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Relevant bibliographies by topics / Glycosurie

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Academic literature on the topic 'Glycosurie'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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Journal articles on the topic "Glycosurie"

1

Brochériou, I., A. Duquesne, L. Garderet, et al. "Une pseudotumeur inflammatoire rétro-orbitaire révélatrice d’une glycosurie normoglycémique." Néphrologie & Thérapeutique 8, no. 1 (2012): 66. http://dx.doi.org/10.1016/j.nephro.2011.07.423.

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Neil Dalton, R., Martin J. Wiseman, Charles Turner, and Giancarlo Viberti. "Measurement of urinary para-aminohippuric acid in glycosurie diabetics." Kidney International 34, no. 1 (1988): 117–20. http://dx.doi.org/10.1038/ki.1988.153.

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RØMCKE, OLAF, and ERLING SKOUGE. "»Zur Frage der zentral bedingten Albuminurie, Glycosurie und Azetonurie»." Acta Medica Scandinavica 77, no. 1-3 (2009): 211–23. http://dx.doi.org/10.1111/j.0954-6820.1931.tb15394.x.

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ECKERSTRÖM, STEN. "HyperglycÉmie de type transitoire et glycosurie consÉcutives à un infarctus du myocarde." Acta Medica Scandinavica 95, no. 5 (2009): 528–38. http://dx.doi.org/10.1111/j.0954-6820.1938.tb16404.x.

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Schlienger, J. L. "Dans les coulisses de la chimie clinique : histoire de la glycémie et de la glycosurie." Médecine des Maladies Métaboliques 7, no. 1 (2013): 81–85. http://dx.doi.org/10.1016/s1957-2557(13)70496-6.

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Carpentier, C., G. Velho, K. Mohammedi, et al. "Abondance de la glycosurie pendant l’hyperglycémie et risque de néphropathie diabétique à long terme dans le diabète de type 1." Annales d'Endocrinologie 78, no. 4 (2017): 297. http://dx.doi.org/10.1016/j.ando.2017.07.252.

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Falcão, Mário Cícero, Cléa Rodrigues Leone, and José Lauro Araújo Ramos. "Is glycosuria a reliable indicator of adequacy of glucose infusion rate in preterm infants?" Sao Paulo Medical Journal 117, no. 1 (1999): 19–24. http://dx.doi.org/10.1590/s1516-31801999000100004.

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CONTEXT: Adequacy of glucose infusion may be monitored via the glycosuria levels, as there is a relationship between glycemia and glycosuria regulated by the renal glucose threshold. In the neonatal period, however, this relationship is not so clear. OBJECTIVE: To evaluate the occurrence of glycosuria in preterm infants submitted to glucose infusion and to verify the relationship between glycosuria and blood glucose level. DESIGN: Accuracy study. SETTING: Neonatal intensive care unit of General Maternity Hospital. PATIENTS: 40 preterm newborns receiving glucose infusion. PROCEDURES: 511 concom

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Kaufmann, R. C., F. K. Khosho, and K. S. Amankwah. "Scanning Electron Microscopy of the glomerulus in diabetic BB/S wistar rats." Proceedings, annual meeting, Electron Microscopy Society of America 45 (August 1987): 730–31. http://dx.doi.org/10.1017/s0424820100127967.

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Renal damage secondary to diabetes seems to be related to the severity and duration of the diabetes. In streptozotocin and alloxan-induced diabetic rats, renal disease is found only in those rats that have glycosuria and then only after the glycosuria has been present for many months. In these animals, the longer they have glycosuria, the more severe the renal damage. In our colony of BB/S Wistar rats, animals that are going. to become frankly diabetic demonstrate clinical diabetes before they begin spilling glucose in their urine. After glycosuria develops, the condition of the animals worsen

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Lee, Matthew A., George McMahon, Ville Karhunen, et al. "Common variation at 16p11.2 is associated with glycosuria in pregnancy: findings from a genome-wide association study in European women." Human Molecular Genetics 29, no. 12 (2020): 2098–106. http://dx.doi.org/10.1093/hmg/ddaa054.

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Abstract Glycosuria is a condition where glucose is detected in urine at higher concentrations than normal (i.e. not detectable). Glycosuria at some point during pregnancy has an estimated prevalence of 50% and is associated with adverse outcomes in both mothers and offspring. Little is currently known about the genetic contribution to this trait or the extent to which it overlaps with other seemingly related traits, e.g. diabetes. We performed a genome-wide association study (GWAS) for self-reported glycosuria in pregnant mothers from the Avon Longitudinal Study of Parents and Children (cases

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G., V. "Glycosuria as a sign of pregnancy. Seit-z and Jess (Münch, ni. Woch .. 1922, no. 1)." Kazan medical journal 18, no. 2 (2021): 108. http://dx.doi.org/10.17816/kazmj79920.

