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Relevant bibliographies by topics / Gene expression in C. fusca

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Academic literature on the topic 'Gene expression in C. fusca'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Gene expression in C. fusca"

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Deng, Yu, and Stephen S. Fong. "Development and Application of a PCR-Targeted Gene Disruption Method for Studying CelR Function in Thermobifida fusca." Applied and Environmental Microbiology 76, no. 7 (January 22, 2010): 2098–106. http://dx.doi.org/10.1128/aem.02626-09.

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ABSTRACT Thermobifida fusca is a high-G+C-content, thermophilic, Gram-positive soil actinobacterium with high cellulolytic activity. In T. fusca, CelR is thought to act as the primary regulator of cellulase gene expression by binding to a 14-bp inverted repeat [5′-(T)GGGAGCGCTCCC(A)] that is upstream of many known cellulase genes. Previously, the ability to study the roles and regulation of cellulase genes in T. fusca has been limited largely by a lack of established genetic engineering methods for T. fusca. In this study, we developed an efficient procedure for creating precise chromosomal gene disruptions and demonstrated this procedure by generating a celR deletion strain. The celR deletion strain was then characterized using measurements for growth behavior, cellulase activity, and gene expression. The celR deletion strain of T. fusca exhibited a severely crippled growth phenotype with a prolonged lag phase and decreased cell yields for growth on both glucose and cellobiose. While the maximum endoglucanase activity and cellulase activity were not significantly changed, the endoglucanase activity and cellulase activity per cell were highly elevated. Measurements of mRNA transcript levels in both the celR deletion strain and the wild-type strain indicated that the CelR protein potentially acts as a repressor for some genes and as an activator for other genes. Overall, we established and demonstrated a method for manipulating chromosomal DNA in T. fusca that can be used to study the cellulolytic capabilities of this organism. Components of this method may be useful in developing genetic engineering methods for other currently intractable organisms.

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Wei, Yu-Tuo, Qi-Xia Zhu, Zhao-Fei Luo, Fu-Shen Lu, Fa-Zhong Chen, Qing-Yan Wang, Kun Huang, Jian-Zhong Meng, Rong Wang, and Ri-Bo Huang. "Cloning, Expression and Identification of a New Trehalose Synthase Gene from Thermobifida fusca Genome." Acta Biochimica et Biophysica Sinica 36, no. 7 (July 1, 2004): 477–84. http://dx.doi.org/10.1093/abbs/36.7.477.

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Abstract A new open reading frame in Thermobifida fusca sequenced genome was identified to encode a new trehalose synthase, annotated as “glycosidase” in the GenBank database, by bioinformatics searching and experimental validation. The gene had a length of 1830 bp with about 65% GC content and encoded for a new trehalose synthase with 610 amino acids and deduced molecular weight of 66 kD. The high GC content seemed not to affect its good expression in E. coli BL21 in which the target protein could account for as high as 15% of the total cell proteins. The recombinant enzyme showed its optimal activities at 25 °C and pH 6.5 when it converted substrate maltose into trehalose. However it would divert a high proportion of its substrate into glucose when the temperature was increased to 37 °C, or when the enzyme concentration was high Its activity was not inhibited by 5 mM heavy metals such as Cu2+, Mn2+, and Zn2+ but affected by high concentration of glucose. Blasting against the database indicated that amino acid sequence of this protein had maximal 69% homology with the known trehalose synthases, and two highly conserved segments of the protein sequence were identified and their possible linkage with functions was discussed.

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Béki, Emese, István Nagy, Jos Vanderleyden, Szilvia Jäger, László Kiss, László Fülöp, László Hornok, and József Kukolya. "Cloning and Heterologous Expression of a β-d-Mannosidase (EC 3.2.1.25)-Encoding Gene from Thermobifida fusca TM51." Applied and Environmental Microbiology 69, no. 4 (April 2003): 1944–52. http://dx.doi.org/10.1128/aem.69.4.1944-1952.2003.

