Relevant bibliographies by topics / Genetic Epigenesis

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
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Academic literature on the topic 'Genetic Epigenesis'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 July 2024

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Journal articles on the topic "Genetic Epigenesis"

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Szabad, János. "Előprogramozott gének." Orvosi Hetilap 158, no. 34 (August 2017): 1323–30. http://dx.doi.org/10.1556/650.2017.30837.

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Abstract: Cells feel good and carry on perfect functions when they contain the right types of proteins in the right concentration, at the right time and sites. There are mechanisms that ensure the right level of gene expression in the different cell types: the formation of protein molecules based on the DNA-encoded genetic information. Gene expression can also be regulated through the compactness of chromatin, i.e. the accessibility of the genes. The chromosomes are repositories of the genetic information – the sequence of base pairs – and also of the so-called epigenetic mechanisms that control gene expression through the regulation of chromatin compactness. The epigenetic mechanisms operate through DNA methylation and/or the regulation of chromatin compactness. The present overview takes a look into the phenomenon of epigenesis. It summarizes how genetic crosses reveal the involvement of epigenesis, explains its meaning and impact on life of the organisms. An understanding of epigenesis provides guidance to improve our life. Orv Hetil. 2017; 158(34): 1323–1330.
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Miller, Alexander, Catherine Malabou, Emily Apter, Peter Szendy, Emanuela Bianchi, and Alexander R. Galloway. "On Epigenesis." October, no. 175 (2021): 109–44. http://dx.doi.org/10.1162/octo_a_00418.

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Abstract “On Epigenesis” consists of a series of interrelated short articles examining the philosophical concept of epigenesis, with a particular focus on Catherine Malabou's development of it in contemporary thought. Alexander Miller introduces the topic of epigenesis and considers its significance as a new paradigm. He also presents the reader with an overview of Malabou's work on the topic: Drawing from recent advances in the life sciences as well as the Western philosophical tradition, he claims, Malabou has proposed “an epigenetic paradigm for rationality” for the 21st century. Catherine Malabou explains that when, in 2001, the scientific journal Nature published virtually the entire sequence of three billion bases that make up the human genome, people were surprised: Only five percent of the sequence turned out to actually be genes. Assembled in bunches and clusters, they are separated by vast expanses of so-called gene deserts made up of DNA characterized as “junk” or “repetitive,” which is to say, non-coding. The sequencing of the genome did not offer the revelations that people had expected, marking the end of the “everything is genetic” creed and announcing the rise of the “epigenetic paradigm.” The present article analyzes the implications of this new paradigm in biology, philosophy, and hermeneutics. Emily Apter situates Catherine Malabou's theory of epigenesis within a broader disciplinary context of Continental philosophy, the cognitive turn, and what a brain does or “is” as an object of aesthetic representation. Peter Szendy argues that even if they are not the central focus of her philosophical work, media and medial metaphors play a key role in Catherine Malabou's understanding of epigenetics. Indeed, her views on the epigenetic paradigm shift could lead to a rethinking of mediality. A medium, according to such an epigenetic approach, would be neither simply a storage space nor a carrier: It would be what happens along with the events (whether they involve works or data) that it hosts or transports. Emanuela Bianchi asks whether the epigenesis of “pure reason” can in any sense be “pure,” since epigenesis necessarily involves empirical processes. Foregrounding the topological involvement of the developing organism in its environment in both biological and psychoanalytic registers, she suggests a way forward can be found in thinking of the genesis of reason as both empirical and rational. Alexander R. Galloway traces an etymological path from “epigenetic” back to the Greek verb “gignomai,” meaning “to be born” or “to become.” But what is becoming? And why is becoming better than (mere) being? One answer is that becoming helps one to escape the confines of identity and rote determination. But what happens when the epigenetic paradigm becomes dominant, when contingency, evolution, and becoming prevail over essence, stasis, and determinism?
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Martinson, T. J. "“The Utter Blankness Found Within”: Epigenetic Formalism in House of Leaves." Poetics Today 44, no. 3 (September 1, 2023): 435–62. http://dx.doi.org/10.1215/03335372-10578513.

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Abstract This article examines epigenetic relations through the study of “blankness” in Mark Z. Danielewski's House of Leaves (2000). The novel's experimental form, as well as the eponymous House on Ash Tree Lane, provide particularly productive models for envisioning the structural and mutative agency of immaterial relations responsible for epigenesis, or the molecular signals that alter genetic expression. To examine the agency of blankness as it applies across literary theory and epigenesis, this essay borrows from science studies, new materialisms, biosemiotics, new formalisms, and Derridean deconstruction to offer an interdisciplinary hermeneutics deemed “epigenetic formalism” by which to better conceive of a network—whether biological or literary—whose form absorbs its environmental milieu via the agency of blankness. In this way, examining epigenesis alongside House of Leaves allows for crucial insight into the relationality and formal composition of the human genome, as well as insight into the relationality and composition of forms in a literary text.
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Lapin, A. V., A. V. Tolstov, and I. M. Kulikova. "Unique Ultra-Rich Rare Metal Ores of the Tomtor Complex and Problem of Their Genesis." МИНЕРАЛОГИЯ (MINERALOGY) 5 (October 27, 2019): 70–88. http://dx.doi.org/10.35597/2313-545x-2019-5-3-70-88.

