Relevant bibliographies by topics / Gene mrs

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Gene mrs'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Gene mrs"

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Hwang, Seung Rim, Sook Hee Ku, Min Kyung Joo, Sun Hwa Kim, and Ick Chan Kwon. "Theranostic nanomaterials for image-guided gene therapy." MRS Bulletin 39, no. 1 (January 2014): 44–50. http://dx.doi.org/10.1557/mrs.2013.312.

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Bawazer, Lukmaan A. "Bio Focus: Building synthetic organelles from the gene up." MRS Bulletin 40, no. 1 (January 2015): 9. http://dx.doi.org/10.1557/mrs.2014.315.

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Nakai, T., R. Ishima, H. Sakahara, K. Endo, J. Konishi, and K. Akasaka. "An in vitro1H-MRS Model of Oncogene Transfection." Acta Radiologica 38, no. 6 (November 1997): 1083–86. http://dx.doi.org/10.1080/02841859709172136.

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Purpose: Malignancy is an abnormality of cell division and differentiation based on abnormal expression of oncogenes. This note describes the in vitro1H-NMR spectral features of oncogene-transfected NIH3T3 fibroblast cells compared to non-transfected cells Material and Methods: 1H-NMR spectra of cultured NIH3T3 cells and c-erbB-2 or c-Ha-ras gene-transfected cells were obtained by 400 MHz high resolution NMR. the peaks were assigned by 2D HOHAHA spectra of the cell suspension and the spectral changes were evaluated in 1D and 1D differential spectra Results: the 1H spectra obtained from both transfected cell lines were broadened over all peaks, suggesting reduced mobility in plasma membrane lipid molecules. No other differential spectra for characterizing metabolic change was detected Conclusion: Broadened 1H spectra observed after c-erbB-2 or c-Ha-ras transfection suggest changes of plasma membrane viscosity, which may be related to the oncogene expression
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Mifsud, Karen R., and Johannes M. H. M. Reul. "Acute stress enhances heterodimerization and binding of corticosteroid receptors at glucocorticoid target genes in the hippocampus." Proceedings of the National Academy of Sciences 113, no. 40 (September 21, 2016): 11336–41. http://dx.doi.org/10.1073/pnas.1605246113.

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A stressful event results in secretion of glucocorticoid hormones, which bind to mineralocorticoid receptors (MRs) and glucocorticoid receptors (GRs) in the hippocampus to regulate cognitive and affective responses to the challenge. MRs are already highly occupied by low glucocorticoid levels under baseline conditions, whereas GRs only become substantially occupied by stress- or circadian-driven glucocorticoid levels. Currently, however, the binding of MRs and GRs to glucocorticoid-responsive elements (GREs) within hippocampal glucocorticoid target genes under such physiological conditions in vivo is unknown. We found that forced swim (FS) stress evoked increased hippocampal RNA expression levels of the glucocorticoid-responsive genes FK506-binding protein 5 (Fkbp5), Period 1 (Per1), and serum- and glucocorticoid-inducible kinase 1 (Sgk1). Chromatin immunoprecipitation (ChIP) analysis showed that this stressor caused substantial gene-dependent increases in GR binding and surprisingly, also MR binding to GREs within these genes. Different acute challenges, including novelty, restraint, and FS stress, produced distinct glucocorticoid responses but resulted in largely similar MR and GR binding to GREs. Sequential and tandem ChIP analyses showed that, after FS stress, MRs and GRs bind concomitantly to the same GRE sites within Fkbp5 and Per1 but not Sgk1. Thus, after stress, MRs and GRs seem to bind to GREs as homo- and/or heterodimers in a gene-dependent manner. MR binding to GREs at baseline seems to be restricted, whereas after stress, GR binding may facilitate cobinding of MR. This study reveals that the interaction of MRs and GRs with GREs within the genome constitutes an additional level of complexity in hippocampal glucocorticoid action beyond expectancies based on ligand–receptor interactions.
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Shen, Yao, Gabriel Chan, Michael Xie, Wangyong Zeng, and Liang Liu. "Identification of master regulator genes of UV response and their implications for skin carcinogenesis." Carcinogenesis 40, no. 5 (November 19, 2018): 687–94. http://dx.doi.org/10.1093/carcin/bgy168.

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AbstractSolar UV radiation is a major environmental risk factor for skin cancer. Despite decades of robust and meritorious investigation, our understanding of the mechanisms underlying UV-induced skin carcinogenesis remain incomplete. We previously performed comprehensive transcriptomic profiling in human keratinocytes following exposure to different UV radiation conditions to generate UV-specific gene expression signatures. In this study, we utilized Virtual Inference of Protein Activity by Enriched Regulon (VIPER), a robust systems biology tool, on UV-specific skin cell gene signatures to identify master regulators (MRs) of UV-induced transcriptomic changes. We identified multiple prominent candidate UV MRs, including forkhead box M1 (FOXM1), thyroid hormone receptor interactor 13 and DNA isomerase II alpha, which play important roles in cell cycle regulation and genome stability. MR protein activity was either activated or suppressed by UV in normal keratinocytes. Intriguingly, many of the UV-suppressed MRs were activated in human skin squamous cell carcinomas (SCCs), highlighting their importance in skin cancer development. We further demonstrated that selective inhibition of FOXM1, whose activity was elevated in SCC cells, was detrimental to SCC cell survival. Taken together, our study uncovered novel UV MRs that can be explored as new therapeutic targets for future skin cancer treatment.
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Freitas Ribeiro, Laryssa, Rafael Akira Sato, Andressa de Souza Pollo, Gabriel Augusto Marques Rossi, and Luiz Augusto do Amaral. "Occurrence of Methicillin-Resistant Staphylococcus spp. on Brazilian Dairy Farms that Produce Unpasteurized Cheese." Toxins 12, no. 12 (December 8, 2020): 779. http://dx.doi.org/10.3390/toxins12120779.