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ABSTRACT Thermobifida fusca TM51, a thermophilic actinomycete isolated from composted horse manure, was found to produce a number of lignocellulose-degrading hydrolases, including endoglucanases, exoglucanases, endoxylanases, β-xylosidases, endomannanases, and β-mannosidases, when grown on cellulose or hemicellulose as carbon sources. β-Mannosidases (EC 3.2.1.25), although contributing to the hydrolysis of hemicellulose fractions, such as galacto-mannans, constitute a lesser-known group of the lytic enzyme systems due to their low representation in the proteins secreted by hemicellulolytic microorganisms. An expression library of T. fusca, prepared in Streptomyces lividans TK24, was screened for β-mannosidase activity to clone genes coding for mannosidases. One positive clone was identified, and a β-mannosidase-encoding gene (manB) was isolated. Sequence analysis of the deduced amino acid sequence of the putative ManB protein revealed substantial similarity to known mannosidases in family 2 of the glycosyl hydrolase enzymes. The calculated molecular mass of the predicted protein was 94 kDa, with an estimated pI of 4.87. S. lividans was used as heterologous expression host for the putative β-mannosidase gene of T. fusca. The purified gene product obtained from the culture filtrate of S. lividans was then subjected to more-detailed biochemical analysis. Temperature and pH optima of the recombinant enzyme were 53°C and 7.17, respectively. Substrate specificity tests revealed that the enzyme exerts only β-d-mannosidase activity. Its kinetic parameters, determined on para-nitrophenyl β-d-mannopyranoside (pNP-βM) substrate were as follows: Km = 180 μM and V max = 5.96 μmol min−1 mg−1; the inhibition constant for mannose was Ki = 5.5 mM. Glucono-lacton had no effect on the enzyme activity. A moderate trans-glycosidase activity was also observed when the enzyme was incubated in the presence of pNP-αM and pNP-βM; under these conditions mannosyl groups were transferred by the enzyme from pNP-βM to pNP-αM resulting in the synthesis of small amounts (1 to 2%) of disaccharides.

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4

Morandin, C., A. Hietala, and H. Helanterä. "Vitellogenin and vitellogenin-like gene expression patterns in relation to caste and task in the ant Formica fusca." Insectes Sociaux 66, no. 4 (September 25, 2019): 519–31. http://dx.doi.org/10.1007/s00040-019-00725-9.

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Abstract Social insect colonies are characterized by division of labour, and extensive morphological, physiological and behavioural differences between queens and workers. The storage protein vitellogenin (Vg) affects multiple aspects of social insect life histories, and has been suggested as a key player for caste differentiation and maintenance. Recently, three genes homologous to Vg have been described in the ant Formica exsecta. Their role is currently unclear but their structural variation suggests variable functions. We examined the expression patterns of the conventional Vg and the three Vg-like genes using qRT-PCR in the common black ant Formica fusca between queens and workers, between nurse and foragers workers, and across social contexts (queenless vs. queenright nests), and sampling time. As expected, we found a significant queen caste and nurse task-related increase for the conventional Vg, while Vg-like-C displayed a consistent forager-biased expression pattern. Task (forager vs. nurse) was the only factor that explained expression variation among workers in any of the studied genes. The removal of the queen did not affect expression, although the proportion of fertile nurses increased in queenless nests. The observed expression biases suggest that in Formica fusca, the ancestral duplication has led to alternative social functions for Vg-like genes across castes and tasks. To get a broader picture of the role of gene duplications in social evolution and the roles of Vg-like genes in caste differentiation and maintenance, how these genes achieve these roles at a molecular level need to be investigated further.

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Chen, Wei-Lin, Jo-Chieh Hsu, Chui-Li Lim, Cheng-Yu Chen, and Chao-Hsun Yang. "Expression of the Thermobifida fusca β-1,3-Glucanase in Yarrowia lipolytica and Its Application in Hydrolysis of β-1,3-Glucan from Four Kinds of Polyporaceae." Processes 9, no. 1 (December 29, 2020): 56. http://dx.doi.org/10.3390/pr9010056.

