Relevant bibliographies by topics / Gestational trophoblastic disease. eng

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  2. Dissertations / Theses
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Academic literature on the topic 'Gestational trophoblastic disease. eng'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Gestational trophoblastic disease. eng"

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Begum, Shirin Akter, Md Zillur Rahman Bhuiyan, Rehana Akhter, Romena Afroz, Afroza Khanom, and Kashfia Ahmed Keya. "A review on gestational trophoblastic disease." Bangladesh Medical Journal 44, no. 1 (January 12, 2016): 51–56. http://dx.doi.org/10.3329/bmj.v44i1.26357.

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Molar pregnancy occurs when the fertilization of the egg by the sperm goes wrong and leads to the growth of abnormal cells or clusters of water filled sacs inside the womb. This condition is one of a group of conditions known as gestational trophoblastic tumours (GTTs). Molar pregnancies used to be called hydatidiform mole but now most people call them molar pregnancies. Molar pregnancies are rare but they are the most common type of gestational trophoblastic tumour. In the UK, about 1 in 590 pregnancies is a molar pregnancy. In Asian women, molar pregnancies are about twice as common as in Caucasian women. Most molar pregnancies are benign. They can spread beyond the womb in some women, but are still curable. Molar pregnancies can either be complete or partial. In case of complete mole, no parts of foetal tissue are formed. In case of partial mole there may be some foetal tissue in the womb, alongside the molar tissue. By measuring the levels of ?hCG in blood and urine in high dilution helps to diagnose a molar pregnancy; an ultrasound scan can also diagnose many women with molar pregnancy. The molar tissue needs to be surgically removed. Afterwards, in around 10 to 15 out of 100 women, some molar tissue remains in the deeper tissues of the womb or other parts of the body. This is called a persistent gestational tumour. Invasive mole, choriocarcinoma, and placental site trophoblastic tumor (PSTT) termed as “gestational trophoblastic neoplasia” (GTN), which can progress, invade, metastasize, and lead to death if left untreated.These women need to have chemotherapy completely get rid of the abnormal cells.Bangladesh Med J. 2015 Jan; 44 (1): 51-56
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Lehman, E., D. M. Gershenson, T. W. Burke, C. Levenback, E. G. Silva, and M. Morris. "Salvage surgery for chemorefractory gestational trophoblastic disease." Journal of Clinical Oncology 12, no. 12 (December 1994): 2737–42. http://dx.doi.org/10.1200/jco.1994.12.12.2737.

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PURPOSE To investigate and define better the role of salvage surgery for patients with chemorefractory gestational trophoblastic disease (GTD). PATIENTS AND METHODS A retrospective review of medical records at The University of Texas M.D. Anderson Cancer Center identified 33 patients with chemorefractory GTD who underwent salvage surgery between 1962 and 1991. The end points selected for analysis were serologic response and survival. RESULTS Initial salvage procedures consisted of 29 hysterectomies, four thoracotomies, and one nephrectomy (in conjunction with a hysterectomy). Fourteen patients (42%) had a serologic complete response (CR) to surgery (normalization of human chorionic gonadotropin [hCG]), 10 (30%) had a partial response (> 50% decrease in hCG level), and nine had no response (< or = 50% decrease in hCG level). Of 19 patients who received further chemotherapy, eight (42%) attained a CR. Four patients underwent a second salvage surgery: two thoracotomies, one craniotomy, and one partial hepatectomy. All achieved a CR. The probability of achieving a CR was influenced by the time from diagnosis to surgery, number of preoperative disease sites, preoperative World Health Organization (WHO) score, and histologic type. Survival was influenced by the type of antecedent pregnancy, number of preoperative regimens, number of preoperative disease sites, time from diagnosis to surgery, and preoperative WHO score. CONCLUSION Based on the findings of this study, it appears that a select subset of patients with chemorefractory GTD who have a limited number of clinically detectable tumor foci may benefit from salvage surgery.
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Kubelka-Sabit, KB, I. Prodanova, D. Jasar, G. Bozinovski, V. Filipovski, S. Drakulevski, and D. Plaseska-Karanfilska. "Molecular and immunohistochemical characteristics of complete hydatidiform moles." Balkan Journal of Medical Genetics 20, no. 1 (June 30, 2017): 27–34. http://dx.doi.org/10.1515/bjmg-2017-0009.