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Main genetic models of the formation of ultra-rich rare metal ores of the Tomtor deposit (Yakutia) are compared on the basis of their structural-textural features, mineral composition, and geologic setting: 1) reducing epigenesis of laterite weathering crusts of carbonatites and 2) redeposition of weathering products of carbonatites. It is shown that the unique features of the Tomtor deposit are explained by more complex (compared with other deposits of weathering crusts of carbonatites) evolution, which is expressed in two consecutive stages of supergenesis: laterite weathering and reducing epigenesis and their total ore-concentrating efects. The searching-forecast criteria for ores of the Tomtor type are suggested on the basis of epigenetic model of their formation.
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Ether, Yashoda, Pravin Jadhav, MP Moharil, MS Dudhare, P. Kale, and R. Dani. "Epigenesis ThroughIn-vitroRegeneration in Soybean Amenable to Genetic Transformation." Vegetos- An International Journal of Plant Research 26, no. 2 (2013): 245. http://dx.doi.org/10.5958/j.2229-4473.26.2.081.

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Vitale, Francesco. "Differing the Ecological Event: Interpretive Mutations between Bio- and Eco-Deconstruction." Oxford Literary Review 45, no. 1 (July 2023): 57–73. http://dx.doi.org/10.3366/olr.2023.0403.

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In Biodeconstruction (2018) I argued that Derrida, in the Life Death seminar (1975–76), would have anticipated the most recent developments in epigenetics, a field in which the dogma of genetic determinism is radically challenged by noting the influence of the environment in the production of mutations in the genetic program, particularly when a genetic population is faced with a radical change in its environmental conditions, which I propose to call an ‘ecological event’. I explore a comparison between the Derridean deconstruction of genetic determinism and the theoretical elaborations of epigenesis, referring to the work of Eva Jablonka and Marion J. Lamb, Evolution in Four Dimensions (2005). Jablonka and Lamb propose a theory of genetic variation in which mutations would be the result of the interpretation of unpredictable environmental events by the individual whose survival would be in danger. Through this comparison, I show that the study of ‘interpretive Mutations’ as reactions to unpredictable environmental events can be helpful in understanding the Derridean theme of the ‘event’, rearticulating it in relation to the radical environmental changes that humanity will sooner or later face.
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Ramozzi-Chiarottino, Zelia. "Jean Piaget’s Genetic Epistemology as a Theory of Knowledge Based on Epigenesis." ATHENS JOURNAL OF HUMANITIES & ARTS 8, no. 3 (June 11, 2021): 209–30. http://dx.doi.org/10.30958/ajha.8-3-2.

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This article aims to highlight Jean Piaget’s theory of knowledge and situate it in this context since its beginnings in Ancient Greece where, in Plato, we already find this seminal idea: knowledge is acquired in successive and upward moments (dialektikê), starting from an opinion on the sensible world (doxa) towards the épistêmê of the intelligible world, the world of Ideas or concepts. Piaget’s Theory of Knowledge, we believe, was determined by four moments: 1) his research as a malacologist under the guidance of Godet and Raymond, 2) the acquaintance with Kant’s philosophy at age of 21, 3) his internship at the Binet/Simon laboratory, 4) his studies on the Limnaea Stagnalis. His core idea: it is possible for human beings to attain the necessary and universal knowledge due to the exchange processes of their organisms with the environment, which give rise to the epigenetic ontogenesis of their specific organic mental structures, framed for the specific act of knowing. Epigenetic ontogenesis begins with the infans first actions in the world, from the very moment of birth. Around two years of age, these actions will be represented and organized in groups linked to empirical experience, until the brain be able to perform the operations of the Abelian Group. The physiological development ends here, and the logico-mathematical knowledge becomes possible.
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Walker, Stephen F. "Is human language just another neurobiological specialization?" Behavioral and Brain Sciences 19, no. 4 (December 1996): 649–50. http://dx.doi.org/10.1017/s0140525x00043508.

