Relevant bibliographies by topics / Gene structure in plasmodia

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  2. Dissertations / Theses
  3. Books
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Academic literature on the topic 'Gene structure in plasmodia'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Gene structure in plasmodia"

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Knapp, Bernhard, Erika Hundt, and Hans A. Küpper. "Plasmodium falciparum aldolase: gene structure and localization." Molecular and Biochemical Parasitology 40, no. 1 (April 1990): 1–12. http://dx.doi.org/10.1016/0166-6851(90)90074-v.

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Lal, Altaf A., Vidal F. de la Cruz, Gary H. Campbell, Patricia M. Procell, William E. Collins, and Thomas F. McCutchan. "Structure of the circumsporozoite gene of Plasmodium malariae." Molecular and Biochemical Parasitology 30, no. 3 (September 1988): 291–94. http://dx.doi.org/10.1016/0166-6851(88)90099-0.

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Lajoie-Mazenc, I., C. Detraves, V. Rotaru, M. Gares, Y. Tollon, C. Jean, M. Julian, M. Wright, and B. Raynaud-Messina. "A single gamma-tubulin gene and mRNA, but two gamma-tubulin polypeptides differing by their binding to the spindle pole organizing centres." Journal of Cell Science 109, no. 10 (October 1, 1996): 2483–92. http://dx.doi.org/10.1242/jcs.109.10.2483.

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Cells of eukaryotic organisms exhibit microtubules with various functions during the different developmental stages. The identification of multiple forms of alpha- and beta-tubulins had raised the question of their possible physiological roles. In the myxomycete Physarum polycephalum a complex polymorphism for alpha- and beta-tubulins has been correlated with a specific developmental expression pattern. Here, we have investigated the potential heterogeneity of gamma-tubulin in this organism. A single gene, with 3 introns and 4 exons, and a single mRNA coding for gamma-tubulin were detected. They coded for a polypeptide of 454 amino acids, with a predicted molecular mass of 50,674, which presented 64–76% identity with other gamma-tubulins. However, immunological studies identified two gamma-tubulin polypeptides, both present in the two developmental stages of the organism, uninucleate amoebae and multinucleate plasmodia. The two gamma-tubulins, called gamma s- and gamma f-tubulin for slow and fast electrophoretic mobility, exhibited apparent molecular masses of 52,000 and 50,000, respectively. They were recognized by two antibodies (R70 and JH46) raised against two distinct conserved sequences of gamma-tubulins. They were present both in the preparations of amoebal centrosomes possessing two centrioles and in the preparations of plasmodial nuclear metaphases devoid of structurally distinct polar structures. These two gamma-tubulins exhibited different sedimentation properties as shown by ultracentrifugation and sedimentation in sucrose gradients. Moreover, gamma s-tubulin was tightly bound to microtubule organizing centers (MTOCs) while gamma f-tubulin was loosely associated with these structures. This first demonstration of the presence of two gamma-tubulins with distinct properties in the same MTOC suggests a more complex physiological role than previously assumed.
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Li, Wu-Bo, David J. Bzik, Toshihiro Horii, and Joseph Inselburg. "Structure and expression of the Plasmodium falciparum SERA gene." Molecular and Biochemical Parasitology 33, no. 1 (February 1989): 13–25. http://dx.doi.org/10.1016/0166-6851(89)90037-6.

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Robson, Kathryn J. H., and M. W. Jennings. "The structure of the calmodulin gene of Plasmodium falciparum." Molecular and Biochemical Parasitology 46, no. 1 (May 1991): 19–34. http://dx.doi.org/10.1016/0166-6851(91)90195-c.

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Nunes, Alvaro, Vandana Thathy, Thomas Bruderer, Ali A. Sultan, Ruth S. Nussenzweig, and Robert Ménard. "Subtle Mutagenesis by Ends-in Recombination in Malaria Parasites." Molecular and Cellular Biology 19, no. 4 (April 1, 1999): 2895–902. http://dx.doi.org/10.1128/mcb.19.4.2895.

