Relevant bibliographies by topics / Gentamicine

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Gentamicine'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 July 2024

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Journal articles on the topic "Gentamicine"

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Glesberts, Marlon. "Gentamicine." Nursing 22, no. 4 (2016): 43. http://dx.doi.org/10.1007/s41193-016-0076-4.

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Stanić, Momčilo, Radoslav Mitić, and Milan Jakovljević. "Aminoglycosides and Kidney Function." Journal of Medical Biochemistry 26, no. 4 (2007): 294–99. http://dx.doi.org/10.2478/v10011-007-0037-1.

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Aminoglycosides and Kidney FunctionThe aminoglycosides are bactericide antibiotics with predominant effecting on the Gramm-negative bacteries, then staphylococci and mycobacteria with a small therapeutic range and with expressive ototoxic, nephrotoxic and with rare neurotoxic side effects. In this work the value of creatinine are investigated in serum of patients who are hospitalized in Internal department of the Health Center in Kosovska Mitrovica, before, in the course of and after therapy with the aminoglycosides antibiotics were given in different doses. The creatinine was determined by kinetic method (Jaffe). The significant increasing of creatinine values in serum was recorded with the groups of patients who have got gentamicine 2x120 mg i.m., gentamicine 2x120 mg in combination with furosemide, the gentamicine 2x120 mg with the patients with diabetes mellitus, gentamicine 2x120 mg in the course of 14 days until with the groups who have been getting gentamicine 1x120 mg, gentamicine 2x80 mg and amikacine 2x500 mg were not recorded the significant increased values of creatinine. The determination of creatinine for the sake of the following degree damage of kidney function in the course therapy of the aminoglycosides was very important.
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Ionescu (Filip), Oana, Maria Viorica Ciocilteu, Costel Valentin Manda, et al. "Bone - Graft Delivery Systems of Type PLGA- gentamicin and Collagen - hydroxyapatite - gentamicine." Materiale Plastice 56, no. 3 (2019): 534–27. http://dx.doi.org/10.37358/mp.19.3.5224.

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The purpose of this study was the synthesis of two types of biodegradable materials with synthetic polymers (PLGA) or natural polymers (collagen) and hydroxyapatite, followed by determination of the encapsulation percentage of the drug in the polymer. Regardless of the chosen method, the percentage of the encapsulated drug was found to be quite high: 15.92% in the Coll-HA-Genta material and 19.59% respectively in the PLGA-Genta biocomposite. The therapeutic value of gentamicin was improved by encapsulating it in delivery systems, contributing to sustained release for a long time (about 30 days).
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Garg Satish, K., and B. D. Garg. "Cinétique d’absorption et d’élimination de la gentamicine chez le bufflon (Bubalus bubalis) après administration parentérale répétée." Revue d’élevage et de médecine vétérinaire des pays tropicaux 45, no. 3-4 (1992): 315–17. http://dx.doi.org/10.19182/remvt.8924.

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La cinétique d'absorption et d'élimination de la gentamicine a été déterminée chez des bufflons après administration parentérale répétée de 5 mg/kg de poids vif. Les demi-vie d'absorption (t 1/2Ka) et d'élimination (t 1/2 béta) ont été respectivement de 0,40 + ou - 0,12 et 4,33 + ou - 0,39 h. La comparaison statistique des valeurs des déterminants pharmacocinétiques établis dans la présente étude, avec les valeurs correspondantes obtenues après une seule injection intramusculaire de la même dose que dans une étude antérieure de Garg et Garg en 1990, a révélé que l'administration répétée de gentamicine influence son profil pharmacocinétique, la demi-vie d'élimination étant statistiquement plus longue (P < 0,05). La valeur constante du taux d'élimination étant diminuée, les doses ultérieures doivent être réduites, en particulier en cas d'insuffisance rénale. Dans le cas contraire, les doses de gentamicine à administrer n'ont pas lieu d'être modifiées.
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Lacarelle, Bruno, A. Baltasat, S. Bouquet, and N. Venisse. "Suivi thérapeutique pharmacologique de la gentamicine." EMC - Biologie Médicale 1, no. 1 (2006): 1–4. http://dx.doi.org/10.1016/s2211-9698(06)76297-5.