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AbstractMolar pregnancy is a gestational trophoblastic disease that belongs to the category of precancerous lesions. On the other end of the spectrum are gestational trophoblastic neoplasms such as invasive mole, choriocarcinoma, placental site trophoblastic tumor and epithelioid trophoblastic tumor, which are considered malignant tumors. Based on defined histopathological criteria, molar pregnancy is divided into partial and complete hydatidiform mole. Especially in the case of early complete mole, the diagnosis can be quite challenging and often necessitates additional molecular or immunohistochemical methods. The aim of this study was to assess the importance of additional molecular and immunohistochemical methods to accurately diagnose complete hydatidiform mole and to stress the importance of correct diagnosis and close follow-up of these patients. A total of 367 consecutive cases of spontaneous abortion were analyzed in a 3-year period. Eight cases with histopathological diagnosis of complete molar pregnancy were selected for further analysis. Apart from standard microscopic analysis, additional molecular and immunohistochemical analyses were performed in all eight cases. Most of the histopathological characteristics of complete molar pregnancy were present in all cases, together with complete absence of positivity for the p57 immunohistochemical marker in the cytotrophoblasts and villous stromal cells. The molecular analysis revealed androgenetic diploidy in seven cases and biparental diploidy in one case with more than three consecutive complete molar pregnancies. Additional immunohistochemical and molecular methods can considerably aid in the correct diagnosis of molar pregnancy.
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Deep, JP, LB Sedhai, J. Napit, and J. Pariyar. "Gestational Trophoblastic Disease." Journal of Chitwan Medical College 3, no. 2 (August 13, 2013): 4–11. http://dx.doi.org/10.3126/jcmc.v3i2.8434.

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Gestational trophoblastic disease (GTD) is a group of tumors that arise from placental tissue and secrete β-hCG. GTD is a combination of benign or invasive mole and malignant known as Gestational Trophoblastic Neoplasia (GTN). Prevalence, diagnosis and treatment of GTD have drastically changed in recent years. DOI: http://dx.doi.org/10.3126/jcmc.v3i2.8434 Journal of Chitwan Medical College Vol.3(2) 2013 4-11
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Bolck, F. "Gestational Trophoblastic Disease." Experimental pathology 35, no. 1 (January 1988): 56. http://dx.doi.org/10.1016/s0232-1513(88)80123-3.

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Bannatyne, Patricia M. "Gestational Trophoblastic Disease." Pathology 20, no. 4 (1988): 401. http://dx.doi.org/10.1016/s0031-3025(16)36572-2.

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Zivaljevic, Milica, Marija Tesic, Tamara Vujkov, Jelka Rajovic, and Marina Popovic. "Gestational trophoblastic disease." Archive of Oncology 10, no. 2 (2002): 71–75. http://dx.doi.org/10.2298/aoo0202071z.

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Gestational trophoblastic disease belongs to a spectrum of rare tumors originating from trophoblast. It spreads from the benignant disease uncomplicated partial mole to the most malignant choriocarcinoma in stage IV of disease with brain metastases. Fortunately, with adequate chemotherapy even patients in advanced stage of the disease have significant chances to be cured. In estimating prognosis and adequate therapy of disease, the most significant are clinical factors: serum hCG level, duration of the disease from termination of antecedent pregnancy, prior chemotherapy, brain or liver metastases. hCG is an ideal tumor marker for follow up and early diagnosis of recidivism and metastases. In the Institute of Oncology in Sremska Kamenica 32 patients with gestational trophoblastic disease were treated in the period from 1987 to 2001. All the patients with non-metastatic disease and low risk metastatic disease (stage I-III FIGO) were successfully cured. Five patients died, all in stage IV of the disease (FIGO) with liver and brain metastases; in 4 of them disease occurred after term pregnancy. Overall survival was 85%. Treatment of non-metastatic and low risk metastatic disease was successful in all cases. Treatment failures occurred in advanced disease with brain and liver metastasis Specificity and low incidence of this disease ask for the treatment to be carried out in specialized centers, as it is in developed countries (Trophoblastic Disease Centers).
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Shapter, Anne P., and Robert McLellan. "GESTATIONAL TROPHOBLASTIC DISEASE." Obstetrics and Gynecology Clinics of North America 28, no. 4 (December 2001): 805–17. http://dx.doi.org/10.1016/s0889-8545(05)70237-0.