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Methicillin-resistant Staphylococcus spp. (MRS) have been identified in several foods, including dairy products. Studies are needed about their occurrence and genetic diversity in the dairy production chain in order to gain a better understanding of their epidemiology and control. This study therefore focuses on isolating and characterizing MRS strains detected in milk used in the production of Brazilian artisanal unpasteurized cheeses. To this end, samples were collected from bovine feces, the hands of milkmen, milking buckets, sieves, unpasteurized milk, whey, water, artisanal unpasteurized cheeses, cheese processing surfaces, cheese handlers, cheese trays, cheese molds, and skimmers at five dairy farms located in the state of São Paulo, Brazil. Colonies suggestive of Staphylococcus spp. were subjected to multiplex PCR to confirm the presence of Staphylococcus aureus and to detect the mecA gene. Sixteen isolates containing mecA gene were detected in samples from unpasteurized cheese and from cheese handlers. None of these isolates were positive to enterotoxin genes. These 16 isolates were subjected to antimicrobial susceptibility tests, which revealed they were resistant to oxacillin, penicillin, and cefepime. Using gene sequencing, the MRS isolates were identified as S. haemolyticus, S. hominis, and S. epidermidis. Furthermore, isolates from cheese handlers’ hands and artisanal unpasteurized cheese presented high genetic similarity by random amplified polymorphic DNA (RAPD-PCR) analysis, which indicates cross contamination during cheese production. Thus, we found that people directly involved in milking and cheese processing activities at small dairy farms are a potential source of contamination of MRS strains in unpasteurized milk and cheese, representing a risk to public health.
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Shovlin, Claire L. "Glaxo/MRS Young Investigator Medal: Molecular Studies on Adenosine Deaminase Deficiency and Hereditary Haemorrhagic Telangiectasia." Clinical Science 94, no. 3 (March 1, 1998): 207–18. http://dx.doi.org/10.1042/cs0940207.

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1. This manuscript describes two different strategies to progress from the clinical assessment of patients to the identification of disease-causing mutations. In the first disease, recognition of a metabolic abnormality allowed direct molecular analysis of the causal gene. In contrast, localization of the second disease gene by linkage analysis was critical to implicate a gene with a previously unsuspected disease role. 2. Two sisters with chronic respiratory disease and recurrent infections were identified as the first cases of adult onset immunodeficiency due to adenosine deaminase deficiency. Autosomal recessive inheritance of two mutations in the adenosine deaminase gene was demonstrated. Enzyme replacement therapy improved the patients' immunological and clinical status. 3. Individuals with pulmonary arteriovenous malformations were used to identify families with hereditary haemorrhagic telangiectasia (HHT, Rendu-Osler-Weber Syndrome). Linkage studies mapped the HHT disease gene in some families to chromosome 9, and demonstrated genetic heterogeneity. The chromosome 9 disease interval was refined, and several candidate genes were assessed. Following the first description of disease-segregating mutations, a complete analysis of the endoglin gene (which encodes an endothelial cell transforming growth factor-β receptor) identified seven novel mutations. Two mutations did not produce mutant mRNA, and disease severity was comparable between families, indicating that HHT results from stoichiometric insufficiency of endoglin. 4. Each study has implications extending beyond the relatively rare disease analysed. The adenosine-deaminase-deficient patients highlight a treatable cause of HIV-negative CD4+ lymphopenia in adults, perhaps accounting for further cases of ‘non-HIV AIDS’. The HHT studies have illuminated a novel area of vascular pathophysiology, with potential relevance to further disease states.
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Trott, Jamie, Yunus Alpagu, Ee Kim Tan, Mohammad Shboul, Yousif Dawood, Michael Elsy, Heike Wollmann, et al. "Mitchell-Riley syndrome iPSCs exhibit reduced pancreatic endoderm differentiation due to a mutation in RFX6." Development 147, no. 21 (October 8, 2020): dev194878. http://dx.doi.org/10.1242/dev.194878.

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ABSTRACTMitchell-Riley syndrome (MRS) is caused by recessive mutations in the regulatory factor X6 gene (RFX6) and is characterised by pancreatic hypoplasia and neonatal diabetes. To determine why individuals with MRS specifically lack pancreatic endocrine cells, we micro-CT imaged a 12-week-old foetus homozygous for the nonsense mutation RFX6 c.1129C>T, which revealed loss of the pancreas body and tail. From this foetus, we derived iPSCs and show that differentiation of these cells in vitro proceeds normally until generation of pancreatic endoderm, which is significantly reduced. We additionally generated an RFX6HA reporter allele by gene targeting in wild-type H9 cells to precisely define RFX6 expression and in parallel performed in situ hybridisation for RFX6 in the dorsal pancreatic bud of a Carnegie stage 14 human embryo. Both in vitro and in vivo, we find that RFX6 specifically labels a subset of PDX1-expressing pancreatic endoderm. In summary, RFX6 is essential for efficient differentiation of pancreatic endoderm, and its absence in individuals with MRS specifically impairs formation of endocrine cells of the pancreas head and tail.
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Lee, Eun-Jae, Mi-Sun Oh, Jong S. Kim, Dae-Il Chang, Jong-Ho Park, Jae-Kwan Cha, Ji Hoe Heo, et al. "Serotonin transporter gene polymorphisms may be associated with poststroke neurological recovery after escitalopram use." Journal of Neurology, Neurosurgery & Psychiatry 89, no. 3 (October 13, 2017): 271–76. http://dx.doi.org/10.1136/jnnp-2017-316882.

