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Journal articles on the topic 'Glycosuria'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Mng’agi, Mohamed O., and Ambele M. Mwandigha. "Gender-Inclined Young Age Glycosuria: Contribution to Late Age Chronic Renal Diseases, Type 2 Diabetes Mellitus and Cardiovascular Diseases." East African Health Research Journal 7, no. 1 (2023): 88–93. http://dx.doi.org/10.24248/eahrj.v7i1.713.

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Background: Chronic kidney diseases (CKD), Type 2 Diabetes Mellitus (T2DM) and cardiovascular diseases (CVDs) are the recent worldwide late age chronic conditions that could be a consequence of renal glycosuria during childhood. This study aimed at determining the extent of glycosuria in secondary school students to obtain information that could be predictive of the situation in late age life of Tanzanians living in Mkuranga District. Methodology: This was school-based cross-sectional study that was conducted in assenting and consenting 800 students from July to October 2019 in Mkuranga distri

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2

Falcão, Mário Cícero, Cléa Rodrigues Leone, and José Lauro Araújo Ramos. "Is glycosuria a reliable indicator of adequacy of glucose infusion rate in preterm infants?" Sao Paulo Medical Journal 117, no. 1 (1999): 19–24. http://dx.doi.org/10.1590/s1516-31801999000100004.

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CONTEXT: Adequacy of glucose infusion may be monitored via the glycosuria levels, as there is a relationship between glycemia and glycosuria regulated by the renal glucose threshold. In the neonatal period, however, this relationship is not so clear. OBJECTIVE: To evaluate the occurrence of glycosuria in preterm infants submitted to glucose infusion and to verify the relationship between glycosuria and blood glucose level. DESIGN: Accuracy study. SETTING: Neonatal intensive care unit of General Maternity Hospital. PATIENTS: 40 preterm newborns receiving glucose infusion. PROCEDURES: 511 concom

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3

Kaufmann, R. C., F. K. Khosho, and K. S. Amankwah. "Scanning Electron Microscopy of the glomerulus in diabetic BB/S wistar rats." Proceedings, annual meeting, Electron Microscopy Society of America 45 (August 1987): 730–31. http://dx.doi.org/10.1017/s0424820100127967.

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Renal damage secondary to diabetes seems to be related to the severity and duration of the diabetes. In streptozotocin and alloxan-induced diabetic rats, renal disease is found only in those rats that have glycosuria and then only after the glycosuria has been present for many months. In these animals, the longer they have glycosuria, the more severe the renal damage. In our colony of BB/S Wistar rats, animals that are going. to become frankly diabetic demonstrate clinical diabetes before they begin spilling glucose in their urine. After glycosuria develops, the condition of the animals worsen

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4

Lee, Matthew A., George McMahon, Ville Karhunen, et al. "Common variation at 16p11.2 is associated with glycosuria in pregnancy: findings from a genome-wide association study in European women." Human Molecular Genetics 29, no. 12 (2020): 2098–106. http://dx.doi.org/10.1093/hmg/ddaa054.

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Abstract Glycosuria is a condition where glucose is detected in urine at higher concentrations than normal (i.e. not detectable). Glycosuria at some point during pregnancy has an estimated prevalence of 50% and is associated with adverse outcomes in both mothers and offspring. Little is currently known about the genetic contribution to this trait or the extent to which it overlaps with other seemingly related traits, e.g. diabetes. We performed a genome-wide association study (GWAS) for self-reported glycosuria in pregnant mothers from the Avon Longitudinal Study of Parents and Children (cases

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5

Ekun, Oloruntoba Ayodele, Franklin Kayode Ayenogun, Nkeiruka Ogo Ogidi, and Esther Ngozi Adejumo. "Assessment of the Risk of Future Metabolic Syndrome among Non-Hypertensive and Non-Diabetic Nigerian Pregnant Women Presenting with either Glycosuria or Proteinuria at Different Trimesters." Rwanda Journal of Medicine and Health Sciences 6, no. 3 (2023): 315–25. http://dx.doi.org/10.4314/rjmhs.v6i3.5.