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Tse, Ka Yu, Karen K. L. Chan, Kar Fai Tam, and Hextan Y. S. Ngan. "Gestational trophoblastic disease." Obstetrics, Gynaecology & Reproductive Medicine 19, no. 4 (April 2009): 89–97. http://dx.doi.org/10.1016/j.ogrm.2008.12.002.

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Shanbhogue, Alampady K. P., Neeraj Lalwani, and Christine O. Menias. "Gestational Trophoblastic Disease." Radiologic Clinics of North America 51, no. 6 (November 2013): 1023–34. http://dx.doi.org/10.1016/j.rcl.2013.07.011.

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Dissertations / Theses on the topic "Gestational trophoblastic disease. eng"

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Soares, Patrícia Daniela Paranhos Batista. "Distribuição geográfica e características demográficas da doença trofoblástica gestacional em centro de referência terciária do Estado da Bahia, Brasil /." Botucatu : [s.n.], 2009. http://hdl.handle.net/11449/91394.

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Resumo: Traçar um padrão de distribuição geográfica da doença trofoblástica gestacional (DTG) em centro de referência do estado da Bahia, no Nordeste do Brasil e determinar as características demográficas na apresentação da doença. Estudo observacional descritivo com dados obtidos de prontuários de 140 pacientes com DTG encaminhadas ao Centro de Doença Trofoblástica Gestacional da Maternidade Climério de Oliveira, no estado da Bahia, Brasil, de 2002 a 2007. Foi feita uma distribuição geográfica das pacientes com DTG, nas macrorregiões de saúde e foram avaliadas variáveis demográficas, fonte de referência, e tipo de gestação antecedente. Para análise estatística foi usado teste qui-quadrado (p<0,05%). A principal procedência das pacientes foi da macrorregião de saúde Leste (77,9%). A incidência da DTG no centro de referência foi de 8,5/1.000 partos. A faixa etária foi predominante de 20 a 34 anos (65%). Uma pequena proporção de pacientes estava trabalhando (42,9%). O nível educacional foi baixo: 67,9% das pacientes cursaram apenas o ensino fundamental. Hospitais secundários foram a principal fonte de referência de pacientes (84,3%). A maioria das pacientes teve gestação de termo prévia à DTG (42,1%). Neste estudo, a DTG predominou na melhor faixa etária para fecundidade e em pacientes com aspectos sócio-demográficos desfavoráveis. A tendência de referência dessas pacientes foi principalmente da macrorregião de saúde Leste.
Abstract: To outline the geographical distribution pattern of gestational trophoblastic disease (GTD) in a referral center located in the state of Bahia, northeastern Brazil, and to determine the demographic characteristics in the presentation of the disease. Observational, descriptive study of the data retrieved from the medical records of 140 GTD patients referred to the Gestational Trophoblastic Disease Center of Climério de Oliveira Maternity, Bahia, Brazil between 2002 and 2007. The geographical distribution of GTD patients across healthcare macroregions was determined and demographic variables, referral sources and type of previous gestation were assessed. Statistical analysis was performed using the chi-square test (p<0.05%). Results: The majority of the patients originated from the East Healthcare Macroregion (77.9%). DTG incidence at the referral Center was 8.5/1.000 deliveries. The 20-34-year age group predominated (65%). A small percentage of the patients was employed (42.9%). Education level was low: 67.9% of the patients attended only elementary school. Secondary hospitals were the principal sources of patient referral (84.3%). In most cases (42.1%), GTD was preceded by term gestation. DTG predominated in the peak fertility age group and among patients of unfavorable socio-demographic status. Most referred patients tended to come from the East Healthcare Macroregion.
Orientador: Marilza Vieira Cunha Rudge
Coorientador: Izildinha Maestá
Coorientador: Olívia Lúcia Nunes Costa
Banca: Raul Cortes Charry
Banca: Sue Iazaki Sua
Mestre
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Braga, Neto Antônio Rodrigues. "Influência da quimioterapia para neoplasia trofoblástica gestacional sobre a gravidez: resultados maternos e perinatais /." Botucatu : [s.n.], 2007. http://hdl.handle.net/11449/95363.