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ObjectiveSelective serotonin reuptake inhibitors (SSRIs) putatively improve neurological recovery after stroke. We aimed to investigate whether serotonin transporter (SERT) gene polymorphisms are related to the responsiveness to SSRIs in the poststroke neurological recovery.MethodsThis was a post hoc analysis of the EMOTION study (ClinicalTrials.gov NCT01278498), a randomised, placebo-controlled, double-blind trial examining the efficacy of escitalopram on emotional and neurological disturbances after acute stroke. Patients with no/minimal disability initially (modified Rankin Scale (mRS) 0–1) were excluded. Of the participants, 301 underwent genetic studies of the STin2 (a variable number tandem repeat (VNTR) in intron 2) (STin2 12/10 and STin2 12/12 genotypes) and 5-HTTLPR (a variable-length repeat in the promoter region) polymorphisms of SERT. We explored whether neurological function (National Institutes of Health Stroke Scale (NIHSS) score and mRS) at 3 months would differ according to SERT polymorphisms within each treatment arm (escitalopram and placebo).ResultsAmong the escitalopram users (n=159), neurological function in subjects with STin2 12/10 (n=29) improved significantly more than that in STin2 12/12 carriers (n=130) at 3 months. After adjusting for age, initial NIHSS and depression, STin2 12/10 independently predicted a good clinical outcome (mRS 0–1) (OR 2.99, 95% CI 1.04 to 8.58) at 3 months. However, differences between STin2 polymorphisms were not shown in the placebo group (n=142). 5-HTTLPR polymorphisms were not associated with neurological recovery in any treatment group.ConclusionSTin2 VNTR polymorphisms may be associated with poststroke neurological recovery after SSRI therapy. Further studies are needed to identify the role of serotonin in neurological recovery after stroke.
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Ngassam Tchamba, Cyrille, Jean-Noël Duprez, Pierrick Lucas, Yannick Blanchard, Filip Boyen, Freddy Haesebrouck, Maria A. Argudín, Jacques Mainil, and Damien Thiry. "Comparison of the Staphylococcal Chromosome Cassette (SCC) mec in Methicillin-Resistant Staphylococcus aureus (MRSA) and Non-aureus Staphylococci (MRNAS) from Animals and Humans." Antibiotics 10, no. 3 (March 4, 2021): 256. http://dx.doi.org/10.3390/antibiotics10030256.

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Methicillin-resistant Staphylococcus aureus (MRSA) and non-aureus staphylococci (MRNAS) cause different infections in animals, including mastitis, in livestock and humans. This study aimed to identify and compare the staphylococcal chromosome cassette mec (SCCmec) types of MRSA or MRNAS isolated from several animal species and humans in different countries. Of 1462 S. aureus and non-aureus staphylococci, 68 grew on Chrom MRSA ID® agar, were phenotypically resistant to cefoxitin and tested positive with the PCR for the mecA gene. These 60 MRSA and 8 MRNAS were isolated in Belgium mainly from cows (livestock-associated (LA) MRS) and humans (community-acquired (CA) MRS) and in Japan from dogs and cats. The SCCmec cassettes were identified by multiplex PCR in 52 MRSA and 7 MRNAS and by whole genome sequencing (WGS) in 8 additional MRSA. The SCCmec types IV and V were the most frequent in Belgian LA-MRS and CA-MRS, while the SCCmec type II was identified in four of the five Japanese MRSA. The remaining isolate was a bovine S. haemolyticus in which no SCCmec was identified. These results confirm the high prevalence of the SCCmec types IV and V in LA-MRS and CA-MRS in Belgium, emphasizing the possible public health hazard of the former, and the absence of SCCmec in some MRNAS.
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Dissertations / Theses on the topic "Gene mrs"

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Schwob, Étienne. "Methionyl-trna synthetase mitochondriale de saccharomyces cerevisiae : purification et tentative de clonage de son gene, caracterisation des genes rpk1 codant pour une proteine-kinase et act2 codant pour une nouvelle forme d'actine." Université Louis Pasteur (Strasbourg) (1971-2008), 1988. http://www.theses.fr/1988STR13195.

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Carniel, Fagner. "O telejornal que "Fala pra gente, mas não fala da gente"." reponame:Repositório Institucional da UFPR, 2012. http://hdl.handle.net/1884/28028.

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Resumo: Entendendo que o discurso hegemônico do telejornalismo rural, especificamente na figura do Globo Rural, atua na promoção da modernização agrícola, procuro refletir neste trabalho sobre o papel da televisão nos processos de (re)construção do meio rural em Dois Vizinhos (PR). A hipótese é de que a "presença" da televisão neste contexto acirra a revisão e ressignificação das atividades e condições de vida no contato com experiências e estilos de vida rurais altamente inseridos no mercado e no consumo, veiculados pelo telejornalismo rural. São dinâmicas sociais que configuram a própria agricultura familiar a partir de processos identitários híbridos, forjados no encontro da vida local com a sociedade envolvente. Ativada para designar uma identidade aberta às tensões estabelecidas entre o que se vê na televisão e o que se vive nas comunidades, a agricultura familiar marca a própria diversidade do rural na região, tanto na (re)produção como na representação da vida familiar, abrigando uma polissemia de perspectivas e estratégias identitárias que disputam espaço e ligitimidade nos contextos locais de Dois Vizinhos.
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Yogalingam, Gouri. "Molecular characterisation of feline MPS VI and evaluation of gene therapy /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phy54.pdf.