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Glycosuria as a sign of pregnancy. Seit-z and Jess (Mnch, ni. Woch .. 1922, No. 1) found that when administered to pregnant women (at II-VIII months of pregnancy) 100 grm. of grape sugar, half of them get glycosuria.

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Dissertations / Theses on the topic "Glycosurie"

1

David, Hélène. "Le diabète rénal et les glycosuries chez l'adulte jeune." Bordeaux 2, 1990. http://www.theses.fr/1990BOR25203.

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Jeanneau, Michèle. "Dépistage du diabète en médecine du travail : recherche systématique de glycosurie sur 52 911 sujets en Aquitaine." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25037.

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Filipe, Ana Isabel Pinto. "Cetoacidose diabética : estudo retrospetivo em cães e gatos." Master's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2013. http://hdl.handle.net/10400.5/5993.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>A Cetoacidose Diabética (CAD) é uma complicação grave da Diabetes Mellitus (DM), caracterizada pela tríade de hiperglicémia, acidose metabólica e hipercetonémia (com cetonúria). Resulta da deficiência insulínica, aliada a um excesso de glucagina e outras hormonas diabetogénicas, muitas vezes como resultado da presença de doenças concomitantes. Na tentativa de apresentar uma proposta de diagnóstico, de monitorização e de um tratamento adequado da CAD, que diminua a taxa de mortalidade desta doença, este estudo teve como objetivos: -

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Rahman, Mahfuzar. "Nonmalignant health effects of arsenic exposure /." Linköping : Univ, 1999. http://www.bibl.liu.se/liupubl/disp/disp99/Med612s.htm.

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Morouço, Paulo Jorge Frade. "Diabetes Mellitus felina : proposta de protocolo de monitorização da glicemia em ambulatório." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2008. http://hdl.handle.net/10400.5/956.

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Dissertação de Mestrado Integrado em Medicina Veterinária.<br>A Diabetes Mellitus (DM) é definida como um grupo de doenças metabólicas caracterizadas por hiperglicemia, que resultam de defeitos na secreção de insulina, acção da insulina, ou ambos. É uma das endocrinopatias mais comuns no gato, e a sua prevalência tem vindo a aumentar ao longo dos tempos. A classificação actual divide a DM em Tipo 1, Tipo 2 e Outros Tipos Específicos. O gato é uma das poucas espécies que desenvolve uma forma de DM que é clínica e histologicamente análoga à DM Tipo 2 Humana, verificando-se essa analogia em

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Huang, Chiung-Yu, and 黃炯毓. "Effect of Glucose Injection on Blood Glucose and Glycosuria for Tilapia（Oreochromis niloticus × O. aureus）." Thesis, 1999. http://ndltd.ncl.edu.tw/handle/35881882636994240956.

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碩士<br>國立海洋大學<br>水產養殖學系<br>87<br>Fishes appear to utilize dietary carbohydrate poorly. It has been shown that carbohydrate is generally the major dietary source of energy in most domestic animals, however, it is considered to be of limited value in the nutrition of fish. Differences in carbohydrate digestion、utilization、absorption and metabolism between fish and mammals the major reasons. Causing dietary differences between these two animal groups. The objective of this experiment was to observe the variation of the blood glucose and glycosuria concentration within 24 hours after each injection

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Books on the topic "Glycosurie"

1

Wheeler, Linda A. Maternal assessment: Blood pressure. 2nd ed. Edited by Raff Beverly S, Albers Lolita, and March of Dimes Birth Defects Foundation. March of Dimes Birth Defects Foundation, 1988.

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Wheeler, Linda A. Maternal assessment: Urine evaluation. 2nd ed. Edited by Raff Beverly S, Albers Lolita, and March of Dimes Birth Defects Foundation. March of Dimes Birth Defects Foundation, 1987.

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3

Bockenhauer, Detlef, and Robert Kleta. Approach to the patient with renal Fanconi syndrome, glycosuria, or aminoaciduria. Edited by Robert Unwin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0041_update_001.

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Up to 80% of filtered salt and water is returned back into the circulation in the proximal tubule. Several solutes, such as phosphate, glucose, low-molecular weight proteins, and amino acids are exclusively reabsorbed in this segment, so their appearance in urine is a sign of proximal tubular dysfunction. An entire orchestra of specialized apical and basolateral transporters, as well as paracellular molecules, mediate this reabsorption. Defects in proximal tubular function can be isolated (e.g. isolated renal glycosuria, aminoacidurias, or hypophosphataemic rickets) or generalized. In the latt

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Westman, Eric C., Emily Maguire, and William S. Yancy. Ketogenic Diets as Highly Effective Treatments for Diabetes Mellitus and Obesity. Edited by Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0037.

