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Relevant bibliographies by topics / Goiter, Endemic

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Academic literature on the topic 'Goiter, Endemic'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 March 2023

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Journal articles on the topic "Goiter, Endemic"

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Koutras, Demetrios. "Endemic Goiter - an update." HORMONES 1, no. 3 (2002): 157–64. http://dx.doi.org/10.14310/horm.2002.1163.

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Scrimshaw, N. S. "ENDEMIC GOITER." Nutrition Reviews 15, no. 6 (2009): 161–64. http://dx.doi.org/10.1111/j.1753-4887.1957.tb00524.x.

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Benitez, Rodrigo, Leslie Degroot, Mario Paredes, and Wilson Pañafiel. "Yodo, Bocio y Cretinismo endémicos en la región Andina del Ecuador." Revista Ecuatoriana de Medicina y Ciencias Biológicas 5, no. 1 (2017): 15–30. http://dx.doi.org/10.26807/remcb.v5i1.106.

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An inventory of the total population of isolated rural villages of the most affected Ecuadorian Andean provinces by endemic goiter was made. In each of these villages iodine content in urine, salt and water was determined. It was conclusive that the chronic iodine deficiency is sine qua non condition for the endemic; but the severity of the endemic; but the severity of the endemic doesn’t have relationship with the magnitude of the lack, rather due to the intercourse of socio-economic and biological intercurrent deficitary factors. Even with the iodine deficiency and the intercourse of the communities located more than 3,500 m, adove sea level, did not present goiter with endemic characteristics. The severity of the endemic was determined to be correlated with: incidence of nodular goiters, in the general population in the first two decades of life, presence of big goiters, incidence of goiters mean in the relation to that in woman. All of wich is epidemiologically related to the incidence of defects that would begin appearing in endemic form in a community, and in accord to the severity would follow the following order of appearing in the endemic form in a community, and in accord to the severity would follow the following order od appearance: motor abnormalities, mental deficiency, deafmutism, deafness and mutism. Finally, a familiar tendency was clearly demonstrated so that a cretin child would be born, and no relationship, on familial level, between incidence and type of goiter and the appearance.

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Cuevas-Ramos, Daniel, and Bernardo Pérez-Enríquez. "Endemic Multinodular Goiter." New England Journal of Medicine 356, no. 26 (2007): e27. http://dx.doi.org/10.1056/nejmicm065299.

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Naramore, Sarah E. "Making Endemic Goiter an American Disease, 1800-1820." Journal of the History of Medicine and Allied Sciences 76, no. 3 (2021): 239–63. http://dx.doi.org/10.1093/jhmas/jrab018.

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Abstract In 1800, American physician and naturalist Benjamin Smith Barton (1766-1815) published A Memoir Concerning the Disease of Goitre as it Prevails in Different Parts of North-America. The text documented the nature of the disease in the United States and highlighted how it differed from the ailment’s presentation in European patients. While medical topographies were common during this period, Barton’s goiter research and the steady stream of American goiter research that followed are worth special attention. This body of literature demonstrates how American physicians understood their relationship to transnational medical discussions and the unique perspective they brought to them. Goiter literature was common in European medical and travel writing during this period and intensely focused on the appearance of the disease in the mountains of Switzerland and Northern Italy. American goiter by its very appearance in non-mountainous regions of the United States contradicted nearly all of the received wisdom about the ailment’s cause and potential cure. For two decades, American writers leveraged their own observations and local knowledge to challenge larger narratives in their field.

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Gaitan, Eduardo, Norman C. Nelson, and Galen V. Poole. "Endemic goiter and endemic thyroid disorders." World Journal of Surgery 15, no. 2 (1991): 205–15. http://dx.doi.org/10.1007/bf01659054.

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Sauiin, Clark T. "Goiter and Endemic Cretinism." Endocrinologist 9, no. 3 (1999): 157–58. http://dx.doi.org/10.1097/00019616-199905000-00001.