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The gene encoding a thermostable β-1,3-glucanase was cloned from Thermobifida fusca and expressed constitutively by Yarrowia lipolytica using plasmid pYLSC1. The expression level of the recombinant β-1,3-glucanase reached up to 270 U/mL in the culture medium. After a treatment with endo-β-N-acetyl-glucosaminidase H, the recombinant protein appeared as a single protein band, with a molecular size of approximately 66 kDa on the SDS-polyacrylamide gel. The molecular weight was consistent with the size predicted from the nucleotide sequence. The optimum temperature and pH of the transformant β-1,3-glucanase were 60 °C and pH 8.0, respectively. This β-1,3-glucanase was tolerant to 10% methanol, ethanol, and DMSO, retaining 70% activity. The enzyme markedly hydrolyzed Wolfiporia cocos and Pycnoporus sanguineus glucans. The DPPH and ABTS scavenging potential, reducing power and total phenolic contents of these two Polyporaceae hydrolysates, were significantly increased after 18 h of the enzymatic reaction. The present results indicate that T. fusca β-1,3-glucanase from Y. lipolytica transformant (pYLSC1-13g) hydrolyzes W. cocos and P. sanguineus glucans and improves the antioxidant potential of the hydrolysates.

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Vigliotta, Giovanni, Eliana Nutricati, Elisabetta Carata, Salvatore M. Tredici, Mario De Stefano, Paola Pontieri, Domenica Rita Massardo, Maria Vittoria Prati, Luigi De Bellis, and Pietro Alifano. "Clonothrix fusca Roze 1896, a Filamentous, Sheathed, Methanotrophic γ-Proteobacterium." Applied and Environmental Microbiology 73, no. 11 (April 6, 2007): 3556–65. http://dx.doi.org/10.1128/aem.02678-06.

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ABSTRACT Crenothrix polyspora Cohn 1870 and Clonothrix fusca Roze 1896 are two filamentous, sheathed microorganisms exhibiting complex morphological differentiation, whose phylogeny and physiology have been obscure for a long time due to the inability to cultivate them. Very recently, DNA sequencing data from uncultured C. polyspora-enriched material have suggested that Crenothrix is a methane-oxidizing γ-proteobacterium (39). In contrast, the possible ecological function of C. fusca, originally considered a developmental stage of C. polyspora, is unknown. In this study, temporal succession of two filamentous, sheathed microorganisms resembling Cohn's Crenothrix and Roze's Clonothrix was observed by analyzing the microbial community of an artesian well by optical microscopy. Combined culture-based and culture-independent approaches enabled us to assign C. fusca to a novel subgroup of methane-oxidizing γ-proteobacteria distinct from that of C. polyspora. This assignment was supported by (i) methane uptake and assimilation experiments, (ii) ultrastructural data showing the presence in C. fusca cytoplasm of an elaborate membrane system resembling that of methanotrophic γ-proteobacteria, and (iii) sequencing data demonstrating the presence in its genome of a methanol dehydrogenase α subunit-encoding gene (mxaF) and a conventional particulate methane mono-oxygenase α subunit-encoding gene (pmoA) that is different from the unusual pmoA (u-pmoA) of C. polyspora.

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Yang, Chao-Hsun, Kun-I. Lin, Gen-Hung Chen, Yu-Fen Chen, Cheng-Yu Chen, Wei-Lin Chen, and Yu-Chun Huang. "Constitutive Expression of Thermobifida fusca Thermostable Acetylxylan Esterase Gene in Pichia pastoris." International Journal of Molecular Sciences 11, no. 12 (December 15, 2010): 5143–51. http://dx.doi.org/10.3390/ijms11125143.

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Nayak, Kinshuk C. "Mutational Bias and Gene Expression Level Shape Codon Usage in Thermobifida fusca YX." In Silico Biology 9, no. 5,6 (2009): 337–53. http://dx.doi.org/10.3233/isb-2009-0421.

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9

Ghangas, Gurdev S., and David B. Wilson. "Expression of a Thermomonospora fusca Cellulase Gene in Streptomyces lividans and Bacillus subtilis." Applied and Environmental Microbiology 53, no. 7 (1987): 1470–75. http://dx.doi.org/10.1128/aem.53.7.1470-1475.1987.