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AbstractOne can disagree with Müller that it is neurobiologically questionable to suppose that human language is innate, specialized, and species-specific, yet agree that the precise brain mechanisms controlling language in any individual will be influenced by epigenesis and genetic variability, and that the interplay between inherited and acquired aspects of linguistic capacity deserves to be investigated.
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Schaafsma, S. M., B. J. Riedstra, K. A. Pfannkuche, A. Bouma, and T. G. G. Groothuis. "Epigenesis of behavioural lateralization in humans and other animals." Philosophical Transactions of the Royal Society B: Biological Sciences 364, no. 1519 (December 4, 2008): 915–27. http://dx.doi.org/10.1098/rstb.2008.0244.

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Despite several decades of research, the epigenesis of behavioural and brain lateralization is still elusive, although its knowledge is important in understanding developmental plasticity, function and evolution of lateralization, and its relationship with developmental disorders. Over the last decades, it has become clear that behavioural lateralization is not restricted to humans, but a fundamental principle in the organization of behaviour in vertebrates. This has opened the possibility of extending descriptive studies on human lateralization with descriptive and experimental studies on other vertebrate species. In this review, we therefore explore the evidence for the role of genes and environment on behavioural lateralization in humans and other animals. First, we discuss the predominant genetic models for human handedness, and conclude that their explanatory power alone is not sufficient, leaving, together with ambiguous results from adoption studies and selection experiments in animals, ample opportunity for a role of environmental factors. Next, we discuss the potential influence of such factors, including perinatal asymmetrical perception induced by asymmetrical head position or parental care, and social modulation, both in humans and other vertebrates, presenting some evidence from our own work on the domestic chick. We conclude that both perinatal asymmetrical perception and later social modulation are likely candidates in influencing the degree or strength of lateralization in both humans and other vertebrates. However, in most cases unequivocal evidence for this is lacking and we will point out further avenues for research.
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Tikhonovich, Igor A., and Nikolay A. Provorov. "Epigenetics of ecological niches." Ecological genetics 8, no. 4 (December 15, 2010): 30–38. http://dx.doi.org/10.17816/ecogen8430-38.

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The development of symbioses ensures formation of the super-organism systems for heredity (symbiogenomes) which represent the products of joint adaptations of partners towards an unfavorable environment. Using the examples of symbioses which enable plants and microorganisms to cooperatively overcome the limitations in the major biogenic elements (C, N, P) or impacts of the biotic and abiotic stresses we demonstrate that symbiosis involves not only the de novo formation (epigenesis) by plant of the ecological niches for hosting the microsymbionts, but also the reorganizations of relevant genetic systems in accordance to the partners’ genotypes and environmental conditions. A possibility to address the ongoing processes in terms of epigenetics is evident when the microsymbionts occurring in the novel niches are included into the host reproduction cycle ensuring a stable maintenance of novel adaptation in the next generations suggesting that the newly formed symbiogenome have acquired the properties of a system for inheritance of the newly acquired adaptive traits.
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Dissertations / Theses on the topic "Genetic Epigenesis"

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Moraes, Alberto da Silva. "Supraorganização e extensibilidade da cromatina, e composição nuclear em celulas de camundongo." [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317803.

