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Journal articles on the topic 'Genetic risk factor'

Author: Grafiati

Published: 7 July 2024

Last updated: 31 July 2025

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1

Koch, Linda. "Genetic T2DM risk factor found." Nature Reviews Endocrinology 10, no. 3 (2014): 128. http://dx.doi.org/10.1038/nrendo.2013.273.

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2

Sokolova, I. V., D. A. Mustafina, A. G. Sadertdinova, et al. "GENETIC RISK FACTOR FOR IDIOPATHIC SCOLIOSIS." International Journal of Applied and Fundamental Research (Международный журнал прикладных и фундаментальных исследований), no. 1 2023 (2023): 25–29. http://dx.doi.org/10.17513/mjpfi.13501.

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3

Reitsma, Pieter H. "Genetic Risk Factors of Thrombosis." Blood 114, no. 22 (2009): SCI—43—SCI—43. http://dx.doi.org/10.1182/blood.v114.22.sci-43.sci-43.

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Abstract Abstract SCI-43 Venous thrombosis is a common episodic disease with a steep age gradient. Interactions between various risk factors determine the development of the disease, and the proportion of variance attributable to genetic factors may be as high as 50-60%. There are six (moderately) strong genetic risk factors. First there are heterozygous deficiencies of the natural anticoagulants protein C, protein S, and antithrombin. These deficiency states are quite rare in the general population (in all races) and their genetic architecture is complex with hundreds of documented mutations.

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Ohishi, Mitsuru, Kenshi Fujii, Takazo Minamino, et al. "A potent genetic risk factor for restenosis." Nature Genetics 5, no. 4 (1993): 324–25. http://dx.doi.org/10.1038/ng1293-324.

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5

Senior, Kathryn. "New genetic risk factor for sporadic PD." Nature Reviews Neurology 5, no. 7 (2009): 354. http://dx.doi.org/10.1038/nrneurol.2009.79.

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6

Bertina, Rogier M. "Factor V Leiden and other coagulation factor mutations affecting thrombotic risk." Clinical Chemistry 43, no. 9 (1997): 1678–83. http://dx.doi.org/10.1093/clinchem/43.9.1678.

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Abstract Five genetic defects have been established as risk factors for venous thrombosis. Three are protein C, protein S, and antithrombin deficiencies, defects in the anticoagulant pathways of blood coagulation. Together they can be found in ∼15% of families with inherited thrombophilia. Their laboratory diagnosis is hampered by the large genetic heterogeneity of these defects. The other two genetic risk factors, resistance to activated protein C associated with the factor V Leiden mutation and increased prothrombin associated with the prothrombin 20210 A allele, are much more prevalent and

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Schwab, Manfred, Andreas Claas, and Larissa Savelyeva. "BRCA2: a genetic risk factor for breast cancer." Cancer Letters 175, no. 1 (2002): 1–8. http://dx.doi.org/10.1016/s0304-3835(01)00752-2.

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8

EVANS, JEFF. "Major Genetic Risk Factor Is Discovered for Parkinson's." Clinical Psychiatry News 38, no. 2 (2010): 33. http://dx.doi.org/10.1016/s0270-6644(10)70097-7.

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9

Medda, Emanuela, Serena Donati, Angela Spinelli, and Gian Carlo Di Renzo. "Genetic amniocentesis: a risk factor for preterm delivery?" European Journal of Obstetrics & Gynecology and Reproductive Biology 110, no. 2 (2003): 153–58. http://dx.doi.org/10.1016/s0301-2115(03)00106-4.

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10

Utermann, G. "Lipoprotein(a) ? A genetic risk factor for CHD." Fresenius' Journal of Analytical Chemistry 343, no. 1 (1992): 37. http://dx.doi.org/10.1007/bf00331978.

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11

Bertina, Rogier M., and Hans L. Vos. "A Novel Genetic Risk Factor for Venous Thrombosis." Clinical Chemistry 57, no. 4 (2011): 637–38. http://dx.doi.org/10.1373/clinchem.2010.158998.

