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Relevant bibliographies by topics / Glycolysis pathway

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Academic literature on the topic 'Glycolysis pathway'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Glycolysis pathway"

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Kuijpers, Niels G. A., Daniel Solis-Escalante, Marijke A. H. Luttik, et al. "Pathway swapping: Toward modular engineering of essential cellular processes." Proceedings of the National Academy of Sciences 113, no. 52 (2016): 15060–65. http://dx.doi.org/10.1073/pnas.1606701113.

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Recent developments in synthetic biology enable one-step implementation of entire metabolic pathways in industrial microorganisms. A similarly radical remodelling of central metabolism could greatly accelerate fundamental and applied research, but is impeded by the mosaic organization of microbial genomes. To eliminate this limitation, we propose and explore the concept of “pathway swapping,” using yeast glycolysis as the experimental model. Construction of a “single-locus glycolysis” Saccharomyces cerevisiae platform enabled quick and easy replacement of this yeast’s entire complement of 26 g

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Yumnamcha, Thangal, Michael Guerra, Lalit Pukhrambam Singh, and Ahmed S. Ibrahim. "Metabolic Dysregulation and Neurovascular Dysfunction in Diabetic Retinopathy." Antioxidants 9, no. 12 (2020): 1244. http://dx.doi.org/10.3390/antiox9121244.

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Diabetic retinopathy is a major cause of ocular complications in patients with type 1 and type 2 diabetes in developed countries. Due to the continued increase in the number of people with obesity and diabetes in the United States of America and globally, the incidence of diabetic retinopathy is expected to increase significantly in the coming years. Diabetic retinopathy is widely accepted as a combination of neurodegenerative and microvascular changes; however, which change occurs first is not yet understood. Although the pathogenesis of diabetic retinopathy is very complex, regulated by nume

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Lund, Jenny, D. Margriet Ouwens, Marianne Wettergreen, Siril S. Bakke, G. Hege Thoresen, and Vigdis Aas. "Increased Glycolysis and Higher Lactate Production in Hyperglycemic Myotubes." Cells 8, no. 9 (2019): 1101. http://dx.doi.org/10.3390/cells8091101.

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Previous studies have shown that chronic hyperglycemia impairs glucose and fatty acid oxidation in cultured human myotubes. To further study the hyperglycemia-induced suppression of oxidation, lactate oxidation, mitochondrial function and glycolytic rate were evaluated. Further, we examined the intracellular content of reactive oxygen species (ROS), production of lactate and conducted pathway-ANOVA analysis on microarray data. In addition, the roles of the pentose phosphate pathway (PPP) and the hexosamine pathway were evaluated. Lactic acid oxidation was suppressed in hyperglycemic versus nor

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DANDEKAR, Thomas, Stefan SCHUSTER, Berend SNEL, Martijn HUYNEN, and Peer BORK. "Pathway alignment: application to the comparative analysis of glycolytic enzymes." Biochemical Journal 343, no. 1 (1999): 115–24. http://dx.doi.org/10.1042/bj3430115.

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Comparative analysis of metabolic pathways in different genomes yields important information on their evolution, on pharmacological targets and on biotechnological applications. In this study on glycolysis, three alternative ways of comparing biochemical pathways are combined: (1) analysis and comparison of biochemical data, (2) pathway analysis based on the concept of elementary modes, and (3) a comparative genome analysis of 17 completely sequenced genomes. The analysis reveals a surprising plasticity of the glycolytic pathway. Isoenzymes in different species are identified and compared; dev

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Reiter, Russel J., Ramaswamy Sharma, and Sergio Rosales-Corral. "Anti-Warburg Effect of Melatonin: A Proposed Mechanism to Explain its Inhibition of Multiple Diseases." International Journal of Molecular Sciences 22, no. 2 (2021): 764. http://dx.doi.org/10.3390/ijms22020764.