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CORVILAIN, B., J. VAN SANDE, J. E. DUMONT, P. BOURDOUX, and A. M. ERMANS. "Autonomy in Endemic Goiter." Thyroid 8, no. 1 (1998): 107–13. http://dx.doi.org/10.1089/thy.1998.8.107.

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Fernando, Ranil, Pramodh Chandrasinghe, Sumal Nandasena, and Arunasalam Pathmeswaran. "Epidemiology of Goiters in Sri Lanka with Geographic Information System Mapping: Population-based Cross-sectional Study." World Journal of Endocrine Surgery 7, no. 3 (2015): 55–59. http://dx.doi.org/10.5005/jp-journals-10002-1170.

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ABSTRACT Background Sri Lanka is considered an endemic region for goiter. Early researchers have suggested the existence of a goiter belt based on rainfall pattern. Subsequent studies done in school children have challenged this theory. Current study is a community-based cross-sectional study done to assess the epidemiology of goiters. Geographic information system (GIS) mapping of the goiter prevalence is developed for the first time in Sri Lanka. Methods Study subjects were selected using a multi-staged cluster sampling with probability proportionate to size (PPS) method. Examination for the presence of goiters was done by two trained investigators and graded. ArcGIS 10 software was used for geostatistical analysis and developing a map by interpolating the data collected for the first time in Sri Lanka. Results A total of 5200 individuals (female—66%, median age—38 years; range 10–92 years) were assessed and 426 goiters were detected. The adjusted prevalence rate of goiters was 6.8%. Kriging interpolation method was used to develop the most appropriate epidemiological map clustering of high prevalence areas with scattered pockets of high prevalence was observed. Current map does not show a prevalence pattern in relation to the rainfall or elevation above sea level as proposed in the past. Conclusion Goiter prevalence in Sri Lanka appears to be different from earlier proposed patterns. Use of GIS has contributed to develop a comprehensive epidemiological map of goiters demonstrating the absence of an endemic goiter belt in the wet zone as proposed earlier. How to cite this article Chandrasinghe P, Fernando R, Nandasena S, Pathmeswaran A. Epidemiology of Goiters in Sri Lanka with Geographic Information System Mapping: Population-based Cross-sectional study. World J Endoc Surg 2015;7(3):55-59.

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Volodchenko, N. P., N. V. Valova, V. V. Ishchenko, S. V. Kravtsova, E. A. Shevcheko, and T. K. Ishchenko. "RESULTS OF SURGICAL TREATMENT OF NODULAR GOITER IN THE REGION OF ENDEMIC GOITER." Amur Medical Journal, no. 3 (2017): 79–80. http://dx.doi.org/10.22448/amj.2017.3.79-80.

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Dissertations / Theses on the topic "Goiter, Endemic"

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Sheremet, M. I. "Prognosis of remote results of surgical treatment of nodular endemic goiter with autoimmune thyroiditis." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18147.

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Duarte, Glaucia Cruzes. "Avaliação ultra-sonográfica da tiróide, excreção urinária de iodo em escolares de 6 a 14 anos e grau de iodação de sal, em diferentes regiões do estado de São Paulo." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-19072007-121746/.