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Lao, G., and D. B. Wilson. "Cloning, sequencing, and expression of a Thermomonospora fusca protease gene in Streptomyces lividans." Applied and environmental microbiology 62, no. 11 (1996): 4256–59. http://dx.doi.org/10.1128/aem.62.11.4256-4259.1996.

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Dissertations / Theses on the topic "Gene expression in C. fusca"

1

Urwin, Nigel A. R. "Studies on the structure and expression of the isocitrate lyase gene of Chlorella fusca." Thesis, University of the West of Scotland, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254324.

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Sasmono, R. Tedjo. "Transcriptional regulation of c-fms gene expression /." [St. Lucia, Qld.], 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17479.pdf.

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Thirumalai, Avinash N. "Regulation of C-reactive Protein Gene Expression and Function." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etd/2467.

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Human C-reactive protein (CRP) is the prototypic acute phase protein whose serum concentration increases rapidly during inflammation. CRP is also associated with atherosclerosis; it is deposited at lesion sites where it may interact with modified lipoproteins. There are 2 major questions regarding CRP: 1. How is the serum concentration of CRP regulated? 2. What are the functions of CRP in atherosclerosis? Our first aim was to determine the role of the constitutively expressed transcription factor Oct-1 in regulating CRP gene expression. We found that Oct-1 overexpression inhibited (IL-6+IL-1β)- induced CRP gene expression; maximal inhibition required the binding of Oct-1 to an octamer motif at (-59 to -66) on the CRP promoter. Oct-1 overexpression inhibited both (IL-6+IL-1β)- induced and C/EBPβ-induced CRP gene expression even when the Oct-1 site was deleted. These findings suggest that Oct-1 is a repressor of CRP gene expression that acts via binding to its cognate site on the CRP promoter as well as through indirect interactions with other promoterbound transcription factors. Our second aim was to investigate the interaction of CRP with oxidized low density lipoprotein (ox-LDL). Acidic pH, a hallmark of atherosclerotic lesions, reversibly alters CRP structure and exposes a hidden binding site that enables CRP to bind ox-LDL. Using site-directed mutagenesis we constructed a CRP mutant (E42Q) that showed significant binding to ox-LDL at physiological pH. E42Q CRP required a less acidic pH for maximal binding and bound ox-LDL more efficiently than wild type CRP at any pH. We then examined if reactive oxygen species also induced CRP – ox-LDL interaction. H2O2-treated CRP bound ox-LDL at physiological pH. Like acidic pH, H2O2-treatment induced only a local structural change exposing the ox-LDL binding site. E42Q and H2O2-modified CRP are tools to study the function of CRP in animal models of atherosclerosis, which may not have an inflammatory environment sufficient to modify CRP and induce binding to atherogenic ox-LDL. We conclude that Oct-1 is one of the critical regulators of CRP gene expression, and that CRP can be modified in vitro to convert it into an atherogenic LDL-binding molecule.

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John, Christopher Robert. "Gene expression associated with the evolution of C₄ photosynthesis." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709401.

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Voleti, Bhavya. "Mechanism of Transcriptional Regulation of C-Reactive Protein Gene Expression." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etd/2058.

