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Relevant bibliographies by topics / Glucose metabolism / Dissertations / Theses

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Dissertations / Theses on the topic 'Glucose metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Pennant, Mary Elizabeth. "Measuring glucose metabolism." Thesis, University of Cambridge, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611215.

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2

Roberts, Justin D. "Post exercise glucose metabolism." Thesis, Brunel University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249789.

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3

Thiên. "Glucose metabolism in falciparum Malaria." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2004. http://dare.uva.nl/document/74040.

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4

Williams, Andrew C. "Glucose metabolism in human spermatozoa." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302101.

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5

Rotondo, Floriana. "Glucose and white adipose tissue metabolism. Effects of site and sex on the fate of glucose." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/482142.

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The current theories about endothelial inflammation in the adipose tissue in metabolic syndrome point to hypoxia as one of its main causes. Nevertheless, our group has found that both 3T3L1 adipocytes, under normoxic conditions, and WAT, in vivo, consume large amount of glucose, with high production of lactate and glycerol irrespective of oxygen availability. This thesis is based on the hypothesis that adipocytes act essentially as glycolytic cells impervious to hypoxia, and their metabolism may help reduce the blood glucose levels. We assumed that hypoxia, could affect the cells of the stro

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6

Fraser, Bernadine Heather. "Glycogen and glucose metabolism in cardioprotection." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0028/NQ34764.pdf.

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7

Butler, Mark Henry. "Information processing in liver glucose metabolism." Thesis, University of Liverpool, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367289.

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8

Vo, Annie Phuong. "Glucose Metabolism in Cancer-Associated Fibroblasts." Thesis, Harvard University, 2013. http://dissertations.umi.com/gsas.harvard:11025.

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Under normal conditions, non-transformed cells rely on glycolysis followed by oxidative phosphorylation to generate ATPs. When oxygen is scarce or when cells are actively proliferating, cellular ATPs come mainly from glycolysis. Pyruvate is converted into lactate to allow glycolysis to continue. Interestingly, cancer cells have adapted to favor lactate production even at normal oxygen tensions, exhibiting a metabolic shift known as the Warburg effect. However, the metabolic state of other cellular constituents within the tumor remains mostly unknown. Cancer-associated fibroblasts (CAFs) are

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9

Karki, Rabindra. "Regulation of glucose metabolism by Alox8." OpenSIUC, 2014. https://opensiuc.lib.siu.edu/theses/1507.

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Type II diabetes is one of the leading cause of morbidity in the U.S. and other parts of the world. Insulin resistance which precedes Type II diabetes is a complex state of the body where the body fails to respond to insulin. Its complexity lies in its multifactorial origin that is to say various environmental and polygenic components come into play. Here we try to dissect one of these components - `Alox8' in transgenic mice and try to see if it affects blood glucose homeostasis. Comparison of glucose tolerance and insulin sensitivity among sixteen mice comprising of six wild type, five hetero

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10

Segarra, Mondéjar Marc. "Estudio de la regulación del metabolismo de la glucosa por la actividad sináptica." Doctoral thesis, Universitat de Barcelona, 2019. http://hdl.handle.net/10803/668643.

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La actividad sináptica regula una larga lista de procesos esenciales para el desarrollo y la supervivencia neuronal mediante cambios en los niveles de Ca2+ citoplasmático. Modula desde la morfología del árbol dendrítico neuronal hasta la plasticidad neuronal o los mecanismos de protección contra el daño oxidativo. La entrada de Ca2+ a través de los receptores sinápticos promueve diferentes cambios a nivel celular que van desde la remodelación del citoesqueleto a la estimulación de diferentes cascadas de señalización que finalmente activan diferentes factores de transcripción implicados en la r

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11

Ling, Zong-Chao. "Islet glucose metabolism and insulin release in two animal models of glucose intolerance /." Stockholm, 1999. http://diss.kib.ki.se/1999/19990517ling/.

