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Books on the topic 'Genetic risk factor'

Author: Grafiati

Published: 7 July 2024

Last updated: 31 July 2025

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1

National Institute on Alcohol Abuse and Alcoholism (U.S.) and Editorial Experts Inc, eds. Alcoholism, an inherited disease. U.S. Department of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1985.

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2

Petrakis, Peter L. Alcoholism, and inherited disease. U.S. Dept. of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1985.

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3

David, Brusick, ed. Methods for genetic risk assessment. Lewis Publishers, 1994.

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4

Offit, Kenneth. Clinical cancer genetics: Risk counseling and management. Wiley-Liss, 1998.

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5

Kåre, Berg, ed. Genetic approaches of coronary heart disease and hypertension. Springer-Verlag, 1991.

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6

1946-, Choy Wai Nang, ed. Genetic toxicology and cancer risk assessment. Marcel Dekker, 2001.

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7

Symposium on Phenotypic Variation in Populations: Relevance to Risk Assessment (1986 Brookhaven National Laboratory). Phenotypic variation in populations: Relevance to risk assessment. Plenum Press, 1988.

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8

A, Levin Morris, and Strauss Harlee S, eds. Risk assessment in genetic engineering. McGraw-Hill, 1991.

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9

G, Evers-Kiebooms, ed. Genetic risk, risk perception, and decision making: Proceedings of a conference held July 28-29, 1986, Leuven, Belgium. A.R. Liss, 1987.

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10

Gould, Randi L. Cancer and genetics: Answering your patients' questions. PRR, 1997.

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11

1960-, Foulkes William D., and Hodgson S. V, eds. Inherited susceptibility to cancer: Clinical, predictive, and ethical perspectives. Cambridge University Press, 1998.

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12

Batsheva, Bonné-Tamir, and Adam Avinoam, eds. Genetic diversity among Jews: Diseases and markers at the DNA level. Oxford University Press, 1992.

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13

Hodgson, S. V. A practical guide to human cancer genetics. Cambridge University Press, 1993.

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14

V, Hodgson S., ed. A practical guide to human cancer genetics. 3rd ed. Cambridge University Press, 2007.

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15

Ueland, Per Magne, and Rima Rozen. MTHFR polymorphisms and disease. Landes Bioscience, 2005.

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16

Teichler-Zallen, Doris. To test or not to test: A guide to genetic screening and risk. Thorndike Press, 2009.

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17

Rebecca, Sutphen, and Steligo Kathy, eds. Confronting hereditary breast and ovarian cancer: Identify your risk, understand your options, change your destiny. Johns Hopkins University Press, 2012.

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18

Robert, Cloninger C., and Begleiter Henri, eds. Genetics and biology of alcoholism. Cold Spring Harbor Laboratory Press, 1990.

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19

Constantine, Zervos, North Atlantic Treaty Organization. Scientific Affairs Division., and NATO Advanced Research Workshop on Oncogene and Transgenics Correlates of Cancer Risk Assessments (1991 : Attikē, Greece), eds. Oncogene and transgenics correlates of cancer risk assessments. Plenum Press, 1992.

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20

1961-, Rodríguez Santiago, ed. Genetic endocrinology of the metabolic syndrome. Nova Science Publishers, 2009.

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21

Seshadri, Sudha, and Stéphanie Debette, eds. Risk Factors for Cerebrovascular Disease and Stroke. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199895847.001.0001.

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Risk Factors for Cerebrovascular Disease and Stroke describes environmental and genetic determinants for cerebrovascular disease and stroke from the perspective of an international group of neurologists, epidemiologists, and geneticists who are at the forefront of research and education on these issues. Unlike other books in the field, which solely deal with physiology, diagnosis, and management of stroke, this essential book discusses prevention factors as well as the causes. This unique book takes a comprehensive approach to risk prediction while integrating epidemiological, genetic, and sta

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22

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0043.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now an additional 12 JIA susceptibility loci with evidence for association in more than one study. In addition, some subtype-specific associations are emerging. These risk loci now need to be investigated fur

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23

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0043_update_002.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and large-scale genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now 17 JIA susceptibility loci which reach the genome-wide significance threshold for association and a further 7 regions with evidence for association in more than one study. In addition, some su

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24

Hinks, Anne, and Wendy Thomson. Genetics of juvenile rheumatic diseases. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199642489.003.0043_update_003.

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Juvenile rheumatic diseases are heterogeneous, complex genetic diseases; to date only juvenile idiopathic arthritis (JIA) has been extensively studied in terms of identifying genetic risk factors. The MHC region is a well-established risk factor but in the last few years candidate gene and large-scale genome-wide association studies have been utilized in the search for non-HLA risk factors. There are now 17 JIA susceptibility loci which reach the genome-wide significance threshold for association and a further 7 regions with evidence for association in more than one study. In addition, some su

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25

Gaff, Clara, Louise Keogh, and Elizabeth Lobb. Communicating genetic risk. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198736134.003.0034.