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Resumo: Objetivo. Avaliar a influência da quimioterapia no resultado da primeira gravidez após a neoplasia trofoblástica gestacional (NTG). Pacientes e Métodos. Trata-se do estudo observacional restropsectivo e comparativo, realizado em 3590 pacientes acompanhadas com doença trofoblástica gestacional (DTG) no centro de NTG da Santa Casa Misericórdia do Rio de Janeiro, entre janeiro de 1960 e dezembro de 2005. Destas 867 reternaram grávidas e tiveram seus resultados obstétricos e perinatais confrontados entre grupos que cursaram com remissão espontânea (RE) e NTG, subgrupos de pacientes com NTG que engravidaram com menos de 6 meses, entre os 6 e 12 meses e com mais de 12 meses da última sessão de quimioterapia, e que receberam quioterapia por agente único (NTG-M) ou multiplos agentes (NTG-P)... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Objective. To evaluate the influence to chemoterapy on the results of the first pregnancy after gestational trophoblastic neoplasia (GTN). Patients and methods. It is an obsrvational, retrospective and comparative study comprising 3590 patients followed for gestational trophoblastic disease (GTD) at Santa Casa de Misericórdia do Rio de Janeiro GTN Center between January 1960 and December 2005... (Complete abstract click electronic access below)
Orientador: Izildinha Maestá
Coorientador: Odair Carlito Michelin
Banca: Luiz Camano
Banca: Paulo Belfort
Mestre
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Ferreira, Érika Goulart Veloso. "Avaliação da qualidade de vida e aspectos psicológicos em pacientes com doença trofoblástica gestacional /." Botucatu : [s.n.], 2008. http://hdl.handle.net/11449/99253.

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Resumo: Clicar acesso eletrônico abaixo.
Abstract: Objective: to assess quality of life and psychological aspects in patients with gestational trophoblastic disease (GTD). Methods: This cross-sectional self-report study was carried out among 54 women treated at Botucatu Trophoblastic Diseases Center, São Paulo, Brazil. Validated questionnaires were used to assess quality of life (QoL) (WHOQOL-bref), symptoms of depression (Beck Depression Inventory-BDI) and anxiety (State-Trait Anxiety Inventory -STAI). Results: Most patients rated overall QoL as good (44.44%), and were satisfied with their health status (42.59%). However, these findings did not reach statistical significance. Mean QoL domain score was the highest for psychological health (53.86 ± 21.46), and the lowest for social relationships (65.74 ± 22.41). BDI mean was 15.81 ± 11.15, indicating dysphoria. STAI means were 46 ± 6.46 for trait-anxiety, and 43.72 ± 4.23 for state-anxiety, both evidencing medium-high anxiety. Among patients who were employed, the environment domain mean was the highest (p=0.024). The presence of children prior to disease onset resulted in the lowest means for physical health (p=0,041) and environment (p=0.045). The patients desiring to have children showed significantly higher means for physical health (p=0.004), psychological health (p=0.021) and environment (p=0.003). The need for chemotherapy to achieve complete response had no significant influence on QoL. Conclusion: This study evidenced the psychological impact on GTD patients. This information suggests that specialized care centers should provide psychological interventions during the treatment and follow-up of GTD patients... (Complete abstract click electronic access below)
Orientador: Marilza Vieira Cunha Rudge
Coorientador: Izildinha Maestá
Banca: Rafael Cortes-Chary
Banca: Olivia Lucia Costa
Mestre
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Lai, Yau-lin Caroline, and 黎幼蓮. "Genotyping of gestational trophoblastic disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hdl.handle.net/10722/209581.