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KERDOUN, Mohamed Amine, Hadjer BALLOUTI, Yasmine GUERGOUR, et al. "Demonstration of inter and intraindividual pharmacokinetic variability during gentamicin treatment in hospitals." Batna Journal of Medical Sciences (BJMS) 5, no. 1 (2018): 53–56. http://dx.doi.org/10.48087/bjmsoa.2018.5113.

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Introduction : les aminosides et notamment la gentamicine possèdent un intérêt thérapeutique certain dans les infections sévères. Ces médicaments possèdent cependant un index thérapeutique étroit justifiant le suivi thérapeutique pharmacologique de ces derniers. L’objectif de cette étude est de montrer, par un cas clinique, l’importance relative des variabilités interindividuelles et intra-individuelles et l’intérêt du suivi thérapeutique de la gentamicine en milieu hospitalier pour éviter la survenue de toxicité et l’apparition de germes résistants. Observation : Nous présentons le cas d’une patiente de 53 ans pesant 88 kg, avec une créatininémie de 10,1 mg/l, est traitée par gentamicine durant sept jours. Ses paramètres pharmacocinétiques ont été estimés durant le traitement. La variabilité intra-individuelle des principaux paramètres a été quantifiée et comparé à la variabilité interindividuelle retrouvée dans la littérature. Discussion : Pour la demi-vie, la variabilité intra-individuelle représente la moitié de la valeur atteinte par la variabilité interindividuelle (25 versus 40 %). Celle de la clairance et du volume central est plus importante et supérieure à la variabilité interindividuelle (38 et 52 % versus 23 et 35 % respectivement). Conclusion : Cette forte variabilité pharmacocinétique a des conséquences cliniques importantes. A la notion de dose unique standard s’oppose des concentrations sanguines variées. Pour garantir l’efficacité du traitement, il est nécessaire d’évaluer périodiquement la situation des patients pour tenir compte de la variabilité pharmacocinétique
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Drachuk, V. M., I. I. Zamorskiy, and O. M. Horoshko. "ANTIOXIDANT POTENTIAL OF ADEMETIONINE IN GENTAMICINE NEPHROPATHY PROGRESSION." Актуальні проблеми сучасної медицини: Вісник Української медичної стоматологічної академії 20, no. 1 (2020): 8–12. http://dx.doi.org/10.31718/2077-1096.20.1.8.

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Acute kidney injury has been globally considered as a serious medical issue. Its incidence rate is increasing over the years despite the introduction of new therapies, the cause of which is often a side effect to drugs, or due to the impact of toxins, trauma. Nephrotoxicity is one of the most important side effects of aminoglycoside antibiotics, and on gentamicin, in particular. Despite rigorous patient monitoring, this type of nephrotoxicity appears in 10–30% of therapeutic courses. Therefore, searching for new therapies, which would be able to prevent or decrease toxic kidney injury during the treatment with aminoglycosides is quite relevant. Ademetionine is an amino acid derivative, which possesses various effects such as antioxidant, membrane stabilizing, anti-inflammatory and regenerative.
 Aim of the research is to study an antioxidative activity of ademetionine in conditions of gentamicin nephropathy progression in rats. Materials and methods. Research was conducted on non-linear mature white rats weighting 130-180 g, randomly divided into 3 groups (n = 7): I group included intact control animals, II group included rodents with gentamicin nephropathy (injection of 4% Gentamicin sulphate solution in a dosage of 80 mg/kg for 6 days), III group involved the rats receiving ademetionine («Abbott SpA», Italy) in a dosage of 20 mg/kg. Peroxidation processes in blood and kidneys were evaluated by the content of active products that react with thiobarbituric acid and oxidative modification of proteins levels, antioxidant defence was assessed by catalase and glutathione peroxidase activity, SH-groups and ceruloplasmin content.
 Results. In the course of experimental studies on the model of gentamicin nephropathy, the expressive antioxidant activity of ademetionine was proven: it was demonstrated by reducing the intensity of lipid peroxidation (a decrease in the content of active products that react with thiobarbituric acid) and proteins (a decrease in the content of oxidative modified proteins) in the blood and kidney homogenate, next with increased activity of the enzymatic link of antioxidant protection (glutathione peroxidase and catalase) and non-enzymatic (ceruloplasmin and compounds with SH-groups). The antioxidant potential of ademetionine has been confirmed by an increase in the antioxidant-prooxidant index in kidney tissue and a significant decrease in the index of oxidative stress in the blood of treated animals.
 Conclusion. Thus, the results of experimental studies indicate the antioxidative effect of ademetionine in modelled gentamicin nephropathy, and point out the necessity of further studying its potential nephroprotectective properties.
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Homs, J. B., and G. O’tterbein. "Toxicité de la gentamicine administrée par autotransfusion du sang drainé après chirurgie prothétique du genou utilisant un ciment à la gentamicine." Annales Françaises d'Anesthésie et de Réanimation 14, no. 2 (1995): 238. http://dx.doi.org/10.1016/s0750-7658(95)70027-2.