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Jokubka, Ramūnas. "Kiaulių genetinių žymenų įtaka produktyvumo savybėms." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2005. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2005~D_20051122_142646-74982.

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The study presents evaluation of pig genetic markers (MHS and MC4R) associated with performance traits in the Lithuanian White pig breed. The study presents a direct approach the testing and explanation of the quantitative part of the trait and QTL with marker based on estimated breeding values in the Lithuanian White population. Information about the strategies for association analysis and improvement can be applied for further characterization of the Lithuanian White population. The use of breeding values instead of single measurements reduces the bias in the recorded performance traits, therefore, the results obtained by using the marker for the Lithuanian White population gives animal breeders the opportunities for realization of a short-term goal in their selection criteria.
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Pradhan, Anjala Vinayak. "Genes differentially expressed in adult familial myelodysplastic syndromes (MDS)." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418063.

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Silva, Ivanilde da. "De quem nós/a gente está(mos) falando afinal?" Florianópolis, SC, 2004. http://repositorio.ufsc.br/xmlui/handle/123456789/87528.

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Dissertação (mestrado) - Universidade Federal de Santa Catarina, Centro de Comunicação e Expressão. Programa de Pós-graduação em Linguística
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O objetivo deste trabalho é descrever e analisar a intercambialidade de nós e a gente (e suas respectivas realizações #mos e zero) atrelada à peculiaridade de serem pronomes multirreferenciais, podendo designar, dentro de uma escala de possibilidades, desde as pessoas do discurso a referentes genéricos, a ponto de não se precisar o referente. A análise está apoiada nas concepções teóricas de Benveniste (1988 e 1989) no que se refere às discussões sobre pronomes e subjetividade das pessoas do discurso; nos processos de referenciação e de categorização de referentes do discurso, baseados na abordagem lingüística e sócio-cognitiva de Mondada e Dubois (1995/2003), Apothéloz (1995/2003) e Milner (1995/2003) principalmente, e nos pressupostos teóricos e metodológicos da sociolingüística variacionista. As amostras da pesquisa foram constituídas por 32 entrevistas, 16 colhidas na cidade de Blumenau/SC, da fala de profissionais, muitos deles vinculados a um hospital da mesma cidade, e os demais dados de fala foram colhidos do Programa do Jô, atração televisiva veiculada pela Rede Globo de Televisão. As entrevistas, coletadas na cidade catarinense foram realizadas entre os anos de 2001 e 2002 e as exibidas pelo Programa do Jô no período de 2003 a 2004. As amostras possuem a mesma distribuição de informantes, conforme dito acima, todos com grau de escolaridade superior, classificados de acordo com o sexo e duas faixas etárias. Os resultados gerais da utilização dos pronomes nós e a gente indicam mudança: na medida em que o pronome a gente se estabiliza como pronome pessoal, ele disputa cada vez mais espaço no campo da determinação, concorrendo com o pronome nós.
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Lenormand, Jean-Luc. "Proto-oncogène c-mos et différenciation myogénique." Paris 6, 1994. http://www.theses.fr/1994PA066626.

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Le proto-oncogène c-mos, l'homologue cellulaire de la séquence transformante du virus de souris du sarcome de Moloney, code pour une serine/thréonine kinase cytoplasmique. Nous avons mis en évidence une expression du proto-oncogène c-mos au cours de la myogenèse in vitro dans des cellules musculaires l6 de rat. Nous montrons également qu'il existe une accumulation concomitante du messager et de la protéine c-mos au cours de sa maturation du muscle squelettique chez le rat. Une analyse fine réalisée à partir du muscle squelettique de rat adulte, indique que la protéine mos de 43kda n'est pas associée a un des constituants de la matrice musculaire, mais a deux polypeptides de 80kda et 34kda. Par différentes approches expérimentales, le peptide de 34kda se révèle être la protéine p34cdc2 qui intervient dans la régulation du cycle cellulaire dans tous les organismes étudiés. Nous démontrons, pour la première fois que dans des cellules et tissus somatiques non transformes, une association entre un des éléments indispensables à la régulation du cycle cellulaire et un proto-oncogène. Cette association entre ces deux facteurs dans le muscle squelettique adulte, reflète une activité cytostatique (csf) pour la protéine p43c-mos, qui en inhibant l'activité de la protéine p34cdc2, empêcherait les cellules différenciées de réinitier un cycle cellulaire.
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Li, Xingru. "Wilms' tumor gene 1 in different types of cancer." Doctoral thesis, Umeå universitet, Institutionen för medicinsk biovetenskap, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-103389.