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C-reactive protein (CRP) is an acute phase protein produced by hepatocytes whose serum concentration increases in inflammatory conditions including cardiovascular complications. Statins that are used in the treatment of cardiovascular diseases to reduce cholesterol also lower serum CRP levels. In human hepatoma Hep3B cells, CRP is induced in response to cytokines IL-6 and IL-1β. The objective of the study was to determine the mechanism of regulation of CRP gene expression in Hep3B cells in response to cytokines and to determine the effect of statins on CRP expression. Key findings of our research were: 1. IL-1β-activated NF-κB p50/p65 acted synergistically with IL-6-activated C/EBPβ in inducing CRP transactivation through the proximal CRP promoter. 2. A NF-κB site was localized in the proximal CRP promoter centered at position -69 overlapping the known OCT-1/HNF-1/HNF-3 sites. 3. The synergy between IL-6 and IL-1β in inducing CRP gene expression was partially mediated through the NF-κB site. 4. In the absence of C/EBPβ, a complex containing C/EBPζ and RBP-Jκ was formed at the C/EBP-p50-site. 5. Overexpressed C/EBPζ repressed both (IL-6+IL-1β)-induced and C/EBPβ-induced CRP expression. 6. OCT-1 repressed (IL-6+IL-1β)-induced CRP transactivation through the proximal CRP promoter. 7. Statins reduce cytokine-induced CRP gene expression at the transcriptional level. These findings led us to conclude that: 1. CRP transcription is determined by the relative levels of various transcription factors such as C/EBPβ, C/EBPζ, NF-κB and OCT-1 and their interaction with the proximal CRP promoter. 2. Inhibition of CRP transcription by statins is not due to an anti-inflammatory effect but due to the direct effect on CRP gene expression.

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Hachicha, Mohamed. "Regulation of C-C and C-X-C chemokine gene expression in synovial fibroblasts and peripheral blood neutrophils." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1996. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ25241.pdf.

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Dörr, Katrin Zaragoza. "Molecular mechanisms of gene activation and gene expression mediated by CCAAT/enhancer binding proteins." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2008. http://dx.doi.org/10.18452/15873.

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Der Transkriptionsfaktor CCAAT/Enhancer-Binding Protein alpha (C/EBPa) koordiniert Proliferationshemmung und Differenzierung von myeloiden VorlŠuferzellen und Adipozyten. C/EBPa ist ein transkriptioneller Aktivator von abstammungspezifischen Genen und blockiert den Zellzyklus durch Repression von proliferationsfšrdernden E2F Zielgenen. Die hier gezeigten Daten zeigen, dass auch umgekehrt E2F die transkriptionelle und differenzierungsfšrdernde AktivitŠt von C/EBPa entgegenwirkt. Somit besitzen E2F-C/EBPa eine zentrale Schalterfunktion zwischen Proliferation und Differenzierung. Der Repressionsmechanismus durch E2F ist in mehreren Aspekten neuartig: Zum erstenmal wurde gezeigt, dass E2F einen anderen Transkriptionsfaktor reprimieren kann. E2F reprimiert die transkriptionelle AktivitŠt von C/EBPa ohne Bindung an cis-regulatorischen Elemente, sondern durch direkte Protein-Protein Interaktionen, die die Bindung von C/EBPa an DNA verhindern. Diese Form der transkriptionellen Repression geschieht unabhŠngig von "Pocket-Proteinen''". Patienten mit Akuter Myeloiden LeukŠmie (AML) weisen hŠufig eine gestšrte DNA Bindung von C/EBPa auf, welche ursachlich fŸr granulozitŠren Funktionsstšrungen sein kšnnte. Daher wŠre es wichtig zu analysieren ob E2F die DNA Bindung von C/EBPa in AML Patienten beeintrŠchtigt und ob auf E2F gerichtete Therapien granulozitŠre Reifung wiederherstellen. C/EBPa blockiert Zellproliferation durch vielseitigen Mechanismen. Hier wurde gezeigt, dass C/EBPa mit UBF1, dem Co-Aktivator der RNA Polymerase I, an chromosomalen Foci positioniert wird. Eine €hnlichkeit zu anderen fokalen Strukturen suggeriert, dass C/EBPa die Transkription von Polymerase I regulierten rRNA Gene reprimieren und somit ribosomale Biogenese beeintrŠchtigen kšnnte. Die Assoziation zwischen C/EBPa und UBF1 wird durch die Histon-Methyltransferase SUV39H1 stimuliert. Demnach kšnnte die antiproliferative Funktion von C/EBPa nicht nur auf der Regulierung von RNA Pol II-abhŠngiger Transkription, sondern auch auf der Repression von RNA Pol I regulierter rRNA Synthese basieren.
The transcription factor CCAAT/Enhancer-Binding Protein alpha (C/EBPa) coordinates proliferation arrest and differentiation of myeloid progenitors and adipocytes. C/EBPa acts as a transcriptional activator of lineage specific genes and blocks the cell cycle by repressing transcription of E2F-regulated genes. Data presented here suggest that also inversely E2F interferes with the transcriptional activity of C/EBPa, counteracting C/EBPa-mediated differentiation processes. Thus, E2F-C/EBPa are part of a switch mechanism between proliferation and differentiation. The mechanism by which E2F suppresses C/EBPa-mediated transactivation is novel in several aspects. E2F acts as a co-repressor of another transcription factor, C/EBPa, without binding to cis-regulatory elements, but by direct protein-protein interactions that abolish the binding of C/EBPa to DNA. This mechanism of transcriptional repression occurs independent of pocket proteins. Disturbed DNA binding of C/EBPa is often observed in AML patients suggesting a causative role in granulocytic disorders. Thus, it would be of main interest to analyze whether E2F mediates disruption of C/EBPa''s DNA-binding in AML patients and whether therapies directed against E2F could restore granulocytic maturation. Despite the extensive knowledge of mechanisms involved in the inhibitory function of C/EBPa, it has not been addressed whether C/EBPa may impinge on cell proliferation by affecting the ribosomal biogenesis of a cell. This work demonstrates an association of C/EBPa to the RNA Pol I transcription factor UBF1, both proteins retained in large chromosomal foci. Similarities to other focal structures associated to UBF1, suggest that C/EBPa may repress transcription of Pol I-transcribed rRNA genes, and thus affect ribosomal biogenesis. The enrichment of C/EBPa at sites of UBF1 is induced by the histone methyltransferase SUV39H1. Thus, C/EBPa may not only control lineage commitment and cell proliferation by regulating genes transcribed by RNA Pol II, but also may act as a repressor of RNA Pol I mediated rRNA synthesis.