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Obesity and type 2 diabetes mellitus (T2DM) have reached epidemic proportions worldwide. While characterized by chronic hyperglycemia, the underlying cause of T2DM is insulin resistance—most often related to an increase in abdominal adiposity caused by obesity. The goal of treatment of T2DM is to put the disease into remission by targeting the underlying insulin resistance. The observation that dietary carbohydrate is the major factor to cause glycosuria and hyperglycemia, has been known since the early days of modern medicine. As a result, low-carbohydrate, ketogenic diets were employed to tr

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Erickson, Stephen B., Hatem Amer, and Timothy S. Larson. Urolithiasis, Kidney Transplantation, and Pregnancy and Kidney Disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0475.

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It was previously assumed that all kidney stones crystallized as urine passed through the renal tubules and were retained by means of crystal-tubular cell interactions. Recently uroscopy with papillary biopsies has shown 2 different pathways for stone formation, both mediated by calcium phosphate crystals. Kidney transplant has become the preferred treatment for patients with end-stage renal disease. Those benefiting from transplant included patients who would be deemed "high risk," such as those with diabetes mellitus and those older than 70 years. Anatomical changes associated with pregnancy

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Walsh, Stephen B. Approach to the patient with renal tubular acidosis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0036.

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The renal tubular acidoses are a collection of syndromes characterized by defective urinary acidification. These syndromes have classically caused some confusion, and many opine that the widely used numerical system (type 1, 2) should be abandoned. We consider distal renal tubular acidosis and proximal renal tubular acidosis separately, and briefly cover hypoaldosteronism. Distal (Type 1) renal tubular acidosis is a syndrome of hypokalaemia, metabolic acidosis, kidney stones, nephrocalcinosis, and osteomalacia or rickets. It is caused by failure of the acid secreting α‎‎‎-intercalated cells in

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Izzedine, Hassan, and Victor Gueutin. Drug-induced chronic tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0087.

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The chronic form of drug-induced tubulointerstitial nephritis (CTIN) is an insidious disease and most probably represents the common final response pattern of the kidney to a variety of agents (including analgesics, lithium, antineoplastic chemotherapeutic agents, like cisplatin and nitrosoureas, and immunosuppressive drugs, such as ciclosporin and tacrolimus). Drug-induced CTIN is usually asymptomatic, presenting with slowly progressive renal impairment. Because of its insidious nature, CTIN is often diagnosed incidentally on routine laboratory screening or evaluation of CKD. The diagnosis of

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Book chapters on the topic "Glycosurie"

1

Zelikovic, Israel. "Aminoaciduria and Glycosuria." In Pediatric Nephrology. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-76341-3_37.

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Zelikovic, Israel. "Aminoaciduria and Glycosuria in Children." In Pediatric Nephrology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-43596-0_33.

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Zelikovic, Israel. "Aminoaciduria and Glycosuria in Children." In Pediatric Nephrology. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27843-3_33-1.

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Serrano Ríos, M. "Renal Diabetes (primary renal glycosuria): A Short Overview." In Verhandlungen der Deutschen Gesellschaft für Innere Medizin. J.F. Bergmann-Verlag, 1987. http://dx.doi.org/10.1007/978-3-642-85460-6_130.

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De Marchi, S., E. Cecchin, F. Zanello, and E. Bartoli. "Abnormal Blood Glucose and Insulin Response during Oral Glucose Tolerance Test in Familial Renal Glycosuria." In Hereditary Kidney Diseases. KARGER, 1997. http://dx.doi.org/10.1159/000059902.

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"Glycosuria." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_7013.

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Dawlatly, Bashier, and Rina Davison. "Glycosuria of Pregnancy." In Differential Diagnosis in Obstetrics and Gynaecology: An A-Z. CRC Press, 2015. http://dx.doi.org/10.1201/b18646-35.

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Agarwal, Amit. "Pheochromocytoma Presenting with Glycosuria." In Endocrine Surgery Made Easy. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10258_55.

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9

Gupta, MC. "Chapter-03 Renal Glycosuria in Pregnancy." In Step by Step Diabetes in Pregnancy. Jaypee Brothers Medical Publishers (P) Ltd., 2006. http://dx.doi.org/10.5005/jp/books/10807_3.

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10

Knoers, Nine V. A. M., and Elena N. Levtchenko. "Disorders of tubular electrolyte handling." In Oxford Textbook of Medicine. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.2116_update_001.

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Physiology—glucose reabsorption in the proximal tubule is carried out by two different pairs of apical Na<sup>+</sup>-dependent (SGLT1 and 2) and basolateral Na<sup>+</sup>-independent (GLUT1 and 2) glucose transporters. Clinical disorders—abnormalities in renal glucose transport can be seen in association with other defects of proximal tubular transport (Fanconi syndrome, see below). Familial renal glycosuria is a rare autosomal recessive condition caused by mutations in the SGLT2-encoding gene, ...

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