/vp.38-6-689.

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Eleven gastrointestinal neoplasms from 10 aged horses and 1 pony were examined grossly, histologically, immunohistochemically, and (in two cases) ultrastructurally. Clinical signs were associated with two neoplasms, and the other nine tumors were incidental findings at laparotomy or necropsy. The neoplasms were solitary (9/11) or multifocal (2/11), well demarcated, serosal or mural masses of stomach (1), jejunum (1), ileum (3), cecum (5), and/or colon (2). Microscopic examination revealed discrete spindle cells arranged in compact patterns with fascicles and whorls or cribriform pattern with fascicles and rare palisades, often with a myxoid interstitial matrix. Three tumors infiltrated between the muscularis interna and the muscularis externa at the myenteric plexi. All neoplasms were vimentin positive, 3/11 were S-100 positive, 2/11 were muscle actin positive, and no neoplasm was positive for glial fibrillary acid protein, desmin, factor VIII, chromogranin, or neuron-specific enolase. Of the two tumors studied ultrastructurally, one contained an admixture of smooth muscle cells and cells resembling Schwann cells, and the second was populated by homogeneous fusiform mesenchymal cells separated by homogeneous matrix. Gastrointestinal stromal tumors (GIST) have been recognized in humans, more recently in dogs and nonhuman primates, and now in equids. Most of these tumors are comprised of a loosely arranged network of spindled cells separated by myxoid matrix. GIST may be composed of myogenic, neurogenic, combined myogenic and neurogenic, and undifferentiated mesenchymal cells.
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Binkowska-Borgosz, Izabela, Teresa Starzyńska, and Wojciech Błogowski. "Obesity and gastrointestinal neoplasms." Postępy Higieny i Medycyny Doświadczalnej 68 (October 17, 2014): 1193–98. http://dx.doi.org/10.5604/17322693.1125648.

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NARDI, PETER M. "AIDS-Related Gastrointestinal Neoplasms." Contemporary Diagnostic Radiology 17, no. 5 (1994): 1–6. http://dx.doi.org/10.1097/00219246-199417050-00001.

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Turner, Matthew S., and Jeffrey D. Goldsmith. "Best Practices in Diagnostic Immunohistochemistry: Spindle Cell Neoplasms of the Gastrointestinal Tract." Archives of Pathology & Laboratory Medicine 133, no. 9 (2009): 1370–74. http://dx.doi.org/10.5858/133.9.1370.

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Abstract Context.—The proper classification of spindle cell neoplasms of the gastrointestinal tract frequently requires the use of immunohistochemistry, as the histologic appearance of these lesions often overlaps. Objective.—To review the antibodies used in the diagnosis of spindle cell neoplasms of the gastrointestinal tract, and to outline an approach to the evaluation of these lesions by using immunohistochemistry. Data Sources.—The authors' experience and a review of the English literature from 1976 to 2008. Conclusions.—The most common spindle cell neoplasm of the gastrointestinal tract is gastrointestinal stromal tumor; this lesion is readily diagnosed with c-kit immunohistochemistry in most cases. Other stains, such as smooth muscle actin, desmin, S100 protein, and β-catenin, are also useful in the diagnosis of smooth muscle tumors, schwannomas, desmoid-type fibromatoses, and metastatic melanoma.
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AbdullGaffar, Badr. "Gastrointestinal Stromal Tumors and Extra-Gastrointestinal Tract Neoplasms." Southern Medical Journal 103, no. 10 (2010): 1004–8. http://dx.doi.org/10.1097/smj.0b013e3181ef2f41.

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Hernández-Luna, Marco Antonio, Sergio López-Briones, and Rosendo Luria-Pérez. "The Four Horsemen in Colon Cancer." Journal of Oncology 2019 (September 29, 2019): 1–12. http://dx.doi.org/10.1155/2019/5636272.

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Worldwide, neoplasms of the gastrointestinal tract have a very high incidence and mortality. Among these, colorectal cancer, which includes colon and rectum malignancies, representing both highest incidence and mortality. While gallbladder cancer, another neoplasm associated to gastrointestinal tract occurs less frequently. Genetic factors, inflammation and nutrition are important risk factors associated with colorectal cancer development. Likewise, pathogenic microorganisms inducing intestinal dysbiosis have become an important scope to determine the role of bacterial infection on tumorigenesis. Interestingly, in human biopsies of different types of gastrointestinal tract cancer, the presence of different bacterial strains, such as Fusobacterium nucleatum, Escherichia coli, Bacteroides fragilis and Salmonella enterica have been detected, and it has been considered as a high-risk factor to cancer development. Therefore, pathogens infection could contribute to neoplastic development through different mechanisms; including intestinal dysbiosis, inflammation, evasion of tumoral immune response and activation of pro-tumoral signaling pathways, such as β catenin. Here, we have reviewed the suggested bacterial molecular mechanisms and their possible role on development and progression of gastrointestinal neoplasms, focusing mainly on colon neoplasms, where the bacteria Fusobacterium nucleatum, Escherichia coli, Bacteroides fragilis and Salmonella enterica infect.
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Muralidhar, Aparna, and Pushpa Mahadevan. "Spectrum of neuroendocrine neoplasms of GIT – a histomorphological study in a tertiary care centre." Journal of Medical Science 92, no. 2 (2023): e813. http://dx.doi.org/10.20883/medical.e813.

