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Dissertations / Theses on the topic 'GM-CSF'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Bailie, Karen Elizabeth Margaret. "G-CSF and GM-CSF : effects on neonatal neutrophils." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.482043.

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2

Chevalier, Anne Sophie. "Utilisation thérapeutique du G-CSF et du GM-CSF en hématologie." Paris 5, 1993. http://www.theses.fr/1993PA05P248.

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3

Hallsworth, Matthew Pearce. "GM-CSF and eosinophil survival in asthma." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341883.

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4

Bernstone, Laura. "Characterisation of HIV-1 infection and M-CSF and GM-CSF macrophages." Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572833.

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Macrophages are a natural target cell for HIV-1 infection, and they contribute to the development of disease as they are important for transmission, dissemination and persistence of the virus in an infected patient. Macrophages are less well-studied than T cells and cell lines in relation to HIV-1 infection, yet macrophages are highly specialised and key aspects of the HIV-1 life cycle in these cells are already known to differ compared to other cell types. HIV-1 entry into macrophages has been suggested to occur by macropinocytosis, however the entry route in these cells has not been fully ch

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5

Lin, Tony Wei Ting. "The role of GM-CSF/G-CSF & BKLF in the development of atherosclerosis." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/10407.

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The studies described in this thesis are aimed to investigate the role of the pro-inflammatory cytokine GM-CSF/G-CSF, transcriptional factor KLF3 and psychological stress in KLF3 deficiency mice on neointimal formation, a common feature of atherosclerosis with the aid of perivascular collar model on murine model. The present study supports an important role for GM/CSF-G/CSF and KLF in atherosclerosis. There is now overwhelming evidence that the monocyte/macrophage has a crucial role in the initiation and progression of atherosclerotic plaque and this has led to the view that the recruitment an

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6

Mirabella, Fabio. "Regulation of the human IL-3/GM-CSF locus." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487744.

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The human Interleukin-3 (IL-3) and Granulocyte-Macrophage Colony-Stimulating-Factor (GM-CSF) genes are two closely linked cytokine genes that are located within a conserved cytokine gene cluster. They are expressed in an inducible tissue-specific manner in a variety of haemopoietic cell types, and they contribute to the regulation of inflammatory responses arid haemopoiesis. For this reason their expression is strictly regulated. The experiments in this thesis describe an investigation into the factors and DNA elements involved in the control and regulation of these genes during T cell develop

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7

Grant, Olivia M. "GM-CSF Stress-Induced Priming of the Dendritic Cell." Ohio Dominican University Honors Theses / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=oduhonors1449522036.

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8

Adkins, Karissa Kathleen 1971. "Glucocorticoid regulation of GM-CSF in bronchial epithelial cells." Diss., The University of Arizona, 1998. http://hdl.handle.net/10150/282844.

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Inflammation plays a central role in the pathogenesis of asthma. Glucocorticoids are first line antiinflammatory therapy in the treatment of asthma and are effective inhibitors of inflammatory cytokines. Clinical data demonstrate that granulocyte-macrophage colony-stimulating factor (GM-CSF) production by airway epithelial cells may be an important target of inhaled glucocorticoid therapy. In this study, the regulatory mechanisms of GM-CSF expression by interleukin-1β (IL-1β) and the synthetic glucocorticoid dexamethasone (DEX) were examined in the BEAS-2B human bronchial epithelial cell line.

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9

Aziz, Khalil Abdul. "Influence of GM-CSF and G-CSF on the mutual interactions between platelets and neutrophils." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241473.

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10

Osborne, Cameron Stuart. "Transcriptional regulation of the GM-CSF gene in T lymphocytes /." Title page, contents and summary only, 1996. http://web4.library.adelaide.edu.au/theses/09PH/09pho81.pdf.

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11

Yang, Nianji Eric. "THE ROLE OF GM-CSF IN MACROPHAGE POLARISATION IN RESPONSE TO TROPOELASTIN-COATED POLYETHYLENE IMPLANTS IN VIVO." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15866.