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Lai, Yau-lin Caroline. "Genotyping of gestational trophoblastic disease." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23427383.

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Cheung, Nga-yin Annie. "Pathobiological study of gestational trophoblastic disease." Click to view the E-thesis via HKUTO, 1999. http://sunzi.lib.hku.hk/hkuto/record/B31981690.

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Cheung, Nga-yin Annie, and 張雅賢. "Pathobiological study of gestational trophoblastic disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31981690.

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何柏松 and Pak-chung Ho. "Immunological studies in gestational trophoblastic disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1989. http://hub.hku.hk/bib/B31981343.

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方佩儀 and Pui-yee Fong. "Differential gene expression in gestational trophoblastic disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31224362.

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Lee, Lee, and 李莉. "The ASPP family in gestational trophoblastic disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40738486.

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Books on the topic "Gestational trophoblastic disease. eng"

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Freedman, Ralph S. Gestational trophoblastic disease. Bethesda, MD (Bldg. 82, Rm. 103, Bethesda 20892): U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Cancer Institute, International Cancer Research Data Bank, 1988.

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Hui, Pei, ed. Gestational Trophoblastic Disease. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-61779-394-3.

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Szulman, Aron E., and Herbert J. Buchsbaum, eds. Gestational Trophoblastic Disease. New York, NY: Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4698-5.

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Nayak, Bhagyalaxmi, and Uma Singh, eds. Gestational Trophoblastic Disease. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-4878-3.

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Gestational trophoblastic disease: Diagnostic and molecular genetic pathology. New York: Springer, 2012.

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World Congress on Gestational Trophoblastic Disease (4th 1988 Beijing, China). Papers from the IV World Congress on Gestational Trophoblastic Disease, Beijing, China, September 1988. [New Haven, Conn: E.I. Kohorn, 1989.

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E, Szulman Aron, and Buchsbaum Herbert J. 1934-, eds. Gestational trophoblastic disease. New York: Springer-Verlag, 1987.

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H, Ngan, ed. Gestational trophoblastic disease. London: Baillière Tindall, 2003.

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W, Hancock Barry, Newlands E. S, and Berkowitz Ross Stuart, eds. Gestational trophoblastic disease. London: Chapman & Hall Medical, 1997.

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Buchsbaum, Herbert J., and Aron E. Szulman. Gestational Trophoblastic Disease. Springer, 1988.

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Book chapters on the topic "Gestational trophoblastic disease. eng"

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Hui, Pei. "Gestational Choriocarcinoma." In Gestational Trophoblastic Disease, 127–37. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-61779-394-3_8.

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Ladner, H. A., and S. Ladner. "Gestational Trophoblastic Disease." In Radiation Oncology of Gynecological Cancers, 429–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60334-1_14.

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Lurain, John, Michael Seckl, and Julian Schink. "Gestational Trophoblastic Disease." In Textbook of Uncommon Cancer, 653–62. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119196235.ch45.

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Pant, Alok, and John R. Lurain. "Gestational Trophoblastic Disease." In The American Cancer Society's Oncology in Practice, 318–28. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781118592168.ch24.

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Growdon, Whitfield. "Gestational Trophoblastic Disease." In Uncommon Gynecologic Cancers, 245–59. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118655344.ch23.

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Khong, T. Yee, Annie N. Y. Cheung, and Wenxin Zheng. "Gestational trophoblastic disease." In Diagnostic Endometrial Pathology, 1–50. 2e. | Boca Raton : CRC Press, 2018. | Preceded by Handbook of endometrial pathology / T. Yee Khong, Sezgin M. Ismail. 2005.: CRC Press, 2019. http://dx.doi.org/10.1201/9781315228686-10.

