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1

Tambouret, Rosemary H., and David C. Wilbur. Glandular Lesions of the Uterine Cervix. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1989-5.

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2

Rosenstein, Marvin. Handbook of glandular tissue doses in mammography. Rockville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Food and Drug Administration, Center for Devices and Radiological Health, 1985.

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3

Beatriz, Jiménez-Ayala Portillo, ed. Cytopathology of the glandular lesions of the female genital tract. Basel: Karger, 2011.

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4

McCluggage, W. Glenn, John Tidy, and John H. F. Smith. Cellular Pathology of Glandular Lesions and Uncommon Neoplasms of the Cervix. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-2210-4.

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5

Dadzie, Ophelia E. Pearls and pitfalls in neoplastic dermatopathology. Cambridge, United Kingdom: Cambridge University Press, 2016.

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6

Halpin, Nick. Coping with fatigue due to the effects of glandular fever or flu viruses. Dundee: University of Dundee, Student Counselling and Advisory Service, 1986.

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7

Rosenbaum, Michael E. Super supplements: Your guide to today's newest vitamins, minerals, enzymes, amino acids, and glandulars. New York, N.Y: New American Library, 1989.

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8

Fedikow, Mark Albert Fredrick. Results of a vegetation geochemical survey near Bissett, southeastern Manitoba. Winnipeg: Manitoba Energy and Mines, Geological Services, 1987.

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9

1952-, Wick Mark R., and Swanson Paul E. 1957-, eds. Immunohistology and electron microscopy of anaplastic and pleomorphic tumors. Cambridge: Cambridge University Press, 1997.

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10

Kurman, Robert J. Tumors of the cervix, vagina, and vulva. Washington, D.C: Armed Forces Institute of Pathology, 1993.

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11

Kurman, Robert J. Tumors of the cervix, vagina, and vulva. Washington, D.C: Armed Forces Institute of Pathology, 1992.

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12

Kurman, Robert J. Tumors of the cervix, vagina, and vulva. Washington, D.C: Armed Forces Institute of Pathology, 1992.

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13

Escovich, Novelli. Glandulas Salivales. Corpus, 2005.

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14

Fernández-Flórez, Wénceslao. El ladrón de glándulas. Peninsular Publishing Company, 1998.

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15

Facchinetti, F. Cellular Communication in Reproduction (Endocrine Updates). Society for Endocrinology, 1993.

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16

Glandular Extracts. McGraw-Hill, 1995.

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17

Garrett, J. R., J. Ekstr�m, and L. C. Anderson, eds. Glandular Mechanisms of Salivary Secretion. S. Karger AG, 1998. http://dx.doi.org/10.1159/isbn.978-3-318-00266-9.

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18

R, Garrett J., Ekström Jörgen, and Anderson L. C, eds. Glandular mechanisms of salivary secretion. Basel: Karger, 1998.

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19

Novelli. Patologia Quirurgica de La Glandula Tiroides. Unr, 2005.

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20

Glandular Failure - Caused Obesity (and other associated promoters). The Bridge Medical Publishers, 2004.

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21

Cirugia de los tumores de la glandula parotida. Amolca (Actualidades Médico Odontológicas Latinoamericanas), 2007.

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22

Warner and Rosenstien. Handbook of Glandular Tissue Doses in Mammography (Hhs Publication #pb85209914). Us Dept of Health & Human, 1985.

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23

Wilbur, David C., and Rosemary H. Tambouret. Glandular Lesions of the Uterine Cervix: Cytopathology with Histologic Correlates. Springer, 2014.

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24

Jim�nez-Ayala, M., and B. Jim�nez-Ayala Portillo. Cytopathology of the Glandular Lesions of the Female Genital Tract. S. Karger AG, 2010. http://dx.doi.org/10.1159/isbn.978-3-8055-9465-3.

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25

Smith, John, Glenn McCluggage, and John Tidy. Cellular Pathology of Glandular Lesions and Uncommon Neoplasms of the Cervix. Springer, 2014.

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26

Unwanted hair: Its cause and treatment : ancestral curse or glandular disorder? Carnforth: Parthenon Press, 1985.

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27

Boyle, Mary Margaret. Von Brunn's nests and glandular metaplasia in the supramontanal prostatic urethra. 1995.