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12

Chaiteerakij, Roongruedee, and Lewis R. Roberts. "Telomerase mutation: A genetic risk factor for cirrhosis." Hepatology 53, no. 5 (2011): 1430–32. http://dx.doi.org/10.1002/hep.24304.

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13

Seppa, Nathan. "Risk factor: Genetic defect hikes breast cancer threat." Science News 161, no. 17 (2009): 259–60. http://dx.doi.org/10.1002/scin.5591611703.

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Daly, Ann K., Christopher P. Day, Yang-Lin Liu, and Quentin M. Anstee. "TM6SF2 as a genetic risk factor for fibrosis." Hepatology 62, no. 4 (2015): 1321. http://dx.doi.org/10.1002/hep.27656.

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15

RJ, Irmawati, Elsi Tandi Pailan, and Baharuddin Baharuddin. "Risk Factor Analysis of Gout Arthritis." Jurnal Ilmiah Kesehatan Sandi Husada 12, no. 1 (2023): 157–62. http://dx.doi.org/10.35816/jiskh.v12i1.919.

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Gout arthritis is an inflammatory disease in the joints, which is still very high and is still a public health problem, especially in Indonesia with a high prevalence. This study aims to determine the risk factors that influence the incidence of Gout arthritis. Quantitative research with a case control study approach. A sample of 60 respondents was 30 each for the case and control groups. The instrument used in this study was in the form of a questionnaire or questionnaire. The data were analyzed using the chi-square test. Genetic statistical test results with values p = 0.002 (<0.05) OR =

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16

Payne, Thomas H., Lue Ping Zhao, Calvin Le, et al. "Electronic health records contain dispersed risk factor information that could be used to prevent breast and ovarian cancer." Journal of the American Medical Informatics Association 27, no. 9 (2020): 1443–49. http://dx.doi.org/10.1093/jamia/ocaa152.

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Abstract Objective The genetic testing for hereditary breast cancer that is most helpful in high-risk women is underused. Our objective was to quantify the risk factors for heritable breast and ovarian cancer contained in the electronic health record (EHR), to determine how many women meet national guidelines for referral to a cancer genetics professional but have no record of a referral. Methods and Materials We reviewed EHR records of a random sample of women to determine the presence and location of risk-factor information meeting National Comprehensive Cancer Network (NCCN) guidelines for

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17

Masuda, Junichi, Toru Nabika, and Yoshitomo Notsu. "Silent stroke: pathogenesis, genetic factors and clinical implications as a risk factor." Current Opinion in Neurology 14, no. 1 (2001): 77–82. http://dx.doi.org/10.1097/00019052-200102000-00012.

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18

Biguzzi, Eugenia, Filippo Castelli, Willem M. Lijfering, et al. "Rise of levels of von Willebrand factor and factor VIII with age: Role of genetic and acquired risk factors." Thrombosis Research 197 (January 2021): 172–78. http://dx.doi.org/10.1016/j.thromres.2020.11.016.

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19

Cushman, Mary. "Inherited Risk Factors for Venous Thrombosis." Hematology 2005, no. 1 (2005): 452–57. http://dx.doi.org/10.1182/asheducation-2005.1.452.

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Abstract Venous thrombosis occurs as a consequence of genetic and environmental risk factors. Since the discovery of factor V Leiden, the most common genetic risk factor, there has been intense interest in clarifying the roles of genes and the environment with thrombosis risk. The translation of this risk information to clinical practice is a challenging one in the setting of a rapidly expanding knowledge base that includes application of genetic medicine. There are benefits, but also potential harms, of testing for inherited disorders associated with thrombosis. This paper reviews inherited r

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20

Stepler, Kaitlyn E., Taneisha R. Gillyard, Calla B. Reed, Tyra M. Avery, Jamaine S. Davis, and Renã A. S. Robinson. "ABCA7, a Genetic Risk Factor Associated with Alzheimer’s Disease Risk in African Americans." Journal of Alzheimer's Disease 86, no. 1 (2022): 5–19. http://dx.doi.org/10.3233/jad-215306.