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David, John M. "The control of apolipoprotein C-I gene expression during adipocyte differentiation." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file 4.92 Mb., p, 2006. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:1435920.

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Crossland, Nicola. "Expression of the 5-HTâ†2â†c receptor gene in schizophrenia." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393476.

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Xue, Lin. "A VISUALIZATION TOOL FOR CROSS-EXPERIMENT GENE EXPRESSION ANALYSIS OF C. ELEGANS." UKnowledge, 2007. http://uknowledge.uky.edu/gradschool_theses/472.

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Forty-six genomic gene expression studies of free living soil nematode C. eleganshave been published. To facilitate exploratory analysis of those studies, we constructed adatabase containing all the published C. elegans expression datasets. A Perl CGIprogram, called Microarray Analysis Display (MAdisplay), allows gene expressionclustergrams of any combination of entered genes and datasets to be viewed(http://elegans.uky.edu/gl/madisplay). Perl programs were used to preprocess the rawdata from different sources into a common format and to transform the data to displaythe expression changes relative to each experiment's controls. Three hundred lists ofgenes from figures and tables were extracted from the publications and made available inthe GeneLists database, which also contains Gene Ontology and KEGG gene lists. Weused these tools to examine in a systematic fashion the mean expression of gene lists inthe set of microarray and SAGE experiments. Seventy-nine percent of publicationderived gene lists show a strong expression change (p-value andlt;0.001) in more than oneexperiment with the median being fourteen out of the 127 experiments that are derivedfrom the forty-six publications. This indicates that groups of genes identified in onepublication typically show an expression effect in many other experiments.

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Books on the topic "Gene expression in C. fusca"

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Radzio, Renate. Heterologe Genexpression in dem Cephalosporin C produzierenden Hyphenpilz Acremonium chrysogenum. Berlin: J. Cramer, 1997.

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Krasnoshtein, Flora. Analysis of the pattern of expression of the fanconi anemia group C (Fac) gene during murine development. Ottawa: National Library of Canada, 1995.

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The nonlinear workbook: Chaos, fractals, cellular automata, genetic algorithms, gene expression programming, support vector machine, wavelets, hidden Markov models, fuzzy logic with C++, Java and symbolic C++ programs. 6th ed. Hackensack, New Jersey: World Scientific, 2015.