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Background. Neuroendocrine neoplasms are diverse in terms of sites of origin, functional status, and degrees of aggressiveness. Since neuroendocrine cells are ubiquitous in the human body, these neoplasms can arise in different organs, with gastrointestinal tract being most frequently involved. The past few years have seen a surge in the diagnosis of these neoplasms, which were earlier considered to be rare. Their nomenclature, classification, and diagnostic criteria are revamped frequently, as new knowledge emerges. Aim. To study the histopathological spectrum of neuroendocrine neoplasms of gastrointestinal tract and assess the immunohistochemical expression of neuroendocrine markers in them. Material and methods. Ours is a descriptive study of the distribution and pathologic characteristics of gastrointestinal neuroendocrine neoplasms in a tertiary care hospital in Kerala, over a three year period. Neoplasms were categorised based on 2019 updated WHO classification. Results. Among the 59 cases, we observed a male predominance. (Male to female ratio - 1.8:1). Most patients were in 6th and 7th decades of life. Duodenum was most frequently involved followed by rectum and appendix. NET G2 and G1 constituted the predominant histologic grades (47% and 24% respectively). NEC and MiNEN were infrequent. All cases were positive for synaptophysin, with variable positivity for chromogranin. Ki67 helped establish the histologic grade. We also came across a rare case of neuroendocrine tumor with coexisting mucinous neoplasm in appendix. Conclusions. With evolving knowledge and advanced imaging modalities, the incidence of these neoplasms is increasing with time. Histopathology is the mainstay of diagnosis and plays a decisive role in influencing management protocols and prognosis.
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Dissertations / Theses on the topic "Gastrointestinal Neoplasms"

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Larsson, Susanna C. "Diet and gastrointestinal cancer : one-carbon metabolism and other aspects /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-955-6/.

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Nordenvall, Caroline. "Aspects of the etiology and survival of lower gastrointestinal cancers." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-813-6/.

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Sundelöf, Martin. "Aspects on prognosis of cancers of the oesophagus and gastric cardia /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7357-011-7/.

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Lindblad, Mats. "Aspects on the etiology of esophageal and gastric cancer /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-110-5/.

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Swärd, Christina. "Aspects on diagnosis and treatment of gastrointestinal neuroendocrine tumours." Göteborg : Dept. of Surgery, Institute of Clinical Sciences at Sahlgrenska Academy, Lundberg Laboratory for Cancer Research, University of Gothenburg, 2010. http://hdl.handle.net/2077/21863.

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Canena, Jorge Manuel Tavares. "Próteses no tubo digestivo: evolução e aplicações no século XXI." Doctoral thesis, Faculdade de Ciências Médicas. Universidade Nova de Lisboa, 2012. http://hdl.handle.net/10362/7874.

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RESUMO: A utilização das próteses metálicas auto-expansíveis é um tratamento paliativo e definitivo muito eficaz em doentes com obstrução maligna coló-retal e gastroduodenal. Este tratamento está associado a taxas de morbilidade e de intervenção adicional aceitáveis. O recurso a próteses expansíveis em doenças benignas pode constituir uma opção terapêutica válida. As próteses biodegradáveis e as próteses metálicas totalmente cobertas podem ser utilizadas, com eficácia aceitável, num grupo de doentes com estenóses refractárias do esófago e para os quais não estão disponíveis grandes opções de tratamento. As próteses metálicas totalmente cobertas podem ser um tratamento de excepção em situações benignas biliares onde anteriormente a cirurgia era a única opção disponível. As próteses metálicas são a opção de escolha no tratamento paliativo do colangiocarcinoma hilar, independentemente do tipo de lesão. Em doentes com estenoses do tipo II de Bismuth a colocação bilateral de próteses metálicas, sempre que tecnicamente possíveis, deve ser recomendada pois está associada a uma maior patência das próteses e a uma menor taxa de reintervenção.
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Scott, Lucy C. "Biomarker development for gastrointestinal and ovarian cancer : a proteomic approach." Thesis, University of Glasgow, 2010. http://theses.gla.ac.uk/1644/.

