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Journal articles on the topic 'Gemfibrozilo'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Maguiña, Elbis, Fernando Nuevo V., and Jesús Rumiche B. "Análisis costo-efectividad del uso de estatinas frente a gemfibrozilo en pacientes de consultorios externos del Centro Médico Naval: "Cirujano Mayor Santiago Tavara"." Ciencia e Investigación 9, no. 1 (2006): 33–40. http://dx.doi.org/10.15381/ci.v9i1.5090.

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El objetivo de la investigación fue determinar y comparar el costo-efectividad de Atorvastatina 20 mG y Pravastatina 20m mG trente a Gemfibrozilo 600mG, en el tratamiento de hiperlipidemia, e identificar los factores de riesgo. Fue un estudio transversal, descriptivo y retrospectivo, realizado en el Centro Médico Naval, en el período de enero de 2002 a julio de 2005. Se revisaron historias clínicas de 64 pacientes hiperlipidémicos, entre 40 y 64 años, que acuden a los consultorios externos de Cardiología, Endocrinología, Geriatría y Medicina Interna a quienes se les prescribió: Estatinas 20 mg

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2

García Llopis, Paula, Alejandra González D’huicque, and Vicente Palop Larrea. "Edema facial por gemfibrozilo." Medicina Clínica 135, no. 8 (2010): 384. http://dx.doi.org/10.1016/j.medcli.2009.06.040.

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3

Acuña Elvira, Nerea. "Consulta farmacéutica: intoxicación por gemfibrozilo." Farmacéuticos Comunitarios 9, no. 2 (2017): 49–51. http://dx.doi.org/10.5672/fc.2173-9218.(2017/vol9).002.06.

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4

Tormo Molina, J., I. Gázquez Pérez, and A. Matas Hoces. "Cerivastatina y gemfibrozilo: una combinación peligrosa*." Atención Primaria 28, no. 6 (2001): 438–39. http://dx.doi.org/10.1016/s0212-6567(01)70410-6.

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5

de Arriba Méndez, José-Javier, Elena Gómez Merino, José Antonio Sáez Barcelona, and Lourdes Sáez Méndez. "Rabdomiólisis grave asociada a cerivastatina y gemfibrozilo." Medicina Clínica 117, no. 7 (2001): 278–79. http://dx.doi.org/10.1016/s0025-7753(01)72086-6.

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6

Bosch Rovira, T., J. A. Llompart Pou, and J. Forteza-Rey. "Rabdomiólisis asociada al tratamiento combinado con cerivastatina y gemfibrozilo." Revista Clínica Española 201, no. 12 (2001): 731–32. http://dx.doi.org/10.1016/s0014-2565(01)70966-6.

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7

Vascónez Espinosa, F., N. Gómez Rodríguez, A. Martín Joven, and F. J. Posada García. "Rabdomiólisis complicada con insuficiencia renal aguda en un paciente tratado con gemfibrozilo y cerivastatina." Revista Clínica Española 201, no. 4 (2001): 228–29. http://dx.doi.org/10.1016/s0014-2565(01)70806-5.

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8

Alean Flórez, Joel, Daniela Márquez Méndez, Saudith María Burgos Núñez, Germán Enamorado-Montes, and José Marrugo Negrete. "Productos farmacéuticos y de cuidado personal presentes en aguas superficiales, de consumo humano y residuales en el departamento de Córdoba, Colombia." Revista de Investigación Agraria y Ambiental 12, no. 2 (2021): 179–97. http://dx.doi.org/10.22490/21456453.4231.

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Contextualización: gracias al avance de técnicas analíticas sensibles desarrolladas en las últimas décadas, se ha podido identificar y cuantificar la presencia de diversos contaminantes en el medio acuático, dentro de este grupo se encuentran los contaminantes emergentes (CE) conformados por productos de cuidado e higiene personal, antibióticos, hormonas, plastificantes, fármacos y otros que son vertidos a las fuentes de agua alterando el equilibrio de estos ecosistemas.   Vacío de investigación: diversas investigaciones muestran que los contaminantes emergentes pueden comprometer

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9

Jiménez-Bambague, Eliana M., Carlos A. Madera-Parra, and Enrique J. Peña-Salamanca. "Eliminación de compuestos farmacéuticos presentes en el agua residual doméstica mediante un tratamiento primario avanzado." INGENIERÍA Y COMPETITIVIDAD 22, no. 1 (2020): 10. http://dx.doi.org/10.25100/iyc.v22i1.8794.