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Our previous studies have shown that the incorporation of tropoelastin (TE) improves the biocompatibility of silk fibroin and low density polyethylene (PE). With the coating or blending of tropoelastin, both biomaterials underwent a favourable foreign body inflammatory response. Macrophage is a key cell in the biomaterial-induced inflammation and can be polarized into two main phenotypes (M1/M2) capable of inducing either pro-inflammatory or anti-inflammatory cytokine profiles during host responses. The objective of the present study was to investigate the possible underling mechanism of the i

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12

Eubank, Tim. "M-CSF and GM-CSF induce human monocytes to express either pro- or anti-angiogenic factors." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1069772001.

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13

Eubank, Timothy D. "M-CSF and GM-CSF induce human monocytes to express either pro- or anti-angiogenic factors." Columbus, Ohio : Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1069772001.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Title from first page of PDF file. Document formatted into pages; contains xx, 168 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: Clay B. Marsh, Biochemistry Program. Includes bibliographical references (p. 150-168).

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14

Pinder, Emma Muriel. "Does GM-CSF restore effective neutrophil function in critically ill patients?" Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3943.

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Nosocomial infection is an increasing problem worldwide associated with significant morbidity and mortality in addition to heightened healthcare costs. The problem is even greater on the intensive care unit (ICU) where up to 20 of patients develop a secondary infection during their admission. In an era of increasing antibiotic resistance alternative strategies to prevent nosocomial infection must be sought. The intensive care population are recognised to be at high risk of developing immune dysfunction during their critical illness and this has been shown to be associated with an increased ris

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15

CARDONE, MARCO. "Il GM-CSF può promuovere il differenziamento di monociti umani in cellule dendritiche dotate di distinte proprietà immuno-stimolatorie e migratorie: ruolo dell'espressione del recettore del GM-CSF." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2008. http://hdl.handle.net/2108/546.

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Le cellule dendritiche mieloidi (DCs) e i macrofagi evolvono da un precursore comune. Comunque, è ancora poco chiaro quale siano i fattori che controllano il differenziamento dei monociti in DC o macrofagi. Noi abbiamo riportato che la densità di superficie della subunità α del recettore del GM-CSF (GM-CSFR) in monociti del sangue periferico umano varia tra i diversi donatori. Sebbene poi nessuna correlazione sia stata trovata tra l’entità di espressione del GM-CSFR e la capacità di monociti di differenziare in DC in presenza di GM-CSF e IL-4, l’espressione del GM-CSFR è stata vista però infl

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16

Mikkelsen, Ipsen Johanna. "Kan onkolytiskt adenovirus armerat med GM-CSF fungera som behandling mot cancer?" Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-43026.

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Cancer är idag en vanlig sjukdom som uppstår på grund av okontrollerad celltillväxt och kan visa sig som cirka 200 olika sjukdomar beroende på vilken celltyp som drabbas. Trots dagens standardterapier med kirurgi, strålning och kemoterapi så dör årligen runt 23 000 människor av cancer i Sverige. Dödsfallen beror ofta på svåråtkomliga metastaser som bildats från ursprungstumören och sedan spridits med blod och lymfa ut i kroppen. En långt gången cancer kan även visa upp resistens mot vissa terapier, vilket försvårar behandling ytterligare. Det är därför viktigt att nya effektiva läkemedel och

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17

Ladds, Simon John. "The upregulation of neutrophil protein biosynthesis in response to GM-CSF stimulation." Thesis, University of Liverpool, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366718.

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18

Razanajaona, Diane. "Régulation de l'expression génique du GM-CSF dans les cellules hématopoiétiques humaines." Aix-Marseille 2, 1992. http://www.theses.fr/1992AIX22022.