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BackgroundIn pregnancy, women experience physiological changes which could increase the risk of insulin resistance, and metabolic syndrome later in life. This study assessed the risk of future metabolic syndrome among pregnant women with either glycosuria or proteinuria at different gestational ages. MethodsEight-Six participants were recruited from health facilities in South-west, Nigeria and they were in three categories: those with glycosuria (n = 32), proteinuria (n = 27), and control (n = 27), based on urinalysis result. Data were analyzed using IBM SPSS Statistics for Windows version 25.

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6

Onuigbo, Macaulay, and Cynthia Washington. "Glycosuria in Alcoholics." Nephron 87, no. 3 (2001): 287–88. http://dx.doi.org/10.1159/000045930.

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7

MELLON, J. P. "GLYCOSURIA AND PHENYLKETONURIA." Journal of Intellectual Disability Research 7, no. 2 (2008): 122–28. http://dx.doi.org/10.1111/j.1365-2788.1963.tb00791.x.

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8

Challen, Antoinette. "Glycosuria in childhood." Practical Diabetes International 4, no. 3 (1987): 114–15. http://dx.doi.org/10.1002/pdi.1960040306.

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9

G., V. "Glycosuria as a sign of pregnancy. Seit-z and Jess (Münch, ni. Woch .. 1922, no. 1)." Kazan medical journal 18, no. 2 (2021): 108. http://dx.doi.org/10.17816/kazmj79920.

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Glycosuria as a sign of pregnancy. Seit-z and Jess (Mnch, ni. Woch .. 1922, No. 1) found that when administered to pregnant women (at II-VIII months of pregnancy) 100 grm. of grape sugar, half of them get glycosuria.

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10

TRACHTMAN, HOWARD, and RONALD KAHN. "Renal Glycosuria in Pyelonephritis." Pediatrics 83, no. 4 (1989): 652–53. http://dx.doi.org/10.1542/peds.83.4.652a.

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Standard pediatric nephrology textbooks do not include acute pyelonephritis in the differential diagnosis of renal glycosuria. Recently, we encountered a 9-month-old girl who had fever up≤39.4°C(103°F) for five to six days. On the sixth day of her illness, a Chemstrip urinalysis was performed and demonstrated the presence of 2+ pyuria, 1+ protein, 1+ hematuria, and 4+ glycosuria. A simultaneous blood glucose concentration was 107 mg/ dL. A repeat urinalysis, done 32 hours after a feeding, confirmed the presence of renal glycosuria, a concurrent serum glucose of 129 mg/dL, pyuria, minimal hemat

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11

Lushnikova, L. A. "Towards recognizing renal diabetes." Kazan medical journal 43, no. 3 (2021): 54–55. http://dx.doi.org/10.17816/kazmj83817.

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Even Lenin (1895) and Klemperer (1896) described chronic glycosuria, which by pathogenesis, clinical picture and prognosis has nothing to do with classical diabetes mellitus. The authors called this form renal diabetes, considering that glycosuria is based on an increase in the permeability of the renal barrier for sugar.

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12

Imad Mahi, Dounia El Moujtahide, El Houcine Sebbar, and Mohammed Choukri. "Verification of analytical performance of glycosuria assay on the Abbott Architect® analyzer: Experience of the Biochemistry Laboratory, Mohammed VI University Hospital of Oujda." World Journal of Biology Pharmacy and Health Sciences 21, no. 1 (2025): 511–15. https://doi.org/10.30574/wjbphs.2025.21.1.0058.

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The aim of our study was to evaluate the analytical performance of the glycosuria assay method using the ARCHITECT c Systems automated analyzer in the biochemistry laboratory of the Mohammed VI University Hospital in Oujda. Glycosuria, defined as the presence of glucose in urine, is a key biomarker in the diagnosis and monitoring of diabetes mellitus, reflecting disturbances in glucose metabolism. We conducted a performance study of the ARCHITECT c Systems analyzer, focusing on repeatability and reproducibility, in accordance with the COFRAC GTA 04 accreditation technical guide, which aligns w

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13

Rukh, Mah, Aqsa Naeem, Abida Bibi, Syed Bilal Hussain, and Muhammad Imran Qadir. "Can Perfume Allergy Cause Glycosuria?" Mediterranean Journal of Basic and Applied Sciences 07, no. 01 (2023): 27–28. http://dx.doi.org/10.46382/mjbas.2023.7103.