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Glucose is an essential nutrient for every cell but its metabolic fate depends on cellular phenotype. Normally, the product of cytosolic glycolysis, pyruvate, is transported into mitochondria and irreversibly converted to acetyl coenzyme A by pyruvate dehydrogenase complex (PDC). In some pathological cells, however, pyruvate transport into the mitochondria is blocked due to the inhibition of PDC by pyruvate dehydrogenase kinase. This altered metabolism is referred to as aerobic glycolysis (Warburg effect) and is common in solid tumors and in other pathological cells. Switching from mitochondri

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Fontaine, Krystal A., Erica L. Sanchez, Roman Camarda, and Michael Lagunoff. "Dengue Virus Induces and Requires Glycolysis for Optimal Replication." Journal of Virology 89, no. 4 (2014): 2358–66. http://dx.doi.org/10.1128/jvi.02309-14.

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ABSTRACTViruses rely on host cellular metabolism to provide the energy and biosynthetic building blocks required for their replication. Dengue virus (DENV), a member of theFlaviviridaefamily, is one of the most important arthropod-borne human pathogens worldwide. We analyzed global intracellular metabolic changes associated with DENV infection of primary human cells. Our metabolic profiling data suggested that central carbon metabolism, particularly glycolysis, is strikingly altered during a time course of DENV infection. Glucose consumption is increased during DENV infection and depriving DEN

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Alfarouk, Khalid O., Samrein B. M. Ahmed, Robert L. Elliott, et al. "The Pentose Phosphate Pathway Dynamics in Cancer and Its Dependency on Intracellular pH." Metabolites 10, no. 7 (2020): 285. http://dx.doi.org/10.3390/metabo10070285.

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The Pentose Phosphate Pathway (PPP) is one of the key metabolic pathways occurring in living cells to produce energy and maintain cellular homeostasis. Cancer cells have higher cytoplasmic utilization of glucose (glycolysis), even in the presence of oxygen; this is known as the “Warburg Effect”. However, cytoplasmic glucose utilization can also occur in cancer through the PPP. This pathway contributes to cancer cells by operating in many different ways: (i) as a defense mechanism via the reduced form of nicotinamide adenine dinucleotide phosphate (NADPH) to prevent apoptosis, (ii) as a provisi

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Shin, Minhye, and Soo Rin Kim. "Metabolic Changes Induced by Deletion of Transcriptional Regulator GCR2 in Xylose-Fermenting Saccharomyces cerevisiae." Microorganisms 8, no. 10 (2020): 1499. http://dx.doi.org/10.3390/microorganisms8101499.

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Glucose repression has been extensively studied in Saccharomyces cerevisiae, including the regulatory systems responsible for efficient catabolism of glucose, the preferred carbon source. However, how these regulatory systems would alter central metabolism if new foreign pathways are introduced is unknown, and the regulatory networks between glycolysis and the pentose phosphate pathway, the two major pathways in central carbon metabolism, have not been systematically investigated. Here we disrupted gcr2, a key transcriptional regulator, in S. cerevisiae strain SR7 engineered to heterologously

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Janulevicius, Albertas, and G. Sander van Doorn. "Selection for rapid uptake of scarce or fluctuating resource explains vulnerability of glycolysis to imbalance." PLOS Computational Biology 17, no. 1 (2021): e1008547. http://dx.doi.org/10.1371/journal.pcbi.1008547.

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Glycolysis is a conserved central pathway in energy metabolism that converts glucose to pyruvate with net production of two ATP molecules. Because ATP is produced only in the lower part of glycolysis (LG), preceded by an initial investment of ATP in the upper glycolysis (UG), achieving robust start-up of the pathway upon activation presents a challenge: a sudden increase in glucose concentration can throw a cell into a self-sustaining imbalanced state in which UG outpaces LG, glycolytic intermediates accumulate and the cell is unable to maintain high ATP concentration needed to support cellula

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Crowther, Gregory J., William F. Kemper, Michael F. Carey, and Kevin E. Conley. "Control of glycolysis in contracting skeletal muscle. II. Turning it off." American Journal of Physiology-Endocrinology and Metabolism 282, no. 1 (2002): E74—E79. http://dx.doi.org/10.1152/ajpendo.2002.282.1.e74.