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Orientador: Maria Luiza Silveira Mello
Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
Made available in DSpace on 2018-08-10T21:08:10Z (GMT). No. of bitstreams: 1 Moraes_AlbertodaSilva_D.pdf: 18240611 bytes, checksum: 0c094268046690a939c185449986f74f (MD5) Previous issue date: 2008
Resumo: Envelhecimento pode ser definido como as mudanças sofridas por um organismo ao longo do tempo. Esse processo, em biologia, é denominado senescência. A senescência celular é um fenômeno observado em células isoladas, e tem sido estudada tipicamente em células em cultura. Sua ocorrência in vivo foi observada em alguns tecidos de mamíferos. As mudanças na estrutura e organização da cromatina que ocorrem em células senescentes incluem, aumento na resistência da cromatina à digestão por nucleases e acúmulo de modificações de histonas e proteínas associadas à heterocromatina. Embora nem todas as células em um organismo envelhecido estejam em estado de senescência, espera-se que mudanças na estrutura e organização da cromatina ocorram. A restrição calórica é a única intervenção conhecida que tem a capacidade de estender o tempo de vida em mamíferos. Após uma dieta de restrição calórica ou jejum muitos genes, cuja expressão encontra-se alterada em animais idosos, têm sua expressão restabelecida aos níveis observados em animais jovens. Acredita-se que mudanças na cromatina também possam ocorrer durante o jejum, e que induzam mudanças no nível de expressão de diversos genes. No presente trabalho, buscando-se alterações na organização da cromatina em hepatócitos de camundongo ao longo do envelhecimento ou submetidos ao jejum, observou-se um aumento das propriedades viscoelásticas da cromatina ao longo do envelhecimento, de acordo com as mudanças na habilidade dessa cromatina em formar fibras estendidas de cromatina. Essas diferenças foram acompanhadas por um desempacotamento da cromatina. Observou-se também que essa viscoelasticidade da cromatina era dependente principalmente de interações desta com a matriz nuclear, e que cópias de genes cuja atividade transcricional não é mais requerida, ou requerida em um nível menor em animais idosos, podem desligar-se temporariamente da matriz nuclear. Mudanças nas propriedades viscoelásticas da cromatina e no seu grau de compactação já haviam sido observadas previamente em animais em jejum. Apesar disso, no presente trabalho, nenhuma diferença com relação à interação dos genes rDNA com a matriz nuclear foi encontrada em animais em jejum. Contudo, independente da condição fisiológica, o DNA aderido à matriz nuclear parece ser rico em genes, enquanto as seqüências heterocromáticas, pobres em genes, geralmente são encontradas tanto associadas com a matriz nuclear quanto dissociadas desta (cuidado com essa conclusão. Está forte). Em hepatócitos de animais idosos foi observado acúmulo de marcadores heterocromáticos (modificações de histonas) e de outras proteínas (proteínas formadoras de heterocromatina e glicoproteínas presentes principalmente nos cromocentros), assim como diminuição das modificações de histonas associadas com transcrição ativa. Todas essas modificações estão relacionadas com alterações na síntese de RNA já relatadas para animais idosos, e são uma evidência de que o controle da expressão gênica, a organização e a composição da cromatina estão intimamente relacionados. Em um outro tipo celular como espermatozóides de camundongo, uma diferente organização nuclear levou a propriedades diferenciadas de sua cromatina com relação às suas propriedades viscoelásticas (aumentadas). Tais diferenças possivelmente estejam relacionadas com um padrão modificado de expressão gênica, uma vez que em espermatozóides, a atividade transcricional é nula ou quase ausente
Abstract: Aging may be defined as the changes that take place in an organism with time. This process, in biology, is called senescence. Cellular senescence is observed in isolated cells, and has been studied typically in cultured cells, but its occurrence in vivo has been shown only in some mammalian tissues. Chromatin changes that take place with cellular senescence include increase in the resistance of chromatin to nuclease digestion and accumulation of histone modifications and non-histone proteins associated with heterochromatin. Although not all cells in an aged organism are subjected to cellular senescence, it is expected that changes in the chromatin structure and organization still occur. Caloric restriction is the only intervention known to extend life span in mammals. It has been shown that many genes whose expression pattern is altered in aged animals can be reverted to the levels observed in young animals after a caloric restriction diet or complete food withdrawal. Changes in chromatin structure may occur during the starvation period to induce changes in the expression level of several genes. With the aim of screening for alterations in the chromatin organization in mouse hepatocyte nuclei with aging or following starvation, we observed an increase in the viscoelastic properties of chromatin with aging, in terms of changes in the ability of this chromatin to form extended chromatin fibers after a lysis treatment in liver imprints on histological slides. These differences were accompanied by chromatin unpackage. Most of the viscoelasticity of the chromatin were dependent on its interactions with the nuclear matrix, and copies of genes whose transcription are no longer required in aged animals, tended to detach from the nuclear matrix. Changes in the viscoelastic properties and packing degree of chromatin had been shown previously in starved animals. However, no differences regarding this feature were seen in the present work. Nevertheless, regardless the physiological condition, DNA attached to the nuclear matrix seems to be gene-rich, while heterochromatic gene-poor regions were found both attached and detached from the nuclear matrix. We observed accumulation of heterochromatic marks (histone modifications) and non-histone proteins (heterochromatin proteins and glycoproteins present mainly in the chromocenters), as well as decreased histone modifications associated with transcription in hepatocyte nuclei of aged mice. All these changes are related to altered RNA synthesis observed in aged animals and are an evidence of the strong relationship between chromatin organization, composition, and control of gene expression. In another cell type, mouse sperm cells, its nuclear organization lead to different chromatin properties regarding its viscoelastic properties (increased). These differences are possibly related to a modified pattern of gene expression since gene transcription is almost or completely absent in sperm cells
Doutorado
Biologia Celular
Doutor em Biologia Celular e Estrutural
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Zhu, Xianmin Elefant Felice. "The histone acetyltransferase Dmel\TIP60 Is essential for multicellular development in Drosophila /." Philadelphia, Pa. : Drexel University, 2007. http://hdl.handle.net/1860/2582.

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Schoenborn, Jamie R. "Comprehensive epigenetic profiling identifies multiple distal regulatory elements directing Ifng transcription /." Thesis, Connect to this title online; UW restricted, 2007. http://hdl.handle.net/1773/5098.

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Ramos, Edward. "Tools for studying gross nuclear organization, dynamics and epigenetic modifications of chromosomes /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/10849.