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Mahdi, Osama S. "Evaluation of inhibitory activity of extracts of Apium gravelens, Coriandrum sativum and Cuminum cyminum against number of pathogenic bacteria." Kufa Journal For Veterinary Medical Sciences 2, no. 2 (2011): 37–50. http://dx.doi.org/10.36326/kjvs/2011/v2i23912.

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Natural resources especially plants are useful bactericides for the control of bacterial infection .To evaluate the antibacterial potential of three types of plants, essential oils were extracted from the seeds of Apium gravelens, Coriandrum sativum and Cuminum cyminum and assayed in vitro for antibacterial activity aganist most prevalent pathogens Staphylococcus aureus, Salmonella spp.,Escherishia coli and Pseudomonas aeruginosa at 37 º C and 25 ºC.The antimicrobial effect was assessed using agar diffusion method by applying ethanolic solutions of extracts using it in two different temperatures 37 ºC and 25 ºC. The result shows that the extract of Apium gravelens, Coriandrum sativum and Cuminum cyminum alcoholic extract at 25 ºC exhibited an inhibition zone on S. aureus in the concentration 200 mg/ml and had a exhibited significantly (p < 0.05) greater than that produced by gentamicine,also the extract of Apium gravelens at 25 ºC in the concentration 100 mg/ml had a significant antibacterial activity on S. aureus, and the extract of Apium gravelens alcoholic extract at 25 ºC and 37 ºC pronounced antibacterial activity against Pseudomonas aeruginosa in the concentration 100 and 200 mg/ml respectively moreover the inhibition produced by gentamicine. While the antibacterial effect Apium gravelens and Coriandrum sativum at 37 ºC on S. aureus in the concentration 200 mg/ml show very similar effect with gentamicine. The antibacterial activity against Salmonella spp.and E. coli were moderate in action It was found that the investigated extracts of Apium gravelens , Coriandrum sativum and Cuminum cyminum were exhibited a considerable inhibitory effect against S. aureus and extracts of Apium gravelens and Coriandrum sativum against P. aerogene. The significant antibacterial activity appears promising.
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Eva, Jouvin, Estelle Moreau, Laeticia Lambert, and Anne-Claire Bursztejn. "Utilisation de la gentamicine dans l’épidermolyse bulleuse dystrophique dominante." Annales de Dermatologie et de Vénéréologie - FMC 1, no. 8 (2021): A231. http://dx.doi.org/10.1016/j.fander.2021.09.189.

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Dissertations / Theses on the topic "Gentamicine"

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Morin, Jean-Paul. "Etude des mécanismes de l'action nephrotoxique de la gentamicine." Rouen, 1987. http://www.theses.fr/1987ROUES046.