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ABSTRACT The recent advent of gene-targeting techniques in malaria (Plasmodium) parasites provides the means for introducing subtle mutations into their genome. Here, we used the TRAPgene of Plasmodium berghei as a target to test whether an ends-in strategy, i.e., targeting plasmids of the insertion type, may be suitable for subtle mutagenesis. We analyzed the recombinant loci generated by insertion of linear plasmids containing either base-pair substitutions, insertions, or deletions in their targeting sequence. We show that plasmid integration occurs via a double-strand gap repair mechanism. Although sequence heterologies located close (less than 450 bp) to the initial double-strand break (DSB) were often lost during plasmid integration, mutations located 600 bp and farther from the DSB were frequently maintained in the recombinant loci. The short lengths of gene conversion tracts associated with plasmid integration intoTRAP suggests that an ends-in strategy may be widely applicable to modify plasmodial genes and perform structure-function analyses of their important products.
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Sharma, Yagya D., and Araxie Kilejian. "Structure of the knob protein (KP) gene of Plasmodium falciparum." Molecular and Biochemical Parasitology 26, no. 1-2 (November 1987): 11–16. http://dx.doi.org/10.1016/0166-6851(87)90124-1.

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Schwarz, R. T., V. Riveros-Moreno, M. J. Lockyer, S. C. Nicholls, L. S. Davey, Y. Hillman, J. S. Sandhu, R. R. Freeman, and A. A. Holder. "Structural diversity of the major surface antigen of Plasmodium falciparum merozoites." Molecular and Cellular Biology 6, no. 3 (March 1986): 964–68. http://dx.doi.org/10.1128/mcb.6.3.964.

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The structures of the major merozoite surface antigen of Plasmodium falciparum and the gene encoding it were indistinguishable for the Wellcome strain and the Thai clone T9/94 but different for clones T9/96, T9/98, and T9/101. The central portion of the gene is subject to the greatest variation in structure. The protein from all five lines was found to be posttranslationally modified by covalent addition of both carbohydrate and fatty acid.
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Schwarz, R. T., V. Riveros-Moreno, M. J. Lockyer, S. C. Nicholls, L. S. Davey, Y. Hillman, J. S. Sandhu, R. R. Freeman, and A. A. Holder. "Structural diversity of the major surface antigen of Plasmodium falciparum merozoites." Molecular and Cellular Biology 6, no. 3 (March 1986): 964–68. http://dx.doi.org/10.1128/mcb.6.3.964-968.1986.

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The structures of the major merozoite surface antigen of Plasmodium falciparum and the gene encoding it were indistinguishable for the Wellcome strain and the Thai clone T9/94 but different for clones T9/96, T9/98, and T9/101. The central portion of the gene is subject to the greatest variation in structure. The protein from all five lines was found to be posttranslationally modified by covalent addition of both carbohydrate and fatty acid.
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Tsuboi, Takafumi, Osamu Kaneko, Chiho Eitoku, Nantavadee Suwanabun, Jetsumon Sattabongkot, Joseph M. Vinetz, and Motomi Torii. "Gene structure and ookinete expression of the chitinase genes of Plasmodium vivax and Plasmodium yoelii." Molecular and Biochemical Parasitology 130, no. 1 (August 2003): 51–54. http://dx.doi.org/10.1016/s0166-6851(03)00140-3.

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Dissertations / Theses on the topic "Gene structure in plasmodia"

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Watts, David Ian. "A comparison of gene structure in amoebae and plasmodia of Physarum polycephalum." Thesis, University of Leicester, 1987. http://hdl.handle.net/2381/35177.