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28

Tidy, John, W. Glenn McCluggage, and John H. F. Smith. Cellular Pathology of Glandular Lesions and Uncommon Neoplasms of the Cervix. Springer, 2016.

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29

Tidy, John, W. Glenn McCluggage, and John H. F. Smith. Cellular Pathology of Glandular Lesions and Uncommon Neoplasms of the Cervix. Springer, 2014.

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30

Taguchi, Yosh. La prostata: Todo lo que necesita saber sobre la glandula masculina. Gestion 2000, 2004.

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31

Keshav, Satish, and Alexandra Kent. Pancreatic disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0201.

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Acute pancreatitis is an acute inflammatory process of the pancreas and is potentially reversible. It is characterized by oedema and necrosis of peripancreatic fat and may progress to necrosis of glandular and surrounding tissue. Activation of pancreatic enzymes leads to pancreatic autodigestion and systemic effects.
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32

Carcangiu, Maria Luisa, Ronald A. DeLelli, and Juan Rosai. Atlas of Tumor Pathology: Tumors of the Thyroid Gland [Third Series - Fascicle 5]. Armed Forces Institute of Pathology, 1992.

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33

Publications, ICON Health. Glandular Fever: A Medical Dictionary, Bibliography, And Annotated Research Guide To Internet References. Icon Health Publications, 2004.

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34

Dr Donsbach Tells You What You Always Wanted to Know About Glandular Extracts. Wholistic Publishers, 1988.

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35

The Foot-and-Mouth Disease (Sera and Glandular Products) (Revocation) Order 1993 (Statutory Instruments: 1993: 1332). Stationery Office Books, 1993.

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36

Ajzensztejn, Daniel. Breast cancer. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0327.

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Breast cancer is the commonest female cancer, with a lifetime risk of approximately 1 in 9. There are approximately 40 000 new cases and 11 000 deaths from the disease in England and Wales each year. Breast cancer is an adenocarcinoma which arises from the glandular tissue of the breast. Its etiology is complex, with hormonal, genetic, and modifiable lifestyle factors all involved in developing the disease. Prognosis is related to the anatomical extent of the cancer, and other factors. This chapter discusses the definition and etiology of breast cancer, as well as its typical symptoms, less common symptoms, demographics, natural history, complications, diagnosis, treatment, and prognosis.
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37

Sekhar, Rajagopal V. Endocrine Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0035.

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HIV infection is now a chronic disease, and it is associated with an increasing prevalence of metabolic and endocrine abnormalities. The underlying etiology of these disorders can be attributed to multiple factors, including, but not limited to, the effects of HIV itself, antiretroviral drugs, the effects of immune dysfunction, and other opportunistic infections. Any endocrine glandular system can be involved; hence, appropriate clinical suspicion, endocrinological dynamic testing for accurate diagnosis, and effective therapy are important in the identification and management of these disorders. HIV infection, antiretroviral drugs, and other factors play a role in the development of endocrine disease. For endocrine disorders in HIV, early referral to an endocrinologist is suggested.
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38

Provine, Robert R. Beyond the Smile. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780190613501.003.0011.

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With the expectation that innovation, insight, and discovery will come from researching neglected topics, this chapter explores human instincts, including yawning, laughing, vocal crying, emotional tearing, coughing, nausea and vomiting, itching and scratching, and changes in scleral color. The critical change approach is exploited to analyze recently evolved, uniquely human traits (e.g., human-type laughter and speech, emotional tearing, scleral color cues) and compare them with thir primate antecendents, seeking the specific neurological, glandular, and muscular processes responsible for their genesis. Particular attention is paid to contagious behaviors, with the anticipation that they may reveal the roots of sociality and empathy. Few of these curious behaviors are traditionally considered in the context of facial expression or emotion, but they deserve recognition for what they can contribute to behavioral neuroscience and social biology.
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39

DeBruhl, Nanette D., and Nazanin Yaghmai. Breast Implants. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0060.