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La gentamicine s'accumule sélectivement dans les lysosomes des cellules tubulaires proximales de rein de rat ou de lapin, au sein desquels elle provoque une phospholipidose. L'étude morphométrique de la cellule tubulaire proximale en ultrastructure a révélé que la gentamicine provoque une augmentation de la densité de lysosomes par unité de volume cellulaire, et une augmentation de la taille de ces organites, parallèlement à la survenue d'une surcharge par des corps myéloïdes. L'étude biochimique du cortex rénal a révélé que la gentamicine provoque une diminution de l'activité des phospholipases A1, A2 et C et de la sphingomyélinase ayant pour conséquence la survenue de la phospholipidose. L'activité de la cathepsine B, le turnover des phosphoinositides membranaires et le transport cellulaire du calcium sont aussi diminués lors du traitement par la gentamicine. L'étude fonctionnelle a révélé que la gentamicine provoque l'apparition d'une polyurie, d'une élévation de l'excrétion urinaire de N-acetyl-β-D-glucosaminidase et de calcium, d'une forte diminution de l'excrétion urinaire d'AMP cyclique et peu de modifications de l'excrétion urinaire de sodium, de potassium, de magnésium et de phosphore. L'étude de l'éventuelle modulation de la toxicité rénale de la gentamicine par l'administration conjointe de Muzolimine* (un diurétique de l'anse), de Perindopril* (un inhibiteur de l'enzyme de conversion) ou d'une surcharge alimentaire en calcium a montré que : - la surcharge calcique protège contre l'atteinte fonctionnelle du rein. - L'administration conjointe d'un diurétique de l'anse ne modifie pas l'atteinte rénale induite par la seule gentamicine. - L'inhibition du système rénine angiotensine potentialise la toxicité rénale de la gentamicine. Les deux cibles privilégiées de la toxicité rénale de la gentamicine sont l'appareil lysosomal d'une part et d'autre part la membrane péricellulaire du pôle apical des cellules tubulaires proximales. Le rôle de l'obstruction tubulaire dans le développement de l'insuffisance rénale aiguë provoquée par la gentamicine est discutée.
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LeBrun, Michel. "Signalisation intracellulaire et mécanismes de néphrotoxicité de la gentamicine." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ60775.pdf.

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Robert, Nathalie. "La néphrotoxicité de la gentamicine et les intéractions médicamenteuses." Paris 5, 1992. http://www.theses.fr/1992PA05P086.

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Morin, Jean-Paul. "Etude des mécanismes de l'action néphrotoxique de la gentamicine." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37608163g.

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Benkirane, Abdelkrim. "Emergence de "Staphylococcus aureus" résistants à la méticilline et sensibles à la gentamicine en milieu hospitalier entre 1993 et 1995 : aspects microbiologiques et épidémiologiques." Paris 5, 1997. http://www.theses.fr/1997PA05P070.

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Mugabe, Clement, and Abdelwahab Omri. "La gentamicine sous la forme liposomale : aspects technologique et microbiologique." Acfas-Sudbury, 2005. https://zone.biblio.laurentian.ca/dspace/handle/10219/63.

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Poirier, André. "Influence de l'infection rénale sur la néphrotoxicité de la gentamicine." Master's thesis, Université Laval, 1985. http://hdl.handle.net/20.500.11794/33572.

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Dans le but d'évaluer l'influence de l'infection rénale sur la toxicité de la gentamicine, une pyélonéphrite expérimentale à Escherichia coli est induite chez les rats. Plusieurs groupes d'animaux : infectés, traités ou infectés et traités ont été étudiés. Deux traitements à la gentamicine furent institués: 1- 10 mg/kg Q8H et 2- 30 mg/kg Q8H, pendant 3 jours. Plusieurs paramètres de toxicité rénale ont été analysés soient : la diurèse, l'osmolalité urinaire, la protéinurie, la B-galactosurie et Y-glutamyltransférase. Que ce soit a basse ou à haute dose, l'infection rénale semble potentialiser la néphrotoxicité de la gentamicine<br>Montréal Trigonix inc. 2018
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SARTRE, JACQUES. "Determination de l'effet bactericide, sur des souches de staphylocoques, des glycopeptides associes a une fluoroquinolone ou a un aminoside, par une methode cinetique, au moyen de l'ensemenceur spiral." Lyon 1, 1990. http://www.theses.fr/1990LYO1M317.