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Mazur, Michael T., and Robert J. Kurman. "Gestational Trophoblastic Disease." In Blaustein’s Pathology of the Female Genital Tract, 835–75. New York, NY: Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4757-1942-0_24.

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Mazur, Michael T., and Robert J. Kurman. "Gestational Trophoblastic Disease." In Diagnosis of Endometrial Biopsies and Curettings, 67–99. New York, NY: Springer New York, 2005. http://dx.doi.org/10.1007/978-0-387-26321-2_4.

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Chhieng, David, and Pei Hui. "Gestational Trophoblastic Disease." In Cytology and Surgical Pathology of Gynecologic Neoplasms, 131–38. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-164-6_8.

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Dickson, Elizabeth L., and Sally A. Mullany. "Gestational Trophoblastic Disease." In Gynecologic Oncology, 175–201. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1976-5_5.

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Conference papers on the topic "Gestational trophoblastic disease. eng"

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Gandhi, Krati, and Pushpa Dahiya. "A rare case of invasive mole following evacuation of molar pregnancy and its management." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685366.

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Introduction: Gestational trophoblastic disease (GTD) is a spectrum of abnormal growth and proliferation of trophoblasts that continue even beyond the end of pregnancy. It comprises of hydatidiform mole, invasive mole, choriocarcinoma and placental site tumor. Invasive mole (Choreoadenoma destruens) comprises about 5-8% of all GTD. It has invasive and destructive potentialities. Case Report: We report a case of 22 yr old female, G3P0A2, with 3 months amenorrhea with c/o pain abdomen since 4 days with no c/o bleeding p/v, with raised level of β hcg after two spontaneous abortions. On clinical examination vitals were stable. P/A ut 16-18 wks, doughy feel, slight tender. P/V os closed, ut 16-18 wks, bpv+. Ultrasonography shows multicystic lesion in cervix and vagina with loss of fat planes with UB. β hcg level was more than 5,00,000. Suction evacuation was done and products sent for histopathology. MRI Pelvis was also done in which invasive mole was diagnosed. 4 doses of inj. Methotrexte f/b folinic acid was given but β hcg levels did not fall by log 10. On histopath there was no evidence of invasive mole but 2nd line chemotherapy (EMACO) was started on the basis of MRI findings. Patient has received 5 cycles of EMACO REGIME with β hcg level being followed and is on decreasing trend, has reached to 5.90 mIU/ml. Conclusion: Patient of molar pregnancy should be followed regularly for early diagnosis of persistent gestational trophoblastic disease and adequate management as loss to follow up patients may land up into complications.
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Hemida, R., A. Zakaria, W. Elrefaie, and E. Refaie. "EP1136 Outcome of gestational trophoblastic disease in Lower Egypt." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.1178.

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Baptista de Almeida, S., F. Ferreira da Silva, and F. Gomes. "EP1134 Gestational trophoblastic disease (GTD): a 23-years single institution experience." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.1176.

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Freitas, Pedro Corvelo, Beatriz Mira, António Guimarães, Ana Opinião, Hugo Nunes, Ana Francisca Jorge, Fátima Vaz, and António Moreira. "520 Long term follow up after diagnosis of gestational trophoblastic disease." In ESGO SoA 2020 Conference Abstracts. BMJ Publishing Group Ltd, 2020. http://dx.doi.org/10.1136/ijgc-2020-esgo.176.

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Costigan, Catherine, Sabin Tabirca, John Coulter, and Ernest Scheiber. "Mathematically Modelling HCG In Women With Gestational Trophoblastic Disease Using Exponential Interpolation." In 30th Conference on Modelling and Simulation. ECMS, 2016. http://dx.doi.org/10.7148/2016-0520.