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The control of gene expression by rearrangement of DNA sequences, in prokaryotes and eukaryotes, is recorded in several instances. These accompany the differentiation of cells, yielding a new phenotype. The possibility of such a means of gene control operating in Physarum was considered; this organism undergoes marked changes in cell morphology and function during the amoebal-plasmodial transition. Genes activated or inactivated in this transition were examined for possible changes in structure. This was done by using amoebal- and plasmodial-specific cDNAs to probe Southern blots of amoebal and plasmodial DNA, digested with restriction endonucleases. This procedure should have revealed any restriction enzyme polymorphisms that might have existed between amoebae and plasmodia as a result of DNA rearrangements. However, no changes in DNA structure were observed between amoebae and plasmodia. The scope of this investigation is critically assessed. The methylation of cytosine residues has also been proposed as a means of controlling gene expression in eukaryotes. The available amoebal- and plasmodial-specific cDNAs were used therefore to probe Southern blots of amoebal and plasmodial DNA digested with methylation sensitive and insensitive restriction enzymes, in order to examine the methylation patterns of DNA from the two forms. For all phase-specific genes tested, the patterns in amoebae and plasmodia were identical, suggesting that no changes had occurred. Again, the scope of this investigation is assessed, and the possibility of a more extensive search for putative DNA rearrangements or changes in methylation pattern is mooted. To study closely the structure of three plasmodial-specific genes, attempts were made to clone regions of Physarum genomic DNA containing these sequences. It was not possible to isolate positive clones in any useful quantity. The probable reasons for the difficulties encountered are discussed.
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Holding, Thomas Mitchell. "Multi-scale immune selection and the maintenance of structured antigenic diversity in the malaria parasite Plasmodium falciparum." Thesis, University of Exeter, 2018. http://hdl.handle.net/10871/33217.

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The most virulent malaria parasite, Plasmodium falciparum, makes use of extensive antigenic diversity to maximise its transmission potential. Parasite genomes contain several highly polymorphic gene families, whose products are the target of protective immune responses. The best studied of these are the PfEMP1 surface proteins, which are encoded by the var multi-gene family and are important virulence factors. During infection, the parasite switches expression between PfEMP1 variants in order to evade adaptive immune responses and prolong infection. On the population level, parasites appear to be structured with respect to their var genes into non-overlapping repertoires, which can lead to high reinfection rates. This non-random structuring of antigenic diversity can also be found at the level of individual var gene repertoires and var genes themselves. However, not much is known about the evolutionary determinants which select for and maintain this structure at different ecological scales. In this thesis I investigate the mechanisms by which multi-scale immune selection and other ecological factors influence the evolution of structured diversity. Using a suite of theoretical frameworks I show that treating diversity as a dynamic property, which emerges from the underlying infection and transmission processes, has a major effect on the relationship between the parasite’s transmis- sion potential and disease prevalence, with important implications for monitoring control efforts. Furthermore, I show that an evolutionary trade-off between within-host and between-host fitness together with functional constraints on diversification can explain the structured diversity found at both the repertoire and parasite population level and might also account for empirically observed exposure-dependent acquisition of immunity. Together, this work highlights the need to consider evolutionary factors acting at different ecological scales to gain a more comprehensive understanding of the complex immune-epidemiology of P. falciparum malaria.
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Rono, Evans Kiplangat. "Variation in the Anopheles gambiae TEP1 Gene Shapes Local Population Structures of Malaria Mosquitoes." Doctoral thesis, Humboldt-Universität zu Berlin, 2017. http://dx.doi.org/10.18452/18573.