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The development of new biomarkers for cancer patients would be advantageous in population screening for the early detection of cancers, pathological diagnosis, assessment of prognosis, tailoring treatment to individuals, and assessment of treatment response. With this in mind different proteomic approaches were used to identify biomarkers which could potentially aid prognosis and predict response in gastrointestinal and ovarian cancer. Raf Kinase Inhibitor Protein (RKIP) was originally purified from bovine brain extracts and named phosphatidylethanolamine-binding protein (PEBP). It has subsequently been shown to be a widely expressed and highly conserved protein. Several recent studies have suggested that RKIP may suppress metastasis in melanoma, prostate, and breast cancer, as reduction or loss of RKIP expression was observed in metastatic cell lines and metastatic tissue. In this part of the project RKIP expression was assessed by immunohistochemistry in tissue microarrays (TMA) from patients with colorectal and ovarian cancer. The results confirmed the findings of earlier studies and suggest that the level of RKIP expression is significantly and inversely associated with metastatic disease and can predict the risk of metastatic relapse in patients with no evidence of metastases at presentation. The level of RKIP expression as a prognostic factor was independent of sex, age, tumour site, mitotic index, lymphovascular invasion and tumour stage. Cytokeratin 18 (CK18) is an epithelial-specific cytokeratin that undergoes cleavage by caspases during apoptosis. Measurement of caspase-cleaved (CK18-NE) or total cytokeratin 18 (CK18) from epithelial-derived tumours could be a simple, non-invasive way to monitor or predict responses to treatment. Soluble plasma CK18-NE and CK18 were measured by ELISA from 73 patients with advanced gastrointestinal adenocarcinomas before treatment and during chemotherapy, as well as 100 healthy volunteers. Both CK18-NE and total CK18 plasma levels were significantly higher in patients compared to the healthy volunteers (p=0.015, p<0.001). The total CK18 baseline plasma levels prior to treatment were significantly higher (p=0.009) in patients who develop progressive disease than those who achieve partial response or stable disease and this correlation was confirmed in an independent validation set. The peak plasma levels of CK18 occurring in any cycle following treatment were also found to be associated with tumour response, but peak levels of CK18-NE did not reach significance (p=0.01, and p=0.07, respectively). A surface-enhanced laser desorption-ionisation mass spectrometry (SELDI-MS) pilot study on serum from 8 oesophageal cancer patients and 8 healthy volunteers revealed a novel biomarker, ~4kDa, downregulated in patients (p=0.012). An expanded 30 tumour/normal study was performed for validation which confirmed the down-regulation of this potential biomarker (p<0.0001). Attempts to identify tentatively suggested that the peptide may be inter-alpha-trypsin inhibitor heavy chain H4 precursor, which was interesting as a cleavage fragment of inter-alpha -trypsin inhibitor heavy chain H4 had been previously found to be up-regulated in patients with ovarian cancer, and down-regulated in patients with breast cancer. However, it was not possible to confidently confirm this identification. In a further part of this study, haptoglobin was found to be significantly more abundant in the serum from patients with oesophageal cancer compared to healthy volunteers. It was straightforward to isolate and identify and would be amenable to immunoassay as there are good antibodies available for confirmation. In conclusion, with the current lack of effective markers of metastatic relapse in colorectal cancer, a straightforward test like RKIP expression in the primary tumour may be a very cost-effective way to identify which patients may derive greater benefit from adjuvant treatment and closer post-operative surveillance, and in patients with advanced gastrointestinal malignancy levels of plasma CK18 are a potential marker of tumour response.
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Held, Maria. "Epidemiological studies of Helicobacter pylori and its relation to cancer and precancerous lesions in the upper gastrointestinal tract /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-944-7/.

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Beamish, Andrew James. "Applied physiology in upper gastrointestinal cancer surgery : perioperative risk stratification and management." Thesis, Cardiff University, 2014. http://orca.cf.ac.uk/71521/.

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This thesis examines methods of perioperative risk stratification and outcome in patients receiving multidisciplinary stage-directed treatment for oesophagogastric cancer. The hypotheses tested were: Suboptimal bioelectrical impedance analysis (BIA) body composition variables predict poor outcomes in oesophagogastric cancer (OGC) surgery; low CT-measured psoas muscle density (PMD) predicts poor outcomes in OGC surgery; suboptimal cardiopulmonary exercise (CPX) performance predicts poor outcomes following OGC surgery; the literature offers evidence in support of enhanced recovery programmes in OGC surgery; the use of an enhanced recovery programme in OGC surgery is feasible, safe and not associated with adverse outcomes. High values for BIA-derived measures of fat-free mass and muscle mass respectively predicted longer survival (p=0.047, p=0.011), but were not associated with reduced 30-day mortality, major morbidity or length of stay. CT-measured psoas muscle density greater than the median of 48.7 Hounsfield Units predicted longer survival (p=0.046), but was not associated with reduced 30-day mortality, major morbidity or length of stay (LOHS). Multivariable analysis demonstrated radiological TNM stage (p=0.015), and both left (p=0.046) and right PMD (p=0.047), as significant and independent predictors of survival. Cardiopulmonary exercise testing results materially altered the management plan in 6.8% patients. Major morbidity (p=0.049) and poor survival (p=0.048) were associated with a high ventilatory equivalent for carbon dioxide (VE/VCO2), but not with the anaerobic threshold (AT) or peak oxygen uptake (VO2peak). VE/VCO2 also emerged on multivariable analysis as an independent and significant predictor of LOHS (p=0.001). Systematic review and meta-analysis revealed enhanced recovery programmes (ERPs) in OGC surgery to be feasible, safe and costeffective, significantly shortening length of stay (LOHS, p<0.0001). In our unit, the introduction of ERPs in gastric and oesophageal cancer surgery respectively, significantly reduced LOHS (p=0.004; p=0.032), critical care stay (p<0.0001; p<0.0001) and overall cost (p=0.001; p<0.0001).
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Derakhshan, Mohammad H. "Upper gastrointestinal adenocarcinoma : associations with gastric secretory function and gender." Thesis, University of Glasgow, 2008. http://theses.gla.ac.uk/479/.