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The nonlinear workbook: Chaos, fractals, cellular automata, neural networks, genetic algorithms, gene expression programming, support vector machine, wavelets, hidden Markov models, Fuzzy logic with C++, Java and SymbolicC++ programs. 5th ed. New Jersey: World Scientific, 2011.

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The nonlinear workbook: Chaos, fractals, cellular automata, neural networks, genetic algorithms, gene expression programming, support vector machine, wavelets, hidden Markov models, Fuzzy logic with C++, Java and SymbolicC++ programs. 3rd ed. Hackensack, NJ: World Scientific, 2005.

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The nonlinear workbook: Chaos, fractals, cellular automata, neural networks, genetic algorithms, gene expression programming, support vector machine, wavelets, hidden Markov models, Fuzzy logic with C++, Java and SymbolicC++ programs. 5th ed. New Jersey: World Scientific, 2011.

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The nonlinear workbook: Chaos, fractals, cellular automata, neural networks, genetic algorithms, gene expression programming, support vector machine, wavelets, hidden Markov models, Fuzzy logic with C++, Java and SymbolicC++ programs. 4th ed. New Jersey: World Scientific, 2008.

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Li, Fugen. Rainbow trout cystatin C: Gene expression, heterologous production and characterization. 1998.

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Heterologe Gene Expression in Dem Cephalosporin C Produzierenden Hyphenpilz Acremonium Chrysogenum (Bibliotheca Mycologica). Gebruder Borntraeger Verlagsbuchhandlung, 1997.

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(Editor), John N. Abelson, Melvin I. Simon (Editor), and P. Michael Conn (Editor), eds. Methods in Enzymology, Volume 294: Ion Channels, Part C (Methods in Enzymology). Academic Press, 1998.

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Book chapters on the topic "Gene expression in C. fusca"

1

Lanctôt, Christian, and Peter Meister. "Microscopic Analysis of Chromatin Localization and Dynamics in C. elegans." In Imaging Gene Expression, 153–72. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-526-2_11.

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Cahill, Michael A., Ralf Janknecht, and Alfred Nordheim. "Signal uptake by the c-fos serum response element." In Inducible Gene Expression, Volume 2, 39–72. Boston, MA: Birkhäuser Boston, 1995. http://dx.doi.org/10.1007/978-1-4684-6837-3_2.

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Duarte, T. L., and I. F. Almeida. "Vitamin C, gene expression and skin health." In Handbook of diet, nutrition and the skin, 114–27. Wageningen: Wageningen Academic Publishers, 2012. http://dx.doi.org/10.3920/978-90-8686-729-5_7.

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Sotiroudis, Theodore G., Symeon M. Kyriakidis, Leonidas G. Baltas, Vasilis G. Zevgolis, and Athanasios E. Evangelopoulos. "Ca2+-Calmodulin-Dependent Protein Kinases and Protein Kinase C: Functional Similarities." In Evolutionary Tinkering in Gene Expression, 59–69. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5664-6_6.

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Cantrell, D. A., A. A. Davies, G. Krissansen, and M. J. Crumpton. "Interactions between protein kinase C and the T3/T cell antigen receptor complex." In Regulation of Immune Gene Expression, 119–33. Totowa, NJ: Humana Press, 1986. http://dx.doi.org/10.1007/978-1-4612-5014-2_11.

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Tripp, Vanessa, and Lennart Randau. "Evolution of C/D Box sRNAs." In RNA Metabolism and Gene Expression in Archaea, 201–24. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-65795-0_9.

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Belasco, Joel G., Ann-Bin Shyu, and Michael E. Greenberg. "Rapid Degradation of the c-FOS Proto-Oncogene Transcript." In Post-Transcriptional Control of Gene Expression, 65–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75139-4_7.

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Van Beveren, Charles, Richard L. Mitchell, Cynthia Henning-Chubb, Eliezer Huberman, and Inder M. Verma. "Expression of the C-Fos Gene During Differentiation." In Mechanisms of Lymphocyte Activation and Immune Regulation, 263–74. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5323-2_26.