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The main aim of this research is to correlate relation between ornament allergy and glycosuria. Glycosuria is a process in which glucose is removed from urine. The value of glucose in blood must be less than 7 mmol/L. Glycosuria is caused by diabetes, high sugar diet, emotion and liver cirrhosis. Some people are sensitive to some fragrances or ornaments when they come in contact with these ornaments, they show marks of allergy like disease in respiratory track, fever, rashes and headache. The best treatment is to avoid such ornaments having unpleasant sharp smell which cause rashes. 17% males

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14

Mah, Rukh, Naeem Aqsa, Bibi Abida, Bilal Hussain Syed, and Imran Qadir Muhammad. "Can Perfume Allergy Cause Glycosuria?" Mediterranean Journal of Basic and Applied Sciences (MJBAS) 7, no. 1 (2023): 27–28. https://doi.org/10.46382/MJBAS.2023.7103.

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he main aim of this research is to correlate relation between ornament allergy and glycosuria. Glycosuria is a process in which glucose is removed from urine. The value of glucose in blood must be less than 7 mmol/L. Glycosuria is caused by diabetes, high sugar diet, emotion and liver cirrhosis. Some people are sensitive to some fragrances or ornaments when they come in contact with these ornaments, they show marks of allergy like disease in respiratory track, fever, rashes and headache. The best treatment is to avoid such ornaments having unpleasant sharp smell which cause rashes. 17% males h

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15

DONATO, LUIGI, and GIOVANNI TURCHETTI. "Renal Glycosuria in Pregnancy." Acta Medica Scandinavica 152, no. 3 (2009): 223–30. http://dx.doi.org/10.1111/j.0954-6820.1955.tb03481.x.

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16

Sivaji, Vaishnavi, Prasanna Raju, Soundarrajan Marimuthu, and Subash Sundar. "Familial renal glycosuria identified in an Indian family." BMJ Case Reports 17, no. 9 (2024): e258408. http://dx.doi.org/10.1136/bcr-2023-258408.

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Glycosuria can be isolated or it can be associated with other tubulopathies like proximal renal tubular acidosis, Fanconi syndrome and endocrine conditions like diabetes mellitus.The SLC5A2gene codes for the SGLT2 transporter, which is responsible for glucose reabsorption in the proximal tubule. Previously reported cases show that mutation in this gene is associated with intellectual disability, seizure disorder and renin and angiotensin system dysfunction. In his early childhood, a male child displayed persistently high urine glucose levels. We ruled out diabetes mellitus and other tubulopath

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17

Davies, Melanie, and John Day. "Screening for Non-Insulin-Dependent Diabetes Mellitus (NIDDM): How Often Should it Be Performed?" Journal of Medical Screening 1, no. 2 (1994): 78–81. http://dx.doi.org/10.1177/096914139400100202.

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Objectives— To examine whether a screening programme for diabetes repeated after an interval of 30 months is worthwhile both in terms of yield of new cases and continued high response rate. Methods— Self testing for postprandial glycosuria was used as it has been shown to have a good response rate, a good yield of cases of diabetes, and a sensitivity and specificity which compares favourably with more expensive and invasive screening methods. A total of 3231 subjects aged 45–70 years in one practice were screened on two occasions 30 months apart. Eighty seven subjects known to have diabetes we

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18

Khatoon, Tayyaba, Ataullah Fahad, and Naved Ahmad. "Unveiling Renal Glycosuria: A Case Study Integrating Conventional and Unani Medicine Approaches." International Journal of AYUSH Case Reports 8, no. 4 (2024): 531–36. https://doi.org/10.70805/ija-care.v8i4.636.

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A male patient of 60 years of age presented with chief complaints of Chronic Obstructive Pulmonary Disease (COPD) at the OPD of Ajmal Khan Tibbiya College Hospital, without any past-history of diabetes, TB, or other systemic illnesses. The patient underwent clinical examination and routine investigations, including urine analysis and blood tests. Urine analysis revealed unexpected glycosuria (glucose positive) despite normal blood glucose levels (80mg/dl). Other parameters were within normal limits, except for mildly elevated ALP (176 IU/L). The diagnosis of renal glycosuria was established af

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19

KAYNAR, Kübra, Canan ŞEHİT, Arif Mansur COŞAR, and Gül CEBECİOĞLU HASANÇEBİ. "A Case with Renal Glycosuria." Turkiye Klinikleri Journal of Case Reports 26, no. 4 (2018): 213–15. http://dx.doi.org/10.5336/caserep.2018-61441.