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Gastric and oesophageal cancers were responsible for more than one million deaths in 2002. Although global incidence of gastric cancer is decreasing, this malignancy is still the fourth most common cause of cancer worldwide. The incidence of oesophageal adenocarcinoma is rising rapidly, three-fold in the last two decades. The incidence of adenocarcinoma of gastric cardia is stable. In the pathogenesis of both gastric and oesophageal adenocarcinomas, the state of the gastric mucosa and its secretory function plays a central role. Non-cardia adenocarcinoma develops in subjects with H.pylori associated atrophic gastritis and hypochlorhydria. Little is known about the gastric phenotype in patients with adenocarcinoma of the cardia and gastroesophageal junction. Another important but poorly understood risk factor for upper GI adenocarcinoma is male gender. In the first study we aimed to investigate the association between the pattern of H.pylori gastritis and gastric secretory function in 255 H.pylori-infected patients with dyspepsia showing normal endoscopy. Our findings showed that maximal acid output correlates inversely with severity of corpus gastritis, corpus atrophy, and positively related to male gender and serum pepsinogen I. In the second study we compared cancers at the cardia and non-cardia subsites with respect to pre-morbid gastric mucosal atrophy and acid secretion. In a nested case-control study comprising 101,601 men and women enrolled in the Norwegian JANUS cohort, 230 cases of gastric cancer were identified. 173 cases including 144 non-cardia and 44 cardia cancer were enrolled to study. Three controls were matched to each case. Serum pepsinogen I, pepsinogen II, anti-H.pylori IgG antibody and gastrin were measured using serums which had been collected a median of 11.9 years before cancer diagnosis radioimmunoassay method. Non-cardia cancer was positively associated with H.pylori and gastric atrophy. The diffuse and intestinal histological subtypes of non-cardia cancer were of similar proportions and both showed a positive association with H.pylori and atrophy. Cardia cancer was negatively associated with H.pylori, but H.pylori positive cardia cancer showed a positive association with gastric atrophy. The predominant histological subtype of cardia cancer was intestinal and it was not associated with gastric atrophy compared to the diffuse subtype. Cardia cancer in atrophic patients had an intestinal: diffuse ratio similar to non-cardia cancer, whereas cardia cancers in persons without atrophy were predominantly intestinal. These findings indicate two aetiologies of cardia cancer, one associated with H.pylori atrophic gastritis, resembling non-cardia cancer, and the other associated with non-atrophic gastric mucosa, resembling oesophageal adenocarcinoma. Serological markers of gastric atrophy may provide the key to determining gastric versus oesophageal origin of cardia cancer. In the third study we extended our investigation of the aetiology of cardia cancer by examining the association of both serological evidences of gastric atrophy and gastroesophageal reflux disease (GORD) symptoms with adenocarcinoma of the oesophagus, cardia and non-cardia regions of the stomach. This has been performed for the different histological subtypes of the cancer. We have also included H.pylori status and smoking history which are other well established risk factors for upper GI cancer. This has been undertaken in a population in Northwest Iran with a high incidence of upper gastrointestinal cancer. Serum pepsinogen I/II was used as a marker of atrophic gastritis and categorised to five quintiles. History of GORD symptoms, smoking and H.pylori infection was incorporated in logistic regression analysis. Lauren classification was used to subtype gastric and oesophageal adenocarcinoma. Non-cardia cancer was associated with atrophic gastritis but not with GORD symptoms; 55% of these cancers were intestinal subtype. Oesophageal adenocarcinoma was associated with GORD symptoms, but not with atrophic gastritis; 84% were intestinal subtype. Cardia cancer was positively associated with both severe gastric atrophy and with frequent GORD symptoms though the latter was only apparent in the non-atrophic subgroup and in the intestinal subtype. The association of cardia cancer with atrophy was stronger for the diffuse versus intestinal subtype and this was the converse of the association observed with non-cardia cancer. These findings indicate two distinct aetiologies of cardia cancer, one arising from severe atrophic gastritis and being of intestinal or diffuse subtype similar to non-cardia cancer, and one related to GORD and intestinal in subtype, similar to oesophageal adenocarcinoma. Gastric atrophy, GORD symptoms and histological subtype may distinguish between gastric versus oesophageal origin of cardia cancer. In the fourth study we investigated the relationship between gender and upper gastrointestinal adenocarcinoma. Male gender is a well-established risk factor for oesophageal adenocarcinoma. Male predominance of gastric cancer is related to the histological subtype of the tumour being more marked in the intestinal versus diffuse histological subtype. In addition, global data suggests that the male predominance of upper gastrointestinal cancer is related to the anatomical location, being higher for proximal and lower for distal tumours. However, the proportion of the intestinal histological subtype differs according to anatomical site and it is unclear whether it is the anatomical site or the histological subtype which is associated with the gender phenomenon. We have conducted a population-based study to investigate this. The study was based upon 3270 gastric and oesophageal cancers recorded in West of Scotland Cancer Registry between 1998 and 2002. The Lauren subtype of adenocarcinoma was determined by reviewing 1204 reports and 3241 slides in a sample of 812 cases. Logistic regression models were used to estimate relationship between male predominance and histological subtype, tumour location and age. We found that the crude incidence rate of intestinal subtype was higher in males (23.86/ 100,000 person-years) versus females (9.00/ 100,000 person-years), giving M/F of 2.65. M/F ratio of intestinal subtype cancer was 3.41 at age <50, reached a peak of 7.86 at age 50-59, and then showed a progressive decrease throughout the life. In contrast, the incidence rate of diffuse subtype adenocarcinoma was similar in both sexes (5.58 vs. 5.20 /100,000 person-years) yielding M/F of 1.07. Multivariate analyses including histological subtype, tumour location and age indicated that the male predominance was related to the histological type rather than anatomical location. Intestinal type tumour showed similar male predominance of incidence irrespective of its anatomical location (OR, 95% CI: 2.6, 1.78 – 3.9). Further analysis of the age-specific incidence curves indicated that the male predominance of intestinal subtype was due to a 17.2-year delay of development of this cancer in females.
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Books on the topic "Gastrointestinal Neoplasms"