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Le gm-csf est une glycoproteine de la famille des csf (colony-stimulating factor), jouant un role crucial dans la croissance et la differenciation hematopoietique. Son messager appartient au groupe des messagers instables, et son expression est inductible dans les cellules hematopoietiques normales, mais constitutive dans certaines cellules tumorales, telles que les cellules de leucemies aigues myeloides (lam). L'etude de la relation entre ce facteur et les lam montre que son expression est constitutive dans ces cellules, que ce facteur est implique dans leur proliferation autocrine in vitro,

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19

Rothchild, Alissa Chen. "Antimicrobial Roles for iNKT Cells and GM-CSF in Mycobacterium Tuberculosis Infection." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11371.

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Despite effective antibiotics, Mycobacterium tuberculosis (Mtb), the causative agent of tuberculosis, still infects nearly one-third of the world's population. While key immune factors including CD4+ T cells and IFNg production have been identified, there are still many antimicrobial mechanisms yet to be explored. Here we characterized the role of invariant natural killer T (iNKT) cells and GM-CSF during Mtb infection.

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20

Goyal, Girija. "Novel Role of PPAR-Gamma in GM-CSF Induced Anti-Tumor Immunity." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:14226102.

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Granulocyte macrophage colony stimulating factor (GM-CSF) mediates context dependent anti- or pro-inflammatory functions through cells of the myeloid lineage. GM-CSF signaling induces the expression of the transcription factor peroxisome proliferator-activated receptor gamma (PPAR-γ). We examined the role PPAR-γ in myeloid cells in the anti-tumor response to GVAX, a GM-CSF based cancer immunotherapy using the B16 model of murine melanoma. We found that selective loss of PPAR-γ in the myeloid lineage using LysM-Cre reduces the efficacy of GVAX which could not be explained by known mechanisms.

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21

Kühnle, Simone. "The reconstitution of immunocompetence by GM-CSF or IFN after pharmacological suppression." [S.l. : s.n.], 2001. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB9073784.

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22

Sylvain-Prévost, Stéphanie. "Implication de TAK1 dans la modulation des réponses du neutrophile humain au fMLP et au GM-CSF." Mémoire, Université de Sherbrooke, 2012. http://hdl.handle.net/11143/6362.

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Les neutrophiles sont d'une grande importance dans la première ligne de défense de l'organisme contre les pathogènes. Ils participent activement par leurs actions antimicrobiennes, comme la phagocytose et la relâche de granules, mais influencent également la réponse immunitaire par les différentes cytokines et chimiokines qu'ils produisent. L'étude des différentes fonctions du neutrophile a permis d'établir les étapes clés de la signalisation intracellulaire qui mène à ces différentes fonctions. De plus, les études dé signalisation, dans différents organismes, ont placé TAK1, une MAP3K, à l'av

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23

Jasper, Melinda Jane. "Paracrine regulation of ovarian function by granulocyte-macrophage colony-stimulating factor (GM-CSF) & colony-stimulating factor-1 (CSF-1) /." Title page and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phj39.pdf.

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24

Xu, Jian. "Selective reconstitution by GM-CSF of the immune response in human immunosuppressed cells." Konstanz, 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=965669238.

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25

Hellebrand, Eva. "Entwicklung von GM-CSF-produzierenden EBV-Vektoren für die Immuntherapie der B-CLL." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-12575.

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26

Cowling, Randy T. "Effect of GM-CSF on RNA and protein in human peripheral blood granulocytes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ36768.pdf.

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27

Arcanjo, Katia Denise de Souza. "Organização molecular do espaço intercelular na sinalização do GM-CSF em sistemas hematopoeticos." [s.n.], 2002. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317887.

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Orientador : Radovan Borojevic<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-01T08:59:37Z (GMT). No. of bitstreams: 1 Arcanjo_KatiaDenisedeSouza_D.pdf: 8227428 bytes, checksum: ce60d7dcab3f2fe523a6be9c914002b6 (MD5) Previous issue date: 2002<br>Resumo: O microambiente da medula óssea desempenha importância fundamental na proliferação e diferenciação das células progenitoras hematopoéticas bem como no controle do egresso dessas células para o sangue periférico. Embora moléculas da matriz extracelular, juntamente com citoc

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28

Stomski, Frank Charles. "The molecular basis of IL-3, Il-5 and GM-CSF receptor activation /." Title page, contents and abstract only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09phs8766.pdf.