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McEwen, Kirsten Rose. "Epigenetic regulation of imprinted loci in the mouse." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609297.

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Ko, Soyoung. "Androgen receptor expression and activity roles of inflammation, age-induced oxidative stress, and epigenetic modifications : a dissertation /." San Antonio : UTHSC, 2009. http://learningobjects.library.uthscsa.edu/cdm4/item_viewer.php?CISOROOT=/theses&CISOPTR=42&CISOBOX=1&REC=2.

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Zhang, Le, and 张乐. "Epigenetic regulation in laminopathy-based premature aging." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B46337672.

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Phipps, Sharla Marion Ostein. "Genetic and epigenetic modulation of telomerase activity in development and disease." Birmingham, Ala. : University of Alabama at Birmingham, 2007. https://www.mhsl.uab.edu/dt/2008r/phipps.pdf.

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Thesis (Ph. D.)--University of Alabama at Birmingham, 2007.
Additional advisors: Vithal K. Ghanta, J. Michael Ruppert, Theresa V. Strong, R. Douglas Watson. Description based on contents viewed Oct. 3, 2008; title from PDF t.p. Includes bibliographical references.
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Johansson, Sofia. "The effect of chronic alcoholism on epigenetic patterns regulating gene expression and neurodegeneration in the human brain /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/91-7357-071-0/.

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Loewy, Amanda Duvall 1981. "Hypermethylation of the MMACHC promoter is associated with methionine dependence in the human malignant melanoma cell line Me-Wo-LC1." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=116118.

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Methionine dependence, the inability of cells to grow when the amino acid methionine is replaced in culture medium by its metabolic precursor homocysteine, is characteristic of many cancer cell lines. Most cells proliferate normally under these conditions. The methionine dependent tumorigenic human melanoma cell line MeWo-LC1 was derived from the methionine independent non-tumorigenic line MeWo. The MeWo-LC1 cell line has been shown to have a cellular phenotype similar to that of cells from patients with the cblC inborn error of cobalamin metabolism, with decreased synthesis of cobalamin coenzymes and decreased activity of the cobalamin dependent enzymes methionine synthase and methylmalonyl-CoA mutase. Inability of cblC cells to complement the defect in cobalamin metabolism in MeWo-LC1 suggested that the defect was caused by decreased activity of the MMACHC gene product. However, no potentially disease causing mutations could be detected in the coding sequence of MMACHC in MeWo-LC1. No MMACHC expression could be detected in MeWo-LC1, and there was virtually complete methylation of a CpG island at the 5' end of the MMACHC gene in MeWo-LC1, consistent with inactivation of the gene by methylation; the CpG island was partially methylated in MeWo and only lightly methylated in control fibroblasts. Transfection of MeWo-LC1 with wild type MMACHC with a constitutive promoter resulted in correction of the defect in cobalamin metabolism and restoration of the ability of cells to grow in medium containing homocysteine. We conclude that epigenetic inactivation of the MMACHC gene is responsible for methionine dependence in MeWo-LC1.
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Books on the topic "Genetic Epigenesis"

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Epigenetic regulation of lymphocyte development. Heidelberg: Springer, 2011.

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1918-, Preer John R., ed. Paramecium: Genetics and epigenetics. Boca Raton: CRC Press, 2008.

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Beale, Geoffrey. Paramecium: Genetics and epigenetics. Boca Raton: CRC Press, 2008.

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Beale, Geoffrey. Paramecium. London: Taylor and Francis, 2008.

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Pintér, Balázs. Epigenetics: Mechanisms, functions and human effects. New York: Nova Science Publishers, 2010.

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Balázs, Pintér, and Mészáros Zsolt, eds. Epigenetics: Mechanisms, functions, and human effects. Hauppauge, NY: Nova Science Publishers, 2009.

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Bruce, Stillman, Stewart, David J., Ph.D., and Cold Spring Harbor Laboratory, eds. Epigenetics. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 2004.

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David, Allis C., Jenuwein Thomas, and Reinberg Danny, eds. Epigenetics. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory Press, 2007.

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Jörg, Tost, ed. Epigenetics. Norfolk, UK: Caister Academic Press, 2008.

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Jörg, Tost, ed. Epigenetics. Norfolk, UK: Caister Academic Press, 2008.

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Book chapters on the topic "Genetic Epigenesis"

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Petronis, Arturas, James L. Kennedy, and Andrew D. Paterson. "Genetic Anticipation: Fact or Artifact, Genetics or Epigenetics?" In Epigenetics and Anticipation, 199–200. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-17678-4_11.

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Graw, Jochen. "Epigenetik." In Genetik, 291–338. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-44817-5_8.