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L, Fielding J. W., and Priestman Terry J, eds. Gastrointestinal oncology. Lea & Febiger, 1986.

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P, Griffin-Sobel Joyce, and Oncology Nursing Society, eds. Gastrointestinal cancers. Oncology Nursing Society, 2007.

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Abbruzzese, James L. Gastrointestinal oncology. Oxford University Press, 2004.

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M, Daly John, Hennessy T. P. J, and Reynolds John V, eds. Management of upper gastrointestinal cancer. W.B. Saunders, 1999.

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J, Belcastro V., and Dobelbower Ralph R, eds. Gastrointestinal cancer: Radiation therapy. Springer-Verlag, 1990.

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R, Dobelbower Ralph, ed. Gastrointestinal cancer: Radiation therapy. Springer-Verlag, 1989.

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David, Kelsen, ed. Principles and practice of gastrointestinal oncology. 2nd ed. Lippincott, Williams & Wilkins, 2008.

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A, Ajani Jaffer, and Hoff Paulo M, eds. Atlas of gastrointestinal cancers. Current Medicine, 2006.

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Wolfgang, Fischbach, ed. Gastrointestinal lymphoma: Future perspectives. Springer, 2000.

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Cady, Blake. Polyps of the gastrointestinal tract. W.B. Saunders, 1996.

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Book chapters on the topic "Gastrointestinal Neoplasms"

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Colloca, Giuseppe A., and Antonella Venturino. "Neuroendocrine neoplasms." In The Prognosis of Metastatic Gastrointestinal Cancer. CRC Press, 2024. http://dx.doi.org/10.1201/9781032703350-7.

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Rosty, Christophe, Michael B. Wallace, and Till S. Clauditz. "Epithelial Neoplasms of the Large Bowel." In Gastrointestinal Pathology. John Wiley & Sons, Ltd, 2021. http://dx.doi.org/10.1002/9781119073048.ch12.

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Larson, Brent K., and Deepti Dhall. "Neuroendocrine Neoplasms of the Gastrointestinal Tract." In Practical Gastrointestinal Pathology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-51268-2_17.

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Chun, Hoon Jai, Seun Ja Park, Yun Jeong Lim, and Si Young Song. "Pancreatic Neuroendocrine Neoplasms. V-2. Staging and Treatment." In Gastrointestinal Cancer. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-0815-8_65.

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Geboes, K., and C. De Wolf-Peeters. "Neoplasms of the Small Intestine." In Gastrointestinal and Liver Tumors. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18629-5_7.

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Chun, Hoon Jai, Seun Ja Park, Yun Jeong Lim, and Si Young Song. "Pancreatic Neuroendocrine Neoplasms. V-1. Epidemiology and Clinical Features." In Gastrointestinal Cancer. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-0815-8_64.

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Jung, Hwoon-Yong, and Ho June Song. "Endoscopic Submucosal Dissection for Early Esophageal Neoplasms." In Therapeutic Gastrointestinal Endoscopy. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-1184-0_7.

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Singh, Harkirat, and Asif Khalid. "Pancreatic Cystic Neoplasms in Women: Mucinous Cystic Neoplasms, Serous Cystadenomas, and Solid Pseudopapillary Neoplasms." In Gastrointestinal and Liver Disorders in Women’s Health. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25626-5_9.

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Chun, Hoon Jai, Seun Ja Park, Yun Jeong Lim, and Si Young Song. "Staging and Treatment. II-3. Surgical Resection for Appendiceal Neoplasms." In Gastrointestinal Cancer. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-0815-8_29.

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Walsh, Shaun V., and Frank A. Carey. "Malignant Epithelial Neoplasms of the Large Bowel." In Morson and Dawson's Gastrointestinal Pathology. Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118399668.ch38.