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The presence of breast implants limits the amount of tissue that can be visualized on mammography and tomosynthesis. The proper mammographic positioning of the breasts of women with implants requires special training. More tissue can be visualized in women with sub-pectoral implants than in women with sub-glandular implants. Women with implants are recommended to have age-appropriate routine interval screening mammography for detection of cancer. If an implant rupture is suspected, ultrasound and MRI are used as adjunct imaging modalities. This chapter, appearing in the section on breast implants, reviews the key imaging and clinical features, imaging protocols and pitfalls, and management recommendations for breast implants. Topics discussed include types of implants, imaging findings of intact implants, and signs of ruptured implants, using mammography, ultrasound, and magnetic resonance imaging.
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40

Kettler, Mark D. Large Circumscribed Mass in Young Female. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0021.

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Fibroepithelial lesions account for the overwhelming majority of solid breast masses affecting women younger than age 20. Nearly all present as self-detected or provider-detected palpable masses. All fibroepithelial lesions are composed of stromal (fibrous) and glandular (epithelial) elements and variable histology. Rapidly growing mobile breast masses in girls or female adolescents may represent juvenile fibroadenomas, which have different but benign histological features when compared to typical fibroadenomas. Benign phyllodes tumors closely resemble usual fibroadenomas and juvenile fibroadenomas on imaging. Decisions whether to biopsy these tumors are made clinically; the diagnosis of phyllodes tumor depends on histological assessment.This chapter, appearing in the section on circumscribed mass, reviews the key clinical and imaging features, differential diagnosis, and management recommendations of large solid breast masses affecting young women, including typical fibroadenomas, giant fibroadenomas, juvenile fibroadenomas, and phyllodes tumors.
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41

Apple, Sophia K., and Lawrence W. Bassett. Normal Breast Anatomy and Histology. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0003.

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In this chapter, normal breast anatomy is discussed, with a succinct pictorial summary of breast glandular elements, the chest wall, and the axilla. The locations of breast lesions are often arbitrarily divided into upper outer, upper inner, lower inner, and upper inner quadrants. However, the lobes within a specific quadrant cross over into adjacent quadrants. The terminal duct lobular unit (TDLU) is composed of an interlobular duct and associated lobules with multiple grape-like structures where the milk is secreted and drains into the terminal ducts, interlobular ducts, excretory ducts, lactiferous sinus, lactiferous duct, and the nipple. Axillary lymph nodes are divided into three levels, based on their location in relation to the pectoralis minor muscle. Level I axillary lymph nodes are located below the edge of the pectoralis minor; level II lies posterior to the pectoralis minor; and level III lies medial to the pectoralis minor.
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42

Scrofula and Its Gland Diseases: An Introduction to the General Pathology of Scrofula, With an Account of the Histology, Diagnosis and Treatment of Its Glandular Affections. Wentworth Press, 2016.

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43

Series, Michigan Historical Reprint. Alphabetical repertory of the skinsymptoms and external alterations of substance : Together with the morbid phenomena observed in the glandular, osseous, ... remarks on the diseases of the skin by. Scholarly Publishing Office, University of Michigan Library, 2005.

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44

Hormonal regulation of prostate-specific antigen and human glandular kallikrein in males and females in vivo: Effects of androgens and antiandrogens on plasma and urinary PSA and hK2 levels. Ottawa: National Library of Canada, 2000.

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45

Ng, Wan-Fai, Arjan Vissink, Elke Theander, and Francisco Figueiredo. Sjögren’s syndrome—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0128.

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Management of Sjögren's syndrome (SS) encompasses confirmation of diagnosis, disease assessment, and treatment of glandular and systemic manifestations including special situations such as pregnancy and SS-related lymphoma. The American European Consensus Group classification criteria 2002 are the current gold standard for the diagnosis of SS. Salivary gland sialometry, sialochemistry, and ultrasound and tear osmolarity may be useful adjuncts. Symptoms of SS are non-specific and must be actively explored. When assessing patients with SS, it is important to consider not only objective parameters such as abnormalities in blood tests and changes in tear and salivary flow, but also patient-reported outcome measures and impact on quality of life. Current management of patients with SS is hampered by the lack of evidence-based strategies. The symptoms experienced by patients with SS are often not fully appreciated by clinicians, which may contribute to the suboptimal management of the condition. Management of fatigue remains a major challenge and a holistic, multidisciplinary approach is recommended. Factors that may contribute to fatigue should be fully addressed. Recent advances in the understanding of the pathogenic mechanisms of SS have informed more targeted therapeutic strategies with some promising data. Optimal management of SS requires expertise from different disciplines. Combined clinics with rheumatology, oral medicine, and ophthalmology input will improve care and communications as well as reduce the number of clinic visits for patients and healthcare-related cost. Effective link between pSS specialists, dentists, opticians, and general practitioners will facilitate early diagnosis and reduce risk of long-term disability of SS.
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46

Ng, Wan-Fai, Arjan Vissink, Elke Theander, and Francisco Figueiredo. Sjögren’s syndrome—management. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199642489.003.0128_update_001.