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20

Ferrannini, Ele. "Learning From Glycosuria: FIG. 1." Diabetes 60, no. 3 (2011): 695–96. http://dx.doi.org/10.2337/db10-1667.

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21

Holst, J. E. "Investigation into the adrenalin-glycosuria." Acta Medica Scandinavica 74, S34 (2009): 270–76. http://dx.doi.org/10.1111/j.0954-6820.1930.tb14963.x.

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22

WATSON, WILLIAM J. "Screening for Glycosuria During Pregnancy." Southern Medical Journal 83, no. 2 (1990): 156–58. http://dx.doi.org/10.1097/00007611-199002000-00006.

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23

MØLLER, ELSE. "NONDIABETIC GLYCOSURIA IN CHRONIC SCHIZOPHRENIA1." Acta Psychiatrica Scandinavica 24, no. 2 (2007): 223–50. http://dx.doi.org/10.1111/j.1600-0447.1949.tb03495.x.

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24

Bacalov, Iurie, Aurelia Crivoi, Elena Chirita, and Adriana Druta. "Evaluation of the efficacy of phytopreparations in preventing glycosuria, ketonuria, and proteinuria in experimental models of diabetes." Studia Universitatis Moldaviae. Seria Științe ale Naturii, no. 6 (176) (December 2024): 133–39. https://doi.org/10.59295/sum6(176)2024_18.

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Diabetes mellitus is a chronic disease that requires close monitoring to prevent complications such as ketonuria, proteinuria, and glycosuria, each indicating poor disease control. Ketonuria occurs when the body breaks down fats instead of glucose for energy and is more common in type 1 diabetes. Proteinuria indicates kidney damage caused by high blood sugar levels. Glycosuria reflects the kidneys’ inability to fully reabsorb glucose, occurring when blood sugar levels are too high. Herbal preparations can provide complementary support to conventional treatment. Plants such as mulberry, ginseng

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25

Carel, R. S., D. S. Silverberg, R. Kaminsky, and A. Aviram. "Routine urinalysis (dipstick) findings in mass screening of healthy adults." Clinical Chemistry 33, no. 11 (1987): 2106–8. http://dx.doi.org/10.1093/clinchem/33.11.2106.

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Abstract We present results of simple urinalysis examinations (dipstick) in a large (approximately 21,000 people) working population. In about 10% of the screened individuals at least one abnormality was found. In men the most common finding was proteinuria (4.9%). Hematuria was found in 2.6% and glycosuria in 0.6%. The most prevalent finding in women was hematuria (8.1%), followed by proteinuria (3.9%) and glycosuria (0.6%). Only about 1% of the examinees had two or more abnormalities in their urine examinations. These findings attest to the utility and practicability of incorporating urine t

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26

Nielsen, A. Levin. "ON THE MECHANISM OF GLYCOSURIA. II1)." Acta Medica Scandinavica 131, S213 (2009): 273–83. http://dx.doi.org/10.1111/j.0954-6820.1948.tb15137.x.

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27

FABER, KNUD, and A. NORGAARD. "Studies on the Threshold for Glycosuria." Acta Medica Scandinavica 54, no. 1 (2009): 289–322. http://dx.doi.org/10.1111/j.0954-6820.1921.tb15181.x.

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28

HOLST, JOHAN. "Glycosuria and diabetes in exophthalmic goitre." Acta Medica Scandinavica 55, no. 1 (2009): 302–22. http://dx.doi.org/10.1111/j.0954-6820.1921.tb15214.x.

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29

NIELSEN, A. LEVIN. "On the Mechanism of Glycosuria. I1." Acta Medica Scandinavica 130, no. 3 (2009): 219–31. http://dx.doi.org/10.1111/j.0954-6820.1948.tb10060.x.

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30

Rebelo, Joana Chaves, Felisbela Rocha, Susana Gama de Sousa, and Paulo Teixeira. "Renal glycosuria: report of two cases." Jornal Brasileiro de Nefrologia 34, no. 3 (2012): 291–92. http://dx.doi.org/10.5935/0101-2800.20120013.

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31

Goto, Masakatsu, W. Patrick Zeller, and R. Morrison Hurley. "Glycosuria and maternal licking in rats." Laboratory Animals 26, no. 4 (1992): 295–98. http://dx.doi.org/10.1258/002367792780745751.