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A nutrição de iodo de uma população é avaliada por indicadores: a iodação do sal, excreção urinária de iodo e volume tiroidiano. A iodação do sal é eficiente no combate às doenças decorrentes da deficiência de iodo, sendo empregada em todo território nacional desde 1995 e o estado de São Paulo é considerado uma área iodo-suficiente. Para avaliar esta ingesta de iodo, foram selecionados aleatoriamente 964 escolares entre 6 a 14 anos, de seis regiões do Estado. Foram avaliados sob o ponto de vista ecográfico da tiróide, 484 meninas e 480 meninos. Os volumes da glândula tiróide elevaram-se progressivamente com a idade, guardando correlação positiva e significativa com a superfície corporal. Cerca de 2,9% dos escolares apresentava bócio. Outras anormalidades encontradas foram hemiagenesia (0,5%), nódulos (0,2%), cistos (0,7%) e hipoecogenicidade (11,7%). Notamos excessiva excreção urinária de iodo nesta população, com valores superiores a 300ug Iodo/L em 76,8% das amostras. As amostras de sal doméstico apresentavam valores entre 34,0 e 68,3mg Iodo/Kg de sal. Concluímos que a população de escolares estudados no estado de São Paulo apresenta excessiva ingestão diária de iodo, a qual, extrapolada para população em geral poderá induzir várias alterações da função tiroidiana, como hipertiroidismo subclínico (em idosos) e tiroidite crônica auto-imune na população adulta.<br>The salt iodization is efficient to prevent iodine deficient disorders and began in all of Brazil in 1995 and the state of São Paulo is considered an iodine sufficient area. To evaluate the iodine nutrition, 964 schoolchildren, aged between 6 and 14 years old (484 girls, 480 boys) were examined by ultrasonographic studies. There was a progressive increase of the thyroid volume with aging with a positive and significant correlation with the body surface area. The presence of enlarged thyroid gland was rarely seen, being present in only 2.9% of the studied cohort. A few thyroid gland abnormalities were noticed such as hemiagenesia (0.5%), nodules (0.2%), cysts (0.7%) and hypoechogenicity (11,7%). It was clearly demonstrated that the urinary excretion of iodine was elevated being above 300ug Iodine/L in 76,8% of the schoolchildren examined. Assays for the iodine concentration in domestic salt samples revealed values between 34.0 and 68.3 mgI/kg of salt. We conclude that the schoolchildren population of the State of São Paulo may be under an excessive daily ingestion of iodine. This may induce, if extrapolated to the general population, subclinical hyperthyroidism in the elderly and possibly an increment in the prevalence of cronic autoimmune thyroiditis.

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Vanderpas, Jean. "L'hypothyroïdie juvénile endémique en Ubangi, Zaïre." Doctoral thesis, Universite Libre de Bruxelles, 1994. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/213084.