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12

Helminen, O. (Olli). "Glucose metabolism in preclinical type 1 diabetes." Doctoral thesis, Oulun yliopisto, 2016. http://urn.fi/urn:isbn:9789526213255.

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Abstract Type 1 diabetes is considered to be a T cell-mediated autoimmune disease characterized by destruction of the pancreatic beta cells. Its prediction is currently based on diabetes-associated autoantibodies, giving a cumulative risk of 84% during 15 years of follow-up since seroconversion. Prediction of the timing of clinical onset has remained challenging, however. This thesis examines glucose metabolism in autoantibody-positive children with a high risk of developing type 1 diabetes. Out of a total of 14,876 children with an increased genetic risk followed up from birth in the Finnish

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13

Robertson, Tracey M. "The effects of coffee on glucose metabolism." Thesis, University of Surrey, 2016. http://epubs.surrey.ac.uk/812361/.

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It has been suggested that coffee drinking may confer a beneficial effect on health by reducing the risk of developing Type 2 Diabetes Mellitus (T2DM) and indeed there is much epidemiological evidence for a reduced incidence of T2DM in habitual coffee drinkers. However, many acute studies have reported a temporary worsening in postprandial glycaemia following caffeinated coffee (CC) consumption. Varied methodologies have been employed by these studies with many giving their participants large doses of coffee. In the acute studies conducted for this thesis, a single serving of CC increased the

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14

Jovanovic, Ana. "Postprandial Interactions between Glucose and Lipid Metabolism." Thesis, University of Newcastle Upon Tyne, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.512173.

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15

Karim, S. A. M. "Influence of glucose metabolism on retinal transdifferentiation." Thesis, University of Nottingham, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374805.

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16

Shmueli, Ehoud. "Glucose metabolism and insulin resistance in cirrhosis." Thesis, University of Newcastle Upon Tyne, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308777.

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17

Bitler, Chad. "Vitamin D and Markers of Glucose Metabolism." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1416231511.

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18

Lindfelt, Jan O. W. "Hepatic nerves in hemostasis and glucose metabolism :." Lund : Dept. of Surgery, Lund University, 1993. http://catalog.hathitrust.org/api/volumes/oclc/39654187.html.

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19

Tejedor, Vaquero Sonia 1988. "Influence of metabolism in the regulation of T cell differentiation." Doctoral thesis, Universitat Pompeu Fabra, 2018. http://hdl.handle.net/10803/664638.

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Glucose is a key nutrient for T cells. Despite that T cell activation is impaired when they are deprived of glucose, it has also been shown that T effector responses can be elicited in vivo in glucose-poor environments, such as the intratumoral niche, which raises the question of how these cells can maintain their function in nutrient-restricted sites. In this work, we analyzed the ability of T effector cells to be activated by pro-inflammatory polarizing conditions under limiting glucose availability, using an in vitro model in which effector Th0 cells were restimulated to secondary effector

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20

Jackson, Alicia R. "Transport and Metabolism of Glucose in Intervertebral Disc." Scholarly Repository, 2010. http://scholarlyrepository.miami.edu/oa_dissertations/479.

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Low back pain is a major social and economic dilemma in the United States. Despite its high impact, the origins of low back remain unclear. Nonetheless, degenerative changes to the intervertebral discs (IVD) of the spine have been implicated as a possible source leading to pain. Poor nutritional supply to the IVD is believed to play a primary role in the pathophysiology of disc degeneration. Since the disc is avascular, vital nutrients, such as glucose, must be supplied by surrounding blood vessels. However, the transport and metabolic properties of glucose in the IVD have not been full

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21

Wakil, Ammar. "Fatty acids and the modulation of glucose metabolism." Thesis, University of Hull, 2011. http://hydra.hull.ac.uk/resources/hull:4938.