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Die Allele (*R1, *R2, *S1 und *S2) des A. gambiae complement-like thioester-containing Protein 1 (TEP1) bestimmen die Fitness der Mücken, welches die männlichen Fertilität und den Resistenzgrad der Mücke gegen Pathogene wie Bakterien und Malaria-Parasiten. Dieser Kompromiss zwischen Reproduktion und Immunnität hat Auswirkungen auf die Größe der Mückenpopulationen und die Rate der Malariaübertragung. Wie die genetische Diversität von TEP1 die genetische Struktur natürlicher Vektorpopulationen beeinflusst, ist noch unklar. Die Zielsetzung dieser Doktorarbeit waren: i) die biogeographische Kartographierung der TEP1 Allele und Genotypen in lokalen Malariavektorpopulationen in Mali, Burkina Faso, Kamerun, und Kenia, und ii) die Bemessung des Einflusses von TEP1 Polymorphismen auf die Entwicklung humaner P. falciparum Parasiten in der Mücke. Die Analysen der TEP1 Polymorphismen zeigten, dass die natürliche Selektion auf Exone, sowie Introne wirkt, was auf eine starke funktionale Beschränkung an diesem Lokus hindeutet. Außerdem zeigen unsere Daten die strukturierte Erhaltung natürlicher genetischer Variation im TEP1 Lokus, in welchem die Allele und Genotypen spezifische evolutionäre Wege verfolgen. Diese Ergebnisse weisen auf die Existenz von arten- und habitatspezifischen Selektionsdrücken hin, die auf den TEP1 Lokus wirken. Resultate haben gezeigt, dass TEP1*S1 und *S2 Mücken gleichermassen empfänglich für Plasmodium-Infektionen sind. Insgesamt tragen die Resultate der biogeographischen Kartographierung des TEP1 Lokus und der Züchtungs- und Infektionsexperimente zu einem besseren Verständnis über den Einfluss der verschiedenen Vektorarten und lokale Umwelteinflüsse auf die Vektorpopulationen und Malariaübertragung bei. Des weiteren kann die hier beschriebene hochdurchsatz-genotypisierungs Methode, zur Studie lokaler A. gambiae Mückenpopulationen, in der Feldforschungsarbeit eingesetzt werden. Dieser neue Ansatz wird die epidemiologisch relevante Überwachung und Vorhersage dynamischer Prozesse in lokalen Malariavektorpopulationen unterstützen, welche die Entwicklung neuer Strategien der Vektorkontrolle ermöglichen könnten.
The alleles (*R1, *R2, *S1 and *S2) and genotypes of A. gambiae complement-like thioester-containing protein 1 (TEP1) determine the fitness in male fertility and the degree of mosquito resistance to pathogens such as bacteria and malaria parasites. This trade-off between the reproduction and the immunity impacts directly on mosquito population abundance and malaria transmission respectively. How TEP1 genetic diversity influences the genetic structure of natural vector populations and development of human malaria parasites is unclear. The aims of this thesis were to: i) map distribution of TEP1 alleles and genotypes in local malaria vector populations in Mali, Burkina Faso, Cameroon and Kenya, and ii) assess the impact of TEP1 polymorphism on development of human P. falciparum parasites in mosquitoes. Analyses of TEP1 polymorphism revealed that natural selection acts in concert on both exons and introns, suggesting strong functional constrains acting at this locus. Moreover, our data demonstrate a structured maintenance of natural TEP1 genetic variation, where the alleles and the genotypes follow distinct evolutionary paths. These findings suggest the existence of species- and habitat-specific selection patterns that act on TEP1 locus. Results revealed that the TEP1*S1 and *S2 mosquitoes are equally susceptible to Plasmodium infections. Collectively, results of my thesis on the biogeographic TEP1 mapping, and on the breeding and infection experiments contribute to a better understanding of how the vector species and local environmental factors, shape vector population structures and malaria transmission. Furthermore, the high throughput TEP1 genotyping approach reported here could be used for field studies of local A. gambiae mosquito populations. This new approach will benefit surveilance and prediction of dynamics in local malaria vector populations that may have epidemiological significance, and therefore inform the development of novel vector control measures.
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Jones, Piet. "Structure learning of gene interaction networks." Thesis, Stellenbosch : Stellenbosch University, 2014. http://hdl.handle.net/10019.1/86650.