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African American/Black adults are twice as likely to have Alzheimer’s disease (AD) compared to non-Hispanic White adults. Genetics partially contributes to this disparity in AD risk, among other factors, as there are several genetic variants associated with AD that are more prevalent in individuals of African or European ancestry. The phospholipid-transporting ATPase ABCA7 (ABCA7) gene has stronger associations with AD risk in individuals with African ancestry than in individuals with European ancestry. In fact, ABCA7 has been shown to have a stronger effect size than the apolipoprotein E (APO

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21

Horne, McDonald K., and Donna Jo McCloskey. "Factor V Leiden as a Common Genetic Risk Factor for Venous Thromboembolism." Journal of Nursing Scholarship 38, no. 1 (2006): 19–25. http://dx.doi.org/10.1111/j.1547-5069.2006.00072.x.

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22

Cheekurthy, Alice Jayapradha. "Predisposition of Obesity through Genetic and Non-Genetic Risk Factors." Journal of Endocrinology Research 2, no. 2 (2021): 27. http://dx.doi.org/10.30564/jer.v2i2.2767.

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Globally there is an increase in the number of people affected by obesity.This has increased the count of individuals to double,triple, and even quadruple. Obesity is a complex disease that has a genetic, behavioural,socioeconomic, and environmental effect. This raises morbidity and mortality in obesity. It is an important predisposition for diabetes as well as the current pandemic COVID-19. The rationale of this case-control observational study is to identify obese individuals among the diabetic and non-diabetic population. The study includes non - genetic factors like lipid profiles with gen

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23

Cattaneo, Marco, Emanuela Taioli, Valerio De Stefano, Patrizia Chiusolo, Pier Mannuccio Mannucci, and Ida Martinelli. "Genetic Risk Factors for Superficial Vein Thrombosis." Thrombosis and Haemostasis 82, no. 10 (1999): 1215–17. http://dx.doi.org/10.1055/s-0037-1614362.

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SummaryInherited thrombophilic states are associated with an increased risk for deep vein thrombosis (DVT), but whether they are also risk factors for superficial vein thrombosis (SVT) is uncertain. We assessed the risk conferred by inherited thrombophilic states in patients with a first episode of SVT in whom the coexistence of DVT had been ruled out by ultrasonography. Sixty-three patients with SVT, after exclusion of patients with varicose veins, malignant or autoimmune disease, and 537 healthy individuals were investigated. The G1691A mutation in the factor V gene, the G20210A mutation in

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24

Weng, Lu-Chen, Sarah R. Preis, Olivia L. Hulme, et al. "Genetic Predisposition, Clinical Risk Factor Burden, and Lifetime Risk of Atrial Fibrillation." Circulation 137, no. 10 (2018): 1027–38. http://dx.doi.org/10.1161/circulationaha.117.031431.

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25

Carulli, Lucia. "Telomere shortening as genetic risk factor of liver cirrhosis." World Journal of Gastroenterology 21, no. 2 (2015): 379. http://dx.doi.org/10.3748/wjg.v21.i2.379.

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26

Gallone, Salvatore, Silvia Boschi, Elisa Rubino, et al. "Is HCRTR2 a Genetic Risk Factor for Alzheimer's Disease?" Dementia and Geriatric Cognitive Disorders 38, no. 3-4 (2014): 245–53. http://dx.doi.org/10.1159/000359964.

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27

Kotyuk, Eszter, Viktor Biro, Julianna Bircher, Zsuzsanna Elek, Maria Sasvari, and Anna Szekely. "ABCA1 Polymorphism, a Genetic Risk Factor of Harm Avoidance." Journal of Individual Differences 38, no. 3 (2017): 189–95. http://dx.doi.org/10.1027/1614-0001/a000235.