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Se evaluó la presencia de compuestos farmacéuticos de los grupos terapéuticos antiepilépticos, agentes hipolipemiantes, analgésicos antiinflamatorios y tranquilizantes en tres jornadas de muestreo, además se estimó la eficiencia de eliminación mediante un tratamiento primario avanzado (TPA) con procesos de coagulación, floculación y sedimentación en la planta de tratamiento de agua residual de la ciudad de Cali (Colombia). De los compuestos medidos, 10 estuvieron por encima del límite de detección de la técnica analítica usada: carbamazepina, 10,11-Dihidro-10,11-dihidroxicarbamazepina, gabapen

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10

Martín-Navarro, Juan Antonio, Vladimir Petkov-Stoyanov, María José Gutiérrez-Sánchez, and Luis Pedraza-Cezón. "Fracaso renal agudo por nefritis intersticial aguda con síndrome de Fanconi en relación con metamizol y gemfibrozilo." Nefrología 36, no. 3 (2016): 321–23. http://dx.doi.org/10.1016/j.nefro.2015.06.022.

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11

Rotllan, Noemí, Joan Carles Escolà-Gil, Laura Calpe-Berdiel, and Francisco Blanco Vaca. "Efecto de la expresión de la PTEC, el gemfibrozilo y la rosiglitazona en el transporte inverso de colesterol desde macrófagos a heces in vivo." Clínica e Investigación en Arteriosclerosis 21, no. 5 (2009): 232–39. http://dx.doi.org/10.1016/s0214-9168(09)72685-3.

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12

Estrela, Gabriel Rufino, Adriano Cleis Arruda, Heron Fernandes Vieira Torquato та ін. "Gemfibrozil Induces Anemia, Leukopenia and Reduces Hematopoietic Stem Cells via PPAR-α in Mice". International Journal of Molecular Sciences 21, № 14 (2020): 5050. http://dx.doi.org/10.3390/ijms21145050.

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Hypercholesterolemia, also called high cholesterol, is a form of hyperlipidemia, which may be a consequence of diet, obesity or diabetes. In addition, increased levels of low-density lipoprotein (LDL) and reduced levels of high-density lipoprotein (HDL) cholesterol are associated with a higher risk of atherosclerosis and coronary heart disease. Thus, controlling cholesterol levels is commonly necessary, and fibrates have been used as lipid-lowering drugs. Gemfibrozil is a fibrate that acts via peroxisome proliferator-activated receptor alpha to promote changes in lipid metabolism and decrease

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13

Ni, Yu-Fei, Hao Wang, Qiu-Yan Gu, et al. "Gemfibrozil has antidepressant effects in mice: Involvement of the hippocampal brain-derived neurotrophic factor system." Journal of Psychopharmacology 32, no. 4 (2018): 469–81. http://dx.doi.org/10.1177/0269881118762072.

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Major depressive disorder has become one of the most serious neuropsychiatric disorders worldwide. However, currently available antidepressants used in clinical practice are ineffective for a substantial proportion of patients and always have side effects. Besides being a lipid-regulating agent, gemfibrozil is an agonist of peroxisome proliferator-activated receptor-α (PPAR-α). We investigated the antidepressant effects of gemfibrozil on C57BL/6J mice using the forced swim test (FST) and tail suspension test (TST), as well as the chronic unpredictable mild stress (CUMS) model of depression. Th

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14

&NA;. "Gemfibrozil see Lovastatin/gemfibrozil." Reactions Weekly &NA;, no. 291 (1990): 8. http://dx.doi.org/10.2165/00128415-199002910-00028.

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15

Song, Danjun, Min Luo, Manyun Dai та ін. "PPARα-dependent increase of mouse urine output by gemfibrozil and fenofibrate". Canadian Journal of Physiology and Pharmacology 95, № 2 (2017): 199–205. http://dx.doi.org/10.1139/cjpp-2016-0140.