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Thesis (MSc)--Stellenbosch University, 2014.
ENGLISH ABSTRACT: There is an ever increasing wealth of information that is being generated regarding biological systems, in particular information on the interactions and dependencies of genes and their regulatory process. It is thus important to be able to attach functional understanding to this wealth of information. Mathematics can potentially provide the tools needed to generate the necessary abstractions to model the complex system of gene interaction. Here the problem of uncovering gene interactions is cast in several contexts, namely uncovering gene interaction patterns using statistical dependence, cooccurrence as well as feature enrichment. Several techniques have been proposed in the past to solve these, with various levels of success. Techniques have ranged from supervised learning, clustering analysis, boolean networks to dynamical Bayesian models and complex system of di erential equations. These models attempt to navigate a high dimensional space with challenging degrees of freedom. In this work a number of approaches are applied to hypothesize a gene interaction network structure. Three di erent models are applied to real biological data to generate hypotheses on putative biological interactions. A cluster-based analysis combined with a feature enrichment detection is initially applied to a Vitis vinifera dataset, in a targetted analysis. This model bridges a disjointed set of putatively co-expressed genes based on signi cantly associated features, or experimental conditions. We then apply a cross-cluster Markov Blanket based model, on a Saccharomyces cerevisiae dataset. Here the disjointed clusters are bridged by estimating statistical dependence relationship across clusters, in an un-targetted approach. The nal model applied to the same Saccharomyces cerevisiae dataset is a non-parametric Bayesian method that detects probeset co-occurrence given a local background and inferring gene interaction based on the topological network structure resulting from gene co-occurance. In each case we gather evidence to support the biological relevance of these hypothesized interactions by investigating their relation to currently established biological knowledge. The various methods applied here appear to capture di erent aspects of gene interaction, in the datasets we applied them to. The targetted approach appears to putatively infer gene interactions based on functional similarities. The cross-cluster-analysis-based methods, appear to capture interactions within pathways. The probabilistic-co-occurrence-based method appears to generate modules of functionally related genes that are connected to potentially explain the underlying experimental dynamics.
AFRIKAANSE OPSOMMING: Daar is 'n toenemende rykdom van inligting wat gegenereer word met betrekking tot biologiese stelsels, veral inligting oor die interaksies en afhanklikheidsverhoudinge van gene asook hul regulatoriese prosesse. Dit is dus belangrik om in staat te wees om funksionele begrip te kan heg aan hierdie rykdom van inligting. Wiskunde kan moontlik die gereedskap verskaf en die nodige abstraksies bied om die komplekse sisteem van gene interaksies te modelleer. Hier is die probleem met die beraming van die interaksies tussen gene benader uit verskeie kontekste uit, soos die ontdekking van patrone in gene interaksie met behulp van statistiese afhanklikheid , mede-voorkoms asook funksie verryking. Verskeie tegnieke is in die verlede voorgestel om hierdie probleem te benader, met verskillende vlakke van sukses. Tegnieke het gewissel van toesig leer , die groepering analise, boolean netwerke, dinamiese Bayesian modelle en 'n komplekse stelsel van di erensiaalvergelykings. Hierdie modelle poog om 'n hoë dimensionele ruimte te navigeer met uitdagende grade van vryheid. In hierdie werk word 'n aantal benaderings toegepas om 'n genetiese interaksie netwerk struktuur voor te stel. Drie verskillende modelle word toegepas op werklike biologiese data met die doel om hipoteses oor vermeende biologiese interaksies te genereer. 'n Geteikende groeperings gebaseerde analise gekombineer met die opsporing van verrykte kenmerke is aanvanklik toegepas op 'n Vitis vinifera datastel. Hierdie model verbind disjunkte groepe van vermeende mede-uitgedrukte gene wat gebaseer is op beduidende verrykte kenmerke, hier eksperimentele toestande . Ons pas dan 'n tussen groepering Markov Kombers model toe, op 'n Saccharomyces cerevisiae datastel. Hier is die disjunkte groeperings ge-oorbrug deur die beraming van statistiese afhanklikheid verhoudings tussen die elemente in die afsondelike groeperings. Die nale model was ons toepas op dieselfde Saccharomyces cerevisiae datastel is 'n nie- parametriese Bayes metode wat probe stelle van mede-voorkommende gene ontdek, gegee 'n plaaslike agtergrond. Die gene interaksie is beraam op grond van die topologie van die netwerk struktuur veroorsaak deur die gesamentlike voorkoms gene. In elk van die voorgenome gevalle word ons hipotese vermoedelik ondersteun deur die beraamde gene interaksies in terme van huidige biologiese kennis na te vors. Die verskillende metodes wat hier toegepas is, modelleer verskillende aspekte van die interaksies tussen gene met betrekking tot die datastelle wat ons ondersoek het. In die geteikende benadering blyk dit asof ons vermeemde interaksies beraam gebaseer op die ooreenkoms van biologiese funksies. Waar die a eide gene interaksies moontlik gebaseer kan wees op funksionele ooreenkomste tussen die verskeie gene. In die analise gebaseer op die tussen modelering van gene groepe, blyk dit asof die verhouding van gene in bekende biologiese substelsels gemodelleer word. Dit blyk of die model gebaseer op die gesamentlike voorkoms van gene die verband tussen groepe van funksionele verbonde gene modelleer om die onderliggende dinamiese eienskappe van die experiment te verduidelik.
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呂志恆 and Chi-hang Vincent Lui. "Gene structure and expression of human pro-alpha2(XI) collagen (col11A2) gene." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1995. http://hub.hku.hk/bib/B31234355.