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The Wilms’ tumor gene 1 (WT1) was first reported as a tumor suppressor gene in Wilms’ tumor. However, later studies have shown the oncogenic properties of WT1 in a variety of tumors. It was recently proposed that WT1 was a chameleon gene, due to its dual functions in tumorigenesis. We aimed to investigate the clinical significance of WT1 as biomarker in acute myeloid leukemia (AML) and clear cell renal cell carcinoma (ccRCC) and to elucidate the function of WT1 as an oncogene in squamous cell carcinoma of head and neck (SCCHN). In AML, it was suggested that WT1 expression was an applicable marker of minimal residual disease (MRD). In adult patients with AML, we found a good correlation between WT1 expression levels normalized to two control genes, β-actin and ABL. Outcome could be predicted by a reduction in WT1 expression in bone marrow (≥ 1-log) detected less than 1 month after diagnosis, when β-actin was used as control. Also, irrespective of the control gene used, outcome could be predicted by a reduction in WT1 expression in peripheral blood (≥ 2-log) detected between 1 and 6 months after treatment initiation. Previous studies in RCC demonstrated that WT1 acted as a tumor suppressor. Thus, we tested whether single nucleotide polymorphisms (SNPs) or mutations in WT1 might be associated with WT1 expression and clinical outcome in patients with ccRCC. We performed sequencing analysis on 10 exons of the WT1 gene in a total of 182 patient samples, and we identified six different SNPs in the WT1 gene. We found that at least one or two copies of the minor allele were present in 61% of ccRCC tumor samples. However, no correlation was observed between WT1 SNP genotypes and RNA expression levels. Moreover, none of the previously reported WT1 mutations were found in ccRCC. Nevertheless, we found that a favorable outcome was associated the homozygous minor allele for WT1 SNP. We then further investigated whether WT1 methylation was related to WT1 expression and its clinical significance. Methylation array and pyrosequencing analyses showed that the WT1 promoter region CpG site, cg22975913, was the most frequently hypermethylated CpG site. We found a trend that showed nearly significant correlation between WT1 mRNA levels and hypermethylation in the 5’-untranslated region. Hypermethylation in the WT1 CpG site, cg22975913, was found to be associated with patient age and a worse prognosis. One previous study reported that WT1 was overexpressed in SCCHN. That finding suggested that WT1 might play a role in oncogenesis. We found that both WT1 and p63 could promote cell proliferation. A positive correlation between WT1 and p63 expression was observed, and we identified p63 as a WT1 target gene. Furthermore, several known WT1 and p63 target genes were affected by knocking down WT1. Also, co-immunoprecipitation analyses demonstrated a protein interaction between WT1 and p53. In summary, WT1 gene expression can provide useful information for MRD detection during treatment of patients with AML. In RCC, our results suggested that the prognostic impact of WT1 SNPs was limited to the subgroup of patients that were homozygous for the minor allele, and that WT1 promoter hypermethylation could be used as a prognostic biomarker. In SCCHN, WT1 and p63 acted as oncogenes by affecting multiple genes involved in cancer cell growth.
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Pereira, Monalessa Fábia. "Estudo funcional do gene que codifica um transportador de membrana MFS em Colletotrichum lindemuthianum." Universidade Federal de Viçosa, 2011. http://locus.ufv.br/handle/123456789/5341.

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Conselho Nacional de Desenvolvimento Científico e Tecnológico
Colletotrichum lindemuthianum is the causal agent of common bean antracnose. This fungus, like other phytopatogens, is constantly exposed to several toxic compounds from many sources, what makes indispensable the development of protection strategies against these products. One of these strategies is related to membrane transporters proteins like the Major Facilitator Superfamily (MFS), that could provide protection against toxic compounds or minimizing its action, being essential for the fungal cellular viability maintenance. In this context, this work aimed to inactivate the mfs1 gene encoding a MFS membrane transporter and investigate the phenotypic alterations entailed in an isolate C. lindemuthianum mutant LV49 (race 89) for this gene. To obtain the mutant, it was necessary to confirm if mfs1 gene was organized as a single copy in the C. lindemuthianum genome. The mfs1 gene can be organized in a cluster in view that a second open reading frame, which corresponds to a transcription factor superfamily containing a Zn2-Cys6 domain, identified as clft1, was observed in 3` downstream region of this gene. The mfs1 promoter analysis revealed a putative mfs1 element that is recognized by proteins of this family, what suggests that this protein could be related to the mfs1 expression regulation. The Split-Marker technique proved to be efficient in C. lindemuthianum mfs1 gene inactivation enabling the study of mfs1 function in a mutant by specific integrations without ectopic integrations. The Δmfs1 mutant showed no differences in drug sensibility profile when commonly drugs employed in antracnose control was used and in relation to pathogenicity, the mutant symptoms started earlier on susceptible bean leaves, showing a stress situation due to the genic product absence. It was also observed that mfs1 presents a primordial role in C. lindemuthianum cellular viability maintenance, what was confirmed by the altered conidiation observed, confirming that this gene encodes for a specific hexose membrane transporter, specifically carbon sources like glucose, mannose and fructose. The protein Mfs1 phylogenetic analysis allow us to conclude that this transporter is a SP family member and the MFS proteins are strongly related with the transported substance. Studies conducted with MFS transporters are important to broaden the knowledge of these proteins and to understand the cell viability in C. lindemuthianum.
O fungo Colletotrichum lindemuthianum é o agente causal da antracnose do feijoeiro comum. Este fungo, assim como outros fungos fitopatógenos estão constantemente expostos a uma grande variedade de compostos tóxicos provenientes de várias fontes, o que torna imprescindível para estes o desenvolvimento de mecanismos de proteção contra estes produtos. Uma dessas estratégias está relacionada com a presença de proteínas transportadoras de membrana, como as pertencentes à Principal Superfamília Facilitadora (MFS), que podem fornecer aos fungos proteção contra compostos tóxicos evitando ou minimizando a ação destes, sendo em sua maioria essenciais para a manutenção da viabilidade celular. Este trabalho teve como objetivo inativar o gene mfs1 que codifica para um transportador de membrana da família MFS e investigar as alterações fenotípicas ocasionadas em um mutante C. lindemuthianum isolado LV49 (raça 89) para este gene. Para a obtenção do mutante foi necessário confirmar que o gene mfs1 encontrava-se presente em cópia única no genoma de C. lindemuthianum. O gene mfs1 pode estar organizado em um conjunto de genes com funções relacionadas, uma vez que downstream à região 3’ deste foi identificada uma segunda janela aberta de leitura correspondente a uma proteína da superfamília de fatores de transcrição contendo o domínio Zn2-Cys6, identificado como clft1. A análise do promotor do gene mfs1 revelou um putativo cis elemento de reconhecimento por proteínas desta família, o que sugere que esta proteína possa estar relacionada à regulação da expressão de mfs1. A técnica de Split- Marker mostrou-se eficiente na inativação do gene mfs1 de C. lindemuthianum, possibilitando o estudo da função do gene mfs1, em um mutante com integração específica e livre de integrações ectópicas. O mutante Δmfs1 não mostrou diferenças no perfil de sensibilidade a drogas comumente empregadas no controle da antracnose e em relação à patogenicidade, o mutante induziu mais precocemente os sintomas em folhas de feijoeiro susceptível, evidenciando uma situação de estresse decorrente da ausência do produto gênico. Foi observado também que o gene mfs1 exerce um papel primordial na manutenção da viabilidade celular de C. lindemuthianum, fato este confirmado pela conidiação alterada e pela confirmação de que este gene codifica para um transportador de membrana específico no transporte de hexoses, especificamente glicose, manose e frutose, uma vez que o mutante Δmfs1 mostrou crescimento reduzido quando cultivado em meios contendo apenas glicose, manose e frutose como fontes de carbono. A análise filogenética da proteína Mfs1 associada aos outros resultados obtidos nos sugere que este transportador é um membro da família SP, e que as proteínas MFS estão fortemente relacionadas com o tipo de substância que é transportada. Estudos de natureza básica sobre transportadores MFS são importantes para ampliar os conhecimentos sobre estas proteínas e a viabilidade celular em C. lindemuthianum.
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Croft, S. M. "Mammalian-wide interspersed repeats (MIRs) and their role in mammalian gene function and evolution." Thesis, Nottingham Trent University, 2009. http://irep.ntu.ac.uk/id/eprint/104/.