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Brunschwig, Caroline. "Entérocoques résistants aux béta-lactamines : vitesses de bactéricidie d'associations d'antibiotiques : étude in vitro." Montpellier 1, 1991. http://www.theses.fr/1991MON11186.

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Bunetel, Laurence. "Ciments et antibiotique : étude expérimentale de la diffusion." Rennes 1, 1988. http://www.theses.fr/1988REN1B006.

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Books on the topic "Gentamicine"

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Mugabe, Clement. Contribution à l'étude de l'incorporation de la gentamicine dans les vésicules phospholipidiques. Université Laurentienne, 2003.

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Rhomberg, Brigitte. Einheilung einer [beta]-Tricalciumphosphat-Gentamicin-Kombination [Beta-Tricalciumphosphat-Gentamicin-Kombination] im Tierversuch. [s.n.], 1986.

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Maes, R. A. A. 1937-, Oellerich Michael, and Deutsche Forschungsgemeinschaft, eds. Tobramycin profile. New York, 1988.

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Seguin, Catherine L. Gentamicin: A spin-label electron spin resonance study. Laurentian University, 2004.

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Ho, Katherine Ka-Ling. Gentamicin pharmacokinetics, efficacy and nephrotoxicity in pediatric oncology patients. National Library of Canada, 1990.

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Simjee, Shabbir Ahmed Salim. Molecular characterisation of mobile high-level gentamicin resistance determinants in enterococci. University of Birmingham, 1998.

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Cheng, Judy W. M. Implementation and evaluation of impact of the formulary conversion of tobramycin to gentamicin. St. Michael's Hospital, 1991.

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Rukholm, Gavin. Liposomal delivery of gentamicin: A time-killing study against clinical isolates of Pseudomonas aeruginosa. Laurentian University, 2004.

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Barrington, Frank. Conjugative transfer of high-level gentamicin resistance in clinical and community isolates of enterococci. The Author], 1993.

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Dawson, Pamela. The effect of gentamicin therapy on urinary excretion of retinol-binding protein, lysozyme and electrolytes. Aston University. Department of Pharmaceutical Sciences, 1986.

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Book chapters on the topic "Gentamicine"

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Beyer, Karl-Heinz. "Gentamicin." In Biotransformation der Arzneimittel. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74386-3_148.

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Vidal, C., and W. R. Külpmann. "Gentamicin." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_1223-1.

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Vidal, C., and W. R. Külpmann. "Gentamicin." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_1223.

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Wilson, John Fawcett. "Gentamicin." In The Immunoassay Kit Directory. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0679-5_22.

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de Groot, Anton C. "Gentamicin." In Monographs In Contact Allergy. CRC Press, 2022. http://dx.doi.org/10.1201/9781003158004-238.

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Rodriguez, H. H., and O. Martinez-Alvarez. "Hidrotalcite with Gentamicine, of the Type Mg0.68Al0.32(OH)2(NO3)0.32*0.1H2O, Formed by Chemical Coprecipitation in Controlled Atmosphere." In Supplemental Proceedings. John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118356074.ch22.

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Dafotakis, M. "Gentamicin-induzierte Vestibulopathie." In Komplikationen in der Neurologie. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-47880-6_14.

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Klemm, K., and R. Schnettler. "Gentamicin-PMMA-Ketten." In Chirurgische Infektionen von Knochen, Gelenken und Weichteilen, edited by K. H. Schultheis, K. E. Rehm, and H. Ecke. De Gruyter, 1991. http://dx.doi.org/10.1515/9783110886511-007.

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Schomburg, Dietmar, and Dörte Stephan. "Gentamicin 2”-nucleotidyltransferase." In Enzyme Handbook. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59025-2_127.

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Chan, M. K., and W. L. Ng. "Silymarin Ameliorates Gentamicin Nephrotoxicity." In Nephrotoxicity. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-2040-2_29.