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<p align="justify"><i>Note des Bibliothèques :la thèse du Dr Vanderpas a été défendue en 1991 mais il n'est techniquement pas possible d'indiquer cette date dans le logiciel Bictel/e.</i></p><p><p><p align="justify"><u>Première partie</u> :Fonction thyroïdienne de la naissance à 7 ans chez les enfants d’un essai clinique de supplémentation d’huile iodée versus placebo à la femme enceinte.</p><p><p><p align="justify">L’endémie goitreuse du Nord-Congo (République démocratique, ex-Zaïre) a fait l’objet d’un programme de santé publique de prévention du goitre et du crétinisme dans le cadre du Centre d’Etudes Médicales de l’Université Libre de Bruxelles pour les actions de coopération de 1974 à 1995. Le partenaire congolais était l’Institut de Recherche Scientifique et le Bureau National des Troubles dus à la Carence Iodée.</p><p><p><p align="justify">Le présente travail s’inscrit dans ce contexte et analyse plus particulièrement la fonction thyroïdienne chez l’enfant de zéro à sept ans, dans la continuité d’un suivi d’un essai clinique pharmacologique randomisé et contrôlé (RCT, Randomised Clinical Trial) de phase 2 consistant à administrer une huile iodée (Lipiodol®) à des femmes enceintes se présentant à la maternité de Karawa. Cette cohorte de femmes enceintes a été précédemment étudiée par le Professeur Claude-Hector Thilly*.</p><p><p><p align="justify">Chez les enfants nés de mères non supplémentées en iode, l’histoire fonction thyroïdienne se caractérise comme suit :<p><li>Une fonction thyroïdienne relativement stable au ours de la première année de vie par rapport aux valeurs de TSH et de T4 sériques du sang de cordon ;les moyennes de ces marqueurs biologiques sont clairement indicateurs d’un niveau de carence iodée par rapport aux normes d’une population d’enfants belges d’âge comparable (T4 sérique abaissée et TSH sérique élevée) ;</li><p><li>Une aggravation des altérations de la TSH et de la T4 sériques au cours de la deuxième année de vie, aggravation qui se poursuit jusqu’à la quatrième année ;</li><p><li>Un maintien de marqueurs biologiques de TSH et T4 sérique fortement altérés au moins jusqu’à l’âge de 7 ans (étendue d’âge étudiée).</li></p><p><p><p align="justify">Dans cette région, le manioc est connu pour son rôle goitrogène, au travers de son contenu en glucosides cyanogènes, et il avait été précédemment démontré que le thiocyanate élevé des mères passait librement la barrière placentaire. Au cours de la première année de vie, lorsque les nourrissons sont essentiellement alimentés au sein, le thiocyanate sérique diminue fortement et se rapproche de valeurs observées chez des enfants d’autres régions non exposés au manioc. La dégradation de la fonction thyroïdienne au cours de la deuxième année de vie coïncide avec l’introduction du manioc dans l’alimentation. Pour une valeur de concentration urinaire en iode stable au cours des 7 premières années de vie, la prévalence de goitre et les variations de T4 et TSH sériques suivent celles du thiocyanate sérique. Cela est confirmé au travers d’une analyse multi-variée qui met en évidence l’association entre les valeurs moyennes de TSH et T4 et les concentrations urinaires en iode et en thiocyanate.</p> <p><p><p align="justify">L’administration intra-musculaire d’huile iodée prévient les altérations de la fonction thyroïdienne chez la mère (Thilly 1978), et cette protection s’étend chez l’enfant jusqu’à 24 mois, c’est-à-dire jusqu’à ce que l’allaitement maternel reste le principal apport nutritionnel. Au-delà de 24 mois, des altérations de la fonction thyroïdienne apparaissent chez certains de ces enfants (Elévation de la TSH et abaissement de la T4), et au-delà de 4 ans, la fréquence des altérations de la fonction thyroïdienne est aussi fréquente chez les enfants de mères traitées que chez les enfants de mères non traitées.</p><p><p><p align="justify">Au vu de la fréquence fort élevée d’altérations de la fonction thyroïdienne entre 4 et 7 ans (2/3 ont une TSH anormalement élevée > 10 mU/L), seuls certains enfants présentent les stigmates d’une hypothyroïdie prolongée depuis le début de l’existence. Il apparaît qu’il y a lieu de distinguer des hypothyroïdies juvéniles de durée, de sévérité, et de timing différents. Si l’hypothyroïdie juvénile est aussi fréquente au-delà de 4 ans dans les deux groupes de l’étude, les stigmates cliniques d’hypothyroïdie persistante sont plus fréquemment observés chez les enfants nés de mères non supplémentées en iode que chez les autres. De plus, la sévérité des stigmates cliniques (degré d’arriération mentale ;importance du retard de développement statural) démontre que l’hypothyroïdie persistante s’est installée plus précocement chez ertains enfants nés de mères non supplémentées en iode que chez les autres. Dans les formes les plus sévères, l’évolution staturale et le niveau d’intelligence de ces enfants avec hypothyroïdie persistante sont compatibles avec le tableau clinique de crétinisme myxédémateux endémique décrits chez le sujet adulte par les Professeurs François Delange et Jacques Dumont.</p><p><p><p><p align="justify"><u>Deuxième partie</u>: étude du métabolisme iodé chez les enfants hypothyroïdiens et mise en évidence de la carence combinée en iode et en sélénium.</p> <p><p><p align="justify">Certains enfants hypothyroïdiens le sont depuis longtemps (depuis la naissance, éventuellement), d’autres le sont transitoirement, sans que leur hypothyroïdie passagère ne laisse de séquelles évidentes en termes de retard statural ou d’arriération mentale.</p><p><p><p align="justify">Ceux qui sont en hypothyroïdie persistante au-delà de 4 ans ont une fonction thyroïdienne altérée :lorsqu’on leur administre de l’iode, leur glande ne répond pas à cette correction de carence iodée, et ils demeurent profondément hypothyroïdiens. Ce phénomène de non réponse à la correction de la carence iodée n’estpas observé chez les enfants hypothyroïdiens plus jeunes :cela démontre qu’il y a, chez certains enfants, une perte progressive de la capacité fonctionnelle de la thyroïde à répondre à la supplémentation iodée. Ces sujets développent le tableau clinique de crétin myxédémateux endémique.</p><p><p><p align="justify">On constate que l’hypothyroïdie juvénile recouvre un vaste spectre depuis les cas d’hypothyroïdie transitoire jusqu’aux cas d’hypothyroïdie irréversible, même après correction de la carence iodée.</p><p><p><p align="justify">Sur base d’hypothèse physiopathologique de cette perte de capacité fonctionnelle de la thyroïde chez certains jeunes enfants, il a été proposé qu’une carence combinée en iode et en sélénium pourrait expliquer ce processus. Une telle carence combinée a été décrite dans notre travail dans la région goitreuse du Nord-Congo, et pas dans d’autres régions non goitreuses du même pays ou dans d’autres endémies goitreuses avec peu de crétinisme myxédémateux endémique (Soudan, Sénégal).</p> <p><p><p align="justify"><FONT size=1>*Thilly Claude-Hector, Delange François, Lagasse Raphael, Bourdoux Pierre, Ramioul L, Berquist Helen, Ermans André-Marie. Fetal hypothyroidism and maternal thyroid status in severe endemic goiter. Journal of Clinical Endocrinology and Metabolism.</FONT></P><p><p><br>Agrégation de l'enseignement supérieur, Orientation médecine<br>info:eu-repo/semantics/nonPublished