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Cardiovascular disease is the leading cause of death globally. This thesis has critically evaluated aspects related to two cardiovascular risk factors, namely hyperlipidaemia and hyperglycaemia, in healthy volunteers. Free fatty acids produced by visceral fat and transported to the liver in the portal circulation are thought to be one mechanism by which obesity contributes to increased insulin resistance and therefore reduced glucose disposal and a propensity to hyperglycaemia. The first study assessed the bioavailability of eicosapentaenoic acid, a cardio-protective fatty acid, in different f

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22

Chung, Tien-jung Albert, and 鍾典融. "Hepatitis B and glucose metabolism : a systematic review." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206934.

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Background/Aim: Hepatitis C virus infection is a known risk factor of impaired glucose metabolism and diabetes mellitus. Whether hepatitis B virus (HBV) infection is also associated with impaired glucose tolerance remains uncertain. The aim of the study was to conduct a systematic review on the association between HBV infection and impaired glucose metabolism Methods: Studies reporting the association between HBV infection and markers of impaired glucose metabolism were identified through keyword search in PubMed and Google Scholar. 10 studies (out of 320) were included in this systematic rev

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23

McCauley, Sydney Russelle. "Glucose Metabolism in Low Birth Weight Neonatal Pigs." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/99450.

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The neonatal period in mammals is characterized by high growth rates and is dominated by skeletal muscle hypertrophy. Low birth weight (LBWT) neonates experience restricted growth and development of skeletal muscle, leading to metabolic perturbations later in life. The overall hypothesis of this dissertation was that in utero disturbances in glucose metabolism and increased energy requirements predisposes LBWT neonatal pigs to metabolic disturbances after birth. We sought to increase growth of skeletal muscle and improve glucose production through increasing dietary energy and to determine

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24

Nyirenda, Moffat J. "Glucocorticoids and the programming of hepatic glucose metabolism." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/22529.

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Since steroid hormones have well characterised programming properties, excessive fetal exposure to glucocorticoids in early in life may produce lifelong effects. In order to examine the role of glucocorticoids in the programming of glucose metabolism, fetal glucocorticoid load was increased by administration of dexamethasone, a poor substrate for 11β-HSD, during the different trimesters of rat pregnancy. Dexamethasone given selectively in the last week of pregnancy reduced birth weight by 10%, and produced hyperglycaemia and hyperinsulinaemia in the adult offspring. In addition, rats exposed t

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25

Panes, Alison. "Aspects of the glucose metabolism of Trypanosoma brucei." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380419.

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26

Denti, Paolo. "Nonlinear Mixed-Effects Modelling of Glucose-Insulin Metabolism." Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3426016.

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Diabetes mellitus is not only a very serious disease, causing discomfort and pain to millions across the world, but with the aging of the population and the prevalence of a sedentary lifestyle, it is assuming the proportion of a real epidemic, becoming a public health and social emergency. In answer to this call, research on diabetes has been intensely carried out in the past decades and the knowledge and understanding of its etiology have been significantly improved. However, investigation is still ongoing, many important questions are still unanswered, and the causes eliciting the pathogenes

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27

Hicks, Meghan. "Impact of a Smartphone-delivered Sedentary Behavior Intervention on Glucose Metabolism in Prediabetic Adults." Thesis, The University of Arizona, 2018. http://hdl.handle.net/10150/626855.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>This study investigates whether an 8-week lifestyle-based, smartphone-delivered intervention targeting reduction in sedentary behavior (i.e., sitting) significantly reduces objectively measured time spent sitting and improves fasting glucose and insulin. The incidence of type II diabetes has continued to increase in the United States and increases in sedentary behavior along with reductions in physical activity throughout the d

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28

Rincón, Viatela Jorge E. "Regulation of glucose transport and insulin-stimulated glut4 translocation in skeletal muscle /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-3297-2/.

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29

Kawano, Yuichi. "Effect of hyperglycemia on glucose transport and intracellular signal transduction in skeletal muscle /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3593-9/.