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Conference papers on the topic "Gentamicine"

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Morris, James, Dulip Jayasinghe, Adriece Al Rifai, and Stella Tse. "P49 Gentamicin safety in neonatal sepsis following maternal gentamicin given during labour." In Abstracts from the NPPG Conference 2023. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/bmjpo-2024-nppg.59.

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Gianecini, Ricardo Ariel, Noelia Cuenca, Melisa Gonzalez, Paula Cristaldo, Claudia Oviedo, and Patricia Galarza. "Sensibilidad a gentamicina en aislamientos de neisseria gonorrhoeae con fenotipos MDR Y NO-MDR en Argentina, 2017–2019." In XIII Congresso da Sociedade Brasileira de DST - IX Congresso Brasileiro de AIDS - IV Congresso Latino Americano de IST/HIV/AIDS. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/dst-2177-8264-202133p127.

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Introducción : Actualmente, la emergencia de aislamientos de Neisseria gonorrhoeae resistentes a la azitromicina y/o ceftriaxona pone en riesgo la efectividad de la terapia antimicrobiana dual. Gentamicina en combinación con azitromicina surge como alternativa terapéutica principalmente ante falla de tratamiento y/o pacientes alérgicos a cefalosporinas. Objetivo: El objetivo de este estudio fue determinar la actividad in vitro de gentamicina en aislamientos de N. gonorrhoeae con fenotipo multirresistente (MDR) y no-MDR en Argentina. Métodos: Se estudiaron 2595 aislamientos colectados por el PROVSAG entre 2017 y 2019. La CIM a los antimicrobianos se realizó de acuerdo al documento CLSI M07-A10. Un aislamiento MDR fue definido como: sensibilidad disminuida/resistencia a un antimicrobiano actualmente recomendado (ceftriaxona, cefixima o azitromicina), más resistencia, al menos a otros dos antimicrobianos (penicilina-G, tetraciclina y/o ciprofloxacina). Resultados: Un alto porcentaje de aislamientos resulto resistente a penicilina-G (47,1%), tetraciclina (34,3%) y ciprofloxacina (74,3%). Aislamientos no-sensibles a azitromicina (CIM:8805,2μg/mL) se incrementaron de 1,7% (2017) a 4,2% (2019). Aunque no se detectaron aislamientos resistentes a ceftriaxona, un total de 2,3% (2017) y 4,6% (2019) mostraron valores de CIM 8805,0,06 μg/mL (considerados con sensibilidad-reducida). Un 7,0% del total de aislamientos presento un fenotipo MDR. Las CIMs a gentamicina estuvieron comprendidas entre 1–16 956,g/mL y la CIM50/90 fue 8 μ/mL. No se observaron diferencias significativas en las CIMs a gentamicina en aislamientos con fenotipo MDR y no-MDR. Los aislamientos con fenotipo MDR y no-MDR en Argentina no mostraron resistencia a gentamicina. Sin embargo, en similitud a estudios en otras regiones geográficas, un elevado porcentaje de aislamientos mostró sensibilidad intermedia (CIM:8–16 μg/mL). Conclusion: Estos resultados muestran a gentamicina como una posible opción de tratamiento en Argentina. La emergencia de aislamientos de N. gonorrhoeae con fenotipo MDR llevan a la necesidad de un fortalecimiento de los sistemas de vigilancia para la detección de resistencias emergente y búsqueda de nuevas opciones tratamiento.
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Trah, Julian, Philipp Deindl, Alexandra Luister, Claudia Langebrake, Dominique Singer, and Chinedu Ebenebe. "Risikofaktoren für supratherapeutische Gentamicin-Talspiegeln bei Neugeborenen." In Abstracts zur 49. Jahrestagung der Gesellschaft fär Neonatologie und Pädiatrische Intensivmedizin (GNPI). Georg Thieme Verlag KG, 2023. http://dx.doi.org/10.1055/s-0043-1769392.

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Chen, Jim S., Narasimham Pulugundla, Marla R. Wolfson, and Thomas H. Shaffer. "Pharmacokinetics of gentamicin by intravenous and intratracheal administrations." In 2014 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2014. http://dx.doi.org/10.1109/spmb.2014.7002952.