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Conference papers on the topic "Gastrointestinal Neoplasms"

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Bertoldi, P., J. Aguirre, and E. Barbosa. "Traction-assisted versus conventional endoscopic submucosal dissection for superficial gastrointestinal neoplasms: a systematic review and meta-analysis of randomized controlled trials." In ESGE Days 2024. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1783089.

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Nayak, Hemanta Kumar, Shubham Gupta, Abhijeet Rai, et al. "Endoscopic Ultrasound (EUS)-Guided Tissue Acquisition and Histomorphologic Analysis for Suspected Spindle Cell Neoplasms of the Upper Gastrointestinal Tract: Does the Needle Size and Type Matter?" In ENDOCON 2024. Thieme Medical and Scientific Publishers Pvt. Ltd., 2024. http://dx.doi.org/10.1055/s-0044-1786310.

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Pereira, Igor, Júlia Pizelli, Lara Salim, José de Assis Silva Júnior, Lara Vianna de Barros Lemos, and Luisa Aguirre Buexm. "Perfil clínico e sobrevida dos pacientes com neoplasias malignas gastrointestinais do Hospital Escola Álvaro Alvim." In Semana Científica da Faculdade de Medicina de Campos. Faculdade de Medicina de Campos, 2022. http://dx.doi.org/10.29184/anaisscfmc.v12022p10.

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Introdução: Anualmente mais de 18,1 milhões de pessoas no mundo são diagnosticadas com câncer, sendo que mais da metade morrem vítimas dessa doença. Sabe-se que 50% dos casos no Brasil são diagnosticados em fases avançadas e 70% estão relacionados a fatores ambientes e poderiam ser evitados com uma mudança no estilo de vida. No que tange a mortalidade, as neoplasias malignas gastrointestinais estão entre as maiores causadoras de morte. Objetivo: Esse trabalho visa analisar o perfil clínico e sobrevida de pacientes com neoplasias malignas gastrointestinais tratados no Hospital Escola Álvaro Alvim, durante cinco anos. Métodos: Estudo observacional, retrospectivo e descritivo, realizado no setor de oncologia do Hospital Escola Álvaro Alvim (HEAA), com análise secundária e anônima de prontuários médicos de 675 pacientes com neoplasias malignas de gastrointestinais entre de 01 de janeiro de 2012 a 31 de dezembro de 2016, e aprovado pelo Comitê de Ética. Foram incluídos os maiores de 18 anos diagnosticados com neoplasia maligna gastrointestinais confirmado por exame histopatológico no HEAA, e excluídos os menores de 18 anos com exame negativo para essas neoplasias. e que, portanto, não são acompanhados no hospital. As análises das variáveis sociodemográficas e clínico-patológicas foram realizadas através de proporções e médias, utilizando gráficos e tabelas, com auxílio de programa estatístico. Resultados e Discussão: Do total de 675 pacientes, 25,8% apresentavam tumores localizados na região gástrica e 74,2% na região intestinal. Homens (52,7%), brancos (31,1%), casados (49,6%), com 60 anos ou mais (54,2%), procedentes de Campos dos Goytacazes (71,7%), com histórico de câncer familiar (24,6%), sem hábitos, como tabagismo (36,3%) e etilismo (36,1%), com tumores classificados histopatologicamente como adenocarcinoma (68,9%), em estadiamento clínico avançado (42,2%), submetidos a tratamento cirúrgico primário (37,5%), que não apresentavam metástase (85,6%), nem progressão da doença (61%) e que não vieram a óbito (32%) foram os mais acometidos pelas neoplasias malignas de gastrointestinais. Foram observadas associações entre estadiamento avançado e tipo histopatológico adenocarcinoma (p=0,024), tratamento quimioterápico (p=0,010) e óbito (p≤0,0001). Já as neoplasias intestinais foram associadas ao sexo feminino (p≤0,0001), não tabagismo (p=0,002) e não etilismo (p=0,001). A sobrevida global (SG) média da população do estudo foi de 36,2 meses. Pacientes do sexo masculino (p=0,014), com câncer gástrico (p≤0,0001), em estadiamento clínico IV (p≤0,0001), com metástase ao diagnóstico (p≤0,0001) apresentaram uma pior SG. Conclusões: As neoplasias malignas de gastrointestinais apresentam uma incidência elevada na população do Norte Fluminense. O câncer intestinal foi a neoplasia maligna de maior incidência, seguida do câncer gástrico, corroborando com os dados da literatura. Este levantamento epidemiológico contribui para traçar um perfil clínico, estratificando os grupos de risco e auxilia na prevenção, detecção precoce e tratamento para as neoplasias malignas gastrointestinais.
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Marques, Marina Trombin, Leonardo de Sousa Bernardes, Rafael Zini Moreira da Silva, et al. "Trousseau Syndrome in a patient on Direct Oral Anticoagulant use: A Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.508.