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30

Martino, Paul F. "The effects of dantrolene on post exercise glucose uptake." Virtual Press, 1996. http://liblink.bsu.edu/uhtbin/catkey/1020145.

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The purpose of this investigation was to determine the relationship between calcium and glucose uptake following muscle contraction with the use of the calcium channel blocker dantrolene. In previous studies an exercise model has been used to investigate the role of calcium during post-exercise glucose uptake. This study utilized electrical stimulation. It has been shown that exercise-induced glucose uptake is calciummediated, but to date no one has shown that glucose transport induced by electrical stimulation is calcium-mediated. Twenty four male Sprague Dawley rats weighing 140 g were sacri

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31

Whetstone, Heather Catherine. "Role of quinoprotein glucose dehydrogenase in metabolism of glucose and gluconate in Agrobacterium tumefaciens." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0011/MQ31475.pdf.

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32

Roohi, Mohammad Sadegh. "Regulation of sorbitol metabolism by glucose in Clostridium pasteurianum." Thesis, Heriot-Watt University, 1987. http://hdl.handle.net/10399/1042.

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33

Aiston, Susan Michelle. "Regulation of hepatic glucose metabolism by leptin and insulin." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341441.

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34

Pariagh, Sandra. "Glucose metabolism in hepatocytes exposed to free radical stress." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341783.

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35

Hampson, Laura Jane. "Control of hepatocyte glucose metabolism by hormones & neurotransmitters." Thesis, University of Newcastle Upon Tyne, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.438399.

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36

Bingham, Emma Margaret. "Physiological studies into human brain glucose sensing and metabolism." Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.433672.

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37

Dufort, Fay Josephine. "Contribution of Glucose Metabolism to the B Lymphocyte Responses." Thesis, Boston College, 2012. http://hdl.handle.net/2345/3161.

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Thesis advisor: Thomas C. Chiles<br>B-lymphocytes respond to environmental cues for their survival, growth, and differentiation through receptor-mediated signaling pathways. Naïve Blymphocytes must acquire and metabolize external glucose in order to support the bioenergetics associated with maintaining cell volume, ion gradients, and basal macromolecular synthesis. The up-regulation of glycolytic enzyme expression and activity via engaged B-cell receptor mediated-events was glucose-dependent. This suggests an essential role for glucose energy metabolism in the promotion of B cell growth, survi

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38

Sen, Satarupa. "Regulation of cellular glucose metabolism by HIV-1 infection." Diss., Temple University Libraries, 2014. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/280025.

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Biology<br>Ph.D.<br>Regulation of Glucose metabolism is known to play an important role in pathogenesis of many diseases. Primarily because deregulation of this metabolic pathway can lead to either apoptosis or extended life span of the cells involved. Viruses are parasitic in nature, they utilize the host cellular pathways to support their own progeny; hence it is expected that viruses would regulate the central glucose metabolism of infected host cells. Human immunodeficiency virus type 1 (HIV-1) causes acquired immune deficiency syndrome, and it uniquely infects both activated CD4+ T cells

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39

Maddegoda, Vidanelage Thilini Nilusha Jayasinghe. "Effect of Krüppel-like factor 3 on glucose metabolism." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/10186.

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Type 2diabetes is an adult-onset condition in which the body is not able to tightly regulate the amount of glucose in the blood and it results from a combination of causes including genetic, behavioral and environmental factors. Krüppel-like factor 3 (KLF3) is a zinc finger transcription factor encoded by the klf3 gene. It has three Krüppel-like zinc fingers that bind to DNA. KLF3 inhibits adipogenesis in vitro through its ability to inhibit an adipogenic master regulator, C/EBPα expression (Sue et al., 2008). klf3-/- mice have fewer and smaller fat cells in adipose tissue. Reasons for differ

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40

Chen, Guoqing. "Halofuginone modulates glucose metabolism and autophagy in colorectal cancer." HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/309.