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Le, Couteur Claire Elizabeth. "Dentist, Doctor, Dean : Professor Sir Charles Hercus and his record of fostering research at the Otago Medical School, 1921-1958." Thesis, University of Canterbury. History, 2014. http://hdl.handle.net/10092/9538.

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This thesis investigates the development of medical research at the Otago Medical School in Dunedin, New Zealand under Sir Charles Hercus, Dean from 1937-1958. It also explores his interest and participation in research from his student days and the years before becoming Dean, as well as the influence of the First World War on his career. The study draws upon unpublished material in New Zealand archives and a collection of student projects investigating public health issues. Hercus, as Professor of Public Health and Bacteriology incorporated these projects into the curriculum in the early 1920s. The thesis uses many original papers published in scientific and medical journals by Hercus and his colleagues at the School. Building on a base of archival material including contemporary newspaper accounts, which have lately become available on the Papers Past website, this thesis draws together the individual disease studies undertaken by other thesis writers to give an account of Hercus’s achievements in fostering medical research. A key finding of this thesis is that Hercus was instrumental in building up the research capability of the School. He accomplished this through his own investigations and by helping to establish the New Zealand Medical Research Council. The thesis illustrates the multitude of studies that Hercus undertook personally or facilitated others to pursue, beyond the elimination of endemic goitre, for which he perhaps is best known. Another outcome of this study is an understanding of the difficult path that scientists faced in the early years of the twentieth century in New Zealand if they wished to carry out research. This thesis follows the origins of the Department of Scientific Research in the 1920s and the frequent collaboration Hercus made with scientists outside of the School. It will also demonstrate Hercus’s compassion and foresight in employing several Jewish refugee doctors as researchers at the School, who brought expertise into the research programmes. The era was one of great interest in improving the health and wellbeing of a generation affected by wars and deprivation caused by them. A key finding of this thesis is that researchers at the School took steps to mitigate these through making New Zealand more self-sufficient in foodstuffs and to improve the national diet. As well, Hercus lobbied for the establishment of a School of Physical Education within the university to improve the physical fitness of the population.

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Books on the topic "Goiter, Endemic"

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Dennison, John. Endemic Cretinism. Springer Science+Business Media, LLC, 2011.