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Transposable elements (TEs) are ubiquitous components of plant and animal genomes and constitute more than ~45% of the human genome. Though originally considered as 'parasitic' or 'junk' DNA, TEs are now thought to have played a role in shaping genomes during evolution, contributing to genome plasticity and diversity. All classes of retrotransposons accumulate in the genome via a process termed retrotransposition, wherein the elements are reverse transcribed into RNA and inserted into the genome as DNA. Exaptation of these elements can provide additional or novel function for endogenous genes. Mammalian-wide interspersed repeats (MIRs) are short interspersed nuclear elements (SINEs), belonging to the non-autonomous class of retroelements and are found in all mammals. The recruitment of an MIR element by a gene may provide insight into mammalian evolution and gene function. The human genome was screened for genes that have exaptated MIR elements and the compiled dataset was analysed to determine any commonality which may suggest conserved function(s). Subsequently 1359 genes were identified that have exaptated MIR elements, constituting 5% of the total genes in the human genome. MIR elements may be multifunctional, as 1% of the total human genes contain MIRs that are spliced and/or are contributing to protein coding sequences. Subsequently sequence motifs were identified in the MIR consensus sequences which resemble canonical mammalian splice sites; therefore MIR elements recruited in the 5'-UTR and coding sequence may be a result of the exonisation of intronic elements. The MIR-containing transcripts are frequently expressed in neurological tissue, suggesting a role in neuronal function. Moreover a number of MIR-containing mRNA transcripts are known to be localised to the dendritic compartment of the neurone, and ciliated region of photoreceptors. Some of the localised mRNAs contain putative microRNA binding sites within the MIR sequence, and possible dsRNA structures were noted between MIR elements. It is proposed that exaptated MIR elements may be a source of cis-acting regulatory elements, involved in post-transcriptional control of gene expression, including localisation of mRNA to distinct intracellular compartments.
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Books on the topic "Gene mrs"

1

Michaud, Josélito. Dans mes yeux à moi: Récit. Montréal: Libre expression, 2011.

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Michaud, Josélito. Dans mes yeux à moi: Récit. Montréal]: Édition du Club Québec loisirs, 2011.

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Lacerte, A. B. Mes trois Castels: Castel-Isolé, Castel-Joli, Castel-Hanté. [Ottawa?: s.n.], 1995.

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Franco, Lucía Londoño de. El viaje de mis genes: Del valle del Eufrates al valle de Aburrá. [Bogotá], Colombia: Tercer Mundo Editores, 1989.

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Naw mis a mwy: Arweiniad cyflawn i feichiogi, geni a'r wythnosau cyntaf gyda'ch babi newydd. Caerdydd: Llywodraeth Cynulliad Cymru, 2005.

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Yusa, I. Gede. Demokrasi, HAM, dan konstitusi: Perspektif negara-bangsa untuk menghadirkan keadilan : kado untuk guru Prof. Dr. I Dewa Gede Atmadja, S.H., M.S. Edited by Atmadja, I Dewa Gede, 1944-. Malang, Jatim: Setara Press, 2011.

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RICH. Totally Unauthorized Sega Games Guide. Indianapolis, IN: BradyGames, 1994.

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Tom, Badgett, ed. Official Sega Genesis and Game Gear strategies, 2ND Edition. Toronto: Bantam Books, 1991.

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Sandler, Corey. Official Sega Genesis and Game Gear strategies, 3RD Edition. New York: Bantam Books, 1992.

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Zhao, Yong, Han Zhang, Jiansheng Chen, Peng Wu, Guangfeng Chen, and Jichun Tian. Genetic Analyses of Wheat and Molecular Marker-Assisted Breeding, Volume 2: Conditional QTL Analysis and MAS. Springer, 2015.