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Costigan, Catherine, Sabin Tabirca, and John Coulter. "Mathematically Modelling hCG in Women with Gestational Trophoblastic Disease Using Logarithmic Transformations." In 2016 UKSim-AMSS 18th International Conference on Computer Modelling and Simulation (UKSim). IEEE, 2016. http://dx.doi.org/10.1109/uksim.2016.60.

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Kaur, Paramjeet, Ashok K. Chauhan, Anil Khurana, Yashpal Verma, and Nupur Bansal. "Gestational trophoblastic neoplasia: Retrospective analysis of clinical profile, treatment pattern and outcome." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685377.

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Abstract:
Background: Gestational trophoblastic disease is a spectrum of cellular proliferation arising from the placental villous trophoblast. Gestational triphoblastic neoplasia (GTN) is a collective term for GTD that invade locally or metastasize. GTD includes hydatidiform mole (complete and partial) and GTN include invasive mole, choricocarcinoma, placental site trophoblastic tumor and epitheliod trophoblastic tumor. Aim: To evaluate clinicopathological profile, treatment pattern and clinical outcome in patients with gestational trophoblastic neoplasia (GTN). Materials and Methods: Twelve cases of gestational trophoblastic neoplasia treated between 2012 to November 2015 in deptt of Radiotherapy – II, PGIMS, Rohtak were evaluated in this retrospective study. Data was analyzed on the basis of age, histopathology, stage, type of treatment received and treatment related toxicities. Disease free survival was estimated. Results: Out of 12 women 7 (58 %) had hydatidiform mole, 4 (33%) invasive mole and 01 (8%) had choriocarcinoma. All the cases were given chemotherapy. Two patients had low risk disease. Among high risk group seven patients had score of less than 7 and five patients had risk score of 7 or higher. Five patients were given single agent methotrexate, seven patients received multidrug regimens. All patients are on regular follow up. One patient (high risk group) expired as she did not receive treatment. Conclusion: GTN are rare and proliferative disorders with proper diagnosis and treatment most of the cases are amenable to treatment with favorable outcome.
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8

Kaur, Paramjeet, Ashok K. Chauhan, Anil Khurana, Yashpal Verma, and Nupur Bansal. "Gestational trophoblastic neoplasia: Retrospective analysis of clinical profile, treatment pattern and outcome." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685306.

Full text
Abstract:
Background: Gestational trophoblastic disease is a spectrum of cellular proliferation arising from the placental villous trophoblast. Gestational triphoblastic neoplasia (GTN) is a collective term for GTD that invade locally or metastasize. GTD includes hydatidiform mole (complete and partial) and GTN include invasive mole, choricocarcinoma, placental site trophoblastic tumor and epitheliod trophoblastic tumor. Aim: To evaluate clinicopathological profile, treatment pattern and clinical outcome in patients with gestational trophoblastic neoplasia (GTN). Materials and Methods: Twelve cases of gestational trophoblastic neoplasia treated between 2012 to November 2015 in Department of Radiotherapy – II, PGIMS, Rohtak were evaluated in this retrospective study. Data was analyzed on the basis of age, histopathology, stage, type of treatment received and treatment related toxicities. Disease free survival was estimated. Result: Out of 12 women 7 (58 %) had hydatidiform mole, 4 (33%) invasive mole and 1 (8%) had choriocarcinoma. All the cases were given chemotherapy. Two patients had low risk disease. Among high risk group seven patients had score of less than 7 and five patients had risk score of 7 or higher. Five patients were given single agent methotrexate, seven patients received multidrug regimens. All patients are on regular follow up. One patient (high risk group) expired as she did not receive treatment. Conclusion: GTN are rare and proliferative disorders with proper diagnosis and treatment most of the cases are amenable to treatment with favorable outcome.
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Yarandi, F., and E. Shir Ali. "EP1146 Clinical response to a second uterine curettage in patients with low-risk gestational trophoblastic disease: a pilot study." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.1187.

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Funk, A. "Enhanced myometrial vascularity and persistence of beta hCG: grey area between retained products of conception, AV-malformation and gestational trophoblastic disease." In 62. Kongress der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe – DGGG'18. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1671581.

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