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Glycolytic flux in muscle declines rapidly after exercise stops, indicating that muscle activation is a key controller of glycolysis. The mechanism underlying this control could be 1) a Ca2+-mediated modulation of glycogenolysis, which supplies substrate (hexose phosphates, HP) to the glycolytic pathway, or 2) a direct effect on glycolytic enzymes. To distinguish between these possibilities, HP levels were raised by voluntary 1-Hz exercise, and glycolytic flux was measured after the exercise ceased. Glycolytic H+ and ATP production were quantified from changes in muscle pH, phosphocreatine con

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Dissertations / Theses on the topic "Glycolysis pathway"

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Shao, Wei 1970. "Identification of caspase-1 and caspase-3 substrates and study on caspase-1 substrates in glycolytic pathway." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100248.

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Apoptosis is executed by caspase-mediated cleavage of various proteins. Elucidating the consequence of substrate cleavage provides us with insight into cell death and other biological processes. In this study, we applied the diagonal gel approach, a proteomic strategy, to identify substrates of the inflammatory caspase, caspase-1 and the cell death executioner caspase, caspase-3. Our results showed significant overlap between the substrates cleaved by both caspase-1 and -3. Such substrates are implicated in common cellular functions, including maintenance of the cytoskeleton, folding of protei

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Barger, Jennifer F. "An Oncogenic Signal Pathway Dictates the Metabolic Requirements for Survival." University of Cincinnati / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321888795.

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Poulain, Laury. "Etude du métabolisme du glucose dans les leucémies aigües myéloïdes et implication de la voie de signalisation mTORC1." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB028/document.

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Les Leucémies Aigües Myéloïdes (LAM) sont des hémopathies malignes hétérogènes de mauvais pronostic qui se caractérisent par une expansion clonale de progéniteurs immatures. De nombreuses dérégulations de voies de signalisation sont retrouvées dans les cellules leucémiques et leur confèrent un avantage de prolifération et de survie. La voie de signalisation mTORC1, qui contrôle la traduction protéique, l’autophagie et plusieurs voies métaboliques, est ainsi constitutivement activée dans les cellules leucémiques. La reprogrammation métabolique notamment via « l’effet Warburg » est un phénomène

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Zheng, Chaoqun. "Molecular mechanisms of the anti-cancer action of schweinfurthins." Diss., University of Iowa, 2015. https://ir.uiowa.edu/etd/1815.

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Schweinfurthins are a family of natural products with significant anti-cancer activities. They were originally identified in the National Cancer Institute (NCI) human 60 cancer cell line screening. The growth inhibition profile of schweinfurthins is distinct from other clinically used anti-cancer agents, indicating that they have a novel mechanism of action or have a previously unrecognized protein target. Previous studies showed that schweinfurthins affect multiple cellular processes in cancer cells. For example, schweinfurthins can alter cytoskeleton organization, induce ER stress and apopto

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Bauffe, Frédérique. "Etude de protéines parasitaires pour l'amélioration des tests de diagnostic rapide du paludisme." Thesis, Aix-Marseille, 2012. http://www.theses.fr/2012AIXM5068.

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Le paludisme est un problème de santé public dans de nombreux pays. Cinq espèces infectent l'homme : P. falciparum, responsable de la grande majorité des décès, et P. vivax, P. ovale, P. malariae et P. knowlesi qui provoquent des formes bénignes de la maladie. Le diagnostic qui fait partie des moyens de lutte, est une urgence médicale. Les tests de diagnostic rapides (TDRs) dont l'usage est recommandés par l'OMS, sont donc de plus en plus employés. Cependant, la détection et l'identification des espèces non P. falciparum par ces tests est insuffisante. Le besoin en nouveaux couples « antigènes

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Zeng, Sheng. "Mycobacterium bovis BCG chaperonin 60.1 contributes to adaptations under stresses: implication for escaping isoniazid bactericidal mechanism and for mycobacterial biofilm growth." Doctoral thesis, Universite Libre de Bruxelles, 2019. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/286394.