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Lui, Chi-hang Vincent. "Gene structure and expression of human pro-alpha2(XI) collagen (col11A2) gene /." [Hong Kong] : University of Hong Kong, 1995. http://sunzi.lib.hku.hk/hkuto/record.jsp?B14394856.

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McCabe, Veronica Mary. "Domain structure of the mouse Xist gene." Thesis, Imperial College London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286333.

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Pearce, Marcela. "Genomic structure of the human utrophin gene." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318897.

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Chua, Y. L. "Chromatin structure of the pea plastocyanin gene." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597674.

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The pea plastocyanin gene (PetE) is a single-copy, nuclear photosynthesis gene. Pea PetE is flanked by an enhancer/5' matrix attachment region (MAR) and a 3' MAR. When linked upstream to uidA directed by the CaMV 35S promoter, the enhancer/5' MAR increased reporter gene expression in transgenic tobacco plants. In contrast, the 3' MAR increased expression only when linked downstream of the reporter gene. The 3' MAR, but not the 5' MAR, decreased variation in reporter gene expression. These results indicate that the two MARs surrounding PetE have different effects on transgene expression. The chromatin structure of PetE was examined at three different transcriptional states by investigating the nuclease accessibility of the gene in pea roots, etiolated shoots and green shoots. Time-course digestions of nuclei with micrococcal nuclease and DNaseI indicated that the enhancer/5' MAR and promoter regions were more resistant to digestion in the inactive gene in pea roots than the same regions in the active gene in shoots, whereas the transcribed region of PetE was digested similarly amongst the tissues. PetE transcription is hence accompanied by changes in the nuclease accessibility of the enhancer/5' MAR and promoter regions only. The acetylation states of histone H3 and H4 proteins associated with PetE were analysed by chromatin immunoprecipitation with antibodies specific for acetylated or non-acetylated histone tails followed by polymerase chain reaction quantification. Comparison of pea tissue indicated that histone acetylation was associated with increased PetE transcription in green shoots. Moreover, acetylation of both histone H3 and H4 proteins was targeted to the enhancer/5' MAR and promoter regions in green shoots, suggesting that only specific nucleosomes along the gene were modified.
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李福基 and Fuk-ki Lee. "Sorbitol dehydrogenase: gene structure, function and mutation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31237241.

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Books on the topic "Gene structure in plasmodia"

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Gene structure and expression. Cambridge [Cambridgeshire]: Cambridge University Press, 1985.

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Julian, Burke, ed. Gene structure and transcription. 2nd ed. Oxford: IRL Press at Oxford University Press, 1992.