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Background: The Three Territory Sign (TTS) is a radiologic marker of ischemic stroke associated with malignant neoplastic diseases (Trousseau Syndrome) and corresponds to a rare stroke etiology. Case Report: Female, 62-year-old patient, with comorbidities of smoking, hypertension and diabetes, presented with a sudden faciobrachial-predominant left hemiparesis settled in the day before the admission. Diagnosed with a metastatic rectal adenocarcinoma seven months before, she underwent a rectosigmoidectomy three months ago and developed deep vein thrombosis, starting anticoagulant therapy with rivaroxaban 20mg daily. A Magnetic Resonance Imaging (MRI) revealed several lesions with restricted diffusion in multiple vascular territories, bilaterally, corresponding to ischemic stroke. Etiologic investigation did not detect signs of cardioembolism, nor significant vessel stenosis or unstable atherosclerotic plaques. In admission, she had a D-dimer level of 11,43μg (0- 0,5μg/mL). Conclusion: The evidence of TTS is about six times more frequent in stroke related to malignancies compared to cardioembolic etiology. The D-dimer is a marker of malignancies in cryptogenic stroke, elevated in 75% of cases. The most common associated neoplasms are pulmonary (40%) and gastrointestinal (33,3%). In the MRI, the lesions can be isolated or gathered, generally small and peripheral. There is no evidence regarding the ideal preventive therapy. It is necessary to reinforce the importance of investigating malignancies in patients presenting with cryptogenic stroke and TTS, a syndrome that is still poorly recognized.
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Kandolf Sekulović, Lidija. "TOXICITIES OF TARGETED THERAPY AND IMMUNE-RELATED ADVERSE DRUG REACTIONS OF IMMUNOTHERAPY IN THE TREATMENT OF METASTATIC MELANOMA." In Okrugli sto s međunarodnim učešćem "Melanom". Akademija nauka i umjetnosti Bosne i Hercegovine, 2018. http://dx.doi.org/10.5644/pi2019.180.04.

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Targeted therapy and immunotherapy changed the treatment landscape for metastatic melanoma, which is chemotherapy resistant cancer. In pre-innovation era, the overall survival of patients with metastatic melanoma was 6 months, while today 5-year overall survival rate of 34% (and 50% in good prognostic groups) is evident. However, both treatments have their side effects, and cutaneous are the most frequent. Treating physicians in oncology centres, but also primary care specialists, need to be aware of their spectrum which differs for each class of drug: BRAF inhibitors, MEK inhibitors and immunotherapy with anti-PD1 and anti-CTLA4. While BRAF inhibitors have the most prominent adverse effects which are class specific, there are also drug-specific adverse effects. For example, vemurafenib causes photosensitivity, which is not specific for dabrafenib, while dabrafenib induces pyrexia, that occurs much less frequently with other BRAF inhibitors. Cutaneous rash and cutaneous neoplasms which develop due to paradoxical activation of RAS signalling are described with BRAF inhibitor monotherapy. These side-effects are much less common in combination therapy with BRAF and MEK inhibitor, but MEK inhibitor itself causes characteristic acneiform eruption, and serous retinopathy. Immune related adverse drug reactions are a hallmark of the immune checkpoint inhibitor immunotherapy, which can affect every organ system, and most commonly skin, lungs and gastrointestinal system, with differential frequencies recorded with anti-CTLA4 therapy and anti PD-1 therapy. Skin reactions most frequently include pruritus and eczematous reactions, as well as vitiligo-like hypopigmentation, which is linked Melanom 45 to the better response to treatment. In this review, frequent and rare side effects are presented, as well as the current algorithms for their treatment.
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Gabriela Prandini Nunes Cota, Maria, and Luis Felipe Malaquias Ferreira. "A RELAÇÃO ENTRE A MICROBIOTA INTESTINAL E A OCORRÊNCIA DE NEOPLASIAS DO TRATO GASTROINTESTINAL." In Semana Online Acadêmica de Medicina. Congresse.me, 2022. http://dx.doi.org/10.54265/jnco9736.