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Tuberculosis, caused by Mycobacterium tuberculosis, still poses a huge global health threat today. During infection, the bacilli are believed to confront with various stresses, including hypoxia. Hypoxia is known to trigger the bacteria to adapt into a nonreplicating dormant state associated with reduced drug susceptibility. In addition to dormancy, mycobacteria, like other bacteria, may switch to sessile biofilm growth that is generally associated with augmented drug and stress tolerance. Bacterial biofilm is physically heterogeneous and may harbor cells displaying distinct metabolic activiti

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Tulsian, Richa. "Circadian Clock as the mechanism of Caloric Restriction in regulating mTOR Signaling and Glucose Homeostasis." Cleveland State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=csu1539971252367088.

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Williams, Jonathan Glyn. "Isoenzyme specific PFK-2/FBPase-2 inhibition as an anti-cancer strategy." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:7f47d9bb-7a9d-4dbc-92fa-57d2654640d1.

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High aerobic glycolytic capacity is correlated with poor prognosis and increased tumour aggressiveness. 6Phosphofructo-1-kinase catalyses the first irreversible step of glycolysis, and is activated by fructose-2,6-bisphosphate, a product of the kinase activity of four bifunctional isoenzymes, 6-phosphofructo-2-kinase/fructose-2,6-biphosphatase (PFK-2/FBPase-2:PFKFB1-4). These are potential anti-tumour targets, but their individual and collective role requires further investigation. This thesis had three aims; to validate the PFK-2/FBPase-2 isoenzymes as anti-cancer targets, to investigate the

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Dwyer, Trisha A. "Brain Hypometabolism and Seizures: The Dynamics of Hypoxia and Hypoglycemia in Brain Energy Homeostasis." University of Toledo Health Science Campus / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=mco1313737400.

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Sahin, Ceylan. "Contributions To The Kinetic Modeling Of Glycolytic Pathway In Yeast." Phd thesis, METU, 2009. http://etd.lib.metu.edu.tr/upload/3/12610535/index.pdf.

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Being at the center of most metabolic pathways and also one of the best known pathways, the glycolytic pathway has been of interest to modeling studies. This study is composed of our attempts to model ethanolic fermentation by yeast through kinetic equations of glycolytic steps and its branches. Model was based totally on experimentally measured kinetics of enzymes and transport steps, either obtained in this study or from the literature. Effect of ethanol on enzyme activities was tested in the range of ethanol 0 to 20% (v/v) in assay mixture. All enzymes were inhibited by ethanol to some deg

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Books on the topic "Glycolysis pathway"

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Schurr, Avital, and Evelyne Gozal, eds. Glycolysis at 75: Is it Time to Tweak the First Elucidated Metabolic Pathway in History? Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-586-2.

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Ross, John, Igor Schreiber, and Marcel O. Vlad. Determination of Complex Reaction Mechanisms. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195178685.001.0001.

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In a chemical system with many chemical species several questions can be asked: what species react with other species: in what temporal order: and with what results? These questions have been asked for over one hundred years about simple and complex chemical systems, and the answers constitute the macroscopic reaction mechanism. In Determination of Complex Reaction Mechanisms authors John Ross, Igor Schreiber, and Marcel Vlad present several systematic approaches for obtaining information on the causal connectivity of chemical species, on correlations of chemical species, on the reaction pathw

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Merriman, Tony R. The genetic basis of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0040.

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An individual’s risk of gout is determined by a complex relationship between inherited genetic variants and environmental exposures. Genetic variants that control hyperuricaemia and subsequent progression to clinical gout specify pathogenic pathways that could be therapeutically targeted. Genome-wide association studies (GWAS) have provided novel insights into the pathways leading to hyperuricaemia. GWAS have identified the renal uric acid transporter SLC2A9/GLUT9 and the gut excretory molecule ABCG2, which each have very strong genetic effects in the control of urate levels and risk of gout.