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Graw, Jochen. "Epigenetik." In Genetik, 361–422. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-60909-5_8.

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Thorson, Jennifer L. M., Millissia Ben Maamar, and Michael K. Skinner. "Epigenetics in Sperm, Epigenetic Diagnostics, and Transgenerational Inheritance." In Handbook of Genetic Diagnostic Technologies in Reproductive Medicine, 61–71. 2nd ed. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003024941-7.

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Pascual, Marién, and Sergio Roa. "Epigenetics." In SpringerBriefs in Genetics, 23–50. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6366-5_3.

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Henderson, Mark. "Epigenetik." In 50 Schlüsselideen Genetik, 188–91. Heidelberg: Spektrum Akademischer Verlag, 2010. http://dx.doi.org/10.1007/978-3-8274-2381-8_48.

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Buttlar, Jann, Carlo Klein, Alexander Bruch, Alexandra Fachinger, Johanna Funk, Harmen Hawer, and Aaron Kuijpers. "Die Epigenetik." In Tutorium Genetik, 299–327. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-56067-9_13.

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Siegmund, K. D., and S. Lin. "Epigenetics." In Handbook of Statistical Genetics, 1301–21. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470061619.ch40.

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Schuol, Sebastian. "Epigenetics and Genetic Determinism (in Popular Science)." In Epigenetics, 41–54. Wiesbaden: Springer Fachmedien Wiesbaden, 2017. http://dx.doi.org/10.1007/978-3-658-14460-9_4.

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Horsthemke, Bernhard. "Epigenetics." In Vogel and Motulsky's Human Genetics, 299–318. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-37654-5_11.

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Conference papers on the topic "Genetic Epigenesis"

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Andrade, Gustavo Moreira, Antonio Márcio Teodoro Cordeiro Silva, Amanda Hasan Figueiredo, Ana Luiza Gomes Monteiro, Leonardo Chaves de Oliveira Moraes, and Giovanna Silva Quirino. "The use of epigenetics in the treatment of triplenegative breast cancer, focusing on IncRNA." In Brazilian Breast Cancer Symposium 2023. Mastology, 2023. http://dx.doi.org/10.29289/259453942023v33s1032.

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Objective: In triple-negative breast cancer (CMTN), the standard therapeutic procedure is usually not very effective due to the aggressiveness of the disease. Therefore, it is important to identify and characterize new forms of treatment for this neoplasm. In this context, the study of the genetic material of diseases has gained notoriety among alternative forms of therapy, as long non-coding RNAs (IncRNAs) have been identified in neoplastic cells. Therefore, the aim of this study was to evaluate the use of epigenetics in the treatment of CMTN, with emphasis on lncRNAs. Methodology: A systematic review of the specialized scientific literature was carried out, in the PubMed database, with the descriptors: “breast cancer,” “epigenetic,” and “treatment”; the Boolean operator: “AND”; and the filters: “free full text,” “adults: 19+ years,” and publication date from 2021 to 2023. A total of 32 articles were identified, with 3 included Results: Epigenetics influences the treatment of breast cancer; as the lncRNA was found in neoplastic cells, it was possible to monitor the prognosis of the disease. The lncRNA Uc003xsl.1 was associated with a poor prognosis, as it was related to advanced stages of CMTN, increasing the transcriptional activity of NFkB, which promotes tumor progression. On the contrary, the lncRNA LINC00472 proved to be a marker of good prognosis, as it inhibited the proliferation, invasion, and migration of neoplastic cells in the CMTN. Furthermore, with regard to breast cancer, lncRNA IGF-2AS proved to be an important biomarker, as it slows tumor growth in vivo, repressing malignancy and tumor progression. Therefore, IncRNAs have gained notoriety in treatment as regulators of breast cancer tumorigenesis. Conclusion: Thus, the use of epigenetics in the treatment of CMTN has proven to be essential to curbing neoplastic cells, as it interferes with tumor proliferation in different ways, either by influencing transcription or by slowing down growth.
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Son, Joo-Hiuk. "Active Demethylation of Cancer Cells using Terahertz Radiation for Potential Cancer Treatment." In Conference on Lasers and Electro-Optics/Pacific Rim. Washington, D.C.: Optica Publishing Group, 2022. http://dx.doi.org/10.1364/cleopr.2022.cmp3a_02.

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Carcinogenesis involves DNA methylation which is a primary alteration in DNA in the development of cancer before genetic mutation. Because the abnormal DNA methylation is found in most cancer cells, the assessment of DNA methylation using terahertz radiation can be a novel optical method to detect and control cancer. The methylation has been directly observed by terahertz time-domain spectroscopy and this epigenetic chemical change could be manipulated to the state of demethylation using resonant terahertz radiation. Demethylation of cancer cells is a key issue in epigenetic cancer therapy and our results may lead to the treatment of cancer using electromagnetic waves.
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Novohatin, Vladimir, Vladimir Vorob'ev, and Viktor Dragavcev. "New algorithms of phenotyping for seven genetic-physiological systems which maximising yield of future varieties." In Multifunctional adaptive fodder production. ru: Federal Williams Research Center of Forage Production and Agroecology, 2022. http://dx.doi.org/10.33814/mak-2022-28-76-103-115.