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Abstract. Even though cholesterol homeostasis and self-harm behaviors have shown to be associated, gene polymorphisms of the cholesterol system have not been studied yet in the context of self-harm related personality traits. Here we present an association study between six ABCA1 polymorphisms and temperament scales measured by Cloninger’s Temperament and Character Inventory on 253 young adults. An association between ABCA1 rs4149264 and harm avoidance has been observed. This association remained significant after Bonferroni correction. Haplotype analysis confirmed an independent association b

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28

Watanabe, Takehiro, Yi-Qiang Liang, Hyoe Inomata, et al. "Genetic analysis of multiple risk factor syndrome in rats." Journal of the American College of Cardiology 41, no. 6 (2003): 276. http://dx.doi.org/10.1016/s0735-1097(03)82297-0.

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29

Nelen, Willianne LDM, Eric AP Steegers, Tom KAB Eskes, and Henk J. Blom. "Genetic risk factor for unexplained recurrent early pregnancy loss." Lancet 350, no. 9081 (1997): 861. http://dx.doi.org/10.1016/s0140-6736(97)24038-9.

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30

Cunha, Madalena, Graça Aparício, João Duarte, Anabela Pereira, Carlos Albuquerque, and António Oliveira. "Genetic heritage as a risk factor enabling chilhood obesity." Atención Primaria 45 (May 2013): 201–7. http://dx.doi.org/10.1016/s0212-6567(13)70023-4.

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31

Jones, Rachel. "Genetic risk factor identified for cystic fibrosis liver disease." Nature Reviews Gastroenterology & Hepatology 6, no. 11 (2009): 627. http://dx.doi.org/10.1038/nrgastro.2009.171.

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32

Gabriel, Carmen Comas, Margarita Torrents Muns, Ana Munoz Prades, Ignacio Rodriguez Garcia, and Bernat Serra Zantop. "468: Genetic amniocentesis: a risk factor for preterm delivery?" American Journal of Obstetrics and Gynecology 204, no. 1 (2011): S187. http://dx.doi.org/10.1016/j.ajog.2010.10.487.

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33

Kueppers, Friedrich. "AATD as a genetic risk factor for aneurysmal disease." Lancet 402, no. 10413 (2023): 1625–26. http://dx.doi.org/10.1016/s0140-6736(23)01747-6.

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34

Krug, Tiago, Helena Manso, Liliana Gouveia, et al. "Kalirin: a novel genetic risk factor for ischemic stroke." Human Genetics 127, no. 5 (2010): 513–23. http://dx.doi.org/10.1007/s00439-010-0790-y.

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35

Farré, Xavier, Roderic Espín, Alexandra Baiges, et al. "Evidence for shared genetic risk factors between lymphangioleiomyomatosis and pulmonary function." ERJ Open Research 8, no. 1 (2021): 00375–2021. http://dx.doi.org/10.1183/23120541.00375-2021.

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IntroductionLymphangioleiomyomatosis (LAM) is a rare low-grade metastasising disease characterised by cystic lung destruction. The genetic basis of LAM remains incompletely determined, and the disease cell-of-origin is uncertain. We analysed the possibility of a shared genetic basis between LAM and cancer, and LAM and pulmonary function.MethodsThe results of genome-wide association studies of LAM, 17 cancer types and spirometry measures (forced expiratory volume in 1 s (FEV1), forced vital capacity (FVC), FEV1/FVC ratio and peak expiratory flow (PEF)) were analysed for genetic correlations, sh

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36

Walter, S., M. M. Glymour, K. Koenen, et al. "Do genetic risk scores for body mass index predict risk of phobic anxiety? Evidence for a shared genetic risk factor." Psychological Medicine 45, no. 1 (2014): 181–91. http://dx.doi.org/10.1017/s0033291714001226.