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Burke, David, and James Howells. The motor unit. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0002.

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The motor unit represent the final output of the motor system. Each consists of a motoneuron, its axon, neuromuscular junctions, and muscle fibres innervated by that axon. The discharge of a motor unit can be followed by recording its electromyographic signature, the motor unit action potential. Motoneurons are not passive responders to the excitatory and inhibitory influences on them from descending and segmental sources. Their properties can change, e.g. due to descending monoaminergic pathways, which can alter their responses to other inputs (changing ‘reflex gain’). Contraction strength de

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Book chapters on the topic "Glycolysis pathway"

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Alemanno, Fernando. "Anaerobic Glycolysis or Embden–Meyerhof Pathway." In Biochemistry for Anesthesiologists and Intensivists. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26721-6_2.

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Wamelink, Mirjam M. C., Vassili Valayannopoulos, and Barbara Garavaglia. "Disorders of Glycolysis and the Pentose Phosphate Pathway." In Inborn Metabolic Diseases. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49771-5_7.

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ap Rees, T. "The Organization of Glycolysis and the Oxidative Pentose Phosphate Pathway in Plants." In Higher Plant Cell Respiration. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70101-6_14.

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Li, Ting, Christopher Copeland, and Anne Le. "Glutamine Metabolism in Cancer." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_2.

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AbstractMetabolism is a fundamental process for all cellular functions. For decades, there has been growing evidence of a relationship between metabolism and malignant cell proliferation. Unlike normal differentiated cells, cancer cells have reprogrammed metabolism in order to fulfill their energy requirements. These cells display crucial modifications in many metabolic pathways, such as glycolysis and glutaminolysis, which include the tricarboxylic acid (TCA) cycle, the electron transport chain (ETC), and the pentose phosphate pathway (PPP) [1]. Since the discovery of the Warburg effect, it has been shown that the metabolism of cancer cells plays a critical role in cancer survival and growth. More recent research suggests that the involvement of glutamine in cancer metabolism is more significant than previously thought. Glutamine, a nonessential amino acid with both amine and amide functional groups, is the most abundant amino acid circulating in the bloodstream [2]. This chapter discusses the characteristic features of glutamine metabolism in cancers and the therapeutic options to target glutamine metabolism for cancer treatment.

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Chong, Chuii Khim, Mohd Saberi Mohamad, Safaai Deris, et al. "Using an Improved Differential Evolution Algorithm for Parameter Estimation to Simulate Glycolysis Pathway." In Advances in Intelligent and Soft Computing. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28765-7_85.

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Teramoto, Haruhiko, and Masayuki Inui. "Regulation of Sugar Uptake, Glycolysis, and the Pentose Phosphate Pathway in Corynebacterium glutamicum." In Corynebacterium glutamicum. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-29857-8_9.

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Chong, Chuii Khim, Mohd Saberi Mohamad, Safaai Deris, Yee Wen Choon, and Lian En Chai. "Parameter Estimation for Simulation of Glycolysis Pathway by Using an Improved Differential Evolution." In Communications in Computer and Information Science. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-32826-8_37.

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Chassagnole, Christophe, Juan-Carlos A. Rodriguez, Andrei Doncescu, and Laurence T. Yang. "Differential Evolutionary Algorithms for In Vivo Dynamic Analysis of Glycolysis and Pentose Phosphate Pathway inEscherichia coli." In Parallel Computing for Bioinformatics and Computational Biology. John Wiley & Sons, Inc., 2005. http://dx.doi.org/10.1002/0471756504.ch3.

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Cocuron, Jean-Christophe, and Ana Paula Alonso. "Liquid Chromatography Tandem Mass Spectrometry for Measuring 13C-labeling in Intermediates of the Glycolysis and Pentose Phosphate Pathway." In Plant Metabolic Flux Analysis. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-688-7_9.