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O’Hagan, Shaun, Peter Mallett, Lynne Speirs, et al. "1266 Standardising care – Northern Ireland regional paediatric gentamicin chart." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.519.

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Ortega-Garcia, MP, M. Zaragozá- González, P. Pérez-Villalón, et al. "4CPS-239 Therapeutic drug monitoring of gentamicin in neonates." In 25th Anniversary EAHP Congress, Hospital Pharmacy 5.0 – the future of patient care, 23–28 March 2021. British Medical Journal Publishing Group, 2021. http://dx.doi.org/10.1136/ejhpharm-2021-eahpconf.71.

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Halleron, Kevin, Mary-Beth Toner, and Bharathi Rao. "1285 Postnatal gentamicin administration and prescription: mind the gap!" In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Liverpool, 28–30 June 2022. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2022. http://dx.doi.org/10.1136/archdischild-2022-rcpch.778.

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Eljarrat-Binstock, Esther, Frederik Raiskup, Joseph Frucht-Pery, and Abraham J. Domb. "Hydrogel iontophoresis for gentamicin administration to the rabbit eye." In Biomedical Optics 2005, edited by Fabrice Manns, Per G. Soederberg, Arthur Ho, Bruce E. Stuck, and Michael Belkin. SPIE, 2005. http://dx.doi.org/10.1117/12.593844.

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9

van den Heuvel, Robert. "Gentamicin improves symptoms in paediatric Nagashima-type palmoplantar keratosis." In 25th World Congress of Dermatology, edited by Peter van de Kerkhof. Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/3d7a0ada.

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Wójcik, Michał, Anna Witczyńska, Anna Wilczyńska, and Agata Przekora. "Biopolymer-based gentamicin-enriched biomaterial as a wound dressing." In 1st International PhD Student’s Conference at the University of Life Sciences in Lublin, Poland: ENVIRONMENT – PLANT – ANIMAL – PRODUCT. Publishing House of The University of Life Sciences in Lublin, 2022. http://dx.doi.org/10.24326/icdsupl1.a034.

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Reports on the topic "Gentamicine"

1

Brown, Michael. Effect of Ototoxic Drugs on the Amphibian Auditory System: Injection of Gentamicin Sulfate into Anuran Otic Capsules and Recovery of Thresholds. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.6734.

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Jorgensen, Frieda, Andre Charlett, Craig Swift, Anais Painset, and Nicolae Corcionivoschi. A survey of the levels of Campylobacter spp. contamination and prevalence of selected antimicrobial resistance determinants in fresh whole UK-produced chilled chickens at retail sale (non-major retailers). Food Standards Agency, 2021. http://dx.doi.org/10.46756/sci.fsa.xls618.