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While gemfibrozil and fenofibrate are prescribed for anti-dyslipidemia treatment, a rational basis for the use of these drugs for treatment of dyslipidemia with concurrent metabolic syndrome has not been established. In this study, wild-type and Pparα-null mice were fed gemfibrozil- or fenofibrate-containing diets for 14 days. Urine output (24 h) was monitored, and urine, serum, and liver and kidney tissues were subjected to toxicity assessment. A 2-month challenge followed by a 2-week wash-out was performed for gemfibrozil to determine urine output and the potential toxicity. A therapeuticall

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16

Hari, Rosida. "PROFIL KADAR TRIGLISERIDA PASIEN HIPERTRIGLISERIDEMIA SETELAH PENGGUNAAN GEMFIBROZIL 300 mg DI POLIKLINIK PRATAMA POLITEKNIK NEGERI JEMBER." JURNAL ILMIAH FARMASI AKADEMI FARMASI JEMBER 4, no. 1 (2021): 32–37. http://dx.doi.org/10.53864/jifakfar.v4i1.44.

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Penyakit kardiovaskular merupakan penyebab kematian utama didunia dan jumlah kasus penyakit ini telah mengalami peningkatan selama satu decade terakhir. Salah satu penyebabnya adalah hipertrigliserida. Hipertrigliseridemia atau tingginya kadar trigliserida darah merupakan salah satu kelainan fraksi lipid yang berhubungan dengan peningkatan resiko penyakit kardiovaskular Penurunan kadar trigliserida dalam darah dapat dilakukan dengan terapi Gemfibrozil. Gemfibrozil adalah regulator lipid plasma yang dapat menurunkan kadar kolesterol total, kolesterol LDL, kolesterol VLDL dan trigliserida serta

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17

Bröijersén, Anders, Anders Hamsten, Angela Silveira, et al. "Gemfibrozil Reduces Thrombin Generation in Patients with Combined Hyperlipidaemia, without Influencing Plasma Fibrinogen, Fibrin Gel Structure or Coagulation Factor VII." Thrombosis and Haemostasis 76, no. 02 (1996): 171–76. http://dx.doi.org/10.1055/s-0038-1650548.

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SummaryA double-blind, placebo-controlled, cross-over study was conducted in 21 men with combined hyperlipoproteinaemia to examine if lipid-lowering treatment with gemfibrozil (10-12 weeks) affects blood coagulation and fibrin gel structure at rest or during mental stress. Gemfibrozil lowered plasma triglycerides by 57 ± 4%, whereas high density lipoprotein (HDL) cholesterol increased by 22 ± 5%. Gemfibrozil lowered the triglyceride content of low density lipoprotein (LDL). Gemfibrozil reduced the plasma concentrations of thrombin-antithrombin complex (TAT) and prothrombin fragment F1+2 (F1+2)

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18

Andersen, P., P. Smith, I. Seljeflot, S. Brataker, and H. Arnesen. "Effects of Gemfibrozil on Lipids and Haemostasis after Myocardial Infarction." Thrombosis and Haemostasis 63, no. 02 (1990): 174–77. http://dx.doi.org/10.1055/s-0038-1645040.

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SummaryThe effects of gemfibrozil on haemostatic variables were studied in 43 survivors of myocardial infarction with serum triglycerides (TG) ≥2 mmol/1 2 weeks prior to randomization. The study was double-blind, placebo-controlled and stratified for chronic betablockade. Twenty-two individuals were given gemfibrozil 600 mg twice daily and 21 individuals received matching placebo. After 8 weeks the TG level was unchanged in the placebo group, whereas a 44% reduction was noted in the gemfibrozil group (p <0.0001). Fibrinogen increased in both groups, while bleeding time and platelet count we

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19

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 1323 (2010): 23. http://dx.doi.org/10.2165/00128415-201013230-00079.

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20

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 437 (1993): 7. http://dx.doi.org/10.2165/00128415-199304370-00025.

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21

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 443 (1993): 9. http://dx.doi.org/10.2165/00128415-199304430-00036.

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22

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 856 (2001): 9. http://dx.doi.org/10.2165/00128415-200108560-00024.

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23

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 314 (1990): 7. http://dx.doi.org/10.2165/00128415-199003140-00034.

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24

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 317 (1990): 5–6. http://dx.doi.org/10.2165/00128415-199003170-00026.

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25

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 329 (1990): 6. http://dx.doi.org/10.2165/00128415-199003290-00021.

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26

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 331 (1990): 8. http://dx.doi.org/10.2165/00128415-199003310-00039.

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27

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 478 (1993): 8. http://dx.doi.org/10.2165/00128415-199304780-00042.