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1951-, Rossbach M., ed. Total diet survey in goiter endemic areas in Indonesia. Forschungszentrum Jülich GmbH, 1995.

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José, Varea, Paredes Suárez Mario, Ochoa Bailón Carlos, and Fundación Ciencia para Estudios del Hombre y la Naturaleza (Ecuador), eds. Detección y significación de anomalías del desarrollo asociadas al bocio endémico: Informe ecuatoriano. Fundación Ciencia para Estudio del Hombre y la Naturaleza, 1987.

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Zhengzhou Shi (China). Wei sheng ju. Zhengzhou Shi si chong bing fang zhi zhi. Zhengzhou Shi wei sheng ju, Zhengzhou Shi ji kong zhong xin, 2002.

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International, Thyroid Symposium (1984 Budapest Hungary). Treatment of endemic and sporadic Goitre: International thryoid symposium, October 18-21, 1984, Budapest. Schattauer, 1985.

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1932-, Dunn John T., ed. Towards the eradication of endemic goiter, cretinism, and iodine deficiency: Proceedings of the V Meeting of the PAHO/WHO Technical Group on Endemic Goiter, Cretinism, and Iodine Deficiency. Pan American Health Organization, Pan American Sanitary Bureau, 1986.

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Lamberg, Bror-Axel. Kupukaula on jo menneisyyttä: Struuman historiaa Suomessa. Societas Scientiarum Fennica, 2003.

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National Workshop on the Control of Iodine Deficiency Disorders (IDD) (2nd 1985 Arusha, Tanzania). Towards the eradication of endemic goitre, cretinism, and iodine deficiency in Tanzania: Proceedings of the Second National Workshop on the Control of Iodine Deficiency Disorders (IDD), held at the Arusha International Conference Centre, November 18-20th, 1985. TFNC, 1988.

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Eduardo, Gaitan, ed. Environmental goitrogenesis. CRC Press, 1989.

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Riggs, Douglas S., John B. Stanbury, and Gordon L. Brownell. Endemic Goiter. Harvard University Press, 2014.

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Book chapters on the topic "Goiter, Endemic"

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Theisler, Charles. "Goiter (Endemic Goiter)." In Adjuvant Medical Care. CRC Press, 2022. http://dx.doi.org/10.1201/b22898-158.

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Sobrinho, L. G., and A. L. Oliveira. "Endemic Goiter in Portugal." In Iodine Deficiency in Europe. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1245-9_51.

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Kusić, Zvonko, Nikola Daković, François Delange, et al. "Endemic Goiter in Croatia." In Iodine Deficiency in Europe. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1245-9_54.

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Fernando, Ranil. "Management of Endemic Goiter." In Endocrine Surgery. CRC Press, 2021. http://dx.doi.org/10.1201/9780429197338-12.

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Hatemi, Husrev, and Irfan Urgancioglu. "Endemic Goiter and Iodine Deficiency in Turkey." In Iodine Deficiency in Europe. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1245-9_57.

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Szybiński, Z., and E. Mazurek. "Prevalence of medullary thyroid cancer in the endemic goiter area." In New Aspects in Thyroid Diseases, edited by H. F. Deckart and E. Strehlau. De Gruyter, 1992. http://dx.doi.org/10.1515/9783110874051-041.

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Huang, Gao-Sheng, and Yan-fang Liu. "Chronic Lymphocytic Thyroiditis in Endemic Goiter: Local Ig Production and Deposition." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_111.

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Langer, Pavel, Mária Tajtáková, Ján Podoba, et al. "Some Problems of Endemic Goiter in Czecho-Slovakia Yesterday, Today and Tomorrow." In Iodine Deficiency in Europe. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1245-9_44.

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Gerasimov, Gregory, Oleg Judenitch, and Ivan Dedov. "Iodine Deficiency Disorders and Endemic Goiter in the Commonwealth of Independent States (CIS)." In Iodine Deficiency in Europe. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1245-9_43.