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Cell metabolism disorder is considered as both direct and indirect consequence of oncogenic mutations for tumoigenesis. Autophagy is a metabolic stress response and a mechanism of natural cellular degradation. It is believed that autophagy, as well as metabolism, is a crucial process for the adaptation of cancer cells to changes in nutrient availability. Understanding the relationship between metabolism and autophagy and targeting on the key steps are regarded as a promising strategy to treat cancer. Halofuginone (HF), a semisynthetic quinazolinone alkaloid originally derived from the plant Di

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41

Budgen, Nigel. "The catabolism of glucose by the thermoacidophilic archaebacterium Thermoplasma acidophilum." Thesis, University of Bath, 1988. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383178.

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42

Strömmer, Lisa. "Insulin action and secretion after surgical trauma : an experimental study in the rat /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4295-1/.

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43

Abbot, Emily L. "Regulation of the pyruvate dehydrogenase complex (PDC) in cultured human muscle cells." Thesis, University of Newcastle Upon Tyne, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275560.

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44

Choi, Kevin. "Glucose Kinetics of Hyperglycemic Rainbow Trout: Effects of Exogenous Glucose and Exercise." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/32856.

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This thesis investigates the ability of rainbow trout to modulate hepatic glucose production (Ra) and disposal (Rd). My goals were to determine: (1) if resting trout can modulate fluxes to cope with exogenous glucose; (2) how fluxes change during graded swimming; (3) how exogenous glucose affects swimming kinetics; and (4) if exogenous glucose affects cost of transport or performance. Results show that resting trout suppress Ra completely and stimulate Rd from 10.6 to 27.6 μmol kg-1 min-1. During swimming, fluxes increase from 15.6 to 21.9 μmol kg-1 min-1, but only at speeds >2.4 BL s-1. When

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45

Al-Basheri, Khalid Ali. "The biochemistry and genetics of maltose metabolism in clostridium acetobutylicum." Thesis, Heriot-Watt University, 1994. http://hdl.handle.net/10399/1383.

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46

Lévy, Pierre. "Hepatitis C virus-induced reprogramming of glutamine metabolism." Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10328.

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L'hépatite C chronique est une des étiologies principales du carcinome hépatocellulaire. En revanche, les mécanismes de tumorigenèse sont peu connus. Récemment, plusieurs modifications du métabolisme du glucose ont été décrites dans les cellules infectées par le HCV. Celles-ci évoquent les reprogrammations métaboliques mises en place dans les cellules cancéreuses. L'effet Warburg, ou glycolyse aérobie, est une des caractéristiques principales des cellules tumorales. Ce phénomène permet d'assurer une production énergétique ainsi qu'un stock en précurseurs de macromolécules suffisants pour perme

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47

Salem, Nicolas. "[(Methyl)1-^11C]-Acetate Metabolism in Hepatocellular Carcinoma." Cleveland, Ohio : Case Western Reserve University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1238772847.

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48

Douen, A. G. "The mechanism of D-glucose uptake in rat adipocytes." Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.378811.

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49

Stevens, Joseph R. "The Effects of Low Dose Endotoxin on Glucose Homeostasis." Diss., Virginia Tech, 2014. http://hdl.handle.net/10919/64849.

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Obese individuals present with an increased inflammatory tone as compared to healthy, normal-weight individuals, which is associated with insulin resistance. One factor hypothesized to contribute to increased inflammation in obese and diabetic states is elevated blood endotoxin levels, also known as metabolic endotoxemia. In healthy rodents (non-obese and insulin sensitive), there is evidence that blood endotoxin levels fluctuate over the course of the day with elevations in the post-prandial state that return to baseline levels in the post-absorptive state. High-fat feeding in these animals a

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50

Trajanoski, Zlatko. "Techniques for in vivo assessment of glucose and FFA metabolism /." Aachen : Shaker, 1998. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=015159660&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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