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Book chapters on the topic "Gene mrs"

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Rotini, Alessio, Giorgia Giacomazzi, Ester Sara Di Filippo, and Maurilio Sampaolesi. "MicroRNAs (miRs) in Muscle Gene Therapy." In Muscle Gene Therapy, 99–119. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03095-7_6.

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Allen, George C., Steven Spiker, and William F. Thompson. "Use of matrix attachment regions (MARs) to minimize transgene silencing." In Plant Gene Silencing, 241–56. Dordrecht: Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4183-3_17.

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Schulz, Nicholas, and George F. Vande Woude. "mos Proto-Oncogene Product and Cytostatic Factor." In Signaling Mechanisms and Gene Expression in the Ovary, 112–28. New York, NY: Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4612-3200-1_9.

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Macrina, Francis L., and C. Jeffrey Smith. "Gene Transmission, MLS, and Tetracycline Resistance in Bacteroides." In Brock/Springer Series in Contemporary Bioscience, 474–89. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_35.

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Najeeb, Sofi, Anumalla Mahender, Annamalai Anandan, Waseem Hussain, Zhikang Li, and Jauhar Ali. "Genetics and Breeding of Low-Temperature Stress Tolerance in Rice." In Rice Improvement, 221–80. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-66530-2_8.

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AbstractLow-temperature stress (LTS) is one of the major abiotic stresses that affect crop growth and ultimately decrease grain yield. The development of rice varieties with low-temperature stress tolerance has been a severe challenge for rice breeders for a long time. The lack of consistency of the quantitative trait loci (QTLs) governing LTS tolerance for any given growth stage over different genetic backgrounds of mapping populations under different low-temperature stress conditions remains a crucial barrier for adopting marker-assisted selection (MAS). In this review, we discuss the ideal location and phenotyping for agromorphological and physiological parameters as indicators for LTS tolerance and also the traits associated with QTLs that were identified from biparental mapping populations and diverse rice accessions. We highlight the progress made in the fields of genome editing, genetic transformation, transcriptomics, and metabolomics to elucidate the molecular mechanisms of cold tolerance in rice. The stage-specific QTLs and candidate genes for LTS tolerance brought out valuable information toward identifying and improving LTS tolerance in rice varieties. We showed 578 QTLs and 38 functionally characterized genes involved in LTS tolerance. Among these, 29 QTLs were found to be colocalized at different growth stages of rice. The combination of stage-specific QTLs and genes from biparental mapping populations and genome-wide association studies provide potential information for developing LTS-tolerant rice varieties. The identified colocalized stage-specific LTS-tolerance QTLs will be useful for MAS and QTL pyramiding and for accelerating mapping and cloning of the possible candidate genes, revealing the underlying LTS-tolerance mechanisms in rice.
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Aragon-Martin, Jose Antonio, and Anne H. Child. "Marfan Syndrome (MFS): Inherited Microfibrillar Disorder Caused by Mutations in the Fibrillin-1 Gene." In Diagnosis and Management of Marfan Syndrome, 233–43. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-5442-6_22.

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Lynen, P., P. Goretzki, V. Gorelov, B. Ebling, T. Mueller, G. Kielbach, and H. D. Röher. "Die Bedeutung des MIS II-Gens für das Wachstumsverhalten des Adenokarzinoms des Pankreas." In Chirurgisches Forum ’97 für experimentelle und klinische Forschung, 91–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60717-2_19.

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Seedhouse, Claire, and Nigel Russell. "Susceptibility Susceptibility to MDS: DNA Repai DNA repair r and Detoxification Detoxification Genes." In The Myelodysplastic Syndromes, 5–24. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0440-4_2.

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Walter, Lutz, Jung Won Seo, and Eberhard Günther. "Comparative aspects of the MHC class I-related MR1, CD1D, and MIC genes in primates." In Major Histocompatibility Complex, 213–21. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-65868-9_15.

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van der Velden, Vincent H. J., and Jacques J. M. van Dongen. "MRD Detection in Acute Lymphoblastic Leukemia Patients Using Ig/TCR Gene Rearrangements as Targets for Real-Time Quantitative PCR." In Leukemia, 115–50. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-418-6_7.

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Conference papers on the topic "Gene mrs"

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Tsurkas, Andrew, and Gang Bao. "Molecular Beacons for Sensitive Gene Detection in Living Cells." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-42959.

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Real-time imaging of gene expression in living cells has the potential to significantly impact clinical and laboratory studies of cancer, including cancer diagnosis and analysis. Molecular beacons (MBs) provide a simple and promising tool for the detection of target mRNA as tumor markers due to their high signal-to-background ratio, and their improved specificity in detecting point mutations. However, the harsh intracellular environment does limit the sensitivity of MB-based gene detection. Specifically, MBs bound to target mRNAs cannot be distinguished from those degraded by nucleases, or opened due to non-specific interactions. To overcome this difficulty, we have developed a novel dual FRET molecular beacons approach in which a pair of molecular beacons, one with a donor fluorophore and a second with an acceptor fluorophore, hybridize to adjacent regions on the same target resulting in fluorescence resonance energy transfer (FRET). The detection of a FRET signal leads to a substantially increased signal-to-background ratio compared with that in single molecular beacon assays and enables discrimination between fluorescence due to specific probe/target hybridization and a variety of false-positive events. We have performed systematic in-solution and cellular studies of dual FRET molecular beacon and demonstrated that this new approach allows for real-time imaging of gene expression in living cells.
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Shimizu, Kazunori, Shigeru Kawakami, Kouji Hayashi, Shingo Katano, Guangyuan Zhang, Dai Maekawa, Mitsuru Hashida, and Satoshi Konishi. "Development of in vivo gene delivery methods in mice using tissue suction devices for abdominal endoscopic gene therapy." In 2012 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2012. http://dx.doi.org/10.1109/mhs.2012.6492389.