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INTRODUÇÃO: A microbiota intestinal se relaciona simbioticamente e desempenha funções essenciais à homeostase humana, sobretudo do ponto de vista imunopatológico, sendo a sua composição influenciada pelo estilo de vida e dieta do indivíduo. Há indícios que o desequilíbrio da microbiota intestinal estaria relacionado ao desenvolvimento de neoplasias do trato gastrointestinal (TGI) OBJETIVO: Identificar a relação entre a microbiota intestinal e a ocorrência de neoplasias do TGI. MÉTODOS: revisão narrativa nas bases PubMed e LILACS com os seguintes descritores: “gastrointestinal tract”, “câncer”, e “microbiota”. RESULTADOS: A carcinogênese de neoplasias do TGI relaciona-se à fatores genéticos, ambientais e comportamentais. O aumento do consumo de produtos industrializados, que alteram a microbiota intestinal, está relacionado a um concomitante aumento da incidência de neoplasias do TGI. Esse efeito relaciona-se à uma diminuição de microrganismos importantes para a homeostase e a proliferação de agentes como H. pylori e Fusobacterium nucleatum. A disbiose gera um ambiente próinflamatório, por meio da maior produção de ácidos graxos de cadeia curtas e toxinas como a 4-hidroxi-2-nonenal, que relaciona-se à desregulação do inflamassoma e dos genes de supressão de células tumorais, predispondo mecanismos carcinogênicos. Do contrário, alguns estudos indicam um possível efeito protetor de microbiotas equilibradas na prevenção e melhor prognóstico dessas neoplasias. CONCLUSÃO: As evidências atuais sugerem um papel protetor de uma microbiota diversificada e equilibrada em prevenir neoplasias TGI, bem como o efeito nocivo da disbiose sobre a carcinogênese. Estudos prospectivos mais abrangentes ainda são necessários para especificar tais efeitos e promover medidas profiláticas e terapêuticas. PALAVRAS-CHAVE: Microbiota, Neoplasias, Trato Gastrointestinal
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Borisova, Ekaterina G., Tsanislava Genova, Ivan Terziev, Borislav Vladimirov, Hristo Valkov, and Oxana Semyachkina-Glushkovskaya. "Autofluorescence detection of lower tract gastrointestinal neoplasia." In Saratov Fall Meeting 2020: Optical and Nano-Technologies for Biology and Medicine, edited by Valery V. Tuchin and Elina A. Genina. SPIE, 2021. http://dx.doi.org/10.1117/12.2589442.

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Borisova, Ekaterina G., Borislav Vladimirov, Ivan Terziev, Radina Ivanova, and Latchezar Avramov. "5-ALA/PpIX fluorescence detection of gastrointestinal neoplasia." In European Conference on Biomedical Optics. OSA, 2009. http://dx.doi.org/10.1364/ecbo.2009.7368_24.

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Borisova, Ekaterina G., Borislav Vladimirov, Ivan Terziev, Radina Ivanova, and Latchezar Avramov. "5-ALA/PpIX fluorescence detection of gastrointestinal neoplasia." In European Conferences on Biomedical Optics, edited by Irene Georgakoudi, Jürgen Popp, and Katarina Svanberg. SPIE, 2009. http://dx.doi.org/10.1117/12.831604.

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Azevedo, Ingrid Nayara Duarte, Caroline De Brito Ferreira, Josiany Souza Lins De Vasconcelos, Maria Beatriz Medeiros Vale Costa, and Yade Farias Nunes. "ALTERAÇÕES CLÍNICAS E LABORATORIAIS EM GATOS ACOMETIDOS POR LINFOMA ALIMENTAR." In I Congresso On-line Nacional de Clínica Veterinária de Pequenos Animais. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1845.

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Introdução: O linfoma é uma neoplasia originária da proliferação clonal de linfócitos malignos frequentemente diagnosticada em felinos, correspondendo a 90% das neoplasias hematopoiéticas nesses animais. São classificadas de acordo com formas anatômicas: multicêntrico, mediastinal, alimentar, cutâneo e de tecidos extranodais (nasal, renal e sistema nervoso). Sendo o linfoma alimentar a forma mais diagnosticada em gatos e que possui crescente incidência nos últimos anos, tornando essencial que o clínico veterinário tenha conhecimento sobre os aspectos diagnósticos em gatos acometidos por essa neoplasia. Objetivo: Descrever as alterações clínicas e laboratoriais encontrados em gatos acometidos por linfoma alimentar. Material e Métodos: A revisão de literatura foi desenvolvida a partir de pesquisas no Google Acadêmico no qual buscou-se diversos artigos atuais referentes ao tema abordado. Resultados: O linfoma alimentar felino não apresenta predileção sexual, raça ou idade, embora a maioria dos gatos acometidos sejam idosos, machos e portadores de doenças imunossupressoras. Essa patologia acomete principalmente o trato gastrointestinal, linfonodos regionais, fígado e baço e se manifesta de duas formas: linfocítico (células pequenas e bem diferenciadas) e linfoblástico (células grandes e pouco diferenciadas), sendo o linfoma linfoblástico a forma mais agressiva. O diagnóstico consiste nos achados clínicos e na realização de exames citológicos e histopatológicos, ultrassonográficos e laboratoriais como o hemograma, a bioquímica e os testes sorológicos para a imunodeficiência felina e a leucemia felina. As principais alterações clínicas consistem na perda de peso, anorexia, êmese, diarreia e desconforto abdominal. As alterações laboratoriais incluem: anemia normocítica normocrômica não regenerativa, leucocitose com aumento de neutrófilos bastonetes, hipercalcemia, hipoalbuminemia e hipoproteinemia e são decorrentes do comprometimento da médula óssea pela neoplasia ou pela destruição celular ou pelo sequestro esplênico. Sendo o prognóstico dependente da resposta inicial à quimioterapia e da ocorrência de remissão. Conclusão: O linfoma alimentar é uma doença frequentemente diagnosticada na medicina felina e que pode levar a uma diminuição na sobrevida desses animais, o que ressalta a importância da determinação do diagnóstico precoce através da identificação dos sinais clínicos iniciais da doença bem como a realização de exames laboratoriais para se fechar um diagnóstico correto, e também para a avaliação do prognóstico.
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