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BackgroundObesity and anxiety are often linked but the direction of effects is not clear.MethodUsing genetic instrumental variable (IV) analyses in 5911 female participants from the Nurses' Health Study (NHS, initiated 1976) and 3697 male participants from the Health Professional Follow-up Study (HPFS, initiated 1986), we aimed to determine whether obesity increases symptoms of phobic anxiety. As instrumental variables we used the fat mass and obesity-associated (FTO) gene, the melanocortin 4 receptor (MC4R) gene and a genetic risk score (GRS) based on 32 single nucleotide polymorphisms (SNPs)

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37

Djordjevic, Valentina, Ljiljana Rakicevic, and Dragica Radojkovic. "An overview of genetic risk factors in thrombophilia." Srpski arhiv za celokupno lekarstvo 138, suppl. 1 (2010): 79–81. http://dx.doi.org/10.2298/sarh10s1079d.

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Thrombophilia is a multifactorial disorder, involving both genetic and acquired risk factors that affect the balance between procoagulant and anticoagulant factors and lead to increased tendency to thrombosis. The concept that thrombophilia could be associated with genetic defects was first proposed in 1965 after the discovery of familiar antihrombin III deficiency. Further family studies showed that deficiency of protein C or protein S also increased thrombotic risk. In the coming years the advent in DNA technology, especially the invention of PCR reaction, played an important role in the ide

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38

Keïta, Kaly, Cheick Oumar Kamissoko, Ibrahima Amadou Dembélé, et al. "Environmental Factors (External Exposome) and Rare Immunological Diseases (Systemic Lupus Erythematosus): Is Smoking a Risk Factor or Protective Factor?" SAS Journal of Medicine 11, no. 04 (2025): 272–77. https://doi.org/10.36347/sasjm.2025.v11i04.003.

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Introduction: The interaction between genetic and environmental factors influencing susceptibility to the development of systemic lupus erythematosus (SLE) is widely described in the literature. The strongest epidemiological evidence, largely from the North, exists in favor of an increased risk of SLE associated with exposure to current smoking, and a protective effect of moderate alcohol consumption associated against the development of SLE. The north-south differences in genetic and environmental characteristics in patients with SLE is openned research question in literature. We do not know

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39

Simmonds, Rachel, José Hermida, Suely Rezende, and David Lane. "Haemostatic Genetic Risk Factors in Arterial Thrombosis." Thrombosis and Haemostasis 86, no. 07 (2001): 374–85. http://dx.doi.org/10.1055/s-0037-1616235.

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SummaryHaemostasis plays an integral role in arterial thrombotic disease. However, establishing which of the factors are risk factors has proven surprisingly difficult. Because of its technical simplicity and digital nature, the study of haemostatic polymorphisms as risk factors has grown in popularity. Once established as a risk factor, a genetic polymorphism has the potential to aid selective prophylaxis and therapy of disease. Numerous reports have now been published on polymorphisms of coagulation and fibrinolytic factors, of coagulation and fibrinolytic inhibitory proteins, and of platele

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40

Burgess, Stephen, Christopher N. Foley, and Verena Zuber. "Inferring Causal Relationships Between Risk Factors and Outcomes from Genome-Wide Association Study Data." Annual Review of Genomics and Human Genetics 19, no. 1 (2018): 303–27. http://dx.doi.org/10.1146/annurev-genom-083117-021731.

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An observational correlation between a suspected risk factor and an outcome does not necessarily imply that interventions on levels of the risk factor will have a causal impact on the outcome (correlation is not causation). If genetic variants associated with the risk factor are also associated with the outcome, then this increases the plausibility that the risk factor is a causal determinant of the outcome. However, if the genetic variants in the analysis do not have a specific biological link to the risk factor, then causal claims can be spurious. We review the Mendelian randomization paradi

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41

Shanmugapriya, Dr V. "BREAST CANCER EPIDEMIOLOGY, CLASSIFICATION AND RISK FACTOR." International Scientific Journal of Engineering and Management 04, no. 03 (2025): 1–6. https://doi.org/10.55041/isjem02339.