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Management of Sjögren’s syndrome (SS) encompasses confirmation of diagnosis, disease assessment, and treatment of glandular and systemic manifestations including special situations such as pregnancy and SS-related lymphoma. The American European Consensus Group (AECG) classification criteria 2002 are the current gold standard for the diagnosis of SS. Salivary gland sialometry, sialochemistry, and ultrasound and tear osmolarity may be useful adjuncts. Recently, preliminary classification criteria of the American College of Rheumatology have been introduced as an alternative to the AECG criteria. Symptoms of SS are non-specific and must be actively explored. When assessing patients with SS, it is important to consider not only objective parameters such as abnormalities in blood tests and changes in tear and salivary flow, but also patient-reported outcome measures and impact on quality of life. Current management of patients with SS is hampered by the lack of evidence-based strategies. The symptoms experienced by patients with SS are often not fully appreciated by clinicians, which may contribute to the suboptimal management of the condition. Management of fatigue remains a major challenge and a holistic, multidisciplinary approach is recommended. Factors that may contribute to fatigue should be fully addressed. Recent advances in the understanding of the pathogenic mechanisms of SS have informed more targeted therapeutic strategies with some promising data. Optimal management of SS requires expertise from different disciplines. Combined clinics with rheumatology, oral medicine, and ophthalmology input will improve care and communications as well as reduce the number of clinic visits for patients and healthcare-related cost. Effective link between pSS specialists, dentists, opticians, and general practitioners will facilitate early diagnosis and reduce risk of long-term disability of SS.
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47

Nithikathkul, Choosak, Prasert Saichua, Louis Royal, and John H. Cross. Capillariosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0065.

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Capillaria species are members of the superfamily Trichinelloidae. These worms have a filamentous thin anterior end and a slightly thicker oesophagus which is surrounded by glandular cells or stichocytes. This oesophageal pattern is called stichosomal oesophagus. Capillaria species are parasites which are found in many vertebrate animals. More than two hundred species have been reported in several vertebrate species, including fish, amphibians, reptiles, birds, and mammals (Cross 1992; Chitwood et al. 1968), but only three species infect humans. These are Capillaria hepatica , C. aerophila and C. philippinensis (McCarthy and Moore 2000). Of these intestinal capillariosis, a fish-borne parasitic zoonosis caused by C. philippinensis , is the most important. Humans acquire the parasite, C. philippinensis, by eating uncooked or raw freshwater fish (Cross and Basaca-Sevilla 1991). The disease is endemic mainly in Philippines and Thailand where there are many reported fatalities.Although C . hepatica is found in rodents worldwide, only a few cases of hepatic capillariosis have been reported in humans from Europe, Asia, Africa, North and South America. The infection is acquired by the ingestion of embryonated eggs from the soil. Female worms deposit eggs in the liver tissue and granulomas develop around the egg. The eggs are released after the rodent is eaten and the liver digested. Eggs pass in the faeces and are deposited in the soil where they embryonate. Avoidance of contaminated soil would prevent human infection and destruction of rodents would control animal infections.Only 12 cases of human infection caused by Capillaria aerophila have been reported, the majority from Russia. The parasite is found within tissue of the respiratory passages of canines and felines worldwide.Anatrichosoma cutaneum (Nematoda, Trichosomoididae), also included in this chapter, is primarily a subcutaneous parasite of monkeys, but there are two reports of cutaneous infections in humans resulting in serpiginous lesions in the skin of the soles, palms, and nasal passages. In addition there is a further suspected case isolated from a breast nodule and a possible case of mucosal lesions in the mouth reported. Whole monkey colonies can be infected with this parasite and control is difficult.
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48

Tumors of the Cervix, Vagina, and Vulva (Atlas of Tumor Pathology 3rd Series). American Registry of Pathology, 1992.

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