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Joseph, K., U. Welcke, H. Höffken, H. W. Fritsch, T. Koppelberg, and M. Rothmund. "Imaging of Parathyroid Adenomas Using Technetium-99m-Sestamibi in an Endemic Goiter Area." In Radioactive Isotopes in Clinical Medicine and Research. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7340-6_65.

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Conference papers on the topic "Goiter, Endemic"

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Latifah, Leny, Yusi Dwi Nurcahyani, Suryati Kumorowulan, and Diah Yunitawati. "Iodine Deficiency Associated with Adolescent Cognitive Performance in Endemic Iodine Deficiency Endemic Area." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.05.24.

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ABSTRACT Background: Iodine deficiency is one of the most important public health problems in the world today, especially among children and pregnant women who are considered to be the highest-risk groups. These diseases hinder the socio-economic development of the affected areas. This study aimed to determine iodine deficiency associated with adolescent cognitive performance in endemic iodine deficiency endemic area. Subjects and Method: This is a cross-sectional study conducted in Bulu district, Temanggung. A sample of 120 adolescents range age 11-14 years old was selected by cluster sampling. The dependent variable was Cognitive performance. The independent variable was Iodine deficiency. The data were collected by questionnaire, measurement and assessment. This study was analyzed by multiple logistic regression. Results: On cognitive performance. Adolescent IQs are below average (69.2%), mathematics tests (64.2%), and Indonesian language tests are below average (47.5%). A total of (30.8%) adolescents showed insufficient iodine intake, goiter enlargement (30.2%), stunting (35%), and anemia (20.8%). The linear regression model on IQ showed goiter enlargement as a factor associated with decreased IQ, even after accounting for confounding factors (p = 0.013; R2 = 0.07). Subjects with goiter enlargement had lower mean IQ (Mean = 10.32). IQ was related to Indonesian achievement (p = 0.031; R2 = 0.13), while UIE (p = 0.031), father’s education (p = 0.011), and IQ (p <0.001) were identified as factors related to math test scores (R2 = 0.23). after accounting for confounding factors. Conclusion: Iodine deficiency is negatively associated with cognitive performance in areas of endemic infested iodine deficiency. Goiter enlargement, which is an indicator of long-term iodine deficiency status, correlates with IQ. UIE, which reflects current indicators of iodine deficiency status, affects academic achievement. The cognitive impact of iodine deficiency lasts into adolescence. Therefore, ensuring adequate iodine intake in adolescents is essential to optimize their cognitive performance. Keywords: cognitive performance, iodine deficiency, early adolescence, nutrition, nutrition Correspondence: Leny Latifah. Research and Development Center for Health, Magelang, Central Java, Indonesia. Email: lenylatifah1@gmail.com DOI: https://doi.org/10.26911/the7thicph.05.24

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Cilmiaty, Risya, Selfi Handayani, and Widia Susanti. "DENTAL MATURITY, ORAL HYGIENE AND HEIGHT OF JUNIOR HIGH SCHOOL STUDENTS IN GOITER ENDEMIC AREA IN KARANGANYAR REGENCY." In INTERNATIONAL CONFERENCE ON PUBLIC HEALTH. Graduate Studies in Public Health, Graduate Program, Sebelas Maret University Jl. Ir Sutami 36A, Surakarta 57126. Telp/Fax: (0271) 632 450 ext.208 First website:http//:s2ikm.pasca.uns.ac.id Second website: www.theicph.com. Email: theicph2016@gmail.com, 2016. http://dx.doi.org/10.26911/theicph.2016.059.

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"Boron and Silicon Content in the Hair of Schoolchildren with Endemic Goiter in Oil-Gas Producing Areas of West Kazakhstan." In International Institute of Chemical, Biological & Environmental Engineering. International Institute of Chemical, Biological & Environmental Engineering, 2015. http://dx.doi.org/10.15242/iicbe.c0615017.

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