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Onoda, Naoyoshi, Kiyoshi Maeda, Itsuo Nakanishi, Yasuyuki Kondo, Nobuya Yamada, Yoshito Yamashita, Yong-Suk Chung, and Michio Sowa. "c-myc Gene Expression in Primary Gastric Cancer." In Recent Advances in Management of Digestive Cancers, 242–44. Tokyo: Springer Japan, 1993. http://dx.doi.org/10.1007/978-4-431-68252-3_55.

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Sherman, Fred, Richard P. Moerschell, Susumu Tsunasawa, Rolf Sternglanz, and Mark E. Dumont. "Co- and Posttranslational Processes and Mitochondrial Import of Yeast Cytochrome c." In Translational Regulation of Gene Expression 2, 117–41. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2894-4_6.

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Conference papers on the topic "Gene expression in C. fusca"

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Wang, Yu, and Maia Angelova. "Weighted Kernel Fuzzy C-Means Method for Gene Expression Analysis." In 2012 Spring Congress on Engineering and Technology (S-CET). IEEE, 2012. http://dx.doi.org/10.1109/scet.2012.6342018.

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Zhang, Mingrui, Beya Adamu, Chi-Cheng Lin, and Ping Yang. "Gene expression analysis with integrated fuzzy C-means and pathway analysis." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6090211.

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Finigan, JH, A. Boueiz, R. Damico, HH Pae, DN Grigoryev, C. Cheadle, and PM Hassoun. "Gene Expression Changes Mediated by Activated Protein C in Ventilator Associated Lung Injury." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5551.

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Suzuki, Motoshi, Yasuhiro Kamei, Shunsuke Yuba, and Shin Takagi. "A technique enabling the controlled gene expression in targeted single cells of C. elegans." In 2006 IEEE International Symposium on MicroNanoMechanical and Human Science. IEEE, 2006. http://dx.doi.org/10.1109/mhs.2006.320244.

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Novianti, Titta, Febriana Wahyuni, It Jamilah, and Syafruddin Ilyas. "The Peak of Cytochrome-c (Cyt-c) Gene Expression in Inflammatory Stage after Amputation of Digit Tip Mice (Mus musculus)." In 1st International Conference on Health. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009566300780082.

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"Mining Association Rules that Incorporate Transcription Factor Binding Sites and Gene Expression Patterns in C. elegans." In International Conference on Bioinformatics Models, Methods and Algorithms. SciTePress - Science and and Technology Publications, 2013. http://dx.doi.org/10.5220/0004252300810089.

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Stur, Elaine, Shelia Thomas, Francine Rezzoug, and Donald Miller. "Abstract 1520: Down-regulation of c-MYC and hTERT gene expression in triple negative breast cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1520.

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Tao, Yongguang, Shuang Liu, and Yiqun Jiang. "Abstract 4317: EGLN1/c-Myc induced lymphoid-specific helicase inhibits ferroptosis through lipid metabolic gene expression changes." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-4317.

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Chen, Ben-Kuen, and Wan-Chen Huang. "Abstract LB-19: Involvement of aryl hydrocarbon receptor nuclear translocator in EGF-induced c-Jun/Sp1-mediated gene expression." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-19.

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Roos, AB, JL Barton, L. Didon, and M. Nord. "A Role for C/EBPβ in theIn VivoEffects of Budesonide and Formoterol on Inflammatory and Host-Defense Gene Expression." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5087.

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Reports on the topic "Gene expression in C. fusca"

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Melkoumian, Zaroui. Regulation of C-myc Gene Expression by Potassium Channel Blocker Quindine in MCF-7 Human Breast Cancer Cell Line. Fort Belvoir, VA: Defense Technical Information Center, July 2000. http://dx.doi.org/10.21236/ada384096.

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Horio, M., F. R. Collart, and E. Huberman. Heterogeneity in c-jun gene expression in normal and malignant cells exposed to either ionizing radiation or hydrogen peroxide. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/10173201.

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Ye, Shanli. DNA Sequences Involved in the Regulation of Human c-myc Gene Expression by Herpes Simplex Virus Type 1 (HSV-1). Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7097.

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