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The discovery of cancer predisposing genetic mutations has heightened community awareness of the link between family history, genetic constitution, and personal risk. The component of an individual’s cancer risk that is due to their genetic make-up can be described as their ‘genetic risk’. Knowledge of genetic risk can assist both individuals with cancer and unaffected individuals to make decisions about healthcare and inform relatives who may share that genetic risk. Accordingly, patients seek advice about their risk and its implications and management from general practitioners (primary heal

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26

Introduction to risk calculation in genetic counseling. 2nd ed. Oxford University Press, 1999.

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27

Introduction to risk calculation in genetic counseling. 3rd ed. Oxford University Press, 2007.

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28

Putaala, Jukka, and Nicolas Martinez-Majander. Risk factors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0002.

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Risk factors in young-onset stroke include both traditional and unconventional as well as both chronic and temporal ones. In young patients, unconventional risk factors such as oral contraceptive use, antiphospholipid antibodies, genetic thrombophilia, acute infections, illicit drug use, and migraine may play a greater role than in elderly patients. However, recent large studies have challenged this traditional view suggesting that young adult stroke would occur mostly due to such unconventional risk factors. These studies have shown a high prevalence of in particular modifiable behavioural ri

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29

Holdt, Lesca M., and Daniel Teupser. Genetic background of atherosclerosis and its risk factors. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0002.

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This chapter is concerned with how atherosclerosis risk is modulated by a complex interplay between genetic and environmental risk factors. The contribution of genetics to the variability of atherosclerosis risk is estimated as 50%. Recent genome-wide association studies have led to the identification of over 50 gene variants which modulate atherogenesis. Risk factors for atherosclerosis are also partly genetically determined and some of the variants which play a role in atherogenesis overlap with those modulating its risk factors. However, the current relevance of these findings for clinical

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30

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Informa Healthcare, 2001.

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31

Introduction to risk calculation in genetic counselling. Oxford University Press, 1991.

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32

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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33

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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34

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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35

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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36

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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37

Genetic susceptibility to cancer. Kluwer Academic, 1998.

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38

Garte, Seymour. Genetic Susceptibility to Cancer. Springer London, Limited, 2012.

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39

Garte, Seymour. Genetic Susceptibility to Cancer. Springer London, Limited, 2013.

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40

Choy, Wai Nang. Genetic Toxicology and Cancer Risk Assessment. Taylor & Francis Group, 2001.

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41

Bianchi, Filippo, and Salvatore De Rossi. Aneuploidy: Etiology, Disorders, and Risk Factors. Nova Science Publishers, Incorporated, 2012.

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42

Woodhead, Avril. Phenotypic Variation in Populations:Relevance to Risk Assessment (Basic Life Sciences). Springer, 1988.

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43

Laboratory), Symposium on Phenotypic Variation in Populations: Relevance to Risk Assessment (1986 Brookhaven National. Phenotypic variation in populations. Plenum, 1988.

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44

Young, Ian D. Introduction to Risk Calculation in Genetic Counseling. Oxford University Press, USA, 2006.

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45

Young, Ian D. Introduction to Risk Calculation in Genetic Counseling. Oxford University Press, 2007.

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46

CLARK, ROSS D. Medical, Genetic & Behavioral Risk Factors of the Sporting Breeds. Xlibris, 2017.

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47

Banerjee, Amitava, and Kaleab Asrress. Risk factors for cardiovascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0086.

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The most prevalent cardiovascular diseases (CVDs) are atherosclerotic, affecting all arterial territories. Epidemiologic studies such as the Framingham and INTERHEART studies have firmly established the commonest or ‘traditional’ risk factors for CVD; namely, smoking, hypertension, diabetes mellitus, hypercholesterolaemia, and a family history of CVD. The ‘risk-factors approach’ to CVD looks at these factors, individually and in combination, in the causation of disease. The complex causation pathways involve interplay of individual factors, whether genetic or environmental. More recently, ther

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48

Previvors Facing The Breast Cancer Gene And Making Lifechanging Decisions. Avery Publishing Group, 2010.

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49

Clark, Ross D. Medical, Genetic and Behavioral Risk Factors of the Terrier Breeds. XLIBRIS, 2017.

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50

Roman, Shiloh Angelus. Anxiety Disorders: Risk Factors, Genetic Determinants and Cognitive-Behavioral Treatment. Nova Science Publishers, Incorporated, 2014.

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