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28

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 1291 (2010): 26. http://dx.doi.org/10.2165/00128415-201012910-00078.

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29

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 1298 (2010): 18. http://dx.doi.org/10.2165/00128415-201012980-00053.

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30

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 960 (2003): 11. http://dx.doi.org/10.2165/00128415-200309600-00036.

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31

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 991 (2004): 9. http://dx.doi.org/10.2165/00128415-200409910-00024.

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32

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 726 (1998): 8. http://dx.doi.org/10.2165/00128415-199807260-00025.

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33

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 899 (2002): 11. http://dx.doi.org/10.2165/00128415-200208990-00035.

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34

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 914 (2002): 8. http://dx.doi.org/10.2165/00128415-200209140-00025.

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35

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 631 (1996): 9. http://dx.doi.org/10.2165/00128415-199606310-00025.

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36

Todd, Peter A., and Alan Ward. "Gemfibrozil." Drugs 36, no. 3 (1988): 314–39. http://dx.doi.org/10.2165/00003495-198836030-00004.

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37

Spencer, Caroline M., and Lee B. Barradell. "Gemfibrozil." Drugs 51, no. 6 (1996): 982–1018. http://dx.doi.org/10.2165/00003495-199651060-00009.

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38

Ahmad, Saeed. "Gemfibrozil." Southern Medical Journal 84, no. 1 (1991): 102. http://dx.doi.org/10.1097/00007611-199101000-00027.

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39

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 394 (1992): 7. http://dx.doi.org/10.2165/00128415-199203940-00026.

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40

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 374 (1991): 5. http://dx.doi.org/10.2165/00128415-199103740-00019.

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41

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 421 (1992): 8. http://dx.doi.org/10.2165/00128415-199204210-00033.

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42

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 1383 (2012): 21. http://dx.doi.org/10.2165/00128415-201213830-00072.

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43

&NA;. "Gemfibrozil." Reactions Weekly &NA;, no. 1390 (2012): 18–19. http://dx.doi.org/10.2165/00128415-201213900-00067.

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44

Atmaca, Hulusi, Hayriye Sayarlıoğlu, Eyüp Külah, Nejat Demircan, and Tekin Akpolat. "Rhabdomyolysis Associated with Gemfibrozil—Colchicine Therapy." Annals of Pharmacotherapy 36, no. 11 (2002): 1719–21. http://dx.doi.org/10.1345/aph.1c028.

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OBJECTIVE: To report a case of rhabdomyolysis possibly due to combination therapy with colchicine and gemfibrozil. CASE SUMMARY: A 40-year-old man with amyloidosis and hepatitis B virus—related chronic liver disease was admitted to the university hospital because of fatigue, lack of appetite, dark brownish urine, and myalgia for 2 weeks. The patient was receiving colchicine and gemfibrozil. Elevations of serum creatine kinase, lactate dehydrogenase, and aspartate aminotransferase concentrations with myalgia were compatible with the diagnosis of rhabdomyolysis. DISCUSSION: To our knowledge, myo

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45

Werremeyer, Amy B., Ji M. Koo, and Justin M. Welch. "A Comparison of Adverse Effects of Simvastatin plus Gemfibrozil and Atorvastatin plus Gemfibrozil." Hospital Pharmacy 42, no. 7 (2007): 631–36. http://dx.doi.org/10.1310/hpj4207-631.

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Background The manufacturer of simvastatin recommends a dose limitation of 10 mg daily when used in combination with gemfibrozil, due to increased risk of myopathy and rhabdomyolysis. Little information is available regarding the risk of adverse effects of atorvastatin when used in combination with gemfibrozil. Purpose To compare the rate of discontinuation or dose reduction due to adverse effects with simvastatin and gemfibrozil versus atorvastatin and gemfibrozil. Methods Retrospective review of patients taking gemfibrozil in combination with simvastatin 10 mg, simvastatin 80 mg, or atorvast

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46

Cao, Suzanne, NhuChi Dao, Kristina Roloff, and Guillermo Valenzuela. "Pregnancies Complicated by Familial Hypertriglyceridemia: A Case Report." American Journal of Perinatology Reports 08, no. 04 (2018): e362-e364. http://dx.doi.org/10.1055/s-0038-1676832.