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Breast cancer remains one of the leading causes of cancer-related morbidity and mortality worldwide. This journal explores the latest advancements in breast cancer research, covering key areas such as epidemiology, risk factors, early detection methods, molecular and genetic influences, novel treatment approaches, and patient care strategies. Emphasis is placed on innovative diagnostic tools, targeted therapies, and personalized medicine, which have significantly improved survival rates and treatment outcomes.

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42

Vorobev, A. V., A. D. Makatsariya, V. O. Bitsadze, A. G. Solopova, and D. A. Ponomarev. "Thrombophilia identified as a risk factor for thrombogenesis in cancer patients." Obstetrics, Gynecology and Reproduction 15, no. 3 (2021): 228–35. http://dx.doi.org/10.17749/2313-7347/ob.gyn.rep.2021.232.

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Aim: to assess a rate and range of genetic and acquired thrombophilia in onco-gynecologic patients with ovarian cancer, uterine corpus cancer and cervical cancer.Materials and Мethods. A prospective controlled cohort non-randomized interventional study was conducted: within the years 2014 to 2020, there were examined 546 women with genital malignancies, divided into 2 groups: group I – 155 cancer patients with former thrombosis, group II – 391 women with female genital cancer without former thrombotic complications. Control group consisted of 137 patients with benign female genital tumors. The

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43

Stickeler, Elmar, and Ingo B. Runnebaum. "Genetic risk factors for familial ovarian cancer." British Menopause Society Journal 8, no. 3 (2002): 92–97. http://dx.doi.org/10.1258/136218002100321776.

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In Europe ovarian cancer represents the third most common cancer of the female genital tract, with 30,000 newly diagnosed patients per year. Family history is the most significant risk factor. Lifetime risk for ovarian cancer increases from 1.4% for women with a negative family history to 14.6-32.2% in women from affected families. About 5-10% of ovarian cancers are hereditary and supposed to occur in three different forms: hereditary breast and ovarian cancer syndrome (HBOC), site-specific hereditary ovarian cancer (HOC) and hereditary nonpolyposis colorectal cancer syndrome (HNPCC). HBOC and

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44

Santos, Izabela, Ana Fernandes, Marinez Sousa, Cláudia Ferreira, and Karina Gomes. "Genetic polymorphisms as a risk factor for dyslipidemia in children." Journal of Pediatric Genetics 02, no. 02 (2015): 069–75. http://dx.doi.org/10.3233/pge-13050.

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45

OOKI, Syuichi. "Statistical genetic analysis of multiple risk factor syndrome in Japan." Japanese Journal of Health and Human Ecology 68, no. 4 (2002): 123–32. http://dx.doi.org/10.3861/jshhe.68.123.

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Kwinta, Przemko, and Jacek J. Pietrzyk. "Retinopathy of prematurity: is genetic predisposition an important risk factor?" Expert Review of Ophthalmology 2, no. 2 (2007): 275–83. http://dx.doi.org/10.1586/17469899.2.2.275.

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47

Bubnov, Yu, and N. Erina. "THE BLOOD GENETIC MARKERS AS RISK FACTOR IN ARTERIAL HYPERTENSION." Journal of Hypertension 22, Suppl. 2 (2004): S349. http://dx.doi.org/10.1097/00004872-200406002-01219.

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48

Ahrenstorf, Gerrit, Hui Zhi Low, Katja Kniesch, et al. "LILRA3 deletion is a genetic risk factor of HIV infection." AIDS 31, no. 1 (2017): 25–34. http://dx.doi.org/10.1097/qad.0000000000001304.

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49

Haiman, Christopher A., Loïc Le Marchand, Jennifer Yamamato, et al. "A common genetic risk factor for colorectal and prostate cancer." Nature Genetics 39, no. 8 (2007): 954–56. http://dx.doi.org/10.1038/ng2098.

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50

Sullivan, P. F. "Questions about DISC1 as a genetic risk factor for schizophrenia." Molecular Psychiatry 18, no. 10 (2013): 1050–52. http://dx.doi.org/10.1038/mp.2012.182.

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