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29

Pfeiffer, Hella [Verfasser], and Walburga [Akademischer Betreuer] Dieterich. "Bestimmung der Oberflächenmarker und des Aktivierungsgrades von CD14+ Zellen aus humanem Blut nach Stimulation und Reifung mit IL-4/GM-CSF oder IL-15/GM-CSF / Hella Pfeiffer. Gutachter: Walburga Dieterich." Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2015. http://d-nb.info/1075838045/34.

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30

Baker, David Alan. "Mutational analysis of the proto-oncogenes c-fms and c-kit." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362390.

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31

Curé, Hervé. "Intensification de la chimiotherapie neo-adjuvante du cancer du sein : impact sur la reponse et la survie et etude des cellules souches hematopoietiques peripheriques (doctorat : hemato-cancerologie)." Clermont-Ferrand 1, 1999. http://www.theses.fr/1999CLF1MM15.

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32

DE, GENTILE ALBANE. "Etude de l'expression et de la modulation par l'acide retinoique des recepteurs du gm-csf et de cytokines dans les leucemies aigues promyelocytaires. Production d'anticorps diriges contre le recepteur du gm-csf." Paris 6, 1994. http://www.theses.fr/1994PA066580.

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Ce travail comporte deux parties. Dans un premier temps, nous presentons les resultats de l'etude in vitro: 1) de l'expression des recepteurs du gm-csf (r-gm-csf) par les cellules leucemiques de patients atteints de leucemie aigue promyelocytaire. (lam 3) ; 2) de la production de cytokines par ces memes cellules leucemiques ; 3) de la modulation de cette production ainsi que de l'expression des recepteurs au cours de la differenciation granulocytaire induite par l'acide retinoique tout trans (atra). Nos travaux montrent que les cellules de lam 3 expriment les recepteurs du gm-csf et que cette

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33

Szymanski, Silvia. "Evaluation of different approaches to enhancing arteriogenesis using isolated monocytes and GM-CSF treatment in an ischemic mouse hind limb model." Giessen : VVB Laufersweiler, 2006. http://geb.uni-giessen.de/geb/volltexte/2007/4335/index.html.

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34

Feil, Bertram. "Genexpression des Adaptorproteins Shc bei Patienten mit Juveniler Myelomonozytärer Leukämie (JMML) Charakterisierung neuer Spleißformen der SH2-Domäne von Shc /." [S.l. : s.n.], 2000. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB8937685.

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35

Houzet, Laurent. "Etude du rôle des séquences AU riches de l'ARNm du GM-CSF in vivo." Doctoral thesis, Universite Libre de Bruxelles, 2001. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211667.

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36

LICARI, VERONIQUE. "Maladie de kaposi localisee chez un sujet hiv negatif : action antitumorale du gm-csf." Aix-Marseille 2, 1994. http://www.theses.fr/1994AIX20131.

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37

Kühnle, Simone. "The reconstitution of immunocompetence by GM-CSF or (IFN-g) [(IFN-gamma)] after pharmacological suppression." [S.l.] : [s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=961227540.

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38

Harris, Robert John. "Studies of the production and function of GM-CSF in LTBMC and mature B cells." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266492.

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39

Fernandez, Marilyn Cecilia. "Mechanisms regulating GM-CSF and TNF-alpha induced IL-1(beta) gene expression in PMN /." The Ohio State University, 1997. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487942739808581.

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40

Deane, David Leslie. "The characterisation of a GM-CSF and IL-2 inhibitory protein encoded by Orf virus." Thesis, University of Edinburgh, 2001. http://hdl.handle.net/1842/23323.