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Gene structure and expression. 3rd ed. Cambridge: Cambridge University Press, 1996.

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Gene structure and expression. 2nd ed. Cambridge [England]: Cambridge University Press, 1991.

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Julian, Burke, ed. Gene structure and transcription. Oxford, England: IRL Press, 1988.

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S, Holmes R., and Lim Hwa A, eds. Gene families: Structure, fuction, genetics and evolution. Singapore: World Scientific, 1996.

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S, Dillon Lawrence. The gene: Its structure, function, and evolution. New York: Plenum Press, 1987.

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Shafai, Roshan. The polynucleotide structure of a germin gene. Ottawa: National Library of Canada, 1990.

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Evans, D. E. Plant nuclear structure, genome architecture and gene regulation. Chichester, West Sussex, UK: Wiley-Blackwell, 2013.

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NATO Advanced Research Workshop on Human Apolipoprotein Mutants: from Gene Structure to Phenotypic Expression (1988 Limone sul Garda, Italy). Human apolipoprotein mutants 2: From gene structure to phenotypic expression. New York: Plenum Press, 1989.

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Book chapters on the topic "Gene structure in plasmodia"

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Mukhopadhyay, Tapas, Steven A. Maxwell, and Jack A. Roth. "Gene Structure." In p53 Suppressor Gene, 13–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22275-1_2.

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Neale, David B., and Nicholas C. Wheeler. "Gene Structure and Gene Families." In The Conifers: Genomes, Variation and Evolution, 75–90. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-46807-5_5.

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Axelson-Fisk, Marina. "Gene Structure Submodels." In Comparative Gene Finding, 201–67. London: Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6693-1_5.

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Lyubich, Yuri I., and Ethan Akin. "Stationary Gene Structure." In Mathematical Structures in Population Genetics, 169–209. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76211-6_4.

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Boedtker, Helga, and Sirpa Aho. "Collagen Gene Structure." In Biology of Invertebrate and Lower Vertebrate Collagens, 135–55. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-7636-1_11.

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Axelson-Fisk, Marina. "Gene Structure Submodels." In Comparative Gene Finding, 181–244. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84996-104-2_5.

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Dillon, Lawrence S. "Viral Genes — Structure and Controls." In The Gene, 599–633. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4899-2007-2_10.

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Bagyan, I. L., I. V. Gulina, A. S. Kraev, V. N. Mironov, L. V. Padegimas, M. M. Pooggin, E. V. Revenkova, et al. "Plant Gene Technology." In Genome Structure and Function, 279–318. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5550-2_14.

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Reeck, Gerald R. "Nucleosome Structure." In Chromosomal Proteins and Gene Expression, 1–16. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-7615-6_1.

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Habener, Joel F. "Gene Structure and Regulation." In Molecular Cloning of Hormone Genes, 11–51. Totowa, NJ: Humana Press, 1987. http://dx.doi.org/10.1007/978-1-4612-4824-8_2.

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Conference papers on the topic "Gene structure in plasmodia"

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Batzoglou, Serafim, Lior Pachter, Jill Mesirov, Bonnie Berger, and Eric S. Lander. "Human and mouse gene structure." In the fourth annual international conference. New York, New York, USA: ACM Press, 2000. http://dx.doi.org/10.1145/332306.332326.

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Panfilio, Kristen A. "Trends in bug genome size, gene structure, and gene repertoires." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93920.

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Zhang, Shuqin. "Hierarchical modular structure in gene coexpression networks." In 2012 IEEE 6th International Conference on Systems Biology (ISB). IEEE, 2012. http://dx.doi.org/10.1109/isb.2012.6314123.

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Ben-Dor, Amir, Benny Chor, Richard Karp, and Zohar Yakhini. "Discovering local structure in gene expression data." In the sixth annual international conference. New York, New York, USA: ACM Press, 2002. http://dx.doi.org/10.1145/565196.565203.