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Background Although rare, familial hypertriglyceridemia can cause acute and life-threatening complications in pregnancy. Cases The first patient's pregnancy was complicated by multiple admissions for pancreatitis due to hypertriglyceridemia and noncompliance with gemfibrozil. In her second pregnancy, she was compliant with gemfibrozil and only experienced pancreatitis episodes toward the end of pregnancy. The second patient had diabetes mellitus and familial hypertriglyceridemia. She required multiple hospitalizations for diabetic ketoacidosis secondary to insulin noncompliance. In both pregna

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47

Chakkarwar, Vishal Arvind, and Pravin Kawtikwar. "EFFECT OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-ALPHA-AGONISTS ON DIABETES-INDUCED ACUTE KIDNEY INJURY: ROLE OF OXIDATIVE STRESS AND HYPERLIPIDEMIA." Asian Journal of Pharmaceutical and Clinical Research 12, no. 1 (2019): 143. http://dx.doi.org/10.22159/ajpcr.2018.v12i1.29397.

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Objective: The present study investigated the possible effect of fenofibrate and gemfibrozil peroxisome proliferator-activated receptor-alpha agonist in diabetes-induced acute kidney injury (AKI) in rats.Methods: Rats were administered streptozotocin (STZ) (50 mg/kg, i.p., single dose) to induce experimental diabetes mellitus. The development of diabetic AKI was assessed biochemically and histologically. In addition, the diabetes-induced lipid profile and renal oxidative stress were assessed. The single dose of STZ produced diabetes, which induced renal oxidative stress, altered the lipid prof

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48

Chakkarwar, Vishal Arvind, and Pravin Kawtikwar. "EFFECT OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-ALPHA-AGONISTS ON DIABETES-INDUCED ACUTE KIDNEY INJURY: ROLE OF OXIDATIVE STRESS AND HYPERLIPIDEMIA." Asian Journal of Pharmaceutical and Clinical Research 12, no. 1 (2019): 143. http://dx.doi.org/10.22159/ajpcr.2019.v12i1.29397.

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Objective: The present study investigated the possible effect of fenofibrate and gemfibrozil peroxisome proliferator-activated receptor-alpha agonist in diabetes-induced acute kidney injury (AKI) in rats.Methods: Rats were administered streptozotocin (STZ) (50 mg/kg, i.p., single dose) to induce experimental diabetes mellitus. The development of diabetic AKI was assessed biochemically and histologically. In addition, the diabetes-induced lipid profile and renal oxidative stress were assessed. The single dose of STZ produced diabetes, which induced renal oxidative stress, altered the lipid prof

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49

Wilkes, H. C., T. W. Meade, S. Barzega, et al. "Gemfibrozil Reduces Plasma Prothrombin Fragment F1+2 Concentration, a Marker of Coagulability, in Patients with Coronary Heart Disease." Thrombosis and Haemostasis 67, no. 05 (1992): 503–6. http://dx.doi.org/10.1055/s-0038-1648481.

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SummaryThe effects of gemfibrozil on several indices of haemostatic activity were explored in male patients with coronary heart disease (CHD). Sixty-three of 71 patients completed a crossover study in which gemfibrozil 1,200 mg/day and matching placebo were each taken in randomised order for 2 months in a doubleblind manner, separated by a 2-month washout period. Serum cholesterol decreased by an average (95% confidence interval) of 12 (9 to 15)% and non-fasting triglyceride concentration by 43 (34 to 51)% during active treatment. Plasma prothrombin fragment Fi + 2 concentration, a marker of t

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50

Backes, Jim M., Patrick M. Moriarty, and Cheryl A. Gibson. "The Effect of Micronized Fenofibrate on Lipid Profiles of Patients Converted from Gemfibrozil." Hospital Pharmacy 37, no. 9 (2002): 953–56. http://dx.doi.org/10.1177/001857870203700915.

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Although numerous studies have established the efficacy of micronized fenofibrate (MF) and gemfibrozil in improving lipid profiles, there are limited comparative data on the lipid-lowering effects of these two agents. The objective of this study was to evaluate the mean changes in lipid values of hypertriglyceridemic patients crossed over from gemfibrozil to MF. The Medical charts of 21 patients were analyzed retrospectively. Patients were maintained on gemfibrozil 600 mg twice daily for a minimum of 3 months. The patient's last fasting lipid profile on gemfibrozil was compared to the first li

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