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Ochs, Raymond S. "Carbohydrate Pathways Related to Glycolysis." In Biochemistry, 2nd ed. CRC Press, 2021. http://dx.doi.org/10.1201/9781003029649-13.

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Conference papers on the topic "Glycolysis pathway"

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Wang, Li, Yongqian Zhang, Hong Qing, et al. "Retention Time Prediction Based Proteomic Analysis on Proteins from Glycolysis/Gluconeogenesis Pathway of E. coli." In 2012 International Conference on Biomedical Engineering and Biotechnology (iCBEB). IEEE, 2012. http://dx.doi.org/10.1109/icbeb.2012.344.

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Gershon, Eli. "Robust H∞ and peak to peak output-feedback control analysis of the Glycolysis pathway in yeasts." In 2016 24th Mediterranean Conference on Control and Automation (MED). IEEE, 2016. http://dx.doi.org/10.1109/med.2016.7535990.

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Feng, Yan, Weiguo Lv, Xing Xie, and Yan Yu. "Abstract 5420: PGAM1-REDD1 pathway promotes paclitaxel resistance through enhancing aerobic glycolysis in ovarian cancer cells." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5420.

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Yuan, Tai-Yi, Hanan N. Fernando, Jessica Czamanski, Chong Wang, Wei Yong Gu, and Chun-Yuh Huang. "Effects of Static Compression on Energy Metabolism of Porcine Intervertebral Disc." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19600.

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Degeneration of the intervertebral disc (IVD) has been associated with low back pain, which is one of the major socio-economic problems in the United States. Since IVD is the largest avascular cartilaginous structure in the human body, poor nutrient supply has been suggested as a potential mechanism for IVD degeneration. Biosynthesis of extracellular matrix is an energy demanding process which is required to maintain tissue integrity [1]. Cells consume glucose and oxygen to produce adenosine triphosphate (ATP), the main energy form in cells. Glycolysis, the primary metabolic pathway for produc

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Nakamura, Takuya, Satoru Shinriki, Mitsuhiro Hayashi, et al. "Abstract 5149: CYLD downregulation induces aerobic glycolysis via mTOR pathway in human oral squamous cell carcinoma cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5149.

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Liu, Wen, Benjamin Beck, Kedar S. Vaidya, Kevin T. Nash, Scott W. Ballinger та Danny R. Welch. "Abstract 126: The KISS1 metastasis suppressor appears to integrate glycolysis, mitochondrial biogenesis and metastasis via regulation of a PGC1α pathway". У Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-126.

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Verma, Nisha, Mario Pink, Albert W. Rettenmeier, and Simone Schmitz-Spanke. "722 Induction of metabolic shift from glycolysis to pentose phosphate pathway in human bladder cancer cells exposed to benzo[a]pyrene." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.1176.

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Verdone, James E., Jelani C. Zarif, and Kenneth J. Pienta. "Abstract 5100: Aerobic glycolysis is the primary metabolic pathway used for prostate cancer cell motility and invasion processes regardless of androgen sensitivity." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5100.

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El-fadl, Rihab, Nasser Rizk, Amena Fadel, and Abdelrahman El Gamal. "The Profile of Hepatic Gene Expression of Glucose Metabolism in Mice on High Fat Diet." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0213.

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Obesity is a growing problem worldwide, and recent data indicated that 20% of the populations would be obese. Obesity arises as a multifactorial disease caused by inherited traits that interact with lifestyle factors such as diet and physical activity. The liver plays an essential role in the gluco-regulation via regulating glucose, lipid and protein metabolism. The process of glucose metabolism is controlled by a range of molecular mechanisms and genes which affect the metabolism of the liver during intake of high fat diet (HFD). The objective of this research is to investigate the profile of

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Lo, Kwok Fung, Christopher W. Dawson, Lawrence S. Young, and Kwok Wai Lo. "Abstract LB-093: Activation of the FGFR1 signaling pathway by the epstein-barr virus-encoded LMP1 promotes aerobic glycolysis and transformation of human nasopharyngeal epithelial cells." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-lb-093.

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