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Maternal anogenital licking (MAGL) has been studied to understand the mechanism of maternal behaviour. The present study showed that rats had glycosuria at the concentration of 18-20 mg/dl and glucose was the preference of postpartum rats. MAGL increased on suckling rats separated for 24 h. However, wiping anogenital region attenuated the increase of MAGL. Therefore, glucose preference of postpartum rats may be involved in MAGL.

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32

Milavetz, James J. "Angiotensin-Converting Enzyme Inhibitors and Glycosuria." Archives of Internal Medicine 152, no. 5 (1992): 1081. http://dx.doi.org/10.1001/archinte.1992.00400170151027.

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33

Milavetz, J. J. "Angiotensin-converting enzyme inhibitors and glycosuria." Archives of Internal Medicine 152, no. 5 (1992): 1081–83. http://dx.doi.org/10.1001/archinte.152.5.1081.

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34

TUCKER, MIRIAM E. "Dapagliflozin Lowers Glucose by Raising Glycosuria." Clinical Endocrinology News 3, no. 7 (2008): 15. https://doi.org/10.1016/s1558-0164(08)70261-x.

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35

Dr., Aruna Maramulla* and Dr. Latha Carey1. "Diabetes Mellitus (Madhumeh)." Science World a monthly e magazine 3, no. 7 (2023): 1684–88. https://doi.org/10.5281/zenodo.8192814.

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<strong>Definition:</strong> It is a chronic complex metabolic disorder caused by insulin insufficiency/deficiency or impaired insulin action and characterized by glucose in tolerance, persistent hyperglycemia and glycosuria.

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36

Gruzdev, V. "Floridzin glycosuria as a sign of pregnancy." Kazan medical journal 20, no. 6 (2021): 644. http://dx.doi.org/10.17816/kazmj76805.

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Numerous observations have convinced Klaft'a (Zentr. F. Gyn., 1924, No. 17) that if after intravenous administration of 2 mg. floridzin in a woman does not get glycosuria, then pregnancy with a high probability can be excluded.

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37

Sahu, S., P. K. Jain, N. Mudgal, and G. Singh. "Glycosuria sensing based on nanometric plasmonic polaritons." IOP Conference Series: Materials Science and Engineering 1136, no. 1 (2021): 012064. http://dx.doi.org/10.1088/1757-899x/1136/1/012064.

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38

Murphy, Mary, and Nancy Risser. "Routine Urine Screening for Glycosuria and Proteinuria." Nurse Practitioner 22, no. 3 (1997): 211. http://dx.doi.org/10.1097/00006205-199703000-00024.

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39

HOLST, J. E. "Investigations into Benign Glycosuria and Diabetes Mellitus." Acta Medica Scandinavica 63, no. 1 (2009): 47–98. http://dx.doi.org/10.1111/j.0954-6820.1925.tb15344.x.

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40

Savit, Russ M., and David S. Udis. "“Upside-Down” Contrast-Urine Level in Glycosuria." Journal of Computer Assisted Tomography 11, no. 5 (1987): 911–12. http://dx.doi.org/10.1097/00004728-198709000-00036.

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41

Seedorff, H. H. "HEREDITARY RENAL GLYCOSURIA WITH CENTRAL MACULAR DEGENERATION." Acta Ophthalmologica 40, no. 1 (2009): 91–95. http://dx.doi.org/10.1111/j.1755-3768.1962.tb02356.x.

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42

Limsuwat, Chok, and Sharma S. Prabhakar. "Reversible Renal Glycosuria in Acute Interstitial Nephritis." American Journal of the Medical Sciences 344, no. 3 (2012): 245–47. http://dx.doi.org/10.1097/maj.0b013e318254bd71.

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43

Mavrakanas, Thomas A., Constantinos Tomos, Paraskevi Pratsiou, Ioanna Seventekidou, and George Tzouvelekis. "Transient glycosuria during a urinary tract infection." La Presse Médicale 39, no. 1 (2010): 145–46. http://dx.doi.org/10.1016/j.lpm.2009.09.002.

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44

Toka, Hakan R., Jun Yang, Chloe A. Zera, Jeremy S. Duffield, Martin R. Pollak, and David B. Mount. "Pregnancy-Associated Polyuria in Familial Renal Glycosuria." American Journal of Kidney Diseases 62, no. 6 (2013): 1160–64. http://dx.doi.org/10.1053/j.ajkd.2013.05.018.

