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1

-C, Fehmann H., and Göke B. (Burkhard), eds. The insulinotropic gut hormone glucagon-like peptide-1. Basel: Karger, 1997.

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2

Joseph, Jamie William. Oral delivery of glucagon-like peptide-1 using PLGA-COOH microspheres. Ottawa: National Library of Canada, 1999.

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3

Kim, Julie. The roles of glucagon-like peptide-1 (GLP-1) in the mouse brain. Ottawa: National Library of Canada, 1998.

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4

Jeng, Winnie. Structural and functional studies of the glucagon-like peptide-1 (GLP-1) receptor. Ottawa: National Library of Canada, 1998.

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5

Sandhu, Harmanjit Singh. The effect of GLP-1, glucagon-like peptide 1, on insulin sensitivity in diabetic dogs. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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6

Mendosa, David. Losing weight with your diabetes medication: How Byetta and other drugs can help you lose more weight than you ever thought possible. Philadelphia, PA: Da Capo Life Long, 2008.

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7

Cook, Sonya M. Characterization of mice with a null mutation in the glucagon-like peptide-1 receptro gene. Ottawa: National Library of Canada, 1998.

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8

Mendosa, David. Losing weight with your diabetes medication: How Byetta and other drugs can help you lose more weight than you ever thought possible. Philadelphia, PA: Da Capo Life Long, 2008.

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9

Satkunarajah, Malathy. Studies of the incretins, glucagon-like peptide-1 and glucose-dependent insulinotropic polypeptide, and their receptors. Ottawa: National Library of Canada, 1998.

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10

Green, Brian Desmond. Amino-terminally modified analogues of glucagon-like Peptide-1(7-36)Amide: Activity and antidiabetic potential. [S.l: The Author], 2003.

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11

Mooney, Mark H. Glucagon-like peptide-1 and gastric inhibitory polypeptide: Effects of N-terminal glycation on hormone degradation, insulin secretion and antihyperglycaemic activity. [S.l: The Author], 2000.

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12

Fehmann, H. C., and B. G�ke, eds. The Insulinotropic Gut Hormone Glucagon-Like Peptide-1. S. Karger AG, 1997. http://dx.doi.org/10.1159/isbn.978-3-318-01934-6.

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13

Ranganath, Lakshminarayan Rao. The role of glucagon-like peptide-1 and lipoprotein lipase in health and obesity. 1996.

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14

Huang, Ya-Chi. Glucagon-like peptide-1-induced suppression on glucagon secretion in pancreatic alpha-cells: A study using phosphoinositol 3-kinasegamma deficient mice. 2006.

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15

Mendosa, David, and Joe Prendergast. Losing Weight with Your Diabetes Medication: How Byetta and Other Drugs Can Help You Lose More Weight Than You Ever Thought Possible. Hachette Books, 2008.

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16

David, Mendosa, and David Mendosa. Losing Weight with Your Diabetes Medication. Hachette Books, 2008.

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17

(Foreword), Joe Prendergast, ed. Losing Weight with Your Diabetes Medication: How Byetta and Other Drugs Can Help You Lose More Weight than You Ever Thought Possible (Marlowe Diabetes Library). Marlowe & Company, 2008.

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18

Dorizas, Petros. Investigation of the effect of glucagon like peptide-1 (glp-1) in gastric motility and satiety in man. 1997.

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19

Aziz, Alfred. The role of glucagon-like peptide-1 receptors in protein-induced suppression of food intake in rats. 2004.

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20

Rocca, Antonio Stevereves. Direct and indirect effects of fatty acids on secretion of the antidiabetic hormone, glucagon-like peptide-1. 2000.

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21

Professor R., M.D. Arnold. Digestion/International Symposium on Glucagon-Like Peptide-1 (International Journal of Gastroenterology, Vol 54, No 6 1993). S Karger Pub, 1993.

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22

Truncated glucagon-like peptide-1: Mechanism of action and effects on hormone synthesis and secretion in isolated rat pandreatic islets. Ottawa: National Library of Canada, 1993.

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23

Hinder, Lucy M., Kelli A. Sullivan, Stacey A. Sakowski, and Eva L. Feldman. Mechanisms Contributing to the Development and Progression of Diabetic Polyneuropathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0114.

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Advances in our understanding of diabetes in human patients and experimental models indicate that a number of mechanisms may contribute to sensory nerve damage in diabetic polyneuropathy (DPN). In addition to oxidative stress, hyperglycemia and hyperlipidemia, recent research in pain, advanced glycation endproduct (AGE), and proteomics specify a contributory role for altered neuronal calcium homeostasis in DPN. Technology advances indicate neuronal energy balance and mitochondrial biogenesis, fission, and fusion are additional potential mechanisms. The effects of dysregulation or loss of insulin signaling and the effects of glucagon-like peptide-1 (GLP-1) and its receptor (GLP-1R) are also implicated.
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24

Nutt, David J., and Liam J. Nestor. Appetite hormones and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0012.

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Many of the same behavioural and brain disturbances observed in addiction are also seen in obesity and binge-eating disorder. This suggests that there are shared neural substrates between substance addiction and compulsive food consumption. Food intake and appetite are regulated by numerous appetite hormones that exert their effects through brain systems involved in reward sensitivity, stress, impulsivity, and compulsivity. There is now emerging evidence that appetite hormones (e.g. ghrelin, glucagon-like peptide-1, orexin) can modulate addictive behaviours (e.g. craving) and the intake of alcohol and drugs. Therefore, there is an emerging shift into a new field of testing drugs that affect appetite hormones and their receptors in the brain, and their use in regulating the brain mechanisms that lead to relapse in addiction disorders.
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