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Improvement of hereditary drought resistance in crops is recently being among the main objectives for food security of humanity because of global warming and the growing costs for bakery and forage grain. Analysis of complex properties of drought resistance in cereal (phenotyping) shows the limitation of a canonical genocentric approach and the approaches based on molecular genetics to solve the problem of significant hereditary improvement of drought resistance. The priority epigenetic approach that we propose is based on the Theory of eco-genetic organization of quantitative traits (TEGOQT). In TEGOQT seven genetic-physiological systems (GPS) involved in harvest increasing, but not particular traits of product ivity, are to be operated with. These GPS are attractions; micro-distribution of attractive plastics substances between grains and chaff in ear; adaptability to drought, cold, frost, heat, salt, etc; horizontal immunity; ''payment'' by dry biomass for a limiting factor of soil nutrition — N, P, K, etc.; tolerance to plant density in phytocenosis; hereditary variability in duration of the phases of ontogenesis. In this paper we discuss drought adaptability as a part of GPS complex. It is shown that phenotyping evaluation is necessary to analize drought tolerance, the complex property to which no less than 22 components characters contribute.
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Fonck, E., G. Feigl, L. Augsburger, D. A. Rüfenacht, and N. Stergiopulos. "Structural Properties of Human Cerebral Arteries as Assessed by a Constituent-Based Biomechanical Model." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176122.

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Human cerebral arteries may develop aneurysms, as a result of geometrical and structural modifications in the arterial wall resulting from a variety of genetic, epigenetic and biomechanical factors. Aneurysms exhibit a markedly different wall composition and structure, characterized by relatively low elastin content as shown in Figure 1.
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La Cava, William, Thomas Helmuth, Lee Spector, and Kourosh Danai. "Genetic Programming with Epigenetic Local Search." In GECCO '15: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2739480.2754763.

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Vesnina, Anna. "INFLUENCE OF GENETIC FEATURES ON THE DEVELOPMENT OF ATHEROSCLEROSIS." In I International Congress “The Latest Achievements of Medicine, Healthcare, and Health-Saving Technologies”. Kemerovo State University, 2023. http://dx.doi.org/10.21603/-i-ic-22.

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In the course of the work, it was found that a large number of genetic features affect the development of atherosclerosis (gene SNPs, telomere length, epigenetic factors, circadian rhythms). Studies of SNP genes, in particular those affecting lipid metabolism, predominate. Relatively new is the study of the relationship between chrononutrition and the development of metabolic diseases.
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Johnson, Brennan M., Deborah M. Garrity, and Lakshmi P. Dasi. "Quantifying the Biomechanics of the Embryonic Zebrafish Heart." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80730.

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Congenital heart defects are present in 4 to 50 per 1000 live births[1]. Most of these defects begin within the first few weeks post fertilization. Ample evidence exists which shows that mechanical epigenetic factors, such as pressure and shear stress, play key roles in heart development [2–3]. It has been shown in-vitro that cardiomyocytes are able to sense and respond to the presence of pulsatile flow[4], and that shear stress can activate genetic pathways which might ultimately dictate the morphological development of the cardiac tissue[5]. When blood flow characteristics have been changed experimentally, embryonic hearts consistently develop serious malformations. In order to understand mechanical epigenetic factors and their role in heart development, it is critical to contrive techniques for quantitatively measuring the biomechanics of the embryonic heart.
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Couvertier, Daniel J., Erik Goodman, and Kalyanmoy Deb. "Towards an epigenetics-inspired control system for power dispatch problem." In GECCO '17: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3067695.3076104.

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Yuen, Sizhe, Thomas H. G. Ezard, and Adam J. Sobey. "The effect of epigenetic blocking on dynamic multi-objective optimisation problems." In GECCO '22: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3520304.3529022.

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Wang, Yajuan, Onur Dur, Michael J. Patrick, Joseph P. Tinney, Kimimasa Tobita, Kerem Pekkan, and Bradley B. Keller. "Hemodynamic Investigation of Normal Developing Aortic Arch in the Chick Embryo." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193264.