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45

Kim, Kyeong Min, Soon Kil Kwon, and Hye-Young Kim. "A Case of Isolated Glycosuria Mediated by anSLC5A2Gene Mutation and Characterized by Postprandial Heavy Glycosuria Without Salt Wasting." Electrolytes & Blood Pressure 14, no. 2 (2016): 35. http://dx.doi.org/10.5049/ebp.2016.14.2.35.

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46

Sapienza, Giada Maria, Martina Greco, Marco Cardilicchia, et al. "Dal rene all'occhio: che cos'è la TINU?" Medico e Bambino Pagine elettroniche 27, no. 26 (2024): 2. http://dx.doi.org/10.53126/mebxxviisup2.

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An eight-year-old boy presenting with interstitial nephritis (abdominal pain, microhematuria, glycosuria, proteinuria, elevated urinary beta2-microglobulin levels) was eventually diagnosed with TINU syndrome (tubulointerstitial nephritis associated with uveitis) following the late onset of uveitis.

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47

Profili, Nicia I., Roberto Castelli, Antonio Gidaro, Roberto Manetti, Margherita Maioli, and Alessandro P. Delitala. "Sodium-Glucose Cotransporter-2 Inhibitors in Diabetic Patients with Heart Failure: An Update." Pharmaceuticals 17, no. 11 (2024): 1419. http://dx.doi.org/10.3390/ph17111419.

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Diabetes mellitus and heart failure are two diseases that are commonly found together, in particular in older patients. High blood glucose has a detrimental effect on the cardiovascular system, and worse glycemic control contributes to the onset and the recrudesce of heart failure. Therefore, any specific treatment aimed to reduce glycated hemoglobin may, in turn, have a beneficial effect on heart failure. Sodium-glucose cotransporter-2 inhibitors have been initially developed for the treatment of type 2 diabetes mellitus, and their significant action is to increase glycosuria, which in turn c

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48

Basso, Paulo José, Lucas Favaretto Tazinafo, Mauro Ferreira Silva, and Maria José Alves Rocha. "An alternative to the use of animals to teach diabetes mellitus." Advances in Physiology Education 38, no. 3 (2014): 235–38. http://dx.doi.org/10.1152/advan.00051.2014.

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We developed an alternative approach to teach diabetes mellitus in our practical classes, replacing laboratory animals. We used custom rats made of cloth, which have a ventral zipper that allows stuffing with glass marbles to reach different weights. Three mock rats per group were placed into metabolic cages with real food and water and with test tubes containing artificial urine, simulating a sample collection of 24 h. For each cage, we also provided other test tubes with artificial blood and urine, simulating different levels of hyperglycemia. The artificial “diabetic” urine contained differ

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49

Lytvyn, Yuliya, Marko Škrtić, Gary K. Yang, Paul M. Yip, Bruce A. Perkins, and David Z. I. Cherney. "Glycosuria-mediated urinary uric acid excretion in patients with uncomplicated type 1 diabetes mellitus." American Journal of Physiology-Renal Physiology 308, no. 2 (2015): F77—F83. http://dx.doi.org/10.1152/ajprenal.00555.2014.

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Plasma uric acid (PUA) is associated with metabolic, cardiovascular, and renal abnormalities in patients with type 2 diabetes but is less well understood in type 1 diabetes (T1D). Our aim was to compare PUA levels and fractional uric acid excretion (FEUA) in patients with T1D vs. healthy controls (HC) during euglycemia and hyperglycemia. PUA, FEUA, blood pressure (BP), glomerular filtration rate (GFR-inulin), and effective renal plasma flow (ERPF-paraaminohippurate) were evaluated in patients with T1D ( n = 66) during clamped euglycemia (glucose 4–6 mmol/l) and hyperglycemia (9–11 mmol/l), and

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50

Woronik, V., I. F. Freitas, L. B. Saldanha, E. Sabbaga, and M. Marcondes. "Glycosuria in glomerular diseases: histopathology and clinical correlations." Brazilian Journal of Medical and Biological Research 31, no. 5 (1998): 633–37. http://dx.doi.org/10.1590/s0100-879x1998000500005.

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