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Ram, Ramesh, and Madhu Chetty. "Learning Structure of a Gene Regulatory Network." In 6th IEEE/ACIS International Conference on Computer and Information Science (ICIS 2007). IEEE, 2007. http://dx.doi.org/10.1109/icis.2007.127.

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O'hara, Patrick J., Frank A. Grant, A. Betty, J. Haldmen, and Mark J. Murray. "Structure of the Human Factor VII Gene." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643786.

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Abstract:
Factor VII is a member of a family of vitamin K-dependent, gamma-carboxylated plasma protein which includes factor IX, factor X, protein C, protein S and prothrombin. Activated factor VII (factor Vila) is a plasma serine protease which participates in a cascade of reactions leading to the coagulation of blood. Two overlapping genomic clones containing sequences encoding human factor VII were isolated and characterized. The complete sequence of the gene was determined and found to span 12.8 kilobases. The mRNA for factor VII as demonstrated by cDNA cloning is polyadenylated at multiple sites but contains only one AAUAAA poly-A signal sequence. The mRNA can undergo alternative splicing forming one transcript containing eight segments as exons and another with an additional exon which encodes a larger pre-pro leader sequence. The portion of the pre-pro leader coded for by the additional exon has no known counterpart in the other vitamin K-dependent proteins. The positions of the introns with respect to the amino acid sequence encoded by the eight essential exons of factor VII are the same as those present in factor IX, factor X, protein C and the first three exons of prothrombin. These exons code for domains generally conserved among members of this gene family, including a pre-pro leader (the essential exon la and alternative exon lb), a gamma-carboxylated domain (exons 2 and 3) a growth factor domain (exons 4 and 5) an activation region (exon 6) and a serine protease (exon 8). The corresponding introns in these genes are dissimilar with respect to size and sequence, with the exception of the third intron in factor VII and protein C. Four introns and a portion of exon 8 in factor VII contain regions made up of tandem repeats of oligonucleotide monomer elements. More than a quarter of the intron sequences and more than a third of the 3' untranslated portion of the mRNA transcript consist of these minisatellite tandem repeats. This type of structure is responsible for polymorphisms due to allelic variation in repeat copy number in other areas of the human genome. Tandem repeats can evolve as a result of random crossover in DNA whose sequence is not maintained by selection. This suggests that much of the sequence information present in the introns and untranslated portion of the message is dispensable.
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Holmes, Roger S., and Hwa A. Lim. "Gene Families: Structure, Function, Genetics and Evolution." In VIII International Congress on Isozymes. WORLD SCIENTIFIC, 1996. http://dx.doi.org/10.1142/9789814531344.

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OGREN, P. V., K. B. COHEN, G. K. ACQUAAH-MENSAH, J. EBERLEIN, and L. HUNTER. "THE COMPOSITIONAL STRUCTURE OF GENE ONTOLOGY TERMS." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812704856_0021.

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"Neurotransmitter gene network reconstruction and analisis." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-177.

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"Phase portraits of gene networks models." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-088.

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Reports on the topic "Gene structure in plasmodia"

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Gatewood, J. M. Novel gene complex structure determination. Office of Scientific and Technical Information (OSTI), August 1997. http://dx.doi.org/10.2172/524855.

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Glesne, D., E. Huberman, F. Collart, T. Varkony, and H. Drabkin. Chromosomal localization and structure of the human type II IMP dehydrogenase gene. Office of Scientific and Technical Information (OSTI), May 1994. http://dx.doi.org/10.2172/10148872.

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Derewenda, Zygmunt W. Structure-Function Relationships in Merlin, the Product of the NF2 Causal Gene. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada429527.

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Chisholm, Sally, Martin F. Polz, and Eric J. Alm. Final Report: DOE award: ER64516-1031199-0013966 2007-2011 Genomic Structure, Metagenomics, Horizontal Gene Transfer, and Natural Diversity of Prochlorococcus and Vibrio. Office of Scientific and Technical Information (OSTI), August 2013. http://dx.doi.org/10.2172/1089668.

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