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Governed by genetic and epigenetic feedback [1], during embryonic cardiac development, the anatomy of aortic arches demonstrates drastic three dimensional (3D) changes that interact with the function of cardiovascular system. Six major pairs of aortic arches appear at different embryonic periods and eventually form the two brachiocephalic arteries (left and right third), an aortic arch (left fourth) and pulmonary arteries and ductus arteriosus (left and right sixth) [2–4], Fig 1. Flow-driven hemodynamic loading plays a major role in this dynamic process. Morphological studies on the embryonic aortic arches began over 100 years ago while the recent remarkable developments include understanding genetic determinants such as the effects of neural crest cells [5,6]. However the relationship between hemodynamic factors and the dynamic 3D geometry changes is still limited requiring an interdisciplinary research effort [7,8].
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Reports on the topic "Genetic Epigenesis"

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Seale, Maria, Natàlia Garcia-Reyero, R. Salter, and Alicia Ruvinsky. An epigenetic modeling approach for adaptive prognostics of engineered systems. Engineer Research and Development Center (U.S.), July 2021. http://dx.doi.org/10.21079/11681/41282.

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Prognostics and health management (PHM) frameworks are widely used in engineered systems, such as manufacturing equipment, aircraft, and vehicles, to improve reliability, maintainability, and safety. Prognostic information for impending failures and remaining useful life is essential to inform decision-making by enabling cost versus risk estimates of maintenance actions. These estimates are generally provided by physics-based or data-driven models developed on historical information. Although current models provide some predictive capabilities, the ability to represent individualized dynamic factors that affect system health is limited. To address these shortcomings, we examine the biological phenomenon of epigenetics. Epigenetics provides insight into how environmental factors affect genetic expression in an organism, providing system health information that can be useful for predictions of future state. The means by which environmental factors influence epigenetic modifications leading to observable traits can be correlated to circumstances affecting system health. In this paper, we investigate the general parallels between the biological effects of epigenetic changes on cellular DNA to the influences leading to either system degradation and compromise, or improved system health. We also review a variety of epigenetic computational models and concepts, and present a general modeling framework to support adaptive system prognostics.
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Schorry, Elizabeth K. Genetic and Epigenetic Differences in Monozygotic Twins with NF1. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada554129.

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Ruvinsky, Alicia, Maria Seale, R. Salter, and Natàlia Garcia-Reyero. An ontology for an epigenetics approach to prognostics and health management. Engineer Research and Development Center (U.S.), March 2023. http://dx.doi.org/10.21079/11681/46632.

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Techniques in prognostics and health management have advanced considerably in the last few decades, enabled by breakthroughs in computational methods and supporting technologies. These predictive models, whether data-driven or physics-based, target the modeling of a system’s aggregate performance. As such, they generalize assumptions about the modelled system’s components, and are thus limited in their ability to represent individual components and the dynamic environmental factors that affect composite system health. To address this deficiency, we have developed an epigenetics-inspired knowledge representation for engineered system state that encompasses components and environmental factors. Epigenetics is concerned with explaining how environmental factors affect the expression of an organism’s genetic material. The field has derived important in-sights into the development and progression of disease states based on how environmental factors impact genetic material, causing variations in how a gene is expressed. The health of an engineered system is similarly influenced by its environment. A foundation for a new approach to prognostics based on epigenetics must begin by representing the entities and relationships of an engineered system from the perspective of epigenetics. This paper presents an ontology for an epigenetics-inspired representation of an engineered system. An ontology describing the epigenetics of an engineered system will enable the composition of a formal model and the incremental development of a more robust, causal reasoning system.
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Park, Jong Y. Genetic and Epigenetic Biomarkers for Recurrent Prostate Cancer After Radiotherapy. Fort Belvoir, VA: Defense Technical Information Center, May 2014. http://dx.doi.org/10.21236/ada609389.

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Park, Jong. Genetic and Epigenetic Biomarkers for Recurrent Prostate Cancer After Radiotherapy. Fort Belvoir, VA: Defense Technical Information Center, May 2013. http://dx.doi.org/10.21236/ada581491.

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Yamamoto, Fumiichiro. Identification of Tumor Suppressor Genes by Genetic and Epigenetic Genome-Scanning. Fort Belvoir, VA: Defense Technical Information Center, April 2008. http://dx.doi.org/10.21236/ada485734.

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Fu, Xiang-Dong. Chemical Strategy to Translate Genetic/Epigenetic Mechanisms to Breast Cancer Therapeutics. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada610934.

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Fu, Xiang-Dong, and Betty Diamond. Chemical Strategy to Translate Genetic/Epigenetic Mechanisms to Breast Cancer Therapeutics. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada562003.

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Ding, Sheng. Chemical Strategy to Translate Genetic/epigenetic Mechanisms to Breast Cancer Therapeutics. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada564152.

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Geck, Peter. Genetic and Epigenetic Silencing of the AS3 Proliferative rrest Gene in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada443071.

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