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Huang, Alice H., and Robert L. Mauck. "Repeated Dynamic Loading Modulates Cartilage Gene Expression but Does Not Improve Mechanical Properties of MSC-Laden Hydrogels." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204339.

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Mesenchymal stem cells (MSCs) are a multi-potential cell type that can differentiate toward a variety of tissue-specific phenotypes, including cartilage. Given their chondrogenic potential, MSCs are a promising cell source for cartilage tissue engineering (TE). However, while MSCs readily undergo chondrogenesis in 3D culture and deposit a cartilage-like matrix, the mechanical properties of MSC-seeded constructs are greatly inferior to chondrocyte-seeded constructs similarly maintained [1]. To date, optimization strategies for enhancing functional MSC chondrogenesis, including increasing seeding density and transient application of growth factor, have shown limited success [3]. Using microarray analysis, we have recently demonstrated that mis-expression of certain genes, including lubricin, chondromodulin and RGD-CAP, a collagen associated protein, may underlie this disparity in mechanical function [2]. In this study, we examined dynamic compression as an alternative method to enhance MSC differentiation. Previous work using chondrocyte-based constructs have demonstrated that matrix biosynthesis and mechanical properties were improved with the application of cyclic compression [4]. Furthermore, upregulation of lubricin was observed when surface motion was applied to chondrocyte-seeded porous scaffolds [5]. While significant effort has gone toward optimizing loading parameters to direct tissue growth of chondrocyte-based constructs, few studies have examined the effects of mechanical stimulation on MSC-based constructs. Some have demonstrated positive effects on MSC chondrogenesis with application of compressive loading [6, 7], while others have shown that long-term loading may adversely affect the developing mechanical properties of MSC-seeded constructs [8]. In this study, we examined the effects of repeated dynamic compressive loading on MSC chondrogenesis and showed that mechanical properties and gene expression were modulated by this loading modality.
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Mazda, Osam, Tsunao Kishida, Masahiro Matsui, Hiroshi Nakano, Koichiro Yoshimoto, Taketoshi Shimada, Shigeru Nakai, Jiro Imanishr, and Yasuo Hisa. "Nonviral gene administration by means of the Epstein-Barr virus (EBV)-based episomal vectors and it application to gene therapy and regenerative medicine." In 2009 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2009. http://dx.doi.org/10.1109/mhs.2009.5351980.

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Niimi, Miyako, Taisuke Masuda, Kunihiro Kaihatsu, Nobuo Kato, and Fumihito Arai. "Compiled chip for all pretreatment processes of virus gene analysis." In 2013 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2013. http://dx.doi.org/10.1109/mhs.2013.6710394.

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Aljawarneh, Shadi A., Reem Jaradat, Abdelsalam M. Maatuk, and Abdullah Alhaj. "Gene profile classification: A proposed solution for predicting possible diseases and initial results." In 2016 International Conference on Engineering & MIS (ICEMIS). IEEE, 2016. http://dx.doi.org/10.1109/icemis.2016.7745342.

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Srikanth, Panigrahi, and N. Rajasekhar. "A novel cluster analysis for gene-miRNA interactions documents using improved similarity measure." In 2016 International Conference on Engineering & MIS (ICEMIS). IEEE, 2016. http://dx.doi.org/10.1109/icemis.2016.7745383.

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Mehri, Sounira, and Mohamed Hammami. "ACE gene I/D polymorphism and obesity in patients with type 2 diabetes mellitus." In 2017 International Conference on Engineering & MIS (ICEMIS). IEEE, 2017. http://dx.doi.org/10.1109/icemis.2017.8273107.

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Suzuki, Motoshi, Yasuhiro Kamei, Shunsuke Yuba, and Shin Takagi. "IR-LEGO : a tool for gene induction in targeted single cells." In 2008 International Symposium on Micro-NanoMechatronics and Human Science (MHS). IEEE, 2008. http://dx.doi.org/10.1109/mhs.2008.4752440.

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Walter, Matthew J. "Abstract IA11: Spliceosome gene mutations in MDS: Biology and potential therapeutic strategies." In Abstracts: Second AACR Conference on Hematologic Malignancies: Translating Discoveries to Novel Therapies; May 6-9, 2017; Boston, MA. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1557-3265.hemmal17-ia11.

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Reports on the topic "Gene mrs"

1

Schnall, Mitchell D. MRI-Based Screen for Breast Cancer Patients Carrying a Breast Susceptibility Gene. Fort Belvoir, VA: Defense Technical Information Center, July 2001. http://dx.doi.org/10.21236/ada398696.

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Verma, Amit. Meta-Analytical Online Repository of Gene Expression Profiles of MDS Stem Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada603210.

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Schnall, Michael D. MRI-Based Screen for Breast Cancer Patients Carrying a Breast Cancer Susceptibility Gene. Fort Belvoir, VA: Defense Technical Information Center, October 1998. http://dx.doi.org/10.21236/ada359858.

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Schnall, Mitchell D. MRI-Based Screen for Breast Cancer Patients Carrying a Breast Cancer Susceptibility Gene. Fort Belvoir, VA: Defense Technical Information Center, October 1997. http://dx.doi.org/10.21236/ada338681.

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Schnall, Mitchell D. MRI-Based Screen for Breast Cancer Patients Carrying a Breast Cancer Susceptibility Gene. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada390928.

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