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Abstract:
Campylobacter spp. are the most common bacterial cause of foodborne illness in the UK, with chicken considered to be the most important vehicle for this organism. The UK Food Standards Agency (FSA) agreed with industry to reduce Campylobacter spp. contamination in raw chicken and issued a target to reduce the prevalence of the most contaminated chickens (those with more than 1000 cfu per g chicken neck skin) to below 10 % at the end of the slaughter process, initially by 2016. To help monitor progress, a series of UK-wide surveys were undertaken to determine the levels of Campylobacter spp. on whole UK-produced, fresh chicken at retail sale in the UK. The data obtained for the first four years was reported in FSA projects FS241044 (2014/15) and FS102121 (2015 to 2018). The FSA has indicated that the retail proxy target for the percentage of highly contaminated raw whole retail chickens should be less than 7% and while continued monitoring has demonstrated a sustained decline for chickens from major retailer stores, chicken on sale in other stores have yet to meet this target. This report presents results from testing chickens from non-major retailer stores (only) in a fifth survey year from 2018 to 2019. In line with previous practise, samples were collected from stores distributed throughout the UK (in proportion to the population size of each country). Testing was performed by two laboratories - a Public Health England (PHE) laboratory or the Agri-Food &amp; Biosciences Institute (AFBI), Belfast. Enumeration of Campylobacter spp. was performed using the ISO 10272-2 standard enumeration method applied with a detection limit of 10 colony forming units (cfu) per gram (g) of neck skin. Antimicrobial resistance (AMR) to selected antimicrobials in accordance with those advised in the EU harmonised monitoring protocol was predicted from genome sequence data in Campylobacter jejuni and Campylobacter coli isolates The percentage (10.8%) of fresh, whole chicken at retail sale in stores of smaller chains (for example, Iceland, McColl’s, Budgens, Nisa, Costcutter, One Stop), independents and butchers (collectively referred to as non-major retailer stores in this report) in the UK that are highly contaminated (at more than 1000 cfu per g) with Campylobacter spp. has decreased since the previous survey year but is still higher than that found in samples from major retailers. 8 whole fresh raw chickens from non-major retailer stores were collected from August 2018 to July 2019 (n = 1009). Campylobacter spp. were detected in 55.8% of the chicken skin samples obtained from non-major retailer shops, and 10.8% of the samples had counts above 1000 cfu per g chicken skin. Comparison among production plant approval codes showed significant differences of the percentages of chicken samples with more than 1000 cfu per g, ranging from 0% to 28.1%. The percentage of samples with more than 1000 cfu of Campylobacter spp. per g was significantly higher in the period May, June and July than in the period November to April. The percentage of highly contaminated samples was significantly higher for samples taken from larger compared to smaller chickens. There was no statistical difference in the percentage of highly contaminated samples between those obtained from chicken reared with access to range (for example, free-range and organic birds) and those reared under standard regime (for example, no access to range) but the small sample size for organic and to a lesser extent free-range chickens, may have limited the ability to detect important differences should they exist. Campylobacter species was determined for isolates from 93.4% of the positive samples. C. jejuni was isolated from the majority (72.6%) of samples while C. coli was identified in 22.1% of samples. A combination of both species was found in 5.3% of samples. C. coli was more frequently isolated from samples obtained from chicken reared with access to range in comparison to those reared as standard birds. C. jejuni was less prevalent during the summer months of June, July and August compared to the remaining months of the year. Resistance to ciprofloxacin (fluoroquinolone), erythromycin (macrolide), tetracycline, (tetracyclines), gentamicin and streptomycin (aminoglycosides) was predicted from WGS data by the detection of known antimicrobial resistance determinants. Resistance to ciprofloxacin was detected in 185 (51.7%) isolates of C. jejuni and 49 (42.1%) isolates of C. coli; while 220 (61.1%) isolates of C. jejuni and 73 (62.9%) isolates of C. coli isolates were resistant to tetracycline. Three C. coli (2.6%) but none of the C. jejuni isolates harboured 23S mutations predicting reduced susceptibility to erythromycin. Multidrug resistance (MDR), defined as harbouring genetic determinants for resistance to at least three unrelated antimicrobial classes, was found in 10 (8.6%) C. coli isolates but not in any C. jejuni isolates. Co-resistance to ciprofloxacin and erythromycin was predicted in 1.7% of C. coli isolates. 9 Overall, the percentages of isolates with genetic AMR determinants found in this study were similar to those reported in the previous survey year (August 2016 to July 2017) where testing was based on phenotypic break-point testing. Multi-drug resistance was similar to that found in the previous survey years. It is recommended that trends in AMR in Campylobacter spp. isolates from retail chickens continue to be monitored to realise any increasing resistance of concern, particulary to erythromycin (macrolide). Considering that the percentage of fresh, whole chicken from non-major retailer stores in the UK that are highly contaminated (at more than 1000 cfu per g) with Campylobacter spp. continues to be above that in samples from major retailers more action including consideration of interventions such as improved biosecurity and slaughterhouse measures is needed to achieve better control of Campylobacter spp. for this section of the industry. The FSA has indicated that the retail proxy target for the percentage of highly contaminated retail chickens should be less than 7% and while continued monitoring has demonstrated a sustained decline for chickens from major retailer stores, chicken on sale in other stores have yet to meet this target.
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