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In this study, the gene encoding the GM-CSF inhibitory factor (GIF) was isolated and mapped to the right terminal quarter of the orf virus genome. The orf virus GIF cDNA was expressed as a secreted protein in Chinese hamster ovarian cells as detected by GM-CSF inhibition ELISA. Recombinant GIF was purified by ovine GM-CSF affinity chromatography and gel filtration. Sequence analysis of the 20 N-terminal amino acids was performed on the purified GIF. The GIF gene encodes a 28 kDa protein that exhibits 32% amino acid sequence similarity to the predicted sequence of the A41L gene product encoded

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41

Paolini, Léa. "Impact de l’acide lactique sur le phénotype et le métabolisme des macrophages humains." Thesis, Angers, 2018. http://www.theses.fr/2018ANGE0036.

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Les macrophages associés aux tumeurs (TAM) orchestrent l'inflammation nécessaire à la croissance tumorale (propriétés de type M1) et favorisent les métastases et l'angiogenèse (caractéristiques de type M2). Cependant, la nature des facteurs capables de conférer des propriétés M1 et M2 aux macrophages humains demeure inconnue. L'acide lactique (AL) est un métabolite produit par les cellules tumorales connu pour moduler les fonctions des cellules présentes dans le microenvironnement tumoral. Dans cette étude, nous avons analysé son impact sur la différenciation des monocytes humains. Les résulta

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42

Robertson, Sarah A. "Granulocyte-macrophage colony stimulating factor (GM-CSF) : a paracrine regulator in the pre-implantation mouse uterus." Title page, abstract and contents only, 1993. http://web4.library.adelaide.edu.au/theses/09PH/09phr6515.pdf.

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43

Rajotte, Daniel. "Étude sur la signalisation intracellulaire et la relation structure fonction du récepteur pour le GM-CSF." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1996. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21506.pdf.

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44

Kidger, Simone Verina. "The impact of the synovial environment and GM-CSF on the myeloid compartment in rheumatoid arthritis." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8012/.

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The synovial environment in rheumatoid arthritis (RA) is a milieu of Damage Associated Molecular Patterns (DAMPs), cytokines and immune complexes, which can modulate the activation or polarisation of myeloid cells. GM-CSF, which is a pivotal myeloid cell growth factor, is also a pro-inflammatory cytokine that drives aspects of RA immunopathogenesis. Inhibition of GM-CSF signalling has been successful in both mouse models and in clinical trials for RA, however, the specific effect of GM-CSF on myeloid cells in a synovial setting is not well understood. The aim of this thesis was to investigate

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45

Nataf, Éric. "Etude cytogenetique du myelome multiple : a propos de 49 patients ; interet de l'utilisation gm. - csf - il6 - il3." Lille 2, 1992. http://www.theses.fr/1992LIL2M180.

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46

OLAGNIER, VALERIE. "Essai multicentrique en double aveugle ou cho-cell-gm-csf dans les neuroblastomes de haut grade traites par double autogreffe medullaire." Lyon 1, 1989. http://www.theses.fr/1989LYO1M323.

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47

Weininger, Eva-Maria [Verfasser], Juan Manuel [Akademischer Betreuer] Maler, Jun Manuel [Gutachter] Maler, and Philipp [Gutachter] Spitzer. "GM-CSF und M-CSF in Serum und Liquor bei der Alzheimer-Erkrankung / Eva-Maria Weininger ; Gutachter: Jun Manuel Maler, Philipp Spitzer ; Betreuer: Juan Manuel Maler." Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2020. http://d-nb.info/1203377649/34.

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48

Hüttinger, Cornelia. "Nonviraler Gentransfer des felinen Zytokin-Gens GM-CSF mittels Magnetofektion als neoadjuvante Immuntherapie beim Fibrosarkom der Katze." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-89747.

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49

Walsch, Florian Markus. "Nonviraler Gentransfer des felinen Zytokin-Gens GM-CSF mittels Magnetofektion als neoadjuvante Immuntherapie beim Fibrosarkom der Katze." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-147715.

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50

Buatois, Vanessa. "CX3CR1, et le GM-CSF, sont requis dans le développement de l'asthme allergique dans un modèle murin." Nice, 2005. http://www.theses.fr/2005NICE4035.

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